DOCSLIB.ORG

Hematomas in Children L

Total Page：16

File Type：pdf, Size：1020Kb

Download full-text PDF Read full-text
Hematomas in Children L Hematomas in children L. van Duurling, forensic specialist KNMG / manager FPKM Subcutaneous bruising (hematoma) A so-called hematoma (‘black and blue spot’) is caused by a haemorrhage in the skin after collision and/or compressive external force with or by a blunt object. Hematomas are the result of the presence of blood in the space between the cells of the skin (interstitial space), often as a result of the tearing (off) of blood vessels (then there is a hematoma, whereby the discoloration of the skin sharp is clearly demarcated), or as a consequence of the leakage of blood from blood vessels (then there is a bruise whereby the discoloration of the skin is more diffuse). A local inflammatory response and the dilation of capillaries (small blood vessels) may contribute to the redness of a recent hematoma.1 Hematomas usually disappear completely within about 2 weeks.2 Distinction between accidental and non-accidental hematomas Hematomas are extremely rare in children younger than 6 months. The stage of development plays an important role: children who are not mobile rarely have hematomas. The frequency of the occurrence of hematoma increases as children become older (and therefore more mobile) and enterprising.3 Accidental hematomas in children who can walk are generally small and are located at locations on the skin where bone protrudes slightly and/or is relatively close to the skin surface (such as the tibia). This usually involves locations at the front of the body, because the child is moving forward. Accidental injuries occur during sports and play or from (traffic) accidents. The explanation given for these injuries is usually adequate. Non-accidental hematomas are often located at locations where no bone is just under the skin, such as the neck, face (cheeks), the buttocks, torso (abdomen) and the (upper) arms. Non-accidental hematomas are often larger than accidental ones, and are more often clustered, sometimes showing an impression of an object (for example fingers, ‘fingerprint bruising’).4 5 Hematomas in the anogenital region (at the penis and the vulva), on the inner side of the upper and lower arms and the thighs (for example in the form of 'fingerprint bruising’), in the armpits and at the point of the shoulder blade may be indicative of sexual and/or physical abuse. Encountering multiple hematomas close to each other (clustered) in the form of ‘fingerprints' on the chest and/or (simultaneously) on the back may indicate a mechanism 1 Kollias N. The physical basis of skin color and its evaluation. Clin Dermatol 1995;13:361-7. 2 Taitz LS. Child abuse: some myths and shibboleths. Hospital Update 1991 May:400-4. 3 Labbe J, Caouette G. Recent skin injuries in normal children. Pediatrics 2001;108:271-6. 4 Maguire S, Mann MK, Sibert J, Kemp A. Are there patterns of bruising in childhood which are diagnostic or suggestive of abuse? Arch Dis Child 2005; 90:182-6. 5 Naidoo NS. A profile of the orofacial injuries in child physical abuse at a children's hospital. Child Abuse Neglect. 2000;24:521-34. www.thenextpage.nl whereby the chest cage is firmly enclosed, such as in roughly executed care procedures or after a violent shaking incident.6 6 Hobbs CJ, Osman J. Genital injuries in boys and abuse. Arch Dis Child 2007;92:328-31. www.thenextpage.nl Compression of the trunk and/or neck, such as in an attempt at strangling, leads to a pressure increase in the blood flow toward the heart (increased venous pressure), which can result in punctiform haemorrhages (petechiae), specifically on the face, neck and/or in the conjunctiva of the eyes.7 A blow to the side of the face can cause punctiform haemorrhages (petechiae) or small skin discolorations (purpura) on, in or behind the ear.8. In the figure below it is shown at which locations hematomas are suspicious for child abuse. Minimally Very suspicious suspicious 7 Reece RM, Christian CW. Child abuse, medical diagnosis and management. AAP, third edition, 2009:19-51 8 Feldman KW. Patterned abusive bruises of the buttocks and pinnae. Pediatrics 1992;90:633-6. www.thenextpage.nl Tramline bruising If an object is used for inflicting injury, the object can cause injury with visible edges. From hitting with the flat of the hand it is possible that capillaries in the skin between the fingers tear, because the blood is pushed away under the fingers. Solid objects that encounter the skin with force can result, by the stretching of the skin at the location of the edges from these objects, in stripe-like skin discolorations at the site of the edges of the object, leaving behind a pattern of tracks (‘tramline bruising), see figure 2).9 Fig. 2: Tramline bruising 9 Stark MM, Payne-James J, Crane J, Hincliffe JA. Clinical Forensic Medicine. Humana Press, Second edition, 2005. www.thenextpage.nl .
Load more

Recommended publications
  • Bruise, Contusion & Ecchymosis Conventions
    Bruise, Contusion and Ecchymosis MedDRA Proactivity Proposal Implementation MedDRA Version 16.0 I. MSSO Recognized Definitions of Concepts and Terms The MSSO has designated Dorland’s Illustrated Medical Dictionary as the standard reference for medical definitions. The following definitions are cited from Dorland’s 27th edition: Bruise – A superficial injury produced by impact without laceration; a contusion Contusion – A bruise; an injury of a part without a break in the skin Ecchymosis – A small hemorrhagic spot, larger than a petechia, in the skin or mucous membrane forming a nonelevated, rounded or irregular, blue or purplish patch. Hematoma – A localized collection of blood, usually clotted, in an organ, space, or tissue, due to a break in the wall of a blood vessel. Hemorrhage – The escape of blood from the vessels; bleeding. Petechia – A pinpoint, non-raised, perfectly round, purplish red spot caused by intradermal or submucous hemorrhage. Additional comments regarding the definitions: Bruise and contusion are synonymous, and are often used in a colloquial context. Bruise and contusion are each considered a result of injury. Bruise and contusion have been used to describe minor hemorrhage within tissue, where traumatized blood vessels leak blood into the interstitial space. Commonly, capillaries and sometimes venules are injured within skin, subcutaneous tissue, muscle, or bone. In addition to trauma, the terms bruise, ecchymosis, and to a lesser extent, contusion, have also been used as clinical signs of disorders of platelet function, coagulopathies, venous congestion, allergic reactions, etc. Hemorrhage may be used to describe blood escaping from vessels and retained in the interstitial space, and perhaps more commonly, to describe the escape of blood from vessels, and flowing freely external to the tissues.
    [Show full text]
  • Immune Thrombocytopenic Purpura in a Twin Girl Revealed by a Traumatic Injury in Parakou (North Benin)
    Immune thrombocytopenic purpura in a twin girl revealed by a traumatic injury in parakou (North Benin) Adedemy JD 1*, Noudamadjo A 1, Kpanidja G 1, Agossou J 1, Agbeille Mohamed F 1, Dovonou CA 2 1 Mother and Child Department, Parakou Teaching Hospital, Republic of Benin, and Faculty of Medicine, University of Parakou West Africa 2 Department of Medicine, Parakou Teaching Hospital, and Faculty of Medicine, University of Parakou, West Africa Abstract Background: ITP seems to be rare but in tropical settings thrombocytopenia is often encountered among children. Objective: Authors through this case report are putting emphasy on the diagnosis and management of ITP in a 4 year old twin girl admitted in the pediatric emergency ward for hematuria and bleeding from various origins seen in the context of a domestic trauma. Results: The various clinical signs have been analyzed to confirm ITP through exclusion of other possible health conditions. The management of ITP depend on the severity of clinical signs and in some cases the situations can be life threatening. In this case report, Blood transfusion and corticosteroids were the main treatment tools. The hospital stay was about 47 days and an ambulatory follow up was conducted for almost 6 months. Conclusion: In the context of various bleeding disorders, hematuria and thrombocytopenia, autoimmune thrombocytopenia in a twin girl was revealed by a domestic trauma. Citation: Adedemy JD, Noudamadjo A, Kpanidja G, Agossou J, Agbeille MF, Dovonou CA (2019) Immune Thrombocytopenic Purpura in a twin girl revealed by a traumatic injury in Parakou (North Benin). Adv Pediatr Res 6:27.
    [Show full text]
  • How Significant Is Bleeding in Antiphospholipid Antibody
    Treatment of Anti-Phospholipid Syndrome and Prothrombin Deficiency with Plasma Exchange Lowell Tilzer KU Medical Center Department of Pathology & Lab Medicine Case 72 YEARS OLD FEMALE PRESENTED TO KUMC WITH BLEEDING FOLLOWING ROUTINE HEMORRHOIDECTOMY SURGERY Past Medical History • Surgeries: Bilateral tubal ligation, appendectomy, partial hysterectomy, bilateral bunion surgery – No bleeding complications • 2008: Presented to ER with chest pain – Incidentally found prolonged PT and PTT + Lupus anticoagulant and anticardiolipin antibodies • 2009: Melanotic stool with severe anemia (Hb 4) – Blood transfusion (>10 units) – Attributed to long-term use of Aspirin Brief course Initial Work-up Reported Normal Test Value Range PT/INR 2.2 (HIGH) 0.8-1.2 PTT 87.4 (HIGH) 24.0-40.0 PT mixing study @ 60 mins 1.5 (HIGH) PTT mixing study @ 60 mins 82.8 (HIGH) Factor 2 assay 10% (LOW) 50-150% Factor 5 assay 78% 50-150% Factor 7 assay 153% (HIGH) 50-150% Factor 8 assay 235% (HIGH) 50-150% Factor 10 assay 68% 50-150% 300 Linear dilutions 250 200 150 100 % Normal control 50 0 Factor 2 Factor 5 Factor 7 Factor 8 Factor 10 Additional coagulation study Test Result Interpretation dRVVT Prolonged Lupus anticoagulant Abs Hexagonal Lupus Positive Lupus anticoagulant Abs anticoagulant Anti-b2 GPI IgG Positive Anti-b2 GPI IgM Positive Support APS diagnosis Anti-cardiolipin IgG Positive Anti-cardiolipin IgM Positive There is NO Factor 2 inhibitor, therefore previous result of low Factor 2 inhibitor <0.4 BU Factor 2 level was due to lupus (activity-based) anticoagulant Abs against phospholipids in the assay. Algorithm for Screening tests (PT/INR, aPTT) coagulopathies Mixing study Heparin, DTI PL dependent assays (aPTT or dRVVT + Hexa) PTT PT PT PTT LA+ Factor LA- Intrinsic Common Extrinsic inhibitors pathway pathway pathway Revised Criteria for Antiphospholipid Syndrome (APS) (Sydney Criteria) APS: ≥ 1 Laboratory Criteria AND ≥ 1 Clinical Criteria • Laboratory Criteria: “≥ 2 occasions 3 months apart” 1.
    [Show full text]
  • Intracranial Haemorrhage in a 26 Year-Old Woman with Idiopathic Thrombocytopenic Purpura
    Postgraduate Medical Journal (1987) 63, 781-783 Postgrad Med J: first published as 10.1136/pgmj.63.743.781 on 1 September 1987. Downloaded from Intracranial haemorrhage in a 26 year-old woman with idiopathic thrombocytopenic purpura Gavin Awerbuch and Reuven Sandyk Department ofNeurology, University ofArizona Health Sciences Center, Tucson, AZ. 85724, USA. Summary: Intracranial haemorrhage (ICH), a rare complication of idiopathic thrombocytopenic purpura (ITP), described only once previously in an adult, is usually fatal. We report a previously healthy 26 year old woman with chronic ITP in whom spontaneous ICH developed. The eventual favourable outcome in this case despite severe initial neurological deficit makes this case unusual. The importance of aggressive management in an ITP associated ICH is stressed and a plan for management is suggested. Introduction Although relatively rare, intracranial haemorrhage of 5.7 g/dl, white blood count of 15.5 x I09/l with a (ICH) is the most serious complication of idiopathic normal differential and a platelet count of 6.0 x 109/l, thrombocytopenic purpura (ITP) and is the leading reticulocyte count was 3.2%. The prothrombin time, reported cause ofdeath.'`3 In children, nearly 20 cases partial thromboplastin time, thrombin time, fibrin- Protected by copyright. of acute ITP complicated by ICH have been reported. ogen, fibrin monomers, and fibrin split products were Previous reports have alluded to ICH associated with all normal. An electroencephalogram (EEG), gallium ITP in adults,4`7 but ICH has been documented only scan of the abdomen, chest X-ray, and computerized once in adults.8 We report the occurrence of an axial tomography (CT scan) of the head were normal.
    [Show full text]
  • Cerebral Venous Thrombosis After Intravenous Immunoglobulin Therapy in Immune Thrombocytopenic Purpura
    Case Report Cerebral Venous Thrombosis after Intravenous Immunoglobulin Therapy in Immune Thrombocytopenic Purpura Joe James, P. V. Shiji, Chandni Radhakrishnan Department of Internal Medicine, Government Medical College, Kozhikode, Kerala, India Abstract A common misconception is that immune thrombocytopenic purpura (ITP) causes only bleeding diathesis. From this case vignette of a young male with ITP who had cerebral venous thrombosis, we highlight the importance of considering venous thrombosis in such patients when they present with focal cerebral signs. Keywords: Immune thrombocytopenic purpura, intravenous immunoglobulin, venous thrombosis 3 INTRODUCTION blood cell count of 21,800/mm with 84% neutrophils, 12% lymphocytes, and 4% mixed cells; hemoglobin of 11.2 g/dl; and Immune thrombocytopenic purpura (ITP) is a disorder platelet count of 65,000/mm3. Serum sodium was 136 mEq/L, characterized by autoimmune destruction of platelets, potassium 3.9 mEq/L, calcium 9.1 mg/dL, and plasma glucose which commonly presents as bleeding diathesis. However, 105 mg/dL. Serum creatinine was 1.1 mg/dL, total bilirubin thromboembolic complications can also occur in ITP. Here, was 1.5 mg/dL, and alanine aminotransferase was 30 IU. we describe a case of ITP who presented with cerebral venous International normalized ratio was 1.03 and activated partial thrombosis (CVT) after treatment with high‑dose intravenous thromboplastin time was 31.3 (reference <28). Anti‑nucleosome immunoglobulin (IVIg). antibody, anti ds‑DNA, C3 and C4 levels were normal. HIV, HBsAg, and HCV serology were negative. Computed CASE REPORT tomography (CT) of the head showed a small intraparenchymal A 26‑year‑old‑male with a history of ITP, presented with hematoma in the right frontal lobe [Figure 1a].
    [Show full text]
  • Protein C 결핍에서 발생한 소모성 응고질환으로 인한 뇌내출혈로 야기된 뇌성마비 환자에 대한 증례기록 연세대학교 의과대학 세브란스병원 재활의학과1, 소아청소년과2 조유나1・이영 목 2・박 은 숙 1・최 자 영 1・박 천 웅 1・나 동 욱 1
    J Korean Child Neurol Soc 2017;25(1):44-47 Case report pISSN 1226-6884•eISSN 2383-8973 Protein C 결핍에서 발생한 소모성 응고질환으로 인한 뇌내출혈로 야기된 뇌성마비 환자에 대한 증례기록 연세대학교 의과대학 세브란스병원 재활의학과1, 소아청소년과2 조유나1・이영 목 2・박 은 숙 1・최 자 영 1・박 천 웅 1・나 동 욱 1 Cerebral Palsy due to Intracranial Hemorrhage Yoona Cho, MD1, Young-Mock Lee, MD2, Eun Sook Park, MD1, Ja Young Choi, MD1, Chunung Caused by Consumptive Coagulopathy in Protein C Park, MD1, Dong-wook Rha, MD, PhD1 Deficiency: A Case Report 1Department and Research Institute of Rehabi- Protein C (PROC) is a potent anticoagulant inactivating coagulation factors Va and litation Medicine, 2Department of Pediatrics, VIIIa. PROC deficiency is very rare condition inherited as an autosomal dominant Gangnam Severance Hospital, Yonsei University or recessive trait, and associated with various thromboembolic and ischemic College of Medicine, Seoul, Korea conditions. Moreover, severe form of PROC deficiency can cause fatal hemorrhagic complications due to consumptive coagulopathy. We reported two children with Submitted: 24 August, 2016 Revised: 13 September, 2016 hemorrhagic stroke who were diagnosed as severe PROC deficiency caused by two Accepted: 13 September, 2016 different types of compound heterozygous PROC gene mutations. We described results of laboratory tests, genetic analysis, brain magnetic resonance images, and Correspondence to Dong-wook Rha, MD, PhD functional outcomes. Both children received prophylactic anticoagulation therapy Department and Research Institute of Rehabilitation, and presented with purple-colored skin lesions during rehabilitation. Purpura Medicine Yonsei University College of Medicine, Severance Rehabilitation Hospital, BioMechanics and fulminans caused by insufficient anticoagulation should be differentiated from Robotic Rehabilitation Laboratory, 50-1 Yonsei-ro hematoma caused by excessive anticoagulation therapy in these children.
    [Show full text]
  • Thrombocytopenia and Intracranial Venous Sinus Thrombosis After “COVID-19 Vaccine Astrazeneca” Exposure
    Journal of Clinical Medicine Article Thrombocytopenia and Intracranial Venous Sinus Thrombosis after “COVID-19 Vaccine AstraZeneca” Exposure Marc E. Wolf 1,2 , Beate Luz 3, Ludwig Niehaus 4 , Pervinder Bhogal 5, Hansjörg Bäzner 1,2 and Hans Henkes 6,7,* 1 Neurologische Klinik, Klinikum Stuttgart, D-70174 Stuttgart, Germany; [email protected] (M.E.W.); [email protected] (H.B.) 2 Department of Neurology, Universitätsmedizin Mannheim, University of Heidelberg, D-68167 Mannheim, Germany 3 Zentralinstitut für Transfusionsmedizin und Blutspendedienst, Klinikum Stuttgart, D-70174 Stuttgart, Germany; [email protected] 4 Neurologie, Rems-Murr-Klinikum Winnenden, D-71364 Winnenden, Germany; [email protected] 5 Department of Interventional Neuroradiology, The Royal London Hospital, Barts NHS Trust, London E1 1FR, UK; [email protected] 6 Neuroradiologische Klinik, Klinikum Stuttgart, D-70174 Stuttgart, Germany 7 Medical Faculty, University Duisburg-Essen, D-47057 Duisburg, Germany * Correspondence: [email protected]; Fax: +49-711-278-345-09 Abstract: Background: As of 8 April 2021, a total of 2.9 million people have died with or from the coronavirus infection causing COVID-19 (Corona Virus Disease 2019). On 29 January 2021, the European Medicines Agency (EMA) approved a COVID-19 vaccine developed by Oxford University Citation: Wolf, M.E.; Luz, B.; and AstraZeneca (AZD1222, ChAdOx1 nCoV-19, COVID-19 vaccine AstraZeneca, Vaxzevria, Cov- Niehaus, L.; Bhogal, P.; Bäzner, H.; ishield). While the vaccine prevents severe course of and death from COVID-19, the observation of Henkes, H. Thrombocytopenia and pulmonary, abdominal, and intracranial venous thromboembolic events has raised concerns. Objec- Intracranial Venous Sinus Thrombosis after “COVID-19 Vaccine tive: To describe the clinical manifestations and the concerning management of patients with cranial AstraZeneca” Exposure.
    [Show full text]
  • Modern Management of Traumatic Hemothorax
    rauma & f T T o re l a t a m n r e u n o t J Mahoozi, et al., J Trauma Treat 2016, 5:3 Journal of Trauma & Treatment DOI: 10.4172/2167-1222.1000326 ISSN: 2167-1222 Review Article Open Access Modern Management of Traumatic Hemothorax Hamid Reza Mahoozi, Jan Volmerig and Erich Hecker* Thoraxzentrum Ruhrgebiet, Department of Thoracic Surgery, Evangelisches Krankenhaus, Herne, Germany *Corresponding author: Erich Hecker, Thoraxzentrum Ruhrgebiet, Department of Thoracic Surgery, Evangelisches Krankenhaus, Herne, Germany, Tel: 0232349892212; Fax: 0232349892229; E-mail: [email protected] Rec date: Jun 28, 2016; Acc date: Aug 17, 2016; Pub date: Aug 19, 2016 Copyright: © 2016 Mahoozi HR. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Abstract Hemothorax is defined as a bleeding into pleural cavity. Hemothorax is a frequent manifestation of blunt chest trauma. Some authors suggested a hematocrit value more than 50% for differentiation of a hemothorax from a sanguineous pleural effusion. Hemothorax is also often associated with penetrating chest injury or chest wall blunt chest wall trauma with skeletal injury. Much less common, it may be related to pleural diseases, induced iatrogenic or develop spontaneously. In the vast majority of blunt and penetrating trauma cases, hemothoraces can be managed by relatively simple means in the course of care. Keywords: Traumatic hemothorax; Internal chest wall; Cardiac Hemodynamic response injury; Clinical manifestation; Blunt chest-wall injuries; Blunt As above mentioned the hemodynamic response is a multifactorial intrathoracic injuries; Penetrating thoracic trauma response and depends on severity of hemothorax according to its classification.
    [Show full text]
  • ACR Appropriateness Criteria: Suspected Retroperitoneal Bleed
    New 2021 American College of Radiology ACR Appropriateness Criteria® Suspected Retroperitoneal Bleed Variant 1: Clinically suspected retroperitoneal bleed. Initial imaging. Procedure Appropriateness Category Relative Radiation Level CT abdomen and pelvis with IV contrast Usually Appropriate ☢☢☢ CT abdomen and pelvis without and with IV Usually Appropriate contrast ☢☢☢☢ CTA abdomen and pelvis with IV contrast Usually Appropriate ☢☢☢☢☢ Aortography abdomen and pelvis May Be Appropriate (Disagreement) ☢☢☢☢ CT abdomen and pelvis without IV contrast May Be Appropriate ☢☢☢ US abdomen and pelvis Usually Not Appropriate O Radiography abdomen and pelvis Usually Not Appropriate ☢☢☢ MRA abdomen and pelvis with IV contrast Usually Not Appropriate O MRA abdomen and pelvis without and with Usually Not Appropriate IV contrast O MRA abdomen and pelvis without IV contrast Usually Not Appropriate O MRI abdomen and pelvis without and with IV Usually Not Appropriate contrast O MRI abdomen and pelvis without IV contrast Usually Not Appropriate O RBC scan abdomen and pelvis Usually Not Appropriate ☢☢☢ ACR Appropriateness Criteria® 1 Suspected Retroperitoneal Bleed SUSPECTED RETROPERITONEAL BLEED Expert Panel on Vascular Imaging: Nupur Verma, MDa; Michael L. Steigner, MDb; Ayaz Aghayev, MDc; Ezana M. Azene, MD, PhDd; Suzanne T. Chong, MD, MSe; Benoit Desjardins, MD, PhDf; Riham H. El Khouli, MD, PhDg; Nicholas E. Harrison, MDh; Sandeep S. Hedgire, MDi; Sanjeeva P. Kalva, MDj; Yoo Jin Lee, MDk; David M. Mauro, MDl; Hiren J. Mehta, MDm; Mark Meissner, MDn; Anil K. Pillai, MDo; Nimarta Singh, MD, MPHp; Pal S. Suranyi, MD, PhDq; Eric E. Williamson, MDr; Karin E. Dill, MD.s Summary of Literature Review Introduction/Background Retroperitoneal bleeding is a hemorrhage into the retroperitoneal space, the space located posterior to the parietal peritoneum and anterior to the transversalis fascia.
    [Show full text]
  • Delayed Traumatic Intracranial Bleeding and Herniation After Initial Negative Computed Tomography Scan
    ACS Case Reviews in Surgery Vol. 2, No. 3 Delayed Traumatic Intracranial Bleeding and Herniation after Initial Negative Computed Tomography Scan AUTHORS: CORRESPONDENCE AUTHOR: AUTHOR AFFILIATIONS: Philip L. Rosen, MDa; Daniel J. Gross, MDa; Fulfilled Dr. Philip L. Rosen a. SUNY Downstate Medical Center Ighalob; Vladimir Rubinshteyn, MD, FACSb; Akella SUNY Downstate Medical Center Department of Surgery Chendrasekhar, MD, FACSb Department of Surgery, c/o Dr. Philip Rosen 450 Clarkson Ave, Box 40 450 Clarkson Ave, Box 40 Brooklyn, NY 11203 Brooklyn, NY 11203 b. Richmond University Medical Center Phone: (718) 270-3302 Department of Surgery Email: [email protected] 355 Bard Ave Staten Island, NY 10310 Background Delayed acute subdural hematoma (SDH) is defined as an acute SDH that is not apparent on the initial computed tomography (CT) scan but appears on a repeat CT scan. Delayed acute SDH occurs is uncommon and mainly occurs in middle-aged and elderly persons who are either on anticoagulation or antiplatelet therapy. Summary We describe the unusual case of a healthy adult male, not on anticoagulants or antiplatelets, who was a pedestrian struck by a moving vehicle, with an initial head CT negative for intracranial bleeding, who developed a delayed acute SDH 36 hours later with devastating consequences. This is notable because in our protocols at our Level I trauma center, we do not routinely reimage head trauma patients after a CT that is negative for intracranial bleeding if they are not on anticoagulants or antiplatelets. Conclusion A CT scan that is negative for an initial intracranial bleed, does not rule out a subsequent severe intracranial bleed, even in a healthy patient who is not on anticoagulation or antiplatelets.
    [Show full text]
  • Extrapleural Hematoma Cases Treated with Video-Assisted Thoracoscopic Surgery
    CASE REPORT Extrapleural hematoma cases treated with video-assisted thoracoscopic surgery Mehmet Gökhan Pirzirenli, M.D., Burçin Çelik, M.D., Selçuk Gürz, M.D., Zeynep Pelin Sürücü, M.D. Department of Thoracic Surgery, Ondokuz Mayis University Faculty of Medicine, Samsun ABSTRACT Extrapleural hematoma is a rare clinical condition whose most common causes are blunt chest trauma, central venous catheterization and ruptured thoracic aortic aneurysm. This study aimed to present three cases that underwent video-assisted thoracoscopic surgery due to extrapleural hematoma. Etiologic factors in these cases were falls from height in two cases and a dialysis catheter placed into the right internal jugular vein in one case. Keywords: Extrapleural; hematoma; treatment; video-assisted thoracoscopic surgery. INTRODUCTION Anesthesia Management Electrocardiogram, heart rate and blood pressure were Extrapleural hematoma (EPH) is a rare clinical condition monitored in all patients. Venous cannula was inserted into since the expansion of extrapleural space and accumulation a peripheral vein and a radial arterial cannula was inserted of blood in this region is quite difficult. EPH is classified into under local anesthesia. Additional monitoring included SpO , three groups including spontaneous, iatrogenic and traumat- 2 urine output and peak airway pressure. After inhaling 100% ic.[1] Blunt chest trauma, central venous catheterization and oxygen for 5 minutes, anesthesia was induced with 0.2 µg/kg/ ruptured thoracic aortic aneurysm are the most frequent eti-
    [Show full text]
  • Concomitant Central Venous Sinus Thrombosis and Subdural Hematoma in Acute Promyelocytic Leukemia: Middle Meningeal Artery Embolization Enables Safe Anticoagulation
    J Neurosurg Case Lessons 1(23):CASE2080, 2021 DOI: 10.3171/CASE2080 Concomitant central venous sinus thrombosis and subdural hematoma in acute promyelocytic leukemia: middle meningeal artery embolization enables safe anticoagulation. Illustrative case Kushagra Maini, MD,1 Feroze Afzal, MD,1 Dan-Victor Giurgiutiu, MD,1 Scott Y. Rahimi, MD,2 Manan Shah, MD,1 Jeffrey A. Switzer, DO,1 Fernando L. Vale, MD,2 and Klepper Alfredo Garcia, MD1 Departments of 1Neurology and 2Neurosurgery, Augusta University, Augusta, Georgia BACKGROUND Acute promyelocytic leukemia (APL) has long been associated with coagulation disorders. The proposed mechanism is a combination of fibrinolysis, proteolysis, platelet dysfunction, thrombocytopenia, and possibly disseminated intravascular coagulation. Hemorrhagic complications are prominent. OBSERVATIONS In this case, a 25-year-old female with newly diagnosed APL developed extensive cerebral venous thrombosis (CVT) and was initiated on a protocol with idarubicin and all-trans retinoic acid. The general recommendation for treating CVT is anticoagulation to stabilize the existing thrombus and prevent propagation. The patient was initiated on a heparin drip, but her clinical course was complicated by subdural hemorrhage (SDH) and epidural hemorrhage in the setting of thrombocytopenia. Anticoagulation was held, and her CVT propagated on follow-up imaging. To restart anticoagulation for CVT with a limited risk of SDH, the authors pursued middle meningeal artery (MMA) embolization. The patient was transitioned to apixaban and discharged to home. LESSONS MMA embolization enables safe anticoagulation in patients with concomitant CVT and SDH. The authors report the complex clinical course and effective management of this rare clinical scenario. https://thejns.org/doi/abs/10.3171/CASE2080 KEYWORDS leukemia; brain bleed; thrombosis; embolization; surgical technique; oncology Cerebral venous sinus thrombosis is a rare complication of differential regulation on acute myeloid leukemia (AML) adhesion acute promyelocytic leukemia (APL).
    [Show full text]