Examination of Abdomen
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General Signs and Symptoms of Abdominal Diseases
General signs and symptoms of abdominal diseases Dr. Förhécz Zsolt Semmelweis University 3rd Department of Internal Medicine Faculty of Medicine, 3rd Year 2018/2019 1st Semester • For descriptive purposes, the abdomen is divided by imaginary lines crossing at the umbilicus, forming the right upper, right lower, left upper, and left lower quadrants. • Another system divides the abdomen into nine sections. Terms for three of them are commonly used: epigastric, umbilical, and hypogastric, or suprapubic Common or Concerning Symptoms • Indigestion or anorexia • Nausea, vomiting, or hematemesis • Abdominal pain • Dysphagia and/or odynophagia • Change in bowel function • Constipation or diarrhea • Jaundice “How is your appetite?” • Anorexia, nausea, vomiting in many gastrointestinal disorders; and – also in pregnancy, – diabetic ketoacidosis, – adrenal insufficiency, – hypercalcemia, – uremia, – liver disease, – emotional states, – adverse drug reactions – Induced but without nausea in anorexia/ bulimia. • Anorexia is a loss or lack of appetite. • Some patients may not actually vomit but raise esophageal or gastric contents in the absence of nausea or retching, called regurgitation. – in esophageal narrowing from stricture or cancer; also with incompetent gastroesophageal sphincter • Ask about any vomitus or regurgitated material and inspect it yourself if possible!!!! – What color is it? – What does the vomitus smell like? – How much has there been? – Ask specifically if it contains any blood and try to determine how much? • Fecal odor – in small bowel obstruction – or gastrocolic fistula • Gastric juice is clear or mucoid. Small amounts of yellowish or greenish bile are common and have no special significance. • Brownish or blackish vomitus with a “coffee- grounds” appearance suggests blood altered by gastric acid. -
USMLE and COMLEX II
USMLE and COMLEX II CE / CK Review General Surgery Northwestern Medical Review www.northwesternmedicalreview.com Lansing, Michigan 2014-2015 1. Northwestern Medical Review Acute Abdomen 4. What is the most common confirmatory physical 1. Your patient is a 45-year-old woman who is finding for peritonitis? presented with the complaint of severe abdominal ________________________________________ pain. You made the initial diagnosis of acute abdomen based on her history and examination. To determine the exact etiology, you want to 5. In a stable patient who is suspected of having perform a laparotomy on the patient. Before acute abdomen what is the next best course of ordering the procedure you re-evaluate the findings action? more thoroughly and come to the conclusion that you should NOT perform laparotomy on the A. Administering opiate analgesics patient. Which of the following clinical suspicions B. Laparotomy and/or findings was the CONTRAINDICATION to the laparotomy procedure on this patient? C. Serial abdominal exams D. Abdominal CT scan A. Suspicion of bacterial peritonitis E. Serial abdominal exams and CT scan B. Presence of acute right lower quadrant abdominal pain 6. What would you do if the above patient were to C. History indicating that the abdominal pain is become unstable? chronic D. Presence of a palpable abdominal mass ________________________________________ E. Alvarado score of 9 ________________________________________ 7. What are the top-tested causes of acute abdomen that do not require laparotomy? 2. What is acute abdomen? -
PERFORATED PEPTIC ULCER. Patient Usually Experiences
Postgrad Med J: first published as 10.1136/pgmj.12.134.470 on 1 December 1936. Downloaded from 470 POST-GRADUATE MEDICAL JOURNAL December, 1936 PERFORATED PEPTIC ULCER. By RONALD W. RAVEN, F.R.C.S. (Assistant Surgeon to T'he French Hospital, Assistant Surgeon to The Gordon Hospital for Rectal Diseases and Swrgical Registrar to The Royal Cancer Hospital.) INTRODUCTION. Peptic ulceration is a crippling disease judged from the stand-point of morbidity, and is also dangerous to life on account of serious complications, such as haemorrhage or perforation which may supervene during the course of the disease. These complications may occur in any patient and there are no criteria which will indicate whether or not an ulcer will bleed or perforate. When the treatment of peptic ulceration is under review it must be remembered that from 20 to 30 per cent. of these ulcers perforate. In a large series of cases I found that the incidence of perforation was 27 per cent. It is thus essential that patients suffering with peptic ulcer should be kept under continuous careful observation. Unfortunately, however, a small percentage of patients give no previous history of the peptic ulcer syndrome and perforation of the ulcer is the first indication of its presence. Recently, when considering the role of surgery in the treatment of chronic peptic ulcer, Joll stated that there has been a rise in the incidence of perforation as a complication of peptic ulcer since medical treatment has become systematized in the treatment of this disease. It must also be remembered that medical treat- Protected by copyright. -
Pathophysiology, Diagnosis, and Management of Pediatric Ascites
INVITED REVIEW Pathophysiology, Diagnosis, and Management of Pediatric Ascites ÃMatthew J. Giefer, ÃKaren F. Murray, and yRichard B. Colletti ABSTRACT pressure of mesenteric capillaries is normally about 20 mmHg. The pediatric population has a number of unique considerations related to Intestinal lymph drains from regional lymphatics and ultimately the diagnosis and treatment of ascites. This review summarizes the physio- combines with hepatic lymph in the thoracic duct. Unlike the logic mechanisms for cirrhotic and noncirrhotic ascites and provides a sinusoidal endothelium, the mesenteric capillary membrane is comprehensive list of reported etiologies stratified by the patient’s age. relatively impermeable to albumin; the concentration of protein Characteristic findings on physical examination, diagnostic imaging, and in mesenteric lymph is only about one-fifth that of plasma, so there abdominal paracentesis are also reviewed, with particular attention to those is a significant osmotic gradient that promotes the return of inter- aspects that are unique to children. Medical and surgical treatments of stitial fluid into the capillary. In the normal adult, the flow of lymph ascites are discussed. Both prompt diagnosis and appropriate management of in the thoracic duct is about 800 to 1000 mL/day (3,4). ascites are required to avoid associated morbidity and mortality. Ascites from portal hypertension occurs when hydrostatic Key Words: diagnosis, etiology, management, pathophysiology, pediatric and osmotic pressures within hepatic and mesenteric capillaries ascites produce a net transfer of fluid from blood vessels to lymphatic vessels at a rate that exceeds the drainage capacity of the lym- (JPGN 2011;52: 503–513) phatics. It is not known whether ascitic fluid is formed predomi- nantly in the liver or in the mesentery. -
Tutorial 9 the Abdomen.Pdf
117 TUTORIAL 9 THE EXAMINATION OF ABDOMEN OVERALL OBJECTIVE At the end of this module, the student should be able to: 1. Do a full clinical examination of abdomen and able to make a reasonable provisional and differential diagnosis of vomiting, gastroenteritis, failure to thrive, hepatomegaly, splenomegaly and jaundice in infants and children. 2. To list the complications and principles of management of chronic diarrhoea, malnutrition, chronic liver disease and portal hypertension. EXTRA GENERAL FEATURES INCLUDE Signs of Chronic Liver Disease Signs of Chronic Liver Failure Jaundice Foetor hepaticus, scratch marks, pruritus Ascites Malabsorption of vitamin D: Rickets Spider telangiectasia Malabsorption of Vit K: Bleeding/bruising/ Spider navi haemorrhage/ epitasis/ purpura Palmer erythema Splenomegaly - Portal hypertension Finger clubbing Hepatic encephalopathy Pigmentation Vit A deficiency: Xerophthalmia, corneal Itching xerosis, follicular hyperkeratosis Spider telangiectasia It is a branched group of dilated capillary blood vessels forming a spiderlike image on the skin. It is compressible and blanchable with adequate pressure (glass slide). Generally, as the pressure is released the central feeding vessel can be seen and which often pulsates. Spider navi These are spider shaped small reddish marks formed by the dilatation of central arterioles from which small vessels radiate. They are found on the chest above the nipples, face forearm, and sometimes dorsum of the hands. These are present in case of liver cirrhosis. Complications of cirrhosis (HEPATIC) Hepatic encephalopathy: hepatorenal syndrome, hepatopulmonary syndrome Esophageal varices Portal hypertension Protein energy malnutrition(PEM) Ascites Thrombosis of portal vein Infection/peritonitis Coagulopathy Tutorials on Clinical Methods in Paediatrics. Dr M R Ghuman et al. -
Abdominal Examination
Abdominal Examination Introduction Wash hands, Introduce self, ask Patients name & DOB & what they like to be called, Explain examination and get consent Expose and lie patient flat General Inspection Patient: stable, pain/discomfort, jaundice, pallor, muscle wasting/cachexia Around bed: vomit bowels etc Hands Flapping tremor (hepatic encephalopathy) Nails: clubbing (cirrhosis, IBD, coeliacs), leukonychia (hypoalbuminemia in liver cirrhosis), koilonychia (iron deficiency anaemia) Palms: palmar erythema (hyperdynamic circulation due to ↑oestrogen levels in liver disease/ pregnancy), Dupuytren’s contracture (familial, liver disease), fingertip capillary glucose monitoring marks (diabetes) Head Eyes: sclera for jaundice (liver disease), conjunctival pallor (anaemia e.g. bleeding, malabsorption), periorbital xanthelasma (hyperlipidaemia in cholestasis) Mouth: glossitis/stomatitis (iron/ B12 deficiency anaemia), aphthous ulcers (IBD), breath odor (e.g. faeculent in obstruction; ketotic in ketoacidosis; alcohol) Neck and torso Ask patient to sit forwards: Neck: feel for lymphadenopathy from behind – especially Virchow's node (gastric malignancy) Back inspection: spider naevi (>5 significant), skin lesions (immunosuppression) Ask patient to relax back: Chest inspection: spider naevi (>5 significant), gynaecomastia, loss of axillary hair (all due to ↑oestrogen levels in liver disease/ pregnancy) Abdomen Inspection: distension (Fluid, Flatus, Fat, Foetus, Faeces), incisional hernias (ask patient to cough), scars, striae (pregnancy, -
A Rare Case of Ascites
Clinical Medicine 2019 Vol 19, No 2: s73 CLINICAL A r a r e c a s e o f a s c i t e s Authors: S h u a n n S h w a n a , A d n a n U r R a h m a n a n d E l i z a b e t h S l o w i n s k a A i m s most cases depends on whether it is idiopathic or secondary. The mainstay of treatment is corticosteroids; if there is no response, A 69-year-old gentleman presented with a 5-week history of immunosuppressive therapy can be used. Case-series data exist, abdominal distension. He had a past history of diabetes and which show that high-dose corticosteroids like prednisolone are myocardial infarction, and was an ex-smoker with no significant effective in reducing the chronic inflammatory response caused by history of alcohol intake. Examination demonstrated a distended, retroperitoneal fibrosis; however, there is a high rate of recurrence non-tender abdomen with shifting dullness, no organomegaly and once the steroids are withdrawn. Mycophenolate mofetil in addition no signs of chronic liver disease. to corticosteroids has been shown to reduce duration of steroid use without affecting the efficacy and reduces disease recurrence. ■ M e t h o d s Investigations included an ultrasound scan of the abdomen, an Conflict of interest statement ascitic tap, a computed tomography (CT) of the abdomen/pelvis N o n e d e c l a r e d . -
Ilbpnp55ecxs1d55pt2h4cnta 12-Year-Old Girl with Chronic Vomiting
A 12-year-old girl with chronic vomiting and epigastric pain History: A 12-year-old girl has presented with vomiting for 1 month. The frequency is about 2-3 times a day. The vomitus contains digested food without bile stain. The emesis is not related to meals. After vomiting, she can normally eat. Her bowel movement is once daily with normal stools. There is no fever. After 2 days of symptoms, she was treated with antibiotics and antiemesis with a diagnosis of gastroenteritis. However, the symptom persists. Three weeks prior to admission, the vomiting became worsening (7-8 times a day) and occurred a half an hour after meals. Its characteristics remained the same. At this time, she also complained of epigastric pain, weakness, and anorexia. She was admitted to a primary care hospital and treated with intravenous fluid. The symptoms seemed to be improved. However, the vomiting resumed with a history of foul-smell watery diarrhea 2 time a day for 1 weeks before admission to Children's hospital. She lost significant weight during the illness. Past history: She was admitted because of Dengue hemorrhagic fever 2 years ago. Perinatal and immunization history are normal. There is no genetic and atopic diseases in the family. The patient refuses a history of contact tuberculosis. She denies regular uncooked food ingestion. Physical examination: General appearance- Thai girl, obesity, alert Body weight 80 kg, height 158 cm Vital signs: T 37 C, PR 80/min, RR 18/min, BP 110/70 mmHg HEENT: not pale, no jaundice, pharynx and tonsils-not injected, normal TM Heart: regular rhythm, no murmur Lungs: normal breath sound, no adventitious sound Abdomen: soft, mild distension, mild tenderness at epigastrium, no guarding, no rigidity, active bowel sound, no abnormal mass, fluid thrill and shifting dullness can not be evaluated. -
The Differential Diagnosis of Abdominal Masses Prof
1 The differential diagnosis of Abdominal masses Prof. Dr. Mohamed I. Kassem ILOs for DD of abdominal masses: To know the different divisions and compartments of the abdomen. To be oriented with the anatomical locations of the different abdominal organs. Describe the differential diagnosis of parietal swellings. To know the differential diagnosis of the intra-abdominal swellings in each compartment with their management. 2 Abdominal Examination Exposure Nipple to knee, If embarrassing cover the lower abdomen with sheet. Inspection Abdominal contour ⚫ Normal --- flat from xiphoid to pubis, ⚫ umbilicus is at the center of the abdomen. Abdominal contour ⚫ Generalized distension ⚫ fat, ⚫ fetus, ⚫ feces, ⚫ flatus, ⚫ fluid, ⚫ full-sized tumors. ⚫ Localized bulge ⚫ Mass, organomegaly, hernia. Palpation Superficial • Gain patient’s confidence • Temperature • Parietal mass • Tenderness • Hyperthesia 3 Deep • Liver • Spleen • Kidney • Abdominal Aorta • Masses If masses are felt, note: • Site, • size, • Shape • Surface • skin overlying • special character: • consistency • tenderness • pulsations • mobility with respiration or with hand. 4 Parietal versus intra-abdominal mass Rising up test Imaging 1. PARIETAL SWELLINGS • Extends over the costal margin 5 • Moves anterior and posterior with respiration • More prominent on rising up test 2. INTRA-ABDOMINAL SWELLINGS • Disappear beneath costal margin • Moves up and down with respiration • Less prominent on rising up test PARIETAL SWELLINGS (common for different quadrants) Skin: • Sebaceous cyst • Papilloma • Melanoma, SCC Subcutaneous tissue: • Lipoma: SC, intermuscular • Neurofibroma • Hamangioma, lymphangioma Muscles: • Rectus sheath haematoma • Desmoid tumour 6 ✓ Mid-clavicular lines are the vertical planes 7 In a patient presenting with mass abdomen, generally following clinical features should be assessed care fully. _ Pain: Site, nature, aggravating or relieving factors, duration of pain, referred pain. -
ORIGINAL RESEARCH PAPER Dr. Priya Verma Dr. Bhupendra Narain*
PARIPEX - INDIAN JOURNAL OF RESEARCH Volume-8 | Issue-2 | February-2019 | PRINT ISSN - 2250-1991 ORIGINAL RESEARCH PAPER Paediatrics STUDY OF THE COMPARATIVE UTILITY OF THE SERUM ASCITES ALBUMIN GRADIENT (SAAG) AND KEY WORDS: Ascites , Portal THE ASCITIC FLUID TOTAL PROTEIN IN THE Hypertension , SAAG , AFTP DIFFERENTIAL DIAGNOSIS OF ASCITES Dr. Priya Verma MBBS,MD Dept.Of Pediatrics , Patna Medical College ,Patna Dr. Bhupendra Asst. Prof. Dept. Of Pediatrics, Patna Medical College ,Patna*Corresponding Narain* Author Background The ascitic fluid total protein , cell counts , lactate dehydrogenase activity (LDH ), ascites - to - serum protein and LDH ratios have traditionally been used to classify ascites . However , none of these parameters have been found to be completely discriminating . Objective In this study we compared the utility of the serum ascitic fluid albumin gradient and ascitic fluid total protein levels in establishing the differential diagnosis of ascites . Methodology - 60 children between the ages of 1- 15 years were included in the study .Paired Serum and Ascitic fluid samples were taken simultaneously . Results The SAAG had greater Positive and Negative Predictive values as compared to that of AFTP . The 'p ' value ABSTRACT for SAAG ( > 1.1gm % ) in detecting portal hypertension was < 0.001 which was highly significant as against that of AFTP , 'p' value < 0.02. Conclusion Serum Ascitic fluid Albumin gradient (SAAG ) is a better indicator of portal hypertension than the Ascitic fluid Total Protein (AFTP). INTRODUCTION reflecting the oncotic pressure gradient between the Ascites denotes the pathological accumulation of fluid in vascular bed and the interstitial or ascitic fluid ; the the peritoneal cavity . It is derived from a Greek word two values being measured simultaneously 3 . -
Examination of the Abdomen
06/11/1431 Examination of the Abdomen Chapter 10 Ra'eda Almashaqba 1 Review Anatomy Rectus Abdominis Xiphoid Process Costal Margin Linea Alba Anterior Superior Iliac Spine Symphysis Pubis Ra'eda Almashaqba 2 Inguinal Ligament 1 06/11/1431 Ra'eda Almashaqba 3 9 Regions Ra'eda Almashaqba 4 2 06/11/1431 Ra'eda Almashaqba 5 Ra'eda Almashaqba 6 3 06/11/1431 Location! Location! Location! RUQ liver gallbladder duodenum (small intestine) pancreas head right kidney and adrenal Ra'eda Almashaqba 7 Location! Location! Location! RLQ cecum appendix right ovary and tube Ra'eda Almashaqba 8 4 06/11/1431 Location! Location! Location! LLQ sigmoid colon left ovary and tube LUQ stomach spleen pancreas left kidney and adrenal Ra'eda Almashaqba 9 GI Variations Due to Age Aging- should not affect GI function unless associated with a disease process Decreased: salivation, sense of taste, gastric acid secretion, esophageal emptying, liver size, bacterial flora Increased: constipation! Ra'eda Almashaqba 10 5 06/11/1431 Health History Gastrointestinal Disorder Indigestion, N&V, Anorexia, Hematemesis - Ask the pt how is your appetite Indigestion ----- distress associated with eating Heartburn ---- sense of burning or warmth that is retrosternal and may radiate to the neck Excessive gas: frequent belching, distention or flatulence ,Abd fullness. Dysphagia & odynophagia Change in bowel function Constipation or diarrhea Jaundice Ra'eda Almashaqba 11 Abdominal pain: Visceral : Occur in all the abd, burning, aching, difficult to localize, varies in quality -
Evaluation and Management of Acute Abdominal Pain in the Emergency Department
International Journal of General Medicine Dovepress open access to scientific and medical research Open Access Full Text Article REVIEW Evaluation and management of acute abdominal pain in the emergency department Christopher R Macaluso Abstract: Evaluation of the emergency department patient with acute abdominal pain is Robert M McNamara sometimes difficult. Various factors can obscure the presentation, delaying or preventing the correct diagnosis, with subsequent adverse patient outcomes. Clinicians must consider multiple Department of Emergency Medicine, Temple University School of Medicine, diagnoses, especially those life-threatening conditions that require timely intervention to limit Philadelphia, PA, USA morbidity and mortality. This article will review general information on abdominal pain and discuss the clinical approach by review of the history and the physical examination. Additionally, this article will discuss the approach to unstable patients with abdominal pain. Keywords: acute abdomen, emergency medicine, peritonitis For personal use only. Introduction Abdominal pain is the most common reason for a visit to the emergency department (ED), accounting for 8 million (7%) of the 119 million ED visits in 2006.1 Obviously, anyone practicing emergency medicine (EM) must be skilled in the assessment of abdominal pain. Although a common presentation, abdominal pain must be approached in a serious manner, as it is often a symptom of serious disease and misdiagnosis may occur. Abdominal pain is the presenting issue in a high percentage