Table of Communicable Diseases

Total Page:16

File Type:pdf, Size:1020Kb

Table of Communicable Diseases TABLE OF COMMUNICABLE DISEASES Disease Signs & symptoms Incubation Communicability Prevention Chicken pox – Esp seen winter & spring. Resp 10-21 days Thru inhalation of Mask patient. Provider varicella zoster symptoms, malaise (not feeling well), airborne droplets should avoid contact if virus; viral low-grade fever followed by rash & direct contact they’ve never had disease starting on face & trunk spreading to of weeping chicken pox. rest of body. Fluid filled vesicles lesions & Vaccination now rupture & scab over within 1 week. contaminated available (1995) and part linens. of childhood immunizations. Pt isolated until all lesions crusted over and dry. Common cold >200 strains of viruses cause the 12 hours – 5 Direct contact, Handwashing (viral rhinitis) common cold. Course mild, often days (average 48 airborne droplet, without fever and without muscle hours) contaminated aching. hands and linens. Conjunctivitis The clinical syndrome begins with 24-72 hours Contact with Good personnel (pink eye) tearing, irritation & redness of eye(s) discharge or hygiene. Daily followed by edema of lids, upper respiratory laundering of bed linens photophobia (light sensitivity) & pus tract of infected including pillowcase and drainage. Course lasts from 2 days up persons (fingers, towels. Use wash cloth to 2-3 weeks. clothing, eye on unaffected eye first make-up). and then launder after Communicable use. No school during during course of acute stage. Tx with active infection. antibiotic eye medications. Hepatiti s – Signs & symptoms generally same for Most important is inflammation of all forms: avoidance of contact the liver due to Headache; fever; weakness; joint with blood and body multiple causes pain; anorexia; nausea; vomiting; fluids of all persons. (virus most RUQ pain; jaundice; dark urine; clay- common) colored stools Hepatitis A – May have no symptoms. Adults may 15-50 days; Fecal-oral route. Vaccines in active areas infectious or viral have abdominal pain, loss of appetite, average 30 days. Virus lasts on (active immunity). Good nausea, diarrhea, light colored stools, Disease follows hands about 4 handwashing. dark urine, fatigue, fever & jaundice. mild course & hours. More Disease Signs & symptoms Incubation Communicability Prevention Hepatitis A lasts 2-6 weeks comm. latter half There is no long term of incubation & chronic infection. most during 1 st week of symptoms Hepatitis B – It can take 1-9 months before 4-25 weeks; Direct contact Vaccination 90% serum hepatitis symptoms develop. Some have mild average 8-12 (blood, semen, effective. Virus stable on flu-like symptoms. Dark urine, light weeks vaginal fluid, surfaces with dried blood colored stools, fatigue, fever & saliva). Can for 7 days. jaundice. Can develop acute hepatitis, become cirrhosis, liver cancer. asymptomatic chronic carrier capable of transmitting disease to others. Hepatitis C Chronic condition in 85% of infected 2-25 weeks; avge Contact with Since 1989 screen blood Leading cause of people. Liver fibrosis into cirrhosis in 7-9 weeks. infected blood for HCV. No vaccine due cirrhosis & liver 20% of infected people. Disease may be primarily with IV to high mutation rate. cancer. dormant 10-20 years drug use & before symptoms. sexual contact. HIV – a virus that Mono-like syndrome, fatigue, fever, Variable. May Bloodborne Universal standard attacks the sore throat, lymphadenopathy, develop through blood & precautions immune system & splenomegaly, rash, diarrhea. Skin detectable body fluids causes AIDS (a lesions (Kaposi’s sarcoma); antibodies 1-3 Death is usually from the collection of signs opportunistic infections (Pneumocystic months. Variable opportunistic diseases & symptoms) carinii pneumonia, Tb) time from HIV that take advantage of infection to the patient’s weakened diagnosis of systems. AIDS. Influenza (flu) Epidemics usually in winter. Sudden 1-4 days Direct contact Vaccination available onset fever for 3-5 days, chills, especially in annually; most effective Viral disease tiredness, malaise (not feeling well), Peak flu season crowded areas if received from musculoskeletal aches, nasal is late December via airborne. The September to mid- discharge, dry cough, mild sore throat. through March. virus can persist November. Children can also experience GI on surfaces for Treatment is symptoms of nausea, vomiting & hours but indirect symptomatic (rest, fluids, diarrhea although this is uncommon in contact is less OTC med for fever & Disease Signs & symptoms Incubation Communicability Prevention Influenza adults. “Stomach flu” with GI common. aches). symptoms is caused by other viruses. Contagious 1 day prior to being sick up to 3-7 days after 1 st symptom. Measles Initially symptoms of severe cold with 7-14 days; Inhalation of Handwashing critical. (rubeola, hard fever, conjunctivitis, swollen eyelids, average 10 days infective droplets MMR vaccination part of measles) photophobia, malaise, cough, & direct contact. childhood program. nasopharyngeal congestion, red Highly bumpy rash lasting about 6 days communicable virus mostly before prodrome starts (early or impending disease time), to about 4 days after rash appears. Meningi tis – Viral meningitis – most common type 2-4 days up to 10 Resp droplets; Practice good inflammation of of meningitis; self-limited disease days contact with oral handwashing. Mask for meninges caused lasting 7-10 days. secretions, pt and self. Universal by bacteria & Bacterial – very serious infection; crowding, close precautions. Post viruses fever, chills, headache, nuchal rigidity contact, smoking, exposure antibiotics (stiff neck) with flexion, arthralgia lower started within 24 hours. (achy joints), lethargy, malaise (ill socioeconomic Vaccination now part of feeling), altered mental status, status. Viral childhood series vomiting, seizures. meningitis can (Haemophilus influenza also be spread type B). via contact with feces of infected person. Monkeypox Rare viral disease. 12 days after 12 days From an animal No specific treatment. exposure get fever, headache, muscle with monkeypox Possibly the smallpox aches, backache, swollen lymph if bitten or touch vaccine to prevent nodes, tired. Rash 1-3 days after the animal’s against getting. Disease Signs & symptoms Incubation Communicability Prevention fever; often starts on face as fluid filled blood, body Monkeypox bumps & the spreads. fluids, or its rash. Person-to-person from large respiratory droplets during long periods of face-to-face contact or touching body fluids or contaminated objects of infected persons. MRSA – Usually found in ill patients who are Usually spread Handwashing after any methicillin multidrug resistant. Often in open from infected patient contact. Wear resistant wounds, post-op wounds, around G- patients via gloves when doing pt staphylococcus tube sites. hands of HCW & contact. Protective aureus inanimate objects gowns when in contact (B/P cuff, with infected linens. stethoscope). Avoid sharing of equipment. HCW can be colonized with MRSA (not common) but often are not ill & are not at risk to other healthy persons (peers, family). Mumps Painful enlargement of salivary 12-25 days Resp droplets & Standard BSI. (Acute viral glands. Feverish cold followed by direct contact MMR vaccination is disease) swelling & stiffening of parotid salivary with saliva of standard for childhood gland in front of ear. Often bilateral. infected pt. immunizations. Adults Earache, difficulty chewing & Communicable 3 born after 1956 should swallowing. Glands tender to days before to get at least 1 dose of palpation. about 4 days MMR. after symptoms start. Risk of contracting Disease Signs & symptoms Incubation Communicability Prevention mumps disease is minimal. Pertussis – 1st phase – common cold symptoms 6-20 days Transmitted via Mask pt. whooping cough lasts 1-2 weeks. 2 nd phase lasts respiratory DPT vaccination in month or longer. No fever. Mild cough secretions or in childhood series (not that can become severe & violent, an aerosolized sure how long immunity productive. 3 rd phase – frequency and form. Highly lasts). severity of coughing decreases. contagious except in 3rd phase. Communicability greatest before 2nd phase. Pneumonia Chills, high fever, dyspnea, pleuritic Highest risk are Masks. Vaccination chest pain worsened by deep the non-healthy available esp for children inspiration, cough, crackles & populations <2 years old and adults wheezes heard on breath sounds >65 and for those post- splenectomy. Rubella – Generally milder than measles. Sore 12-19 days Inhalation of Mask pt. German throat, low grade fever. Fine pink rash infective droplets MMR vaccination part of measles ; virus on face, trunk & extremities lasting childhood program. about 3 days. SARS (severe Viral disease. Fever >100.4 oF, chills, Typically 2-7 Respiratory Fit tested N-95 acute respiratory headache, body achiness, respiratory days up to 10 droplets when respirators for caregivers syndrome) complaints (cough, SOB, dyspnea, days coughing or within 6 feet of patient. pneumonia), pulse ox <94% room air, sneezing droplets Patient to also wear N- travel within 10 days of symptoms to into air. Can 95 mask. Caregivers to Ontario, Canada, People’s Republic of touch infectious wear gloves, gowns, China, Vietnam, Taiwan, &/or material on goggles, and face Singapore OR close contact with environmental shields. Proper symptomatic person within 10 days of surfaces and handwashing extremely symptoms. bring to your important. Wear eyes, nose, protective gear when Disease Signs & symptoms Incubation
Recommended publications
  • Unilateral Hyperhidrosis Associated with Underlying Intrathoracic Neoplasia
    Thorax: first published as 10.1136/thx.41.10.814 on 1 October 1986. Downloaded from Thorax 1986;41:814-815 Unilateral hyperhidrosis associated with underlying intrathoracic neoplasia D C LINDSAY, J G FREEMAN, C 0 RECORD From the Department ofMedicine, Royal Victoria Infirmary, and University ofNewcastle upon Tyne Intrathoracic neoplasia is notable for the many ways in wall. There were metastatic plaques in the right hemithorax which it may present. We would like to report two cases and abdominal cavity but no evidence of metastases in the demonstrating a rare association between unilateral local- brain or the spinal cord. ised hyperhidrosis of the thoracic cage and underlying intra- thoracic neoplasm. Discussion Case reports The association of intrathoracic malignancy with sym- pathetic neurological complications, especially Homer's CASE 1 syndrome, is well recognised, particularly in the case of A 67 year old retired shotblaster complained ofa 3 kg weight tumours occurring at the thoracic inlet. Unilateral hyper- loss, mild dyspnoea, chest pain localised to the right costal hidrosis is an unusual phenomenon which has been reported margin, and profuse sweating localised to an area below the sporadically in association with various conditions, includ- right scapula. He smoked 15 cigarettes per day. Examination ing intracranial malignancy, encephalitis, syringomyelia, confirmed a right sided localised band of sweating at the trauma, neuritis, cervical rib, osteoma of the dorsal spine, level ofT6-9 posteriorly. Apart from minimal winging ofthe and chickenpox; in several cases no obvious underlying right scapula and some wasting of the right suprascapular cause has been evident. muscles no abnormal neurological signs were detected.
    [Show full text]
  • Unraveling the Complexity of Chronic Pain and Fatigue
    UNRAVELING THE COMPLEXITY OF CHRONIC PAIN AND FATIGUE LUCINDA BATEMAN, MD & BRAYDEN YELLMAN, MD © UNIVERSITY OF UTAH HEALTH SESSION #3 Effective use of evidence-based clinical diagnostic criteria and symptom management approaches to improve patient outcomes © UNIVERSITY OF UTAH HEALTH THE RATIONALE FOR USING EVIDENCE-BASED CLINICAL DIAGNOSTIC CRITERIA • Widespread pain amplification disorders – 1990 ACR fibromyalgia – 2016 ACR fibromyalgia criteria • Orthostatic Intolerance Disorders – POTS, NMH, OH, CAN, NOH… • ME/CFS 2015 IOM/NAM criteria © UNIVERSITY OF UTAH HEALTH PAIN AMPLIFICATION DISORDERS EX: FIBROMYALGIA ACR 1990 Chronic (>3 months) Widespread Pain (pain in 4 quadrants of body & spine) and Tenderness (>11/18 tender points) PAIN= stiffness, achiness, sharp shooting pains…tingling and numbness…light and sound sensitivity…in muscles, joints, bowel, bladder, pelvis, chest, head… FATIGUE, COGNITIVE and SLEEP disturbances are described in Wolfe et al but were not required for dx. Wolfe F, et al. The American College of Rheumatology 1990 criteria for the classification of fibromyalgia: report of the Multicenter Criteria Committee. Arthritis Rheum 1990;33:160–72 © UNIVERSITY OF UTAH HEALTH FIBROMYALGIA 1990 ACR CRITERIA Pain in four quadrants and the spine © UNIVERSITY OF UTAH HEALTH FIBROMYALGIA 2016 ACR CRITERIA 2016 Revisions to the 2010/2011 fibromyalgia diagnostic criteria, Seminars in Arthritis and Rheumatism. Volume 46, Issue 3. www.semarthritisrheumatism.com/article/S0049-0172(16)30208-6 © UNIVERSITY OF UTAH HEALTH FM IS OFTEN FOUND COMORBID WITH OTHER CONDITIONS Examples of the prevalence of fibromyalgia by 1990 criteria among various groups: General population 2% Women 4% Healthy Men 0.1% IM & Rheum clinics 15% IBS 13% Hemodialysis 6% Type 2 diabetes 15-23% Prevalence of fibromyalgia and co-morbid bipolar disorder: A systematic review and meta-analysis.
    [Show full text]
  • Measles Diagnostic Tool
    Measles Prodrome and Clinical evolution E Fever (mild to moderate) E Cough E Coryza E Conjunctivitis E Fever spikes as high as 105ºF Koplik’s spots Koplik’s Spots E E Viral enanthem of measles Rash E Erythematous, maculopapular rash which begins on typically starting 1-2 days before the face (often at hairline and behind ears) then spreads to neck/ the rash. Appearance is similar to “grains of salt on a wet background” upper trunk and then to lower trunk and extremities. Evolution and may become less visible as the of rash 1-3 days. Palms and soles rarely involved. maculopapular rash develops. Rash INCUBATION PERIOD Fever, STARTS on face (hairline & cough/coryza/conjunctivitis behind ears), spreads to trunk, Average 8-12 days from exposure to onset (sensitivity to light) and then to thighs/ feet of prodrome symptoms 0 (average interval between exposure to onset rash 14 day [range 7-21 days]) -4 -3 -2 -1 1234 NOT INFECTIOUS higher fever (103°-104°) during this period rash fades in same sequence it appears INFECTIOUS 4 days before rash and 4 days after rash Not Measles Rubella Varicella cervical lymphadenopathy. Highly variable but (Aka German Measles) (Aka Chickenpox) Rash E often maculopapular with Clinical manifestations E Clinical manifestations E Generally mild illness with low- Mild prodrome of fever and malaise multiforme-like lesions and grade fever, malaise, and lymph- may occur one to two days before may resemble scarlet fever. adenopathy (commonly post- rash. Possible low-grade fever. Rash often associated with painful edema hands and feet. auricular and sub-occipital).
    [Show full text]
  • Medicine in 18Th and 19Th Century Britain, 1700-1900
    Medicine in 18th and 19th century Britain, 1700‐1900 The breakthroughs th 1798: Edward Jenner – The development of How had society changed to make medical What was behind the 19 C breakthroughs? Changing ideas of causes breakthroughs possible? vaccinations Jenner trained by leading surgeon who taught The first major breakthrough came with Louis Pasteur’s germ theory which he published in 1861. His later students to observe carefully and carry out own Proved vaccination prevented people catching smallpox, experiments proved that bacteria (also known as microbes or germs) cause diseases. However, this did not put an end The changes described in the Renaissance were experiments instead of relying on knowledge in one of the great killer diseases. Based on observation and to all earlier ideas. Belief that bad air was to blame continued, which is not surprising given the conditions in many the result of rapid changes in society, but they did books – Jenner followed these methods. scientific experiment. However, did not understand what industrial towns. In addition, Pasteur’s theory was a very general one until scientists begun to identify the individual also build on changes and ideas from earlier caused smallpox all how vaccination worked. At first dad bacteria which cause particular diseases. So, while this was one of the two most important breakthroughs in ideas centuries. The flushing toilet important late 19th C invention wants opposition to making vaccination compulsory by law about what causes disease and illness it did not revolutionise medicine immediately. Scientists and doctors where the 1500s Renaissance – flushing system sent waste instantly down into – overtime saved many people’s lives and wiped‐out first to be convinced of this theory, but it took time for most people to understand it.
    [Show full text]
  • Bradford Hill Criteria for Causal Inference Based on a Presentation at the 2015 ANZEA Conference
    Bradford Hill Criteria for Causal Inference Based on a presentation at the 2015 ANZEA Conference Julian​ King Director,​ Julian King & Associates – a member of the Kinnect Group www.julianking.co.nz​ | www.kinnect.co.nz 2015 We think we’re good at determining causality, but we suck at it One​ of the great At​ one level, this is an Unfortunately,​ though, the challenges in evaluation is everyday, common sense way we are wired does not determining whether the task. As a species we’ve predispose us to logical results we’re seeing are been making judgments thinking. We are inclined because of the program about causation for a to be led astray by all sorts we’re evaluating, some million years or so. of biases and heuristics. other influences out there in the big world, or random chance. The​ Kinnect Group www.kinnect.co.nz 2 Along came logic Eventually,​ after a very long time, we evolved into philosophers who invented formal logic. Thanks to scientific method, our species has recently triumphed to the extent that we now have cars that drive themselves and flying drones that deliver pizza (don’t confuse this with progress – we still suck at ethics but that’s a story for another day). 3 But we’re still not great at causation Over​ time, the rocket But​ deep down we’re still Such​ a rigid view is not science for dealing with biased, heuristical beings much use in the real causation has become and not very good at world, where there are all more sophisticated – a thinking things through.
    [Show full text]
  • Medical Bacteriology
    LECTURE NOTES Degree and Diploma Programs For Environmental Health Students Medical Bacteriology Abilo Tadesse, Meseret Alem University of Gondar In collaboration with the Ethiopia Public Health Training Initiative, The Carter Center, the Ethiopia Ministry of Health, and the Ethiopia Ministry of Education September 2006 Funded under USAID Cooperative Agreement No. 663-A-00-00-0358-00. Produced in collaboration with the Ethiopia Public Health Training Initiative, The Carter Center, the Ethiopia Ministry of Health, and the Ethiopia Ministry of Education. Important Guidelines for Printing and Photocopying Limited permission is granted free of charge to print or photocopy all pages of this publication for educational, not-for-profit use by health care workers, students or faculty. All copies must retain all author credits and copyright notices included in the original document. Under no circumstances is it permissible to sell or distribute on a commercial basis, or to claim authorship of, copies of material reproduced from this publication. ©2006 by Abilo Tadesse, Meseret Alem All rights reserved. Except as expressly provided above, no part of this publication may be reproduced or transmitted in any form or by any means, electronic or mechanical, including photocopying, recording, or by any information storage and retrieval system, without written permission of the author or authors. This material is intended for educational use only by practicing health care workers or students and faculty in a health care field. PREFACE Text book on Medical Bacteriology for Medical Laboratory Technology students are not available as need, so this lecture note will alleviate the acute shortage of text books and reference materials on medical bacteriology.
    [Show full text]
  • Molecular Pathological Epidemiology in Diabetes Mellitus and Risk of Hepatocellular Carcinoma
    Submit a Manuscript: http://www.wjgnet.com/esps/ World J Hepatol 2016 September 28; 8(27): 1119-1127 Help Desk: http://www.wjgnet.com/esps/helpdesk.aspx ISSN 1948-5182 (online) DOI: 10.4254/wjh.v8.i27.1119 © 2016 Baishideng Publishing Group Inc. All rights reserved. REVIEW Molecular pathological epidemiology in diabetes mellitus and risk of hepatocellular carcinoma Chun Gao Chun Gao, Department of Gastroenterology, China-Japan logy and epidemiology, and investigates the relationship Friendship Hospital, Ministry of Health, Beijing 100029, China between exogenous and endogenous exposure factors, tumor molecular signatures, and tumor initiation, progres- Author contributions: Gao C conceived the topic, performed sion, and response to treatment. Molecular epidemiology research, retrieved concerned literatures and wrote the paper. broadly encompasses MPE and conventional-type mole- cular epidemiology. Hepatocellular carcinoma (HCC) Supported by Beijing NOVA Programme of Beijing Municipal is the third most common cause of cancer-associated Science and Technology Commission, No. Z13110.7000413067. death worldwide and remains as a major public health Conflict-of-interest statement: No conflict of interest. challenge. Over the past few decades, a number of epidemiological studies have demonstrated that diabetes Open-Access: This article is an open-access article which was mellitus (DM) is an established independent risk factor selected by an in-house editor and fully peer-reviewed by external for HCC. However, how DM affects the occurrence and
    [Show full text]
  • Foundations of Epidemiology
    66221_CH01_5398.qxd 6/19/09 11:16 AM Page 1 © Jones and Bartlett Publishers, LLC. NOT FOR SALE OR DISTRIBUTION CHAPTER 1 Foundations of Epidemiology OBJECTIVES After completing this chapter, you will be able to: ■ Define epidemiology. ■ Define descriptive epidemiology. ■ Define analytic epidemiology. ■ Identify some activities performed in epidemiology. ■ Explain the role of epidemiology in public health practice and individual decision making. ■ Define epidemic, endemic, and pandemic. ■ Describe common source, propagated, and mixed epidemics. ■ Describe why a standard case definition and adequate levels of reporting are important in epidemiologic investigations. ■ Describe the epidemiology triangle for infectious disease. ■ Describe the advanced epidemiology triangle for chronic diseases and behavioral disorders. ■ Define the three levels of prevention used in public health and epidemiology. ■ Understand the basic vocabulary used in epidemiology. 66221_CH01_5398.qxd 6/19/09 11:16 AM Page 2 © Jones and Bartlett Publishers, LLC. NOT FOR SALE OR DISTRIBUTION 2 CHAPTER 1 ■ Foundations of Epidemiology In recent years, the important role of epidemiology has become increasingly recognized. Epidemiology is a core subject required in public health and health education programs; it is a study that provides information about public health problems and the causes of those problems. This information is then used to improve the health and social conditions of people. Epidemiology has a population focus in that epidemiologic investigations are concerned with the collective health of the people in a community or population under study. In contrast, a clinician is concerned for the health of an individual. The clinician focuses on treating and caring for the patient, whereas the epidemiologist focuses on iden- tifying the source or exposure of disease, disability or death, the number of persons exposed, and the potential for further spread.
    [Show full text]
  • Molecular Pathological Epidemiology Gives Clues to Paradoxical Findings
    Molecular Pathological Epidemiology Gives Clues to Paradoxical Findings The Harvard community has made this article openly available. Please share how this access benefits you. Your story matters Citation Nishihara, Reiko, Tyler J. VanderWeele, Kenji Shibuya, Murray A. Mittleman, Molin Wang, Alison E. Field, Edward Giovannucci, Paul Lochhead, and Shuji Ogino. 2015. “Molecular Pathological Epidemiology Gives Clues to Paradoxical Findings.” European Journal of Epidemiology 30 (10): 1129–35. https://doi.org/10.1007/ s10654-015-0088-4. Citable link http://nrs.harvard.edu/urn-3:HUL.InstRepos:41392032 Terms of Use This article was downloaded from Harvard University’s DASH repository, and is made available under the terms and conditions applicable to Open Access Policy Articles, as set forth at http:// nrs.harvard.edu/urn-3:HUL.InstRepos:dash.current.terms-of- use#OAP HHS Public Access Author manuscript Author Manuscript Author ManuscriptEur J Epidemiol Author Manuscript. Author Author Manuscript manuscript; available in PMC 2016 October 07. Published in final edited form as: Eur J Epidemiol. 2015 October ; 30(10): 1129–1135. doi:10.1007/s10654-015-0088-4. Molecular Pathological Epidemiology Gives Clues to Paradoxical Findings Reiko Nishiharaa,b,c, Tyler J. VanderWeeled,e, Kenji Shibuyac, Murray A. Mittlemand,f, Molin Wangd,e,g, Alison E. Fieldd,g,h,i, Edward Giovannuccia,d,g, Paul Lochheadi,j, and Shuji Oginob,d,k aDepartment of Nutrition, Harvard T.H. Chan School of Public Health, 655 Huntington Ave., Boston, Massachusetts 02115 USA bDepartment of Medical Oncology, Dana-Farber Cancer Institute and Harvard Medical School, 450 Brookline Ave., Boston, Massachusetts 02215 USA cDepartment of Global Health Policy, Graduate School of Medicine, The University of Tokyo, 7-3-1, Hongo, Bunkyo-ku, Tokyo, Japan dDepartment of Epidemiology, Harvard T.H.
    [Show full text]
  • HIV and AIDS: a Module for Cnas and Hhas
    HIV AND AIDS: A Module for CNAs AND HHAs INTRODUCTION The human immunodeficiency virus (HIV) is a virus that is transmitted through sexual contact or contact with infected blood. HIV causes an illness called acquired immune deficiency syndrome - AIDS. AIDS was first diagnosed in New York City and San Francisco in 1981. However, there is evidence that HIV has actually been infecting people for many, many years before it was recognized and isolated cases of AIDS had occurred many years before the epidemic started. The cause of AIDS, HIV, was finally isolated in 1983. There are now over 33 million people in the world who are infected with HIV. A large number of them will develop AIDS, and AIDS is an enormous public health problem in the United States and worldwide. Modern medications have now allowed many people infected with HIV to avoid developing AIDS. However, there is no cure for HIV infection - the virus cannot be eliminated or eradicated - and there is still no vaccine available that can prevent the spread of HIV. Learning Break: Many people use the terms HIV and AIDS interchangeably, but it is important to remember that they are two different things. HIV is the virus that causes AIDS. AIDS is the disease - or group of diseases - that are caused by infection with HIV. Someone can be infected with HIV but not have AIDS. OBJECTIVES When the student has finished this module, he/she will be able to: 1. Identify a definition of HIV. 2. Identify a definition of AIDS. 3. Identify the basic defense mechanism the body uses against infections.
    [Show full text]
  • Botox® Reconstitution and Dilution Procedures
    BOTOX® RECONSTITUTION AND DILUTION PROCEDURES Indication Chronic Migraine BOTOX® (onabotulinumtoxinA)for injection is indicated for the prophylaxis of headaches in adult patients with chronic migraine (≥ 15 days per month with headache lasting 4 hours a day or longer). Important Limitations Safety and effectiveness have not been established for the prophylaxis of episodic migraine (14 headache days or fewer per month) in 7 placebo-controlled studies. IMPORTANT SAFETY INFORMATION, INCLUDING BOXED WARNING WARNING: DISTANT SPREAD OF TOXIN EFFECT Postmarketing reports indicate that the effects of BOTOX® and all botulinum toxin products may spread from the area of injection to produce symptoms consistent with botulinum toxin effects. These may include asthenia, generalized muscle weakness, diplopia, ptosis, dysphagia, dysphonia, dysarthria, urinary incontinence, and breathing difficulties. These symptoms have been reported hours to weeks after injection. Swallowing and breathing difficulties can be life threatening, and there have been reports of death. The risk of symptoms is probably greatest in children treated for spasticity, but symptoms can also occur in adults treated for spasticity and other conditions, particularly in those patients who have an underlying condition that would predispose them to these symptoms. In unapproved uses, including spasticity in children, and in approved indications, cases of spread of effect have been reported at doses comparable to those used to treat cervical dystonia and upper limb spasticity and at lower
    [Show full text]
  • Colonization V. Infection
    Colonization vs Infection Colonization • The presence of microorganisms in or on a host with growth and multiplication but without tissue invasion or damage • Understanding this concept is essential in the planning and implantation of epidemiological studies in a healthcare infection prevention and control program Infection v. Colonization • Confusing colonization with infection can lead to spurious associations that may lead to expensive, ineffective, and time‐ consuming interventions Multi Drug‐Resistant Organisms Management in Long Term Care • Colonization may become infection when changes in the host Facilities Workshop occur Louisiana Office of Public Health Healthcare‐Associated Infections Program Objectives Colonization: Definition By the end of the presentation, attendees will be able to: • Colonization: presence of a microorganism on/in a host, with • Define colonization growth and multiplication of the organism, but without • Differentiate colonization from infections interaction between host and organism (no clinical expression, no immune response). • Apply appropriate laboratory test by common LTC infectious • agents Carrier: individual which is colonized + more • • Understand the necessity of communicating infectious status Subclinical or unapparent infection: presence of upon patient transfer microorganism and interaction between host and microorganism (sub clinical response, immune response). Often the term colonization is applied for relationship host‐ agent in which the immune response is difficult to elicit. • Contamination: Presence of a microorganism on a body surface or an inanimate object. 1 Colonization vs Infection Spectrum: No Exposure ‐ Exposure ‐ Colonization ‐ Carrier Infection ‐ Disease A carrier is an individual that harbors a specific microorganism Host + Infectious agent What is “Exposed” ? in the absence of discernible clinical disease and serve as a No foothold: Exposed Means of transmission: potential source of infection.
    [Show full text]