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- Mutations in the Drosophila Homolog of Human PLA2G6 Give Rise to Age-Dependent Loss of Psychomotor Activity and Neurodegeneratio
- Intrafamilial Variability and Clinical Heterogeneity in a Family with PLA2G6-Associated Neurodegeneration
- Pla2g6 Mutations Associated with a Continuous Clinical Spectrum from Neuroaxonal Dystrophy to Hereditary Spastic Paraplegia
- The Impact of the Ca2+-Independent Phospholipase A2 (Ipla2) on Immune Cells
- Genetic Analysis in Neurology: the Next 10 Years
- Widespread Lewy Body and Tau Accumulation in Childhood and Adult Onset Dystonia-Parkinsonism Cases with PLA2G6 Mutations Coro Paisán-Ruiza,1, Abi Lia,B,1, Susanne A
- Identification of GPCR-Interacting Cytosolic Proteins Using HDL Particles and Mass Spectrometry-Based Proteomic Approach
- DIS3 Mutations in Multiple Myeloma Impact the Transcriptional Signature
- Migration of Thymic Pre-T Cells Regulates Development, Proliferation, and Heterogeneous Nuclear Ribonucleoprotein L Alternative
- Genetic Imaging of Neuroinflammation in Parkinson's Disease
- Infantile Neuroaxonal Dystrophy Caused by PLA2G6 Gene Mutation in a Chinese Patient: a Case Report
- Parkinson's Disease-Associated Ipla2-VIA/PLA2G6 Regulates Neuronal Functions and Α-Synuclein Stability Through Membrane Remod
- Kinetic Resolution of Ras and Arf Signaling Activation by Gefs on Lipid Membranes and in Live Cells
- Pla2g6-Associated Neurodegeneration (Plan)
- Anti-Phospholipase A2 (Ipla2) (SAB4200129)
- Nomenclature of Genetic Movement Disorders: Recommendations of the International Parkinson and Movement Disorder Society Task Force
- Downloaded on August 14, 2018 and on August 31, 2020)
- Figure S1. Amplification of PEDV M Gene and PCR Sequencing. (A) PCR Products of PEDV M Gene
- PLA2G6 Analysis for Infantile Neuroaxonal Dystrophy
- Hereditary Spastic Paraplegia: from Diagnosis to Emerging Therapeutic Approaches
- Figure S1. Kaplan‑Meier Plots Showing the Prognostic Values of ANLN and KDR in Different Sample Cohorts
- Cooperative Genome-Wide Analysis Shows Increased Homozygosity in Early Onset Parkinson’S Disease
- Infantile Neuroaxonal Dystrophy
- Implications for Melanoma Leonie Roos1,2,3, Johanna K
- Extensive Aggregation of Α-Synuclein and Tau in Juvenile-Onset
- Lipid Metabolism and Ferroptosis
- Metabolomics Reveals That Pparα Activation Protects Against Lithocholic Acid-Induced Liver Injury
- Pla2g6-Associated Neurodegeneration (Plan): Characterization of Patients and Drug Screening
- A Gene Therapy Approach for PLA2G6-Associated Neurodegeneration
- PLA2G6 Guards Placental Trophoblasts Against Ferroptotic Injury
- PARK14 PLA2G6 Mutants Are Defective in Preventing Rotenone- Induced Mitochondrial Dysfunction, ROS Generation and Activation of Mitochondrial Apoptotic Pathway
- PLA2G6 Gene Phospholipase A2 Group VI
- Parkinson's Disease-Associated Ipla2-VIA/PLA2G6 Regulates Neuronal
- Dissecting Molecular Mechanisms of Resistance to NOTCH1-Targeted Therapy in T-Cell Acute Lymphoblastic Leukemia Xenografts
- Proteome Profiling of Different Rat Brain Regions Reveals the Modulatory
- Title 1 Overexpressing PLA2G6 Mutations Cause Symptoms of Young–Onset Dystonia– 2 Parkinsonism Type 14 and Reduction In
- Genetic Ablation of PLA2G6 in Mice Leads to Cerebellar Atrophy Characterized by Purkinje Cell Loss and Glial Cell Activation