Epilepsy Monitoring Unit (EMU) Patient Information
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A Clinical Guide to Epileptic Syndromes and Their Treatment
Published August 13, 2008 as 10.3174/ajnr.A1231 dures and principles of treatment. These chapters are particu- BOOK REVIEW larly important for readers unfamiliar with the overall clinical presentation of the epilepsies and their diagnostic evaluation. A Clinical Guide to Epileptic Omitting these topics was a certain pitfall that has been Syndromes and Their Treatment, avoided. Basic techniques used in the evaluation of patients with of epilepsy, including anatomic and functional neuroim- 2nd ed. aging, are also included. The discussion of the relative value of C.P. Panayiotopoulos, ed. London, UK: Springer-Verlag; 2007, 578 EEG and its problems is particularly valuable. pages, 100 illustrations, $99.00. Subsequent chapters are devoted to the meat of the book, the epilepsy syndromes. These are presented in age-based he syndromic classification of epileptic seizures has been chronologic order, beginning with syndromes in the neonate evolving for more than a quarter century. Piggy-backed T and infant and continuing through syndromes in adults. onto major advances in neurophysiology and neuroimaging, Complete coverage of reflex seizures and reflex epilepsies in the current nomenclature of specific clinical epilepsy syn- chapter 16 is particularly welcome. Chapter 17 covers a group dromes now supersedes earlier seizure classifications based of disorders that are associated with epileptic seizures. The exclusively on seizure types. The practice of epilepsy has thus chapter gives excellent coverage of these disorders but makes joined mainstream medicine through its newfound ability to no pretenses at being comprehensive. For example, coverage link the signs and symptoms of epilepsy with specific under- of important disorders with epilepsy such as the Wolf- lying disorders. -
Myths and Truths About Pediatric Psychogenic Nonepileptic Seizures
Clin Exp Pediatr Vol. 64, No. 6, 251–259, 2021 Review article CEP https://doi.org/10.3345/cep.2020.00892 Myths and truths about pediatric psychogenic nonepileptic seizures Jung Sook Yeom, MD, PhD1,2,3, Heather Bernard, LCSW4, Sookyong Koh, MD, PhD3,4 1Department of Pediatrics, Gyeongsang National University Hospital, 2Gyeongsang Institute of Health Science, Gyeongsang National University College of Medicine, Jinju, Korea; 3Department of Pediatrics, Emory University School of Medicine, Atlanta, GA, USA; 4Department of Pediatrics, Children's Healthcare of Atlanta, Atlanta, GA, USA Psychogenic nonepileptic seizures (PNES) is a neuropsychiatric • PNES are a manifestation of psychological and emotional condition that causes a transient alteration of consciousness and distress. loss of self-control. PNES, which occur in vulnerable individuals • Treatment for PNES does not begin with the psychological who often have experienced trauma and are precipitated intervention but starts with the diagnosis and how the dia- gnosis is delivered. by overwhelming circumstances, are a body’s expression of • A multifactorial biopsychosocial process and a neurobiological a distressed mind, a cry for help. PNES are misunderstood, review are both essential components when treating PNES mistreated, under-recognized, and underdiagnosed. The mind- body dichotomy, an artificial divide between physical and mental health and brain disorders into neurology and psychi- atry, contributes to undue delays in the diagnosis and treat ment Introduction of PNES. One of the major barriers in the effective dia gnosis and treatment of PNES is the dissonance caused by different illness Psychogenic nonepileptic seizures (PNES) are paroxysmal perceptions between patients and providers. While patients attacks that may resemble epileptic seizures but are not caused are bewildered by their experiences of disabling attacks beyond by abnormal brain electrical discharges. -
Epilepsy Syndromes E9 (1)
EPILEPSY SYNDROMES E9 (1) Epilepsy Syndromes Last updated: September 9, 2021 CLASSIFICATION .......................................................................................................................................... 2 LOCALIZATION-RELATED (FOCAL) EPILEPSY SYNDROMES ........................................................................ 3 TEMPORAL LOBE EPILEPSY (TLE) ............................................................................................................... 3 Epidemiology ......................................................................................................................................... 3 Etiology, Pathology ................................................................................................................................ 3 Clinical Features ..................................................................................................................................... 7 Diagnosis ................................................................................................................................................ 8 Treatment ............................................................................................................................................. 15 EXTRATEMPORAL NEOCORTICAL EPILEPSY ............................................................................................... 16 Etiology ................................................................................................................................................ 16 -
Semiological Bridge Between Psychiatry and Epilepsy
Journal of Psychology and Clinical Psychiatry Semiological Bridge between Psychiatry and Epilepsy Abstract Review Article Epilepsy is a paroxysmal disturbance of brain function that presents as behavioral phenomena involving four spheres; sensory, motor, autonomic and consciousness. These behavioural disturbances though transient but may be Volume 8 Issue 1 - 2017 confused with psychiatric disorders. Thus representing a diagnostic problem Department of Neuropsychiatry, Ain Shams University, Egypt for neurophyschiatrists. Here we review the grey zone between psychiatry and epilepsy on three levels. The first level is the semiology itself, that the behavioral *Corresponding author: Ahmed Gaber, Prof. of phenomenon at a time can be the presentation of an epileptic disorder and at Neuropsychiatry, Ain Shams University, Cairo, Egypt, another time a representation of a psychiatric disorder. The second level is Email: the comorbidity between epilepsy and psychiatric disorders namely epileptic psychosis. The third level is the disorders of the brain that can present by both Received: | Published: epileptic and/or psychiatric disorders. We reviewed the current literature in both June 11, 2017 July 12, 2017 epilepsy and Psychiatry including the main presentations that might be confusing. Conclusion: Epilepsy, schizophrenia like psychosis, intellectual disability, autism are different disorders that may share same semiological presentation, comorbidity or even etiology. A stepwise mental approach and decision making is needed excluding seizure disorder first before diagnosing a psychiatric one. Keywords: Epilepsy, Psychosis; Schizophrenia; Semiology; Autoimmune encephalitis Discussion with consciousness are called dialeptic seizures. Seizures Epilepsy is a brain disorder characterized by an enduring consistingseizures are primarily identified of as autonomic auras. Seizures symptoms that interfere are called primarily either predisposition of recurrent seizures. -
Frequently Asked Questions
EPILEPSY AND SEIZURES – GENERAL Frequently Asked Questions What is epilepsy? Seizures are divided into two main categories: Epilepsy is a common neurological disease Generalized seizures characterized by the tendency to have recurrent • Involve both hemispheres of the brain seizures. It is sometimes called a seizure disorder. • Two common types are absence seizures (petit A person has epilepsy if they: mal seizures) and tonic-clonic seizures (grand mal seizures) • Have had at least two unprovoked seizures, or • Have had one seizure and are very likely to Focal seizures (partial seizures) have another, or • Only involve one part of the brain • Are diagnosed with an epilepsy syndrome • Include focal impaired awareness seizures (complex partial seizures) and focal aware seizures (simple partial seizures). What is a seizure? People with epilepsy may experience more than one A seizure is a sudden burst of electrical activity in the type of seizure. For more information about seizure brain that causes a temporary disturbance in the way types, see our Seizure Types Spark sheet. brain cells communicate with each other. The kind of seizure a person has depends on which part and how much of the brain is affected by the electrical FACT: About 1 in 100 Canadians have epilepsy. disturbance that produces the seizure. A seizure may take many different forms, including a blank stare, Why do people have seizures? uncontrolled movements, altered awareness, odd sensations, or convulsions. Seizures are typically brief There are many potential reasons why someone could and can last anywhere from a few seconds to a few have a seizure. Some seizures are a symptom of an minutes. -
Comprehensive Epilepsy Center the Only Level 4 Designated Center in Metropolitan Washington, D.C
Comprehensive Epilepsy Center The Only Level 4 Designated Center in Metropolitan Washington, D.C. 3 Seizing Control On the Road Again Daniele Wishnow endured eight Epilepsy affects nearly 3 million people years of medications and side- in the United States or about one out of every 100 Americans. Many effects, a major car accident and losing the ability to drive before spend years—often their entire lives—taking various medications for she finally discovered the epilepsy experts at MedStar Georgetown their disorder, usually with good results. But uncontrolled, epilepsy University Hospital. Months later, can limit an individual’s ability to drive, work or enjoy other activities. her life was back on track. “At my worst, I had a combination of grand mal seizures—losing However, there is hope. consciousness, collapsing and Neurosurgeon Chris Kalhorn, MD, jerking,” Daniele says, “along with performs delicate and intricate surgery multiple small seizures that made to control seizures. When traditional approaches fail, MedStar Georgetown University me blank out for a few seconds or so. For a long time, medications So on January 2013, Christopher Hospital can help. Our Comprehensive Epilepsy Center features kept them pretty much under Kalhorn, MD—director of experts who can accurately locate the precise area of the brain control, but then they quit functional, pediatric and epilepsy working.” neurosurgery—successfully causing seizures in both adults and children. And with the right removed Daniele’s lesion. Today, Daniele underwent evaluation at Daniele’s back on the road and diagnosis, we can tailor personalized treatment plans to reduce or MedStar Georgetown’s Level 4 back to living a full life. -
Psychogenic Nonepileptic Seizures: Diagnostic Challenges and Treatment Dilemmas Taoufik Alsaadi1* and Tarek M Shahrour2
Alsaadi and Shahrour. Int J Neurol Neurother 2015, 2:1 International Journal of DOI: 10.23937/2378-3001/2/1/1020 Volume 2 | Issue 1 Neurology and Neurotherapy ISSN: 2378-3001 Review Article: Open Access Psychogenic Nonepileptic Seizures: Diagnostic Challenges and Treatment Dilemmas Taoufik Alsaadi1* and Tarek M Shahrour2 1Department of Neurology, Sheikh Khalifa Medical City, UAE 2Department of Psychiatry, Sheikh Khalifa Medical City, UAE *Corresponding author: Taoufik Alsaadi, Department of Neurology, Sheikh Khalifa Medical City, UAE, E-mail: [email protected] They are thought to be a form of physical manifestation of psychological Abstract distress. Psychogenic Non-Epileptic Seizures (PNES) are grouped in Psychogenic Nonepileptic Seizures (PNES) are episodes of the category of psycho-neurologic illnesses like other conversion and movement, sensation or behavior changes similar to epileptic somatization disorders, in which symptoms are psychological in origin seizures but without neurological origin. They are somatic but neurologic in expression [4]. The purpose of this review is to shed manifestations of psychological distress. Patients with PNES are often misdiagnosed and treated for epilepsy for years, resulting in light on this common, but, often times, misdiagnosed problem. It has significant morbidity. Video-EEG monitoring is the gold standard for been estimated that approximately 20 to 30% of patients referred to diagnosis. Five to ten percent of outpatient epilepsy populations and epilepsy centers have PNES [5]. Still, it takes an average of 7 years before 20 to 40 percent of inpatient and specialty epilepsy center patients accurate diagnosis and appropriate referral is made [6]. Early recognition have PNES. These patients inevitably have comorbid psychiatric and appropriate treatment can prevent significant iatrogenic harm, and illnesses, most commonly depression, Post-Traumatic Stress Disorder (PTSD), other dissociative and somatoform disorders, may result in a better outcome. -
How to Identify Migraine with Aura Kathleen B
How to Identify Migraine with Aura Kathleen B. Digre MD, FAHS Migraine is very common—affecting 20% of women and (continuous dots that are present all of the time but do not almost 8% of men. Migraine with aura occurs in about obscure vision and appear like a faulty analog television set), one-third of those with migraine. Visual symptoms such as (Schankin et al), visual blurring and short visual phenomena photophobia, blurred vision, sparkles and flickering are all (Friedman). Migraine visual auras are usually stereotypic for reported in individuals with migraine. But how do you know if an individual. what a patient is experiencing is aura? Figure: Typical aura drawn by a primary care resident: The International Classification of Headache Disorders (ICHD 3) suggests that auras may be visual (most common—90% of all auras), sensory, speech and or language, motor, brainstem or retinal. The typical aura starts out gradually over 5 minutes and lasts 5-60 minutes, is usually unilateral and may be followed by a headache within 60 minutes. To identify aura, we rely on the patient’s description of the phenomena. One helpful question to ask patients to determine if they are experiencing visual aura is: Was this in one eye or both eyes? Many patients will report it in ONE eye but if they haven’t covered each eye when they have the phenomena and try to read text, they may be misled. If you can have them draw their aura (typical zig-zag lines) with scintillations (movement, like a kaleidoscope) across their vision, you know it is an aura. -
Epilepsy & My Child Toolkit
Epilepsy & My Child Toolkit A Resource for Parents with a Newly Diagnosed Child 136EMC Epilepsy & My Child Toolkit A Resource for Parents with a Newly Diagnosed Child “Obstacles are the things we see when we take our eyes off our goals.” -Zig Ziglar EPILEPSY & MY CHILD TOOLKIT • TABLE OF CONTENTS Introduction Purpose .............................................................................................................................................5 How to Use This Toolkit ....................................................................................................................5 About Epilepsy What is Epilepsy? ..............................................................................................................................7 What is a Seizure? .............................................................................................................................8 Myths & Facts ...................................................................................................................................8 How is Epilepsy Diagnosed? ............................................................................................................9 How is Epilepsy Treated? ..................................................................................................................10 Managing Epilepsy How Can You Help Manage Your Child’s Care? ...............................................................................13 What Should You Do if Your Child has a Seizure? ............................................................................14 -
Initiating an Epilepsy Surgery Program with Limited Resources in Indonesia
www.nature.com/scientificreports OPEN Initiating an epilepsy surgery program with limited resources in Indonesia Muhamad Thohar Arifn1*, Ryosuke Hanaya2, Yuriz Bakhtiar1, Aris Catur Bintoro3, Koji Iida4, Kaoru Kurisu4, Kazunori Arita2, Jacob Bunyamin1, Rofat Askoro1, Surya Pratama Brilliantika1, Novita Ikbar Khairunnisa1 & Zainal Muttaqin1 To share the experiences of organizing the epilepsy surgery program in Indonesia. This study was divided into two periods based on the presurgical evaluation method: the frst period (1999–2004), when interictal electroencephalogram (EEG) and magnetic resonance imaging (MRI) were used mainly for confrmation, and the second period (2005–2017), when long-term non-invasive and invasive video-EEG was involved in the evaluation. Long-term outcomes were recorded up to December 2019 based on the Engel scale. All 65 surgical recruits in the frst period possessed temporal lobe epilepsy (TLE), while 524 patients were treated in the second period. In the frst period, 76.8%, 16.1%, and 7.1% of patients with TLE achieved Classes I, II, and III, respectively, and in the second period, 89.4%, 5.5%, and 4.9% achieved Classes I, II, and III, respectively, alongside Class IV, at 0.3%. The overall median survival times for patients with focal impaired awareness seizures (FIAS), focal to bilateral tonic–clonic seizures and generalized tonic–clonic seizures were 9, 11 and 11 years (95% CI: 8.170– 9.830, 10.170–11.830, and 7.265–14.735), respectively, with p = 0.04. The utilization of stringent and selective criteria to reserve surgeries is important for a successful epilepsy program with limited resources. -
September | 2013 ISSN 1660-3656 Epileptologie | 30. Jahrgang
Epileptologie | 30. Jahrgang September | 2013 ISSN 1660-3656 Epilepsie-Liga Seefeldstrasse 84 Postfach 1084 CH-8034 Zürich Redaktionskommission Thomas Dorn | Zürich Reinhard E. Ganz | Zürich Martinus Hauf | Tschugg Hennric Jokeit | Zürich Christian M. Korff | Genève Günter Krämer | Zürich (Vorsitz) Inhalt Oliver Maier | St. Gallen Andrea O. Rossetti | Lausanne Stephan Rüegg | Basel Kaspar Schindler | Bern Editorial 147 – 149 Serge Vulliémoz | Genève Combination of Electroencephalographic and Magnetic Resonance Imaging to Beirat Characterize Epileptic Networks Christoph M. Michel 150 – 157 Alexandre Datta | Basel Thomas Grunwald | Zürich Structure, Function and Genes: Christian W. Hess | Bern What Can We Learn from MRI Anna Marie Hew-Winzeler | Zürich Giorgi Kuchukhidze, Eugen Trinka 158 – 166 Günter Krämer | Zürich Theodor Landis | Genève Hereditäre epileptische Enzephalopathie Malin Maeder | Lavigny mit Amelogenesis imperfecta Klaus Meyer | Tschugg Kohlschütter-Tönz-Syndrom Christoph Michel | Genève (KTZS). Bericht über einen weiteren, Christoph Pachlatko | Zürich posthum erfassten Patienten aus der Monika Raimondi | Lugano Zentralschweiz. Andrea O. Rossetti | Lausanne Otmar Tönz, Bernhard Steiner, Anna Stephan Rüegg | Basel Schossig, Johannes Zschocke, Markus Schmutz | Basel Alfried Kohlschütter 167– 174 Margitta Seeck | Genève Urs Sennhauser | Hettlingen Epilepsie-Liga-Mitteilungen 175 – 198 Franco Vassella | Bremgarten Kongresskalender 199 – 200 Schweizerische Liga gegen Epilepsie Ligue Suisse contre l’Epilepsie Lega Svizzera contro l’Epilessia Swiss League Against Epilepsy Richtlinien für die Autoren Allgemeines - Zusammenfassung, Résumé und englischer Ab- stract (mit Titel der Arbeit): Ohne Literaturzitate Epileptologie veröffentlicht sowohl angeforderte als und Akronyme sowie unübliche Abkürzungen (je auch unaufgefordert eingereichte Manuskripte über maximal 250 Wörter). alle Themen der Epileptologie. Es werden in der Regel - Text: Dabei bei Originalarbeiten Gliederung in Ein- nur bislang unveröffentlichte Arbeiten angenommen. -
Age of Onset in Idiopathic (Genetic) Generalized Epilepsies: Clinical and EEG
View metadata, citation and similar papers at core.ac.uk brought to you by CORE provided by Elsevier - Publisher Connector Seizure 21 (2012) 417–421 Contents lists available at SciVerse ScienceDirect Seizure jou rnal homepage: www.elsevier.com/locate/yseiz Age of onset in idiopathic (genetic) generalized epilepsies: Clinical and EEG findings in various age groups a,b, a b Ali A. Asadi-Pooya *, Mehrdad Emami , Michael R. Sperling a Neurosciences Research Center, School of Medicine, Shiraz University of Medical Sciences, Shiraz, Iran b Jefferson Comprehensive Epilepsy Center, Department of Neurology, Thomas Jefferson University, Philadelphia, USA A R T I C L E I N F O A B S T R A C T Article history: Purpose: The prevalence and differences of idiopathic (genetic) generalized epilepsies (IGEs) with Received 5 February 2012 atypical age of onset compared to classical IGEs is a matter of debate. We tried to determine the clinical Received in revised form 11 April 2012 and EEG characteristics of IGEs in various age groups. Accepted 11 April 2012 Methods: All patients with a clinical diagnosis of IGE were recruited at the outpatient epilepsy clinic at Shiraz University of Medical Sciences from 2008 through 2011. We subdivided the patients into four Keywords: different age groups: 4 years of age and under, 5–11 years, 12–17 years, and finally, 18 years and above, Idiopathic generalized epilepsy at the time of their epilepsy onset. Syndromic diagnosis, sex ratio, seizure types and EEG findings were Age of onset compared. Statistical analyses were performed using Pearson Chi square test. Seizure type EEG Results: 2190 patients with epilepsy were registered.