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COG4
Congenital Disorders of Glycosylation from a Neurological Perspective
Revostmm Vol 10-4-2018 Ingles Maquetaciûn 1
ARP55242 P050) Data Sheet
Molecular Cytogenetic Characterization of Partial Monosomy 2P and Trisomy 16Q in a Newborn: a Case Report
Primepcr™Assay Validation Report
Cog5–Cog7 Crystal Structure Reveals Interactions Essential for the Function of a Multisubunit Tethering Complex
ARF Gtpases and Their Gefs and Gaps: Concepts and Challenges
Choudhury 22.11.2018 Suppl A
Identified Single-Nucleotide Polymorphisms and Haplotypes At
1 Genome-Wide CRISPR-Cas9 Screen Reveals Common Factors in Dsrna and Sindbis Virus
University of Groningen Fatal Outcome Due to Deficiency of Subunit 6
Anti-COG4 (C-Terminal) Produced in Rabbit, Affinity Isolated Antibody
HNRNPA1 Promotes Recognition of Splice Site Decoys by U2AF2 In
Novel Compound Heterozygous COG5 Mutations in a Chinese Male Patient with Severe Clinical Symptoms and Type Iii Congenital Disorder of Glycosylation: a Case Report
Bridging the Gap Between Glycosylation and Vesicle Traffic
Structural Basis for a Human Glycosylation Disorder Caused by Mutation of the COG4 Gene
Computational Modeling of Splicing Regulation
COG4 Gene Component of Oligomeric Golgi Complex 4
Top View
Golgi Function and Dysfunction in the First COG4- Deficient CDG Type II Patient
1 Genome-Wide CRISPR-Cas9 Screen Reveals the Importance of the Heparan Sulfate
You Can Check If Genes Are Captured by the Agilent Sureselect V5 Exome
Title: a Single Heterozygous Mutation in COG4 Disrupts Zebrafish Early Development Via Wnt
Chromosomal Mapping and Phenotypic Characterization of Hereditary Otosclerosis Linked to the OTSC4 Locus
Impact of On-Site Clinical Genetics Consultations on Diagnostic Rate In
University of California Santa Cruz
EGL Test Description
COG Complexes Form Spatial Landmarks for Distinct SNARE Complexes
Anti-COG5 (SAB4200440)
The Mosquito's Innate Sting
Anti-COG4 (SAB4200568)
Structural Variations of the 3D Genome Architecture in Cervical Cancer Development
Edinburgh Research Explorer a Recurrent De Novo Heterozygous COG4 Substitution Leads to Saul-Wilson Syndrome, Disrupted Vesicular Trafficking, And
The Close Relationship Between the Golgi Trafficking Machinery And
Structural Basis for a Human Glycosylation Disorder Caused by Mutation of the COG4 Gene
Golgi Glycosylation and Human Inherited Diseases
Mouse Cog4 Knockout Project (CRISPR/Cas9)