Emerin
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- A Complex LINC Between Nuclear Envelope and Pathology
- Desmoglein 2 Mutation Provokes Skeletal Muscle Actin Expression
- MKL1-Actin Pathway Restricts Chromatin Accessibility and Prevents Mature Pluripotency Activation
- Lamin A/C–Dependent Localization of Nesprin-2, a Giant Scaffolder at the Nuclear Envelope Thorsten Libotte,* Hafida Zaim,* Sabu Abraham,*† V
- Emerin and Emery-Dreifuss Muscular Dystrophy 2573
- Emerin Phosphorylation During the Early Phase of the Oxidative Stress Response Influences Emerin–BAF Interaction and BAF Nucle
- Supplementary Figure S 1. Experimental Workflow to Enrich
- An Emerin LEM-Domain Mutation Impairs Cell Response to Mechanical Stress
- Emerin and the Making of Muscle
- Emerin Represses STAT3 Signaling Through Nuclear Membrane-Based Spatial Control
- Risk Stratification in Laminopathies and Emery Dreifuss Muscular
- Loss of Emerin Alters Myogenic Signaling and Mirna Expression in Mouse Myogenic Progenitors
- Diagnosis of Cardiac Abnormalities in Muscular Dystrophies
- Rules for PP2A-Controlled Phosphosignalling and Drug Responses
- Responsive Nuclear Proteins in Collecting Duct Cells
- N-Terminal Nesprin-2 Variants Regulate Β-Catenin Signalling
- Dystroglycan-Emerin Interaction
- MAN1 and Emerin Have Overlapping Function(S) Essential for Chromosome Segregation and Cell Division in Caenorhabditis Elegans