The Cardiovascular System 8
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General Physical Examination Skills for Ahps
General Physical Examination Techniques for the Rheumatology Clinic This manual has been developed as an overview of the general examination of the rheumatology patient. Certain details have been specifically omitted because they have no relevance when examining a stable out patient in the rheumatology clinic. Vital Signs 1. Heart rate 2. Blood pressure 3. Weight 1. Heart Rate There are two things you want to document when assessing heart rate: The actual rate and the rhythm. Rate: count pulse for at least 15 – 30 seconds (e.g., if you count the rate for 15 seconds, multiply this result by 4 to determine heart rate). The radial pulse is most commonly used to assess the heart rate. With the pads of your index and middle fingers, compress the radial artery until a pulsation is detected. Normal 60-100 bpm Bradycardia <60 bpm Tachycardia >100 bpm 2. Heart Rhythm The rhythm should be regular. A regularly irregular rhythm is one where the pulse is irregular but there is a pattern to the irregularity. For example every third beat is dropped. An irregularly irregular rhythm is one where the pulse is irregular and there is no pattern to the irregularity. The classic example of this is atrial fibrillation. 3. Blood Pressure Blood pressure should be measured in both arms, and should include an assessment of orthostatic change How to measure the Brachial Artery Blood Pressure: • Patient should be relaxed, in a supine or sitting position, with the arms positioned correctly(at, not above, the level of the heart) • Remember that measurements should be taken bilaterally • Choice of appropriate cuff size for the patient is important to accuracy of measured blood pressure. -
Blood Vessels: Part A
Chapter 19 The Cardiovascular System: Blood Vessels: Part A Blood Vessels • Delivery system of dynamic structures that begins and ends at heart – Arteries: carry blood away from heart; oxygenated except for pulmonary circulation and umbilical vessels of fetus – Capillaries: contact tissue cells; directly serve cellular needs – Veins: carry blood toward heart Structure of Blood Vessel Walls • Lumen – Central blood-containing space • Three wall layers in arteries and veins – Tunica intima, tunica media, and tunica externa • Capillaries – Endothelium with sparse basal lamina Tunics • Tunica intima – Endothelium lines lumen of all vessels • Continuous with endocardium • Slick surface reduces friction – Subendothelial layer in vessels larger than 1 mm; connective tissue basement membrane Tunics • Tunica media – Smooth muscle and sheets of elastin – Sympathetic vasomotor nerve fibers control vasoconstriction and vasodilation of vessels • Influence blood flow and blood pressure Tunics • Tunica externa (tunica adventitia) – Collagen fibers protect and reinforce; anchor to surrounding structures – Contains nerve fibers, lymphatic vessels – Vasa vasorum of larger vessels nourishes external layer Blood Vessels • Vessels vary in length, diameter, wall thickness, tissue makeup • See figure 19.2 for interaction with lymphatic vessels Arterial System: Elastic Arteries • Large thick-walled arteries with elastin in all three tunics • Aorta and its major branches • Large lumen offers low resistance • Inactive in vasoconstriction • Act as pressure reservoirs—expand -
Central Venous Pressure Venous Examination but Underestimates Ultrasound Accurately Reflects the Jugular
Ultrasound Accurately Reflects the Jugular Venous Examination but Underestimates Central Venous Pressure Gur Raj Deol, Nicole Collett, Andrew Ashby and Gregory A. Schmidt Chest 2011;139;95-100; Prepublished online August 26, 2010; DOI 10.1378/chest.10-1301 The online version of this article, along with updated information and services can be found online on the World Wide Web at: http://chestjournal.chestpubs.org/content/139/1/95.full.html Chest is the official journal of the American College of Chest Physicians. It has been published monthly since 1935. Copyright2011by the American College of Chest Physicians, 3300 Dundee Road, Northbrook, IL 60062. All rights reserved. No part of this article or PDF may be reproduced or distributed without the prior written permission of the copyright holder. (http://chestjournal.chestpubs.org/site/misc/reprints.xhtml) ISSN:0012-3692 Downloaded from chestjournal.chestpubs.org at UCSF Library & CKM on January 21, 2011 © 2011 American College of Chest Physicians CHEST Original Research CRITICAL CARE Ultrasound Accurately Refl ects the Jugular Venous Examination but Underestimates Central Venous Pressure Gur Raj Deol , MD ; Nicole Collett , MD ; Andrew Ashby , MD ; and Gregory A. Schmidt , MD , FCCP Background: Bedside ultrasound examination could be used to assess jugular venous pressure (JVP), and thus central venous pressure (CVP), more reliably than clinical examination. Methods: The study was a prospective, blinded evaluation comparing physical examination of external jugular venous pressure (JVPEXT), internal jugular venous pressure (JVPINT), and ultrasound collapse pressure (UCP) with CVP measured using an indwelling catheter. We com- pared the examination of the external and internal JVP with each other and with the UCP and CVP. -
Central Venous Pressure: Uses and Limitations
Central Venous Pressure: Uses and Limitations T. Smith, R. M. Grounds, and A. Rhodes Introduction A key component of the management of the critically ill patient is the optimization of cardiovascular function, including the provision of an adequate circulating volume and the titration of cardiac preload to improve cardiac output. In spite of the appearance of several newer monitoring technologies, central venous pressure (CVP) monitoring remains in common use [1] as an index of circulatory filling and of cardiac preload. In this chapter we will discuss the uses and limitations of this monitor in the critically ill patient. Defining Central Venous Pressure What is the Central Venous Pressure? Central venous pressure is the intravascular pressure in the great thoracic veins, measured relative to atmospheric pressure. It is conventionally measured at the junction of the superior vena cava and the right atrium and provides an estimate of the right atrial pressure. The Central Venous Pressure Waveform The normal CVP exhibits a complex waveform as illustrated in Figure 1. The waveform is described in terms of its components, three ascending ‘waves’ and two descents. The a-wave corresponds to atrial contraction and the x descent to atrial relaxation. The c wave, which punctuates the x descent, is caused by the closure of the tricuspid valve at the start of ventricular systole and the bulging of its leaflets back into the atrium. The v wave is due to continued venous return in the presence of a closed tricuspid valve. The y descent occurs at the end of ventricular systole when the tricuspid valve opens and blood once again flows from the atrium into the ventricle. -
Essentials of Bedside Cardiology CONTEMPORARY CARDIOLOGY
Essentials of Bedside Cardiology CONTEMPORARY CARDIOLOGY CHRISTOPHER P. CANNON, MD SERIES EDITOR Aging, Heart Disease and Its Management: Facts and Controversies, edited by Niloo M. Edwards, MD, Mathew S. Maurer, MD, and Rachel B. Wellner, MD, 2003 Peripheral Arterial Disease: Diagnosis and Treatment, edited by Jay D. Coffman, MD, and Robert T. Eberhardt, MD, 2003 Essentials ofBedside Cardiology: With a Complete Course in Heart Sounds and Munnurs on CD, Second Edition, by Jules Constant, MD, 2003 Primary Angioplasty in Acute Myocardial Infarction, edited by James E. Tcheng, MD,2002 Cardiogenic Shock: Diagnosis and Treatment, edited by David Hasdai, MD, Peter B. Berger, MD, Alexander Battler, MD, and David R. Holmes, Jr., MD, 2002 Management of Cardiac Arrhythmias, edited by Leonard I. Ganz, MD, 2002 Diabetes and Cardiovascular Disease, edited by Michael T. Johnstone and Aristidis Veves, MD, DSC, 2001 Blood Pressure Monitoring in Cardiovascular Medicine and Therapeutics, edited by William B. White, MD, 2001 Vascular Disease and Injury: Preclinical Research, edited by Daniell. Simon, MD, and Campbell Rogers, MD 2001 Preventive Cardiology: Strategies for the Prevention and Treatment of Coronary Artery Disease, edited by JoAnne Micale Foody, MD, 2001 Nitric Oxide and the Cardiovascular System, edited by Joseph Loscalzo, MD, phD and Joseph A. Vita, MD, 2000 Annotated Atlas of Electrocardiography: A Guide to Confident Interpretation, by Thomas M. Blake, MD, 1999 Platelet Glycoprotein lIb/IlIa Inhibitors in Cardiovascular Disease, edited by A. Michael Lincoff, MD, and Eric J. Topol, MD, 1999 Minimally Invasive Cardiac Surgery, edited by Mehmet C. Oz, MD and Daniel J. Goldstein, MD, 1999 Management ofAcute Coronary Syndromes, edited by Christopher P. -
Innocent (Harmless) Heart Murmurs in Children
JAMA PATIENT PAGE The Journal of the American Medical Association PEDIATRIC HEART HEALTH Innocent (Harmless) Heart Murmurs in Children murmur is the sound of blood flowing through the heart and the large blood vessels that carry the blood through the body. Murmurs can be a A sign of a congenital (from birth) heart defect or can provide clues to illnesses that start elsewhere in the body and make the heart work harder, such as anemia or fever. In children, murmurs are often harmless and are just the sound of a heart working normally. These harmless murmurs are often called innocent or functional murmurs. Murmurs are easily heard in children because they have thin chests and the heart is closer to the stethoscope. When children have fevers or are scared, their hearts beat faster and murmurs can become even louder than usual. TYPES OF INNOCENT MURMURS • Still murmur is usually heard at the left side of the sternum (breastbone), in line with the nipple. This murmur is harder to hear when a child is sitting or lying on his or her stomach. • Pulmonic murmur is heard as blood flows into the pulmonary artery (artery of the lungs). It is best heard between the first 2 ribs on the left side of the sternum. • Venous hum is heard as blood flows into the jugular veins, the large veins in the neck. It is heard best above the clavicles (collarbones). Making a child look down or sideways can decrease the murmur. CHARACTERISTICS OF INNOCENT MURMURS • They are found in children aged 3 to 7 years. -
Practical Cardiac Auscultation
LWW/CCNQ LWWJ306-08 March 7, 2007 23:32 Char Count= Crit Care Nurs Q Vol. 30, No. 2, pp. 166–180 Copyright c 2007 Wolters Kluwer Health | Lippincott Williams & Wilkins Practical Cardiac Auscultation Daniel M. Shindler, MD, FACC This article focuses on the practical use of the stethoscope. The art of the cardiac physical exam- ination includes skillful auscultation. The article provides the author’s personal approach to the patient for the purpose of best hearing, recognizing, and interpreting heart sounds and murmurs. It should be used as a brief introduction to the art of auscultation. This article also attempts to illustrate heart sounds and murmurs by using words and letters to phonate the sounds, and by presenting practical clinical examples where auscultation clearly influences cardiac diagnosis and treatment. The clinical sections attempt to go beyond what is available in standard textbooks by providing information and stethoscope techniques that are valuable and useful at the bedside. Key words: auscultation, murmur, stethoscope HIS article focuses on the practical use mastered at the bedside. This article also at- T of the stethoscope. The art of the cardiac tempts to illustrate heart sounds and mur- physical examination includes skillful auscul- murs by using words and letters to phonate tation. Even in an era of advanced easily avail- the sounds, and by presenting practical clin- able technological bedside diagnostic tech- ical examples where auscultation clearly in- niques such as echocardiography, there is still fluences cardiac diagnosis and treatment. We an important role for the hands-on approach begin by discussing proper stethoscope selec- to the patient for the purpose of evaluat- tion and use. -
Bates' Pocket Guide to Physical Examination and History Taking
Lynn S. Bickley, MD, FACP Clinical Professor of Internal Medicine School of Medicine University of New Mexico Albuquerque, New Mexico Peter G. Szilagyi, MD, MPH Professor of Pediatrics Chief, Division of General Pediatrics University of Rochester School of Medicine and Dentistry Rochester, New York Acquisitions Editor: Elizabeth Nieginski/Susan Rhyner Product Manager: Annette Ferran Editorial Assistant: Ashley Fischer Design Coordinator: Joan Wendt Art Director, Illustration: Brett MacNaughton Manufacturing Coordinator: Karin Duffield Indexer: Angie Allen Prepress Vendor: Aptara, Inc. 7th Edition Copyright © 2013 Wolters Kluwer Health | Lippincott Williams & Wilkins. Copyright © 2009 by Wolters Kluwer Health | Lippincott Williams & Wilkins. Copyright © 2007, 2004, 2000 by Lippincott Williams & Wilkins. Copyright © 1995, 1991 by J. B. Lippincott Company. All rights reserved. This book is protected by copyright. No part of this book may be reproduced or transmitted in any form or by any means, including as photocopies or scanned-in or other electronic copies, or utilized by any information storage and retrieval system without written permission from the copyright owner, except for brief quotations embodied in critical articles and reviews. Materials appear- ing in this book prepared by individuals as part of their official duties as U.S. government employees are not covered by the above-mentioned copyright. To request permission, please contact Lippincott Williams & Wilkins at Two Commerce Square, 2001 Market Street, Philadelphia PA 19103, via email at [email protected] or via website at lww.com (products and services). 9 8 7 6 5 4 3 2 1 Printed in China Library of Congress Cataloging-in-Publication Data Bickley, Lynn S. Bates’ pocket guide to physical examination and history taking / Lynn S. -
Heart Murmur, Incidental Finding
412 Heart Murmur, Incidental Finding (asymptomatic) mitral valve regurgitation. Technician Tips Count Respirations and Monitor Respiratory Relevant inclusion criteria for the trial that Teaching owners to keep a log of their pet’s Effort) demonstrated this effect were a vertebral resting respiratory rates can allow early detection heart sum > 10.5, an echocardiographic left of HF decompensation so that medications can SUGGESTED READING atrial–aortic ratio > 1.6, and left ventricular be adjusted and hopefully hospitalization for Atkins C, et al: ACVIM consensus statement. enlargement. acute HF can be avoided. Guidelines for the diagnosis and treatment of • ACE inhibition may have a positive effect on canine chronic valvular heart disease. J Vet Intern the time to development of stage C HF in Client Education Med 23:1142-1150, 2009. canine patients with left atrial enlargement Management of the veterinary patient with AUTHOR: Jonathan A. Abbott, DVM, DACVIM due to mitral valve regurgitation. chronic HF requires careful monitoring and EDITOR: Meg M. Sleeper, VMD, DACVIM • Evidence that medical therapy slows the relatively frequent adjustment of medical progression of HCM is lacking. therapy (see client education sheet: How to Client Education Heart Murmur, Incidental Finding Sheet Initial Database BASIC INFORMATION rate or body posture), short (midsystolic), single (unaccompanied by other abnormal • Thoracic radiographs may be considered Definition sounds), and small (not widely radiating). as the initial diagnostic test in small- to A heart murmur that is detected in the process medium-breed dogs with systolic murmurs of an examination that was not initially directed Etiology and Pathophysiology that are loudest over the mitral valve at the cardiovascular system • A heart murmur is caused by turbulent blood region. -
Auscultation of Abdominal Arterial Murmurs
Auscultation of abdominal arterial murmurs C. ARTHUR MYERS, D.O.,° Flint, Michigan publications. Goldblatt's4 work on renal hyperten- sion has stimulated examiners to begin performing The current interest in the diagnostic value of ab- auscultation for renal artery bruits in their hyper- dominal arterial bruits is evidenced by the number tensive patients. of papers and references to the subject appearing in Stenosis, either congenital or acquired, and aneu- the recent literature. When Vaughan and Thoreki rysms are responsible for the vast majority of audi- published an excellent paper on abdominal auscul- ble renal artery bruits (Fig. 2). One should be tation in 1939, the only reference they made to highly suspicious of a renal artery defect in a hy- arterial murmurs was that of the bruit of abdominal pertensive patient with an epigastric murmur. Moser aortic aneurysm. In more recent literature, however, and Caldwell5 have produced the most comprehen- there is evidence of increased interest in auscultat- sive work to date on auscultation of the abdomen ing the abdomen for murmurs arising in the celiac, in renal artery disease. In their highly selective superior mesenteric, splenic, and renal arteries. series of 50 cases of abdominal murmurs in which The purpose of this paper is to review some of aortography was performed, renal artery disease the literature referable to the subject of abdominal was diagnosed in 66 per cent of cases. Their con- murmurs, to present some cases, and to stimulate clusions were that when an abdominal murmur of interest in performing auscultation for abdominal high pitch is found in a patient with hypertension, bruits as a part of all physical examinations. -
Intra-Operative Auscultation of Heart and Lungs Sounds: the Importance of Sound Transmission
Intra-Operative Auscultation of more readily when stethoscopes are used. Loeb Heart and Lungs Sounds: (2) has reported that the response time to detect an abnormal value on an intraoperative The Importance of Sound monitor display and it was 61 seconds with 16% Transmission of the abnormal values not being recognized in 5 minutes. Whereas, Copper et al, (3) found the Anthony V. Beran, PhD* meantime between an event and detection with a stethoscope was 34 seconds. This Introduction suggests that changes in cardio-pulmonary function may be detected more readily with a Sometimes we put so much emphasis on stethoscope (1). Auscultation of heart and lung electronic monitoring devices we forget that sounds during perioperative period is useful our own senses often detect things before a only if the Esophageal Stethoscope provides machine can. Seeing condensation in airway strong, clear transmission of the sounds to the device or clear mask can serve to indicate the anesthesia provider. This study evaluates the presence of ventilation before the signal has sound transmission properties of several even reached the equipment. Sometimes the Esophageal Stethoscopes currently available in sense of smell can be the first thing to aid in the the market. detection of a disconnected airway device or circuit. Similarly, in some situations listening for Methods the presence of abnormal heart or airway sounds can help detect the onset of critical To evaluate the sound transmission properties incidents quicker than electronic monitors. But of the Esophageal Stethoscopes in vitro study in recent years the art of listening has changed was performed. A system that simulates the in the practice of Anesthesia. -
1. Intermittent Chest Pain: Angina: • Stable: (Caused By
CVS: 1. Intermittent chest pain: Angina: • Stable: (caused by chronic narrowing in one or more coronary arteries), episodes of pain are precipitated by exertion and may occur more readily when walking in cold or windy weather, after a large meal or while carrying a heavy load; the pain is promptly relieved by rest and/or sublingual glyceryl nitrate (GTN) spray, and typically lasts for less than 10 minutes. • unstable angina (caused by a sudden severe narrowing in a coronary artery), there is usually an abrupt onset or worsening of chest pain episodes that may occur on minimal exertion or at rest. • Retrosternal/ Progressive onset/ increase in intensity over 1–2 minutes/ Constricting, heavy/ Sometimes arm(s), neck, epigastrium/ Associated with breathlessness/ Intermittent, with episodes lasting 2–10 minutes/ Triggered by emotion, exertion, especially if cold, windy/ Relieved by rest, nitrates Mild to moderate. • Aggravated by thyroxine or drug-induced anemia, e.g. aspirin or NSAIDs Esophageal: • Retrosternal or epigastric/ Over 1–2 minutes; can be sudden (spasm)/ C: Gripping, tight or burning/ R: Often to back, sometimes to arms/ A: Heartburn, acid reflux/ T: Intermittent, often at night-time; variable duration/ Lying flat/some foods may trigger/ Not relieved by rest; nitrates sometimes relieve/ Usually mild but esophageal spasm can mimic myocardial infarction. 2. Acute chest pain: MI: • SOCRATES: Retrosternal/ Rapid over a few minutes/ Constricting, heavy/ Often to arm(s), neck, jaw, sometimes epigastrium/ Sweating, nausea, vomiting, breathlessness, feeling of impending death (angor animi)/ Acute presentation; prolonged duration/ ’Stress’ and exercise rare triggers, usually spontaneous/ Not relieved by rest or nitrates/ Usually severe.