Clinical Cardiology Update 2010
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Essentials of Bedside Cardiology CONTEMPORARY CARDIOLOGY
Essentials of Bedside Cardiology CONTEMPORARY CARDIOLOGY CHRISTOPHER P. CANNON, MD SERIES EDITOR Aging, Heart Disease and Its Management: Facts and Controversies, edited by Niloo M. Edwards, MD, Mathew S. Maurer, MD, and Rachel B. Wellner, MD, 2003 Peripheral Arterial Disease: Diagnosis and Treatment, edited by Jay D. Coffman, MD, and Robert T. Eberhardt, MD, 2003 Essentials ofBedside Cardiology: With a Complete Course in Heart Sounds and Munnurs on CD, Second Edition, by Jules Constant, MD, 2003 Primary Angioplasty in Acute Myocardial Infarction, edited by James E. Tcheng, MD,2002 Cardiogenic Shock: Diagnosis and Treatment, edited by David Hasdai, MD, Peter B. Berger, MD, Alexander Battler, MD, and David R. Holmes, Jr., MD, 2002 Management of Cardiac Arrhythmias, edited by Leonard I. Ganz, MD, 2002 Diabetes and Cardiovascular Disease, edited by Michael T. Johnstone and Aristidis Veves, MD, DSC, 2001 Blood Pressure Monitoring in Cardiovascular Medicine and Therapeutics, edited by William B. White, MD, 2001 Vascular Disease and Injury: Preclinical Research, edited by Daniell. Simon, MD, and Campbell Rogers, MD 2001 Preventive Cardiology: Strategies for the Prevention and Treatment of Coronary Artery Disease, edited by JoAnne Micale Foody, MD, 2001 Nitric Oxide and the Cardiovascular System, edited by Joseph Loscalzo, MD, phD and Joseph A. Vita, MD, 2000 Annotated Atlas of Electrocardiography: A Guide to Confident Interpretation, by Thomas M. Blake, MD, 1999 Platelet Glycoprotein lIb/IlIa Inhibitors in Cardiovascular Disease, edited by A. Michael Lincoff, MD, and Eric J. Topol, MD, 1999 Minimally Invasive Cardiac Surgery, edited by Mehmet C. Oz, MD and Daniel J. Goldstein, MD, 1999 Management ofAcute Coronary Syndromes, edited by Christopher P. -
Pdf Maximiliano Rubín and the Context of Galdos's Medical Knowledge
97 MAXIMILIANO RUBÍN AND THE CONTEXT OF GALDÓS’S MEDICAL KNOWLEDGE Michael W. Stannard The Medical Context Galdós’s interest in doctors, medicine and abnormal mental states is well known and has been the subject of many studies.1 More than 50 doctors populate the pages of his fiction prompting Granjel to refer to a “colegio médico galdosiano” (167). Almost invariably these physicians are portrayed in a favorable light (García Lisbona, 105, note 3), epitomized particularly in the combination of scientific outlook and humane concern of Galdosian characters such as Augusto Miquis, Teodoro Golfín and Moreno Rubio. References to medications abound in the novels 2 while the medical sciences appear in a significant sample of Galdós’s journalistic articles more often than any other science.3 It is somewhat surprising, therefore, that Galdós’s knowledge of the medical sciences of his time should remain incompletely explored. It is the purpose of this paper to draw attention to the depth of Galdós’s understanding of the medical advances of his day, which still remains under-appreciated. Galdós wrote at a time of revolutionary changes in medicine. Gradually replacing the older vitalistic and humoral conceptions of disease, positivist medicine emanating especially from France identified anatomical abnormalities associated with many diseases (Laín Entralgo 273-308). Microscopical studies by Virchow and others from the 1840s onwards identified the cellular basis of disease. From the 1860s Pasteur and Koch showed the role of bacteria in infection while Lister found practical applications in his antiseptic, and later, aseptic techniques that revolutionized the scope and safety of surgery. -
Systolic Murmurs
Murmurs and the Cardiac Physical Exam Carolyn A. Altman Texas Children’s Hospital Advanced Practice Provider Conference Houston, TX April 6 , 2018 The Cardiac Physical Exam Before applying a stethoscope….. Some pearls on • General appearance • Physical exam beyond the heart 2 Jugular Venous Distention Pallor Cyanosis 3 Work of Breathing Normal infant breathing Quiet Tachypnea Increased Rate, Work of Breathing 4 Beyond the Chest Clubbing Observed in children older than 6 mos with chronic cyanosis Loss of the normal angle of the nail plate with the axis of the finger Abnormal sponginess of the base of the nail bed Increasing convexity of the nail Etiology: ? sludging 5 Chest ❖ Chest wall development and symmetry ❖ Long standing cardiomegaly can lead to hemihypertrophy and flared rib edge: Harrison’s groove or sulcus 6 Ready to Examine the Heart Palpation Auscultation General overview Defects Innocent versus pathologic 7 Cardiac Palpation ❖ Consistent approach: palm of your hand, hypothenar eminence, or finger tips ❖ Precordium, suprasternal notch ❖ PMI? ❖ RV impulse? ❖ Thrills? ❖ Heart Sounds? 8 Cardiac Auscultation Where to listen: ★ 4 main positions ★ Inching ★ Ancillary sites: don’t forget the head in infants 9 Cardiac Auscultation Focus separately on v Heart sounds: • S2 normal splitting and intensity? • Abnormal sounds? Clicks, gallops v Murmurs v Rubs 10 Cardiac Auscultation Etiology of heart sounds: Aortic and pulmonic valves actually close silently Heart sounds reflect vibrations of the cardiac structures after valve closure Sudden -
Rhumatisme De Jaccoud : Diagnostic Et Prise En Charge. Jaccoud’S Arthropathy : Diagnosis and Therapeutic Management
3 Disponible en ligne sur FMC www.smr.ma Rhumatisme de Jaccoud : diagnostic et prise en charge. Jaccoud’s arthropathy : diagnosis and therapeutic management. Amina Mounir, Akasbi Nessrine, Harzy Taoufik. Service de Rhumatologie, CHU Hassan II, Faculté de médecine et de pharmacie, Université Sidi Mohammeh Ben Abdellah, Fès - Maroc. DOI: 10.24398/A.317.2019 Rev Mar Rhum 2019; 47:3-7 Résumé Abstract Le rhumatisme de Jaccoud (RJ) est une Jaccoud’s arthropathy (JA) is a rare pathologie rare. Il s’agit d’une arthropathie disorder. It is a chronic and non-erosive chronique non érosive touchant deforming arthropathy, usually affecting essentiellement la main. Il s’associe the hands and associated with connective fréquemment aux connectivites notamment tissue disease especially the systemic le lupus érythémateux systémique (LES). Ce lupus erythematosis. This syndrome is syndrome est caractérisé par une déformation characterized by a painless deformity of the indolore et réductible des rayons lunaires II, digits II, III, IV and V with ulnar dislocation III, IV et V avec luxation ulnaire des tendons of extensor tendons in the metacarpal extenseurs dans les vallées métacarpiennes. valleys. The pathophysiology is poorly La physiopathologie est mal connue mais elle known but involves periarticular structures implique les structures périarticulaires telles such as tendons and the joint capsule. The que les tendons et la capsule. La prise en charge clinical management of JA is always aimed du RJ vise toujours à contrôler rapidement at early control of joint inflammation and l’inflammation des articulations et à prévenir une limitation importante des mouvements et preventing severe limitation of movement une perte persistante de la fonction articulaire. -
Pregnancy and Cardiovascular Disease
Pregnancy and Cardiovascular Disease Cindy M. Martin, M.D. Co-Director, Adult Congenital and Cardiovascular Genetics Center No Disclosures Objectives • Discuss the hemodynamic changes during pregnancy • Define the low, medium and high risk cardiac lesions as related to pregnancy • Review use of cardiovascular drugs in pregnancy Pregnancy and the Heart • 2-4% of pregnancies in women without preexisting cardiac abnormalities are complicated by maternal CV disease • In 2000, there were an estimated 1 million adult patients in the US with congenital heart disease (CHD), with the number increasing by 5% yearly • In 2005, the number of adult patients with CHD surpassed the number of children with CHD in the United States • CV and CHD disease does not always preclude pregnancy but may pose increase risk to mother and fetus Hemodynamic Changes during Pregnancy • Blood Volume – increases 40-50% • Heart rate – increases 10-15 bpm • SVR and PVR – decreases • Blood Pressure – decreases 10mmHg • Cardiac Output – increases 30-50% – Peaks at end of second trimester and plateaus until delivery • These changes are usually well tolerated Physiologic Changes in Pregnancy Hemodynamic Changes in Labor and Delivery • CO increases an additional 50% with each contraction – Uterine contraction displaces 300-500ml blood into the general circulation – Possible for the cardiac output to be 70-80% above baseline during labor and delivery • Mean arterial pressure also usually rises • Volume changes – Increased blood volume with uterine contraction – Increase venous -
Towards a History of Infective Endocarditis
Medical History, 1996, 40: 25-54 Towards a History of Infective Endocarditis ALAIN CONTREPOIS* Infective endocarditis is a serious heart disease which is sometimes very difficult to diagnose and treat despite the progress made over the last few decades. The complex nature of the pathophysiological mechanisms involved in this disease is reflected in the countless number of scientific studies still being done on it.1 At the end of the eighteenth century and during the first half of the nineteenth century, the anatomo-clinical method facilitated the gradual establishment of correlations between a lesion discerned during autopsy and symptoms observed in the live patient. It then became a question of bringing together what were, until then, separate individual observations. The very term "endocarditis", referring to an individual tissue and an inflammatory process, goes back to early-nineteenth-century clinicians such as Broussais and Bouillaud, before the germ theory and the birth of bacteriology. However, it was very difficult for doctors of that period to define endocarditis in a simple, unequivocal manner. There was not always a clear relationship between the ideas which late historians have supposed fed each other; the arguments were confusing, tortuous, circular, and dead-end. In his discussion of syphilis, Ludwik Fleck correctly pointed out that disease is a constructed, ever-changing phenomenon which constantly integrates new information and concepts.2 Thus, the term "endocarditis" continued to be used for a disease which underwent endless development throughout the nineteenth century. There was also modification of the aetiological links between anatomical abnormalities, clinical symptoms and observations during autopsy. -
An Audio Guide to Pediatric and Adult Heart Murmurs
Listen Up! An Audio Guide to Pediatric and Adult Heart Murmurs May 9, 2018 Dr. Michael Grattan Dr. Andrew Thain https://pollev.com/michaelgratt679 Case • You are working at an urgent care centre when a 40 year old recent immigrant from Syria presents with breathlessness. • You hear the following on cardiac auscultation: • What do you hear? • How can you describe what you hear so another practitioner will understand exactly what you mean? • What other important information will help you determine the significance of your auscultation? Objectives • In pediatric and adult patients: – To provide a general approach to cardiac auscultation – To review the most common pathologic and innocent heart murmurs • To emphasize the importance of a thorough history and physical exam (in addition to murmur description) in determining underlying etiology for heart problems Outline • A little bit of physiology and hemodynamics (we promise not too much) • Interactive pediatric and adult cases – https://pollev.com/michaelgratt679 – Get your listening ears ready! • Systolic murmurs (pathologic and innocent) • Diastolic murmurs • Continuous murmurs • Some other stuff Normal Heart Sounds Normal First & Second Sounds Splitting of 2nd heart sound Physiological : • Venous return to right is increased in inspiration – causes delayed closure of the pulmonary valve. • Simultaneously, return to left heart is reduced - premature closure of the aortic valve. • Heart sounds are unsplit when the patient holds breath at end expiration. Fixed: • No alteration in splitting with respiration. • In a patient with ASD – In expiration there is reduced pressure in the right atrium and increased pressure in the left atrium. • Blood is shunted to the right and this delays closure of the pulmonary valve in the same way as would occur in inspiration. -
Cardiology 1
Cardiology 1 SINGLE BEST ANSWER (SBA) a. Sick sinus syndrome b. First-degree AV block QUESTIONS c. Mobitz type 1 block d. Mobitz type 2 block 1. A 19-year-old university rower presents for the pre- e. Complete heart block Oxford–Cambridge boat race medical evaluation. He is healthy and has no significant medical history. 5. A 28-year-old man with no past medical history However, his brother died suddenly during football and not on medications presents to the emergency practice at age 15. Which one of the following is the department with palpitations for several hours and most likely cause of the brother’s death? was found to have supraventricular tachycardia. a. Aortic stenosis Carotid massage was attempted without success. b. Congenital long QT syndrome What is the treatment of choice to stop the attack? c. Congenital short QT syndrome a. Intravenous (IV) lignocaine d. Hypertrophic cardiomyopathy (HCM) b. IV digoxin e. Wolff–Parkinson–White syndrome c. IV amiodarone d. IV adenosine 2. A 65-year-old man presents to the heart failure e. IV quinidine outpatient clinic with increased shortness of breath and swollen ankles. On examination his pulse was 6. A 75-year-old cigarette smoker with known ischaemic 100 beats/min, blood pressure 100/60 mmHg heart disease and a history of cardiac failure presents and jugular venous pressure (JVP) 10 cm water. + to the emergency department with a 6-hour history of The patient currently takes furosemide 40 mg BD, increasing dyspnoea. His ECG shows a narrow complex spironolactone 12.5 mg, bisoprolol 2.5 mg OD and regular tachycardia with a rate of 160 beats/min. -
Ministry of Health of Ukraine Kharkiv National Medical University
Ministry of Health of Ukraine Kharkiv National Medical University AUSCULTATION OF THE HEART. NORMAL HEART SOUNDS, REDUPLICATION OF THE SOUNDS, ADDITIONAL SOUNDS (TRIPLE RHYTHM, GALLOP RHYTHM), ORGANIC AND FUNCTIONAL HEART MURMURS Methodical instructions for students Рекомендовано Ученым советом ХНМУ Протокол №__от_______2017 г. Kharkiv KhNMU 2017 Auscultation of the heart. normal heart sounds, reduplication of the sounds, additional sounds (triple rhythm, gallop rhythm), organic and functional heart murmurs / Authors: Т.V. Ashcheulova, O.M. Kovalyova, O.V. Honchar. – Kharkiv: KhNMU, 2017. – 20 с. Authors: Т.V. Ashcheulova O.M. Kovalyova O.V. Honchar AUSCULTATION OF THE HEART To understand the underlying mechanisms contributing to the cardiac tones formation, it is necessary to remember the sequence of myocardial and valvular action during the cardiac cycle. During ventricular systole: 1. Asynchronous contraction, when separate areas of myocardial wall start to contract and intraventricular pressure rises. 2. Isometric contraction, when the main part of the ventricular myocardium contracts, atrioventricular valves close, and intraventricular pressure significantly increases. 3. The ejection phase, when the intraventricular pressure reaches the pressure in the main vessels, and the semilunar valves open. During diastole (ventricular relaxation): 1. Closure of semilunar valves. 2. Isometric relaxation – initial relaxation of ventricular myocardium, with atrioventricular and semilunar valves closed, until the pressure in the ventricles becomes lower than in the atria. 3. Phases of fast and slow ventricular filling - atrioventricular valves open and blood flows from the atria to the ventricles. 4. Atrial systole, after which cardiac cycle repeats again. The noise produced By a working heart is called heart sounds. In auscultation two sounds can be well heard in healthy subjects: the first sound (S1), which is produced during systole, and the second sound (S2), which occurs during diastole. -
Hypertension Core Curriculum. Final.Indd
MICHIGAN HYPERTENSION CORE CURRICULUM Education modules for training and updating physicians and other health professionals in hypertension detection, treatment and control Developed by the Hypertension Expert Group A Partnership of the National Kidney Foundation of Michigan and the Michigan Department of Community Health 2010 Michigan Hypertension Core Curriculum 2010 Developed by the Hypertension Expert Group A Partnership of the National Kidney Foundation of Michigan and the Michigan Department of Community Health 2 Hypertension Core Curriculum NKFM & MDCH 3 Acknowledgements Hypertension Expert Workgroup Committee April 2010 Ziad Arabi, MD, Senior Staff Physician, Internal Medicine/Hospitalist Medicine, Henry Ford Hospital, Certified Physician Specialist in Clinical Hypertension* Dear Colleague: Aaref Badshah, MD, Chief Medical Resident, Department of Internal Medicine Saint Joseph Mercy-Oakland Hospital * In 2005, the Michigan Department of Community Health (MDCH) and the National Kidney Foundation of Michigan (NKFM) convened a group of hypertension experts to identify strategies that will improve Jason I Biederman DO, FACOI FASN, Hypertension Nephrology Associates, PC* blood pressure control in Michigan. Participants included physicians from across Michigan specializing Joseph Blount, MD, MPH, FACP, Medical Director, OmniCare Health Plan, Detroit, MI in clinical hypertension, leaders in academic research of hypertension and related disorders, and Mark Britton, MD, PhD, Center of Urban and African-American Health Executive Committee, Wayne representatives of key health care organizations that are addressing this condition that afflicts over 70 State University School of medicine, Wayne State University* million U.S. adults. The Hypertension Expert Group has focused on approaches to reduce the burden of kidney and cardiovascular diseases through more effective blood pressure treatment strategies. -
Het Brown-Séquard Syndroom X Verantwoording XI Inleiding ХШ
PDF hosted at the Radboud Repository of the Radboud University Nijmegen The following full text is a publisher's version. For additional information about this publication click this link. http://hdl.handle.net/2066/113632 Please be advised that this information was generated on 2021-10-10 and may be subject to change. Het localisatieconcept in de neurologie van Brown-Séquard P.J. Koehler Vormgeving: Hein Berendsen De Wever-Ziekenhiais/Bureau Public Relations © 1989 Met befiuíp van Apple Macintosh, lettertype Bookman Het localisatieconcept in de neurologie van Brown-Séquard Frontispice: Portret van Brown-Séquard op 66 jarige leefiijd. (Bron: Archives de VAcadémie des Sciences, Parijs). Het localisatieconcept in de neurologie van Brown-Séquard Een wetenschappelijke proeve op het gebied van de Geneeskunde en Tandheelkunde Proefschrift ter verkrijging van de graad van doctor aan de Katholieke Universiteit te Nijmegen, volgens besluit van het college van decanen in het openbaar te verdedigen op vrijdag 12 mei 1989 des namiddags te 1.30 uur precies door Petrus Johannes Koehler geboren op 21 juni 1955 te 's-Gravenhage Uitgever Rodopi Amsterdam 1989 Promotores: Prof. Dr. D. de Moulin en Prof. Dr. B.P.M. Schulte Co-referent: Dr. L.J. Endtz De uitgave van dit proefschrift werd mede mogelijk gemaakt door financiële steun van: • de firma's: Geigy, ICI, MSD, Rhône-Poulenc, Sandoz en UCB. • Tevens werd een bijdrage toegekend uit het onderzoeksfonds van het De Wever-Ziekenhuis. Aan: mijn ouders Mettie Esther Edith Iris Ruben Inhoudsopgave Het Brown-Séquard Syndroom X Verantwoording XI Inleiding ХШ Hoofdstuk 1 ι Biografíe 1.1 Inleiding; reeds bestaande biografieën. -
(Second Heart Sound) Splitting in Normal Physiology and Congenital
Mini Review iMedPub Journals Journal of Pediatric Care 2018 www.imedpub.com ISSN 2471-805X Vol.4 No.2:3 DOI: 10.21767/2471-805X.100035 The Mechanism of S2 (Second Heart Bilton Samuel J1* and 2 Sound) Splitting in Normal Physiology and Carande Elliott J Congenital Heart Defects 1 St George’s University Hospitals NHS Foundation Trust, Blackshaw Road, Tooting, London, UK 2 Royal Gwent Hospital, Aneurin Bevan University Health Board, Cardiff Road, Abstract Newport, UK Splitting of the second heart sound is a difficult concept which is stated, rather than explained, in many medical texts. The splitting into the pulmonary valve and aortic valve components occurs in physiology during inspiration, which is due to later closure of the pulmonary valve as well as earlier closure of the aortic *Corresponding author: Bilton Samuel J valve, determined by changes in intrathoracic pressure and the capacitance of the pulmonary vasculature. Fixed splitting of the second heart sound describes a situation where the second heart sound is split in both inspiration and expiration [email protected] and is classically described in an atrial septal defect. The evidence behind these mechanisms is discussed in the following article. St George’s University Hospitals NHS Keywords: Blood; Pulmonary; Pediatrics; Cardiology; Heart; Hypertension Foundation Trust, Blackshaw Road, Tooting, London, UK. Received: August 13, 2018; Accepted: September 27, 2018; Published: October 03, Tel: 02086721255 2018 Citation: Bilton Samuel J, Carande Elliott J (2018) The Mechanism of S2 (Second Heart Introduction Sound) Splitting in Normal Physiology and Congenital Heart Defects. J Pediatr Care There is a great deal of confusion and complexity surrounding Vol.4 No.2:3 the second heart sound and how it “splits” in normal individuals and in patients with congenital heart defects, such as atrial septal congenital heart defects, we hope to ease how medical students defect and ventricular septal defect.