932 Chest Imaging CPT, HCPCS and Diagnoses Codes
Total Page:16
File Type:pdf, Size:1020Kb

Load more
Recommended publications
-
Hypersensitivity Pneumonitis: Challenges in Diagnosis and Management, Avoiding Surgical Lung Biopsy
395 Hypersensitivity Pneumonitis: Challenges in Diagnosis and Management, Avoiding Surgical Lung Biopsy Ferran Morell, MD1,2 Ana Villar, MD2,3 Iñigo Ojanguren, MD2,3 Xavier Muñoz, MD2,3 María-Jesús Cruz, PhD2,3 1 Vall d’Hebron Institut de Recerca (VHIR), Barcelona, Catalonia, Spain Address for correspondence Ferran Morell, MD, Vall d’Hebron Institut 2 Ciber de Enfermedades Respiratorias (CIBERES), Barcelona, Spain de Recerca (VHIR), PasseigValld’Hebron, 119-129, 08035 Barcelona, 3 Servei de Pneumologia, Hospital Universitari Vall d’Hebron, Catalonia, Spain (e-mail: [email protected]). Barcelona, Spain Semin Respir Crit Care Med 2016;37:395–405. Abstract This review presents an update of the currently available information related to Keywords hypersensitivity pneumonitis, with a particular focus on the contribution of several ► hypersensitivity techniques in the diagnosis of this condition. The methods discussed include proper pneumonitis elaboration of a complete medical history, targeted auscultation, detection of specific ► bronchoalveolar immunoglobulin G antibodies against the most common antigens causing this disease, lavage skin tests, antigen-specific lymphocyte activation assays, bronchoalveolar lavage, and ► fi speci c inhalation cryobiopsy. Special emphasis is placed on the relevant contribution of specificinhalation challenge challenge (bronchial challenge test). Surgical lung biopsy is presented as the ultimate ► bronchial challenge recourse, to be used when the diagnosis cannot be reached through the other methods test covered. -
Workers' Compensation for Occupational Respiratory Diseases
ORIGINAL ARTICLE http://dx.doi.org/10.3346/jkms.2014.29.S.S47 • J Korean Med Sci 2014; 29: S47-51 Workers’ Compensation for Occupational Respiratory Diseases So-young Park,1 Hyoung-Ryoul Kim,2 The respiratory system is one of the most important body systems particularly from the and Jaechul Song3 viewpoint of occupational medicine because it is the major route of occupational exposure. In 2013, there were significant changes in the specific criteria for the recognition of 1Occupational Lung Diseases Institute, Korea Workers’ Compensation & Welfare Service, Ansan; occupational diseases, which were established by the Enforcement Decree of the Industrial 2Department of Occupational and Environmental Accident Compensation Insurance Act (IACIA). In this article, the authors deal with the Medicine, College of Medicine, the Catholic former criteria, implications of the revision, and changes in the specific criteria in Korea by University of Korea, Seoul; 3Department of focusing on the 2013 amendment to the IACIA. Before the 2013 amendment to the IACIA, Occupational and Environmental Medicine, Hanyang University College of Medicine, Seoul, occupational respiratory disease was not a category because the previous criteria were Korea based on specific hazardous agents and their health effects. Workers as well as clinicians were not familiar with the agent-based criteria. To improve these criteria, a system-based Received: 20 December 2013 structure was added. Through these changes, in the current criteria, 33 types of agents Accepted: 6 April 2014 and 11 types of respiratory diseases are listed under diseases of the respiratory system. In Address for Correspondence: the current criteria, there are no concrete guidelines for evaluating work-relatedness, such Hyoung-Ryoul Kim, MD as estimating the exposure level, latent period, and detailed examination methods. -
Silicosis: Pathogenesis and Changklan Muang Chiang Mai 50100 Thailand; Tel: 66 53 276364; Fax: 66 53 273590; E-Mail
Central Annals of Clinical Pathology Bringing Excellence in Open Access Review Article *Corresponding author Attapon Cheepsattayakorn, 10th Zonal Tuberculosis and Chest Disease Center, 143 Sridornchai Road Silicosis: Pathogenesis and Changklan Muang Chiang Mai 50100 Thailand; Tel: 66 53 276364; Fax: 66 53 273590; E-mail: Biomarkers Submitted: 04 October 2018 Accepted: 31 October 2018 1,2 3 Attapon Cheepsattayakorn * and Ruangrong Cheepsattayakorn Published: 31 October 2018 1 10 Zonal Tuberculosis and Chest Disease Center, Chiang Mai, Thailand ISSN: 2373-9282 2 Department of Disease Control, Ministry of Public Health, Thailand Copyright 3 Department of Pathology, Faculty of Medicine, Chiang Mai University, Chiang Mai, Thailand © 2018 Cheepsattayakorn et al. OPEN ACCESS Abstract Ramazzini first described this disease, namely “Pneumonoultramicroscopicsilicovolcanokoniosis” Keywords and then was changed according to the types of exposed dust. No reliable figures on the silica- • Silicosis inhalation exposed individuals are officially documented. How silica particles stimulate pulmonary • Biomarkers response and the exact path physiology of silicosis are still not known and urgently require further • Pathogenesis research. Nevertheless, many researchers hypothesized that pulmonary alveolar macrophages play a major role by secreting fibroblast-stimulating factor and re-ingesting these ingested silica particles by the pulmonary alveolar macrophage with progressive magnification. Finally, ending up of the death of the pulmonary alveolar macrophages and the development of pulmonary fibrosis appear. Various mediators, such as CTGF, FBRS, FGF2/bFGF, and TNFα play a major role in the development of silica-induced pulmonary fibrosis. A hypothesis of silicosis-associated abnormal immunoglobulins has been postulated. In conclusion, novel studies on pathogenesis and biomarkers of silicosis are urgently needed for precise prevention and control of this silently threaten disease of the world. -
Are Serum Aluminum Levels a Risk Factor in the Appearance of Spontaneous Pneumothorax?
Turk J Med Sci 2010; 40 (3): 459-463 Original Article © TÜBİTAK E-mail: [email protected] doi:10.3906/sag-0901-11 Are serum aluminum levels a risk factor in the appearance of spontaneous pneumothorax? Serdar HAN1, Rasih YAZKAN2, Bülent KOÇER3,*, Gültekin GÜLBAHAR3, Serdal Kenan KÖSE4, Koray DURAL3, Ünal SAKINCI3 Aim: To investigate the relationship between aluminum and spontaneous pneumothorax (SP) development. Materials and methods: A patient group and a control group were formed with 100 individuals in each. The serum aluminum levels of the groups were determined and statistically compared. Results: The mean serum aluminum levels were 5.6 ± 2.4 μg/L (1.6-11.9) and 23.2 ± 15.4 μg/L (2-81) in the control and SP groups, respectively (P < 0.001). The specificity and sensitivity of the measurement of aluminum level were 74.4% and 86.4% in the SP group. The risk of SP development was found to be 18 times higher in individuals with high serum levels of aluminum compared to that in individuals with low serum levels of aluminum. Conclusion: A high level of aluminum is a risk factor for the development of SP. Key words: Aluminum, pneumothorax, public health Spontan pnömotoraks gelişiminde serum aluminyum seviyeleri bir risk faktörü müdür? Amaç: Bu çalışmada spontan pnömotoraks (SP) gelişimi ile serum aluminyum düzeyleri arasındaki ilişkinin analiz edilmesi amaçlandı. Yöntem ve gereç: Yüz SP hastasından oluşan hasta grubunun yanı sıra 100 kişiden oluşan sağlıklı kontrol grubu çalışmaya dahil edildi. Gruplar serum aluminyum düzeyleri açısından istatistiksel olarak kıyaslandı. Bulgular: Ortalama serum aluminyum düzeyleri SP ve kontrol grupları için sırasıyla 5,6 ± 2,4 μg/L (1,6-11,9) ve 23,2 ± 15,4 μg/L (2-81) bulundu (P < 0,001). -
Local Coverage Determination for Respiratory Therapy
Local Coverage Determination (LCD): Respiratory Therapy (Respiratory Care) (L34430) Links in PDF documents are not guaranteed to work. To follow a web link, please use the MCD Website. Contractor Information Contractor Name Contract Type Contract Number Jurisdiction State(s) Palmetto GBA A and B MAC 10111 - MAC A J - J Alabama Palmetto GBA A and B MAC 10211 - MAC A J - J Georgia Palmetto GBA A and B MAC 10311 - MAC A J - J Tennessee Palmetto GBA A and B and HHH MAC 11201 - MAC A J - M South Carolina Palmetto GBA A and B and HHH MAC 11301 - MAC A J - M Virginia Palmetto GBA A and B and HHH MAC 11401 - MAC A J - M West Virginia Palmetto GBA A and B and HHH MAC 11501 - MAC A J - M North Carolina Back to Top LCD Information Document Information LCD ID Original Effective Date L34430 For services performed on or after 10/01/2015 Original ICD-9 LCD ID Revision Effective Date L31593 For services performed on or after 01/29/2018 Revision Ending Date LCD Title N/A Respiratory Therapy (Respiratory Care) Retirement Date Proposed LCD in Comment Period N/A N/A Notice Period Start Date Source Proposed LCD 08/18/2016 N/A Notice Period End Date AMA CPT / ADA CDT / AHA NUBC Copyright Statement 10/02/2016 CPT only copyright 2002-2018 American Medical Association. All Rights Reserved. CPT is a registered trademark of the American Medical Association. Applicable FARS/DFARS Apply to Government Use. Fee schedules, relative value units, conversion factors and/or related components are not assigned by the AMA, are not part of CPT, and the AMA is not recommending their use. -
Etiology and Aggravation in Thoracic Medicine
DePaul Law Review Volume 21 Issue 1 Fall 1971: Medico-Legal Symposium II Article 6 Etiology and Aggravation in Thoracic Medicine W. B. Buckingham Follow this and additional works at: https://via.library.depaul.edu/law-review Recommended Citation W. B. Buckingham, Etiology and Aggravation in Thoracic Medicine, 21 DePaul L. Rev. 103 (1971) Available at: https://via.library.depaul.edu/law-review/vol21/iss1/6 This Article is brought to you for free and open access by the College of Law at Via Sapientiae. It has been accepted for inclusion in DePaul Law Review by an authorized editor of Via Sapientiae. For more information, please contact [email protected]. ETIOLOGY AND AGGRAVATION IN THORACIC MEDICINE W. B. BUCKINGHAM* INTRODUCTION N THE practice of thoracic medicine, misconceptions about the etiology and/or the aggravation of thoracic diseases are common- place. These errors are frequently perpetuated by patients and occasionally by well-meaning lawyers and well-trained physicians. Superficial analysis indicates that most of these misconceptions arise from the concept of post hoc ergo propter hoc. In diseases affecting the thorax, multiple etiological factors commonly operate over pro- longed periods of time. Under such circumstances it is extremely difficult to sort out cause and effect. Thus, in light of the imperfec- tions in diagnosis, misconceptions about etiology and aggravation of disease can be appreciated. There are substantial limitations and uncertainties to any medical diagnosis. The semantic derivation of the word "diagnosis" comes through the Greek words whose meaning is "to know through" or "to understand by means of the manifestations of." A complete diagnosis in the modem sense of the term is a disease entity in which the causative mechanisms are clearly understood, and the man- ifestations readily appreciated both in clinical signs and symptoms and in laboratory findings. -
Nebulizers: Diagnosis Codes – Medicare Advantage Policy Appendix
UnitedHealthcare® Medicare Advantage Policy Appendix: Applicable Code List Nebulizers: Diagnosis Codes This list of codes applies to the Medicare Advantage Policy Guideline titled Approval Date: September 8, 2021 Nebulizers. Applicable Codes The following list(s) of procedure and/or diagnosis codes is provided for reference purposes only and may not be all inclusive. The listing of a code does not imply that the service described by the code is a covered or non-covered health service. Benefit coverage for health services is determined by the member specific benefit plan document and applicable laws that may require coverage for a specific service. The inclusion of a code does not imply any right to reimbursement or guarantee claim payment. Other Policies and Guidelines may apply. Diagnosis Code Description For HCPCS Codes A7003, A7004, and E0570 A15.0 Tuberculosis of lung A22.1 Pulmonary anthrax A37.01 Whooping cough due to Bordetella pertussis with pneumonia A37.11 Whooping cough due to Bordetella parapertussis with pneumonia A37.81 Whooping cough due to other Bordetella species with pneumonia A37.91 Whooping cough, unspecified species with pneumonia A48.1 Legionnaires' disease B20 Human immunodeficiency virus [HIV] disease B25.0 Cytomegaloviral pneumonitis B44.0 Invasive pulmonary aspergillosis B59 Pneumocystosis B77.81 Ascariasis pneumonia E84.0 Cystic fibrosis with pulmonary manifestations J09.X1 Influenza due to identified novel influenza A virus with pneumonia J09.X2 Influenza due to identified novel influenza A virus with other -
Statistical Analysis Plan
Non-Interventional Study Protocol Study Code << DXXXRXXX >> Version V1.4 Date 14 July 2017 Decline In lung-function Among Patients with chronic obstructive Lung disease On maintenance therapy (DIAPLO) An observational study evaluating the benefits of early intervention with maintenance therapies to prevent or slow down rapid lung function decline in patients who are at high risk at the time of COPD diagnosis in the combined Optimum Patient Care Research Database and Clinical Practice Research Datalink databases TITLE PAGE Non-Interventional Study Protocol Study Code << DXXXRXXX >> Version 14 July 2017 Date 14 July 2017 TABLE OF CONTENTS PAGE TITLE PAGE ........................................................................................................... 1 TABLE OF CONTENTS ......................................................................................... 2 LIST OF ABBREVIATIONS .................................................................................. 5 RESPONSIBLE PARTIES ...................................................................................... 6 PROTOCOL SYNOPSIS DIAPLO STUDY ........................................................... 7 AMENDMENT HISTORY ................................................................................... 12 MILESTONES ....................................................................................................... 13 1. BACKGROUND AND RATIONALE .................................................................. 14 1.1 Background ........................................................................................................... -
Medicare Approved Six Minute Walk Diagnosis Codes
MEDICARE APPROVED DIAGNOSIS CODES FOR SIX MINUTE WALK TESTING ICD-10 Codes that Support Medical Necessity Group 1 Codes: ICD-10 CODE DESCRIPTION B44.81 Allergic bronchopulmonary aspergillosis C33 Malignant neoplasm of trachea C34.00 Malignant neoplasm of unspecified main bronchus C34.01 Malignant neoplasm of right main bronchus C34.02 Malignant neoplasm of left main bronchus C34.10 Malignant neoplasm of upper lobe, unspecified bronchus or lung C34.11 Malignant neoplasm of upper lobe, right bronchus or lung C34.12 Malignant neoplasm of upper lobe, left bronchus or lung C34.2 Malignant neoplasm of middle lobe, bronchus or lung C34.30 Malignant neoplasm of lower lobe, unspecified bronchus or lung C34.31 Malignant neoplasm of lower lobe, right bronchus or lung C34.32 Malignant neoplasm of lower lobe, left bronchus or lung C34.80 Malignant neoplasm of overlapping sites of unspecified bronchus and lung C34.81 Malignant neoplasm of overlapping sites of right bronchus and lung C34.82 Malignant neoplasm of overlapping sites of left bronchus and lung C34.90 Malignant neoplasm of unspecified part of unspecified bronchus or lung C34.91 Malignant neoplasm of unspecified part of right bronchus or lung C34.92 Malignant neoplasm of unspecified part of left bronchus or lung C78.00 Secondary malignant neoplasm of unspecified lung C78.01 Secondary malignant neoplasm of right lung C78.02 Secondary malignant neoplasm of left lung C78.30 Secondary malignant neoplasm of unspecified respiratory organ C78.39 Secondary malignant neoplasm of other respiratory organs -
Pulmonary Fibrosis with Cholesterol Granulomata in a Patient Working in a Fireworks Factory K M D J Rodrigo1, D a V Rathnapala1, W V Senaratne1, S R Constantine2
Picture stories The sputum culture was negative. Post operatively she polymerase chain reaction (PCR) to detect mycobacterial was treated with anti-tuberculous medications. The wound DNA and enzyme-linked immunosorbent assays (ELISAs) completely healed after 10 months. [4]. Tuberculosis (TB) remains a major global health problem. Isolated sacral tuberculosis usually presents Conflicts of interest as chronic back pain in adults and discharging sinuses We declare that there are no conflicts of interest. or abscess formation in children, with or without neurological deficit [2]. References Definitive diagnosis of tuberculosis involves de- monstration of Mycobacterium tuberculosis by microbio- 1. Kumar A, Varshney MK, Trikha V, Khan SA. Isolated tuberculosis of the coccyx. J Bone Joint Surg Br 2006; 88: logical, cytopathological or histopathological methods. 1388-9. Histological examination of the biopsy usually shows epithelioid granulomas, Langhans’ type multinucleated 2. KimDoUn, Kim SW, Ju CL. Isolated coccygeal tubercu- giant cells and caseous necrosis, as in this patient’s losis. J Korean Neurosurg Soc 2012; 52: 495-7. biopsy specimen [3]. Demonstration of acid fast bacilli 3. Kumar V, Abbas A, Fausto N, Aster J. Robbins and Cotran by special stains is also possible. Only 35-60% of cases Pathologic Basis of Disease: Chronic inflammation. th8 can be diagnosed by demonstrating acid-fast bacilli in edition. China: Saunders Elsevier 2010. the biopsy specimen. Culture of the biopsy material 4. Barker JA, Conway AM, Hill J. Supralevator fistula-in-ano -
Various Types of Pneumoconiosis
Pneumoconiosis MEANING Pneumoconiosis is a chronic lung disease caused due to the inhalation of various forms of dust particles, particularly in industrial workplaces, for an extended period of time. Hence it is also said to be an occupational lung disease, which are a particular subdivision of occupational related diseases that are related primarily to being exposed to harmful substances, whether they are gas or dusts, in the work place, and the pulmonary disorders that may result from it. The severity and type of pneumoconiosis depends on what the dust particles comprise of; for example, small amounts of certain substances, such as asbestos and silica, can lead to serious reactions, while others may not be as harmfulCoalworker's pneumoconiosis Various Types of Pneumoconiosis • The most common types of pneumoconiosis are: • coal workers’ pneumoconiosis, silicosis, • asbestosis. As is evident by their names, these pneumoconioses are caused due to the inhalation of coal mine dust, silica dust, and asbestos fibers. Usually, it takes several years for these pneumoconioses to develop and manifest themselves. However, sometimes, particularly with silicosis, it can develop quite rapidly, within a short period of being exposed to large amounts of silica dust. In their severe form, pneumoconioses often result in the impairment of the lungs, disability, and even untimely death. Asbestosis: This is caused due to the inhalation of fibrous minerals that asbestos is made of. The exposure begins with the baggers, who handle the asbestos by collecting them and packaging them, to workers that make products out of them such as insulation material, cement, and tiles, and people working in the shipbuilding industry, and construction workers. -
Bagassosis, Rare Cause of Hypersensitivity Pneumonitis: a Case Report
IBEROAMERICAN JOURNAL OF MEDICINE 04 (2020) 381-384 Journal homepage: www.iberoamericanjm.tk Case Report Bagassosis, rare cause of hypersensitivity pneumonitis: A case report Tyagi Aruna,*, Jadhav Sagara, Waghmare Manoja, Srivastava AKa, Khare ABa aDepartment of Medicine, DVPPF’s Medical College, Ahmednagar, Maharashtra, PIN-414111, India ARTICLE INFO ABSTRACT Article history: Hypersensitivity pneumonitis (HP), also called extrinsic allergic alveolitis, is a Received 14 May 2020 respiratory syndrome involving the lung parenchyma specifically the alveoli, Received in revised form 18 May terminal bronchioles and alveolar interstitium. HP is caused by delayed allergic 2020 reaction to variety of antigens like microbes, animal proteins, and low-molecular- weight chemicals. Bagassosis is one such HP caused by repetitive inhalation of Accepted 19 May 2020 bagasse. Though use of bagasse as cause of HP is known, use of bagasse in brick- kiln factory as fuel and its association with HP has not been well documented. We Keywords: report a case of HP in a brick-kiln worker where bagasse was used as fuel. Interstitial lung disease A twenty-five years old man, working in a brick kiln for five years, presented with Hypersensitivity pneumonia complaints of dry cough, low grade fever and weight loss for one month. He had Bagassosis bilateral lower lobe crackles on auscultation. The history of bagasse, clay and coal dust exposure, clinical presentation and imaging were suggestive of HP. However, being endemic in India, pulmonary tuberculosis was ruled out by negative Mantoux test, sputum for acid fast bacilli and Cartridge Based Nucleic Acid Amplification Test. He was managed with corticosteroids and symptomatic treatment with good response and resolution of clinical signs and radiological changes.