Medical and Dental Implications of Cerebral Palsy. Part 1 General and Medical Characteristics: a Review

Total Page:16

File Type:pdf, Size:1020Kb

Medical and Dental Implications of Cerebral Palsy. Part 1 General and Medical Characteristics: a Review Central Bringing Excellence in Open Access JSM Dentistry Special Issue on Oral health of children with special health care needs (SHCN) Edited by: Mawlood B. Kowash, BDS, MSc, PhD, FRCDc, FDSRCPS Associate professor in pediatric dentistry, Mohammed Bin Rashid University of Medicine and Health Sciences, United Arab Emirates Review Article *Corresponding author Mawlood B. Kowash, Associate Professor in Paediatric Dentistry, Hamdan Bin Mohammed College of Dental Medical and Dental Implications of Medicine, Mohammed Bin Rashid University of Medical and Health Sciences, Dubai, UAE, Tel: 00971 505939004; Email: [email protected] Cerebral Palsy. Part 1 General and Submitted: 19 April 2017 Accepted: 05 June 2017 Medical Characteristics: A Review Published: 07 June 2017 ISSN: 2333-7133 Haifa Alhashmi, Mawlood Kowash*, and Manal Al Halabi Copyright College of Dental Medicine, Mohammed Bin Rashid University of Medical and Health Sciences, © 2017 Alhashmi et al. UAE OPEN ACCESS Abstract Keywords Cerebral palsy (CP) is a group of neuromuscular disorders that affects the development • Dyskinetic of movement and posture, causing activity limitations. CP is classified into three main groups: • Ataxic spastic, dyskinetic and ataxic. Population-based studies from around the world report estimates • Seizure of CP prevalence ranging from 1.5 to more than 4 per 1,000 live births. The commonest cause • Motor disorders of CP remains unknown in 50% of the cases; prematurity remains the common asterisk factor. • CP CP children suffer from numerous problems and potential disabilities such as mental retardation, epilepsy, feeding difficulties, and ophthalmologic and hearing impairments. This paper reviews and critically discusses the definition, epidemiology, aetiology, classifications, treatments and associated manifestation and complications of CP. INTRODUCTION disturbances of sensation, cognition, communication, perception, and/or by seizure disorders [2]. Cerebral palsy (CP) is a group of neuromuscular disorders, which affect the development of movement and posture, causing [3]. activity limitations. These limitations are attributable to non- CP1)spastic, may be classifiedcharacterized into three by mainincreased groups muscle tone; progressive disturbances, which have occurred in the developing 2) dyskinetic, characterized by hypotonic, slow writhing infant brain. Such disturbances include infection, hypoxia, movements (athetotic), abnormal postural control movements, trauma, and hyper bilirubin anaemia; biochemical and genetic factors may also be involved [1]. ataxic, characterized by involuntary movement, lack of balance The motor disorders of CP are often accompanied by andswallowing depth perception. difficulties, problems of speech and coordination; 3) Cite this article: Alhashmi H, Kowash M, Al Halabi M (2017) Medical and Dental Implications of Cerebral Palsy. Part 1 General and Medical Characteristics: A Review. JSM Dent 5(2): 1088. Alhashmi et al. (2017) Email: [email protected] Central Bringing Excellence in Open Access CP is the most common motor disability which occurs during indicate the prevalence of the CP in the UAE, or Dubai in particular childhood [4]. Population-based studies from around the world report estimates of CP prevalence ranging from 1.5 to more than of Western populations [14]. [5-9]. , experts in the field indicate that it is similar in proportion to that Aetiology In the industrialized nations, the prevalence of cerebral palsy is approximately4 per 1,000 live 2 birthsper 1000 of children live births of a[10] defined. In the age United range States, Congenital aetiology: CP patients have distinctive approximately 10,000 infants and babies are diagnosed with CP deformities which result from defects occurring in normal each year, and a further 1200-1500 are diagnosed at preschool development, and follow outlines based on failures of normal age [10]. formation [15].The earliest acknowledged deformities which lead to survival with motor defects are defects of the neural The prevalence of CP in the developing world is not well tube closure, such as Meningomyelocele (the most common established, but estimated to be 1.5-5.6 cases per 1,000 live neural tube defect occurring in the spine). On the other hand, the births [11]. neural tube defects in the brain known as encephalocele may be LITERATURE REVIEW anterior, with main mid face or nasal defects [16]. Children with Definition including quadriplegic patterns associated with hypotonia, rathersignificant than encephaloceles hypertonia. Segmental have significant defects in motor the brain impairments, known as schizencephaly refer to a cleft in the brain; such schizencephalies famous work of 1862, though he did not apply that term at the vary greatly in their effects, from causing minimal disability time.Sir William John Little Osler, was in histhe book first “Theto define cerebral cerebral palsy palsyof children” in his to causing very severe quadriplegic pattern involvement, and usually present with spasticity and mental retardation [17]. children, grouping them according to their expected aetiology, in(1889), order definedto interpret the clinicalthe physiopathological features, which mechanismspresented 151 of theCP Primary proliferation defects of the brain result in cerebral lesion (damage location). Sigmund Freud, in his “Die infantile Cerebrallahmung” (1897) inspected the causes of these of microencephaly, most of which relate to toxins or infection [15]microencephaly.. Megaloencephaly Regardless, is caused there by arecellular numerous hyper-proliferation, other causes and intrauterine development disorders, in contrast to Little [12]. commonly in syndromes such as sebaceous nerves syndrome, motor disorders, emphasizing the significance of pre-term birth where as macrocephaly is most often due to hydrocephalus. The American Academy for Cerebral Palsy (AACP) was Throughout development neurons transfer toward the founded in 1947 and is considered a multidisciplinary periphery of the brain, a defect in this migration pattern leads professional association, aimed at sponsoring research in the to lissencephaly, which means “smooth brain”; in other words, a child with decreased cerebral gyri. This defect usually leads to severe spastic quadriplegic pattern involvement. There is a field of infant disability. but Mutchoften etchanging, al., (1992) secondary defined CP to as brain “an umbrella lesions termor anomalies covering in a young child may preclude synaptic remodelling through broad-spectrum understanding that significant seizure activity appearinga number ofin syndromesthe early stages with ofmotor brain deficiency, development non-progressive, ”[12]. excitotoxic injury, which may lead to CP. Neonatal aetiology: Neonatal and prenatal causes of CP are et al., (2007) as follows: “Cerebral palsy (CP) describes a group of disordersAnother definitionof the development was then introduced of movement in by and Rosenbaun posture, to numerous patterns [15]. The general trend is that premature chiefly linked to prematurity and birthing problems, which lead causing activity limitation that are attributed to non-progressive infants with more severe bleeds in the brain (in ventricles and the periventricular white matter areas) have a worse prognoses for disturbances that occurred in the developing foetus or infant brain. survival and are at a higher risk of developing CP; nonetheless, The motor disorders of cerebral palsy are often accompanied by there are no restrictions which may accurately predict the risk disturbances of sensation, cognition, communication, perception, of developing CP, much less predict the severity of CP in an and/or behaviour, and/or by a seizure disorder” [12]. individual child. In full-term infants, Hypoxic events occurring Prevalence surrounding delivery usually lead to disability; these events have been called hypoxic-ischemic encephalopathies (HIE) [16]. Cerebral palsy (CP) is the most common motor disability occurring during childhood [4]. Population-based studies from Sub cortical cyst formations develop in severe cases of HIE around the world report prevalence estimates of CP ranging from and are called multi cysticencephalomalacia. When this cystic pattern forms, the prognosis for good function is poor, with most age range [5-9]. In the industrialized world, the prevalence of children developing severe quadriplegic pattern involvement, cerebral1.5 to more palsy than is about4 per 21,000 per 1000live births live births or children [10]. In of the a defined United with severe mental retardation. Further, some of these children States, approximately 10,000 infants and babies are diagnosed develop cysts in the thalamus and basal ganglia, which may lead with CP each year, and a further 1200-1500 are diagnosed at to dystonia [16]. preschool age [10]. The prevalence of CP in the developing world In the preterm or full-term infant, neonatal stroke usually is not well established, however, it is estimated to be 1.5-5.6 affects the middle cerebral artery and presents as a wedge- cases per 1000 live births [11]. In Saudi Arabia, a prevalence shaped defect in one hemisphere. If such awedge-shaped defect is ratio of 5.3 in every 1000 individuals was reported among the Saudi population [13]. While there are no available studies which especially with a cyst, usually presents as hemiplegic pattern small, the child may not be affected; similarly, a significant defect, JSM Dent 5(2): 1088 (2017) 2/6 Alhashmi et al. (2017) Email: [email protected]
Recommended publications
  • Influence of Gestational Age on the Type of Brain Injury and Neuromotor Outcome in High-Risk Neonates
    Eur J Pediatr DOI 10.1007/s00431-007-0629-2 ORIGINAL PAPER Influence of gestational age on the type of brain injury and neuromotor outcome in high-risk neonates Christine Van den Broeck & Eveline Himpens & Piet Vanhaesebrouck & Patrick Calders & Ann Oostra Received: 16 July 2007 /Accepted: 4 October 2007 # Springer-Verlag 2007 Abstract This study was an investigation of a possible periventricular leukomalacia, 24% intraventricular hemor- correlation between either the gestational age (GA) and rhage and 18% persistent flares. There was a significant type of brain injury or between the gestational age and type, correlation between the GA and type of brain injury (P< distribution and severity of cerebral palsy (CP). Four 0.001; Cramer’s V=0.76) and between the GA and type (P= hundred sixty-one children with a birthweight ≥1250 g 0.004; Cramer’s V=0.47) and distribution (P<0.001; and GA ≥30 weeks with a complicated neonatal period and/ Cramer’s V=0.55) of CP. There was no significant correla- or brain injury on serial cerebral ultrasound were selectively tion between the GA and severity of CP. The type of brain followed at the regional Center for Developmental Disor- injury detected by serial ultrasound during the neonatal ders. The children were divided into a preterm and term period, as well as the type and location of CP detected group. There were 40 children with cerebral palsy in the during later childhood, are all GA-dependent in at-risk preterm group and 38 children with cerebral palsy in the newborn infants with a birthweight of ≥1,250 g and term group.
    [Show full text]
  • WCN19 Journal Posters Part 2 Revised V1
    JNS-0000116542; No. of Pages 131 ARTICLE IN PRESS Journal of the Neurological Sciences (2019) xxx–xxx Contents lists available at ScienceDirect Journal of the Neurological Sciences journal homepage: www.elsevier.com/locate/jns WCN19 Journal Posters Part 2 revised_V1 WCN19-2260 WCN19-2269 Poster shift 01 - Channelopathies /neuroethics /neurooncology / Poster shift 01 - Channelopathies /neuroethics /neurooncology / pain - Part I /sleep disorders - Part I /stem cells and gene therapy - pain - Part I /sleep disorders - Part I /stem cells and gene therapy - Part I /stroke /training in neurology - Part I and traumatic brain Part I /stroke /training in neurology - Part I and traumatic brain injury injury Numb chin syndrome- The first finding in metastatic malignancy Results of surgical treatment in patients with moyamoya disease considering CT-perfusion imaging study N. Mustafayev, A. Bayrakoglu, F. Ilgen Uslu, M. Kolukısa Bezmialem University, Neurology, Istanbul, Turkey O. Harmatinaa, V. Morozb, I. Skorokhodab, I. Tyshb, N. Shahinb,R. Hanemb, U. Maliarb a Numb chin syndrome (NCS) is a sensory neuropathy of the SI «Romodanov Institute of Neurosurgery of NAMS of Ukraine», mental nerve, which is accompanied by hypoesthesia and paresthe- Neuroradiology Department, Kyiv, Ukraine b sia of the jaw and lower lip. Although being well known in neurology SI «Romodanov Institute of Neurosurgery of NAMS of Ukraine», practice, most of the physicians who have not experienced this Emergency Department of Vascular Neurosurgery, Kyiv, Ukraine phenomenon are unaware of this phenomenon since it is rare and can be confused with somatic complaints. This case report aims to Aim point out that NCS may be the first sign and symptom of metastatic To improve the results of surgical treatment of patients with cancers in patients who are not diagnosed.
    [Show full text]
  • Hepatic Myelopathy: Case Report And
    LIVER RESEARCH ISSN 2379-4038 http://dx.doi.org/10.17140/LROJ-1-108 Open Journal Case Report Hepatic Myelopathy: Case Report and *Corresponding author Review of the Literature Hua Hong Department of Neurology First Affiliated Hospital Huanquan Liao1#, Zhichao Yan2#, Wei Peng3# and Hua Hong1* Sun Yat-Sen University No. 58 Zhongshan Road 2 #These authors contributed equally. Guangzhou 510080, P.R. China Tel. +008615920500906 1 Fax: +00862087331989 Department of Neurology, The First Affiliated Hospital, Sun Yat-Sen University, Guangzhou E-mail: [email protected] 510080, P.R. China 2State Key Laboratory of Ophthalmology, Zhongshan Ophthalmic Center, Sun Yat-sen Univer- Volume 1 : Issue 2 sity, Guangzhou 510060, Guangdong, P.R. China 3 Article Ref. #: 1000LROJ1108 Department of Stomatology, The First Affiliated Hospital, Sun Yat-Sen University, Guangzhou 510080, P.R. China Article History Received: August 5th, 2015 ABSTRACT Accepted: August 12th, 2015 Published: August 12th, 2015 Background: Hepatic Myelopathy (HM) is a rare complication of chronic liver disease usually associated with extensive portosystemic shunt of blood, which has been created surgically or has occurred spontaneously, causing progressive spastic paraparesis. Some single cases or short Citation clinical reports describing patients suffering from HM have been published worldwide, but are Liao H, Yan Z, Peng W, Hong H. He- patic myelopathy: case report and re- often scattered. view of the literature. Liver Res Open Material and method: One additional case of HM with typical symptoms was presented, and J. 2015; 1(2): 45-55. doi: 10.17140/ a retrospective survey of the literature in a manner of comprehensive review was undertaken.
    [Show full text]
  • A Dictionary of Neurological Signs
    FM.qxd 9/28/05 11:10 PM Page i A DICTIONARY OF NEUROLOGICAL SIGNS SECOND EDITION FM.qxd 9/28/05 11:10 PM Page iii A DICTIONARY OF NEUROLOGICAL SIGNS SECOND EDITION A.J. LARNER MA, MD, MRCP(UK), DHMSA Consultant Neurologist Walton Centre for Neurology and Neurosurgery, Liverpool Honorary Lecturer in Neuroscience, University of Liverpool Society of Apothecaries’ Honorary Lecturer in the History of Medicine, University of Liverpool Liverpool, U.K. FM.qxd 9/28/05 11:10 PM Page iv A.J. Larner, MA, MD, MRCP(UK), DHMSA Walton Centre for Neurology and Neurosurgery Liverpool, UK Library of Congress Control Number: 2005927413 ISBN-10: 0-387-26214-8 ISBN-13: 978-0387-26214-7 Printed on acid-free paper. © 2006, 2001 Springer Science+Business Media, Inc. All rights reserved. This work may not be translated or copied in whole or in part without the written permission of the publisher (Springer Science+Business Media, Inc., 233 Spring Street, New York, NY 10013, USA), except for brief excerpts in connection with reviews or scholarly analysis. Use in connection with any form of information storage and retrieval, electronic adaptation, computer software, or by similar or dis- similar methodology now known or hereafter developed is forbidden. The use in this publication of trade names, trademarks, service marks, and similar terms, even if they are not identified as such, is not to be taken as an expression of opinion as to whether or not they are subject to propri- etary rights. While the advice and information in this book are believed to be true and accurate at the date of going to press, neither the authors nor the editors nor the publisher can accept any legal responsibility for any errors or omis- sions that may be made.
    [Show full text]
  • Spectrum of Nontraumatic Myelopathies in Ethiopian Patients: Hospital-Based Retrospective Study
    Spinal Cord (2016) 54, 604–608 & 2016 International Spinal Cord Society All rights reserved 1362-4393/16 www.nature.com/sc ORIGINAL ARTICLE Spectrum of nontraumatic myelopathies in Ethiopian patients: hospital-based retrospective study NJ Fidèle1 and A Amanuel2 Study design: This is a retrospective hospital-based study. Objectives: The study aimed at a better understanding of the etiology, clinical presentation and treatment outcome of nontraumatic myelopathies in Ethiopian patients. Setting: Etiologies of nontraumatic myelopathies have not been evaluated extensively in most sub-Saharan African countries. The available studies in this region were conducted before the widespread clinical use of modern neuroimaging modalities. This study was conducted in Addis Abba, Ethiopia. Methods: We retrospectively analyzed medical files of patients with a diagnosis of myelopathy (age ⩾ 13 years) admitted or followed up at Tikur Anbesa Hospital between 1 January 2010 and 30 June 2013. Results: Records of 105 patients were analyzed. The male to female ratio was 1.7. The mean age was 38.5 years. Weakness, sensory symptoms (including sensory level), back pain and sphincter dysfunction were the dominant features. Etiologies were dominated by spinal tuberculosis (23.8%) followed by spinal cord neoplastic lesions (primary (10.5%) and secondary neoplasms 8.6%). Other important etiological causes were transverse myelitis (16.2%), degenerative cervical spondylotic myelopathy (15.2%), amyotrophic lateral sclerosis (4.8%) and neuromyelitis optica/multiple sclerosis (3.8%). The mortality rate was 9.5%. Among the patients who died, 40% had chest infection as a complication and 70% presented with complete weakness. Conclusion: Infections remain a major cause of spinal cord disease, and tuberculosis constitutes public health target for reducing the incidence of myelopathies.
    [Show full text]
  • A Dictionary of Neurological Signs.Pdf
    A DICTIONARY OF NEUROLOGICAL SIGNS THIRD EDITION A DICTIONARY OF NEUROLOGICAL SIGNS THIRD EDITION A.J. LARNER MA, MD, MRCP (UK), DHMSA Consultant Neurologist Walton Centre for Neurology and Neurosurgery, Liverpool Honorary Lecturer in Neuroscience, University of Liverpool Society of Apothecaries’ Honorary Lecturer in the History of Medicine, University of Liverpool Liverpool, U.K. 123 Andrew J. Larner MA MD MRCP (UK) DHMSA Walton Centre for Neurology & Neurosurgery Lower Lane L9 7LJ Liverpool, UK ISBN 978-1-4419-7094-7 e-ISBN 978-1-4419-7095-4 DOI 10.1007/978-1-4419-7095-4 Springer New York Dordrecht Heidelberg London Library of Congress Control Number: 2010937226 © Springer Science+Business Media, LLC 2001, 2006, 2011 All rights reserved. This work may not be translated or copied in whole or in part without the written permission of the publisher (Springer Science+Business Media, LLC, 233 Spring Street, New York, NY 10013, USA), except for brief excerpts in connection with reviews or scholarly analysis. Use in connection with any form of information storage and retrieval, electronic adaptation, computer software, or by similar or dissimilar methodology now known or hereafter developed is forbidden. The use in this publication of trade names, trademarks, service marks, and similar terms, even if they are not identified as such, is not to be taken as an expression of opinion as to whether or not they are subject to proprietary rights. While the advice and information in this book are believed to be true and accurate at the date of going to press, neither the authors nor the editors nor the publisher can accept any legal responsibility for any errors or omissions that may be made.
    [Show full text]
  • Cerebral-Palsy-Feeding Executive
    Comparative Effectiveness Review Number 94 Effective Health Care Program Interventions for Feeding and Nutrition in Cerebral Palsy Executive Summary Background Effective Health Care Program Cerebral palsy (CP) is a “group of disorders of the development of The Effective Health Care Program movement and posture, causing was initiated in 2005 to provide activity limitation, that is attributed to valid evidence about the comparative non-progressive disturbances that effectiveness of different medical occurred in the developing fetal or interventions. The object is to help infant brain. The motor disorders of consumers, health care providers, cerebral palsy are often accompanied and others in making informed by disturbances of sensation, cognition, choices among treatment alternatives. communication, perception, and/or Through its Comparative Effectiveness behaviour, and/or by a seizure disorder.”1 Reviews, the program supports This group of syndromes ranges in systematic appraisals of existing severity and is the result of a variety scientific evidence regarding of etiologies occurring in the prenatal, treatments for high-priority health perinatal, or postnatal period. Though conditions. It also promotes and the disorder is nonprogressive, the generates new scientific evidence by clinical manifestations may change over identifying gaps in existing scientific time as the brain develops, with other evidence and supporting new research. neurologic impairments frequently The program puts special emphasis co-occurring.1,2 on translating findings into a variety of useful formats for different More than 100,000 children are estimated stakeholders, including consumers. to be affected with CP in the United States. Due to advances in supportive The full report and this summary are medical care, approximately 90 percent available at www.effectivehealthcare.
    [Show full text]
  • Pediatric Orthopedics in Practice, DOI 10.1007/978-3-662-46810-4, © Springer-Verlag Berlin Heidelberg 2015 880 Backmatter
    879 Backmatter Subject Index – 880 F. Hefti, Pediatric Orthopedics in Practice, DOI 10.1007/978-3-662-46810-4, © Springer-Verlag Berlin Heidelberg 2015 880 Backmatter Subject index Bold letters: Principal article Italics: Illustrations A Acetylsalicylic acid 303, 335 Adolescent scoliosis Amyloidosis 663 Acheiropodia 804 7 Scoliosis Amyoplasia 813–814 Abducent nerve paresis 752, Achievement by proxy 10, 11 AFO 7 Ankle Foot Orthosis Anaerobes 649, 652, 657 816 Achilles tendon Aggrecan 336, 367, 762 ANA 7 antinuclear antibodies Abducted pes planovalgus – lengthening 371, 426, 431, aggressive osteomyelitis Analysis, gait 488, 490–497 433, 434, 436, 439, 443, 464, 7 osteomyelitis, aggressive 7 Gait analysis Abduction contracture 468, 475, 485, 487–490, 493, Agonist 281, 487, 492, 493, Anchor 169, 312, 550, 734 7 contracture 496, 816, 838, 840 495, 498, 664, 832, 835, 840, Andersen classification abduction pants 219–221 – shortening 358, 418, 431, 868 7 classification, Andersen Abduction splint 212, 218–221, 433, 464, 465, 467, 468, 475, Ahn classification 366 Andry, Nicolas 21, 22 248, 850 489, 496, 838 Aitken classification (congenital Anesthesia 26, 38, 135, 154, Abduction Achondrogenesis 750, 751, femoral deficiency ) 7 classi- 162, 174, 221, 243, 247, 248, – hip 195, 198, 199, 212, 213, 756, 758–760, 769 fication, femoral deficiency 255, 281, 303, 385, 386, 400, 214, 218, 219, 220, 221, Achondroplasia 56, 163, 166, Akin osteotomy 477, 479 500, 506, 559, 568, 582–585, 241–245, 247, 248, 251, 255, 242, 270, 271, 353, 409, 628, Albers-Schönberg
    [Show full text]
  • Brainstem and Multiple Cranial Nerve Syndromes
    CHAPTER 21 Brainstem and Multiple Cranial Nerve Syndromes he brainstem is a compact structure, with cra- the lower motor neurons of the CN nuclei. With a nial nerve (CN) nuclei, nerve fascicles, and few exceptions, CNs innervate structures of the head T long ascending and descending tracts all and neck ipsilaterally. A process affecting the brain- closely juxtaposed. Structures and centers in the reticu- stem long tracts on one side causes clinical abnormal- lar formation control many vital functions. Brainstem ities on the opposite side of the body. For this reason, diseases are serious and often life threatening. focal brainstem lesions are characterized by “crossed” Involvement of the intricate network of neural struc- syndromes of ipsilateral CN dysfunction and contra- tures often causes a plethora of clinical findings. lateral long motor or sensory tract dysfunction. For Brainstem syndromes typically involve dysfunction of instance, in the right side of the pons, the nuclei for one or more CNs. Deficits due to dysfunction of indi- CNs VI and VII lie in proximity to the right corti- vidual nerves are covered in the preceding chapters. cospinal tract, which is destined to decussate in the This chapter discusses conditions that cause dysfunc- medulla to innervate the left side of the body. The tion beyond the distribution of a single CN, involving patient with a lesion in the right pons will have CN more than one CN, or conditions that involve brain- findings on the right, such as a sixth or seventh nerve stem structures in addition to the CN nucleus or fasci- palsy, and a hemiparesis on the left.
    [Show full text]
  • Module 2 Neurology and Instrumentation Motor Paralysis The
    Module 2 Neurology and Instrumentation Motor paralysis The term paralysis is derived from the Greek words para, “beside,off, amiss,” and lysis, a “loosening” or “breaking up.” In medicine it has come to refer to an abolition of function, either sensory or motor. When applied to motor function, paralysis means loss of voluntary movement due to interruption of one of the motor pathways at any point from the cerebrum to the muscle fiber. Patterns of paralysis and their diagnosis: The following subdivision, based on the location and distribution of the muscle weakness: 1. Monoplegia : refers to weakness or paralysis of all the muscles of one leg or arm. This term should not be applied to paralysis of isolated muscles or groups of muscles supplied by a single nerve or motor root. 2. Hemiplegia: It is the commonest form of paralysis, involves the arm, the leg, and sometimes the face on one side of the body. With rare exceptions, mentioned further on, hemiplegia is attributable to a lesion of the corticospinal system on the side opposite to the paralysis. 3. Paraplegia: indicates weakness or paralysis of both legs. It is most often the result of diseases of the thoracic spinal cord, caudal equina, or peripheral nerves, and rarely, both medial frontal cortices. 4. Quadriplegia (tetraplegia) :denotes weakness or paralysis of all four extremities. It may result from disease of the peripheral nerves, muscles, or myoneural junctions; gray matter of the spinal cord; or the upper motor neurons bilaterally in the cervical cord, brainstem, or cerebrum. Diplegia is a special form of quadriplegia in which the legs are affected more than the arms.
    [Show full text]
  • Rehabilitation Principles for Motor Dysfunctions According to the Kozyavkin Method
    Kozyavkin V. I. Sak N. N. Kachmar O. O. Babadahly M. O. Rehabilitation principles for motor dysfunctions according to the Kozyavkin Method International Clinic of Rehabilitation www.reha.lviv.ua 2009 1 Universal Decimal classification УДК 616.831 - 009.11 - 053.2 - 085.838 Library and descriptive classifier ББК 57.336.1 Kozyavkin V. I., Sak N. N., Kachmar O. O., Babadagly M. A. Principles of rehabilitation of motor dysfunctions according to Kozyavkin Method. – Lviv: scientific production company ”Ukrayinska tekhnolohiya” (Ukrainian technology), 2007.- 192p. The proposed book is devoted to theoretical principles of motor dysfunction rehabilitation according to Prof. Kozyavkin’s Method and reflects 17 years of experience by the staff at the Institute of Medical Rehabilitation and the International Clinic of Rehabilitation. Readers will be informed about fundamentals related to the organization of human movement systems and rehabilitation principles for disorders of function caused by brain lesions and, in particular, cerebral palsy. They will come to understand how this idea evolved into a fundamentally new tendency in medical treatments and will learn about the effectiveness and application of the given system of rehabilitation. The book will be useful to child neurologists, pediatricians, specialists in medical and physical rehabilitation and students attending related academic institutions. ISBN 978-966-345-118-3 © International Clinic of Rehabilitation 2 Introduction Introduction Motor dysfunctions are one of the main causes of child disabilities and rank the problem of cerebral palsies together with the most important tasks which social pediatrics, child neurology and medical rehabilitation face. For many years, the history of the development of medical treatments for CP was based on attempts to eliminate the most obvious disorders of movement and posture.
    [Show full text]
  • Medical Assistance Eligibility Policy Manual (Archive) - Part 3 of 3
    THIS DOCUMENT IS FOR ARCHIVE PURPOSES ONLY AND MAY NOT REFLECT CURRENT POLICY. Medical Assistance Eligibility Policy Manual (Archive) - Part 3 of 3 Effective until 2021-08-06 THIS DOCUMENT IS FOR ARCHIVE PURPOSES ONLY AND MAY NOT REFLECT CURRENT POLICY. Table of Contents Introduction ................................................................................................................... 11 Getting Started .......................................................................................................... 12 Getting Started ....................................................................................................... 12 Cash Assistance and Nutrition Assistance Policy ...................................................... 18 Example ........................................................................................................................ 19 Examples ................................................................................................................... 19 Introduction ................................................................................................................ 20 E508 Community Spouse Example ........................................................................... 21 E602 Budget Group Examples .................................................................................. 22 E602 Budget Group Examples ............................................................................... 22 A MAGI Budget Group Examples ..........................................................................
    [Show full text]