Breathing Air Brochure
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Hypothermia Brochure
Visit these websites for more water safety and hypothermia prevention in- formation. What is East Pierce Fire & Rescue Hypothermia? www.eastpiercefire.org Hypothermia means “low temperature”. Washington State Drowning When your body is exposed to cold tem- Prevention Coalition Hypothermia www.drowning-prevention.org perature, it tries to protect itself by keeping a normal body temperature of 98.6°F. It Children’s Hospital & tries to reduce heat loss by shivering and Regional Medical Center In Our Lakes moving blood from your arms and legs to www.seattlechildrens.org the core of your body—head, chest and and Rivers abdomen. Hypothermia Prevention, Recognition and Treatment www.hypothermia.org Stages of Hypothermia Boat Washington Mild Hypothermia www.boatwashington.org (Core body temperature of 98.6°— 93.2°F) Symptoms: Shivering; altered judg- ment; numbness; clumsiness; loss of Boat U.S. Foundation dexterity; pain from cold; and fast www.boatus.com breathing. Boat Safe Moderate Hypothermia www.boatsafe.com (Core body temperature of 93.2°—86°F) Symptoms: Semiconscious to uncon- scious; shivering reduced or absent; lips are blue; slurred speech; rigid n in muscles; appears drunk; slow Eve breathing; and feeling of warmth can occur. mer! Headquarters Station Sum Severe Hypothermia 18421 Old Buckley Hwy (Core body temperature below 86°F) Bonney Lake, WA 98391 Symptoms: Coma; heart stops; and clinical death. Phone: 253-863-1800 Fax: 253-863-1848 Email: [email protected] Know the water. Know your limits. Wear a life vest. By choosing to swim in colder water you Waters in Western Common Misconceptions Washington reduce your survival time. -
Is Colonic Propionate Delivery a Novel Solution to Improve Metabolism and Inflammation in Overweight Or Obese Subjects?
Commentary in IgG levels in IPE-treated subjects versus Is colonic propionate delivery a novel those receiving cellulose supplementa- tion. This interesting discovery is the Gut: first published as 10.1136/gutjnl-2019-318776 on 26 April 2019. Downloaded from solution to improve metabolism and first evidence in humans that promoting the delivery of propionate in the colon inflammation in overweight or may affect adaptive immunity. It is worth noting that previous preclinical and clin- obese subjects? ical data have shown that supplementation with inulin-type fructans was associated 1,2 with a lower inflammatory tone and a Patrice D Cani reinforcement of the gut barrier.7 8 Never- theless, it remains unknown if these effects Increased intake of dietary fibre has been was the lack of evidence that the observed are directly linked with the production of linked to beneficial impacts on health for effects were due to the presence of inulin propionate, changes in the proportion of decades. Strikingly, the exact mechanisms itself on IPE or the delivery of propionate the overall levels of SCFAs, or the pres- of action are not yet fully understood. into the colon. ence of any other bacterial metabolites. Among the different families of fibres, In GUT, Chambers and colleagues Alongside the changes in the levels prebiotics have gained attention mainly addressed this gap of knowledge and of SCFAs, plasma metabolome analysis because of their capacity to selectively expanded on their previous findings.6 For revealed that each of the supplementa- modulate the gut microbiota composition 42 days, they investigated the impact of tion periods was correlated with different 1 and promote health benefits. -
The Lower Critical Solution Temperature (LCST) Transition
Copyright by David Samuel Simmons 2009 The Dissertation Committee for David Samuel Simmons certifies that this is the approved version of the following dissertation: Phase and Conformational Behavior of LCST-Driven Stimuli Responsive Polymers Committee: ______________________________ Isaac Sanchez, Supervisor ______________________________ Nicholas Peppas ______________________________ Krishnendu Roy ______________________________ Venkat Ganesan ______________________________ Thomas Truskett Phase and Conformational Behavior of LCST-Driven Stimuli Responsive Polymers by David Samuel Simmons, B.S. Dissertation Presented to the Faculty of the Graduate School of The University of Texas at Austin in Partial Fulfillment of the Requirements for the Degree of Doctor of Philosophy The University of Texas at Austin December, 2009 To my grandfather, who made me an engineer before I knew the word and to my wife, Carey, for being my partner on my good days and bad. Acknowledgements I am extraordinarily fortunate in the support I have received on the path to this accomplishment. My adviser, Dr. Isaac Sanchez, has made this publication possible with his advice, support, and willingness to field my ideas at random times in the afternoon; he has my deep appreciation for his outstanding guidance. My thanks also go to the members of my Ph.D. committee for their valuable feedback in improving my research and exploring new directions. I am likewise grateful to the other members of Dr. Sanchez’ research group – Xiaoyan Wang, Yingying Jiang, Xiaochu Wang, and Frank Willmore – who have shared their ideas and provided valuable sounding boards for my mine. I would particularly like to express appreciation for Frank’s donation of his own post-graduation time in assisting my research. -
The Hazards of Nitrogen Asphyxiation US Chemical Safety and Hazard Investigation Board
The Hazards of Nitrogen Asphyxiation US Chemical Safety and Hazard Investigation Board Introduction • Nitrogen makes up 78% of the air we breath; because of this it is often assumed that nitrogen is not hazardous. • However, nitrogen is safe to breath only if it is mixed with an appropriate amount of oxygen. • Additional nitrogen (lower oxygen) cannot be detected by the sense of smell. Introduction • Nitrogen is used commercially as an inerting agent to keep material free of contaminants (including oxygen) that may corrode equipment, present a fire hazard, or be toxic. • A lower oxygen concentration (e.g., caused by an increased amount of nitrogen) can have a range of effects on the human body and can be fatal if if falls below 10% Effects of Oxygen Deficiency on the Human Body Atmospheric Oxygen Concentration (%) Possible Results 20.9 Normal 19.0 Some unnoticeable adverse physiological effects 16.0 Increased pulse and breathing rate, impaired thinking and attention, reduced coordination 14.0 Abnormal fatigue upon exertion, emotional upset, faulty coordination, poor judgment 12.5 Very poor judgment and coordination, impaired respiration that may cause permanent heart damage, nausea, and vomiting <10 Inability to move, loss of consciousness, convulsions, death Source: Compressed Gas Association, 2001 Statistics on Incidents CSB reviewed cases of nitrogen asphyxiation that occurred in the US between 1992 and 2002 and determined the following: • 85 incidents of nitrogen asphyxiation resulted in 80 deaths and 50 injuries. • The majority of -
Respiratory Physiology - Part B Experimental Determination of Anatomical Dead Space Value (Human 9 - Version Sept
Comp. Vert. Physiology- BI 244 Respiratory Physiology - Part B Experimental Determination of Anatomical Dead Space Value (Human 9 - Version Sept. 10, 2013) [This version has been modified to also serve as a tutorial in how to run HUMAN's artificial organs] Part of the respiratory physiology computer simulation work for this week allows you to obtain a hand-on feeling for the effects of anatomical dead space on alveolar ventilation. The functional importance of dead space can explored via employing HUMAN's artificial respirator to vary the respiration rate and tidal volume. DEAD SPACE DETERMINATION Introduction The lack of unidirectional respiratory medium flow in non-avian air ventilators creates the existence of an anatomical dead space. The anatomical dead space of an air ventilator is a fixed volume not normally under physiological control. However, the relative importance of dead space is adjustable by appropriate respiratory maneuvers. For example, recall the use by panting animals of their dead space to reduce a potentially harmful respiratory alkalosis while hyperventilating. In general, for any given level of lung ventilation, the fraction of the tidal volume attributed to dead space will affect the resulting level of alveolar ventilation, and therefore the efficiency (in terms of gas exchange) of that ventilation. [You should, of course, refresh your knowledge of total lung ventilation, tidal volume alveolar ventilation.] Your objective here is to observe the effects of a constant "unknown" dead space on resulting alveolar ventilation by respiring the model at a variety of tidal volume-frequency combinations. You are then asked to calculate the functional dead space based on the data you collect. -
Clinical Management of Severe Acute Respiratory Infections When Novel Coronavirus Is Suspected: What to Do and What Not to Do
INTERIM GUIDANCE DOCUMENT Clinical management of severe acute respiratory infections when novel coronavirus is suspected: What to do and what not to do Introduction 2 Section 1. Early recognition and management 3 Section 2. Management of severe respiratory distress, hypoxemia and ARDS 6 Section 3. Management of septic shock 8 Section 4. Prevention of complications 9 References 10 Acknowledgements 12 Introduction The emergence of novel coronavirus in 2012 (see http://www.who.int/csr/disease/coronavirus_infections/en/index. html for the latest updates) has presented challenges for clinical management. Pneumonia has been the most common clinical presentation; five patients developed Acute Respira- tory Distress Syndrome (ARDS). Renal failure, pericarditis and disseminated intravascular coagulation (DIC) have also occurred. Our knowledge of the clinical features of coronavirus infection is limited and no virus-specific preven- tion or treatment (e.g. vaccine or antiviral drugs) is available. Thus, this interim guidance document aims to help clinicians with supportive management of patients who have acute respiratory failure and septic shock as a consequence of severe infection. Because other complications have been seen (renal failure, pericarditis, DIC, as above) clinicians should monitor for the development of these and other complications of severe infection and treat them according to local management guidelines. As all confirmed cases reported to date have occurred in adults, this document focuses on the care of adolescents and adults. Paediatric considerations will be added later. This document will be updated as more information becomes available and after the revised Surviving Sepsis Campaign Guidelines are published later this year (1). This document is for clinicians taking care of critically ill patients with severe acute respiratory infec- tion (SARI). -
Exercise and Cellular Respiration Lab
California State University of Bakersfield, Department of Chemistry Exercise and Cellular Respiration Lab Standards: MS-LS1-7 Develop a model to describe how food is rearranged through chemical reactions forming new molecules that support growth and/or release energy as this matter moves through an organism. Introduction: I. Background Information. Cellular respiration (see chemical reaction below) is a chemical reaction that occurs in your cells to create energy; when you are exercising your muscle cells are creating ATP to contract. Cellular respiration requires oxygen (which is breathed in) and creates carbon dioxide (which is breathed out). This lab will address how exercise (increased muscle activity) affects the rate of cellular respiration. You will measure 3 different indicators of cellular respiration: breathing rate, heart rate, and carbon dioxide production. You will measure these indicators at rest (with no exercise) and after 1 and 2 minutes of exercise. Breathing rate is measured in breaths per minute, heart rate in beats per minute, and carbon dioxide in the time it takes bromothymol blue to change color. Carbon dioxide production can be measured by breathing through a straw into a solution of bromothymol blue (BTB). BTB is an acid indicator; when it reacts with acid it turns from blue to yellow. When carbon dioxide reacts with water, a weak acid (carbonic acid) is formed (see chemical reaction below). The more carbon dioxide you breathe into the BTB solution, the faster it will change color to yellow. The purpose of this lab activity is to analyze the effect of exercise on cellular respiration. Background: I. -
QUESTIONS ABOUT YOUR BREATHING Name:______Please Answer the Questions Below for ONLY the PATIENT Seeing the Doctor Today, Date of Birth:______You OR Your Child
QUESTIONS ABOUT YOUR BREATHING Name:_____________________________________ Please answer the questions below for ONLY THE PATIENT seeing the doctor today, Date of Birth:_______________________________ you OR your child. Today’s Date:_______________________________ 1. Have you/has your child had shortness of breath, 9. At what age did you/did your child start having coughing, wheezing (whistling in the chest) during the day? breathing trouble?_____________ r Yes r No 10. Do any blood relatives (parent, brother, sister, child) have: 2. Have you/has your child had breathing trouble at night r Asthma r Allergies or early in the morning r Yes r No 11. Do you or anyone in the family smoke? r Yes r No 3. Has breathing trouble kept you/kept your child from school/ work/normal activities? r Yes r No 12. Are you/is your child ever in smoky places? r Yes r No 4. Have you/has your child ever been to a doctor, urgent care, 13. Check any of the things that make your/your child’s emergency room or a hospital for breathing trouble? r Yes r No breathing worse, or tell us about others. 5. Do you/does your child get colds that settle in the chest, r Breathing in chemicals, dusts, fumes at work r Colds or flu r Strong odors, like cleaners or perfumes or coughing that lasts 10 days or more after a cold is gone? r Animals r Weather r Yes r No r Dust r Exercise 6. Have you/has your child ever needed steroid pills or syrup r Pollen and mold r Cigarette and other smoke (prednisone, prednisolone, prelone) for breathing trouble? r Medicines:___________________________________________ r Yes r No _______________________________________________________ If yes, how many times has this happened? ___________________ r Other things: 7. -
Brownie's THIRD LUNG
BrMARINEownie GROUP’s Owner’s Manual Variable Speed Hand Carry Hookah Diving System ADVENTURE IS ALWAYS ON THE LINE! VSHCDC Systems This manual is also available online 3001 NW 25th Avenue, Pompano Beach, FL 33069 USA Ph +1.954.462.5570 Fx +1.954.462.6115 www.BrowniesMarineGroup.com CONGRATULATIONS ON YOUR PURCHASE OF A BROWNIE’S SYSTEM You now have in your possession the finest, most reliable, surface supplied breathing air system available. The operation is designed with your safety and convenience in mind, and by carefully reading this brief manual you can be assured of many hours of trouble-free enjoyment. READ ALL SAFETY RULES AND OPERATING INSTRUCTIONS CONTAINED IN THIS MANUAL AND FOLLOW THEM WITH EACH USE OF THIS PRODUCT. MANUAL SAFETY NOTICES Important instructions concerning the endangerment of personnel, technical safety or operator safety will be specially emphasized in this manual by placing the information in the following types of safety notices. DANGER DANGER INDICATES AN IMMINENTLY HAZARDOUS SITUATION WHICH, IF NOT AVOIDED, WILL RESULT IN DEATH OR SERIOUS INJURY. THIS IS LIMITED TO THE MOST EXTREME SITUATIONS. WARNING WARNING INDICATES A POTENTIALLY HAZARDOUS SITUATION WHICH, IF NOT AVOIDED, COULD RESULT IN DEATH OR INJURY. CAUTION CAUTION INDICATES A POTENTIALLY HAZARDOUS SITUATION WHICH, IF NOT AVOIDED, MAY RESULT IN MINOR OR MODERATE INJURY. IT MAY ALSO BE USED TO ALERT AGAINST UNSAFE PRACTICES. NOTE NOTE ADVISE OF TECHNICAL REQUIREMENTS THAT REQUIRE PARTICULAR ATTENTION BY THE OPERATOR OR THE MAINTENANCE TECHNICIAN FOR PROPER MAINTENANCE AND UTILIZATION OF THE EQUIPMENT. REGISTER YOUR PRODUCT ONLINE Go to www.BrowniesMarineGroup.com to register your product. -
What Are the Health Effects from Exposure to Carbon Monoxide?
CO Lesson 2 CARBON MONOXIDE: LESSON TWO What are the Health Effects from Exposure to Carbon Monoxide? LESSON SUMMARY Carbon monoxide (CO) is an odorless, tasteless, colorless and nonirritating Grade Level: 9 – 12 gas that is impossible to detect by an exposed person. CO is produced by the Subject(s) Addressed: incomplete combustion of carbon-based fuels, including gas, wood, oil and Science, Biology coal. Exposure to CO is the leading cause of fatal poisonings in the United Class Time: 1 Period States and many other countries. When inhaled, CO is readily absorbed from the lungs into the bloodstream, where it binds tightly to hemoglobin in the Inquiry Category: Guided place of oxygen. CORE UNDERSTANDING/OBJECTIVES By the end of this lesson, students will have a basic understanding of the physiological mechanisms underlying CO toxicity. For specific learning and standards addressed, please see pages 30 and 31. MATERIALS INCORPORATION OF TECHNOLOGY Computer and/or projector with video capabilities INDIAN EDUCATION FOR ALL Fires utilizing carbon-based fuels, such as wood, produce carbon monoxide as a dangerous byproduct when the combustion is incomplete. Fire was important for the survival of early Native American tribes. The traditional teepees were well designed with sophisticated airflow patterns, enabling fires to be contained within the shelter while minimizing carbon monoxide exposure. However, fire was used for purposes other than just heat and cooking. According to the historian Henry Lewis, Native Americans used fire to aid in hunting, crop management, insect collection, warfare and many other activities. Today, fire is used to heat rocks used in sweat lodges. -
Affinity of Small-Molecule Solutes to Hydrophobic, Hydrophilic, and Chemically Patterned Interfaces in Aqueous Solution
Affinity of small-molecule solutes to hydrophobic, hydrophilic, and chemically patterned interfaces in aqueous solution Jacob I. Monroea, Sally Jiaoa, R. Justin Davisb, Dennis Robinson Browna, Lynn E. Katzb, and M. Scott Shella,1 aDepartment of Chemical Engineering, University of California, Santa Barbara, CA 93106; and bDepartment of Civil, Architectural and Environmental Engineering, University of Texas at Austin, Austin, TX 78712 Edited by Peter J. Rossky, Rice University, Houston, TX, and approved November 17, 2020 (received for review September 30, 2020) Performance of membranes for water purification is highly influ- However, a molecular understanding that links membrane enced by the interactions of solvated species with membrane surface chemistry to solute affinity and hence membrane func- surfaces, including surface adsorption of solutes upon fouling. tional properties remains incomplete, due in part to the complex Current efforts toward fouling-resistant membranes often pursue interplay among specific interactions (e.g., hydrogen bonds, surface hydrophilization, frequently motivated by macroscopic electrostatics, dispersion) and molecular morphology (e.g., sur- measures of hydrophilicity, because hydrophobicity is thought to face and polymer configurations) that are difficult to disentangle increase solute–surface affinity. While this heuristic has driven di- (11–14). Chemically heterogeneous surfaces are even less un- verse membrane functionalization strategies, here we build on derstood but can affect fouling in complex ways. -
Breathing Air Quality, Sampling and Testing
Breathing Air Quality, Sampling and Testing Environmental Health Laboratory Department of Environmental and Occupational Health Sciences School of Public Health University of Washington Funding and support from The State of Washington Department of Labor & Industries Safety & Health Investment Projects Medical Aid and Accident Fund Breathing Air Quality, Sampling, and Testing Environmental Health Laboratory Department of Environmental and Occupational Health Sciences School of Public Health University of Washington Funding and support from The State of Washington Department of Labor & Industries Safety & Health Investment Projects Medical Aid and Accident Funds 1 University of Washington Environmental Health Laboratory Table of Contents Overview ...............................................................................1 Background ...........................................................................2 Regulated Components of Breathing Air ...............................7 Performance of Breathing Air Testing Kits ............................9 Laboratory Accreditation .....................................................18 Guidance Summary .............................................................19 Glossary ..............................................................................20 References ...........................................................................22 List of Tables Table 1. Breathing Air Quality Specifications ........................2 Table 2. Description of Kits Tested ........................................9