Eye60. Instrumental Eye Examination.Pdf
Total Page:16
File Type:pdf, Size:1020Kb
Load more
Recommended publications
-
Optic Disc Edema, Globe Flattening, Choroidal Folds, and Hyperopic Shifts Observed in Astronauts After Long-Duration Space Flight
University of Nebraska - Lincoln DigitalCommons@University of Nebraska - Lincoln NASA Publications National Aeronautics and Space Administration 10-2011 Optic Disc Edema, Globe Flattening, Choroidal Folds, and Hyperopic Shifts Observed in Astronauts after Long-duration Space Flight Thomas H. Mader Alaska Native Medical Center, [email protected] C. Robert Gibson Coastal Eye Associates Anastas F. Pass University of Houston Larry A. Kramer University of Texas Health Science Center Andrew G. Lee The Methodist Hospital See next page for additional authors Follow this and additional works at: https://digitalcommons.unl.edu/nasapub Part of the Physical Sciences and Mathematics Commons Mader, Thomas H.; Gibson, C. Robert; Pass, Anastas F.; Kramer, Larry A.; Lee, Andrew G.; Fogarty, Jennifer; Tarver, William J.; Dervay, Joseph P.; Hamilton, Douglas R.; Sargsyan, Ashot; Phillips, John L.; Tran, Duc; Lipsky, William; Choi, Jung; Stern, Claudia; Kuyumjian, Raffi; andolk, P James D., "Optic Disc Edema, Globe Flattening, Choroidal Folds, and Hyperopic Shifts Observed in Astronauts after Long-duration Space Flight" (2011). NASA Publications. 69. https://digitalcommons.unl.edu/nasapub/69 This Article is brought to you for free and open access by the National Aeronautics and Space Administration at DigitalCommons@University of Nebraska - Lincoln. It has been accepted for inclusion in NASA Publications by an authorized administrator of DigitalCommons@University of Nebraska - Lincoln. Authors Thomas H. Mader, C. Robert Gibson, Anastas F. Pass, Larry A. -
Differentiate Red Eye Disorders
Introduction DIFFERENTIATE RED EYE DISORDERS • Needs immediate treatment • Needs treatment within a few days • Does not require treatment Introduction SUBJECTIVE EYE COMPLAINTS • Decreased vision • Pain • Redness Characterize the complaint through history and exam. Introduction TYPES OF RED EYE DISORDERS • Mechanical trauma • Chemical trauma • Inflammation/infection Introduction ETIOLOGIES OF RED EYE 1. Chemical injury 2. Angle-closure glaucoma 3. Ocular foreign body 4. Corneal abrasion 5. Uveitis 6. Conjunctivitis 7. Ocular surface disease 8. Subconjunctival hemorrhage Evaluation RED EYE: POSSIBLE CAUSES • Trauma • Chemicals • Infection • Allergy • Systemic conditions Evaluation RED EYE: CAUSE AND EFFECT Symptom Cause Itching Allergy Burning Lid disorders, dry eye Foreign body sensation Foreign body, corneal abrasion Localized lid tenderness Hordeolum, chalazion Evaluation RED EYE: CAUSE AND EFFECT (Continued) Symptom Cause Deep, intense pain Corneal abrasions, scleritis, iritis, acute glaucoma, sinusitis, etc. Photophobia Corneal abrasions, iritis, acute glaucoma Halo vision Corneal edema (acute glaucoma, uveitis) Evaluation Equipment needed to evaluate red eye Evaluation Refer red eye with vision loss to ophthalmologist for evaluation Evaluation RED EYE DISORDERS: AN ANATOMIC APPROACH • Face • Adnexa – Orbital area – Lids – Ocular movements • Globe – Conjunctiva, sclera – Anterior chamber (using slit lamp if possible) – Intraocular pressure Disorders of the Ocular Adnexa Disorders of the Ocular Adnexa Hordeolum Disorders of the Ocular -
Diagnostic Tools for Dry Eye Disease
Review Ocular Surface Disease Diagnostic Tools for Dry Eye Disease Sarah Dougherty Wood and Shahzad I Mian Department of Ophthalmology and Visual Sciences, Medical School, University of Michigan, Ann Arbor, Michigan, US DOI: https://doi.org/10.17925/EOR.2016.10.02.101 ry eye disease is multifactorial in aetiology and complex in pathophysiology that makes its diagnosis clinically challenging. Although there are numerous tools for assessment of dry eye disease, no single test is sufficient for the diagnosis. Typically a combination of D subjective symptoms and objective tests are used. The aim of this article is to review the available tests, including traditional tools and emerging technologies. This review includes a description of the test methodology, type of data collected, diagnostic reliability of data, benefits and limitations of each test, expected outcomes and tips for practical application. Keywords The International Dry Eye Workshop Dry Eye Workshop (DEWS) defined dry eye as “a multifactorial Dry eye disease, dry eye diagnosis, tear film disease of the tears and ocular surface that results in symptoms of discomfort, visual disturbance and tear film instability with potential damage of the ocular surface. It is accompanied by increased Disclosure: Sarah Dougherty Wood and Shahzad I Mian osmolarity of the tear film and inflammation of the ocular surface”.1 This condition is divided into do not have financial or proprietary interest in any materials or methods mentioned. No funding was two general types: deficient aqueous production by the lacrimal gland and increased evaporation received in the publication of this article. This study of the tear film, with the latter being more prevalent. -
Cataract Surgery Co-Management Information
Cataract Surgery Co-Management Phacoemulsification, Clear-Lens Extraction, and LensX INCLUSION CRITERIA: Significant visual complaints (decreased VA, increased glare, decreased Activities of Daily Living [ADLs], etc.) Treatment of secondary ocular disease (phacomorphic glaucoma, uveitis) Management of ocular disease (diabetic retinopathy, etc.) The patient’s Snellen best-corrected visual acuity must be 20/50 or worse. They may also be eligible for surgery if their BCVA is 20/40 or better and have significant difficulty with glare. Complaints of glare should be confirmed by brightness acuity testing or another suitable diagnostic test. EXCLUSION CRITERIA: Patients who are unable to receive proper postoperative care Patients in poor overall health (Primary Care Physician will not clear patient for surgery) TYPES OF CATARACT PATIENTS: Congenital: If cataract obscures visual axis (> or equal to 3mm) or is causing secondary disease, extraction should be performed within days to weeks after diagnosis in infants and small children to prevent amblyopia. If the cataract is not causing complications, closely observe for progression. Often patients with visually-significant, unilateral, congenital cataracts have strabismus and may require muscle surgery after extraction. Acquired: Most often, senile (Nuclear sclerosis, cortical degeneration, subcapsular) Due to systemic disease: (not limited to those listed below) Diabetes Mellitus Hypocalcemia Myotonic Dystrophy Frabry’s Disease Down’s Syndrome Atopic Dermatitis Wilson’s Disease -
Slit-Lamp Examination of the Vitreous and the Fundus* by H
Br J Ophthalmol: first published as 10.1136/bjo.33.4.242 on 1 April 1949. Downloaded from 242 H. GOLDMANN REFERENCES ANGELUCCI (1905).-Encycloqedie fran9aise. BAILLIART and BIDAULT (1939).-In Traiti d'Ophtalmologie, 8. Masson. COLLE, DUKE-ELDER, P. M., and DUKE-ELDER, W. S. (1931).-Jl. Physiol., 71, 1. COLOMBO (1923).-Boll. d'Ocul., 2. ;CRISTINI, G. (1947) -Ann. d'Ocul., 80, 530. (1948).-Gior. Ital. Oftal., 1, 5. (1948).-ibid., 1, 385. DIETER (1925).-Arch. f. A ugenhesilk., 96, 179 264. DUKE-ELDER, W. S. (1938).-Text-Book of Ophthalmology, Kimpton. DUKE-ELDER, W. S. and DAVSON, H. (1948).-Brit. Ji. Ophthal., 32, 555, ELSCHNIG.-Henke Lubarsch, 11, 911. FORTIN (1929).-Arch. d'Oftal., B.A., 359, 454. (1939).-Semana Medica., 1, 1128. GALA (1939).-Quoted by Magitot in Tratt d'Ophtalmologie, 6, 264. Masson. HAMBURGER (1923).-Med. Klin., 19, 1215. (1924).-lbid., 20, 267, (1925).-Ibid., 21, 1495, IiENDERSON and STARLING (1904).-Jl. Physiol., 31, 305. v. HIPPEL and GRUENHAGEN (1868).-Arch. f. Ophthal., 14,219. KOELLNER (1916).-Arch. f. Augenheilk., 80, 245. KUESEL (1906).-Klin. Monatsbl. f. Augenheilk., 44, 80, 236. LUCIANI-Fisiologia dell'uomo, 1, 381. MAGITOT (1939).-Traite d'Ophtalmologie, 11. Masson. MICHAIL and VANCEA (1926).-Ann. d'Ocul., 126, 561, PARSONS (1902).-The Pathology of the Eye. London. Poos (1931).-Arch. f. Ophthal., 127, 489. STOCKER (1947).-Arch. Ophthal., 37, 583. THIEL (1924).-Zentralbl. f. d. ges. Ophthal., 12, 305. Kurzes Handb,f. Oihthal. (Glaukom.), 781. THOMASSEN (i947).-Acta Ophthal., 25, 221. copyright. - (1947).-Ibid., 25,252. WEBER (1877).-Arch. f. Ophthal., 23, 1. -
CAUSES, COMPLICATIONS &TREATMENT of A“RED EYE”
CAUSES, COMPLICATIONS & TREATMENT of a “RED EYE” 8 Most cases of “red eye” seen in general practice are likely to be conjunctivitis or a superficial corneal injury, however, red eye can also indicate a serious eye condition such as acute angle glaucoma, iritis, keratitis or scleritis. Features such as significant pain, photophobia, reduced visual acuity and a unilateral presentation are “red flags” that a sight-threatening condition may be present. In the absence of specialised eye examination equipment, such as a slit lamp, General Practitioners must rely on identifying these key features to know which patients require referral to an Ophthalmologist for further assessment. Is it conjunctivitis or is it something more Iritis is also known as anterior uveitis; posterior uveitis is serious? inflammation of the choroid (choroiditis). Complications include glaucoma, cataract and macular oedema. The most likely cause of a red eye in patients who present to 4. Scleritis is inflammation of the sclera. This is a very rare general practice is conjunctivitis. However, red eye can also be presentation, usually associated with autoimmune a feature of a more serious eye condition, in which a delay in disease, e.g. rheumatoid arthritis. treatment due to a missed diagnosis can result in permanent 5. Penetrating eye injury or embedded foreign body; red visual loss. In addition, the inappropriate use of antibacterial eye is not always a feature topical eye preparations contributes to antimicrobial 6. Acid or alkali burn to the eye resistance. The patient history will usually identify a penetrating eye injury Most general practice clinics will not have access to specialised or chemical burn to the eye, but further assessment may be equipment for eye examination, e.g. -
Classification of Posterior Vitreous Detachment
Clinical Ophthalmology Dovepress open access to scientific and medical research Open Access Full Text Article EXPERT OPINION Classification of posterior vitreous detachment Akihiro Kakehashi1 Abstract: Diagnosing a posterior vitreous detachment (PVD) is important for predicting the Mikiko Takezawa1 prognosis and determining the indication for vitreoretinal surgery in many vitreoretinal diseases. Jun Akiba2 This article presents both classifications of a PVD by slit-lamp biomicroscopy and of a shallow PVD by optical coherence tomography (OCT). By biomicroscopy, the vitreous condition is deter- 1Department of Ophthalmology, Jichi Medical University, Saitama mined based on the presence or absence of a PVD. The PVD then is classified as either a complete Medical Center, Saitama, 2Kanjodori posterior vitreous detachment (C-PVD) or a partial posterior vitreous detachment (P-PVD). Eye Clinic, Asahikawa, Japan A C-PVD is further divided into a C-PVD with collapse and a C-PVD without collapse, while a P-PVD is divided into a P-PVD with shrinkage of the posterior hyaloid membrane (P-PVD with shrinkage) and a P-PVD without shrinkage of the posterior hyaloid membrane (P-PVD without shrinkage). A P-PVD without shrinkage has a subtype characterized by vitreous gel attachment through the premacular hole in a posterior hyaloid membrane to the macula (P-PVD without shrinkage [M]). By OCT, a shallow PVD is classified as the absence of a shallow PVD or as a shallow PVD. A shallow PVD is then subclassified as a shallow PVD without shrinkage of the posterior vitreous cortex, a shallow PVD with shrinkage of the posterior vitreous cortex, and a peripheral shallow PVD. -
Papilledema Associated with Puberty
CPJXXX10.1177/0009922814554503Clinical PediatricsSun and Horton 554503research-article2014 Resident Round Clinical Pediatrics 2015, Vol. 54(5) 504 –506 Papilledema Associated with Puberty © The Author(s) 2014 Reprints and permissions: sagepub.com/journalsPermissions.nav DOI: 10.1177/0009922814554503 cpj.sagepub.com Lynn W. Sun, BA, PhD1 and Jonathan C. Horton, MD, PhD2 Case Report headache. Fundus examination showed slight reduction of her papilledema. Over the next 6 months her symp- A 9-year-old girl was brought by her mother to a hospi- toms gradually resolved and she stopped attending clinic tal emergency room because of severe headache and appointments. vomiting. The symptoms began 2 weeks earlier while at Two years after her original presentation, she returned summer camp. The child’s examination was benign, and to the eye clinic for a routine examination. On her own she was discharged with a diagnosis of migraine. The initiative, she had discontinued treatment with acetazol- ocular fundi were not examined. The headaches contin- amide 18 months earlier. She denied headache or blurred ued and were sometimes associated with nausea, photo- vision. The height was 152.4 cm and the weight was phobia, and tinnitus. She was evaluated 2 weeks later in 71.67 kg, for a BMI of 30.9 kg/m2 (obese). She had regu- our pediatric ophthalmology clinic because of intermit- lar menstrual periods. The visual acuity was 20/20 in tent horizontal diplopia and blurred vision. each eye without correction. Pupils, eye movements, There was no relevant past medical history. The ocular alignment, visual fields, and slit lamp examination patient did not take tetracycline antibiotics or any other were normal. -
Using Slit-Lamp Images for Deep Learning-Based
diagnostics Article Using Slit-Lamp Images for Deep Learning-Based Identification of Bacterial and Fungal Keratitis: Model Development and Validation with Different Convolutional Neural Networks Ning Hung 1,2,†, Andy Kuan-Yu Shih 3,†, Chihung Lin 3 , Ming-Tse Kuo 4 , Yih-Shiou Hwang 1,2, Wei-Chi Wu 1,2, Chang-Fu Kuo 3, Eugene Yu-Chuan Kang 1,2,* and Ching-Hsi Hsiao 1,2,* 1 Department of Ophthalmology, Chang Gung Memorial Hospital, Linkou Medical Center, No. 5 Fu-Hsin Rd, Kweishan, Taoyuan 333, Taiwan; [email protected] (N.H.); [email protected] (Y.-S.H.); [email protected] (W.-C.W.) 2 College of Medicine, Chang Gung University, No. 261, Wenhua 1st Rd., Kweishan, Taoyuan 333, Taiwan 3 Center for Artificial Intelligence in Medicine, Chang Gung Memorial Hospital, Linkou Medical Center, No. 5 Fu-Hsin Rd, Kweishan, Taoyuan 333, Taiwan; [email protected] (A.K.-Y.S.); [email protected] (C.L.); [email protected] (C.-F.K.) 4 Department of Ophthalmology, Kaohsiung Chang Gung Memorial Hospital, No. 123, Dapi Rd, Niaosong, Kaohsiung 833, Taiwan; [email protected] * Correspondence: [email protected] (E.Y.-C.K.); [email protected] (C.-H.H.); Tel.: +886-3-3281200 (E.Y.-C.K. & C.-H.H.) † These authors contributed equally to this work. Abstract: In this study, we aimed to develop a deep learning model for identifying bacterial keratitis Citation: Hung, N.; Shih, A.K.-Y.; (BK) and fungal keratitis (FK) by using slit-lamp images. We retrospectively collected slit-lamp Lin, C.; Kuo, M.-T.; Hwang, Y.-S.; Wu, images of patients with culture-proven microbial keratitis between 1 January 2010 and 31 December W.-C.; Kuo, C.-F.; Kang, E.Y.-C.; Hsiao, 2019 from two medical centers in Taiwan. -
98796-Anatomy of the Orbit
Anatomy of the orbit Prof. Pia C Sundgren MD, PhD Department of Diagnostic Radiology, Clinical Sciences, Lund University, Sweden Lund University / Faculty of Medicine / Inst. Clinical Sciences / Radiology / ECNR Dubrovnik / Oct 2018 Lund University / Faculty of Medicine / Inst. Clinical Sciences / Radiology / ECNR Dubrovnik / Oct 2018 Lay-out • brief overview of the basic anatomy of the orbit and its structures • the orbit is a complicated structure due to its embryological composition • high number of entities, and diseases due to its composition of ectoderm, surface ectoderm and mesoderm Recommend you to read for more details Lund University / Faculty of Medicine / Inst. Clinical Sciences / Radiology / ECNR Dubrovnik / Oct 2018 Lund University / Faculty of Medicine / Inst. Clinical Sciences / Radiology / ECNR Dubrovnik / Oct 2018 3 x 3 Imaging technique 3 layers: - neuroectoderm (retina, iris, optic nerve) - surface ectoderm (lens) • CT and / or MR - mesoderm (vascular structures, sclera, choroid) •IOM plane 3 spaces: - pre-septal •thin slices extraconal - post-septal • axial and coronal projections intraconal • CT: soft tissue and bone windows 3 motor nerves: - occulomotor (III) • MR: T1 pre and post, T2, STIR, fat suppression, DWI (?) - trochlear (IV) - abducens (VI) Lund University / Faculty of Medicine / Inst. Clinical Sciences / Radiology / ECNR Dubrovnik / Oct 2018 Lund University / Faculty of Medicine / Inst. Clinical Sciences / Radiology / ECNR Dubrovnik / Oct 2018 Superior orbital fissure • cranial nerves (CN) III, IV, and VI • lacrimal nerve • frontal nerve • nasociliary nerve • orbital branch of middle meningeal artery • recurrent branch of lacrimal artery • superior orbital vein • superior ophthalmic vein Lund University / Faculty of Medicine / Inst. Clinical Sciences / Radiology / ECNR Dubrovnik / Oct 2018 Lund University / Faculty of Medicine / Inst. -
Anatomy and Physiology of the Afferent Visual System
Handbook of Clinical Neurology, Vol. 102 (3rd series) Neuro-ophthalmology C. Kennard and R.J. Leigh, Editors # 2011 Elsevier B.V. All rights reserved Chapter 1 Anatomy and physiology of the afferent visual system SASHANK PRASAD 1* AND STEVEN L. GALETTA 2 1Division of Neuro-ophthalmology, Department of Neurology, Brigham and Womens Hospital, Harvard Medical School, Boston, MA, USA 2Neuro-ophthalmology Division, Department of Neurology, Hospital of the University of Pennsylvania, Philadelphia, PA, USA INTRODUCTION light without distortion (Maurice, 1970). The tear–air interface and cornea contribute more to the focusing Visual processing poses an enormous computational of light than the lens does; unlike the lens, however, the challenge for the brain, which has evolved highly focusing power of the cornea is fixed. The ciliary mus- organized and efficient neural systems to meet these cles dynamically adjust the shape of the lens in order demands. In primates, approximately 55% of the cortex to focus light optimally from varying distances upon is specialized for visual processing (compared to 3% for the retina (accommodation). The total amount of light auditory processing and 11% for somatosensory pro- reaching the retina is controlled by regulation of the cessing) (Felleman and Van Essen, 1991). Over the past pupil aperture. Ultimately, the visual image becomes several decades there has been an explosion in scientific projected upside-down and backwards on to the retina understanding of these complex pathways and net- (Fishman, 1973). works. Detailed knowledge of the anatomy of the visual The majority of the blood supply to structures of the system, in combination with skilled examination, allows eye arrives via the ophthalmic artery, which is the first precise localization of neuropathological processes. -
Clinical Findings and Management of Posterior Vitreous Detachment
American Academy of Optometry: Case Report 5 Clinical Findings and Management of Posterior Vitreous Detachment Candidate’s Name, O.D. Candidate’s Address Candidate’s Phone number Candidate’s email Abstract: A posterior vitreous detachment is a degenerative process associated with aging that affects the vitreous when the posterior vitreous cortex separates from the internal limiting membrane of the retina. The composition of the vitreous gel can degenerate two collective ways, including synchysis or liquefaction, and syneresis or shrinking. Commonly, this process of separation occurs with the posterior hyaloid resulting in a Weiss ring overlying the optic nerve. Complications of a posterior vitreous detachment may include retinal breaks or detachments, retinal or vitreous hemorrhages, or vitreomacular traction. This case presentation summarizes the etiology of this ocular condition as well as treatment and management approaches. Key Words: Posterior Vitreous Detachment, Weiss Ring, Vitreous Degeneration, Scleral Depression, Nd:YAG Laser 1 Introduction The vitreous humor encompasses the posterior segment of the eye and fills approximately three quarters of the ocular space.1 The vitreous is a transparent, hydrophilic, “gel-like” substance that is described as a dilute solution of collagen, and hyaluronic acid.2,3,4 It is composed of 98% to 99.7% water.4 As the eye matures, changes may occur regarding the structure and composition of the vitreous. The vitreous functions to provide support to the retina against the choroid, to store nutrients and metabolites for the retina and lens, to protect the retinal tissue by acting as a “shock absorber,” to transmit and refract light, and to help regulate eye growth during fetal development.3,4 Case Report Initial Visit (03/23/2018) A 59-year-old Asian female presented as a new patient for examination with a complaint of a new onset of floaters and flashes of light in her right eye.