Related Study: Rehabilitation of The
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DERMATOLOGY – 15% Acne Vulgaris Inflammatory skin condition assoc. with papules & pustules involving pilosebaceous units Pathophysiology: • 4 main factors – follicular hyperkeratinization with plugging of sebaceous ducts, increased sebum production, Propionibacterium acnes overgrowth within follicles, & inflammatory response • Hormonal activation of pilosebaceous glands which may cause cyclic flares that coincide with menstruation Clinical Manifestations: • In areas with increased sebaceous glands (face, back, chest, upper arms) • Stage I: Comedones: small, inflammatory bumps from clogged pores - Open comedones (blackheads): incomplete blockage - Closed comedones (whiteheads): complete blockage • Stage II: Inflammatory: papules or pustules surrounded by inflammation • Stage III: Nodular or cystic acne: heals with scarring Differential Diagnosis: • Differentiate from rosacea which has no comedones** • Perioral dermatitis based on perioral and periorbital location • CS-induced acne lacks comedones and pustules are in same stage of development Diagnosis: • Mild: comedones, small amounts of papules &/or pustules • Moderate: comedones, larger amounts of papules &/or pustules • Severe: nodular (>5mm) or cystic Management: • Mild: topical – azelaic acid, salicylic acid, benzoyl peroxide, retinoids, Tretinoin topical (Retin A) or topical antibiotics [Clindamycin or Erythromycin with Benzoyl peroxide] • Moderate: above + oral antibiotics [Minocycline 50mg PO qd or Doxycycline 100 mg PO qd], spironolactone • Severe (refractory nodular acne): oral -
Physical Esxam
Pearls in the Musculoskeletal Exam Frank Caruso MPS, PA-C, EMT-P Skin, Bones, Hearts & Private Parts 2019 Examination Key Points • Area that needs to be examined, gown your patients - well exposed • Understand normal functional anatomy • Observe normal activity • Palpation • Range of Motion • Strength/neuro-vascular assessment • Special Tests General Exam Musculoskeletal Overview Physical Exam Preview Watch Your Patients Walk!! Inspection • Posture – Erectness – Symmetry – Alignment • Skin and subcutaneous tissues – Swelling – Redness – Masses Inspection • Extremities – Size – Deformities – Enlargement – Alignment – Contour – Symmetry Inspection • Muscles – Bilateral symmetry – Hypertrophy – Atrophy – Fasciculations – Spasms Palpation • Palpate bones, joints, and surrounding muscles for the following: – Heat – Tenderness – Swelling – Fluctuation – Crepitus – Resistance to pressure – Muscle tone Muscles • Size and strength affected by the following: – Genetics – Exercise – Nutrition • Muscles move joints through range of motion (ROM). Muscle Strength • Compare bilateral muscles – Strength – Symmetry – Equality – Resistance End Feel Think About It!! • The sensation the examiner feels in the joint as it reaches the end of the range of motion of each passive movement • Bone to bone: This is hard, unyielding – normal would be elbow extension. • Soft–tissue approximation: yielding compression that stops further movement – elbow and knee flexion. End Feel • Tissue stretch: hard – springy type of movement with a slight give – toward the end of range of motion – most common type of normal end feel : knee extension and metacarpophalangeal joint extension. Abnormal End Feel • Muscle spasm: invoked by movement with a sudden dramatic arrest of movement often accompanied by pain - sudden hard – “vibrant twang” • Capsular: Similar to tissue stretch but it does not occur where one would expect – range of motion usually reduced. -
Knee Pain in Children: Part I: Evaluation
Knee Pain in Children: Part I: Evaluation Michael Wolf, MD* *Pediatrics and Orthopedic Surgery, St Christopher’s Hospital for Children, Philadelphia, PA. Practice Gap Clinicians who evaluate knee pain must understand how the history and physical examination findings direct the diagnostic process and subsequent management. Objectives After reading this article, the reader should be able to: 1. Obtain an appropriate history and perform a thorough physical examination of a patient presenting with knee pain. 2. Employ an algorithm based on history and physical findings to direct further evaluation and management. HISTORY Obtaining a thorough patient history is crucial in identifying the cause of knee pain in a child (Table). For example, a history of significant swelling without trauma suggests bacterial infection, inflammatory conditions, or less likely, intra- articular derangement. A history of swelling after trauma is concerning for potential intra-articular derangement. A report of warmth or erythema merits consideration of bacterial in- fection or inflammatory conditions, and mechanical symptoms (eg, lock- ing, catching, instability) should prompt consideration of intra-articular derangement. Nighttime pain and systemic symptoms (eg, fever, sweats, night sweats, anorexia, malaise, fatigue, weight loss) are associated with bacterial infections, inflammatory conditions, benign and malignant musculoskeletal tumors, and other systemic malignancies. A history of rash or known systemic inflammatory conditions, such as systemic lupus erythematosus or inflammatory bowel disease, should raise suspicion for inflammatory arthritis. Ascertaining the location of the pain also can aid in determining the cause of knee pain. Anterior pain suggests patellofemoral syndrome or instability, quad- riceps or patellar tendinopathy, prepatellar bursitis, or apophysitis (patellar or tibial tubercle). -
Physical Examination of the Knee: Meniscus, Cartilage, and Patellofemoral Conditions
Review Article Physical Examination of the Knee: Meniscus, Cartilage, and Patellofemoral Conditions Abstract Robert D. Bronstein, MD The knee is one of the most commonly injured joints in the body. Its Joseph C. Schaffer, MD superficial anatomy enables diagnosis of the injury through a thorough history and physical examination. Examination techniques for the knee described decades ago are still useful, as are more recently developed tests. Proper use of these techniques requires understanding of the anatomy and biomechanical principles of the knee as well as the pathophysiology of the injuries, including tears to the menisci and extensor mechanism, patellofemoral conditions, and osteochondritis dissecans. Nevertheless, the clinical validity and accuracy of the diagnostic tests vary. Advanced imaging studies may be useful adjuncts. ecause of its location and func- We have previously described the Btion, the knee is one of the most ligamentous examination.1 frequently injured joints in the body. Diagnosis of an injury General Examination requires a thorough knowledge of the anatomy and biomechanics of When a patient reports a knee injury, the joint. Many of the tests cur- the clinician should first obtain a rently used to help diagnose the good history. The location of the pain injured structures of the knee and any mechanical symptoms were developed before the avail- should be elicited, along with the ability of advanced imaging. How- mechanism of injury. From these From the Division of Sports Medicine, ever, several of these examinations descriptions, the structures that may Department of Orthopaedics, are as accurate or, in some cases, University of Rochester School of have been stressed or compressed can Medicine and Dentistry, Rochester, more accurate than state-of-the-art be determined and a differential NY. -
Joint Range of Motion
JOINT RANGE OF MOTION In addition to joint integrity, adequate muscle length and other soft tissue extensibility must be The amount of motion available at a synovial joint maintained to optimize joint function.3 Most muscles is called the range of motion (ROM). Normal cross more than one joint to allow for shortening ROM varies among individuals and is influenced by over one joint and lengthening over the other during age, gender, body habitus, and whether motion is movement. However, there is potential for a muscle performed actively or passively.1 The type and amount or muscle group to become excessively lengthened of movement that occurs throughout the ROM is or shortened when it crosses more than one joint. If unique to each joint of the body and is dependent a muscle is rendered weak because it is shortened as primarily upon the shape of the articular surfaces. much as it can be as it crosses each joint, it is said to Other factors include the integrity and flexibility of be actively insufficient. An example of this occurs the periarticular soft tissues. when an individual lies prone with the hip extended Joint motion involves rotation or translation of or in neutral, and the individual is asked to perform one articular surface relative to the other about an a “leg curl” to flex the knee as much as possible. axis known as the instantaneous, helical, or screw axis. Active insufficiency occurs in the hamstrings in this Both rotation around the joint axis and translation example because they are shortened over the knee along the instantaneous axis must occur to provide and hip at the same time, and full flexion of the knee normal joint kinematics.2 Joint movements that are may be difficult. -
Neurologic Outcomes in Friedreich Ataxia: Study of a Single-Site Cohort E415
Volume 6, Number 3, June 2020 Neurology.org/NG A peer-reviewed clinical and translational neurology open access journal ARTICLE Neurologic outcomes in Friedreich ataxia: Study of a single-site cohort e415 ARTICLE Prevalence of RFC1-mediated spinocerebellar ataxia in a North American ataxia cohort e440 ARTICLE Mutations in the m-AAA proteases AFG3L2 and SPG7 are causing isolated dominant optic atrophy e428 ARTICLE Cerebral autosomal dominant arteriopathy with subcortical infarcts and leukoencephalopathy revisited: Genotype-phenotype correlations of all published cases e434 Academy Officers Neurology® is a registered trademark of the American Academy of Neurology (registration valid in the United States). James C. Stevens, MD, FAAN, President Neurology® Genetics (eISSN 2376-7839) is an open access journal published Orly Avitzur, MD, MBA, FAAN, President Elect online for the American Academy of Neurology, 201 Chicago Avenue, Ann H. Tilton, MD, FAAN, Vice President Minneapolis, MN 55415, by Wolters Kluwer Health, Inc. at 14700 Citicorp Drive, Bldg. 3, Hagerstown, MD 21742. Business offices are located at Two Carlayne E. Jackson, MD, FAAN, Secretary Commerce Square, 2001 Market Street, Philadelphia, PA 19103. Production offices are located at 351 West Camden Street, Baltimore, MD 21201-2436. Janis M. Miyasaki, MD, MEd, FRCPC, FAAN, Treasurer © 2020 American Academy of Neurology. Ralph L. Sacco, MD, MS, FAAN, Past President Neurology® Genetics is an official journal of the American Academy of Neurology. Journal website: Neurology.org/ng, AAN website: AAN.com CEO, American Academy of Neurology Copyright and Permission Information: Please go to the journal website (www.neurology.org/ng) and click the Permissions tab for the relevant Mary E. -
Tremor, Abnormal Movement and Imbalance Differential
Types of involuntary movements Tremor Dystonia Chorea Myoclonus Tics Tremor Rhythmic shaking of muscles that produces an oscillating movement Parkinsonian tremor Rest tremor > posture > kinetic Re-emergent tremor with posture Usually asymmetric Pronation-supination tremor Distal joints involved primarily Often posturing of the limb Parkinsonian tremor Other parkinsonian features Bradykinesia Rigidity Postural instability Many, many other motor and non- motor features Bradykinesia Rigidity Essential tremor Kinetic > postural > rest Rest in 20%, late feature, only in arms Intentional 50% Bringing spoon to mouth is challenging!! Mildly asymmetric Gait ataxia – typically mild Starts in the arms but can progress to neck, voice and jaw over time Jaw tremor occurs with action, not rest Neck tremor should resolve when patient is lying flat Essential tremor Many other tremor types Physiologic tremor Like ET but faster rate and lower amplitude Drug-induced tremor – Lithium, depakote, stimulants, prednisone, beta agonists, amiodarone Anti-emetics (phenergan, prochlorperazine), anti-psychotics (except clozapine and Nuplazid) Many other tremor types Primary writing tremor only occurs with writing Orthostatic tremor leg tremor with standing, improves with walking and sitting, causes imbalance Many other tremor types Cerebellar tremor slowed action/intention tremor Holmes tremor mid-brain lesion, unilateral Dystonia Dystonia Muscle contractions that cause sustained or intermittent torsion of a body part in a repetitive -
Direct Anterior Total Hip Arthroplasty Gait Biomechanics at Three and Six Months Post Surgery
DIRECT ANTERIOR TOTAL HIP ARTHROPLASTY GAIT BIOMECHANICS AT THREE AND SIX MONTHS POST SURGERY A THESIS SUBMITTED TO THE GRADUATE DIVISION OF THE UNIVERSITY OF HAWAI’I IN PARTIAL FULFILLMENT OF THE REQUIREMENTS FOR THE DEGREE OF MASTER OF SCIENCE IN KINESIOLOGY AND REHABILITATION SCIENCE AUGUST 2012 By: Ryan J. Moizon Thesis Committee: Iris Kimura, Chairperson Ronald Hetzler Christopher Stickley Keywords: Total hip arthroplasty; kinematics; kinetics TABLE OF CONTENTS List of Tables ii List of Figures iii Part I Introduction 1 Methods 4 Results 7 Discussion 12 Partil Review of literature 19 Appendix A: Data Collection Forms 37 Appendix B: Health History Form 40 Appendix C: WWB THA Informed Consent Form 42 Appendix D: WRB Control Informed Consent Form 53 Appendix F: Control Flyer 62 References 64 LIST OF TABLES Table Page 1. Demographic Data: Means and Standard Deviations for DA THA and Control group 7 2. Walking Velocity: Means and Standard Deviations for DA THA and Control group $ 3. Kinematic Variables: Mean and standard deviations for DA THA and Control group 9 4. Kinetic Variables: Mean and standard deviations for DA-THA and Control groups 11 5. Maximum VGRF: Means and Standard Deviations for DA THA and Control groups 11 LIST OF FIGURES Figure Page 1. Mean Values for Walking Velocity for DA THA and Control groups at initial test, 3 and 6 months post-test 13 2. Mean Values for Hip FlexionlExtension Excursion for DA THA and Control groups at initial test, 3 and 6 months post-test 14 3. Mean Values for Maximum VGRF for DA THA and Control groups at initial test, 3 and 6 months post-test 15 4. -
Children with Lower Limb Length Inequality
Children with lower limb length inequality The measurement of inequality. the timing of physiodesis and gait analysis H.I.H. Lampe ISBN 90-9010926-9 Although every effort has been made to accurately acknowledge sources of the photographs, in case of errors or omissions copyright holders arc invited to contact the author. Omslagontwcrp: Harald IH Lampe Druk: Haveka B.V., Alblasserdarn <!) All rights reserved. The publication of Ihis thesis was supported by: Stichling Onderwijs en Ondcrzoek OpJciding Orthopaedic Rotterdam, Stichting Anna-Fonds. Oudshoom B.V., West Meditec B.V., Ortamed B.Y .• Howmedica Nederland. Children with lower limb length inequality The measurement of inequality, the timing of physiodesis and gait analysis Kinderen met een beenlengteverschil Het meten van verschillen, het tijdstip van physiodese en gangbeeldanalyse. Proefschrift ter verkrijging van de graad van doctor aan de Erasmus Universiteit Rotterdam op gezag van de Rector Magnificus Prof. dr P.W.C. Akkermans M.A. en volgens besluit van het College voor Promoties. De openbare verdediging zal plaatsvinden op woen,dag 17 december 1997 om 11.45 uur door Harald Ignatius Hubertus Lampe geboren te Rotterdam. Promotieconmussie: Promotores: Prof. dr B. van Linge Prof. dr ir C.J. Snijders Overige leden: Prof. dr M. Meradji Prof. dr H.J. Starn Prof. dr J.A.N. Verbaar Dr. B.A. Swierstra, tevens co-promotor voor mijn ouders en Jori.nne Contents Chapter 1. Limb length inequality, the problems facing patient and doctor. 9 Review of Ii/era/ure alld aims of /he studies 1.0 Introduction 11 1.1 Etiology, developmental patterns and prediction of LLI 1.1.1 Etiology and developmental pattern 13 I. -
Use of Osteopathic Manipulative Treatment to Manage Compensated Trendelenburg Gait Caused by Sacroiliac Somatic Dysfunction
Editor’s Note: Corrections to this article were published in the March 2010 issue of JAOA—The Journal of the CASE REPORT American Osteopathic Association (2010;110[3]:210). The corrections have been incorporated in this online version of the article, which was posted January 2011. An expla - nation of these changes is available at http://www.jaoa .org/cgi/content /full/110/3/210-a. Use of Osteopathic Manipulative Treatment to Manage Compensated Trendelenburg Gait Caused by Sacroiliac Somatic Dysfunction Adam C. Gilliss, DO; Randel L. Swanson, II, OMS III; Deanna Janora, MD; and Venkat Venkataraman, PhD Gait dysfunctions are commonly encountered in the pri - In the present case report, we provide evidence that com - mary care setting. Compensated Trendelenburg gait is a gait pensated Trendelenburg gait may represent a secondary gait dysfunction that was originally described in patients with dysfunction stemming from somatic dysfunction of the weakness of ipsilateral hip abduction. This condition is sacroiliac joints. We also describe evidence of osteopathic thought to result from neuronal injury or myopathy. No manipulative treatment (OMT) resulting in quantitative treatment modalities currently exist for compensated Tren - improvements in the gait cycle. delenburg gait. The authors present a case in which osteo - pathic manipulative treatment may have improved a Tren - Traditional and Osteopathic Gait Theory delenburg gait dysfunction in a man aged 65 years with The gait cycle is divided into two main phases—stance and multiple sclerosis. Evidence of this improvement was swing, each consisting of numerous subphases. 2,3 Traditionally, obtained with the GaitMat II system for measuring the human gait cycle is considered to have six determinants that numerous gait parameters. -
The Trendelenburg Gait
A Personalised Monitoring and Recommendation Framework for Kinetic Dysfunctions: The Trendelenburg Gait Nikolaos Michalopoulos George E. Raptis Christina Katsini Moment Kinetics Moment Kinetics Moment Kinetics nmichalopoulos@ graptis@ ckatsini@ momentkinetics.com momentkinetics.com momentkinetics.com Theofilos Chrysikos Michail Mamalakis Andrew Vigotsky ECE Department ECE Department Biomedical Engineering Dept. University of Patras University of Patras Northwestern University [email protected] [email protected] [email protected] ABSTRACT bilitative perspective, it is possible to identify the risk of Kinetics and kinematics have been the focus of a lot of re-injury on an individual basis through the application of research throughout the years, and technological advances biomechanical kinematics and kinetics. The identification have contributed towards advanced monitoring and analysing of causality often requires precise analysis, as patients fre- of motion. Focusing on Trendelenburg Gait, an abnormal quently present a plethora of compensatory movement pat- gait, a personalisation framework for monitoring and eval- terns. Such compensatory deficits and impairments are often uating the movement is proposed in this paper based on a more noticeable than the underlying cause [15], making the number of factors, such as individuals' body composition, diagnosis process even more challenging. However, quantify- motion mechanisms, capturing process, analysis tools and ing the mobility state of a medical disorder and determining information -
Hip Arthroscopy PT Protocol
Arthroscopic Hip Surgery Physical Therapy Protocol The intent of this protocol is to provide guidelines for your patient’s therapy progression. It is not intended to serve as a recipe for treatment. We request that the PT/PTA/ATC use appropriate clinical decision- making skills when progressing a patient forward. Please call (833) 872-4477 to obtain the operative report from our office prior to the first post-op visit. Please contact our office if there are any questions about the protocol or your patient’s progression. Please keep in mind common problems that may arise following hip arthroscopy: hip flexor tendonitis, adductor tendonitis, sciatica/piriformis syndrome, ilial upslips and rotations, LB pain from QL hypertonicity and segmental vertebral rotational lesions. If you encounter any of these problems, please evaluate, assess, and treat as you feel appropriate, maintaining American Hip Institute’s precautions and guidelines at all times. Gradual progression is essential to avoid flare-ups. If a flare-up occurs, back off with therapeutic exercises until it subsides. Please reference the exercise progression sheet for timelines and use the following precautions during your treatments. Thank you for progressing all patients appropriately. Successful treatment requires a team approach, and the PT/PTA/ATC is a critical part of the team! Please contact AHI at any time with your input on how to improve the therapy protocol. Please send therapy progress notes and renewal therapy prescription requests with the patient or by fax to (630) 323-5625. Notes by fax must be sent 3 days prior to the patient’s visit to internally process this request.