Rsm Aesthetics 9 Table of Contents
Total Page:16
File Type:pdf, Size:1020Kb
Load more
Recommended publications
-
J.M. Azaña Defez, M.L. Martínez Martínez Doctors in Medicine and Surgery
Acne J.M. Azaña Defez, M.L. Martínez Martínez Doctors in Medicine and Surgery. Consultant Physicians at the Dermatology Service. Pediatric Dermatology Unit. University Hospital Complex of Albacete Abstract Resumen Acne is a chronic inflammatory skin disease of El acné es una enfermedad cutánea inflamatoria the pilosebaceous unit of multifactorial etiology crónica del folículo pilosebáceo, de origen characterized by increased sebaceous secretion, multifactorial, caracterizada por: aumento de la comedone formation, inflammatory lesions and secreción sebácea, formación de comedones, lesiones risk of scarring sequelae. It is undoubtedly one inflamatorias y riesgo de secuelas cicatrizales. of the most frequent dermatological processes Es, sin duda, uno de los procesos dermatológicos in the daily clinical practice, especially in más frecuentes en la práctica clínica diaria, adolescence, although it can also appear in especialmente en la adolescencia, aunque también childhood and persist into adulthood. Adequate puede aparecer en niños y persistir en la edad management of this pathology is relevant, adulta. Es importante un manejo adecuado de esta as it can cause lower self-esteem and social patología, que puede producir una disminución de la dysfunction in patients, with the subsequent autoestima y disfunción social de los pacientes, con impact on quality of life. el consiguiente impacto en la calidad de vida. Key words: Cutibacterium acnes; Grading and classification of acne; Acne management; Isotretinoin. Palabras clave: Acné; Cutibacterium acnes; Graduación y clasificación del acné; Manejo del acné; Isotretinoina. Introduction Etiopathogenesis acne among the Spanish population Acne is a frequent inflammatory skin aged 12 to 18 years is 74%, without Acne is a multifactorial disease, produced disease of chronic course and polymor- significant differences regarding sex by: increased sebaceous secretion, follicu- phous in its clinical expression. -
Prospective Observational Study in a Tertiary
CLINICAL STUDY OF PROFILE OF ADOLESCENT DERMATOSES AND THEIR EFFECT ON QUALITY OF LIFE IN ADOLESCENTS – PROSPECTIVE OBSERVATIONAL STUDY IN A TERTIARY CARE HOSPITAL IN SOUTH INDIA Dissertation Submitted to THE TAMILNADU DR.M.G.R. MEDICAL UNIVERSITY IN PARTIAL FULFILMENT FOR THE AWARD OF THE DEGREE OF DOCTOR OF MEDICINE IN DERMATOLOGY, VENEREOLOGY & LEPROSY Register No.: 201730251 BRANCH XX MAY 2020 DEPARTMENT OF DERMATOLOGY VENEREOLOGY & LEPROSY TIRUNELVELI MEDICAL COLLEGE TIRUNELVELI -11 BONAFIDE CERTIFICATE This is to certify that this dissertation entitled “CLINICAL STUDY OF PROFILE OF ADOLESCENT DERMATOSES AND THEIR EFFECT ON QUALITY OF LIFE IN ADOLESCENTS – PROSPECTIVE OBSERVATIONAL STUDY IN A TERTIARY CARE HOSPITAL IN SOUTH INDIA” is a bonafide research work done by Dr.ARAVIND BASKAR.M, Postgraduate student of Department of Dermatology, Venereology and Leprosy, Tirunelveli Medical College during the academic year 2017 – 2020 for the award of degree of M.D. Dermatology, Venereology and Leprosy – Branch XX. This work has not previously formed the basis for the award of any Degree or Diploma. Dr.P.Nirmaladevi.M.D., Professor & Head of the Department Department of DVL Tirunelveli Medical College, Tirunelveli - 627011 Dr.S.M.Kannan M.S.Mch., The DEAN Tirunelveli Medical College, Tirunelveli - 627011 CERTIFICATE This is to certify that the dissertation titled as “CLINICAL STUDY OF PROFILE OF ADOLESCENT DERMATOSES AND THEIR EFFECT ON QUALITY OF LIFE IN ADOLESCENTS – PROSPECTIVE OBSERVATIONAL STUDY IN A TERTIARY CARE HOSPITAL IN SOUTH INDIA” submitted by Dr.ARAVIND BASKAR.M is a original work done by him in the Department of Dermatology,Venereology & Leprosy,Tirunelveli Medical College,Tirunelveli for the award of the Degree of DOCTOR OF MEDICINE in DERMATOLOGY, VENEREOLOGY AND LEPROSY during the academic period 2017 – 2020. -
Seminar ICLAD 2019 Classification of Silicone Foreign Body Reaction.Pdf
SIMPOSIUM Day/ Date: Friday, 26th 2019 Time Schedule Speaker Time Schedule Speaker 07.30- Registration 08.00 Session I: Moderator: David Session II: Wound Moderator: Larisa Fundamentals of Sudarto Oeiria, Dr. Healing in Paramitha Laser & Other Sp.KK, FINSDV, Dermatologic Wibawa, Dr. Sp.KK, Energy Based FAADV Procedure FINSDV Devices: Update 08.00- Biophysics of Laser David Sudarto 08.00- Basic Wound Theresia L. Toruan, 08.20 and Other Energy Oeiria, Dr. Sp.KK, 08.15 Healing in Reducing Sp.KK(K), Prof. Dr. Based Devices FINSDV, FAADV Scar FINSDV, FAADV 08.20- Skin Conditioning Aryani Sudharmono, 08.15- Silicone Gel Usage in M. Akbar 08.40 Regimen for Laser Dr. Sp.KK(K), 08.30 Invasive Wedyadhana, Dr. and Other Energy FINSDV, FAADV Dermatologic Sp.KK, FINSDV Based Devices Procedure Treatment 08.40- Discussion 08.30- Silicone Gel for Yuli Kurniawati, DR. 08.50 08.45 Keloid and Dr. Sp.KK(K), Hypertrophic Scar FINSDV, FAADV 08.45- Discussion 08.50 Time Schedule Speaker Schedule Speaker Plenary Session I Moderator: Made Swastika Adiguna, Prof. Co moderator: Luh Made Mas Rusyati, DR . Dr. Dr. Sp.KK(K), FINSDV, FAADV Sp.KK(K), FINSDV 08.50- To be confirmed To be confirmed 09.10 09.10- Dermatologic Surgery: The History, Present, Lawrence M. Field, Prof. MD 09.40 and Future 09.40- Lillis Tumescent Anesthesia Patrick J. Lilis, MD 10.10 10.10- Discussion 10.20 10.20- Opening Welcome Speech: 10.30 1. Chairman of ICLAD 2.Chairman of PERDOSKI 10.30- Coffee Break 10.45 Sesi III: Stem Cell Moderator: Session IV: Moderator: Made and Growth Factor: Amaranila Lalita Malignancy Wardhana, DR. -
8.5 X12.5 Doublelines.P65
Cambridge University Press 978-0-521-87409-0 - Modern Soft Tissue Pathology: Tumors and Non-Neoplastic Conditions Edited by Markku Miettinen Index More information Index abdominal ependymoma, 744 mucinous cystadenocarcinoma, 631 adult fibrosarcoma (AF), 364–365, 1026 abdominal extrauterine smooth muscle ovarian adenocarcinoma, 72, 79 adult granulosa cell tumor, 523–524 tumors, 79 pancreatic adenocarcinoma, 846 clinical features, 523 abdominal inflammatory myofibroblastic pulmonary adenocarcinoma, 51 genetics, 524 tumors, 297–298 renal adenocarcinoma, 67 pathology, 523–524 abdominal leiomyoma, 467, 477 serous cystadenocarcinoma, 631 adult rhabdomyoma, 548–549 abdominal leiomyosarcoma. See urinary bladder/urogenital tract clinical features, 548 gastrointestinal stromal tumor adenocarcinoma, 72, 401 differential diagnosis, 549 (GIST) uterine adenocarcinomas, 72 genetics, 549 abdominal perivascular epithelioid cell tumors adenofibroma, 523 pathology, 548–549 (PEComas), 542 adenoid cystic carcinoma, 1035 aggressive angiomyxoma (AAM), 514–518 abdominal wall desmoids, 244 adenomatoid tumor, 811–813 clinical features, 514–516 acquired elastotic hemangioma, 598 adenomatous polyposis coli (APC) gene, 143 differential diagnosis, 518 acquired tufted angioma, 590 adenosarcoma (mullerian¨ adenosarcoma), 523 genetics, 518 acral arteriovenous tumor, 583 adipocytic lesions (cytology), 1017–1022 pathology, 516 acral myxoinflammatory fibroblastic sarcoma atypical lipomatous tumor/well- aggressive digital papillary adenocarcinoma, (AMIFS), 365–370, 1026 differentiated -
2016 Essentials of Dermatopathology Slide Library Handout Book
2016 Essentials of Dermatopathology Slide Library Handout Book April 8-10, 2016 JW Marriott Houston Downtown Houston, TX USA CASE #01 -- SLIDE #01 Diagnosis: Nodular fasciitis Case Summary: 12 year old male with a rapidly growing temple mass. Present for 4 weeks. Nodular fasciitis is a self-limited pseudosarcomatous proliferation that may cause clinical alarm due to its rapid growth. It is most common in young adults but occurs across a wide age range. This lesion is typically 3-5 cm and composed of bland fibroblasts and myofibroblasts without significant cytologic atypia arranged in a loose storiform pattern with areas of extravasated red blood cells. Mitoses may be numerous, but atypical mitotic figures are absent. Nodular fasciitis is a benign process, and recurrence is very rare (1%). Recent work has shown that the MYH9-USP6 gene fusion is present in approximately 90% of cases, and molecular techniques to show USP6 gene rearrangement may be a helpful ancillary tool in difficult cases or on small biopsy samples. Weiss SW, Goldblum JR. Enzinger and Weiss’s Soft Tissue Tumors, 5th edition. Mosby Elsevier. 2008. Erickson-Johnson MR, Chou MM, Evers BR, Roth CW, Seys AR, Jin L, Ye Y, Lau AW, Wang X, Oliveira AM. Nodular fasciitis: a novel model of transient neoplasia induced by MYH9-USP6 gene fusion. Lab Invest. 2011 Oct;91(10):1427-33. Amary MF, Ye H, Berisha F, Tirabosco R, Presneau N, Flanagan AM. Detection of USP6 gene rearrangement in nodular fasciitis: an important diagnostic tool. Virchows Arch. 2013 Jul;463(1):97-8. CONTRIBUTED BY KAREN FRITCHIE, MD 1 CASE #02 -- SLIDE #02 Diagnosis: Cellular fibrous histiocytoma Case Summary: 12 year old female with wrist mass. -
Management of Acne
Review CMAJ Management of acne John Kraft MD, Anatoli Freiman MD cne vulgaris has a substantial impact on a patient’s Key points quality of life, affecting both self-esteem and psychoso- cial development.1 Patients and physicians are faced • Effective therapies for acne target one or more pathways A in the pathogenesis of acne, and combination therapy with many over-the-counter and prescription acne treatments, gives better results than monotherapy. and choosing the most effective therapy can be confusing. • Topical therapies are the standard of care for mild to In this article, we outline a practical approach to managing moderate acne. acne. We focus on the assessment of acne, use of topical • Systemic therapies are usually reserved for moderate or treatments and the role of systemic therapy in treating acne. severe acne, with a response to oral antibiotics taking up Acne is an inflammatory disorder of pilosebaceous units to six weeks. and is prevalent in adolescence. The characteristic lesions are • Hormonal therapies provide effective second-line open (black) and closed (white) comedones, inflammatory treatment in women with acne, regardless of the presence papules, pustules, nodules and cysts, which may lead to scar- or absence of androgen excess. ring and pigmentary changes (Figures 1 to 4). The pathogene- sis of acne is multifactorial and includes abnormal follicular keratinization, increased production of sebum secondary to ing and follicle-stimulating hormone levels.5 Pelvic ultra- hyperandrogenism, proliferation of Propionibacterium acnes sonography may show the presence of polycystic ovaries.5 In and inflammation.2,3 prepubertal children with acne, signs of hyperandrogenism Lesions occur primarily on the face, neck, upper back and include early-onset accelerated growth, pubic or axillary hair, chest.4 When assessing the severity of the acne, one needs to body odour, genital maturation and advanced bone age. -
UC Davis Dermatology Online Journal
UC Davis Dermatology Online Journal Title Morphea-like complications to illicit gluteal silicone injections Permalink https://escholarship.org/uc/item/0xv5g3sn Journal Dermatology Online Journal, 21(4) Authors Tausend, William E Stewart, Larissa R Rapini, Ronald P Publication Date 2015 DOI 10.5070/D3214026271 License https://creativecommons.org/licenses/by-nc-nd/4.0/ 4.0 Peer reviewed eScholarship.org Powered by the California Digital Library University of California Volume 21 Number 4 April 2015 Case presentation Morphea-like complications to illicit gluteal silicone injections William E. Tausend MD1, Larissa R. Stewart MD2, Ronald P. Rapini MD2 Dermatology Online Journal 21 (4): 5 1University of Texas Medical Branch Department of Dermatology 2University of Texas Health Science Center-Houston Department of Dermatology Correspondence: William E. Tausend, MD 1122 Postoffice St. Galveston, Texas 77550 Phone-832-816-5524 [email protected] Abstract We present a case of a 39-year-old Hispanic woman who was referred to our clinic for treatment of several indurated plaques on her buttocks that developed one year prior to presentation, after she received injections of an unknown substance for augmentation. Biopsy of one nodule revealed silicone in the dermis. Keywords: Silicone injection, Procedural complications Introduction Illicit injections of silicone are an increasing and worrisome trend in the United States, especially in the Hispanic and transgender communities [1]. These procedures are often performed by unlicensed individuals who inject large quantities of unknown substances into patients [2,3]. The reasons for this increasing trend are thought to relate to the reduced cost of the procedures [4,5]. -
Qt5085f0pn.Pdf
UC San Diego UC San Diego Previously Published Works Title Foreign Body Granulomas after All Injectable Dermal Fillers: Part 2. Treatment Options Permalink https://escholarship.org/uc/item/5085f0pn Author Lemperle, Gottfried Publication Date 2015-04-05 Peer reviewed eScholarship.org Powered by the California Digital Library University of California SPECIAL TOPIC Foreign Body Granulomas after All Injectable Dermal Fillers: Part 2. Treatment Options Foot Gottfried Lemperle, M.D., Summary: Foreign body granulomas occur at certain rates with all injectable Ph.D. dermal fillers. They have to be distinguished from early implant nodules, which Nelly Gauthier-Hazan, M.D. usually appear 2 to 4 weeks after injection. In general, foreign body granulomas San Diego, Calif.; and Paris, France appear after a latent period of several months at all injected sites at the same time. If diagnosed early and treated correctly, they can be diminished within a few weeks. The treatment of choice of this hyperactive granulation tissue is the intralesional injection of corticosteroid crystals (triamcinolone, betamethasone, or prednisolone), which may be repeated in 4-week cycles until the right dose is found. To lower the risk of skin atrophy, corticosteroids can be combined with antimitotic drugs such as 5-fluorouracil and pulsed lasers. Because foreign body granulomas grow fingerlike into the surrounding tissue, surgical excision should be the last option. Surgery or drainage is indicated to treat normal lumps and cystic foreign body granulomas with little tissue ingrowth. In most patients, a foreign body granuloma is a single event during a lifetime, often triggered by a systemic bacterial infection. (Plast. -
“Highly Cohesive” Silicone Breast Implants: a Histopathologic and Laser Raman Microprobe Analysis
International Journal of Environmental Research and Public Health Article A Case of Silicone and Sarcoid Granulomas in a Patient with “Highly Cohesive” Silicone Breast Implants: A Histopathologic and Laser Raman Microprobe Analysis Todor I. Todorov 1,†, Erik de Bakker 2,3, Diane Smith 4,‡, Lisette C. Langenberg 5, Linda A. Murakata 1,§, Mark H. H. Kramer 6 , Jose A. Centeno 1,k and Prabath W. B. Nanayakkara 4,* 1 Department of Environmental and Infectious Disease Sciences, Division of Biophysical Toxicology, Armed Forces Institute of Pathology, Washington, DC 20306, USA; [email protected] (T.I.T.); [email protected] (L.A.M.); [email protected] (J.A.C.) 2 Department of Plastic Surgery, VU University Medical Centre, P.O. Box 7057, 1007 MB Amsterdam, The Netherlands; [email protected] 3 Department of Molecular Cell Biology and Immunology, VU University Medical Centre, P.O. Box 7057, 1007 MB Amsterdam, The Netherlands 4 Henry Jackson Foundation, Bethesda, MD 20817, USA; [email protected] 5 Department of Internal Medicine, VU University Medical Centre, P.O. Box 7057, 1007 MB Amsterdam, The Netherlands; [email protected] 6 Department of Pathology, VU University Medical Centre, P.O. Box 7057, 1007 MB Amsterdam, The Netherlands; [email protected] * Correspondence: [email protected] † Current address: Center for Food Safety and Applied Nutrition, US Food and Drug Administration, Citation: Todorov, T.I.; de Bakker, E.; College Park, MD 20740, USA. Smith, D.; Langenberg, L.C.; ‡ Current address: Center for Device and Radiological Health, US Food and Drug Administration, Murakata, L.A.; Kramer, M.H.H.; Silver Spring, MD 20993, USA. -
Diagnostic Challenges and Treatment Difficulties in a Patient with Excoriated Acne Conglobata Simona R
Journal of Mind and Medical Sciences Volume 4 | Issue 1 Article 12 2017 Diagnostic challenges and treatment difficulties in a patient with excoriated acne conglobata Simona R. Georgescu Carol Davila University, Department of Dermatology, [email protected] Maria I. Sârbu Carol Davila University, Department of Dermatology Cristina I. Mitran Carol Davila University, Department of Microbiology Mădălina I. Mitran Carol Davila University, Department of Microbiology Vasile Benea Victor Babes Hospital for Infectious and Tropical Diseases, Bucharest, Romania See next page for additional authors Follow this and additional works at: http://scholar.valpo.edu/jmms Part of the Bacterial Infections and Mycoses Commons, and the Skin and Connective Tissue Diseases Commons Recommended Citation Georgescu, Simona R.; Sârbu, Maria I.; Mitran, Cristina I.; Mitran, Mădălina I.; Benea, Vasile; and Tampa, Mircea (2017) "Diagnostic challenges and treatment difficulties in a patient with excoriated acne conglobata," Journal of Mind and Medical Sciences: Vol. 4 : Iss. 1 , Article 12. DOI: 10.22543/7674.41.P7479 Available at: http://scholar.valpo.edu/jmms/vol4/iss1/12 This Case Presentation is brought to you for free and open access by ValpoScholar. It has been accepted for inclusion in Journal of Mind and Medical Sciences by an authorized administrator of ValpoScholar. For more information, please contact a ValpoScholar staff member at [email protected]. Diagnostic challenges and treatment difficulties in a patient with excoriated acne conglobata Authors Simona R. Georgescu, Maria I. Sârbu, Cristina I. Mitran, Mădălina I. Mitran, Vasile Benea, and Mircea Tampa This case presentation is available in Journal of Mind and Medical Sciences: http://scholar.valpo.edu/jmms/vol4/iss1/12 J Mind Med Sci. -
United States Patent (19) 11 Patent Number: 5,952,373 Lanzendörfer Et Al
USOO5952373A United States Patent (19) 11 Patent Number: 5,952,373 Lanzendörfer et al. (45) Date of Patent: Sep. 14, 1999 54 AGENTS ACTING AGAINST 56) References Cited HYPERREACTIVE AND HYPOACTIVE, DEFICIENT SKIN CONDITIONS AND U.S. PATENT DOCUMENTS MANIFEST DERMATITIDES 4,297,348 10/1981 Frazier .................................... 424/180 5,719,129 2/1998 Andary et al. ............................ 514/25 75 Inventors: Ghita Lanzendorfer, Hamburg; Franz St?b, Echem; Sven Untiedt, Hamburg, OTHER PUBLICATIONS all of Germany The Merck Index, 10" Ed., Windholz et al., p. 1315, abstract 73 Assignee: Beiersdorf AG, Hamburg, Germany No. 9021. (1983). Primary Examiner Kevin E. Weddington 21 Appl. No.: 08/849,523 Attorney, Agent, or Firm-Sprung Kramer Schaefer & 22 PCT Filed: Dec. 12, 1995 Briscoe 57 ABSTRACT 86 PCT No.: PCT/EP95/04907 S371 Date: Sep. 8, 1997 The invention relates to the use of a) a compound or several compounds from the group S 102(e) Date: Sep. 8, 1997 consisting of flavonoids 87 PCT Pub. No.: WO96/18381 b) of the antioxidants or c) of the endogenous energy metabolism metabolites or PCT Pub. Date:Jun. 20, 1996 d) of the endogenous enzymatic antioxidant Systems and 30 Foreign Application Priority Data synthetic derivatives thereof (mimics) or Dec. 13, 1994 DE Germany ............................. 44 44238 e) of the antimicrobial action Systems or f) of the antiviral action Systems or 51) Int. Cl. ........................... A61K 31/35; A61K 31/65 g) active compounds of the known, conventional treat 52 U.S. Cl. .......................... 514/456; 514/457; 514/152; ment forms 514/858; 514/859; 514/860; 514/861; 514/863; in each case for the treatment or prophylactic treatment of 514/864 hyperreactive skin predisposed to dermatitis or deficient, 58 Field of Search .................................... -
1. Acne Vulgaris 2
432 Teams Dermatology Acne Related Disorders Done by: Abrar AlFaifi Reviewer: Lama Al Tawil 7 Team Leader:Basil Al Suwaine&Lama Al Tawil Color Code: Original, Team’s note, Important, Doctor’s note, Not important, Old teamwork 432 Dermatology Team Lecture 7: Acne and Acneiform eruption Content 1) Acne Vulgaris 2) Acne Related Disorders P a g e | 1 432 Dermatology Team Lecture 7: Acne and Acneiform eruption Acne and Acneiform Eruptions 1. Acne Vulgaris 2. Hidradenitis Supprative 3. Rosacea 4. Perioral Dermatitis P a g e | 2 432 Dermatology Team Lecture 7: Acne and Acneiform eruption 1. Acne Vulgaris Multifactorial disease of pilosebaceous unit. Affects both males and females. The most common dermatological disease. Mostly prevalent between 12-24 yrs. Affects 8% between 25-34, 4% between 35-44. -It can occur earlier around 8 years of age when the adrenal glands secretes increased levels of androgens without cortisol and the development of new zone occurs –zona reticularis- (this is called adrenarche) -Acne occurrence with aging becomes less yet it can also appear in older age Pathogenesis Increased sebum secretion (Seborrhoea). Ductal cornification and occlusion (micro-comedo). Ductal colonization with propioni bacterium acnes. Fond of lipids and produced by sebaceous gland Rupture of sebaceous gland and inflammation (inflammatory lesion) Pilosebaceous unit: hair follicle sebaceous gland (under the effect of hormones) Specialized terms Microcomedone: Hyperkeratotic plug made of sebum and keratin in follicular canal. Closed comedo ( whitehead1): Closed follicular orifice, accumulation of sebum and keratin 1 Open comedo ( blackhead2): Opened follicular orifice packed with melanin and oxidized lipids -All acne begins with a microcomedo.