Objective Evaluation of Patients with Vestibular Disorders
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Treacher Collins Prize Essay the Significance of Nystagmus
Eye (1989) 3, 816--832 Treacher Collins Prize Essay The Significance of Nystagmus NICHOLAS EVANS Norwich Introduction combined. The range of forms it takes, and Ophthalmology found the term v!to"[<xy!too, the circumstances in which it occurs, must be like many others, in classical Greece, where it compared and contrasted in order to under described the head-nodding of the wined and stand the relationships between nystagmus of somnolent. It first acquired a neuro-ophthal different aetiologies. An approach which is mological sense in 1822, when it was used by synthetic as well as analytic identifies those Goodl to describe 'habitual squinting'. Since features which are common to different types then its meaning has been refined, and much and those that are distinctive, and helps has been learned about the circumstances in describe the relationship between eye move which the eye oscillates, the components of ment and vision in nystagmus. nystagmus, and its neurophysiological, Nystagmus is not properly a disorder of eye neuroanatomic and neuropathological corre movement, but one of steady fixation, in lates. It occurs physiologically and pathologi which the relationship between eye and field cally, alone or in conjunction with visual or is unstable. The essential significance of all central nervous system pathology. It takes a types of nystagmus is the disturbance in this variety of different forms, the eyes moving relationship between the sensory and motor about one or more axis, and may be conjugate ends of the visual-oculomotor axis. Optimal or dysjugate. It can be modified to a variable visual performance requires stability of the degree by external (visual, gravitational and image on the retina, and vision is inevitably rotational) and internal (level of awareness affected by nystagmus. -
Pediatric Ophthalmology/Strabismus 2017-2019
Academy MOC Essentials® Practicing Ophthalmologists Curriculum 2017–2019 Pediatric Ophthalmology/Strabismus *** Pediatric Ophthalmology/Strabismus 2 © AAO 2017-2019 Practicing Ophthalmologists Curriculum Disclaimer and Limitation of Liability As a service to its members and American Board of Ophthalmology (ABO) diplomates, the American Academy of Ophthalmology has developed the Practicing Ophthalmologists Curriculum (POC) as a tool for members to prepare for the Maintenance of Certification (MOC) -related examinations. The Academy provides this material for educational purposes only. The POC should not be deemed inclusive of all proper methods of care or exclusive of other methods of care reasonably directed at obtaining the best results. The physician must make the ultimate judgment about the propriety of the care of a particular patient in light of all the circumstances presented by that patient. The Academy specifically disclaims any and all liability for injury or other damages of any kind, from negligence or otherwise, for any and all claims that may arise out of the use of any information contained herein. References to certain drugs, instruments, and other products in the POC are made for illustrative purposes only and are not intended to constitute an endorsement of such. Such material may include information on applications that are not considered community standard, that reflect indications not included in approved FDA labeling, or that are approved for use only in restricted research settings. The FDA has stated that it is the responsibility of the physician to determine the FDA status of each drug or device he or she wishes to use, and to use them with appropriate patient consent in compliance with applicable law. -
TRAUMATIC BRAIN INJURY Experts Speculate That Perhaps Just As Many – Or More - Hit Their Heads and Never Have It Checked Out
8/1/2019 ‘’POST-CONCUSSION SYNDROME AND VISUAL-VESTIBULAR INTEGRATION DISCLOSURE POLICY DYSFUNCTION” It is the policy of our office, Vincent R. Vicci Jr., O.D., P.A., to state that I have no formal affiliation with other practices, offices, hospitals with the exception of being a staff consultant to the Kessler Institute SEPTEMBER – 2019 for Rehabilitation from which I receive no direct compensation. I have no affiliation with any pharmaceutical company or any company that distributes supplies or instrumentation to doctors and providers of vision care services. I have no copyrighted information, supplies or instrumentation that will be utilized in this presentation. All NEURO-OPTOMETRIC REHABILITATION information supplied during this presentation is based solely upon well established and accepted principles and clinical expertise in the areas LEVEL II EDUCATION discussed. Footnoted materials are presented with all handouts and Power Point presentations. Presentations are prepared and delivered with a high degree of professional conduct to all appropriate VINCENT R. VICCI JR., O.D. participants without discrimination against learners on the basis of gender, age, socioeconomic or ethnic background, sexual orientation WESTFIELD, N.J. or disability. VINCENT R. VICCI JR., O.D., D.P.N.A.P, F.N.O.R.A. • Distinguished Practitioner: National Academies of Practice • Fellow: Neuro-Optometric Rehabilitation Association • Staff consultant - Vision Care Clinics – Kessler Institute for Rehabilitation (West Orange, Saddle Brook, Chester, N.J.) • Consultant: Hartwyck at Oak Tree – J.F.K. Rehabilitation Institute • Seton Hall U.: M.S.O.T. program / yearly guest lecturer • Kean University: O.T. program / yearly guest lecturer • Past President / Co-Founder: Neuro-Optometric Rehabilitation Association (NORA) • Member: O.E.P., N.J.S.O.P, A.O.A. -
Catch One of These Radiology Management Program Webinars
PROVIDERNews Catch One of These Radiology Management Program Webinars Providers who weren’t able to attend the Radiology Management Program overviews held at our recent Mountain State Provider Workshops will have more opportunities to learn this important information. Twelve webinar sessions have been scheduled in November and December to allow providers to hear tips for how to prepare for the program. Representatives from National Imaging Associates, Inc. (NIA) will conduct two 90-minute sessions on each of the following dates: ® Nov. 16, 2010, 8 a.m. and noon ® Nov. 17, 2010, 8 a.m. and noon ® Dec. 7, 2010, 8 a.m. and noon ® Dec. 8, 2010, 8 a.m. and noon ® Dec. 14, 2010, 8 a.m. and noon ® Dec. 15, 2010, 8 a.m. and noon Registration is required. Please click here to access the registration form. (Continued on page 2) Inside This Edition: Radiology Management Program 2 Prescription Drug Benefit Management Moving 10 Paperless EOB and EFT 2 Watch Your Mail for News about FreedomBlueSM NaviNet® Verification Process 3 PPO and BlueRxSM PDP Changes 11 Mountain State Automates Electronic Changes for Service Benefit Plan FEP Members 11 Professional Claim Adjustments Requests 4 National Consumer Cost Tool Initiative 13 Mountain State to Revise COB Process 6 Mountain State to Update Its List of Procedures Coverage for Certain OTC Medications 6 Requiring Authorization 13 October 2010 Medicare Advantage News 7 Contracting/Reimbursement Update 14 Need help getting ready for ICD-10? 8 Please Note Important Holiday Observances 16 HIPAA News 9 Welcome to Our Newest Groups 16 Procedure Codes Relevant to Dental Services 10 Medical Policy Updates 17 PROVIDERNews Catch One of These Radiology Management Program Webinars (Continued from page 1) You will need a computer with Internet access to view the educational materials presented during the webinar. -
Assessment and Management of Infantile Nystagmus Syndrome
perim Ex en l & ta a l ic O p in l h t C h f Journal of Clinical & Experimental a o l m l a o n l r o Atilla, J Clin Exp Ophthalmol 2016, 7:2 g u y o J Ophthalmology 10.4172/2155-9570.1000550 ISSN: 2155-9570 DOI: Review Article Open Access Assessment and Management of Infantile Nystagmus Syndrome Huban Atilla* Department of Ophthalmology, Faculty of Medicine, Ankara University, Turkey *Corresponding author: Huban Atilla, Department of Ophthalmology, Faculty of Medicine, Ankara University, Turkey, Tel: +90 312 4462345; E-mail: [email protected] Received date: March 08, 2016; Accepted date: April 26, 2016; Published date: April 29, 2016 Copyright: © 2016 Atilla H. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Abstract This article is a review of infantile nystagmus syndrome, presenting with an overview of the physiological nystagmus and the etiology, symptoms, clinical evaluation and treatment options. Keywords: Nystagmus syndrome; Physiologic nystagmus phases; active following of the stimulus results in poor correspondence between eye position and stimulus position. At higher velocity targets Introduction (greater than 100 deg/sec) optokinetic nystagmus can no longer be evoked. Unlike simple foveal smooth pursuit, OKN appears to have Nystagmus is a rhythmic, involuntary oscillation of one or both both foveal and peripheral retinal components [3]. Slow phase of the eyes. There are various classifications of nystagmus according to the nystagmus is for following the target and the fast phase is for re- age of onset, etiology, waveform and other characteristics. -
6269 Variation of the Response to the Optokinetic Drum Among Various Strain
[Frontiers in Bioscience 13, 6269-6275, May 1, 2008] Variation of the response to the optokinetic drum among various strains of mice Oliver Puk1, Claudia Dalke1, Martin Hrabé de Angelis2, Jochen Graw1 1 GSF-National Research Center for Environment and Health, Institute of Developmental Genetics, D-85764 Neuherberg, Germany, 2GSF-National Research Center for Environment and Health, Institute of Experimental Genetics, D-85764 Neuherberg, Germany TABLE OF CONTENTS 1. Abstract 2. Introduction 3. Materials and methods 3.1. Animals 3.2. Vision test protocol 3.3. Statistical analysis 3.4. Funduscopy 3.5. Electroretinography 3.6. Histology 4. Results 4.1. Head-tracking behavior 4.2. Electroretinography, funduscopy and histology of DBA/2 and BALB/c mice 4.3. Linkage analysis of BALB/c 5. Discussion 6. Acknowledgement 7. References 1. ABSTRACT 2. INTRODUCTION The mouse is currently an established The optokinetic drum has become an appropriate mammalian model for studying hereditary disorders, which tool to examine visual properties of mice. We performed have an effect on eye structure and function. In order to baseline measurements using mice of the inbred strains select and characterize mouse mutants suffering from C3H, C57BL/6, BALB/c, JF1, 129 and DBA/2 at the age of ocular defects, a variety of test systems are well 8-15 weeks. Each individual C57BL/6, 129 and JF1 mouse established, like slit lamp analysis for detecting lens was reliably identified as non-affected in vision by opacities, iris and corneal abnormalities (1-3), funduscopy determining head-tracking responses. C3H mice were used for abnormalities of the retinal fundus, reflecting retinal as negative control because of their inherited retinal degeneration, vascular problems and optic disc alterations degeneration; as expected, they did not respond to the (4), or electroretinography for functional disorders of the moving stripe pattern. -
Retina Dnb Ophthalmology Question Bank
DNB Ophthalmology Question Bank Retina and Vitreous 1999-2019 Dr. Krati Gupta Dr. Saurabh Deshmukh www.eyelearn.in RETINA, VITREOUS AND CHOROID A. Retinal vascular disorders 1. Anatomy 2. Diabetic retinopathy a) NPDR b) PDR c) Macular edema 3. Retinal vein occlusion a) BRVO b) CRVO 4. Retinal artery occlusion a) CRAO b) OIS 5. Hypertensive retinopathy 6. Pregnancy related hypertensive retinopathy 7. ROP 8. Coats disease 9. Eales disease 10. Radiation retinopathy B. Acquired macular disorders 1. Anatomy 2. Investigations a) Macular function tests b) FFA c) ICG d) OCT e) OCTA 3. Macular degeneration a) ARMD b) CNVM c) IPCV 4. Vitreomacular interface disorders a) ERM b) Macular hole 5. CSR 6. Submacular hemorrhage 7. Macular surgeries 8. Other macular disorders C. Hereditary Fundus Dystrophies 1. Anatomy a) RPE b) Rods and cones 2. Investigations a) VEP b) ERG c) EOG d) Electronystagmometry 3. RP 4. Retinoschisis 5. Bionic eye D. Retinal Detachment 1. Anatomy Dr. Krati Gupta | Dr. Saurabh Deshmukh 2. Investigations a) USG 3. Peripheral retinal degenerations 4. Retinal tears and breaks 5. GRT 6. Retinal Detachment a) RRD b) TRD c) ERD 7. Retinal detachment surgery a) Vitrectomy b) Scleral buckle c) SRF Drainage 8. Vitreous substitutes a) Air b) Gas c) Silicon Oil d) PFCL E. Drugs and LASER 1. Intravitreal drugs a) Steroids b) Anti- VEGF agents 2. Retinal LASERS a) Retinal Micropulsed LASER b) PDT c) TTT F. Vitreous and choroid 1. Vitreous a) Vitreous hemorrhage b) Terson’s syndrome c) PHPV Asteroid hyalosis 2. Choroid a) Choroidal coloboma b) Choroidal effusion G. -
Prolonged Pursuit by Optokinetic Drum Testing in Asymptomatic Female Carriers of Novel FRMD7 Splice Mutation C.1050 ؉5GϾA
OPHTHALMIC MOLECULAR GENETICS SECTION EDITOR: JANEY L. WIGGS, MD, PhD Prolonged Pursuit by Optokinetic Drum Testing in Asymptomatic Female Carriers of Novel FRMD7 Splice Mutation c.1050 ؉5GϾA Arif O. Khan, MD; Jameela Shinwari, MSc; Latifa Al-Sharif, BSc; Dania S. Khalil, BSc; Nada Al Tassan, PhD Objective: To determine the genotype underlying sus- was identified in the 2 affected brothers and in the 3 asymp- pected X-linked infantile nystagmus in a family and to tomatic women only. Allele sharing analysis further con- correlate genotype with clinical examination in poten- firmed that the aunt’s phenotype was not related to the tial female carriers. FRMD7 variant, which was absent in 246 ethnic controls. Her phenotype was also not related to mutation in known Methods: Ophthalmic examination (ophthalmic, or- CFEOM genes (KIF21A, PHOX2A, TUBB3). thoptic, optokinetic [OKN] drum, and electrophysi- ologic when possible) and candidate gene analysis. Conclusions: Prolonged pursuit responses during OKN drum testing in asymptomatic female carriers is consis- Results: Two affected brothers had infantile nystagmus tent with the concept of infantile nystagmus being an ab- with no evidence of associated visual or neurological normally increased pursuit oscillation. Further studies disease. The symptomatic maternal aunt had infantile are required to determine the reproducibility of this po- nystagmus in addition to congenital fibrosis of the ex- tential female carrier sign. Rather than being FRMD7 re- traocular muscles (CFEOM) (bilateral hypotropia, exo- lated, nystagmus in the maternal aunt represented a sec- tropia, ptosis, almost complete ophthalmoplegia, and ond disease in this family, likely related to CFEOM. poorly reactive pupils). A sister, the mother, and the ma- ternal grandmother—all 3 of whom were asymptomatic— Clinical Relevance: Clinicians can use the OKN drum had delayed corrective saccades (prolonged pursuit) dur- to assess obligate female carriers in a family suspected ing OKN drum testing. -
Optical Coherence Tomography in Age-Related Macular Degeneration 1St Edition Principles 1
OPTICAL COHERENCE TOMOGRAPHY IN AGE- RELATED MACULAR DEGENERATION 1ST EDITION DOWNLOAD FREE Gabriel Coscas | 9783662505618 | | | | | Atlas of Retinal OCT: Optical Coherence Tomography Fluoroscopy Dental panoramic radiography X-ray motion analysis. Lay summary — Los Angeles Times September 4, White light is an example of a broadband source with lower power. OCT is based on low-coherence interferometry. October Any light that is outside the short coherence length will not interfere. An imaging approach to temporal OCT was developed by Claude Boccara's team in[24] with an acquisition of the images without beam scanning. Light with broad bandwidths can be generated by using superluminescent diodes or lasers with extremely short pulses femtosecond lasers. Bibcode : OExpr. April Learn how and when to remove this template message. Substances Angiogenesis Inhibitors. Synthetic array heterodyne detection offers another approach to this problem without the need for high dispersion. Free Shipping Free global shipping No minimum order. Therefore, translating one arm of the interferometer has two functions; depth scanning and a Doppler-shifted optical carrier are accomplished by pathlength variation. Optic Neuropathies and Papilledema 6. American Journal of Ophthalmology. We are always looking for ways to improve customer experience on Elsevier. Optical Coherence Tomography in Age-Related Macular Degeneration 1st edition Principles 1. Optical Coherence Tomography in Age-Related Macular Degeneration 1st edition Clinics. A wide range of fundus imaging modalities are now available, and this book explains the respective value of each technique. Intravascular OCT has been investigated for use in neurovascular applications, too, including imaging for guiding endovascular treatment of ischemic stroke and brain aneurysms. -
Glaucoma Symposium
PACIFIC UNIVERSITY COLLEGE OF OPTOMETRY 2015 VICTORIA CONFERENCE July 16 - 19, 2015 Inn at Laurel Point Victoria, B.C. CANADA COPE EVENT #109358 Date Speaker Title COPE Verification Thursday, Dry Eye Etiology and Diagnosis 45687 1 hour July 16, Terry Burris, MD (1 hr) 2015 AS Therapeutic Dry Eye: Current and Future 45701 1 hour Terry Burns, MD Treatment Options (1 hr) AS Therapeutic Danica Marrelli, VEGF Inhibitors in Eye Care 36496 1 hour OD (1 hr) PS Therapeutic Curtis Baxstrom, Prism Applications in Acquired 43108 1 hour OD Brain Injury (1 hr) NO Tad 45456 1 hour Diabetes Potpourri (1 hr) Buckingham, OD SD Therapeutic Glaucoma Case Analysis Friday, Danica Marrelli, Everyday Challenges for the 40184 2 hours July 17 OD Primary Care Optometrist (2 hrs) GL Therapeutic Corneal Degenerations 45688 1 hour Terry Burris, MD (1 hr) AS Therapeutic Tad Pharmaceutical Injections for 45631 1 hour Buckingham, OD Optometrists (1 hr) IS Therapeutic 2015 Update on Corneal 45690 1 hour Terry Burris, MD Procedures Surgery (1 hr) PO Therapeutic Total hours offered: 10 Total hours earned: Name License # Mailing Address ______ Please retain a copy of this stamped form as verification of hours earned. Please be advised that your individual state board makes the final determination of applicable hours. For more information, contact Pacific University College of Optometry, 2043 College Way . Forest Grove, OR 97116 . 503-352-2202 1 of 156 2 of 156 PACIFIC UNIVERSITY COLLEGE OF OPTOMETRY 2015 VICTORIA CONFERENCE July 16 - 19, 2015 Inn at Laurel Point Victoria, -
Nystagmus and Ocular Oscillations in Infancy and Childhood Richard W
9 Nystagmus and Ocular Oscillations in Infancy and Childhood Richard W. Hertle ye care practitioners may be among the first to evaluate Einfants and children with involuntary ocular movements. Pediatric ophthalmologists may, in fact, see more patients with nystagmus than any other specialist because of the frequent association of nystagmus with strabismus.9,16,20,42,54,61,75 Nystag- mus may be covered less frequently in literature and research because there is less we understand or can do about it, compared to strabismus or other childhood eye diseases. HISTORICAL PERSPECTIVE Nystagmus is a rhythmic, involuntary oscillation of one or both eyes. The term comes from the Greek word “nystagmos,” to nod, drowsiness and from “nystazein,” to doze; probably akin to Lithuanian “snusti,” also to doze. Using the information obtained from a complete history, physical examination, and radiographic and oculographic evaluations, more than 40 types of nystagmus can be distinguished (Table 9-1). Some forms of nystagmus are physiological whereas others are pathological. Although the nystagmus is typically described by its more easily observable fast (jerk) phase, the salient clinical and pathological feature is the presence of a slow phase in one or both directions. Clinical descriptions of nystagmus are usually based on the direc- tion of the fast phase and are termed horizontal, vertical, or rotary, or any combination of these (Fig. 9-1). The nystagmus may be conjugate or dysconjugate, indicating whether the eyes move 289 290 handbook of pediatric neuro-ophthalmology TABLE 9-1. Nystagmus Types as Identified by History, Physical Examination, and Ocular Motility Recordings. 1. -
The Influence of Visual Vertigo and Vestibulopathy on Oculomotor
Journal of Vestibular Research 24 (2014) 305–311 305 DOI 10.3233/VES-140519 IOS Press The influence of visual vertigo and vestibulopathy on oculomotor responses Oz Zura,∗, Ruth Dicksteinb, Elizabeth Dannenbaumc,EliCarmelid and Joyce Fungc,e aDepartment of Physical Therapy, Faculty of Social Welfare and Health Sciences, University of Haifa, Haifa, Israel bPhysical Therapy Services, Flieman Geriatric Rehabilitation Hospital, Haifa, Israel cFeil and Oberfeld Research Centre, Jewish Rehabilitation Hospital, Research Site of the Montreal Center for Interdisciplinary Research in Rehabilitation (CRIR), Laval, QC, Canada dPhysical Therapy Department, Haifa University, Social Welfare and Health Sciences, Haifa, Israel eSchool of Physical and Occupational Therapy, McGill University, Montreal, QC, Canada Received 9 May 2013 Accepted 28 March 2014 Abstract. OBJECTIVE: Dynamic visual inputs can cause visual vertigo (VV) in patients with vestibulopathy, leading to dizziness and falls. This study investigated the influence of VV on oculomotor responses. METHODS: In this cross-sectional, single-blind study, with experimental and control groups, 8 individuals with vestibulopathy and VV, 10 with vestibulopathy and no VV, and 10 healthy controls participated. Oculomotor responses were examined with 2- dimensional video-oculography. Participants were exposed to dynamic visual inputs of vertical stripes sweeping across a screen at 20 deg/sec, while seated or in Romberg stance, with and without a fixed target. Responses were quantified by optokinetic nystagmus frequency (OKNf) and gain (OKNg). RESULTS: Seated with no target, VV participants had higher OKNf than controls (37 ± 9vs.24± 9 peaks/sec; P<0.05). In Romberg stance with no target, they had higher OKNf than controls (41 ± 9vs.28± 10 peaks/sec; P<0.05).