A Clinical Review of Orbital Anatomy and Its Relevance to Retrobulbar Anaesthesia
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MR Imaging of the Orbital Apex
J Korean Radiol Soc 2000;4 :26 9-0 6 1 6 MR Imaging of the Orbital Apex: An a to m y and Pat h o l o g y 1 Ho Kyu Lee, M.D., Chang Jin Kim, M.D.2, Hyosook Ahn, M.D.3, Ji Hoon Shin, M.D., Choong Gon Choi, M.D., Dae Chul Suh, M.D. The apex of the orbit is basically formed by the optic canal, the superior orbital fis- su r e , and their contents. Space-occupying lesions in this area can result in clinical d- eficits caused by compression of the optic nerve or extraocular muscles. Even vas c u l a r changes in the cavernous sinus can produce a direct mass effect and affect the orbit ap e x. When pathologic changes in this region is suspected, contrast-enhanced MR imaging with fat saturation is very useful. According to the anatomic regions from which the lesions arise, they can be classi- fied as belonging to one of five groups; lesions of the optic nerve-sheath complex, of the conal and intraconal spaces, of the extraconal space and bony orbit, of the cav- ernous sinus or diffuse. The characteristic MR findings of various orbital lesions will be described in this paper. Index words : Orbit, diseases Orbit, MR The apex of the orbit is a complex region which con- tains many nerves, vessels, soft tissues, and bony struc- Anatomy of the orbital apex tures such as the superior orbital fissure and the optic canal (1-3), and is likely to be involved in various dis- The orbital apex region consists of the optic nerve- eases (3). -
Permeability of the Retina and RPE-Choroid-Sclera to Three Ophthalmic Drugs and the Associated Factors
pharmaceutics Article Permeability of the Retina and RPE-Choroid-Sclera to Three Ophthalmic Drugs and the Associated Factors Hyeong Min Kim 1,†, Hyounkoo Han 2,†, Hye Kyoung Hong 1, Ji Hyun Park 1, Kyu Hyung Park 1, Hyuncheol Kim 2,* and Se Joon Woo 1,* 1 Department of Ophthalmology, Seoul National University College of Medicine, Seoul National University Bundang Hospital, Seongnam 13620, Korea; [email protected] (H.M.K.); [email protected] (H.K.H.); [email protected] (J.H.P.); [email protected] (K.H.P.) 2 Department of Chemical and Biomolecular Engineering, Sogang University, Seoul 04107, Korea; [email protected] * Correspondence: [email protected] (H.K.); [email protected] (S.J.W.); Tel.: +82-2-705-8922 (H.K.); +82-31-787-7377 (S.J.W.); Fax: +82-2-3273-0331 (H.K.); +82-31-787-4057 (S.J.W.) † These authors contributed equally to this work. Abstract: In this study, Retina-RPE-Choroid-Sclera (RCS) and RPE-Choroid-Sclera (CS) were prepared by scraping them off neural retina, and using the Ussing chamber we measured the average time– concentration values in the acceptor chamber across five isolated rabbit tissues for each drug molecule. We determined the outward direction permeability of the RCS and CS and calculated the neural retina permeability. The permeability coefficients of RCS and CS were as follows: ganciclovir, 13.78 ± 5.82 and 23.22 ± 9.74; brimonidine, 15.34 ± 7.64 and 31.56 ± 12.46; bevacizumab, 0.0136 ± 0.0059 and 0.0612 ± 0.0264 (×10−6 cm/s). -
CHQ-GDL-01074 Acute Management of Open Globe Injuries
Acute management of Open Globe Injuries Document ID CHQ-GDL-01074 Version no. 2.0 Approval date 14/05/2020 Executive sponsor Executive Director Medical Services Effective date 14/05/2020 Author/custodian Director Infection Management and Prevention service, Review date 14/05/2022 Immunology and Rheumatology Supersedes 1.0 Applicable to All Children’s Health Queensland (CHQ) staff Authorisation Executive Director Clinical Services (QCH) Purpose This evidence-based guideline provides clinical practice advice for clinicians for the acute management of children with open globe injuries. A paediatric ophthalmology team must be actively involved in the management of all patients presenting with this condition. Scope This guideline applies to all Children’s Health Queensland (CHQ) Staff treating a child presenting for the management of open globe injury. Related documents • CHQ-GDL-01202 CHQ Paediatric Antibiocard: Empirical Antibiotic Guidelines • CHQ-PROC-01035 Antimicrobial Restrictions • CHQ Antimicrobial Restriction list • CHQ-GDL-01023 Tetanus Prophylaxis in Wound Management CHQ-GDL-01074- Acute management of Open Globe Injuries - 1 - Guideline Introduction Ocular trauma is an important cause of eye morbidity and is a leading cause of non-congenital mono-ocular blindness among children.1 A quarter of a million children present each year with serious ocular trauma. The vast majority of these are preventable.2 Open globe injuries are injuries where the cornea and/or sclera are breached and there is a full-thickness wound of the eye wall.3 It can be further delineated into globe rupture from blunt trauma and lacerations from sharp objects. When a large blunt object impacts onto the eye, there is an instant increase in intraocular pressure and the eye wall yields at its weakest point leading to tissue prolapse.4 Open globe lacerations are caused by sharp objects or projectiles and subdivided into either penetrating or perforating injuries. -
Raised Intracranial Pressure Presenting with Spontaneous Periorbital Bruising: Two Case Reports S Hadjikoutis, C Carroll, G T Plant
1192 J Neurol Neurosurg Psychiatry: first published as 10.1136/jnnp.2003.016006 on 16 July 2004. Downloaded from SHORT REPORT Raised intracranial pressure presenting with spontaneous periorbital bruising: two case reports S Hadjikoutis, C Carroll, G T Plant ............................................................................................................................... J Neurol Neurosurg Psychiatry 2004;75:1192–1193. doi: 10.1136/jnnp.2003.016006 The venous drainage of the orbit is known to be via the ophthalmic and vortex veins which communicate with the cavernous sinus. We describe two patients with raised intracranial pressure presenting with periorbital bruising. In one patient dural venous sinus thrombosis was demonstrated and it is suspected that the cause of the raised intracranial pressure may have been the same in the second. We suggest that the abrupt rise of pressure in the cerebral venous system was transmitted via the cavernous sinus to the orbital venous system. Figure 1 Case 1: periorbital bruising more marked on the right. he early diagnosis of raised intracranial pressure can be problematic, especially when the patient first visits an Taccident and emergency department and there are no abnormal physical signs. We describe two patients who presented with headache due to raised intracranial pressure copyright. associated with bilateral periorbital bruising. We suggest that this may be an external sign of raised intracranial pressure under certain circumstances. We then go on to discuss the possible mechanisms whereby an abrupt rise in intracranial pressure may give rise to periorbital bruising. CASE REPORTS Case 1 A 24 year old woman with a history of migraine presented with a three day history of spontaneous periorbital bruising. -
Optic Disc Edema, Globe Flattening, Choroidal Folds, and Hyperopic Shifts Observed in Astronauts After Long-Duration Space Flight
University of Nebraska - Lincoln DigitalCommons@University of Nebraska - Lincoln NASA Publications National Aeronautics and Space Administration 10-2011 Optic Disc Edema, Globe Flattening, Choroidal Folds, and Hyperopic Shifts Observed in Astronauts after Long-duration Space Flight Thomas H. Mader Alaska Native Medical Center, [email protected] C. Robert Gibson Coastal Eye Associates Anastas F. Pass University of Houston Larry A. Kramer University of Texas Health Science Center Andrew G. Lee The Methodist Hospital See next page for additional authors Follow this and additional works at: https://digitalcommons.unl.edu/nasapub Part of the Physical Sciences and Mathematics Commons Mader, Thomas H.; Gibson, C. Robert; Pass, Anastas F.; Kramer, Larry A.; Lee, Andrew G.; Fogarty, Jennifer; Tarver, William J.; Dervay, Joseph P.; Hamilton, Douglas R.; Sargsyan, Ashot; Phillips, John L.; Tran, Duc; Lipsky, William; Choi, Jung; Stern, Claudia; Kuyumjian, Raffi; andolk, P James D., "Optic Disc Edema, Globe Flattening, Choroidal Folds, and Hyperopic Shifts Observed in Astronauts after Long-duration Space Flight" (2011). NASA Publications. 69. https://digitalcommons.unl.edu/nasapub/69 This Article is brought to you for free and open access by the National Aeronautics and Space Administration at DigitalCommons@University of Nebraska - Lincoln. It has been accepted for inclusion in NASA Publications by an authorized administrator of DigitalCommons@University of Nebraska - Lincoln. Authors Thomas H. Mader, C. Robert Gibson, Anastas F. Pass, Larry A. -
The Sclera C
The Sclera c. Stephen Foster Maite Sainz de la Maza The Sclera Foreword by Frederick A. lakobiec With 134 Illustrations and 33 Color Plates Springer Science+Business Media, LLC C. Stephen Foster, MD Associate Professor of Ophthalmology Harvard Medical School Director, Immunology and Uveitis Service Massachusetts Eye and Ear Infirmary Boston, MA 02114 USA Maite Sainz de la Maza, MD, PhD Assistant Professor of Ophthalmology Central University of Barcelona 08036 Barcelona Spain Cover illustration: The eye of a patient with rheumatoid arthritis who has developed pro gressively destructive necrotizing scleritis. Library of Congress Cataloging-in-Publication Data Foster, C. Stephen (Charles Stephen), 1942- The sclera/C. Stephen Foster and Maite Sainz de la Maza. p. cm. Includes bibliographical references and index. ISBN 978-1-4757-2345-8 ISBN 978-1-4757-2343-4 (eBook) DOI 10.1007/978-1-4757-2343-4 1. Sclera-Diseases. I. Maza, Maite Sainz de lao II. Title. [DNLM: 1. Scleritis. 2. Sclera. WW 230 F754s 1993] RE328.F67 1993 617.7' 19-dc20 DNLMIDLC for Library of Congress 93-10235 Printed on acid-free paper. © 1994 Springer Science+ Business Media New York Originally published by Springer-Verlag New York, Inc. in 1994 Softcover reprint of the hardcover 1st edition 1994 All rights reserved. This work may not be translated or copied in whole or in part without the written permission of the publisher, Springer Science+Business Media, LLC. except for brief excerpts in connection with reviews or, scholarly analysis. Use in connection with any form of information storage and retrieval, electronic adaptation, computer software, or by similar or dissimilar methodology now known or hereafter developed is forbidden. -
Branches of the Maxillary Artery of the Dromedary, Camelus Dromedarius
Table 3.5: Branches of the Maxillary Artery of the Dromedary, Camelus dromedarius Artery Origin Course Distribution Departs from common trunk with the deep temporal vessels, close to mandibular foramen; traverses mandibular canal, supplying Mandibular dentition; lower lip; Inferior Alveolar Maxillary Artery mandibular dentition. Terminates as mental anastomoses freely with ventral ramus artery after exiting at mental foramen, of the facial artery. whereupon supplies skin, mucosa, and muscle of the lower lip. Single deep temporal vessel is first major dorsal branch of the MA; ascends deep to the coronoid Deep Temporal Maxillary Artery Temporalis muscle process and fans out on the deep surface of the temporalis muscle. Lower lateral branch of deep temporal artery; passes through the mandibular incisure and Masseteric Deep Temporal Artery Masseter muscle curves rostrally to pierce the internal surface of the masseter muscle. Proximal to the foramen orbitorotundum and optic foramen, numerous rami anastomotica connect the maxillary artery to the carotid rete. Carotid rete, ophthalmic rete, external Ramus anastomoticus Maxillary Artery The network in the dromedary is extensive, ophthalmic artery; intracranial cavity forming a plexus between the carotid and ophthalmic retia, and giving rise to the external ophthalmic artery. Condenses from a dense retial mat (composed of maxillary rami, the extradural/extracranial portion of the carotid rete, and the ophthalmic Extraocular muscles, periorbita, External Ophthalmic MA/CR/OR rete). Perfuses the majority of the periorbita, lacrimal gland including branches to the extraocular muscles and the lacrimal gland Lateral branch of the MA, begins opposite the rami anastomotica; traverses parenchyma orbital Supplies the buccal fat pad, Buccal MA fossa, between malar and anterior border of buccinator; contributes ventral coronoid process. -
Extraocular Muscles Orbital Muscles
EXTRAOCULAR MUSCLES ORBITAL MUSCLES INTRA- EXTRA- OCULAR OCULAR CILIARY MUSCLES INVOLUNTARY VOLUNTARY 1.Superior tarsal muscle. 1.Levator Palpebrae Superioris 2.Inferior tarsal muscle 2.Superior rectus 3.Inferior rectus 4.Medial rectus 5.Lateral rectus 6.Superior oblique 7.Inferior oblique LEVATOR PALPEBRAE SUPERIORIOS Origin- Inferior surface of lesser wing of sphenoid. Insertion- Upper lamina (Voluntary) - Anterior surface of superior tarsus & skin of upper eyelid. Middle lamina (Involuntary) - Superior margin of superior tarsus. (Superior Tarsus Muscle / Muller muscle) Lower lamina (Involuntary) - Superior conjunctival fornix Nerve Supply :- Voluntary part – Oculomotor Nerve Involuntary part – Sympathetic ACTION :- Elevation of upper eye lid C/S :- Drooping of upper eyelid. Congenital ptosis due to localized myogenic dysgenesis Complete ptosis - Injury to occulomotor nerve. Partial ptosis - disruption of postganglionic sympathetic fibres from superior cervical sympathetic ganglion. Extra ocular Muscles : Origin Levator palpebrae superioris Superior Oblique Superior Rectus Lateral Rectus Medial Rectus Inferior Oblique Inferior Rectus RECTUS MUSCLES : ORIGIN • Arises from a common tendinous ring knows as ANNULUS OF ZINN • Common ring of connective tissue • Anterior to optic foramen • Forms a muscle cone Clinical Significance Retrobulbar neuritis ○ Origin of SUPERIOR AND MEDIAL RECTUS are closely attached to the dural sheath of the optic nerve, which leads to pain during upward & inward movements of the globe. Thyroid orbitopathy ○ Medial & Inf.rectus thicken. especially near the orbital apex - compression of the optic nerve as it enters the optic canal adjacent to the body of the sphenoid bone. Ophthalmoplegia ○ Proptosis occur due to muscle laxity. Medial Rectus Superior Rectus Origin :- Superior limb of the tendonous ring, and optic nerve sheath. -
Treatment of Congenital Ptosis
13 Review Article Page 1 of 13 Treatment of congenital ptosis Vladimir Kratky1,2^ 1Department of Ophthalmology, Queen’s University, Kingston, Canada; 21st Medical Faculty, Charles University, Prague, Czech Republic Correspondence to: Vladimir Kratky, BSc, MD, FRCSC, DABO. Associate Professor of Ophthalmology, Director of Ophthalmic Plastic and Orbital Surgery, Oculoplastics Fellowship Director, Queen’s University, Kingston, Canada; 1st Medical Faculty, Charles University, Prague, Czech Republic. Email: [email protected]. Abstract: Congenital ptosis is an abnormally low position of the upper eyelid, with respect to the visual axis in the primary gaze. It can be present at birth or manifest itself during the first year of life and can be bilateral or unilateral. Additionally, it may be an isolated finding or part of a constellation of signs of a specific syndrome or systemic associations. Depending on how much it interferes with the visual axis, it may be considered as a functional or a cosmetic condition. In childhood, functional ptosis can lead to deprivation amblyopia and astigmatism and needs to be treated. However, even mild ptosis with normal vision can lead to psychosocial problems and correction is also advised, albeit on a less urgent basis. Although, patching and glasses can be prescribed to treat the amblyopia, the mainstay of management is surgical. There are several types of surgical procedure available depending on the severity and etiology of the droopy eyelid. The first part of this paper will review the different categories of congenital ptosis, including more common associated syndromes. The latter part will briefly cover the different surgical approaches, with emphasis on how to choose the correct condition. -
Periorbital Sinuses the Periorbital Sinuses Have a Close Anatomical Relationship with the Orbits (Fig 1-8)
12 ● Fundamentals and Principles of Ophthalmology Lacrimal nerve Frontal nerve Trochlear nerve (CN IV) Superior ophthalmic vein Superior division Ophthalmic artery of CN III Nasociliary nerve Abducens nerve (CN VI) Inferior division of CN III Inferior ophthalmic vein A Figure 1-7 A, Anterior view of the right orbital apex showing the distribution of the nerves as they enter through the superior orbital fissure and optic canal. This view also shows the annu- lus of Zinn, the fibrous ring formed by the origin of the 4 rectus muscles. (Continued) The course of the inferior ophthalmic vein is variable, and it can travel within or below the ring as it exits the orbit. The inferior orbital fissure lies just below the superior fissure, between the lateral wall and the floor of the orbit, providing access to the pterygopalatine and inferotemporal fos- sae (see Fig 1-1). Therefore, it is close to the foramen rotundum and the pterygoid canal. The inferior orbital fissure transmits the infraorbital and zygomatic branches of CN V2, an orbital nerve from the pterygopalatine ganglion, and the inferior ophthalmic vein. The inferior ophthalmic vein connects with the pterygoid plexus before draining into the cav- ernous sinus. Periorbital Sinuses The periorbital sinuses have a close anatomical relationship with the orbits (Fig 1-8). The medial walls of the orbits, which border the nasal cavity anteriorly and the ethmoid sinus and sphenoid sinus posteriorly, are almost parallel. In adults, the lateral wall of each orbit forms an angle of approximately 45° with the medial plane. The lateral walls border the middle cranial, temporal, and pterygopalatine fossae. -
Turn-Over Orbital Septal Flap and Levator Recession for Upper-Eyelid
Eye (2013) 27, 1174–1179 & 2013 Macmillan Publishers Limited All rights reserved 0950-222X/13 www.nature.com/eye 1 2 3 1 CLINICAL STUDY Turn-over orbital A Watanabe , PN Shams , N Katori , S Kinoshita and D Selva2 septal flap and levator recession for upper-eyelid retraction secondary to thyroid eye disease Abstract Background A turn-over septal flap has been Keywords: upper-eyelid retraction; orbital reported as a spacer for levator lengthening septal flap; levator recession in a single case report. This study reports the preliminary outcomes of this technique in a series of patients with upper-lid retraction (ULR) associated with thyroid eye disease 1Department of Ophthalmology, Introduction Kyoto Prefectural University of (TED) causing symptomatic exposure Medicine, Kyoto, Japan keratopathy (EK). Achieving a predictable eyelid height and Methods Retrospective, multicenter study contour in the surgical correction of upper- 2 Department of Ophthalmology of 12 eyelids of 10 patients with TED eyelid retraction remains a challenge for and Visual Sciences, South Australian Institute of undergoing a transcutaneous levator- surgeons, as evidenced by the variety of Ophthalmology, Adelaide lengthening technique using the reflected procedures reported.1,2 These techniques are University, Adelaide, South orbital septum (OS) as a spacer. Change in based on weakening or lengthening the Australia, Australia palpebral aperture (PA) and contour, position upper-eyelid retractors and include anterior or 3Department of Oculoplastic of the skin crease (SC), symptoms of EK, and posterior approaches to graded recession and Orbital Surgery, Seirei complications were recorded. or resection of Mu¨ ller’s muscle,3–5 levator Hamamatsu Hospital, Results The average age was 47.5 years. -
CHAPTER 8 Face, Scalp, Skull, Cranial Cavity, and Orbit
228 CHAPTER 8 Face, Scalp, Skull, Cranial Cavity, and Orbit MUSCLES OF FACIAL EXPRESSION Dural Venous Sinuses Not in the Subendocranial Occipitofrontalis Space More About the Epicranial Aponeurosis and the Cerebral Veins Subcutaneous Layer of the Scalp Emissary Veins Orbicularis Oculi CLINICAL SIGNIFICANCE OF EMISSARY VEINS Zygomaticus Major CAVERNOUS SINUS THROMBOSIS Orbicularis Oris Cranial Arachnoid and Pia Mentalis Vertebral Artery Within the Cranial Cavity Buccinator Internal Carotid Artery Within the Cranial Cavity Platysma Circle of Willis The Absence of Veins Accompanying the PAROTID GLAND Intracranial Parts of the Vertebral and Internal Carotid Arteries FACIAL ARTERY THE INTRACRANIAL PORTION OF THE TRANSVERSE FACIAL ARTERY TRIGEMINAL NERVE ( C.N. V) AND FACIAL VEIN MECKEL’S CAVE (CAVUM TRIGEMINALE) FACIAL NERVE ORBITAL CAVITY AND EYE EYELIDS Bony Orbit Conjunctival Sac Extraocular Fat and Fascia Eyelashes Anulus Tendineus and Compartmentalization of The Fibrous "Skeleton" of an Eyelid -- Composed the Superior Orbital Fissure of a Tarsus and an Orbital Septum Periorbita THE SKULL Muscles of the Oculomotor, Trochlear, and Development of the Neurocranium Abducens Somitomeres Cartilaginous Portion of the Neurocranium--the The Lateral, Superior, Inferior, and Medial Recti Cranial Base of the Eye Membranous Portion of the Neurocranium--Sides Superior Oblique and Top of the Braincase Levator Palpebrae Superioris SUTURAL FUSION, BOTH NORMAL AND OTHERWISE Inferior Oblique Development of the Face Actions and Functions of Extraocular Muscles Growth of Two Special Skull Structures--the Levator Palpebrae Superioris Mastoid Process and the Tympanic Bone Movements of the Eyeball Functions of the Recti and Obliques TEETH Ophthalmic Artery Ophthalmic Veins CRANIAL CAVITY Oculomotor Nerve – C.N. III Posterior Cranial Fossa CLINICAL CONSIDERATIONS Middle Cranial Fossa Trochlear Nerve – C.N.