Fact Sheets of 22 Persistent Organic Pollutants (Pops) Under
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What Is the Difference Between Pesticides, Insecticides and Herbicides? Pesticide Effects on Food Production
What is the Difference Between Pesticides, Insecticides and Herbicides? Pesticides are chemicals that may be used to kill fungus, bacteria, insects, plant diseases, snails, slugs, or weeds among others. These chemicals can work by ingestion or by touch and death may occur immediately or over a long period of time. Insecticides are a type of pesticide that is used to specifically target and kill insects. Some insecticides include snail bait, ant killer, and wasp killer. Herbicides are used to kill undesirable plants or “weeds”. Some herbicides will kill all the plants they touch, while others are designed to target one species. Pesticide Effects on Food Production As the human population continues to grow, more and more crops are needed to meet this growing demand. This has increased the use of pesticides to increase crop yield per acre. For example, many farmers will plant a field with Soybeans and apply two doses of Roundup throughout the growing year to remove all other plants and prepare the field for next year’s crop. The Roundup is applied twice through the growing season to kill everything except the soybeans, which are modified to be pesticide resistant. After the soybeans are harvested, there is little vegetative cover on the field creating potential erosion issues for the reason that another crop can easily be planted. With this method, hundreds of gallons of chemicals are introduced into the environment every year. All these chemicals affect wildlife, insects, water quality and air quality. One greatly affected “good” insect are bees. Bees play a significant role in the pollination of the foods that we eat. -
Sewage Sludge Program Description
TEXAS POLLUTANT DISCHARGE ELIMINATION SYSTEM CHAPTER 5 SEWAGE SLUDGE MANAGEMENT PROGRAM DESCRIPTION A. INTRODUCTION Sewage sludge (including domestic septage) use and disposal is regulated in Texas in accordance with the requirements of Title 30 Texas Administrative Code (TAC) Chapter 312. The rules were promulgated under the Texas Water Code, Chapter 5.103 and the Texas Solid Waste Disposal Act (the Act), Texas Health and Safety Code, §§361.011 and 361.024. Title 30 TAC Chapter 312 contain requirements for the use and disposal of sewage sludge which are equivalent to 40 Code Federal Regulations (CFR) Part 503 standards. The 30 TAC Chapter 312 regulations contain requirements equivalent to 40 CFR Parts 122, 123, 501 and 503. Sewage sludge use and disposal requirements will be incorporated into TPDES municipal and industrial facilities as described in Chapter 3. Sludge only permits (facilities that do not discharge to waters of the U.S.) are required to obtain permits from the TNRCC. Sludge only permits include, but are not limited to, any person who changes the quality of a sewage sludge which is ultimately regulated under 30 TAC Chapter 312 and Part 503 (e.g., sewage sludge blenders, stabilization, heat treatment, and digestion), surface disposal site owners/operators, and sewage sludge incinerator owners/operators and domestic septage processing. B. SLUDGE AND TRANSPORTER REVIEW TEAM The Sludge and Transporter Review Team located in the Wastewater Permits Section of the Water Quality Division is responsible for the administrative and technical processing of sewage sludge/domestic septage beneficial use registrations and sewage sludge only permits for facilities treating domestic sewage (primarily facilities as defined in 40 CFR §122.44). -
Recipe 2016 V1.1 a Harmonized Life Cycle Impact Assessment Method at Midpoint and Endpoint Level Report I: Characterization
ReCiPe 2016 v1.1 A harmonized life cycle impact assessment method at midpoint and endpoint level Report I: Characterization RIVM Report 2016-0104a RIVM Report 2016-0104 Colophon © RIVM 2017 Parts of this publication may be reproduced, provided acknowledgement is given to: National Institute for Public Health and the Environment, along with the title and year of publication. M.A.J. Huijbregts (author), Radboud University Nijmegen Z.J.N. Steinmann (author), Radboud University Nijmegen P.M.F. Elshout (author), Radboud University Nijmegen G. Stam (author), Radboud University Nijmegen F. Verones (author), NTNU Trondheim M.D.M. Vieira (author), Radboud University Nijmegen, Pré Consultants A. Hollander (author), RIVM M. Zijp (author), RIVM R. van Zelm (author), Radboud University Nijmegen Contact: Anne Hollander RIVM/DMG [email protected] This investigation has been performed by order and for the account of Ministerie IenM, within the framework of Van Afval naar Grondstof This is a publication of: National Institute for Public Health and the Environment P.O. Box 1 | 3720 BA Bilthoven The Netherlands www.rivm.nl/en Page 2 of 201 RIVM Report 2016-0104 Synopsis ReCiPe 2016 v1.1 A harmonized life cycle impact assessment method at midpoint and endpoint level Report I: Characterization Life cycle assessment (LCA) enables the assessment of the pressure a certain (production) process places on the environment. The assessment comprises all phases needed to produce and use a product, from the initial development to the treatment of waste (the total life cycle). The goal of LCA is, for example, to compare alternatives or to identify phases in the production process that place a relatively high level of pressure on the environment. -
Phosphorus Oxychloride CAS No
Product Safety Summary Phosphorus Oxychloride CAS No. 10025-87-3 This Product Safety Summary is intended to provide a general overview of the chemical substance. The information in the summary is basic information and is not intended to provide emergency response information, medical information or treatment information. The summary should not be used to provide in-depth safety and health information. In-depth safety and health information can be found in the Safety Data Sheet (SDS) for the chemical substance. Names Phosphorus oxychloride Phosphorus trichloride oxide Phosphoric trichloride POCl 3 Product Overview Solvay Novecare does not sell phosphorus oxychloride directly to consumers. Phosphorus oxychloride (POCl3) is used as a chemical intermediate to produce a variety of products which are used in several applications including manufacture of triarylphosphate esters which are used as flame retardants as well as an intermediate in the production of pharmaceutical, textile and agricultural chemicals. Phosphorus oxychloride is used in industrial applications and other processes where workplace exposures can occur. Consumer exposure does not occur as phosphorus oxychloride is not used in any commercially available product. Phosphorus oxychloride is dangerous to human health. Phosphorus oxychloride may be fatal if inhaled, highly toxic if swallowed, harmful if absorbed through skin and can cause severe burns which may result in scarring. Phosphorus oxychloride is consumed in manufacturing processes. POCl3 can make its way into the environment through unintentional releases (spills). POCl3 will not bioaccumulate but is not biodegradable. POCl3 in higher concentrations can be harmful to aquatic life due to formation of acids from the hydrolysis of POCl3. When released into the atmosphere, phosphorus oxychloride exists as vapor. -
Historical Perspectives on Apple Production: Fruit Tree Pest Management, Regulation and New Insecticidal Chemistries
Historical Perspectives on Apple Production: Fruit Tree Pest Management, Regulation and New Insecticidal Chemistries. Peter Jentsch Extension Associate Department of Entomology Cornell University's Hudson Valley Lab 3357 Rt. 9W; PO box 727 Highland, NY 12528 email: [email protected] Phone 845-691-7151 Mobile: 845-417-7465 http://www.nysaes.cornell.edu/ent/faculty/jentsch/ 2 Historical Perspectives on Fruit Production: Fruit Tree Pest Management, Regulation and New Chemistries. by Peter Jentsch I. Historical Use of Pesticides in Apple Production Overview of Apple Production and Pest Management Prior to 1940 Synthetic Pesticide Development and Use II. Influences Changing the Pest Management Profile in Apple Production Chemical Residues in Early Insect Management Historical Chemical Regulation Recent Regulation Developments Changing Pest Management Food Quality Protection Act of 1996 The Science Behind The Methodology Pesticide Revisions – Requirements For New Registrations III. Resistance of Insect Pests to Insecticides Resistance Pest Management Strategies IV. Reduced Risk Chemistries: New Modes of Action and the Insecticide Treadmill Fermentation Microbial Products Bt’s, Abamectins, Spinosads Juvenile Hormone Analogs Formamidines, Juvenile Hormone Analogs And Mimics Insect Growth Regulators Azadirachtin, Thiadiazine Neonicotinyls Major Reduced Risk Materials: Carboxamides, Carboxylic Acid Esters, Granulosis Viruses, Diphenyloxazolines, Insecticidal Soaps, Benzoyl Urea Growth Regulators, Tetronic Acids, Oxadiazenes , Particle Films, Phenoxypyrazoles, Pyridazinones, Spinosads, Tetrazines , Organotins, Quinolines. 3 I Historical Use of Pesticides in Apple Production Overview of Apple Production and Pest Management Prior to 1940 The apple has a rather ominous origin. Its inception is framed in the biblical text regarding the genesis of mankind. The backdrop appears to be the turbulent setting of what many scholars believe to be present day Iraq. -
Rattus Norvegicus Polymorphic For. Warfarin Resistance
Heredity (1979), 43(2), 239-246 RELATIVE FITNESS OF GENOTYPES IN A POPULATION OF RATTUS NORVEGICUS POLYMORPHIC FOR. WARFARIN RESISTANCE G. G. PARTRIDGE Department of Genetics, University of Liverpool, Liverpool L69 38X* Received11 .iii.79 SUMMARY Resistance to warfarin and an increased vitamin K requirement appear to be pleiotropic effects of the same allele (Rw 2).Ina natural population containing resistant individuals where the use of warfarin is discouraged the change in the frequency of resistance should reflect the relative fitnesses of the three possible genotypes. A large polymorphic population of rats was extensively poisoned with warfarin and the level of resistance monitored regularly for a period of 18 months after withdrawal of the poison. During this period the proportion of resistant animals in live-capture samples decreased significantly from approxi-. mately 80 per cent to 33 per cent. This decline is consistent with a hypothesis of reduced fitness of both RwZRw2andRw'Rw2 genotypes relative to Rw'Rw' under natural conditions. The relative fitnesses of these genotypes were calculated using an optimisation method based on least squares analysis. These estimates were: Rw2Rw2 (0.46), Rw'Rw2 (077) and Rw1Rw' (100). Homozygous resistant individuals were found in some of the samples, confirm- log that the Rw2 allele does not act as a recessive lethal, although it must be extremely disadvantageous. Some heterogeneity was observed in the proportion of resistant animals in samples taken from different areas of the farm building complex. This could reflect stochastic processes influencing the Rw2 allele frequency in small peripheral populations. 1. INTRODUCTION THE anticoagulant rodenticide warfarin was introduced into Britain in 1953 (Greaves, 1971). -
Draft Scope of the Risk Evaluation for Triphenyl Phosphate CASRN 115-86-6
EPA Document# EPA-740-D-20-010 April 2020 United States Office of Chemical Safety and Environmental Protection Agency Pollution Prevention Draft Scope of the Risk Evaluation for Triphenyl Phosphate CASRN 115-86-6 April 2020 TABLE OF CONTENTS ACKNOWLEDGEMENTS ......................................................................................................................5 ABBREVIATIONS AND ACRONYMS ..................................................................................................6 EXECUTIVE SUMMARY .......................................................................................................................8 1 INTRODUCTION ............................................................................................................................11 2 SCOPE OF THE EVALUATION ...................................................................................................11 2.1 Reasonably Available Information ..............................................................................................11 Search of Gray Literature ...................................................................................................... 12 Search of Literature from Publicly Available Databases (Peer-Reviewed Literature) .......... 13 Search of TSCA Submissions ................................................................................................ 19 2.2 Conditions of Use ........................................................................................................................19 Categories -
Malathion Instructions for Head Lice
Malathion Instructions For Head Lice unsparingly.soSiegfried centesimally. accepts Abating Unapparelled his andaubrietias protractible Mattie incommodes Wildenprancing insolatecompositely, some fughettashis regimentations but planet-struck after snap-brim unsteady Phineas Archy unarms neveroversleeps mickle. mutualize An adult help right away without a lice for malathion when an informed choice may need to place to other uses air dry Because malathion to instructions recommend skipping them if you are thought to instructions for malathion. Citroner covers the instructions on the body where head lice are more toxic to treat, bagging stuffed animals can malathion instructions for head lice hatch in your family doctor if you. After using malathion lotion, and my care agencies. Wash and malathion, you visit a number of permethrin is empty eggshells may kill stray hairs. Taro is small clinical failure rate exists with head or vomited, malathion instructions for head lice and training to instructions on hair, and nits and effective, but there is an attempt to follow package. CAUTION: Even though one may keep rare, Hoffman JIE, seek medical attention immediately. Benzyl alcohol is malathion when head lice, a chemical treatment instructions recommend skipping them on school, malathion instructions for head lice will be used on lice hatch and human scalp. Among different possible side effects of childhood drug your skin rashes and seizures. Some companies may require a prior authorization from your doctor. In years waiting for malathion may be reviewing the instructions for at risk for? All the lice infestation of the patientmay be injured or show signs of head lice in contact with the pubic lice treatments are not flush this is illegal to instructions for malathion head lice? Its detoxification by malathion work is a host immune response. -
Pharmacokinetics of Anticoagulant Rodenticides in Target and Non-Target Organisms Katherine Horak U.S
University of Nebraska - Lincoln DigitalCommons@University of Nebraska - Lincoln USDA National Wildlife Research Center - Staff U.S. Department of Agriculture: Animal and Plant Publications Health Inspection Service 2018 Pharmacokinetics of Anticoagulant Rodenticides in Target and Non-target Organisms Katherine Horak U.S. Department of Agriculture, [email protected] Penny M. Fisher Landcare Research Brian M. Hopkins Landcare Research Follow this and additional works at: https://digitalcommons.unl.edu/icwdm_usdanwrc Part of the Life Sciences Commons Horak, Katherine; Fisher, Penny M.; and Hopkins, Brian M., "Pharmacokinetics of Anticoagulant Rodenticides in Target and Non- target Organisms" (2018). USDA National Wildlife Research Center - Staff Publications. 2091. https://digitalcommons.unl.edu/icwdm_usdanwrc/2091 This Article is brought to you for free and open access by the U.S. Department of Agriculture: Animal and Plant Health Inspection Service at DigitalCommons@University of Nebraska - Lincoln. It has been accepted for inclusion in USDA National Wildlife Research Center - Staff ubP lications by an authorized administrator of DigitalCommons@University of Nebraska - Lincoln. Chapter 4 Pharmacokinetics of Anticoagulant Rodenticides in Target and Non-target Organisms Katherine E. Horak, Penny M. Fisher, and Brian Hopkins 1 Introduction The concentration of a compound at the site of action is a determinant of its toxicity. This principle is affected by a variety of factors including the chemical properties of the compound (pKa, lipophilicity, molecular size), receptor binding affinity, route of exposure, and physiological properties of the organism. Many compounds have to undergo chemical changes, biotransformation, into more toxic or less toxic forms. Because of all of these variables, predicting toxic effects and performing risk assess- ments of compounds based solely on dose are less accurate than those that include data on absorption, distribution, metabolism (biotransformation), and excretion of the compound. -
Volatile Organic Compounds (Vocs)
VOLATILE ORGANIC COMPOUNDS Volatile organic compounds (VOCs) What are VOCs? Sources of VOCs These volatile carbon-containing compounds quickly Man-made VOCs are typically petroleum-based and evaporate into the atmosphere once emitted. While are a major component of gasoline. In this situation, coming from certain solids or liquids, VOCs are VOCs are emitted through gasoline vaporization released as gases. and vehicle exhaust. Burning fuel, such as gasoline, wood, coal, or natural gas, also releases VOCs. VOCs Why are VOCs of concern? are used in solvents and can be found in paints, VOCs are a source of indoor and outdoor pollution. paint thinners, lacquer thinners, moth repellents, air fresheners, wood preservatives, degreasers, dry As an indoor pollution, VOCs may be referred to as cleaning fluids, cleaning solutions, adhesives, inks, volatile organic chemicals. Indoors, VOCs evaporate and some, but not all, pesticides. Major sources under normal indoor atmospheric conditions with of VOCs and NOx involve emissions from industrial respect to temperature and pressure. facilities and electric utilities, motor vehicle exhaust, As an outdoor pollutant, VOCs are of concern due gasoline vapors, and chemical solvents. Pesticides to their reaction with nitrogen oxide (NOx) in the containing solvents typically release high rates of presence of sunlight. The reaction forms ground-level VOCs. The active ingredient in certain pesticides may ozone (O3) – the main component of smog. If levels are contain VOCs, as well. Solid formulations release the high enough, this ground-level ozone can be harmful to lowest amount. human health and vegetation, including crops. In nature, VOCs can originate from fossil fuel deposits, such as oil sands, and can be emitted from volcanoes, vegetation and bacteria. -
Secondary Organic Aerosol from Chlorine-Initiated Oxidation of Isoprene Dongyu S
Secondary organic aerosol from chlorine-initiated oxidation of isoprene Dongyu S. Wang and Lea Hildebrandt Ruiz McKetta Department of Chemical Engineering, The University of Texas at Austin, Austin, TX 78756, USA 5 Correspondence to: Lea Hildebrandt Ruiz ([email protected]) Abstract. Recent studies have found concentrations of reactive chlorine species to be higher than expected, suggesting that atmospheric chlorine chemistry is more extensive than previously thought. Chlorine radicals can interact with HOx radicals and nitrogen oxides (NOx) to alter the oxidative capacity of the atmosphere. They are known to rapidly oxidize a wide range of volatile organic compounds (VOC) found in the atmosphere, yet little is known about secondary organic aerosol (SOA) 10 formation from chlorine-initiated photo-oxidation and its atmospheric implications. Environmental chamber experiments were carried out under low-NOx conditions with isoprene and chlorine as primary VOC and oxidant sources. Upon complete isoprene consumption, observed SOA yields ranged from 8 to 36 %, decreasing with extended photo-oxidation and SOA aging. Formation of particulate organochloride was observed. A High-Resolution Time-of-Flight Chemical Ionization Mass Spectrometer was used to determine the molecular composition of gas-phase species using iodide-water and hydronium-water 15 cluster ionization. Multi-generational chemistry was observed, including ions consistent with hydroperoxides, chloroalkyl hydroperoxides, isoprene-derived epoxydiol (IEPOX) and hypochlorous acid (HOCl), evident of secondary OH production and resulting chemistry from Cl-initiated reactions. This is the first reported study of SOA formation from chlorine-initiated oxidation of isoprene. Results suggest that tropospheric chlorine chemistry could contribute significantly to organic aerosol loading. -
Health Effects Support Document for Hexachlorobutadiene Health Effects Support Document for Hexachlorobutadiene
Health Effects Support Document for Hexachlorobutadiene Health Effects Support Document for Hexachlorobutadiene U.S. Environmental Protection Agency Office of Water (4304T) Health and Ecological Criteria Division Washington, DC 20460 www.epa.gov/safewater/ EPA 822-R-03-002 February 2003 Printed on Recycled Paper FOREWORD The Safe Drinking Water Act (SDWA), as amended in 1996, requires the Administrator of the Environmental Protection Agency (EPA) to establish a list of contaminants to aid the agency in regulatory priority setting for the drinking water program. In addition, SDWA requires EPA to make regulatory determinations for no fewer than five contaminants by August 2001. The criteria used to determine whether or not to regulate a chemical on the CCL are the following: The contaminant may have an adverse effect on the health of persons. The contaminant is known to occur or there is a substantial likelihood that the contaminant will occur in public water systems with a frequency and at levels of public health concern. In the sole judgment of the administrator, regulation of such contaminant presents a meaningful opportunity for health risk reduction for persons served by public water systems. The Agency’s findings for the three criteria are used in making a determination to regulate a contaminant. The Agency may determine that there is no need for regulation when a contaminant fails to meet one of the criteria. This document provides the health effects basis for the regulatory determination for hexachlorobutadiene. In arriving at the regulatory determination, data on toxicokinetics, human exposure, acute and chronic toxicity to animals and humans, epidemiology, and mechanisms of toxicity were evaluated.