ASCM Web -Ascites.Rtf

Total Page:16

File Type:pdf, Size:1020Kb

ASCM Web -Ascites.Rtf Examination for Ascites Wash your hands & Introduce the exam to your patient Positioning & Draping · Position so that the patient’s abdominal muscles are relaxed. Therefore, the patient: o is lying flat o has arms at their sides o has a pillow · Drape so that the abdomen is visible from the nipples to at least the Anterior Superior Iliac Spines (ASIS’s) Inspection · General -Look for: o masses, scars, and lesions (trauma) o atrophy/hypertrophy o discolouration o swelling o muscle bulk/symmetry o distended abdomen · Specific to Ascites -Look for: o bulging flanks from the foot of the bed o peripheral edema · Ascites often presents with other stigmata of liver disease. Look for: o Jaundice § look at the sclera and frenulum o Hands § palmar erythema § Dupuytren's contracture § clubbing 1 © Michael Colapinto § Terry’s nails § thenar (thumb) wasting § leukonycia § asterixis · get the patient to cock their wrists back while holding their arms out straight · look for intermittent flapping o Estrogen dependent § spider nevi § gynecomastia § frontal balding § testicular atrophy o Liver dependent § jaundice & ascites § hepatomegaly or contracted liver § splenomegaly § hemorrhoids & esophageal varices § caput medusa § easy bruising & petechiae § edema o Miscellaneous § fetor hepaticus § encephalopathy § temporal wasting Special maneuvers · Test for Shifting Dullness (see Figure 1) o percuss at the centre of the abdomen then percuss toward the patient’s right flank and mark where dullness arises o roll patient into the Right lateral decubitus position and repeat your percussion technique 2 o with ascites, the area of dullness will shift to the dependent side (ie) the area of tympany shifts toward the top Figure 1: Shifting Dullness A B © Mary Sims In this diagram, it can be seen how the fluid in the patient’s abdomen shifts as they are moved from (A) the supine position into (B) the Right lateral decubitus position. Thus, the area of dullness on percussion shifts with the position change. · Fluid wave test (see Figure 2) o get the patient to place the radial edge of their hand and their index finger in the head-to-toe direction in the centre of their abdomen o place your hands on either side of the patient o gently tap one side of the abdomen and feel for the tap on the other side 3 Figure 2: Technique for Fluid Wave Test © Mary Sims 4 .
Recommended publications
  • General Signs and Symptoms of Abdominal Diseases
    General signs and symptoms of abdominal diseases Dr. Förhécz Zsolt Semmelweis University 3rd Department of Internal Medicine Faculty of Medicine, 3rd Year 2018/2019 1st Semester • For descriptive purposes, the abdomen is divided by imaginary lines crossing at the umbilicus, forming the right upper, right lower, left upper, and left lower quadrants. • Another system divides the abdomen into nine sections. Terms for three of them are commonly used: epigastric, umbilical, and hypogastric, or suprapubic Common or Concerning Symptoms • Indigestion or anorexia • Nausea, vomiting, or hematemesis • Abdominal pain • Dysphagia and/or odynophagia • Change in bowel function • Constipation or diarrhea • Jaundice “How is your appetite?” • Anorexia, nausea, vomiting in many gastrointestinal disorders; and – also in pregnancy, – diabetic ketoacidosis, – adrenal insufficiency, – hypercalcemia, – uremia, – liver disease, – emotional states, – adverse drug reactions – Induced but without nausea in anorexia/ bulimia. • Anorexia is a loss or lack of appetite. • Some patients may not actually vomit but raise esophageal or gastric contents in the absence of nausea or retching, called regurgitation. – in esophageal narrowing from stricture or cancer; also with incompetent gastroesophageal sphincter • Ask about any vomitus or regurgitated material and inspect it yourself if possible!!!! – What color is it? – What does the vomitus smell like? – How much has there been? – Ask specifically if it contains any blood and try to determine how much? • Fecal odor – in small bowel obstruction – or gastrocolic fistula • Gastric juice is clear or mucoid. Small amounts of yellowish or greenish bile are common and have no special significance. • Brownish or blackish vomitus with a “coffee- grounds” appearance suggests blood altered by gastric acid.
    [Show full text]
  • PERFORATED PEPTIC ULCER. Patient Usually Experiences
    Postgrad Med J: first published as 10.1136/pgmj.12.134.470 on 1 December 1936. Downloaded from 470 POST-GRADUATE MEDICAL JOURNAL December, 1936 PERFORATED PEPTIC ULCER. By RONALD W. RAVEN, F.R.C.S. (Assistant Surgeon to T'he French Hospital, Assistant Surgeon to The Gordon Hospital for Rectal Diseases and Swrgical Registrar to The Royal Cancer Hospital.) INTRODUCTION. Peptic ulceration is a crippling disease judged from the stand-point of morbidity, and is also dangerous to life on account of serious complications, such as haemorrhage or perforation which may supervene during the course of the disease. These complications may occur in any patient and there are no criteria which will indicate whether or not an ulcer will bleed or perforate. When the treatment of peptic ulceration is under review it must be remembered that from 20 to 30 per cent. of these ulcers perforate. In a large series of cases I found that the incidence of perforation was 27 per cent. It is thus essential that patients suffering with peptic ulcer should be kept under continuous careful observation. Unfortunately, however, a small percentage of patients give no previous history of the peptic ulcer syndrome and perforation of the ulcer is the first indication of its presence. Recently, when considering the role of surgery in the treatment of chronic peptic ulcer, Joll stated that there has been a rise in the incidence of perforation as a complication of peptic ulcer since medical treatment has become systematized in the treatment of this disease. It must also be remembered that medical treat- Protected by copyright.
    [Show full text]
  • Pathophysiology, Diagnosis, and Management of Pediatric Ascites
    INVITED REVIEW Pathophysiology, Diagnosis, and Management of Pediatric Ascites ÃMatthew J. Giefer, ÃKaren F. Murray, and yRichard B. Colletti ABSTRACT pressure of mesenteric capillaries is normally about 20 mmHg. The pediatric population has a number of unique considerations related to Intestinal lymph drains from regional lymphatics and ultimately the diagnosis and treatment of ascites. This review summarizes the physio- combines with hepatic lymph in the thoracic duct. Unlike the logic mechanisms for cirrhotic and noncirrhotic ascites and provides a sinusoidal endothelium, the mesenteric capillary membrane is comprehensive list of reported etiologies stratified by the patient’s age. relatively impermeable to albumin; the concentration of protein Characteristic findings on physical examination, diagnostic imaging, and in mesenteric lymph is only about one-fifth that of plasma, so there abdominal paracentesis are also reviewed, with particular attention to those is a significant osmotic gradient that promotes the return of inter- aspects that are unique to children. Medical and surgical treatments of stitial fluid into the capillary. In the normal adult, the flow of lymph ascites are discussed. Both prompt diagnosis and appropriate management of in the thoracic duct is about 800 to 1000 mL/day (3,4). ascites are required to avoid associated morbidity and mortality. Ascites from portal hypertension occurs when hydrostatic Key Words: diagnosis, etiology, management, pathophysiology, pediatric and osmotic pressures within hepatic and mesenteric capillaries ascites produce a net transfer of fluid from blood vessels to lymphatic vessels at a rate that exceeds the drainage capacity of the lym- (JPGN 2011;52: 503–513) phatics. It is not known whether ascitic fluid is formed predomi- nantly in the liver or in the mesentery.
    [Show full text]
  • Hepatálny Ascites a Jeho Komplikácie
    Ascites - principles of diagnosis and treatment Practical course in Internal medicine, 4. year GM I. Department of Internal medicine Faculty of Medicine, Comenius University & University Hospital Bratislava Summer semester 2019/2020 Ascites • Ascites describes the condition of pathologic fluid collection within the abdominal cavity • Word „ascites“ is of Greek origin (askos) - and means bag or sac • Healthy men have little or no intraperitoneal fluid, but women may normally have as much as 20 mL, depending on the phase of their menstrual cycle Ascites - etiology 3 2 5 Liver cirrhosis (ALD 60%, HBV/HCV infection 10%, cryptogenic 10% 10 Malignity Heart failure Tuberculosis Other 80 Ascites – etiology Portal hypertension Portal hypertension PRESENCE ABSENCE SAAG ≥ 11 g/l SAAG < 11 g/l • Hypoalbuminemia • Prehepatic • Nephrotic syndrome • Splenic or portal vein trombosis • Severe malnutrition • Schistosomiasis • Protein-losing enteropathy • Intrahepatic • Malignancy • Pre-/intra-/postsinusoidal • Liver cirrhosis • Infection • Alcohol liver diesease • Tuberculosis • Infective, autimunne, toxic, other ... • Spontaneous bacterial peritonitis • Liver metastases • Pancreatitis • Posthepatic • Polyserositis in systemic or • Budd-Chiari syndrome, veno- occlusive disease endocrinal disorders • Cardiac (hypothyreosis, connestive • Right heart failure tissue diseases, vasculitis ...) • Constrictive pericarditis • Meigs syndrome Ascites – patophysiology disturbance of the balance between the formation and absorption of free abdominal fluid ↓ Fluid absorption ↑ Fluid production Ascites – combination of mechanisms in liver cirrhosis Natural history of chronic liver disease 1. Latent - hidden disorder of water and sodium management - decreased peripheral vascular resistance, increased minute volume, ECT expansion, no edema, normal activity RAAS, ADH, SNS) 2. Positive sodium balance - inability to exclude Na load, ECT expansion, swelling, mild ascites, normal function of RAAS, SNS, ADH 3.
    [Show full text]
  • Abdominal Examination
    Abdominal Examination Introduction Wash hands, Introduce self, ask Patients name & DOB & what they like to be called, Explain examination and get consent Expose and lie patient flat General Inspection Patient: stable, pain/discomfort, jaundice, pallor, muscle wasting/cachexia Around bed: vomit bowels etc Hands Flapping tremor (hepatic encephalopathy) Nails: clubbing (cirrhosis, IBD, coeliacs), leukonychia (hypoalbuminemia in liver cirrhosis), koilonychia (iron deficiency anaemia) Palms: palmar erythema (hyperdynamic circulation due to ↑oestrogen levels in liver disease/ pregnancy), Dupuytren’s contracture (familial, liver disease), fingertip capillary glucose monitoring marks (diabetes) Head Eyes: sclera for jaundice (liver disease), conjunctival pallor (anaemia e.g. bleeding, malabsorption), periorbital xanthelasma (hyperlipidaemia in cholestasis) Mouth: glossitis/stomatitis (iron/ B12 deficiency anaemia), aphthous ulcers (IBD), breath odor (e.g. faeculent in obstruction; ketotic in ketoacidosis; alcohol) Neck and torso Ask patient to sit forwards: Neck: feel for lymphadenopathy from behind – especially Virchow's node (gastric malignancy) Back inspection: spider naevi (>5 significant), skin lesions (immunosuppression) Ask patient to relax back: Chest inspection: spider naevi (>5 significant), gynaecomastia, loss of axillary hair (all due to ↑oestrogen levels in liver disease/ pregnancy) Abdomen Inspection: distension (Fluid, Flatus, Fat, Foetus, Faeces), incisional hernias (ask patient to cough), scars, striae (pregnancy,
    [Show full text]
  • A Rare Case of Ascites
    Clinical Medicine 2019 Vol 19, No 2: s73 CLINICAL A r a r e c a s e o f a s c i t e s Authors: S h u a n n S h w a n a , A d n a n U r R a h m a n a n d E l i z a b e t h S l o w i n s k a A i m s most cases depends on whether it is idiopathic or secondary. The mainstay of treatment is corticosteroids; if there is no response, A 69-year-old gentleman presented with a 5-week history of immunosuppressive therapy can be used. Case-series data exist, abdominal distension. He had a past history of diabetes and which show that high-dose corticosteroids like prednisolone are myocardial infarction, and was an ex-smoker with no significant effective in reducing the chronic inflammatory response caused by history of alcohol intake. Examination demonstrated a distended, retroperitoneal fibrosis; however, there is a high rate of recurrence non-tender abdomen with shifting dullness, no organomegaly and once the steroids are withdrawn. Mycophenolate mofetil in addition no signs of chronic liver disease. to corticosteroids has been shown to reduce duration of steroid use without affecting the efficacy and reduces disease recurrence. ■ M e t h o d s Investigations included an ultrasound scan of the abdomen, an Conflict of interest statement ascitic tap, a computed tomography (CT) of the abdomen/pelvis N o n e d e c l a r e d .
    [Show full text]
  • Advanced Assessment in Clinical Practice: Abdominal Assessment
    Abdominal assessment ______________________________________________________________________ Advanced Assessment in Clinical Practice: Abdominal Assessment I. Assessment of the abdomen A. Basic anatomy and physiology 1. Esophagus 2. Stomach 3. Small intestines 4. Large intestines 5. Liver ∑ Hepatic artery ∑ Portal vein ∑ Hepatic veins ∑ Metabolizes CHO, fats and proteins. ∑ Stores vitamins, minerals, and iron. ∑ Detoxifies harmful substances. ∑ Produces antibodies. ∑ Makes hormones, prothrombin, fibrinogen and protein. 6. Gallbladder 7. Pancreas 8. Spleen 9. Kidneys 10. Bladder Advanced Assessment Page 60 ©Educational Concepts, LLC Abdominal assessment ______________________________________________________________________ B. Areas of the abdomen Right upper quadrant Left upper quadrant Liver and gallbladder Left lobe of the liver Pylorus Spleen Duodenum Stomach Head of the pancreas Body of the pancreas Right adrenal gland Left adrenal gland Portion of the right kidney Portion of the left kidney Hepatic flexure of the colon Splenic flexure of the colon Portions of the ascending and Portions of the transverse and transverse colon descending colon Right lower quadrant Left lower quadrant Lower pole of the right kidney Lower pole of the left kidney Cecum and appendix Sigmoid colon Portion of the ascending colon Portion of the descending colon Bladder if distended Bladder if distended Ovary and salpinx Ovary and salpinx Uterus if enlarged Uterus if enlarged Right spermatic cord Left spermatic cord Right ureter Left ureter C. Assessment parameters
    [Show full text]
  • Ilbpnp55ecxs1d55pt2h4cnta 12-Year-Old Girl with Chronic Vomiting
    A 12-year-old girl with chronic vomiting and epigastric pain History: A 12-year-old girl has presented with vomiting for 1 month. The frequency is about 2-3 times a day. The vomitus contains digested food without bile stain. The emesis is not related to meals. After vomiting, she can normally eat. Her bowel movement is once daily with normal stools. There is no fever. After 2 days of symptoms, she was treated with antibiotics and antiemesis with a diagnosis of gastroenteritis. However, the symptom persists. Three weeks prior to admission, the vomiting became worsening (7-8 times a day) and occurred a half an hour after meals. Its characteristics remained the same. At this time, she also complained of epigastric pain, weakness, and anorexia. She was admitted to a primary care hospital and treated with intravenous fluid. The symptoms seemed to be improved. However, the vomiting resumed with a history of foul-smell watery diarrhea 2 time a day for 1 weeks before admission to Children's hospital. She lost significant weight during the illness. Past history: She was admitted because of Dengue hemorrhagic fever 2 years ago. Perinatal and immunization history are normal. There is no genetic and atopic diseases in the family. The patient refuses a history of contact tuberculosis. She denies regular uncooked food ingestion. Physical examination: General appearance- Thai girl, obesity, alert Body weight 80 kg, height 158 cm Vital signs: T 37 C, PR 80/min, RR 18/min, BP 110/70 mmHg HEENT: not pale, no jaundice, pharynx and tonsils-not injected, normal TM Heart: regular rhythm, no murmur Lungs: normal breath sound, no adventitious sound Abdomen: soft, mild distension, mild tenderness at epigastrium, no guarding, no rigidity, active bowel sound, no abnormal mass, fluid thrill and shifting dullness can not be evaluated.
    [Show full text]
  • Diagnostic Approach of Patient with Ascites: a Case Report
    International Journal of Science and Research (IJSR) ISSN: 2319-7064 SJIF (2019): 7.583 Diagnostic Approach of Patient with Ascites: A Case Report Theodore Dharma Tedjamartono1, Ketut Suryana2 1Intern of Internal Medicine Department in Wangaya Hospital, Denpasar, Bali, Indonesia Correspondence: theodored71[at]gmail.com 2Internist of Internal Medicine Department in Wangaya Hospital, Denpasar, Bali, Indonesia ketutsuryana[at]gmail.com Abstract: Ascites is an accumulation of fluid in the peritoneal cavity due to increase of capillary permeability, portal venous pressure, oncotic pressure, or lymphatic obstruction. From all the pathophysiological mechanisms mentioned, about 80% of the cases occur due to increased portal venous pressure (portal hypertension). It has been known that portal hypertension is associated with chronic liver disease (CLD). Nevertheless, ascites can also be found in several diseases such as kidney disease, heart disease, infection, malignancy or others. Determining the underlying disease of ascites is a challenge for the clinician; anamnesis including the course of the disease, risk factors, comorbidities are needed to be done properly. Ascites usually accompanied by other symptoms, for example in liver disease (signs of portal hypertension), kidney disease (anasarca), heart failure (increased jugular venous pressure, murmurs, gallops), and malignancies (mass, lymphadenopathy), thus physical examination should be performed in every patient carefully. In minimal amount of fluid, abdominal ultrasonography (USG) are recommended.
    [Show full text]
  • Abdominal Pain
    10 Abdominal Pain Adrian Miranda Acute abdominal pain is usually a self-limiting, benign condition that irritation, and lateralizes to one of four quadrants. Because of the is commonly caused by gastroenteritis, constipation, or a viral illness. relative localization of the noxious stimulation to the underlying The challenge is to identify children who require immediate evaluation peritoneum and the more anatomically specific and unilateral inner- for potentially life-threatening conditions. Chronic abdominal pain is vation (peripheral-nonautonomic nerves) of the peritoneum, it is also a common complaint in pediatric practices, as it comprises 2-4% usually easier to identify the precise anatomic location that is produc- of pediatric visits. At least 20% of children seek attention for chronic ing parietal pain (Fig. 10.2). abdominal pain by the age of 15 years. Up to 28% of children complain of abdominal pain at least once per week and only 2% seek medical ACUTE ABDOMINAL PAIN attention. The primary care physician, pediatrician, emergency physi- cian, and surgeon must be able to distinguish serious and potentially The clinician evaluating the child with abdominal pain of acute onset life-threatening diseases from more benign problems (Table 10.1). must decide quickly whether the child has a “surgical abdomen” (a Abdominal pain may be a single acute event (Tables 10.2 and 10.3), a serious medical problem necessitating treatment and admission to the recurring acute problem (as in abdominal migraine), or a chronic hospital) or a process that can be managed on an outpatient basis. problem (Table 10.4). The differential diagnosis is lengthy, differs from Even though surgical diagnoses are fewer than 10% of all causes of that in adults, and varies by age group.
    [Show full text]
  • Examination of the Abdomen
    06/11/1431 Examination of the Abdomen Chapter 10 Ra'eda Almashaqba 1 Review Anatomy Rectus Abdominis Xiphoid Process Costal Margin Linea Alba Anterior Superior Iliac Spine Symphysis Pubis Ra'eda Almashaqba 2 Inguinal Ligament 1 06/11/1431 Ra'eda Almashaqba 3 9 Regions Ra'eda Almashaqba 4 2 06/11/1431 Ra'eda Almashaqba 5 Ra'eda Almashaqba 6 3 06/11/1431 Location! Location! Location! RUQ liver gallbladder duodenum (small intestine) pancreas head right kidney and adrenal Ra'eda Almashaqba 7 Location! Location! Location! RLQ cecum appendix right ovary and tube Ra'eda Almashaqba 8 4 06/11/1431 Location! Location! Location! LLQ sigmoid colon left ovary and tube LUQ stomach spleen pancreas left kidney and adrenal Ra'eda Almashaqba 9 GI Variations Due to Age Aging- should not affect GI function unless associated with a disease process Decreased: salivation, sense of taste, gastric acid secretion, esophageal emptying, liver size, bacterial flora Increased: constipation! Ra'eda Almashaqba 10 5 06/11/1431 Health History Gastrointestinal Disorder Indigestion, N&V, Anorexia, Hematemesis - Ask the pt how is your appetite Indigestion ----- distress associated with eating Heartburn ---- sense of burning or warmth that is retrosternal and may radiate to the neck Excessive gas: frequent belching, distention or flatulence ,Abd fullness. Dysphagia & odynophagia Change in bowel function Constipation or diarrhea Jaundice Ra'eda Almashaqba 11 Abdominal pain: Visceral : Occur in all the abd, burning, aching, difficult to localize, varies in quality
    [Show full text]
  • Abdominal Examination Positioning
    ABDOMINAL EXAMINATION POSITIONING Patients hands remain on his/hers side Legs, straight Head resting on pillow – if neck is flexed, ABD muscles will tense and therefore harder to palpate ABD . INSPECTION AUSCULATION PALPATION PERCUSSION INSPECTION INSPECTION Shape Skin Abnormalities Masses Scars (Previous op's - laproscopy) Signs of Trauma Jaundice Caput Medusae (portal H-T) Ascities (bulging flanks) Spider Navi-Pregnant women Cushings (red-violet) ... Hands + Mouth Clubbing Palmer Erythmea Mouth ulceration Breath (foeter ex ore) ... AUSCULTATION Use stethoscope to listen to all areas Detection of Bowel sounds (Peristalsis/Silent?? = Ileus) If no bowel sounds heard – continue to auscultate up to 3mins in the different areas to determine the absence of bowel sounds Auscultate for BRUITS!!! - Swishing (pathological) sounds over the arteries (eg. Abdominal Aorta) ... PALPATION ALWAYS ASK IF PAIN IS PRESENT BEFORE PALPATING!!! Firstly: Superficial palpation Secondly: Deep where no pain is present. (deep organs) Assessing Muscle Tone: - Guarding = muscles contract when pressure is applied - Ridigity = inidicates peritoneal inflamation - Rebound = Releasing of pressure causing pain ....... MURPHY'S SIGN Indication: - pain in U.R.Quadrant Determines: - cholecystitis (inflam. of gall bladder) - Courvoisier's law – palpable gall bladder, yet painless - cholangitis (inflam. Of bile ducts) ... METHOD Ask patient to breathe out. Gently place your hand below the costal margin on the right side at the mid-clavicular line (location of the gallbladder). Instruct to breathe in. Normally, during inspiration, the abdominal contents are pushed downward as the diaphragm moves down. If the patient stops breathing in (as the gallbladder comes in contact with the examiner's fingers) the patient feels pain with a 'catch' in breath.
    [Show full text]