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SMARCA5
A Computational Approach for Defining a Signature of Β-Cell Golgi Stress in Diabetes Mellitus
Loss of ISWI Atpase SMARCA5 (SNF2H) in Acute Myeloid Leukemia Cells Inhibits Proliferation and Chromatid Cohesion
PAX3-FOXO1 Candidate Interactors
Impaired SNF2L Chromatin Remodeling Prolongs Accessibility at Promoters Enriched for Fos/Jun Binding Sites and Delays Granule Neuron Differentiation
Downloaded Per Proteome Cohort Via the Web- Site Links of Table 1, Also Providing Information on the Deposited Spectral Datasets
KCNMA1 Expression Is Downregulated in Colorectal Cancer Via Epigenetic Mechanisms
Identifying Proteins Bound to Native Mitotic ESC Chromosomes Reveals Chromatin Repressors Are Important for Compaction
Blastic Plasmacytoid Dendritic Cell Neoplasm: Genomics Mark Epigenetic Dysregulation As a Primary Therapeutic Target
The Pdx1 Bound Swi/Snf Chromatin Remodeling Complex Regulates Pancreatic Progenitor Cell Proliferation and Mature Islet Β Cell
Analysis and Functional Evaluation of the Hair-Cell Transcriptome
Chromatin Remodeling in the UV-Induced DNA Damage Response
Histone Deacetylases 1 and 2 Maintain S-Phase Chromatin and DNA Replication Fork Progression Bhaskara Et Al
Snf2h Drives Chromatin Remodeling to Prime Upper Layer Cortical Neuron Development
A Broad Set of Chromatin Factors Influences Splicing
Scalable Pooled CRISPR Screens with Single-Cell Chromatin Accessibility Profiling
Cre-Mediated Deletion of SMARCA5 Disrupts Pluripotency in Mouse Embryonic Stem Cells David P
Systematic Identification of Factors Bound to Isolated Metaphase ESC Chromosomes Reveals a Role for Chromatin Repressors in Compaction
Chromatin Remodeling Factor BAZ1A Regulates Cellular Senescence in Both Cancer and Normal Cells
Top View
ISWI Atpase Smarca5 Regulates Differentiation of Thymocytes Undergoing Β -Selection
Proteome of Mouse Oocytes at Different Developmental Stages
Nucleosomes Around a Mismatched Base Pair Are Excluded Via an Msh2-Dependent Reaction with the Aid of SNF2 Family Atpase Smarcad1
The Emerging Roles of ATP-Dependent Chromatin Remodeling Complexes in Pancreatic Cancer
The ISWI Atpase Smarca5 (Snf2h) Is Required for Proliferation and Differentiation of Hematopoietic Stem and Progenitor Cells
Supporting Information
A Broad Set of Chromatin Factors Influences Splicing Eric Allemand, Michael P
Biochemical and Functional Studies of Histone Deacetylase 3 in Metabolic Tissues
Modeling Gene Regulation from Paired Expression and Chromatin Accessibility Data
Supplemental Table 1: Candidate Sgrna Screening Results
Correlated Gene Modules Uncovered by Single-Cell Transcriptomics with High Detectability and Accuracy Authors: Alec R
The Chromatin Remodeler Snf2h Is Essential for Oocyte Meiotic Cell Cycle Progression
SMARCA5 Fusion in Extraskeletal Ewing Sarcoma&Sol
Computational Chromosome Conformation Capture by Correlation of Chip-Seq at CTCF Motifs
Pathogenic Variants in SMARCA5, a Chromatin Remodeler, Cause a Range of Syndromic
MITF—The First 25 Years
ISWI Atpase Smarca5 Regulates Differentiation of Thymocytes Undergoing Β -Selection
Epigenetic Control of SPI1 Gene by CTCF and ISWI Atpase SMARCA5