What Are Eating Disorders?
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The Impact of Trauma and Attachment on Eating Disorder Symptomology
Loma Linda University TheScholarsRepository@LLU: Digital Archive of Research, Scholarship & Creative Works Loma Linda University Electronic Theses, Dissertations & Projects 9-2014 The mpI act of Trauma and Attachment on Eating Disorder Symptomology Julie A. Hewett Follow this and additional works at: http://scholarsrepository.llu.edu/etd Part of the Clinical Psychology Commons Recommended Citation Hewett, Julie A., "The mpI act of Trauma and Attachment on Eating Disorder Symptomology" (2014). Loma Linda University Electronic Theses, Dissertations & Projects. 210. http://scholarsrepository.llu.edu/etd/210 This Dissertation is brought to you for free and open access by TheScholarsRepository@LLU: Digital Archive of Research, Scholarship & Creative Works. It has been accepted for inclusion in Loma Linda University Electronic Theses, Dissertations & Projects by an authorized administrator of TheScholarsRepository@LLU: Digital Archive of Research, Scholarship & Creative Works. For more information, please contact [email protected]. LOMA LINDA UNIVERSITY School of Behavioral Health in conjunction with the Faculty of Graduate Studies _______________________ The Impact of Trauma and Attachment on Eating Disorder Symptomology by Julie A. Hewett _______________________ A Dissertation submitted in partial satisfaction of the requirements for the degree Doctor of Philosophy in Clinical Psychology _______________________ September 2014 © 2014 Julie A. Hewett All Rights Reserved Each person whose signature appears below certifies that this dissertation in his/her opinion is adequate, in scope and quality, as a dissertation for the degree Doctor of Philosophy. , Chairperson Sylvia Herbozo, Assistant Professor of Psychology Jeffrey Mar, Assistant Clinical Professor, Psychiatry, School of Medicine Jason Owen, Associate Professor of Psychology David Vermeersch, Professor of Psychology iii ACKNOWLEDGEMENTS I would like to express my deepest gratitude to Dr. -
Posttraumatic Stress Disorder in Anorexia Nervosa
NIH Public Access Author Manuscript Psychosom Med. Author manuscript; available in PMC 2012 July 1. NIH-PA Author ManuscriptPublished NIH-PA Author Manuscript in final edited NIH-PA Author Manuscript form as: Psychosom Med. 2011 July ; 73(6): 491±497. doi:10.1097/PSY.0b013e31822232bb. Post traumatic stress disorder in anorexia nervosa Mae Lynn Reyes-Rodríguez, Ph.D.1, Ann Von Holle, M.S.1, T. Frances Ulman, Ph.D.1, Laura M. Thornton, Ph.D.1, Kelly L. Klump, Ph.D.2, Harry Brandt, M.D.3, Steve Crawford, M.D.3, Manfred M. Fichter, M.D.4, Katherine A. Halmi, M.D.5, Thomas Huber, M.D.6, Craig Johnson, Ph.D.7, Ian Jones, M.D.8, Allan S. Kaplan, M.D., F.R.C.P. (C)9,10,11, James E. Mitchell, M.D. 12, Michael Strober, Ph.D.13, Janet Treasure, M.D.14, D. Blake Woodside, M.D.9,11, Wade H. Berrettini, M.D.15, Walter H. Kaye, M.D.16, and Cynthia M. Bulik, Ph.D.1,17 1 Department of Psychiatry, University of North Carolina, Chapel Hill, NC 2 Department of Psychology, Michigan State University, East Lansing, MI 3 Department of Psychiatry, University of Maryland School of Medicine, Baltimore, MD 4 Klinik Roseneck, Hospital for Behavioral Medicine, Prien and University of Munich (LMU), Munich, Germany 5 New York Presbyterian Hospital-Westchester Division, Weill Medical College of Cornell University, White Plains, NY 6 Klinik am Korso, Bad Oeynhausen, Germany 7 Eating Recovery Center, Denver, CO 8 Department of Psychological Medicine, University of Birmingham, United Kingdom 9 Department of Psychiatry, The Toronto Hospital, Toronto, Canada 10 Center for -
Deinstitutionalization: Its Impact on Community Mental Health Centers and the Seriously Mentally Ill Stephen P
Page 40 Deinstitutionalization: Its Impact on Community Mental Health Centers and the Seriously Mentally Ill Stephen P. Kliewer Melissa McNally Robyn L. Trippany Walden University Abstract Deinstitutionalization has had a significant impact on the mental health system, including the client, the agency, and the counselor. For clients with serious mental illness, learning to live in a community setting poses challenges that are often difficult to overcome. Community mental health agencies must respond to these specific needs, thus requiring a shift in how services are delivered and how mental health counselors need to be trained. The focus of this article is to explore the dynamics and challenges specific to deinstitution- alization, discuss implications for counselors, and identify solutions to respond to the identified challenges and resulting needs. State run psychiatric hospitals have traditionally been the primary component in the treatment of people with severe and persistent mental illness. For many years, individuals with severe mental illness (SMI) were kept out of the community setting. This isolation occurred for many reasons: a) the attitude of the public about people with mental illness, b) a belief that the mentally ill could only be helped in such settings, and c) a lack of resources at the community level (Patrick, Smith, Schleifer, Morris & McClennon, 2006). However, the institutional approach was not without its problems. A primary problem was the absence of hope and expecta- tion that patients would recover (Patrick, et al., 2006). In short, institutions seemed to become warehouses where mentally ill were kept for long periods of time with little expectation of improvement. -
Eating Disorders: About More Than Food
Eating Disorders: About More Than Food Has your urge to eat less or more food spiraled out of control? Are you overly concerned about your outward appearance? If so, you may have an eating disorder. National Institute of Mental Health What are eating disorders? Eating disorders are serious medical illnesses marked by severe disturbances to a person’s eating behaviors. Obsessions with food, body weight, and shape may be signs of an eating disorder. These disorders can affect a person’s physical and mental health; in some cases, they can be life-threatening. But eating disorders can be treated. Learning more about them can help you spot the warning signs and seek treatment early. Remember: Eating disorders are not a lifestyle choice. They are biologically-influenced medical illnesses. Who is at risk for eating disorders? Eating disorders can affect people of all ages, racial/ethnic backgrounds, body weights, and genders. Although eating disorders often appear during the teen years or young adulthood, they may also develop during childhood or later in life (40 years and older). Remember: People with eating disorders may appear healthy, yet be extremely ill. The exact cause of eating disorders is not fully understood, but research suggests a combination of genetic, biological, behavioral, psychological, and social factors can raise a person’s risk. What are the common types of eating disorders? Common eating disorders include anorexia nervosa, bulimia nervosa, and binge-eating disorder. If you or someone you know experiences the symptoms listed below, it could be a sign of an eating disorder—call a health provider right away for help. -
Neural Correlates and Treatments Binge Eating Disorder
Running head: Binge Eating Disorder: Neural correlates and treatments Binge Eating Disorder: Neural correlates and treatments Bachelor Degree Project in Cognitive Neuroscience Basic level 22.5 ECTS Spring term 2019 Malin Brundin Supervisor: Paavo Pylkkänen Examiner: Stefan Berglund Running head: Binge Eating Disorder: Neural correlates and treatments Abstract Binge eating disorder (BED) is the most prevalent of all eating disorders and is characterized by recurrent episodes of eating a large amount of food in the absence of control. There have been various kinds of research of BED, but the phenomenon remains poorly understood. This thesis reviews the results of research on BED to provide a synthetic view of the current general understanding on BED, as well as the neural correlates of the disorder and treatments. Research has so far identified several risk factors that may underlie the onset and maintenance of the disorder, such as emotion regulation deficits and body shape and weight concerns. However, neuroscientific research suggests that BED may characterize as an impulsive/compulsive disorder, with altered reward sensitivity and increased attentional biases towards food cues, as well as cognitive dysfunctions due to alterations in prefrontal, insular, and orbitofrontal cortices and the striatum. The same alterations as in addictive disorders. Genetic and animal studies have found changes in dopaminergic and opioidergic systems, which may contribute to the severities of the disorder. Research investigating neuroimaging and neuromodulation approaches as neural treatment, suggests that these are innovative tools that may modulate food-related reward processes and thereby suppress the binges. In order to predict treatment outcomes of BED, future studies need to further examine emotion regulation and the genetics of BED, the altered neurocircuitry of the disorder, as well as the role of neurotransmission networks relatedness to binge eating behavior. -
Eating Disorders and Sexual Function Reviewed: a Trans-Diagnostic, Dimensional Perspective
Current Sexual Health Reports (2020) 12:1–14 https://doi.org/10.1007/s11930-020-00236-w CLINICAL THERAPEUTICS (B MCCARTHY, R SEGRAVES AND R BALON, SECTION EDITORS) Eating Disorders and Sexual Function Reviewed: A Trans-diagnostic, Dimensional Perspective Cara R. Dunkley1,2 & Yana Svatko1 & Lori A. Brotto2,3 Published online: 18 January 2020 # Springer Science+Business Media, LLC, part of Springer Nature 2020 Abstract Purpose of Review Clinical observation and a growing body of empirical research point to an association between disordered eating and sexual function difficulties. The present review identifies and connects the current knowledge on sexual dysfunction in the eating disorders, and provides a theoretical framework for conceptualizing the association between these important health conditions. Recent Findings Research on sexuality and eating pathology has focused on clinical samples of women with anorexia nervosa (AN) and bulimia nervosa (BN). All aspects of sexual response can be impacted in women with an eating disorder, with sexual function in women with AN appearing to be more compromised than in women with BN. Research of this nature is extremely limited with respect to BED, non-clinical samples, men, and individuals with non-binary gender identities. Summary Sexuality should be examined and addressed within the context of eating disorder treatment. Sexual dysfunction and eating disorders, along with commonly comorbid disorders of anxiety and mood, can be seen as separate but frequently overlapping manifestations of internalizing psychopathology. Psychological, developmental, sociocultural, etiological, and bio- physical factors likely represent risk and maintenance factors for internalizing disorders. A dimensional, trans-diagnostic ap- proach to disordered eating and sexuality has promising implications for future research and clinical interventions. -
A Case–Control Study Investigating Food Addiction in Parkinson Patients Ingrid De Chazeron1*, F
www.nature.com/scientificreports OPEN A case–control study investigating food addiction in Parkinson patients Ingrid de Chazeron1*, F. Durif2, C. Lambert3, I. Chereau‑Boudet1, M. L. Fantini4, A. Marques2, P. Derost2, B. Debilly2, G. Brousse1, Y. Boirie5,6 & P. M. Llorca1,2,3,4,5,6 Eating disorders (EDs) in patients with Parkinson’s disease (PD) are mainly described through impulse control disorders but represent one end of the spectrum of food addiction (FA). Although not formally recognized by DSM‑5, FA is well described in the literature on animal models and humans, but data on prevalence and risk factors compared with healthy controls (HCs) are lacking. We conducted a cross‑ sectional study including 200 patients with PD and 200 age‑ and gender‑matched HCs. Characteristics including clinical data (features of PD/current medication) were collected. FA was rated using DSM‑5 criteria and the Questionnaire on Eating and Weight Patterns‑Revised (QEWP‑R). Patients with PD had more EDs compared to HCs (27.0% vs. 13.0%, respectively, p < 0.001). They mainly had FA (24.5% vs. 12.0%, p = 0.001) and night eating syndrome (7.0% vs. 2.5% p = 0.03). In PD patients, FA was associated with female gender (p = 0.04) and impulsivity (higher attentional non‑planning factor) but not with the dose or class of dopaminergic therapy. Vigilance is necessary, especially for PD women and in patients with specifc impulsive personality traits. Counterintuitively, agonist dopaminergic treatment should not be used as an indication for screening FA in patients with PD. In Parkinson’s disease (PD), compulsive eating is part of the spectrum of impulse control disorders (ICDs) that also include pathologic gambling, compulsive buying and hypersexuality1. -
Standpoints on Psychiatric Deinstitutionalization Alix Rule Submitted in Partial Fulfillment of the Requirements for the Degre
Standpoints on Psychiatric Deinstitutionalization Alix Rule Submitted in partial fulfillment of the requirements for the degree of Doctor of Philosophy in the Graduate School of Arts and Sciences COLUMBIA UNIVERSITY 2018 © 2018 Alix Rule All rights reserved ABSTRACT Standpoints on Psychiatric Deinstitutionalization Alix Rule Between 1955 and 1985 the United States reduced the population confined in its public mental hospitals from around 600,000 to less than 110,000. This dissertation provides a novel analysis of the movement that advocated for psychiatric deinstitutionalization. To do so, it reconstructs the unfolding setting of the movement’s activity historically, at a number of levels: namely, (1) the growth of private markets in the care of mental illness and the role of federal welfare policy; (2) the contested role of states as actors in driving the process by which these developments effected changes in the mental health system; and (3) the context of relevant events visible to contemporaries. Methods of computational text analysis help to reconstruct this social context, and thus to identify the closure of key opportunities for movement action. In so doing, the dissertation introduces an original method for compiling textual corpora, based on a word-embedding model of ledes published by The New York Times from 1945 to the present. The approach enables researchers to achieve distinct, but equally consistent, actor-oriented descriptions of the social world spanning long periods of time, the forms of which are illustrated here. Substantively, I find that by the early 1970s, the mental health system had disappeared from public view as a part of the field of general medicine — and with it a target around which the existing movement on behalf of the mentally ill might have effectively reorganized itself. -
Why Do Autistic Women Develop Restrictive Eating Disorders?
Why do autistic women develop restrictive eating disorders? Exploring social risk factors Hannah Baker D.Clin.Psy. thesis (Volume 1), 2020 University College London 2 UCL Doctorate in Clinical Psychology Thesis declaration form I confirm that the work presented in this thesis is my own. Where information has been derived from other sources, I confirm that this has been indicated in the thesis. Signature: Name: Hannah Baker Date: 19/06/2020 3 Overview This thesis seeks to understand why women with Autism Spectrum Disorder (‘autism’) are more likely to develop restrictive eating disorders (‘REDs’). Part 1 is a conceptual introduction exploring the wider topic. To start, I introduce key terms and explore current understanding of autism among females, including the notion of ‘social camouflaging’; the masking of autistic traits and imitation of social behaviours, common among autistic women. Next, I discuss issues around prevalence and diagnosis of autism among those with REDs and the experience of eating disorder treatment for autistic individuals. Finally, a comprehensive review of the literature, outlining the multiple factors which might increase the likelihood of autistic women developing REDs, is presented. Part 2 documents an empirical study investigating the specific role of social risk factors for autistic women with REDs. It is hypothesised that difficulties gaining acceptance from others increases the likelihood of autistic women to perceive themselves as inferior. Moreover, that autistic women who attempt to ‘fit in’ through social camouflaging, are more vulnerable to such risk factors. Two groups of autistic women, with and without REDs, are compared on measures of social comparison, submissive behaviour, fear of negative evaluation and social camouflaging. -
Reading List
Psychiatric Epidemiology Dr. William W. Eaton Textbook Tsuang MT & Tohen M. Textbook in Psychiatric Epidemiology. 2nd ed. New York, Wiley-Liss, 2002. Available online at: http://www3.interscience.wiley.com/cgi-bin/booktoc/104527540 Session 1 Topic: Introduction, Nosology, and History of Psychiatric Epidemiology Assigned Readings: Diagnostic and Statistical Manual of Mental Disorders (DSM-IV). 4th ed. Washington, DC, APA Introduction, XV-XXV. Use of the manual, 1-11. Multiaxial Assessment, 25-35. Available online at: http://online.statref.com/TOC.aspx?grpalias=JHU&FxId=37 Recommended Additional Readings: o Diagnostic and Statistical Manual of Mental Disorders (DSM-IV). 4th ed. Washington, DC, APA. o Choose any other single chapter on mental disorders. Session 2 Topic: Measurement of Psychopathology in Populations Assigned Readings: Tsuang, M.T.& Cohen, M. (2002). Textbook in Psychiatric Epidemiology. 2nd ed. Wiley- Liss. Ch12. Murphy JM. Symptom scales and diagnostic schedules in adult psychiatry. 273-332. Recommended Additional Readings: Choose one Tsuang, M.T.& Cohen, M. (2002). Textbook in Psychiatric Epidemiology. 2nd ed. Wiley- Liss. o Ch 9. Eaton, WW. Studying the Natural History of Psychopathology. 215-238. o Ch 11. Robins, LN. Birth and development of psychiatric interviews. 257-272. o Ch 14. Kessler RC and Walters E. The National Comorbidity Survey. 343-362. Session 3 Topic: Epidemiology of Anxiety Disorders Assigned Readings: Tsuang, M.T.& Cohen, M. (2002). Textbook in Psychiatric Epidemiology. 2nd ed. Wiley- Liss. Ch16. Horwath E, Cohen RS, Weissman MM. Epidemiology of depressive and anxiety disorders. 389-426 Recommended Additional Readings: Choose one o Eaton, W.W. (1995) Progress in the epidemiology of anxiety disorders. -
Two Cases of Food Aversion with Semantic Dementia
View metadata, citation and similar papers at core.ac.uk brought to you by CORE provided by UCL Discovery Neurocase The Neural Basis of Cognition ISSN: 1355-4794 (Print) 1465-3656 (Online) Journal homepage: http://www.tandfonline.com/loi/nncs20 Two cases of food aversion with semantic dementia Alexandra E. Thompson, Camilla N. Clark, Christopher J. Hardy, Phillip D. Fletcher, John Greene, Jonathan D. Rohrer & Jason D. Warren To cite this article: Alexandra E. Thompson, Camilla N. Clark, Christopher J. Hardy, Phillip D. Fletcher, John Greene, Jonathan D. Rohrer & Jason D. Warren (2016): Two cases of food aversion with semantic dementia, Neurocase, DOI: 10.1080/13554794.2016.1149592 To link to this article: http://dx.doi.org/10.1080/13554794.2016.1149592 © 2016 The Author(s). Published by Taylor & Francis. Published online: 10 Mar 2016. Submit your article to this journal Article views: 184 View related articles View Crossmark data Full Terms & Conditions of access and use can be found at http://www.tandfonline.com/action/journalInformation?journalCode=nncs20 Download by: [University of London] Date: 02 June 2016, At: 06:53 NEUROCASE, 2016 http://dx.doi.org/10.1080/13554794.2016.1149592 Two cases of food aversion with semantic dementia Alexandra E. Thompsona,b, Camilla N. Clarka, Christopher J. Hardya, Phillip D. Fletchera, John Greenec, Jonathan D. Rohrera and Jason D. Warrena aDementia Research Centre, UCL Institute of Neurology, University College London, London, UK; bMedical School, University of Adelaide, Adelaide, Australia; cSouthern General Hospital, Glasgow, UK ABSTRACT ARTICLE HISTORY Accounts of altered eating behavior in semantic dementia generally emphasize gluttony and abnormal Received 3 October 2015 food preferences. -
Clinically Useful Brain Imaging for Neuropsychiatry: How Can We Get There?
bioRxiv preprint doi: https://doi.org/10.1101/115097; this version posted March 9, 2017. The copyright holder for this preprint (which was not certified by peer review) is the author/funder, who has granted bioRxiv a license to display the preprint in perpetuity. It is made available under aCC-BY 4.0 International license. Clinically Useful Brain Imaging for Neuropsychiatry: How Can We Get There? Michael P. Milham1,2, R. Cameron Craddock1,2 and Arno Klein1 1Center for the Developing Brain, Child Mind Institute, New York, NY 2Center for Biomedical Imaging and Neuromodulation, Nathan S. Kline Institute for Psychiatric Research, New York, NY Corresponding Author: Michael P. Milham, MD, PhD Phyllis Green and Randolph Cowen Scholar Child Mind Institute 445 Park Avenue New York, NY 10022 [email protected] bioRxiv preprint doi: https://doi.org/10.1101/115097; this version posted March 9, 2017. The copyright holder for this preprint (which was not certified by peer review) is the author/funder, who has granted bioRxiv a license to display the preprint in perpetuity. It is made available under aCC-BY 4.0 International license. Abstract Despite decades of research, visions of transforming neuropsychiatry through the development of brain imaging-based ‘growth charts’ or ‘lab tests’ have remained out of reach. In recent years, there is renewed enthusiasm about the prospect of achieving clinically useful tools capable of aiding the diagnosis and management of neuropsychiatric disorders. The present work explores the basis for this enthusiasm. We assert that there is no single advance that currently has the potential to drive the field of clinical brain imaging forward.