Heart and Neck Vessel Assessment
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Internal Jugular Venous Ectasia in an Adult Female Surgery Section
DOI: 10.7860/JCDR/2018/37443.12129 Case Report Internal Jugular Venous Ectasia in an Adult Female Surgery Section BHARATHGURU NEDUMARAN1, ARUNKUMAR KRISHNASAMY2 ABSTRACT Jugular phlebectasia refers to a saccular or fusiform dilatation of the neck veins. Phlebectasia involving the jugular veins is an entity which is diagnosed more commonly in children presenting with neck swellings, after ruling out other common causes. It is also called a venous aneurysm, venous ectasia or essential venous dilatation. This can affect all neck veins-internal jugular, external jugular, anterior jugular and superficial communicating veins in order of decreasing frequency. Although, various theories have been proposed, there is no general consensus among various authors regarding the cause for its occurrence. It is predominantly congenital in origin. The diagnosis is based on clinical presentation aided by non-invasive imaging modalities. The venous ectasia is being increasingly recognised due to advances in radiographic imaging. Management is mainly conservative and surgery is required only when complications arise. Here, authors present a case of venous ectasia in an adult female who presented with complaints of the presence of a vague swelling in her neck. Keywords: Cervical swelling, Internal jugular vein, Phlebectasia, Venous aneurysm CASE REPORT A 39-year-old lady presented to Department of Cardiothoracic Surgery with complaints of pain and swelling over the right side of the neck for the past six months. She attributed the pain to an injury sustained six months back. She gave a history of palpitations and occasional breathing difficulty. She had no difficulty in swallowing and no hoarseness of voice. On general examination, she was healthy with no other significant abnormalities. -
Right Heart Catheter (Internal Jugular Vein Approach) Designation:
11 .au (Affix identification label here) URN: Family name: Right Heart Catheter (Internal Given name(s): Jugular Vein Approach) Address: Date of birth: Sex: M F I Facility: A. Interpreter / cultural needs • Abnormal heart rhythm that continues for a long time. This may need an electric shock to correct. An Interpreter Service is required? Yes No • The carotid artery (in the neck) is accidentally If Yes, is a qualified Interpreter present? Yes No punctured. This may require surgery to repair. © The State of Queensland (Queensland Health), 20 A Cultural Support Person is required? Yes No Rare risks and complications (less than 1%) If Yes, is a Cultural Support Person present? Yes No include: • Infection. This will need antibiotics. B. Condition and treatment • Allergic reaction to the local anaesthetic. This The doctor has explained that you have the following may require medication to treat. Permission to reproduce should be sought from [email protected] condition: (Doctor to document in patient’s own words) • Blood clot in the neck vein. This may need medication to treat. .................................................................................................................................................................... • Embolism. A blood clot may form and break off .................................................................................................................................................................... from the catheter. This is treated with blood This condition requires the following procedure. thinning medication. • (Doctor to document - include site and/or side where Air in the lung cavity. A chest tube may need to relevant to the procedure) be put in to the chest to drain the air. • Damage to the vein in the neck causing bleeding. .................................................................................................................................................................... This may need surgery to repair. The following will be performed: • Air embolism. Oxygen may be given. -
The Carotid Bruit on September 25, 2021 by Guest
AUGUST 2002 221 Pract Neurol: first published as 10.1046/j.1474-7766.2002.00078.x on 1 August 2002. Downloaded from INTRODUCTION When faced with a patient who may have had a NEUROLOGICAL SIGN stroke or transient ischaemic attack (TIA), one needs to ask oneself some simple questions: was the event vascular?; where was the brain lesion, and hence its vascular territory?; what was the cause? A careful history and focused physical examination are essential steps in getting the right answers. Although one can learn a great deal about the state of a patient’s arteries from expensive vascular imaging techniques, this does not make simple auscultation of the neck for carotid bruits redundant. In this brief review, we will therefore defi ne the place of the bruit in the diagnosis and management of patients with suspected TIA or stroke. WHY ARE CAROTID BRUITS IMPORTANT? A bruit over the carotid region is important because it may indicate the presence of athero- sclerotic plaque in the carotid arteries. Throm- boembolism from atherosclerotic plaque at the carotid artery bifurcation is a major cause of TIA and ischaemic stroke. Plaques occur preferentially at the carotid bifurcation, usually fi rst on the posterior wall of the internal carotid artery origin. The growth of these plaques and their subsequent disintegration, surface ulcera- tion, and capacity to throw off emboli into the Figure 1 Where to listen for a brain and eye determines the pattern of subse- bifurcation/internal carotid quent symptoms. The presence of an arterial http://pn.bmj.com/ artery origin bruit – high up bruit arising from stenosis at the origin of the under the angle of the jaw. -
The Hemodynamic Effect of Unilateral Carotid Ligation on the Cerebral Circulation of Man*
THE HEMODYNAMIC EFFECT OF UNILATERAL CAROTID LIGATION ON THE CEREBRAL CIRCULATION OF MAN* HENRY A. SHENKIN, M.D., FERNANDO CABIESES, M.D., GORDON VAN DEN NOORDT, M.D., PETER SAYERS, M.D., AND REUBEN COPPERMAN, M.A. Neurosurgical Service, Graduate Hospital, and Harrison Department of Surgical Re- search, Schools of Medicine, University of Pennsylvania, Philadelphia (Received for publication July ~5, 1950) HE increasing incidence of surgical attack upon vascular anomalies of the brain has renewed interest in the physiological responses of the T cerebral circulation to occlusion of a carotid vessel. This question has been studied in lower animals by Rein, 5 the Schneiders, 6 Bouckaert and Heymans 1 and by the late Cobb Pilcher. 4 However, because of the lack of an adequate technique for measuring the cerebral blood flow and because of the fact that lower animals have a rich intercommunication between the intracerebral and extracerebral circulations, very few data pertinent to human physiology have been accumulated. There are two important points to be established concerning carotid ligation: firstly, its therapeutic effect, that is the degree of fall of pressure in the vessels distal to the ligation, and secondly, its safety, determined by its effect on the cerebral blood flow. Sweet and Bennett, 8 by careful measurement of pressure changes distal to ligation, have provided informa- tion on the former point. It is the latter problem that is the subject of this paper. METHODS The subjects of this study were 4 patients with intracranial arterial aneurysms subjected to unilateral common carotid artery ligation. They ranged in age from 15 to 53 years (Table 1). -
Problems in Family Practice Heart Murmurs in Infants and Children
Problems in Family Practice Heart Murmurs in Infants and Children Thomas A. Riemenschneider, MD Sacramento, California A system is presented for evaluation of heart murmurs in in fants and children. The system places emphasis on identifica tion of functional murmurs, which the physician encounters so frequently in daily practice. A three-part approach is presented which includes: (1) evaluation of cardiovascular status, (2) as sessment of the heart murmur, and (3) decision regarding the need for further evaluation. This approach relieves the physi cian of the necessity to remember the multiple details of the many congenital cardiac lesions, and requires only the knowl edge of a few easily remembered details about functional murmurs. The system enables the physician to confidently distinguish organic and functional murmurs and to decide which children need further evaluation and referral to the pediatric cardiologist. The physician who cares for infants, children, with “normal” murmurs for reassurance to the and adolescents will frequently encounter heart parents.2 Using his/her knowledge of the myriad murmurs during the course of a careful physical details of the many congenital cardiac malforma examination. It has been estimated that a heart tions, the pediatric cardiologist seeks evidence murmur may be heard at some time in almost that the murmur is due to an organic lesion. The every child.1 Murmurs may be classified as “func family physician cannot expect to retain all of tional” (physiologic, normal, benign, or innocent), these details, and therefore often feels in or “organic” (associated with an anatomic car adequately prepared to assess the child with a diovascular abnormality). -
Outpatient Approach to Palpitations RANDELL K
Outpatient Approach to Palpitations RANDELL K. WEXLER, MD, MPH; ADAM PLEISTER, MD; and SUBHA RAMAN, MD, MSEE The Ohio State University, Columbus, Ohio Palpitations are a common problem seen in family medicine; most are of cardiac origin, although an underlying psychiatric disorder, such as anxiety, is also common. Even if a psychiatric comorbidity does exist, it should not be assumed that palpitations are of a noncardiac etiology. Discerning cardiac from noncardiac causes is important given the potential risk of sudden death in those with an underlying cardiac etiology. History and physical examination followed by targeted diagnostic testing are necessary to distinguish a cardiac cause from other causes of palpitations. Standard 12-lead electrocardiography is an essential initial diagnostic test. Cardiac imaging is recommended if his- tory, physical examination, or electrocardiography suggests structural heart disease. An intermittent event (loop) monitor is preferred for documenting cardiac arrhythmias, particularly when they occur infrequently. Ventricular and atrial premature contractions are common cardiac causes of palpitations; prognostic significance is dictated by the extent of underlying structural heart disease. Atrial fibrillation is the most common arrhythmia resulting in hos- pitalization; such patients are at increased risk of stroke. Patients with supraventricular tachycardia, long QT syn- drome, ventricular tachycardia, or palpitations associated with syncope should be referred to a cardiologist. (Am Fam Physician. 2011;84(1):63-69. Copyright © 2011 American Academy of Family Physicians.) atients often present to family phy- CARDIAC STRUCTURAL CAUSES sicians with palpitations. However, Mitral valve prolapse is the most common this may mean different things structural heart disease leading to palpita- to different people. -
Dysrhythmias
CARDIOVASCULAR DISORDERS DYSRHYTHMIAS I. BASIC PRINCIPLES OF CARDIAC CONDUCTION DISTURBANCES A. Standard ECG and rhythm strips 1. Recordings are obtained at a paper speed of 25 mm/sec. 2. The vertical axis measures distance; the smallest divisions are 1 mm ×1 mm. 3. The horizontal axis measures time; each small division is 0.04 sec/mm. B. Normal morphology Courtesy of Dr. Michael McCrea 1. P wave = atrial depolarization a. Upright in leads I, II, III, aVL, and aVF; inverted in lead aVR b. Measures <0.10 seconds wide and <3 mm high c. Normal PR interval is 0.12–0.20 seconds. 2. QRS complex = ventricular depolarization a. Measures 0.06-0.10 seconds wide b. Q wave (1) <0.04 seconds wide and <3 mm deep (2) Abnormal if it is >3 mm deep or >1/3 of the QRS complex. c. R wave ≤7.5 mm high 3. QT interval varies with rate and sex but is usually 0.33–0.42 seconds; at normal heart rates, it is normally <1/2 the preceding RR interval. 4. T wave = ventricular repolarization a. Upright in leads I, II, V3–V6; inverted in aVR b. Slightly rounded and asymmetric in configuration c. Measures ≤5 mm high in limb leads and ≤10 mm high in the chest leads 5. U wave = a ventricular afterpotential a. Any deflection after the T wave (usually low voltage) b. Same polarity as the T wave c. Most easily detected in lead V3 d. Can be a normal component of the ECG e. Prominent U waves may indicate one of the following: (1) Hypokalemia (<3 mEq/L) (2) Hypercalcemia (3) Therapy with digitalis, phenothiazines, quinidine, epinephrine, inotropic agents, or amiodarone (4) Thyrotoxicosis f. -
Novel Balloon-And-Aspiration Method for Cerebral Venous Sinus Thrombosis: Dental-Floss Technique
NEUROSURGICAL FOCUS Neurosurg Focus 42 (4):E19, 2017 Novel balloon-and-aspiration method for cerebral venous sinus thrombosis: dental-floss technique Yoshikazu Matsuda, MD,1,2 Hideo Okada, MD,1,3 Joonho Chung, MD, PhD,1,4 R. Webster Crowley, MD,1 and Demetrius K. Lopes, MD1 1Department of Neurological Surgery, Rush University Medical Center, Chicago, Illinois; 2Department of Neurosurgery, Wakayama Medical University; 3Department of Neurosurgery, Wakayama Rosai Hospital, Wakayama City, Japan; and 4Department of Neurosurgery, Gangnam Severance Hospital, Yonsei University College of Medicine, Seoul, Republic of Korea Cerebral venous sinus thrombosis is sometimes fatal. The standard treatment for sinus thrombosis is anticoagulation, but endovascular intervention must be considered when medical treatment fails. Mechanical thrombectomy is usually required when a large clot burden exits. Unfortunately, in sinus thrombosis attributable to a clot burden larger than that in an intracranial artery, the conventional technique used for intraarterial acute stroke intervention with a stent retriever and/ or aspiration is not very effective. The authors describe here their endovascular approach to mechanical thrombectomy for sinus thrombosis using aspiration combined with angioplasty balloon support. https://thejns.org/doi/abs/10.3171/2017.1.FOCUS16519 KEY WORDS cerebral venous sinus thrombosis; balloon catheter; aspiration catheter; novel technique ESPITE our improved understanding and diagnosis Operative Technique of sinus thrombosis, its resulting morbidity and mortality rates are still high.6 Standard medical The venous sinus floss technique requires arterial (5- Dtreatment of sinus thrombosis typically consists of heparin Fr catheter) and venous (8-Fr guide catheter or 6-Fr guide in the acute phase and eventual conversion to low-molec- sheath) vascular access. -
Near-Syncope After Exercise
GRAND ROUNDS CLINICIAN’S CORNER AT THE JOHNS HOPKINS BAYVIEW MEDICAL CENTER Near-Syncope After Exercise Roy C. Ziegelstein, MD Syncope and near-syncope are great diagnostic challenges in medicine. On CASE PRESENTATION the one hand, the symptom may result from a benign condition and pose A 72-year-old man complained of near- little or no threat to health other than that related to falling. On the other syncope after exercise that had been oc- hand, syncope or near-syncope can be the manifestation of a serious un- curring for several months. Every morn- derlying condition that poses an imminent threat to life. Patients with a car- ing, he walked at 3.5 mph for about half diac cause of syncope are at far greater risk of dying in the first year after an an hour on his treadmill in the well- ventilated basement of his home. After episode of syncope or near-syncope than individuals with a noncardiac cause. exercising, he would step off the tread- A cardiac cause of syncope should be considered in every patient with syn- mill, check his pulse, and become light- cope or near-syncope, but it is particularly common in older patients or in headed immediately afterward. This feel- patients with known structural heart disease, arrhythmia, or certain electro- ing lasted a few minutes and he would cardiographic abnormalities. Although many diagnostic tests may be help- then go upstairs to eat breakfast and take ful in the evaluation of syncope and near-syncope, the history, physical ex- his medications. amination, and electrocardiogram pinpoint the cause in many circumstances. -
List of Sounds in SAM II
List of Sounds in SAM II Heart and Breath Sound Combined Heart Sounds (cont.) Normal heart and breath sounds Third Heart Sound Mitral valve prolapse with normal breath Fourth Heart Sound sounds Physiologic splitting of second sound Aortic stenosis with crackles with lung sounds Aortic stenosis, mild with normal breath Paradoxical splitting of second sound Sounds with lung sounds Mitral regurgitation with crackles Mitral regurgitation with normal breath Mitral Sounds Sounds Mitral Valve Prolapse Congestive heart failure Mitral Stenosis Atrial fibrillation with normal breath Mitral Stenosis and Regurgitation sounds Mitral Regurgitation, mild Atrial Flutter with low heart rate and Mitral Regurgitation, marked PVCs. Fourth heart sound with respiratory Aortic Sounds variation Aortic Stenosis, mild Aortic Stenosis, severe with aortic Pulmonary related sounds in the neck Wheeze with normal heart sounds Aortic Stenosis and Regurgitation Coarse crackles with normal heart Aortic Stenosis with cooing murmur Sounds Aortic Regurgitation Rhonchus with normal heart sounds Aortic Regurgitation, Acute Emphysema with normal heart sounds Congenital Aortic Stenosis at an accelerated rate Aortic Stenosis with crackles Lobar Pneumonia with normal heart Aortic Stenosis with normal breath Sounds Chronic bronchitis with normal heart Pediatric Sounds Sounds Atrial Septal Defect with lung sounds COPD with diminished normal heart Ventricular Septal Defect with thrill sounds Patent Ductus Arteriosus Tactile Fremitus (palpation on posterior Pulmonary Hypertension -
Right Site, Wrong Route ― Cannulating the Left Internal Jugular Vein
Open Access Case Report DOI: 10.7759/cureus.2044 Right Site, Wrong Route ― Cannulating the Left Internal Jugular Vein Peter Paik 1 , Sanjay K. Arukala 1 , Anupam A. Sule 1 1. Internal Medicine, St. Joseph Mercy Oakland Hospital Corresponding author: Anupam A. Sule, [email protected] Abstract Central venous catheters are placed in approximately five million patients annually in the US. The preferred site of insertion is one with fewer risks and easier access. Although the right internal jugular vein is preferred, on occasion, the left internal jugular may have to be accessed. A patient was admitted for septic shock, cerebrovascular accident, and non-ST-segment elevation myocardial infarction. A central venous line was needed for antibiotic and vasopressor administration. Due to trauma from a fall to the right side and previously failed catheterization attempts at the left subclavian and femoral veins, the left internal jugular vein was accessed. On chest radiography for confirmation, the left internal jugular central venous catheter was seen projecting down the left paraspinal region. It did not take the expected course across the midline toward the right and into the superior vena cava (SVC). A review of a computed tomography (CT) scan of the chest with contrast done on a prior admission revealed a duplicated SVC on the left side that had not been reported in the original CT scan interpretation. A left-sided SVC is present in approximately 0.3% to 0.5% of the population, with 90% of these draining into the coronary sinus. During placements of central venous lines and pacemakers, irritation of the coronary sinus may result in hypotension, arrhythmia, myocardial ischemia, or cardiac arrest. -
The Kinked Innominate Vein
Br Heart J: first published as 10.1136/hrt.22.1.110 on 1 January 1960. Downloaded from THE KINKED INNOMINATE VEIN BY K. SHIRLEY SMITH From the Cardiac Department, Charing Cross Hospital Received July 1, 1959 The examination of the neck is an essential part of every investigation of the heart. Over-filling ofcervical veins had long been known as a sign ofheart failure when Lewis (1930) laid down standard methods for this test, which needs attention to detail and close scrutiny; even so, the appraisal is sometimes difficult. The meaning of fixed bilateral filling as compared with ebb and flow is self- evident, but unilateral venous congestion, when there is no clear causal obstruction is not so easy to understand. In this paper, attention is drawn to the frequent occurrence of unilateral filling of the left external jugular vein and reasons are given for regarding it as a sign of a high and probably rigid aortic arch. Since this sign is caused by the aorta compressing the left innominate vein from below, the sign is named the kinked innominate vein. A high aortic arch frequently causes tortuosity of the right common carotid artery which winds below the clavicular head of the sternomastoid muscle. This physical sign was first fully described by Parkinson et al. (1939) who studied it in 47 patients and named it the "kinked carotid". They found that it was generally associated with hypertension and arteriosclerosis, but in one-quarter of the cases with arteriosclerosis alone. It occurred in women but hardly ever in men. The new sign that is now defined is in a sense a natural counterpart of the arterial sign, both being due to the same cause, namely, a high and probably rigid aortic arch.