Cross-Examination of Neuropsychiatric Testimony in Personal Injury Cases
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Diagnoses to Include in the Problem List Whenever Applicable
Diagnoses to include in the problem list whenever applicable Tips: 1. Always say acute or open when applicable 2. Always relate to the original trauma 3. Always include acid-base abnormalities, AKI due to ATN, sodium/osmolality abnormalities 4. Address in the plan of your note 5. Do NOT say possible, potential, likely… Coders can only use a real diagnosis. Make a real diagnosis. Neurological/Psych: Head: 1. Skull fracture of vault – open vs closed 2. Basilar skull fracture 3. Facial fractures 4. Nerve injury____________ 5. LOC – include duration (max duration needed is >24 hrs) and whether they returned to neurological baseline 6. Concussion with or without return to baseline consciousness 7. DAI/severe concussion with or without return to baseline consciousness 8. Type of traumatic brain injury (hemorrhages and contusions) – include size a. Tiny = <0.6 cm b. Small/moderate = 0.6-1 cm c. Large/extensive = >1 cm 9. Cerebral contusion/hemorrhage 10. Cerebral edema 11. Brainstem compression 12. Anoxic brain injury 13. Seizures 14. Brain death Spine: 1. Cervical spine fracture with (complete or incomplete) or without cord injury 2. Thoracic spine fracture with (complete or incomplete) or without cord injury 3. Lumbar spine fracture with (complete or incomplete) or without cord injury 4. Cord syndromes: central, anterior, or Brown-Sequard 5. Paraplegia or quadriplegia (any deficit in the upper extremity is consistent with quadriplegia) Cardiovascular: 1. Acute systolic heart failure 40 2. Acute diastolic heart failure 3. Chronic systolic heart failure 4. Chronic diastolic heart failure 5. Combined heart failure 6. Cardiac injury or vascular injuries 7. -
Two Types of Delayed Post-Traumatic Intracerebral Hematoma
Two Types of Delayed Post-Traumatic Intracerebral Hematoma Takashi TSUBOKAWA Department of Neurological Surgery, Nihon University School of Medicine, Tokyo 173 Summary The findings of repeated CT scans, clinicalcourses and pathologicalstudies in 28 cases of delayed post-traumatic intracerebral hematoma were studied retrospectivelyto elucidate the mechanism of bleeding and to establish adequate treatment. Based on the results obtained, it became clear that there are two types of delayed hematoma. In 10 of the 28 cases, initial CT findings within 6 hours after head injury revealed cerebral contusion or hemorrhagic contusion, and spots of high density scattered in the low density zone gradually became confluent to form an irregularly shaped hematoma according to follow-up CT findings. This was termed "hematoma within a contusional area." In 15 of the 28 cases, initial CT findings within 6 hours after head injury revealed no abnormal density within the brain and the hematoma appeared suddenly 3 6 days after the injury. In eight of the 15 cases, emergency surgery was performed for the removal of epidural or subdural hematoma. This type of hematoma is termed "contusional hem atoma" and constitutes the second group. In three of the 28 cases, both types of hematoma were observed. Based on histological findings for the two types of delayed hematoma. The first group may be induced by an anoxic vasodilation mechanism (Evans et al.9)), while the second group may be derived from a different mechanism related to ishemic changes and the free radical reaction caused by the reflow phenomenon (Tsubokawa et al.14-16)1 It is important to establish correct diagnoses 1for delayed hematomas based on differences between follow-up findings of repeated CT and an initial CT performed within 6 hours after head injury since the operative indications and operative results for the two groups are different as indicated by our 28 cases. -
Management of the Head Injury Patient
Management of the Head Injury Patient William Schecter, MD Epidemilogy • 1.6 million head injury patients in the U.S. annually • 250,000 head injury hospital admissions annually • 60,000 deaths • 70-90,000 permanent disability • Estimated cost: $100 billion per year Causes of Brain Injury • Motor Vehicle Accidents • Falls • Anoxic Encephalopathy • Penetrating Trauma • Air Embolus after blast injury • Ischemia • Intracerebral hemorrhage from Htn/aneurysm • Infection • tumor Brain Injury • Primary Brain Injury • Secondary Brain Injury Primary Brain Injury • Focal Brain Injury – Skull Fracture – Epidural Hematoma – Subdural Hematoma – Subarachnoid Hemorrhage – Intracerebral Hematorma – Cerebral Contusion • Diffuse Axonal Injury Fracture at the Base of the Skull Battle’s Sign • Periorbital Hematoma • Battle’s Sign • CSF Rhinorhea • CSF Otorrhea • Hemotympanum • Possible cranial nerve palsy http://health.allrefer.com/pictures-images/ Fracture of maxillary sinus causing CSF Rhinorrhea battles-sign-behind-the-ear.html Skull Fractures Non-depressed vs Depressed Open vs Closed Linear vs Egg Shell Linear and Depressed Normal Depressed http://www.emedicine.com/med/topic2894.htm Temporal Bone Fracture http://www.vh.org/adult/provider/anatomy/ http://www.bartleby.com/107/illus510.html AnatomicVariants/Cardiovascular/Images0300/0386.html Epidural Hematoma http://www.chestjournal.org/cgi/content/full/122/2/699 http://www.bartleby.com/107/illus769.html Epidural Hematoma • Uncommon (<1% of all head injuries, 10% of post traumatic coma patients) • Located -
Sports-Related Concussions: Diagnosis, Complications, and Current Management Strategies
NEUROSURGICAL FOCUS Neurosurg Focus 40 (4):E5, 2016 Sports-related concussions: diagnosis, complications, and current management strategies Jonathan G. Hobbs, MD,1 Jacob S. Young, BS,1 and Julian E. Bailes, MD2 1Department of Surgery, Section of Neurosurgery, The University of Chicago Pritzker School of Medicine, Chicago; and 2Department of Neurosurgery, NorthShore University HealthSystem, The University of Chicago Pritzker School of Medicine, Evanston, Illinois Sports-related concussions (SRCs) are traumatic events that affect up to 3.8 million athletes per year. The initial diag- nosis and management is often instituted on the field of play by coaches, athletic trainers, and team physicians. SRCs are usually transient episodes of neurological dysfunction following a traumatic impact, with most symptoms resolving in 7–10 days; however, a small percentage of patients will suffer protracted symptoms for years after the event and may develop chronic neurodegenerative disease. Rarely, SRCs are associated with complications, such as skull fractures, epidural or subdural hematomas, and edema requiring neurosurgical evaluation. Current standards of care are based on a paradigm of rest and gradual return to play, with decisions driven by subjective and objective information gleaned from a detailed history and physical examination. Advanced imaging techniques such as functional MRI, and detailed understanding of the complex pathophysiological process underlying SRCs and how they affect the athletes acutely and long-term, may change the way physicians treat athletes who suffer a concussion. It is hoped that these advances will allow a more accurate assessment of when an athlete is truly safe to return to play, decreasing the risk of secondary impact injuries, and provide avenues for therapeutic strategies targeting the complex biochemical cascade that results from a traumatic injury to the brain. -
Feigned Consensus: Usurping the Law in Shaken Baby Syndrome/ Abusive Head Trauma Prosecutions
View metadata, citation and similar papers at core.ac.uk brought to you by CORE provided by University of Michigan School of Law University of Michigan Law School University of Michigan Law School Scholarship Repository Articles Faculty Scholarship 2020 Feigned Consensus: Usurping the Law in Shaken Baby Syndrome/ Abusive Head Trauma Prosecutions Keith A. Findley University of Wisconsin Law School D. Michael Risinger Seton Hall University School of Law Patrick D. Barnes Stanford University Medical Center Julie A. Mack Pennsylvania State University Medical Center David A. Moran University of Michigan Law School, [email protected] See next page for additional authors Available at: https://repository.law.umich.edu/articles/2102 Follow this and additional works at: https://repository.law.umich.edu/articles Part of the Criminal Procedure Commons, Evidence Commons, Juvenile Law Commons, and the Medical Jurisprudence Commons Recommended Citation Findley, Keith A. "Feigned Consensus: Usurping the Law in Shaken Baby Syndrome/Abusive Head Trauma Prosecutions." Michael Risinger, Patrick Barnes, Julie Mack, David A. Moran, Barry Scheck, and Thomas Bohan, co-authors. Wis. L. Rev. 2019, no. 4 (2019): 1211-268. This Article is brought to you for free and open access by the Faculty Scholarship at University of Michigan Law School Scholarship Repository. It has been accepted for inclusion in Articles by an authorized administrator of University of Michigan Law School Scholarship Repository. For more information, please contact [email protected]. Authors Keith A. Findley, D. Michael Risinger, Patrick D. Barnes, Julie A. Mack, David A. Moran, Barry C. Scheck, and Thomas L. Bohan This article is available at University of Michigan Law School Scholarship Repository: https://repository.law.umich.edu/articles/2102 FEIGNED CONSENSUS: USURPING THE LAW IN SHAKEN BABY SYNDROME/ ABUSIVE HEAD TRAUMA PROSECUTIONS KEITH A. -
What to Expect After Having a Subarachnoid Hemorrhage (SAH) Information for Patients and Families Table of Contents
What to expect after having a subarachnoid hemorrhage (SAH) Information for patients and families Table of contents What is a subarachnoid hemorrhage (SAH)? .......................................... 3 What are the signs that I may have had an SAH? .................................. 4 How did I get this aneurysm? ..................................................................... 4 Why do aneurysms need to be treated?.................................................... 4 What is an angiogram? .................................................................................. 5 How are aneurysms repaired? ..................................................................... 6 What are common complications after having an SAH? ..................... 8 What is vasospasm? ...................................................................................... 8 What is hydrocephalus? ............................................................................... 10 What is hyponatremia? ................................................................................ 12 What happens as I begin to get better? .................................................... 13 What can I expect after I leave the hospital? .......................................... 13 How will the SAH change my health? ........................................................ 14 Will the SAH cause any long-term effects? ............................................. 14 How will my emotions be affected? .......................................................... 15 When should -
Overcoming Defense Expert Testimony in Abusive Head Trauma Cases
NATIONAL CENTER FOR PROSECUTION OF CHILD ABUSE Special Topics in Child Abuse Overcoming Defense Expert Testimony in Abusive Head Trauma Cases By Dermot Garrett Edited by Eleanor Odom, Amanda Appelbaum and David Pendle NATIONAL CENTER FOR PROSECUTION OF CHILD ABUSE Scott Burns Director , National District Attorneys Association The National District Attorneys Association is the oldest and largest professional organization representing criminal prosecutors in the world. Its members come from the offices of district attorneys, state’s attorneys, attorneys general, and county and city prosecutors with responsibility for prosecuting criminal violations in every state and territory of the United States. To accomplish this mission, NDAA serves as a nationwide, interdisciplinary resource center for training, research, technical assistance, and publications reflecting the highest standards and cutting-edge practices of the prosecutorial profession. In 1985, the National District Attorneys Association recognized the unique challenges of crimes involving child victims and established the National Center for Prosecution of Child Abuse (NCPCA). NCPCA’s mission is to reduce the number of children victimized and exploited by assisting prosecutors and allied professionals laboring on behalf of victims too small, scared or weak to protect themselves. Suzanna Tiapula Director, National Center for Prosecution of Child Abuse A program of the National District Attorneys Association www.ndaa.org 703.549.9222 This project was supported by Grants #2010-CI-FX-K008 and [new VOCA grant #] awarded by the Office of Juvenile Justice and Delinquency Prevention. The Office of Juvenile Justice and Delinquency Prevention is a component of the Office of Justice Programs. Points of view in this document are those of the author and do not necessarily represent the official position or policies of the U.S. -
What%Is%Epilepsy?%
What%is%Epilepsy?% Epilepsy(is(a(brain(disorder(in(which(a(person(has(repeated(seizures((convulsions)(over(time.(Seizures(are( episodes(of(disturbed(brain(activity(that(cause(changes(in(attention(or(behavior.( Causes( Epilepsy(occurs(when(permanent(changes(in(brain(tissue(cause(the(brain(to(be(too(excitable(or(jumpy.( The(brain(sends(out(abnormal(signals.(This(results(in(repeated,(unpredictable(seizures.((A(single(seizure( that(does(not(happen(again(is(not(epilepsy.)( Epilepsy(may(be(due(to(a(medical(condition(or(injury(that(affects(the(brain,(or(the(cause(may(be( unknown((idiopathic).( Common(causes(of(epilepsy(include:( •Stroke(or(transient(ischemic(attack((TIA)( •Dementia,(such(as(Alzheimer's(disease( •Traumatic(brain(injury( •Infections,(including(brain(abscess,(meningitis,(encephalitis,(and(AIDS( •Brain(problems(that(are(present(at(birth((congenital(brain(defect)( •Brain(injury(that(occurs(during(or(near(birth( •Metabolism(disorders(present(at(birth((such(as(phenylketonuria)( •Brain(tumor( •Abnormal(blood(vessels(in(the(brain( •Other(illness(that(damage(or(destroy(brain(tissue( •Use(of(certain(medications,(including(antidepressants,(tramadol,(cocaine,(and(amphetamines( Epilepsy(seizures(usually(begin(between(ages(5(and(20,(but(they(can(happen(at(any(age.(There(may(be(a( family(history(of(seizures(or(epilepsy.( Symptoms( Symptoms(vary(from(person(to(person.(Some(people(may(have(simple(staring(spells,(while(others(have( violent(shaking(and(loss(of(alertness.(The(type(of(seizure(depends(on(the(part(of(the(brain(affected(and( cause(of(epilepsy.( -
En 13-Correlation Between.P65
Morgado FL et al. CorrelaçãoARTIGO entre ORIGINAL a ECG e •achados ORIGINAL de imagem ARTICLE de TC no TCE Correlação entre a escala de coma de Glasgow e os achados de imagem de tomografia computadorizada em pacientes vítimas de traumatismo cranioencefálico* Correlation between the Glasgow Coma Scale and computed tomography imaging findings in patients with traumatic brain injury Fabiana Lenharo Morgado1, Luiz Antônio Rossi2 Resumo Objetivo: Determinar a correlação da escala de coma de Glasgow, fatores causais e de risco, idade, sexo e intubação orotraqueal com os achados tomográficos em pacientes com traumatismo cranioencefálico. Materiais e Métodos: Foi realizado estudo transversal prospectivo de 102 pacientes, atendidos nas primeiras 12 horas, os quais receberam pontuação segundo a escala de coma de Glasgow e foram submetidos a exame tomográfico. Resultados: A idade média dos pacientes foi de 37,77 ± 18,69 anos, com predomínio do sexo masculino (80,4%). As causas foram: acidente automobilístico (52,9%), queda de outro nível (20,6%), atropelamento (10,8%), queda ao solo ou do mesmo nível (7,8%) e agressão (6,9%). No presente estudo, 82,4% dos pacientes apresentaram traumatismo cranioencefálico de classificação leve, 2,0% moderado e 15,6% grave. Foram observadas alterações tomográficas (hematoma subgaleal, fraturas ósseas da calota craniana, hemorragia subaracnoidea, contusão cerebral, coleção sanguínea extra-axial, edema cerebral difuso) em 79,42% dos pacientes. Os achados tomográficos de trauma craniano grave ocorreram em maior número em pacientes acima de 50 anos (93,7%), e neste grupo todos necessitaram de intubação orotraqueal. Con- clusão: Houve significância estatística entre a escala de coma de Glasgow, idade acima de 50 anos (p < 0,0001), necessidade de intubação orotraqueal (p < 0,0001) e os achados tomográficos. -
Brain Injury and Opioid Overdose
Brain Injury and Opioid Overdose: Acquired Brain Injury is damage to the brain 2.8 million brain injury related occurring after birth and is not related to congenital or degenerative disease. This includes anoxia and hospital stays/deaths in 2013 hypoxia, impairment (lack of oxygen), a condition consistent with drug overdose. 70-80% of hospitalized patients are discharged with an opioid Rx Opioid Use Disorder, as defined in DSM 5, is a problematic pattern of opioid use leading to clinically significant impairment, manifested by meaningful risk 63,000+ drug overdose-related factors occurring within a 12-month period. deaths in 2016 Overdose is injury to the body (poisoning) that happens when a drug is taken in excessive amounts “As the number of drug overdoses continues to rise, and can be fatal. Opioid overdose induces respiratory doctors are struggling to cope with the increasing number depression that can lead to anoxic or hypoxic brain of patients facing irreversible brain damage and other long injury. term health issues.” Substance Use and Misuse is: The frontal lobe is • Often a contributing factor to brain injury. History of highly susceptible abuse/misuse is common among individuals who to brain oxygen have sustained a brain injury. loss, and damage • Likely to increase for individuals who have misused leads to potential substances prior to and post-injury. loss of executive Acute or chronic pain is a common result after brain function. injury due to: • Headaches, back or neck pain and other musculo- Sources: Stojanovic et al 2016; Melton, C. Nov. 15,2017; Devi E. skeletal conditions commonly reported by veterans Nampiaparampil, M.D., 2008; Seal K.H., Bertenthal D., Barnes D.E., et al 2017; with a history of brain injury. -
Traumatic Brain Injury(Tbi)
TRAUMATIC BRAIN INJURY(TBI) B.K NANDA, LECTURER(PHYSIOTHERAPY) S. K. HALDAR, SR. OCCUPATIONAL THERAPIST CUM JR. LECTURER What is Traumatic Brain injury? Traumatic brain injury is defined as damage to the brain resulting from external mechanical force, such as rapid acceleration or deceleration impact, blast waves, or penetration by a projectile, leading to temporary or permanent impairment of brain function. Traumatic brain injury (TBI) has a dramatic impact on the health of the nation: it accounts for 15–20% of deaths in people aged 5–35 yr old, and is responsible for 1% of all adult deaths. TBI is a major cause of death and disability worldwide, especially in children and young adults. Males sustain traumatic brain injuries more frequently than do females. Approximately 1.4 million people in the UK suffer a head injury every year, resulting in nearly 150 000 hospital admissions per year. Of these, approximately 3500 patients require admission to ICU. The overall mortality in severe TBI, defined as a post-resuscitation Glasgow Coma Score (GCS) ≤8, is 23%. In addition to the high mortality, approximately 60% of survivors have significant ongoing deficits including cognitive competency, major activity, and leisure and recreation. This has a severe financial, emotional, and social impact on survivors left with lifelong disability and on their families. It is well established that the major determinant of outcome from TBI is the severity of the primary injury, which is irreversible. However, secondary injury, primarily cerebral ischaemia, occurring in the post-injury phase, may be due to intracranial hypertension, systemic hypotension, hypoxia, hyperpyrexia, hypocapnia and hypoglycaemia, all of which have been shown to independently worsen survival after TBI. -
Traumatic Brain Injury and Domestic Violence
TRAUMATIC BRAIN INJURY AND DOMESTIC VIOLENCE Women who are abused often suffer injury to their head, neck, and face. The high potential for women who are abused to have mild to severe Traumatic Brain Injury (TBI) is a growing concern, since the effects can cause irreversible psychological and physical harm. Women who are abused are more likely to have repeated injuries to the head. As injuries accumulate, likelihood of recovery dramatically decreases. In addition, sustaining another head trauma prior to the complete healing of the initial injury may be fatal. Severe, obvious trauma does not have to occur for brain injury to exist. A woman can sustain a blow to the head without any loss of consciousness or apparent reason to seek medical assistance, yet display symptoms of TBI. (NOTE: While loss of consciousness can be significant in helping to determine the extent of the injury, people with minor TBI often do not lose consciousness, yet still have difficulties as a result of their injury). Many women suffer from a TBI unknowingly and misdiagnosis is common since symptoms may not be immediately apparent and may mirror those of mental health diagnoses. In addition, subtle injuries that are not identifiable through MRIs or CT scans may still lead to cognitive symptoms. What is Traumatic Brain Injury? Traumatic brain injury (TBI) is defined as an injury to the brain that is caused by external physical force and is not present at birth or degenerative. TBI can be caused by: • A blow to the head, o e.g., being hit on the head forcefully with object or fist, having one’s head smashed against object/wall, falling and hitting head, gunshot to head.