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Oralmedicine
116 Test 98.2 ORAL MEDICINE Developmental Mandibular Salivary Gland Defect The Importance of Clinical Evaluation developmental mandibular salivary gland defect (also known as static A bone cyst, static bone defect, Stafne bone cavity, latent bone cyst, latent bone defect, idiopathic bone cavity, developmen- tal submandibular gland defect of the mandible, aberrant salivary gland defect in the mandible, and lingual mandibular bone Sako Ohanesian, concavity) is a deep, well-defined depression DDS in the lingual surface of the posterior body of the mandible. More precisely, the most common location is within the submandibu- lar gland fossa and often close to the inferi- or border of the mandible. In developmental bone defects investigated surgically, an aberrant lobe of the submandibular gland extends into the bony depression. First recognized by Dr. Edward Stafne in 1942, numerous cases of developmental mandibular salivary gland defect have since been reported, and the lesion should not be considered rare.1 In a study of 4963 pan- Most authorities now agree that this entity is a congenital defect, although it has rarely been observed in children and its precise anatomic nature is still uncertain. oramic images of adult patients, 18 cases of Figure 1. CT slices/panoramic views showing a well-defined radiolucent lesion in the right mandible. salivary gland depression were found by Karmiol and Walsh2, an incidence of nearly 0.4%. Most authorities now agree that this The margins of the radiolucent defect are around an extension of salivary tissue. This entity is a congenital defect, although it has well-defined by a dense radiopaque line. -
Odontogenic Keratocyst with Ameloblastomatous Dentistry Section Transformation: a Rare Case Report
Case Report DOI: 10.7860/JCDR/2020/43336.13636 Odontogenic Keratocyst with Ameloblastomatous Dentistry Section Transformation: A Rare Case Report METEHAN KESKIN1, NILÜFER ÖZKAN2, NIHAT AKBULUT3, MEHMET CIHAN BEREKET4 ABSTRACT Odontogenic Keratocysts (OKC) are a developmental odontogenic cysts arising from remnants of the dental lamina. They differ from other odontogenic cysts due to their aggressive growth behaviour and high recurrence rates. Malignant or benign transformation may develop from their epithelium. Ameloblastomatous transformation of OKC is an extremely rare case. Such lesions have been described as combined or hybrid odontogenic lesions. In this case report, a 22-year-old patient presented with an unusual lesion in the mandible showing histological features of both OKC and ameloblastoma, and review of the available literature regarding the combined lesions. Keywords: Combined lesion, Hybrid lesion, Marginal resection, Mandible CASE REPORT corrugated parakeratosis, approximately 4-6 cell layers and palisaded A systemically healthy 22-year-old male patient was referred to basal cell layer resembling the OKC [Table/Fig-2a]. Some areas Department of Oral and Maxillofacial Surgery, Faculty of Dentistry, inside the cyst wall showed stellate reticulum-like epithelial cells and Ondokuz Mayıs University, Turkey with painless swelling in the a basal cell layer of tall columnar cells with palisaded, revers polarised left lower jaw for 2 months. Three weeks before the first visit, the nuclei resembling the ameloblastomatous epithelium [Table/Fig-2b]. patient was prescribed antibiotics by another dental clinic because The lesion was diagnosed as Odontogenic Keratocyst (OKC) with of swelling in the left side of the jaw. On extraoral examination, a ameloblastomatous transformation. -
Peripheral Giant Cell Reparative Granuloma of Maxilla in a Patient with Aggressive Periodontitis
Peripheral Giant Cell Reparative Granuloma of Maxilla in a Patient with Aggressive Periodontitis E Cayci1, B Kan2, E Guzeldemir-Akcakanat1, B Muezzinoglu3 1Department of Periodontology, Kocaeli University, Faculty of Dentistry, Kocaeli, Turkey. 2Department of Oral and Maxillofacial Surgery, Kocaeli University, Faculty of Dentistry, Kocaeli, Turkey. 3Department of Pathology, Kocaeli University, School of Medicine, Kocaeli, Turkey. Abstract Peripheral giant cell reparative granuloma is a reactive and rare lesion of oral cavity with unknown etiology which is derived from periosteum and periodontal ligament and occurs frequently in young adults. Inflammation or trauma is underlying causative factor of reactive proliferation. In the present case report, a 35 year-old male with aggressive periodontitis and peripheral giant cell reparative granuloma is presented. The patient applied to our clinic with a complaining about a big nodule at his palate. The lesion was pedunculated and localized at his right maxilla between #16 and #17 which arose from distal aspect of #16, and the surface of the lesion was hyperkeratotic and the lesion was measured 22 x 30 mm at the largest diameter. He also had severe generalized aggressive periodontitis and hypertension. Amoxicillin clavulanate 625 mg, three times a day, metronidazole 500 mg three times a day and 0.2% chlorhexidine digluconate oral rinse, twice a day for a week, were prescribed to the patient. Then, scaling and root planing were performed along with systemic antibiotic treatment and he scheduled for surgery. The lesion was excised completely and #16 was extracted. After the healing period, periodontal surgery was planned for the treatment of aggressive periodontitis. Obtained tissue specimen was sent for histopathological examination. -
Iii Bds Oral Pathology and Microbiology
III BDS ORAL PATHOLOGY AND MICROBIOLOGY Theory: 120 Hours ORAL PATHOLOGY MUST KNOW 1. Benign and Malignant Tumours of the Oral Cavity (30 hrs) a. Benign tumours of epithelial tissue origin - Papilloma, Keratoacanthoma, Nevus b. Premalignant lesions and conditions: - Definition, classification - Epithelial dysplasia - Leukoplakia, Carcinoma in-situ, Erythroplakia, Palatal changes associated with reverse smoking, Oral submucous fibrosis c. Malignant tumours of epithelial tissue origin - Basal Cell Carcinoma, Epidermoid Carcinoma (Including TNM staging), Verrucous carcinoma, Malignant Melanoma. d. Benign tumours of connective tissue origin : - Fibroma, Giant cell Fibroma, Peripheral and Central Ossifying Fibroma, Lipoma, Haemangioma (different types). Lymphangioma, Chondroma, Osteoma, Osteoid Osteoma, Benign Osteoblastoma, Tori and Multiple Exostoses. e. Tumour like lesions of connective tissue origin : - Peripheral & Central giant cell granuloma, Pyogenic granuloma, Peripheral ossifying fibroma f. Malignant Tumours of Connective tissue origin : - Fibrosarcoma, Chondrosarcoma, Kaposi's Sarcoma Ewing's sarcoma, Osteosarcoma Hodgkin's and Non Hodgkin's L ymphoma, Burkitt's Lymphoma, Multiple Myeloma, Solitary Plasma cell Myeloma. g. Benign Tumours of Muscle tissue origin : - Leiomyoma, Rhabdomyoma, Congenital Epulis of newborn, Granular Cell tumor. h. Benign and malignant tumours of Nerve Tissue Origin - Neurofibroma & Neurofibromatosis-1, Schwannoma, Traumatic Neuroma, Melanotic Neuroectodermal tumour of infancy, Malignant schwannoma. i. Metastatic -
Oral Path Questions
Oral Pathology Oral Pathology • Developmental Conditions • Mucosal Lesions—Reactive • Mucosal Lesions—Infections • Mucosal Lesions—Immunologic Diseases • Mucosal Lesions—Premalignant • Mucosal Lesions—Malignant • CT Tumors—Benign • CT Tumors—Malignant • Salivary Gland Diseases—Reactive • Salivary Gland Diseases—Benign • Salivary Gland Diseases—Malignant • Lymphoid Neoplasms • Odontogenic Cysts • Odontogenic Tumors • Bone Lesions—Fibro-Osseous • Bone Lesions—Giant Cell • Bone Lesions—Inflammatory • Bone Lesions—Malignant • Hereditary Conditions #1 One of the primary etiologic agents of aphthous stomatitis is proposed to be: A. Cytomegalovirus B. Staphylococcus C. Herpes simplex D. Human leukocyte antigen E. Candidiasis #1 One of the primary etiologic agents of aphthous stomatitis is proposed to be: A. Cytomegalovirus B. Staphylococcus C. Herpes simplex D. Human leukocyte antigen E. Candidiasis #2 Intracellular viral inclusions are seen in tissue specimens of which of the following? A. Solar cheilitis B. Minor aphthous ulcers C. Geographic tongue D. Hairy leukoplaKia E. White sponge nevus #2 Intracellular viral inclusions are seen in tissue specimens of which of the following? A. Solar cheilitis B. Minor aphthous ulcers C. Geographic tongue D. Hairy leukoplakia E. White sponge nevus #3 Sjogren’s Syndrome has been linKed to which of the following malignancies? A. Leukemia B. Lymphoma C. Pleomorphic adenoma D. Osteosarcoma #3 Sjogren’s Syndrome has been linKed to which of the following malignancies? A. Leukemia B. Lymphoma C. Pleomorphic adenoma D. Osteosarcoma #4 Acantholysis, resulting from desmosome weaKening by autoantibodies directed against the protein desmoglein, is the disease mechanism attributed to which of the following? A. Epidermolysis bullosa B. Mucous membrane pemphigoid C. Pemphigus vulgaris D. Herpes simplex infections E. -
O-1 Human Radiation Dosimetry Using Electron Paramagnetic
O-1 Human Radiation Dosimetry Using Electron Paramagnetic Resonance in Tooth Enamel Biopsy Samples Barry Pass1), Alexander Romanyukha2), Tania De1), Lyudmila Romanyukha2), Francois Trompier3), Isabelle Clairand3), Prabhakar Misra1), Luis Benevides2), David Schauer4) 1)Howard University, Washington, DC, 2)Uniformed Services University of the Health Sciences, Bethesda, MD, 3)French Institute for Radiological Protection and Nuclear Safety, Fontenay-aux-roses, 4)National Council on Radiation Protection, Washington, DC e-mail: [email protected] Purposes: Dental enamel is the only living tissue that indefinitely maintains a record of its exposure to ionizing radiation. Electron paramagnetic resonance (EPR) dosimetry in tooth enamel has been applied for dose reconstruction for epidemiological studies of dif- ferent cohorts, including Hiroshima atomic bomb survivors, Chernobyl clean-up workers and other victims of unintended exposures to ionizing radiation. Several international inter-comparisons of EPR enamel dosimetry have demonstrated a high accuracy and reli- ability for this method. The main disadvantage of standard EPR enamel radiation dosimetry, however, is the necessity for large, 100 mg, enamel samples to achieve adequate signal-to-noise. This necessitates the use of extracted teeth for dose measurements, making the application of EPR in dental enamel for immediate, after-the-fact dosimetry problematic. The present study endeavored to improve the sensitivity of EPR measurements sufficiently to make the use of minimally-invasive in vivo enamel biopsies feasible for retrospective radiation dosimetry. Materials and methods: Enamel samples were obtained from teeth extracted in the normal course of dental treatment. Enamel biopsy samples of 2-4 mg in weight were obtained using a high-speed dental hand-piece with a tapered fissure or diamond bur, and an enamel chisel. -
Cryotherapy in the Treatment of Glandular
Cryotherapy in the treatment of glandular odontogenic cyst: case report and review Crioterapia no tratamento de cisto odontogênico glandular: relato de caso e revisão Milene Borges Campagnaro* Raquel Medeiros Farias* Roger Correa de Barros Berthold** Márcia Rejane Brücker*** Fábio Dal Moro Maito**** Claiton Heitz***** Objective: The Glandular Odontogenic Cyst (GOC) is Introduction a rare benign odontogenic lesion, of considerable ag- gression, and often incorrectly diagnosed. We present a Glandular Odontogenic Cyst (GOC) was first patient with a Glandular Odontogenic Cyst in the pos- described by Gardner in 1988 as a distinct clinical terior mandible, its evolution, treatment, and follow-up. pathologic entity, and it was included in the WHO Case report: A female patient, 45 years old, was referred histological typing of odontogenic tumors under to the Oral and Maxillofacial Surgery and Traumatology GOC or sialo-odontogenic cyst1-5. Division at Cristo Redentor Hospital, Porto Alegre, Bra- Glandular Odontogenic Cyst is a rare lesion, of zil, for the assessment of a painful edema on the right considerable aggressive behavior, originated at the hemiface. A unilocular area with well-defined borders 1-5 in the retromolar region, posterior to the third molar on areas of dental support . Clinically, the most affec- the right side of the mandible. The histopathological ted site is the anterior part of the mandible and it examination suggested GOC. Final considerations: The mostly occurs in middle-aged patients with a slight Glandular Odontogenic Cyst needs a complete clinical male prevalence2,4-8. Epidemiological features are assessment associated with image analyses, and espe- scarce due to the rarity of the lesion and a review in cially, with histopathology for the correct diagnosis of 2008 pointed 111 cases published in the literature6. -
Glandular Odontogenic Cyst: Case Series and Summary of the Literature
502 > CLINICAL REVIEW http://dx.doi.org/10.17159/2519-0105/2019/v74no9a6 Glandular odontogenic cyst: case series and summary of the literature SADJ October 2019, Vol. 74 No. 9 p502 - p507 F Opondo1, S Shaik2, J Opperman3, CJ Nortjé4 ABSTRACT The glandular odontogenic cyst (GOC) remains a Histologically, it may mimic any one of a dentigerous rare entity. It was initially named “sialo-odontogenic cyst, radicular cyst, surgical ciliated cyst, lateral perio- cyst” by Padayachee and Van Wyk in 1987 when dontal cyst or a botryoid odontogenic cyst. Importantly, they reported the first two cases. Thereafter the the features of a cystic lesion with squamous and term glandular odontogenic cyst was suggested mucous epithelial elements may cause it to be mis- by Gardner et al. in 1988 and was subsequently diagnosed as a central mucoepidermoid carcinoma. adopted by the WHO.1 With more comprehensive diagnostic criteria, at least 180 In addition to its rarity, it has non-pathognomonic cases have so far been reported in the English literature.4 clinical and radiological features and hence can It is therefore reasonable to assume that the previous mimic other lesions. Since its recognition as an rarity of this entity may be attributable to misdiagnosis. entity by the WHO in 1992, only two further cases of glandular odontogenic cyst have been seen at CASE 1 the authors’ institution and are hereby reported together with a summary of the review articles in A 60-year-old man presented at the diagnostic clinic at the English literature. Tygerberg Oral Health Centre with an asymptomatic swelling of the anterior mandible. -
Summer Journal 2007.Qxp 6/21/2007 9:56 AM Page 1
Summer Journal Cover 2007.qxp 6/21/2007 8:40 AM Page 1 Considerations for Treating the Patient with Scleroderma Summer Journal 2007.qxp 6/21/2007 9:56 AM Page 1 The Best in Dentistry Under One Roof New Location Boston Convention & Exhibition Center January 30 – February 3, 2008 Exhibits, January 31 – February 2 EDUCATION • EXHIBITS • EVENTS • EDUCATION • EXHIBITS • EVENTS Celebrity PROGRAM HIGHLIGHTS Entertainment Bruce Bavitz, DMD, Oral Surgery Sheryl Hal Crossley, DDS, Pharmacology Crow Jennifer de St. Georges, Practice Management FRIDAY Mel Hawkins, DDS, Pharmacology February 1, 2008 Kenneth Koch, DMD, and Dennis Brave, DDS, Endodontics Tickets go on sale Henry Lee, PhD, Forensics September 26, 2007, at 12 noon. John Molinari, PhD, Infection Control Anthony Sclar, DMD, Implants Jane Soxman, DDS, Pediatrics SCENIC SEAPORT Frank Spear, DDS, Restorative Jon Suzuki, DDS, Periodontics YDC HAS John Svirsky, DDS, Oral Pathology BOSTON’S BEST HOTEL . and many more of the best clinicians in dentistry! CHOICES DON’T MISS THESE Visit our Web site NEW PROGRAMS to view our housing blocks Las Vegas Institute of Advanced Dental Studies Medical/Dental Forum—The first program of its kind! BEAUTIFUL BACK BAY New Date! Housing & Registration Open September 26, 2007, at 12:00 noon EST VISIT WWW.YANKEEDENTAL.COM 800-342-8747 (MA) • 800-943-9200 (Outside MA) Summer Journal 2007.qxp 6/21/2007 9:57 AM Page 2 MASSACHUSETTS DENTAL SOCIETY Executive Director Robert E. Boose, EdD Senior Assistant Executive Director, Two Willow Street, Suite 200 Meeting Planning and Education Programs Southborough, MA 01745-1027 Michelle Curtin (508) 480-9797 • (800) 342-8747 • fax (508) 480-0002 Assistant Executive Director, Senior Policy Advisor www.massdental.org Karen Rafeld Chief Financial Officer Kathleen M. -
Oral Pathology Final Exam Review Table Tuanh Le & Enoch Ng, DDS
Oral Pathology Final Exam Review Table TuAnh Le & Enoch Ng, DDS 2014 Bump under tongue: cementoblastoma (50% 1st molar) Ranula (remove lesion and feeding gland) dermoid cyst (neoplasm from 3 germ layers) (surgical removal) cystic teratoma, cyst of blandin nuhn (surgical removal down to muscle, recurrence likely) Multilocular radiolucency: mucoepidermoid carcinoma cherubism ameloblastoma Bump anterior of palate: KOT minor salivary gland tumor odontogenic myxoma nasopalatine duct cyst (surgical removal, rare recurrence) torus palatinus Mixed radiolucencies: 4 P’s (excise for biopsy; curette vigorously!) calcifying odontogenic (Gorlin) cyst o Pyogenic granuloma (vascular; granulation tissue) periapical cemento-osseous dysplasia (nothing) o Peripheral giant cell granuloma (purple-blue lesions) florid cemento-osseous dysplasia (nothing) o Peripheral ossifying fibroma (bone, cartilage/ ossifying material) focal cemento-osseous dysplasia (biopsy then do nothing) o Peripheral fibroma (fibrous ct) Kertocystic Odontogenic Tumor (KOT): unique histology of cyst lining! (see histo notes below); 3 important things: (1) high Multiple bumps on skin: recurrence rate (2) highly aggressive (3) related to Gorlin syndrome Nevoid basal cell carcinoma (Gorlin syndrome) Hyperparathyroidism: excess PTH found via lab test Neurofibromatosis (see notes below) (refer to derm MD, tell family members) mucoepidermoid carcinoma (mixture of mucus-producing and squamous epidermoid cells; most common minor salivary Nevus gland tumor) (get it out!) -
1-1 Introduction the Oral Cavity Diseases Are a Medical Term Used
1-1 Introduction The oral cavity diseases are a medical term used to describe a patient who present with mouth pathology or mouth defect as there are numerous etiologies that can result in oral cavity diseases, prompt, accurate diagnoses is necessary to ensure proper patient management. The study includesalldental patientswho are undergoingscreeningOPGinsections ofdental x-raysin the city ofKhartoum, to assess theoral health through theimageresulting fromthisexaminationanddetermine thefeasibility ofthisexaminationin the diagnosis ofdiseases of the mouthand theknowledge ofthe relationship betweenfood habits of the patientandthe health ofhis mouth, andidentify waysbest fororal hygiene andto maintain his healthanddetermine the effect ofagingon the teethandgums In addition to studyingeffectsfor women. 1-2 Orthopantomogram (OPG) Orthopantogram is a panoramic scanning dental X-ray of the upper and lower jaw. It shows a two-dimensional view of a half-circle from ear to ear. Dental panoramic radiography equipment consists of a horizontal rotating arm which holds an X-ray source and a moving film mechanism (carrying a film) arranged at opposed extremities. The patient's skull sits between the X-ray generator and the film. The X-ray source is collimated toward the film, to give a beam shaped as a vertical blade having a width of 4-7mm when arriving on the film, after crossing the patient's skull. Also the height of that beam covers the mandibles and the maxilla regions .The arm moves and its movement may be described as a rotation around an instant center which shifts on a dedicated trajectory A large number of anatomical structures appear on an OPG: Soft tissue structures and air shadows: demonstrates the main soft tissue structures seen on an OPG, these are usually outlined by air within the nasopharynx and oropharynx. -
Adverse Effects of Medicinal and Non-Medicinal Substances
Benign? Not So Fast: Challenging Oral Diseases presented with DDX June 21st 2018 Dolphine Oda [email protected] Tel (206) 616-4748 COURSE OUTLINE: Five Topics: 1. Oral squamous cell carcinoma (SCC)-Variability in Etiology 2. Oral Ulcers: Spectrum of Diseases 3. Oral Swellings: Single & Multiple 4. Radiolucent Jaw Lesions: From Benign to Metastatic 5. Radiopaque Jaw Lesions: Benign & Other Oral SCC: Tobacco-Associated White lesions 1. Frictional white patches a. Tongue chewing b. Others 2. Contact white patches 3. Smoker’s white patches a. Smokeless tobacco b. Cigarette smoking 4. Idiopathic white patches Red, Speckled lesions 5. Erythroplakia 6. Georgraphic tongue 7. Median rhomboid glossitis Deep Single ulcers 8. Traumatic ulcer -TUGSE 9. Infectious Disease 10. Necrotizing sialometaplasia Oral Squamous Cell Carcinoma: Tobacco-associated If you suspect that a lesion is malignant, refer to an oral surgeon for a biopsy. It is the most common type of oral SCC, which accounts for over 75% of all malignant neoplasms of the oral cavity. Clinically, it is more common in men over 55 years of age, heavy smokers and heavy drinkers, more in males especially black males. However, it has been described in young white males, under the age of fifty non-smokers and non-drinkers. The latter group constitutes less than 5% of the patients and their SCCs tend to be in the posterior mouth (oropharynx and tosillar area) associated with HPV infection especially HPV type 16. The most common sites for the tobacco-associated are the lateral and ventral tongue, followed by the floor of mouth and soft palate area.