Occupational Chemical Respiratory Exposures & Asthma
Total Page:16
File Type:pdf, Size:1020Kb
Load more
Recommended publications
-
Occupational Airborne Particulates
Environmental Burden of Disease Series, No. 7 Occupational airborne particulates Assessing the environmental burden of disease at national and local levels Tim Driscoll Kyle Steenland Deborah Imel Nelson James Leigh Series Editors Annette Prüss-Üstün, Diarmid Campbell-Lendrum, Carlos Corvalán, Alistair Woodward World Health Organization Protection of the Human Environment Geneva 2004 WHO Library Cataloguing-in-Publication Data Occupational airborne particulates : assessing the environmental burden of disease at national and local levels / Tim Driscoll … [et al.]. (Environmental burden of disease series / series editors: Annette Prüss-Ustun ... [et al.] ; no. 7) 1.Dust - adverse effects 2.Occupational exposure 3.Asthma - chemically induced 4.Pulmonary disease, Chronic obstructive - chemically induced 5.Pneumoconiosis - etiology 6.Cost of illness 7.Epidemiologic studies 8.Risk assessment - methods 9.Manuals I.Driscoll, Tim. II.Prüss-Üstün, Annette. III.Series. ISBN 92 4 159186 2 (NLM classification: WA 450) ISSN 1728-1652 Suggested Citation Tim Driscoll, et al. Occupational airborne particulates: assessing the environmental burden of disease at national and local levels. Geneva, World Health Organization, 2004. (Environmental Burden of Disease Series, No. 7). © World Health Organization 2004 All rights reserved. Publications of the World Health Organization can be obtained from Marketing and Dissemination, World Health Organization, 20 Avenue Appia, 1211 Geneva 27, Switzerland (tel: +41 22 791 2476; fax: +41 22 791 4857; email: [email protected]). -
Siphoning Diesel: a Fatal Mistake
CASE REPORT Siphoning diesel: a fatal mistake Leong Wei Cheng, MRCP, Brian Cheong Mun Keong, FRCP Department of Medicine, Hospital Teluk Intan cervical lymphadenopathy and auscultation of his lungs SUMMARY revealed normal findings. Examination of other systems did Diesel is commonly used as fuel for engines and is distilled not reveal any abnormalities. from petroleum. Diesel has toxic potential and can affect multiple organs. Exposure can occur after ingestion, Initial blood investigations showed evidence of acute kidney inhalation or through the dermal route. The practice of injury (urea: 16.7 mmol/l, sodium 129 mmol/l, potassium 3.7 siphoning diesel using a rubber tubing and the mouth is mmol/l and creatinine 937 µmol/l). There was presence of common in rural communities. This can lead to accidental protein and erythrocytes (2+) in his urine. His full blood count ingestion and aspiration. Here we report a case of a patient showed elevated white cell count of 11,600/µl (neutrophil who accidentally ingested diesel during siphoning, which 85%) with normal haemoglobin concentration (15.9 g/dl) caused extensive erosion of the oral cavity and oesophagus and platelets (185,000/µl). Liver transaminases were normal. leading to pneumomediastinum and severe chemical lung Chest radiograph showed clear lung fields. His provisional injury. The patient responded well initially to steroids and diagnosis was acute tonsillitis with post- infectious Rapidly supportive care but required prolonged hospitalisation. He Progressive Glomerolonephritis (RPGN). Intravenous (IV) developed complications of nosocomial infection and Ceftriaxone 2 g daily was started. succumbed 23 days after admission. He became progressively more tachypnoeic in the ward with KEY WORDS: Diesel; siphon; chemical pneumonitis; oesophageal perforation; worsening renal function and metabolic acidosis. -
Dust Exposure and Byssinosis Among Cotton Textile Workers in Dar Es Salaam, Tanzania
MOJ Public Health Research Article Open Access Dust exposure and byssinosis among cotton textile workers in Dar es salaam, Tanzania Abstract Volume 9 Issue 6 - 2020 Background: Cotton dust exposure increases the risk of developing lung diseases including Luco P Mwelange, Simon Mamuya, Gloria Byssinosis. The prevalence of byssinosis is more in developing countries compare to developed countries. For the past forty years there are little information known about the Sakwari, Witness John Axwesso Department of Environmental and Occupational Health, prevalence of byssinosis and its associated risk factors among textile workers in Tanzania. Muhimbili University of Health and Allied Sciences, Tanzania Objective: The study aimed to assess dust exposure and associated risk factors among textile workers, in Dar es Salaam, Tanzania. Correspondence: Luco P Mwelange, Muhimbili University of Health and Allied Sciences, Tanzania, Tel+255655049524, Material and methods: The study design was descriptive cross sectional study conducted Email from March to August 2019. Stratified sampling technique was used to obtain 325 participants (exposed 164 and control 161) respectively. A modified British Medical Received: October 29, 2020 | Published: November 30, 2020 Research Council (BMRC) questionnaire and Side Kick Casella Pump were used for data collection. Data were analyzed using Statistical Package for Social Science software 23 versions. Chi square test and Binary logistic regression were performed to check for association. A 95% confidence Interval with a significance expressed in P˂0.05 was used. Results: Prevalence of byssinosis in the exposed group was 18.9% and 6.2% in the control group. Respiratory symptoms such as Coughing more days in three consecutive months (P˂0.001), wheezing (P˂0.02), dyspnoea I (P˂0.03), dyspnoea II (P˂0.007), and dyspnoea III (P˂0.002), were higher among exposed group compare to control group and the differences were statistically significant. -
Occupational Lung Diseases
24 Occupational lung diseases Introduction i Occupational diseases are often thought to be Key points uniquely and specifically related to factors in the work environment; examples of such diseases are • Systematic under-reporting and the pneumoconioses. However, in addition to other difficulties in attributing causation both contribute to underappreciation of the factors (usually related to lifestyle), occupational burden of occupational respiratory exposures also contribute to the development or diseases. worsening of common respiratory diseases, such • Work-related exposures are estimated as chronic obstructive pulmonary disease (COPD), to account for about 15% of all adult asthma and lung cancer. asthma cases. • Boththe accumulation of toxic dust in the Information about the occurrence of occupational lungs and immunological sensitisation respiratory diseases and their contribution to to inhaled occupational agents can morbidity and mortality in the general population is cause interstitial lung disease. provided by different sources of varying quality. Some • Despite asbestos use being phased European countries do not register occupational out, mesothelioma rates are forecast diseases and in these countries, information about to continue rising owing to the long latency of the disease. the burden of such diseases is completely absent. • The emergence of novel occupational In others, registration is limited to cases where causes of respiratory disease in compensation is awarded, which have to fulfil specific recent years emphasises the need for administrative or legal criteria as well as strict continuing vigilance. medical criteria; this leads to biased information and underestimation of the real prevalence. Under- reporting of occupational disease is most likely to occur in older patients who are no longer at work but whose condition may well be due to their previous job. -
08-0205: N.M. and DEPARTMENT of the NAVY, PUGET S
United States Department of Labor Employees’ Compensation Appeals Board __________________________________________ ) N.M., Appellant ) ) and ) Docket No. 08-205 ) Issued: September 2, 2008 DEPARTMENT OF THE NAVY, PUGET ) SOUND NAVAL SHIPYARD, Bremerton, WA, ) Employer ) __________________________________________ ) Appearances: Oral Argument July 16, 2008 John Eiler Goodwin, Esq., for the appellant No appearance, for the Director DECISION AND ORDER Before: DAVID S. GERSON, Judge COLLEEN DUFFY KIKO, Judge JAMES A. HAYNES, Alternate Judge JURISDICTION On October 30, 2007 appellant filed a timely appeal from a November 17, 2006 decision of the Office of Workers’ Compensation Programs denying his occupational disease claim. Pursuant to 20 C.F.R. §§ 501.2(c) and 501.3, the Board has jurisdiction over the merits of the claim. ISSUE The issue is whether appellant has established that he sustained occupational asthma in the performance of duty due to accepted workplace exposures. On appeal, he, through his attorney, asserts that the Office did not provide Dr. William C. Stewart, the impartial medical examiner, with a complete, accurate statement of accepted facts. FACTUAL HISTORY On December 8, 2004 appellant, then a 57-year-old insulator, filed an occupational disease claim (Form CA-2) asserting that he sustained occupational asthma and increasing shortness of breath due to workplace exposures to fiberglass, silicates, welding smoke, polychlorobenzenes, rubber, dusts, gases, fumes and smoke from “burning out” submarines from 1991 through January -
Occupational Dusts Other Than Silica* by Kenneth M
Thorax: first published as 10.1136/thx.2.2.91 on 1 June 1947. Downloaded from Thorax (1947), 2, 91. DISEASES OF THE LUNG RESULTING FROM OCCUPATIONAL DUSTS OTHER THAN SILICA* BY KENNETH M. A. PERRY London It is only in recent years that considerable interest has been given to the dusts to which men are exposed at their work, even though silicosis is known to have occurred in prehistoric times. Many dusts are now recognized as dangerous, and in the extreme it may even be doubted whether any dust can be regarded as harmless. It is rational at least to suppose that the lung cannot become a physiological dust trap and yet retain its elasticity. It seems possible that any dust, no matter how innocuous in small concentrations, would in large enough quantity eventually overwhelm the defences of the lung and accumulate in such amounts as to impair function; such a form of lung disease would be the result of causes of a mechanical nature-the physical presence of large amounts of inert foreign material. The term "benign pneumoconiosis " has been given to this type of disease in order to contrast it with diseases resulting http://thorax.bmj.com/ from the inhalation of siliceous matter. But besides this group of conditions occupational dust may give rise to inflammatory lesions, allergic responses, and neoplastic changes. INFLAMMATORY CHANGES Inflammatory changes may be caused by inorganic metals, such as manganese, on September 24, 2021 by guest. Protected copyright. beryllium, vanadium, and osmium, giving.rise to a chemical pneumonitis; and by organic matter, such as decaying hay and grain, bagasse, cotton fibre, and similar substances where the aetiology is somewhat obscure, though fungi are frequently blamed. -
National Occupational Research Agenda (Nora)
NATIONAL OCCUPATIONAL RESEARCH AGENDA (NORA) NATIONAL OCCUPATIONAL RESEARCH AGENDA FOR HEALTHCARE AND SOCIAL ASSISTANCE (HCSA) February 2019 Developed by the NORA HCSA Council 1 . For more information about the National Occupational Research Agenda (NORA), visit the web site: https://www.cdc.gov/niosh/nora/ For monthly updates on NORA, subscribe to NIOSH eNews at www.cdc.gov/niosh/eNews Disclaimer This is a product of the National Occupational Research Agenda (NORA) Healthcare and Social Assistance Sector Council. It does not necessarily represent the official position of the National Institute for Occupational Safety and Health, Centers for Disease Control and Prevention, or U.S. Department of Health and Human Services. 2 INTRODUCTION What is the National Occupational Research Agenda? The National Occupational Research Agenda (NORA) is a partnership program to stimulate innovative research and workplace interventions. In combination with other initiatives, the products of this program are expected to reduce the occurrence of injuries and illnesses at work. Unveiled in 1996, NORA has become a research framework for the Nation and the National Institute for Occupational Safety and Health (NIOSH). Diverse parties collaborate to identify the most critical issues in workplace safety and health and develop research objectives for addressing those needs. NORA enters its third decade in 2016 with an enhanced structure. The ten sectors formed for the second decade will continue to prioritize occupational safety and health research by major areas of the U.S. economy. In addition, there are seven cross-sectors organized according the major health and safety issues affecting the U.S. working population. While NIOSH is serving as the steward to move this effort forward, it is truly a national effort. -
Airbag Pneumonitis
Hindawi Publishing Corporation Case Reports in Medicine Volume 2010, Article ID 498569, 2 pages doi:10.1155/2010/498569 Case Report Airbag Pneumonitis Raghav Govindarajan, Gustavo Ferrer, Laurence A. Smolley, Eduardo Araujo Oliveira, and Franck Rahaghi Department of Pulmonary Medicine, Cleveland Clinic Florida, 2950 Cleveland Clinic Boulevard, Weston, FL 33331, USA Correspondence should be addressed to Raghav Govindarajan, [email protected] Received 29 June 2010; Accepted 13 September 2010 Academic Editor: Steven A. Sahn Copyright © 2010 Raghav Govindarajan et al. This is an open access article distributed under the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited. The widespread and mandatory use of airbags has resulted in various patterns of injuries and complications unique to their use. Airbags have been implicated in a spectrum of pulmonary conditions ranging from exacerbation of asthma, reactive airway diseases to new onset asthma. We report a case of inhalational chemical pneumonitis that developed after exposure to the airbag fumes. 1. Introduction room air. There were no significant electrolyte abnormalities. Chest X-ray PA and lateral view revealed left lower lobe and ffi The 2001 United States national highway tra csafety retocardiac infiltrate. CT chest noncontrast revealed tree in administration report estimated that airbags reduce driver bud opacities in the right middle lobe, left lingual. Ground fatality by 12%–14% [1].The widespread and mandatory glass opacity was also seen in the posterior basal segment of use of airbags has resulted in various patterns of injuries the left lower lobe (Figure 1). -
Is Your Patient's Workplace Causing Lung Disease?
IsIs YourYour Patient’sPatient’s WWorkplaceorkplace CausingCausing LungLung Disease?Disease? Occupational lung diseases not only have a significant health impact on the affected individual, but they often result in workplace changes and significant socio-economic impact. By Susan M.Tarlo, MB, BS, FRCPC he range and relative frequency of blasting underground. Conversely, the T occupational lung diseases has diagnosis of occupational asthma caused changed significantly in Canada over the by workplace sensitizers has risen, and past 30 years. Occupational lung diseases this is now the most common chronic that were relatively common before, such occupational lung disease in Canada.1,2 as silicosis and coal miners’ pneumoco- It is estimated that occupational asthma niosis, are now uncommon conditions in (usually due to an immunologic response Canada. Although silicosis can still be to a work agent) accounts for about 7% of caused by sandblasting and occasionally all adult-onset asthma,3 and occupational by other types of exposures, it has become factors may play a role in up to 30% of uncommon in Canadian underground min- adult asthma.4 There has been increased ers. This is due to much improved dust- recognition of the role of workplace irri- control measures, such as spraying water tants in aggravating asthma and even, at to keep dust down while drilling and times, causing asthma due to very high 74 The Canadian Journal of Diagnosis / July 2001 Lung Disease respiratory irritant exposures (termed reactive airways dysfunction syndrome [RADS], or irritant-induced asthma).5,6 Besides causing asthma in some patients, workplace respiratory irritant exposures in accidental high levels (such as nitrogen oxides from silage, or spills of chlorine in chemical plants), can also induce other acute respiratory effects in any part of the respiratory tract.7 These can include acute respiratory distress syn- drome, pneumonitis, bronchiolitis, bron- chiolitis organizing pneumonia (BOOP), bronchiectasis, bronchitis, tracheitis, laryngitis and rhinitis. -
Thirty Workers in Missouri Popcorn Plant Diagnosed With
Occupational Lung SSSEEENNNSSSOOORRR Disease Bulletin Massachusetts Department of Public Health Occupational Health Surveillance Program, 250 Washington Street, 6th Floor, Boston, MA 02108 Tel: (617) 624-5632 Fax: (617) 624-5696 www.mass.gov/dph/ohsp June 2006 Dear Health Care Provider, An investigation of the workplace revealed that diacetyl, Welcome to the June issue of the SENSOR an artificial butter flavoring ingredient, was linked with the Occupational Lung Disease Bulletin! In this issue, we employees’ symptoms. Animal studies conducted by focus on the topic of bronchiolitis obliterans, a severe lung flavoring manufacturers as early as 1993 showed that disease that can occur in workers that make or handle inhalation of diacetyl resulted in severe lung injury. food flavoring. Bronchiolitis obliterans has been linked to Additional studies completed in 2002 confirmed these diacetyl, an artificial butter flavoring ingredient. The findings1. The National Institute for Occupational Safety illness has been called “popcorn lung disease” because of and Health (NIOSH) issued an alert in 2004, warning of the clusters of workers in popcorn plants that have been the danger posed by vapors, dusts or sprays of flavorings diagnosed with the disease. However, workers in plants and recommending controls to ensure worker safety in making food ranging from pastries and frozen food to plants using or making flavorings. NIOSH recommended nacho chips and candy may be exposed to the chemicals substituting less hazardous flavoring ingredients as the in food flavorings, and therefore may be at risk for best option for controlling health risks to workers. bronchiolitis obliterans. In addition, workers who use Additional options include local exhaust ventilation or, if diacetyl to produce artificial flavorings are also at risk. -
Occupational Lung Disease Bulletin
Occupational Lung Disease Bulletin Massachusetts Department of Public Health Winter 2017 The objective of the IARC is to identify hazards that are Dear Healthcare Provider, capable of increasing the incidence or severity of malignant neoplasms. The IARC classifications are not Dr. Neil Jenkins co-wrote this Occupational Lung Disease based on the probability that a carcinogen will cause a Bulletin, during his rotation at MDPH from Harvard School cancer or the dose-response, but rather indicate the of Public Health. He brought expertise in welding from a strength of the evidence that an agent can possibly cause materials science and occupational medicine background, a cancer. as well as involvement in welding oversight with the The IARC classifies the strength of the current evidence American Welding Society. that an agent is a carcinogen as: Remember to report cases of suspected work-related lung Group 1 Carcinogenic to humans disease to us by mail, fax (617) 624-5696 or phone (617) Group 2A Probably carcinogenic to humans 624-5632. The confidential reporting form is available on Group 2B Possibly carcinogenic to humans our website at www.mass.gov/dph/ohsp. Group 3 Not classifiable as to carcinogenicity To receive your Bulletin by e-mail, to provide comments, Group 4 Probably not carcinogenic to humans or to contribute an article to the Bulletin, contact us at [email protected] In 1989, welding fume was classified as Group 2B Elise Pechter MPH, CIH because of "limited evidence in humans" and "inadequate evidence in experimental animals." Since that time, an additional 20 case-control studies and nearly 30 cohort studies have provided evidence of increased risk of lung Welding—impact on occupational health cancer from welding fume exposure, even after Neil Jenkins and Elise Pechter accounting for asbestos and tobacco exposures.3 Studies of experimental animals provide added limited evidence 3 Welding joins metals together into a single piece by for lung carcinogenicity. -
International Statistical Classification of Diseases and Related Health Problems (ICD-10) in Occupational Health Protection of T
World Health Organization WHO/SDE/OEH/99.11 English only Sustainable Development and Distr.: Limited Healthy Environments International Statistical Classification of Diseases and Related Health Problems (ICD-10) in Occupational Health Protection of the Human Environment Occupational and Environmental Health Series Geneva, 1999 INTERNATIONAL STATISTICAL CLASSIFICATION OF DISEASES AND RELATED HEALTH PROBLEMS (ICD-10) IN OCCUPATIONAL HEALTH Antti Karjalainen Finnish Institute of Occupational Health World Health Organization Geneva 1999 WORLD HEALTH ORGANIZATION ¾ WHO/SDE/OEH/99.11 ¾ page i INTERNATIONAL STATISTICAL CLASSIFICATION OF DISEASES AND RELATED HEALTH PROBLEMS (ICD-10) Preface Classifications of occupational diseases have been developed mainly for two purposes: (1) notification for labour safety and health surveillance and (2) compensation. The absence of unified diagnostic criteria, coding systems and classifications reduce the compatibility and comparability of national statistics on occupational diseases. The main purpose of this document is to serve as a guideline for the use of ICD-10 in notification of occupational diseases in countries which do not have a well-established notification system. The document contains general guidelines for the use of ICD-10 codes and a comprehensive list of ICD-10 codes which are relevant for notification of occupational diseases. The list enables one to select, for each country, a selection of occupational disease entities that are the most relevant when building a notification system for that country. The document also provides typical examples of the causative agents/risk factors and risk industries/occupations for each occupational disease. It is to be underlined that these lists are meant to be only examples and should not be taken as exhaustive.