Nr 1. How Long Does It Take for a Dentin Bridge to Form After Calcium Hydroxide Application?
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Glossary for Narrative Writing
Periodontal Assessment and Treatment Planning Gingival description Color: o pink o erythematous o cyanotic o racial pigmentation o metallic pigmentation o uniformity Contour: o recession o clefts o enlarged papillae o cratered papillae o blunted papillae o highly rolled o bulbous o knife-edged o scalloped o stippled Consistency: o firm o edematous o hyperplastic o fibrotic Band of gingiva: o amount o quality o location o treatability Bleeding tendency: o sulcus base, lining o gingival margins Suppuration Sinus tract formation Pocket depths Pseudopockets Frena Pain Other pathology Dental Description Defective restorations: o overhangs o open contacts o poor contours Fractured cusps 1 ww.links2success.biz [email protected] 914-303-6464 Caries Deposits: o Type . plaque . calculus . stain . matera alba o Location . supragingival . subgingival o Severity . mild . moderate . severe Wear facets Percussion sensitivity Tooth vitality Attrition, erosion, abrasion Occlusal plane level Occlusion findings Furcations Mobility Fremitus Radiographic findings Film dates Crown:root ratio Amount of bone loss o horizontal; vertical o localized; generalized Root length and shape Overhangs Bulbous crowns Fenestrations Dehiscences Tooth resorption Retained root tips Impacted teeth Root proximities Tilted teeth Radiolucencies/opacities Etiologic factors Local: o plaque o calculus o overhangs 2 ww.links2success.biz [email protected] 914-303-6464 o orthodontic apparatus o open margins o open contacts o improper -
Oral Health in Prevalent Types of Ehlers–Danlos Syndromes
View metadata, citation and similar papers at core.ac.uk brought to you by CORE provided by Ghent University Academic Bibliography J Oral Pathol Med (2005) 34: 298–307 ª Blackwell Munksgaard 2005 Æ All rights reserved www.blackwellmunksgaard.com/jopm Oral health in prevalent types of Ehlers–Danlos syndromes Peter J. De Coster1, Luc C. Martens1, Anne De Paepe2 1Department of Paediatric Dentistry, Centre for Special Care, Paecamed Research, Ghent University, Ghent; 2Centre for Medical Genetics, Ghent University Hospital, Ghent, Belgium BACKGROUND: The Ehlers–Danlos syndromes (EDS) Introduction comprise a heterogenous group of heritable disorders of connective tissue, characterized by joint hypermobility, The Ehlers–Danlos syndromes (EDS) comprise a het- skin hyperextensibility and tissue fragility. Most EDS erogenous group of heritable disorders of connective types are caused by mutations in genes encoding different tissue, largely characterized by joint hypermobility, skin types of collagen or enzymes, essential for normal pro- hyperextensibility and tissue fragility (1) (Fig. 1). The cessing of collagen. clinical features, modes of inheritance and molecular METHODS: Oral health was assessed in 31 subjects with bases differ according to the type. EDS are caused by a EDS (16 with hypermobility EDS, nine with classical EDS genetic defect causing an error in the synthesis or and six with vascular EDS), including signs and symptoms processing of collagen types I, III or V. The distribution of temporomandibular disorders (TMD), alterations of and function of these collagen types are displayed in dental hard tissues, oral mucosa and periodontium, and Table 1. At present, two classifications of EDS are was compared with matched controls. -
Regional Odontodysplasia: Report of an Unusual Case Involving Mandibular Arch
Regional odontodysplasia: Report of an unusual case involving mandibular arch N. S. Venkatesh Babu, R. Jha Smriti, D. Bang Pratima Abstract Regional odontodysplasia (RO) is a rare developmental anomaly involving both mesodermal and ectodermal components in primary or permanent dentition. It affects the maxilla and the mandible or both; however, maxilla is more commonly involved. This article reports the case of 33-month-old boy who came with the chief complaint of delayed eruption of mandibular teeth. Findings of clinical and radiographic examination were consistent with those of RO. Maxillary dentition was unaffected. Clinical and radiographic features and treatment options are discussed. Keywords: Mandibular arch, primary teeth, regional odontodysplasia Introduction cases of mandibular involvement have been reported so far.[5,8,9] Regional odontodysplasia (RO) is a rare developmental dental anomaly that involves ectoderm and mesoderm The teeth with RO often display a brownish or yellowish derived tissues.[1] It can affect either primary or permanent discoloration and most frequent clinical symptoms dentition.[2] This condition was first described by Hitchin accompanied by this anomaly are failure of eruption and in 1934. The prevalence of this condition is still not clear gingival enlargement. Radiologically, the affected teeth since the studies reported till date have mainly been based illustrate hypoplastic crowns and lack of contrast between on case reports. enamel and dentin is usually apparent. Enamel and the dentin are very thin, -
Management of Dental Trauma in a Primary Care Setting Abstract
Guidance for the Clinician in Rendering Pediatric Care CLINICAL REPORT Management of Dental Trauma in a Primary Care Setting Martha Ann Keels, DDS, PhD, and THE SECTION ON ORAL abstract HEALTH The American Academy of Pediatrics and its Section on Oral Health have KEY WORDS developed this clinical report for pediatricians and primary care physi- dental trauma, dental injury, tooth, teeth, dentist, pediatrician cians regarding the diagnosis, evaluation, and management of dental ABBREVIATION trauma in children aged 1 to 21 years. This report was developed CT—computed tomography through a comprehensive search and analysis of the medical and den- This document is copyrighted and is property of the American tal literature and expert consensus. Guidelines published and updated Academy of Pediatrics and its Board of Directors. All authors have filed conflict of interest statements with the American by the International Association of Dental Traumatology (www.dental- Academy of Pediatrics. Any conflicts have been resolved through traumaguide.com) are an excellent resource for both dental and non- a process approved by the Board of Directors. The American dental health care providers. Pediatrics 2014;133:e466–e476 Academy of Pediatrics has neither solicited nor accepted any commercial involvement in the development of the content of this publication. The guidance in this report does not indicate an exclusive INTRODUCTION course of treatment or serve as a standard of medical care. Variations, taking into account individual circumstances, may be By 14 years of age, 30% of children have experienced a dental injury.1 appropriate. Many of these children are taken directly to their medical home, an urgent care center, or an emergency department for evaluation and treatment. -
Saving Smiles Avulsion Pathway (Page 20) Saving Smiles: Fractures and Displacements (Page 22)
Greater Manchester Local Dental Network SavingSmiles Improving outcomes following dental trauma First Edition I Spring 2017 Practitioners’ Toolkit Contents 04 Introduction to the toolkit from the GM Trauma Network 06 History & examination 10 Maxillo-facial considerations 12 Classification of dento-alveolar injuries 16 The paediatric patient 18 Splinting 20 The AVULSED Tooth 22 The BROKEN Tooth 23 Managing injuries with delayed presentation SavingSmiles 24 Follow up Improving outcomes 26 Long term consequences following dental trauma 28 Armamentarium 29 When to refer 30 Non-accidental injury 31 What should I do if I suspect dental neglect or abuse? 34 www.dentaltrauma.co.uk 35 Additional reference material 36 Dental trauma history sheet 38 Avulsion pathways 39 Fractues and displacement pathway 40 Fractures and displacements in the primary dentition 41 Acknowledgements SavingSmiles Improving outcomes following dental trauma Ambition for Greater Manchester Introduction to the Toolkit from The GM Trauma Network wish to work with our colleagues to ensure that: the GM Trauma Network • All clinicians in GM have the confidence and knowledge to provide a timely and effective first line response to dental trauma. • All clinicians are aware of the need for close monitoring of patients following trauma, and when to refer. The Greater Manchester Local Dental Network (GM LDN) has established a ‘Trauma Network’ sub-group. The • All settings have the equipment described within the ‘armamentarium’ section of this booklet to support optimal treatment. Trauma Network was established to support a safer, faster, better first response to dental trauma and follow up care across GM. The group includes members representing general dental practitioners, commissioners, To support GM practitioners in achieving this ambition, we will be working with Health Education England to provide training days and specialists in restorative and paediatric dentistry, and dental public health. -
Spectrum of Dentin Dysplasia in a Family
CASE REPORT Spectrumof dentin dysplasia in a family: case report and literature review W. Kim Seow, BDS, MDSc, DDSc, PhD Stephen Shusterman, DMD Abstract The dentin dysplasias (DD),which maybe classified as type I (DD1)or type 2 (DD2),form a group of rare, inherited abnormalitiesthat are clinically distinct fromdentinogenesis imperfecta. Studies of affected families mayhelp to distinguish different types of DDand provide further insight into their etiology and clinical management.This report describes a family that showed characteristic dental features of DD1, including clinically normalcrowns in both primaryand permanentdentitions, and mobileteeth that maybe associated with prematureexfoliation. Radiographicfeatures included calcification of the pulp with crescent-shaped, radiolucent pulp remnants, short, tapering, taurodontic roots, and manyperiapical pathoses that maybe q¢sts or granulomas.A spectrumof dentin dysplasia wasnoted within the family. Strategies to prevent pulp and periapical infections and early exfoliation of the teeth include meticulousoral hygieneand effective caries-preventivemeasures. ( P ed iatr Dent16:437-42,1994) Introduction and literature review Histologically, in DD1, most of the coronal and Dentin dysplasias (DD) form a group of rare dentin mantle dentin of the root is usually reported to be abnormalities that are clinically distinct from normal, and the dentin defect is confined mainly to the dentinogenesis imperfecta. 1-3 Since its recognition in root2, s, 10 The dysplastic dentin has been reported to 19203 as "rootless teeth" and as "dentin dysplasia" by consist of numerous denticles, containing whorls of Rushton in 1933,4 the clinical features of DDhave been osteodentin that block the normal course of the den- tinal tubules,s, 10,11 well described. -
Journal 2017
Journal of ENT masterclass ISSN 2047-959X Journal of ENT MASTERCLASS® Year Book 2017 Volume 10 Number 1 YEAR BOOK 2017 VOLUME 10 NUMBER 1 JOURNAL OF ENT MASTERCLASS® Volume 10 Issue 1 December 2017 Contents Free Courses for Trainees, Consultants, SAS grades, GPs & Nurses Welcome Message 3 CALENDER OF FREE RESOURCES 2018-19 Hesham Saleh Increased seats for specialist registrars & exam candidates ENT aspects of cystic fibrosis management 4 Gary J Connett ® 15th Annual International ENT Masterclass Paediatric swallowing disorders 8 Venue: Doncaster Royal Infirmary, 25-27th January 2019 Hayley Herbert and Shyan Vijayasekaran Special viva sessions for exam candidates Paediatric tongue-tie 14 Steven Frampton, Ciba Paul, Andrea Burgess and Hasnaa Ismail-Koch rd ® 3 ENT Masterclass China Paediatric oesophageal foreign bodies 20 Beijing, China, 12-13th May 2018 Emily Lowe, Jessica Chapman, Ori Ron and Michael Stanton Biofilms in paediatric otorhinolaryngology 26 3rd ENT Masterclass® Europe S Goldie, H Ismail-Koch, P.G. Harries and R J Salib Berlin, Germany, 14-15th Sept 2018 Intracranial complications of ear, nose and throat infections in childhood 34 Alice Lording, Sanjay Patel and Andrea Whitney ® ENT Masterclass Switzerland The superior canal dehiscence syndrome 41 Lausanne, 5-6th Oct 2018 Simon Richard Mackenzie Freeman Tympanosclerosis 46 ® ENT Masterclass Sri Lanka Priya Achar and Harry Powell Colombo, 16-17th Nov 2018 Endoscopic ear surgery 49 Carolina Wuesthoff, Nicholas Jufas and Nirmal Patel o Limited places, on first come basis. Early applications advised. o Masterclass lectures, Panel discussions, Clinical Grand Rounds Vestibular function testing 57 o Oncology, Plastics, Pathology, Radiology, Audiology, Medico-legal Karen Lindley and Charlie Huins Auditory brainstem implantation 63 Website: www.entmasterclass.com Harry R F Powell and Shakeel S Saeed CYBER TEXTBOOK on operative surgery, Journal of ENT Masterclass®, Surgical management of temporal bone meningo-encephalocoele and CSF leaks 69 Application forms Mr. -
Pulp Canal Obliteration After Traumatic Injuries in Permanent Teeth – Scientific Fact Or Fiction?
CRITICAL REVIEW Endodontic Therapy Pulp canal obliteration after traumatic injuries in permanent teeth – scientific fact or fiction? Juliana Vilela BASTOS(a) Abstract: Pulp canal obliteration (PCO) is a frequent finding associated (b) Maria Ilma de Souza CÔRTES with pulpal revascularization after luxation injuries of young permanent teeth. The underlying mechanisms of PCO are still unclear, (a) Universidade Federal de Minas Gerais - and no experimental scientific evidence is available, except the results UFMG, School of Dentistry, Department of Restorative Dentistry, Belo Horizonte, MG, of a single histopathological study. The lack of sound knowledge Brazil. concerning this process gives rise to controversies, including the (b) Pontifícia Universidade Católica de Minas most suitable denomination. More than a mere semantic question, Gerais – PUC-MG, Department of Dentistry, the denomination is an important issue, because it reflects the nature Belo Horizonte, MG, Brazil. of this process, and directly impacts the treatment plan decision. The hypothesis that accelerated dentin deposition is related to the loss of neural control over odontoblastic secretory activity is well accepted, but demands further supportive studies. PCO is seen radiographically as a rapid narrowing of pulp canal space, whereas common clinical features are yellow crown discoloration and a lower or non-response to sensibility tests. Late development of pulp necrosis and periapical disease are rare complications after PCO, rendering prophylactic endodontic intervention -
Hereditary Disorders of Dentin: Dentinogenesis Imperfecta Type II
CASE REPORT Hereditary Disorders of Dentin: Dentinogenesis Imperfecta Type II and Dentin Dysplasia Type II Sandhya Shanmugam1, Kuzhanchinathan Manigandan2, Angambakkam Rajasekaran PradeepKumar3 ABSTRACT Dentin is a mineralized tissue in tooth, produced from odontoblasts, that differentiates from the mesenchymal cells of dental papilla. Hereditary dentin defects are broadly classified into two types, namely, dentinogenesis imperfecta (DGI – type I and II) and dentin dysplasia (DD – type I and II). DGI is an autosomal dominant hereditary disorder, and DD is a rare hereditary disturbance of dentin formation that affects both the primary and the permanent dentition. The purpose of this report was to present a case of DGI–type II and a case of DD–type II to highlight the importance of diagnosing hereditary dentin disorders. Keywords: Dentin, Dentin discoloration, Dentin disorders, Dentin dysplasia, Dentinogensis imperfecta. Journal of Operative Dentistry and Endodontics (2020): 10.5005/jp-journals-10047-0091 INTRODUCTION 1,3Department of Conservative Dentistry and Endodontics, Thai Dentin is a mineralized tissue forming the the body of a tooth, which Moogambigai Dental College and Hospital, Dr M.G.R. Educational and serves as a protective covering for the pulp and supports overlying Research Institute, Chennai, Tamil Nadu, India enamel and cementum. Mature dentin is about 70% mineral, 20% 2Department of Conservative Dentistry and Endodontics, Faculty of organic matrix, and 10% water by weight. Dentin is the product of Dental Sciences, Sri Ramachandra Institute of Higher Education and specialized cells called odontoblasts.1 Research, Chennai, Tamil Nadu, India Hereditary developmental disorders affecting dentin are Corresponding Author: Angambakkam Rajasekaran PradeepKumar, rare anomalies occurring due to a genetic defect in structural Department of Conservative Dentistry and Endodontics, Thai or regulatory proteins in dentin. -
Pulp Canal Obliteration- a Daunting Clinical Challenge
IOSR Journal of Dental and Medical Sciences (IOSR-JDMS) e-ISSN: 2279-0853, p-ISSN: 2279-0861.Volume 19, Issue 3 Ser.16 (March. 2020), PP 56-60 www.iosrjournals.org Pulp Canal Obliteration- A Daunting Clinical Challenge Ashish Jain1, P Shanti Priya2, Ronald Tejpaul3, Basa Srinivas Karteek4 1(Reader, Department of Conservative dentistry and Endodontics, PanineeyaInstitute of Dental Sciences, India) 2(Senior lecturer, Department of Conservative dentistry and Endodontics, Panineeya Institute of Dental Sciences, India) 3 (Senior lecturer, Department of Conservative dentistry and Endodontics, Oxford Dental College, India) 4(Senior lecturer, Department of Conservative dentistry and Endodontics, Panineeya Institute of Dental Sciences, India) Abstract: Traumatic injuries of primary or permanent dentition can lead to certain clinical complications and its management presents a considerable challenge for apractitioner. Pulp canal obliteration (PCO) also called as calcific metamorphosis (CM) following dental trauma has been stated to be approximately 37 - 40% and commonly noticed after luxation injuries. The diagnostic status and treatment planning decision regarding PCO always remains controversial. The decision on when to start treatment of such cases, whether on early detection of PCO or to wait until detection of signs and symptoms of pulp necrosis, always remains a clinical dilemma. Following calcific metamorphosis, scouting for the canals and negotiating it to full working length may lead to iatrogenic errors such as perforations or instrument separation. This article emphasizes on protocols to be followed on diagnosing and treating PCO and challenges that are to be encountered while managing these cases.Thorough knowledge of tooth morphology, skill, patience, use of appropriate instruments and materials are essential to successfully manage such cases. -
Veterinary Dentist at Work (VDAW) Index Chronological March 1996 Through Sept 2020
Veterinary Dentist At Work (VDAW) Index Chronological March 1996 through Sept 2020 1996 Vol. 13 No. 1 • Impacted mandibular canine tooth in a dog • Compound odontoma of a maxillary fourth premolar tooth in a dog 1996 Vol. 13 No. 2 • Incisor teeth radiographs in horses • Subcoronal tooth fracture in a dog 1996 Vol. 13 No. 3 • Orthodontic treatment – care is needed during removal of appliances • CT scans of canine molar teeth and salivary glands. 1996 Vol. 13 No. 4 • Endodontic treatment of a sun bear 1997 Vol. 14 No. 1 • Occupational hazard – dermatitis caused by benzoyl peroxide in chemical cured acrylics. 1997 Vol. 14 No. 2 • Interdental wiring and stainless steel arch repair of avulsed mandibular and maxillary incisors in a racehorse. Paul Orsini 1997 Vol. 14 No. 3 • Anomalous extra canine tooth in a cat. Frank Verstraete 1997 Vol. 14 No. 4 • Complex odontoma with necrosis in a dog. Frank Verstraete 1998 Vol. 15 No. 1 • Dental anomaly in a horse – supernumerary cheek tooth. Zack Matzkin • Cat with malpositioned canine tooth tongue trapping. Andries Van Foreest • Dog with lingual papillomatosis. Jeffrey Rhody 1 1998 Vol. 15 No. 2 • Yorkshire terrier with severe periodontal disease. William Rosenblad • Alveolar osteitis post extraction mandibular canine in a cat. Suzy Aller 1998 Vol. 15 No. 3 • Root abscess in an aardvark. 1998 Vol. 15 No. 4 • Fabrication of a veterinary dental teaching model. Ronald Southerland 1999 Vol. 16 No. 1 • Fabrication of a labial maxillary arch bar. Peak, Lobprise, Wiggs • Labial avulsion repair in a cat and a dog. Zlatko Pavlica • TMJ luxation in a rabbit. -
Pediatric Periodontal Disease: a Review of Cases
Pediatric Periodontal Disease: A Review of Cases Dental Acid Erosion: Identification and Management Martha Ann Keels, DDS PhD [email protected] or [email protected] www.dukesmiles.com California Society of Pediatric Dentistry Silverado Resort & Spa Napa, California April 23, 2016 Pediatric Periodontal Matrix Copyright Keels & Quinonez 2003 Healthy Diseased Bone Bone (no alveolar bone loss) (alveolar bone loss) Healthy Gingiva Box 1 Box 2 (pink, firm, stippled) Diseased Gingiva Box 3 Box 4 (erythematous, hemorrhagic) Box 1 – healthy gingiva and no bone loss Box 2 – healthy gingiva and bone loss Hypophosphatasia ** Inconclusive Pediatric Periodontal Disease (LJP) * Dentin Dysplasia Type I Post Avulsion / extraction Box 3 – unhealthy gingiva and no bone loss Gingivitis Eruption related gingivitis Mouthbreating Gingivitis Minimally attached gingival Gingival Fibromatotis Herpetic gingivostomatitis ANUG Thrombocytopenia Leukemia (AML / ALL) Aplastic anemia HIV Acrodynia Vitamin C deficiency Vitamin K deficiency Box 4 – unhealthy gingival and bone loss Neutrophil quantitative defect: (agranulocytosis, cyclic neutropenia, chronic idiopathic neutropenia)* Neutrophil qualitative defect: (Leukocyte adhesion deficiency)* Inconclusive pediatric periodontal disease (LJP) * Langerhan cell histiocytosis X *** Papillon-Lefevre disease * Diabetes mellitus * Down Syndrome * Chediak-Higashi disease * Chronic Granulomatous Disease * Tuberculosis * Ehlers-Danlos (Type VIII) * Osteomyelitis * * bacteriological culture and sensitivity needed ** tooth biopsy