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Benign Cementoblastoma Associated with an Impacted Mandibular Third Molar – Report of an Unusual Case
Case Report Benign Cementoblastoma Associated with an Impacted Mandibular Third Molar – Report of an Unusual Case Chethana Dinakar1, Vikram Shetty2, Urvashi A. Shetty3, Pushparaja Shetty4, Madhvika Patidar5,* 1,3Senior Lecturer, 4Professor & HOD, Department of Oral Pathology and Microbiology, AB Shetty Memorial Institute of Dental Science, Mangaloge, 2Director & HOD, Nittee Meenakshi Institute of Craniofacial Surgery, Mangalore, 5Senior Lecturer, Department of Oral Pathology and Microbiology, Babu Banarasi Das College of Dental Sciences, Lucknow *Corresponding Author: Email: [email protected] ABSTRACT Cementoblastoma is characterized by the formation of cementum-like tissue in direct connection with the root of a tooth. It is a rare lesion constituting less than 1% of all odontogenic tumors. We report a unique case of a large cementoblastoma attached to the lateral root surface of an impacted permanent mandibular third molar in a 33 year old male patient. The association of cementoblastomas with impacted teeth is a rare finding. Key Words: Odontogenic tumor, Cementoblastoma, Impacted teeth, Third molar, Cementum Access this article online opening limited to approximately 10mm. The swelling Quick Response was firm to hard in consistency and tender on palpation. Code: Website: Lymph nodes were not palpable. www.innovativepublication.com On radiographical examination, it showed a large, well circumscribed radiopaque mass attached to the lateral root surface of impacted permanent right mandibular DOI: 10.5958/2395-6194.2015.00005.3 third molar. The mass displayed a radiolucent area at the other end and was seen occupying almost the entire length of the ramus of mandible. The entire lesion was INTRODUCTION surrounded by a thin, uniform radiolucent line (Fig. -
Glossary for Narrative Writing
Periodontal Assessment and Treatment Planning Gingival description Color: o pink o erythematous o cyanotic o racial pigmentation o metallic pigmentation o uniformity Contour: o recession o clefts o enlarged papillae o cratered papillae o blunted papillae o highly rolled o bulbous o knife-edged o scalloped o stippled Consistency: o firm o edematous o hyperplastic o fibrotic Band of gingiva: o amount o quality o location o treatability Bleeding tendency: o sulcus base, lining o gingival margins Suppuration Sinus tract formation Pocket depths Pseudopockets Frena Pain Other pathology Dental Description Defective restorations: o overhangs o open contacts o poor contours Fractured cusps 1 ww.links2success.biz [email protected] 914-303-6464 Caries Deposits: o Type . plaque . calculus . stain . matera alba o Location . supragingival . subgingival o Severity . mild . moderate . severe Wear facets Percussion sensitivity Tooth vitality Attrition, erosion, abrasion Occlusal plane level Occlusion findings Furcations Mobility Fremitus Radiographic findings Film dates Crown:root ratio Amount of bone loss o horizontal; vertical o localized; generalized Root length and shape Overhangs Bulbous crowns Fenestrations Dehiscences Tooth resorption Retained root tips Impacted teeth Root proximities Tilted teeth Radiolucencies/opacities Etiologic factors Local: o plaque o calculus o overhangs 2 ww.links2success.biz [email protected] 914-303-6464 o orthodontic apparatus o open margins o open contacts o improper -
Oral Health in Prevalent Types of Ehlers–Danlos Syndromes
View metadata, citation and similar papers at core.ac.uk brought to you by CORE provided by Ghent University Academic Bibliography J Oral Pathol Med (2005) 34: 298–307 ª Blackwell Munksgaard 2005 Æ All rights reserved www.blackwellmunksgaard.com/jopm Oral health in prevalent types of Ehlers–Danlos syndromes Peter J. De Coster1, Luc C. Martens1, Anne De Paepe2 1Department of Paediatric Dentistry, Centre for Special Care, Paecamed Research, Ghent University, Ghent; 2Centre for Medical Genetics, Ghent University Hospital, Ghent, Belgium BACKGROUND: The Ehlers–Danlos syndromes (EDS) Introduction comprise a heterogenous group of heritable disorders of connective tissue, characterized by joint hypermobility, The Ehlers–Danlos syndromes (EDS) comprise a het- skin hyperextensibility and tissue fragility. Most EDS erogenous group of heritable disorders of connective types are caused by mutations in genes encoding different tissue, largely characterized by joint hypermobility, skin types of collagen or enzymes, essential for normal pro- hyperextensibility and tissue fragility (1) (Fig. 1). The cessing of collagen. clinical features, modes of inheritance and molecular METHODS: Oral health was assessed in 31 subjects with bases differ according to the type. EDS are caused by a EDS (16 with hypermobility EDS, nine with classical EDS genetic defect causing an error in the synthesis or and six with vascular EDS), including signs and symptoms processing of collagen types I, III or V. The distribution of temporomandibular disorders (TMD), alterations of and function of these collagen types are displayed in dental hard tissues, oral mucosa and periodontium, and Table 1. At present, two classifications of EDS are was compared with matched controls. -
Neutropenia Fact Sheet
Neutropenia in Barth Syndrome i ii (Chronic, Cyclic or Intermittent) What problems can Neutropenia cause? Neutrophils are the main white blood cell for fighting or preventing bacterial or fungal infections. They may be referred to as polymorphonuclear cells (polys or PMNs), white cells with segmented nuclei (segs), or neutrophils in the complete blood cell count (CBC) report. Immature neutrophils are referred to as bands. When someone is neutropenic (an abnormally low level of neutrophils in the blood), the risk of infection increases. The absolute neutrophil count (ANC) is a measure of the total number of neutrophils present in the blood. When the ANC is less than 1,000, the risk of infection increases. Most infections occur in the ears, skin or throat and to a lesser extent, the chest. These infections can be very serious and may require antibiotics to clear infections. When someone with Barth syndrome is neutropenic his defenses are weakened, he is likely to become seriously ill more quickly than someone with a normal neutrophil count. Tips: • No rectal temperatures as any break in the skin can lead to an infection. • If the individual has a temperature > 100.4° F (38° C) or has infectious symptoms, the primary physician or hematologist should be notified. The individual may need to be seen. • If the individual has a temperature of 100.4° F (38° C) – 100.5° F (38.05° C)> 8 hours or a temperature > 101.5° F (38.61° C), an immediate examination by the physician is warranted. Some or all of the following studies may be ordered: CBC with differential and ANC Urinalysis Blood, urine, and other appropriate cultures C-Reactive Protein Echocardiogram if warranted • The physician may suggest antibiotics (and G-CSF if the ANC is low) for common infections such as otitis media, stomatitis. -
Benign Fibro-Osseous Lesions Plus…
“Vision is the art of seeing things invisible.” Jonathan Swift 1667 - 1745 Benign Fibro-osseous Lesions Plus… Steven R. Singer, DDS [email protected] 212.305.5674 Benign Fibro-osseous Lesions Fibrous Dysplasia A group of lesions in which normal bone is Localized change in bone metabolism replaced initially by fibrous connective tissue Normal cancellous bone is replaced by Over time, the lesion is infiltrated by osteoid fibrous connective tissue and cementoid tissue The connective tissue contains varying amounts of abnormal bone with irregular This is a benign and idiopathic process trabeculae Trabeculae are randomly oriented. (Remember that normal trabeculae are aligned to respond to stress) Fibrous Dysplasia Fibrous Dysplasia Lesions may be solitary (monostotic) or Fibrous dysplasia is non-hereditary involve more than one bone (polyostotic) Caused by a mutation in a somatic cell. Monostotic form accounts for 70% of all Extent of lesions depends on the timing of cases the mutation. Polyostotic form is more common in the first If the mutation occurs earlier, the disease decade will be more widespread throughout the M=F except in McCune-Albright syndrome, body. An example is McCune-Albright which is almost exclusively found in females Syndrome 1 Fibrous Dysplasia Fibrous Dysplasia McCune-Albright Syndrome • Monostotic and polyostotic forms usually -Almost exclusively begins in the second decade of life females -Polyostotic fibrous • Slow, painless expansion of the jaws dysplasia • Patients may complain of swelling or have -
Misdiagnosis of Osteosarcoma As Cementoblastoma from an Atypical Mandibular Swelling: a Case Report
ONCOLOGY LETTERS 11: 3761-3765, 2016 Misdiagnosis of osteosarcoma as cementoblastoma from an atypical mandibular swelling: A case report ZAO FANG1*, SHUFANG JIN1*, CHENPING ZHANG1, LIZHEN WANG2 and YUE HE1 1Department of Oral Maxillofacial Head and Neck Oncology, Faculty of Oral and Maxillofacial Surgery; 2Department of Oral Pathology, Shanghai Ninth People's Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai Key Laboratory of Stomatology, Shanghai 200011, P.R. China Received December 1, 2014; Accepted January 12, 2016 DOI: 10.3892/ol.2016.4433 Abstract. Cementoblastoma is a form of benign odontogenic of the lesion with extraction of the associated tooth (2); tumor, with the preferred treatment consisting of tooth extrac- however, certain patients may decide against surgery, under- tion and follow-up examinations, while in certain cases, going follow-up alone. Osteosarcoma is a non-hematopoietic, follow-up examinations without surgery are performed. malignant tumor of the bone, with the neoplastic cells of the Osteosarcoma of the jaw is a rare, malignant, mesenchymal lesion producing osteoid (3). This form of tumor is character- tumor, associated with a high mortality rate and low incidence ized by high malignancy, metastasis and mortality rates (4). of metastasis. Cementoblastoma and osteosarcoma of the jaw The tumors are most prevalently located in the metaphyseal are dissimilar in terms of their histological type and prognosis; region of long bones, particularly in the knee and pelvis (5). however, there are a number of covert associations between Osteosarcoma of the jaw is rare, accounting for 5-13% of all them. The present study describes the case of a 20-year-old osteosarcoma cases (6), the majority of which are located in female with an unusual swelling in the left mandible that the mandible. -
Regional Odontodysplasia: Report of an Unusual Case Involving Mandibular Arch
Regional odontodysplasia: Report of an unusual case involving mandibular arch N. S. Venkatesh Babu, R. Jha Smriti, D. Bang Pratima Abstract Regional odontodysplasia (RO) is a rare developmental anomaly involving both mesodermal and ectodermal components in primary or permanent dentition. It affects the maxilla and the mandible or both; however, maxilla is more commonly involved. This article reports the case of 33-month-old boy who came with the chief complaint of delayed eruption of mandibular teeth. Findings of clinical and radiographic examination were consistent with those of RO. Maxillary dentition was unaffected. Clinical and radiographic features and treatment options are discussed. Keywords: Mandibular arch, primary teeth, regional odontodysplasia Introduction cases of mandibular involvement have been reported so far.[5,8,9] Regional odontodysplasia (RO) is a rare developmental dental anomaly that involves ectoderm and mesoderm The teeth with RO often display a brownish or yellowish derived tissues.[1] It can affect either primary or permanent discoloration and most frequent clinical symptoms dentition.[2] This condition was first described by Hitchin accompanied by this anomaly are failure of eruption and in 1934. The prevalence of this condition is still not clear gingival enlargement. Radiologically, the affected teeth since the studies reported till date have mainly been based illustrate hypoplastic crowns and lack of contrast between on case reports. enamel and dentin is usually apparent. Enamel and the dentin are very thin, -
Benign Cementoblastoma Associated with an Unerupted Third Molar - a Case Report
CORE Metadata, citation and similar papers at core.ac.uk Provided by Directory of Open Access Journals BENIGN CEMENTOBLASTOMA ASSOCIATED WITH AN UNERUPTED THIRD MOLAR - A CASE REPORT J.Dinakar* M.S.Senthil Kumar** Shiju Mathew Jacob*** *Professor & HOD, Department of Oral Pathology, ** Reader, *** Lecturer, Department of Oral Surgery, Sri Ramakrishna Dental College and Hospital, Coimbatore, Tamilnadu, India. ABSTRACT: Cementoblastoma is a rare odontogenic tumor derived from odontogenic ectomesenchyme of cementoblast origin that forms cementum layer on the roots of a tooth. A case report is presented of a patient treated with surgical excision of Cementoblastoma associated with an unerupted infected right lower third molar tooth. Key words: Cementoblastoma, Odontogenic tumour, unerupted third molar. The cell of origin is cementoblast. INTRODUCTION: Clinically it causes bony expansion. The commonest site is the posterior region of Cementoblastoma is an odontogenic the mandible. In the radiograph it is seen tumor of ectomesenchymal origin. It is as large radiopaque mass associated with also called cementoma. They are large the root of the tooth. We report a case of bulbous mass of cementum or cementum- Benign Cementoblastoma from Sri like tissue on roots of teeth. Ramakrishna Dental College & Hospital, Coimbatore. restriction in opening the mouth and intra oral examination reveals a partially CASE REPORT: erupted third molar tooth with pus discharge. A panoramic radiograph A 41 year old man presented to our showed a radio-opaque, dense, department with a complaint of pain and amorphous, irregularly shaped mass swelling in the right lower half of the face. measuring 2.2 x 1.5cm attached with the Patient gave history of intermittent pain third molar (Fig 1,1a). -
THE PATHOLOGY of BONE MARROW FAILURE Roos Leguit, Jan G Van Den Tweel
THE PATHOLOGY OF BONE MARROW FAILURE Roos Leguit, Jan G van den Tweel To cite this version: Roos Leguit, Jan G van den Tweel. THE PATHOLOGY OF BONE MARROW FAILURE. Histopathology, Wiley, 2010, 57 (5), pp.655. 10.1111/j.1365-2559.2010.03612.x. hal-00599534 HAL Id: hal-00599534 https://hal.archives-ouvertes.fr/hal-00599534 Submitted on 10 Jun 2011 HAL is a multi-disciplinary open access L’archive ouverte pluridisciplinaire HAL, est archive for the deposit and dissemination of sci- destinée au dépôt et à la diffusion de documents entific research documents, whether they are pub- scientifiques de niveau recherche, publiés ou non, lished or not. The documents may come from émanant des établissements d’enseignement et de teaching and research institutions in France or recherche français ou étrangers, des laboratoires abroad, or from public or private research centers. publics ou privés. Histopathology THE PATHOLOGY OF BONE MARROW FAILURE ForJournal: Histopathology Peer Review Manuscript ID: HISTOP-02-10-0090 Manuscript Type: Review Date Submitted by the 08-Feb-2010 Author: Complete List of Authors: Leguit, Roos; UMC utrecht, Pathology van den Tweel, Jan; UMC Utrecht, Pathology bone marrow, histopathology, myelodysplastic syndromes, Keywords: inherited bone marrow failure syndromes, trephine biopsy Published on behalf of the British Division of the International Academy of Pathology Page 1 of 40 Histopathology THE PATHOLOGY OF BONE MARROW FAILURE Roos J Leguit & Jan G van den Tweel University Medical Centre Utrecht Department of Pathology H4.312 Heidelberglaan 100 For Peer Review 3584 CX Utrecht The Netherlands Running title: Pathology of bone marrow failure Keywords: bone marrow, histopathology, myelodysplastic syndromes, inherited bone marrow failure syndromes, trephine biopsy. -
Pediatric Oral Pathology. Soft Tissue and Periodontal Conditions
PEDIATRIC ORAL HEALTH 0031-3955100 $15.00 + .OO PEDIATRIC ORAL PATHOLOGY Soft Tissue and Periodontal Conditions Jayne E. Delaney, DDS, MSD, and Martha Ann Keels, DDS, PhD Parents often are concerned with “lumps and bumps” that appear in the mouths of children. Pediatricians should be able to distinguish the normal clinical appearance of the intraoral tissues in children from gingivitis, periodontal abnormalities, and oral lesions. Recognizing early primary tooth mobility or early primary tooth loss is critical because these dental findings may be indicative of a severe underlying medical illness. Diagnostic criteria and .treatment recommendations are reviewed for many commonly encountered oral conditions. INTRAORAL SOFT-TISSUE ABNORMALITIES Congenital Lesions Ankyloglossia Ankyloglossia, or “tongue-tied,” is a common congenital condition characterized by an abnormally short lingual frenum and the inability to extend the tongue. The frenum may lengthen with growth to produce normal function. If the extent of the ankyloglossia is severe, speech may be affected, mandating speech therapy or surgical correction. If a child is able to extend his or her tongue sufficiently far to moisten the lower lip, then a frenectomy usually is not indicated (Fig. 1). From Private Practice, Waldorf, Maryland (JED); and Department of Pediatrics, Division of Pediatric Dentistry, Duke Children’s Hospital, Duke University Medical Center, Durham, North Carolina (MAK) ~~ ~ ~ ~ ~ ~ ~ PEDIATRIC CLINICS OF NORTH AMERICA VOLUME 47 * NUMBER 5 OCTOBER 2000 1125 1126 DELANEY & KEELS Figure 1. A, Short lingual frenum in a 4-year-old child. B, Child demonstrating the ability to lick his lower lip. Developmental Lesions Geographic Tongue Benign migratory glossitis, or geographic tongue, is a common finding during routine clinical examination of children. -
Spectrum of Dentin Dysplasia in a Family
CASE REPORT Spectrumof dentin dysplasia in a family: case report and literature review W. Kim Seow, BDS, MDSc, DDSc, PhD Stephen Shusterman, DMD Abstract The dentin dysplasias (DD),which maybe classified as type I (DD1)or type 2 (DD2),form a group of rare, inherited abnormalitiesthat are clinically distinct fromdentinogenesis imperfecta. Studies of affected families mayhelp to distinguish different types of DDand provide further insight into their etiology and clinical management.This report describes a family that showed characteristic dental features of DD1, including clinically normalcrowns in both primaryand permanentdentitions, and mobileteeth that maybe associated with prematureexfoliation. Radiographicfeatures included calcification of the pulp with crescent-shaped, radiolucent pulp remnants, short, tapering, taurodontic roots, and manyperiapical pathoses that maybe q¢sts or granulomas.A spectrumof dentin dysplasia wasnoted within the family. Strategies to prevent pulp and periapical infections and early exfoliation of the teeth include meticulousoral hygieneand effective caries-preventivemeasures. ( P ed iatr Dent16:437-42,1994) Introduction and literature review Histologically, in DD1, most of the coronal and Dentin dysplasias (DD) form a group of rare dentin mantle dentin of the root is usually reported to be abnormalities that are clinically distinct from normal, and the dentin defect is confined mainly to the dentinogenesis imperfecta. 1-3 Since its recognition in root2, s, 10 The dysplastic dentin has been reported to 19203 as "rootless teeth" and as "dentin dysplasia" by consist of numerous denticles, containing whorls of Rushton in 1933,4 the clinical features of DDhave been osteodentin that block the normal course of the den- tinal tubules,s, 10,11 well described. -
Phenotypic and Genotypic Characterisation of Noonan-Like
1of5 ELECTRONIC LETTER J Med Genet: first published as 10.1136/jmg.2004.024091 on 2 February 2005. Downloaded from Phenotypic and genotypic characterisation of Noonan-like/ multiple giant cell lesion syndrome J S Lee, M Tartaglia, B D Gelb, K Fridrich, S Sachs, C A Stratakis, M Muenke, P G Robey, M T Collins, A Slavotinek ............................................................................................................................... J Med Genet 2005;42:e11 (http://www.jmedgenet.com/cgi/content/full/42/2/e11). doi: 10.1136/jmg.2004.024091 oonan-like/multiple giant cell lesion syndrome (NL/ MGCLS; OMIM 163955) is a rare condition1–3 with Key points Nphenotypic overlap with Noonan’s syndrome (OMIM 163950) and cherubism (OMIM 118400) (table 1). N Noonan-like/multiple giant cell lesion syndrome (NL/ Recently, missense mutations in the PTPN11 gene on MGCLS) has clinical similarities with Noonan’s syn- chromosome 12q24.1 have been identified as the cause of drome and cherubism. It is unclear whether it is a Noonan’s syndrome in 45% of familial and sporadic cases,45 distinct entity or a variant of Noonan’s syndrome or indicating genetic heterogeneity within the syndrome. In the cherubism. 5 study by Tartaglia et al, there was a family in which three N Three unrelated patients with NL/MGCLS were char- members had features of Noonan’s syndrome; two of these acterised, two of whom were found to have mutations had incidental mandibular giant cell lesions.3 All three in the PTPN11 gene, the mutation found in 45% of members were found to have a PTPN11 mutation known to patients with Noonan’s syndrome.