Postpartum Hemorrhage: Prevention and Treatment ANN EVENSEN, MD, University of Wisconsin School of Medicine and Public Health, Madison, Wisconsin JANICE M

Total Page:16

File Type:pdf, Size:1020Kb

Postpartum Hemorrhage: Prevention and Treatment ANN EVENSEN, MD, University of Wisconsin School of Medicine and Public Health, Madison, Wisconsin JANICE M This is an updated version of the article that appeared in print. Postpartum Hemorrhage: Prevention and Treatment ANN EVENSEN, MD, University of Wisconsin School of Medicine and Public Health, Madison, Wisconsin JANICE M. ANDERSON, MD, Forbes Family Medicine Residency Program, Pittsburgh, Pennsylvania PATRICIA FONTAINE, MD, MS, HealthPartners Institute for Education and Research, Bloomington, Minnesota Postpartum hemorrhage is common and can occur in patients without risk factors for hemorrhage. Active man- agement of the third stage of labor should be used routinely to reduce its incidence. Use of oxytocin after delivery of the anterior shoulder is the most important and effective component of this practice. Oxytocin is more effective than misoprostol for prevention and treatment of uterine atony and has fewer adverse effects. Routine episiotomy should be avoided to decrease blood loss and the risk of anal laceration. Appropriate management of postpartum hemorrhage requires prompt diagnosis and treatment. The Four T’s mnemonic can be used to identify and address the four most common causes of postpartum hemorrhage (uterine atony [Tone]; laceration, hematoma, inversion, rupture [Trauma]; retained tissue or invasive placenta [Tissue]; and coagulopathy [Thrombin]). Rapid team-based care minimizes morbidity and mortality associated with postpartum hemorrhage, regardless of cause. Massive trans- fusion protocols allow for rapid and appropriate response to hemorrhages exceeding 1,500 mL of blood loss. The National Partnership for Maternal Safety has developed an obstetric hemorrhage consensus bundle of 13 patient- and systems-level recommendations to reduce morbidity and mortality from postpartum hemorrhage. (Am Fam Physi- cian. 2017;95(7):442-449. Copyright © 2017 American Academy of Family Physicians.) More online pproximately 3% to 5% of obstet- Prevention at http://www. ric patients will experience post- Risk factors for postpartum hemorrhage are aafp.org/afp. partum hemorrhage.1 Annually, listed in Table 2.8 However, 20% of postpar- CME This clinical content these preventable events are tum hemorrhage occurs in women with no conforms to AAFP criteria Athe cause of one-fourth of maternal deaths risk factors, so physicians must be prepared for continuing medical 9 education (CME). See worldwide and 12% of maternal deaths in to manage this condition at every delivery. CME Quiz Questions on the United States.2,3 The American College Strategies for decreasing the morbidity and page 417. of Obstetricians and Gynecologists defines mortality associated with postpartum hem- Author disclosure: No rel- early postpartum hemorrhage as at least orrhage are listed in Table 3, 6,10-14 including evant financial affiliations. 1,000 mL total blood loss or loss of blood the choice to deliver infants in women at coinciding with signs and symptoms of high risk of hemorrhage at facilities with hypovolemia within 24 hours after delivery immediately available surgical, intensive of the fetus or intrapartum loss.4,5 Primary care, and blood bank services. postpartum hemorrhage may occur before The most effective strategy to prevent delivery of the placenta and up to 24 hours postpartum hemorrhage is active manage- after delivery of the fetus. Complications ment of the third stage of labor (AMTSL). Scan the QR code below of postpartum hemorrhage are listed in AMTSL also reduces the risk of a postpar- with your mobile device Table 13,6,7; these range from worsening of tum maternal hemoglobin level lower than for easy access to the common postpartum symptoms such as 9 g per dL (90 g per L) and the need for man- patient information hand- fatigue and depressed mood, to death from ual removal of the placenta.11 Components of out on the AFP mobile site. cardiovascular collapse. this practice include: (1) administering oxy- This review presents evidence-based rec- tocin (Pitocin) with or soon after the deliv- ommendations for the prevention of and ery of the anterior shoulder; (2) controlled appropriate response to postpartum hem- cord traction (Brandt-Andrews maneuver) orrhage and is intended for physicians who to deliver the placenta; and (3) uterine mas- provide antenatal, intrapartum, and post- sage after delivery of the placenta.11 Pla- partum care. cental delivery can be achieved using the 442Downloaded American from the Family American Physician Family Physician website at www.aafp.org/afp.www.aafp.org/afp Copyright © 2017 American Academy of VolumeFamily Physicians. 95, Number For the 7 private,◆ April noncom 1, 2017- mercial use of one individual user of the website. All other rights reserved. Contact [email protected] for copyright questions and/or permission requests. Postpartum Hemorrhage Brandt-Andrews maneuver, in which firm traction on cord traction does not prevent severe postpartum hem- the umbilical cord is applied with one hand while the orrhage, but reduces the incidence of less severe blood other applies suprapubic counterpressure15 (eFigure A). loss (500 to 1,000 mL) and reduces the need for manual The individual components of AMTSL have been evalu- extraction of the placenta.21 ated and compared. Based on existing evidence, the most important component is administration of a uterotonic Diagnosis and Management drug, preferably oxytocin.12,16 The number needed to treat Diagnosis of postpartum hemorrhage begins with rec- to prevent one case of hemorrhage 500 mL or greater is 7 ognition of excessive bleeding and targeted examina- for oxytocin administered after delivery of the fetal ante- tion to determine its cause (Figure 16). Cumulative blood rior shoulder or after delivery of the neonate compared loss should be monitored throughout labor and deliv- with placebo.16 The risk of postpartum hemorrhage is also ery and postpartum with quantitative measurement, if reduced if oxytocin is administered after placental deliv- ery instead of at the time of delivery of the anterior shoul- 17 6 der. Dosing instructions are provided in Table 4. Table 3. Strategies to Reduce Morbidity and An alternative to oxytocin is misoprostol (Cytotec), an Mortality from Postpartum Hemorrhage inexpensive medication that does not require injection and is more effective than placebo in preventing postpar- Readiness by every unit 12 tum hemorrhage. However, most studies have shown Have a hemorrhage cart with medications, supplies, checklist, that oxytocin is superior to misoprostol.12,18 Misoprostol and instruction cards immediately available also causes more adverse effects than oxytocin—com- Establish a response team and know who to call when help is monly nausea, diarrhea, and fever within three hours of needed birth.12,18 Establish massive and emergency release transfusion protocols The benefits of controlled cord traction and uterine Institute unit education on protocols and run unit-based drills massage in preventing postpartum hemorrhage are less Recognition and prevention efforts for every patient clear, but these strategies may be helpful.15,19,20 Controlled Antenatal assessment Screen for and treat anemia antenatally Screen for sickle cell disease and thalassemia in women of African, Southeast Asian, or Mediterranean descent Table 1. Complications of Postpartum Obtain sonograms for women at high risk of invasive placenta Hemorrhage Perform delivery in facility with blood bank and in-house surgical services if the patient has a high risk of hemorrhage Anemia Death Identify Jehovah’s Witnesses and other patients who decline Anterior pituitary ischemia Dilutional coagulopathy blood products with delay or failure of Fatigue Intrapartum management lactation (i.e., Sheehan Myocardial ischemia Use active management of the third stage of labor in every syndrome or postpartum delivery pituitary necrosis) Orthostatic hypotension Avoid routine episiotomy Blood transfusion Postpartum depression Avoid instrumented deliveries, especially forceps Information from references 3, 6, and 7. Use perineal warm compresses Measure cumulative blood loss and track postpartum vital signs Response for every hemorrhage Use an emergency management plan with checklists Table 2. Risk Factors for Postpartum Provide support program for patients, families, and staff Hemorrhage Reporting and systems learning for every unit Establish a culture of huddles and postevent debriefs Antepartum hemorrhage Maternal obesity Complete a multidisciplinary review for systems issues Augmented labor Multifetal gestation Establish a perinatal quality improvement committee Chorioamnionitis Preeclampsia Adapted with permission from Council on Patient Safety in Women’s Fetal macrosomia Primiparity Health Care. Obstetric hemorrhage patient safety bundle. http:// Maternal anemia Prolonged labor safehealthcareforeverywoman.org/patient-safety-bundles/obstetric- hemorrhage/ [login required]. Accessed October 16, 2016. Additional Information from reference 8. information from references 6, and 11 through 14. April 1, 2017 ◆ Volume 95, Number 7 www.aafp.org/afp American Family Physician 443 Postpartum Hemorrhage Table 4. Medications Used for Prevention and Treatment of Postpartum Hemorrhage Medication Dosage Prevention Treatment Contraindications and cautions Mechanism of action Adverse effects Cost* First-line agent Oxytocin (Pitocin) Prevention: 10 IU IM or 5 to + + Overdose or prolonged use can cause water Stimulates the upper segment of the Rare $1 ($13) for 10 IU IV bolus intoxication myometrium to contract rhythmically, 10 units of Treatment: 20 to 40 IU in 1 L Possible hypotension with IV use following
Recommended publications
  • OB/GYN EMERGENCIES Elyse Watkins, Dhsc, PA-C, DFAAPA DISCLOSURES
    OB/GYN EMERGENCIES Elyse Watkins, DHSc, PA-C, DFAAPA DISCLOSURES I have no financial relationships to disclose. TOPICS Ovarian torsion Postpartum hemorrhage Ruptured ectopic Acute uterine inversion pregnancy Amniotic fluid embolism Acute menorrhagia Placental abruption OVARIAN TORSION OVARIAN TORSION .Tumors (benign and malignant) are implicated in 50-60% of cases of torsion .20% occur during pregnancy (corpus luteum cyst) .Unilateral or bilateral abdominal-pelvic pain, usually sudden onset .Exercise or movement exacerbates pain .Nausea and vomiting 70% .Pathophys: reduced venous return, stromal edema, internal hemorrhage, and infarction → necrosis OVARIAN TORSION .Physical exam variable .Ultrasonography with color Doppler .Surgical referral RUPTURED ECTOPIC PREGNANCY RUPTURED ECTOPIC PREGNANCY .All patients of reproductive age with a hx of missed menses and pelvic pain should be considered to have an ectopic pregnancy until proven otherwise. .A patient with missed menses, irregular vaginal bleeding, pelvic pain, syncope, abdominal pain, and/or dizziness should be managed as a ruptured ectopic pregnancy until proven otherwise. RUPTURED ECTOPIC PREGNANCY .Physical exam of pts with a ruptured ectopic can reveal pelvic tenderness, an adnexal mass, and evidence of hemodynamic compromise. .A transvaginal ultrasound will often show an adnexal mass and/or fluid in the pouch of Douglas. .The serum qualitative βHCG will be > 5 mIu/mL. RUPTURED ECTOPIC PREGNANCY HTTPS://YOUTU.BE/TNN1FPWHOXS RUPTURED ECTOPIC PREGNANCY .Immediately order an H/H, type and cross, and place large bore IV access for fluid support. .Laparotomy is performed when patients are hemodynamically unstable or if visualization during laparoscopy was difficult. .Patients with a ruptured ectopic pregnancy must be managed emergently and surgically! ACUTE MENORRHAGIA ACUTE MENORRHAGIA .Abnormal uterine bleeding (AUB) can result in acute blood loss that causes hemodynamic compromise so prompt evaluation of vital signs is important.
    [Show full text]
  • Placental Abruption
    Placental Abruption Definition: Placental separation, either partial or complete prior to the birth of the fetus Incidence 0.5 – 1% (4). Risk factors include hypertension, smoking, preterm premature rupture of membranes, cocaine abuse, uterine myomas, and previous abruption (5). Diagnosis: Symptoms (may present with any or all of these) o Vaginal bleeding (usually dark and non-clotting). o Abdominal pain and/or back pain varying from intermittent to severe. o Uterine contractions are usually present and may vary from low amplitude/high frequency to hypertonus. o Fetal distress or fetal death. Ultrasound o Adherent retroplacental clot OR may just appear to be a thick placenta o Resolving hematomas become hypoechoic within one week and sonolucent within 2 weeks May be a diagnosis of exclusion if vaginal bleeding and no other identified etiology Consider in differential with uterine irritability on toco and cat II-III tracing, as small proportion present without bleeding Classification: Grade I: Slight vaginal bleeding and some uterine irritability are usually present. Maternal blood pressure, and fibrinogen levels are unaffected. FHR remains normal. Grade II: Mild to moderate vaginal bleeding seen; tetanic contractions may be present. Blood pressure usually normal, but tachycardia may be present. May be postural hypotension. Decreased fibrinogen; with levels below 250 mg percent; may be evidence of fetal distress. Reviewed 01/16/2020 1 Updated 01/16/2020 Grade III: Bleeding is moderate to severe, but may be concealed. Uterus tetanic and painful. Maternal hypotension usually present. Fetal death has occurred. Fibrinogen levels are less then 150 mg percent with thrombocytopenia and coagulation abnormalities.
    [Show full text]
  • Uterine Atony: Uterus Soft and Relaxed
    Uterine atony: uterus soft and relaxed Placenta not delivered Treat for whole retained placenta If whole placenta still retained ■ Oxytocin ■ Manual removal with prophylactic antibiotics ■ Controlled cord traction ■ Intraumbilical vein injection (if no bleeding) Placenta delivered incomplete Treat for retained placenta fragments If bleeding continues ■ Oxytocin ■ Manage as uterine atony ■ Manual exploration to remove fragments ■ Gentle curettage or aspiration Be ready at all times to transfer to a higher-level facility if the Lower genital tract trauma: Treat for lower genital tract trauma If bleeding continues patient is not responding to the excessive bleeding or shock ■ Repair of tears ■ Tranexamic acid ■ treatment or a treatment cannot contracted uterus Evacuation and repair of haematoma be administered at your facility. Uterine rupture or dehiscence: Treat for uterine rupture or dehiscence If bleeding continues excessive bleeding or shock ■ Laparotomy for primary repair of uterus ■ Tranexamic acid Start intravenous oxytocin infusion ■ Hysterectomy if repair fails and consider: • uterine massage; • bimanual uterine compression; Uterine inversion: Treat for uterine inversion If laparotomy correction not successful • external aortic compression; and uterine fundus not felt ■ Immediate manual replacement ■ Hysterectomy • balloon or condom tamponade. abdominally or visible in vagina ■ Hydrostatic correction ■ Manual reverse inversion Transfer with ongoing intravenous (use general anaesthesia or wait for effect uterotonic infusion. Accompanying
    [Show full text]
  • Uterine Rupture After Misoprostol Use for Termination of Pregnancy in Second Trimester with Previous Caesarean Section: a Case Report Marjan Ghaemi*
    Case Report iMedPub Journals Critical Care Obstetrics and Gynecology 2018 http://www.imedpub.com/ Vol.4 No.3:14 ISSN 2471-9803 DOI: 10.21767/2471-9803.1000167 Uterine Rupture after Misoprostol Use for Termination of Pregnancy in Second Trimester with Previous Caesarean Section: A Case Report Marjan Ghaemi* Yas Hospital, Tehran University of Medical Sciences, Tehran, Iran *Corresponding author: Marjan Ghaemi, Yas Hospital, Tehran University of Medical Sciences, Tehran, Iran, Tel: 0098-912-1967735; E-mail: [email protected] Received date: September 25, 2018; Accepted date: October 16, 2018; Published date: October 22, 2018 Copyright: © 2018 Ghaemi M. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Citation: Ghaemi M (2018) Uterine Rupture after Misoprostol Use for Termination of Pregnancy in Second Trimester with Previous Caesarean Section: A Case Report. Crit Care Obst Gyne Vol.4 No.3:14. cesarean section, hospitalized for severe oligohydramnios with unknown etiology and no history of fluid leakage. In her Abstract previous surgical history, she mentioned laparoscopic cholecystectomy 10 years ago. Considering her medical history Modalities for termination of pregnancy may result in some and normal kidney and bladder in fetal ultrasound, we did not adverse effects especially uterine rupture in women with have proved data for her severe oligohydramnios. Amnio- the previous cesarean section. Here we report uterine infusion was performed to prevent foetal cord compression and rupture in the 23rd week of pregnancy after Misoprostol uses for abortion induction in second pregnancy with the to assess foetal structures.
    [Show full text]
  • Management of Prolonged Decelerations ▲
    OBG_1106_Dildy.finalREV 10/24/06 10:05 AM Page 30 OBGMANAGEMENT Gary A. Dildy III, MD OBSTETRIC EMERGENCIES Clinical Professor, Department of Obstetrics and Gynecology, Management of Louisiana State University Health Sciences Center New Orleans prolonged decelerations Director of Site Analysis HCA Perinatal Quality Assurance Some are benign, some are pathologic but reversible, Nashville, Tenn and others are the most feared complications in obstetrics Staff Perinatologist Maternal-Fetal Medicine St. Mark’s Hospital prolonged deceleration may signal ed prolonged decelerations is based on bed- Salt Lake City, Utah danger—or reflect a perfectly nor- side clinical judgment, which inevitably will A mal fetal response to maternal sometimes be imperfect given the unpre- pelvic examination.® BecauseDowden of the Healthwide dictability Media of these decelerations.” range of possibilities, this fetal heart rate pattern justifies close attention. For exam- “Fetal bradycardia” and “prolonged ple,Copyright repetitive Forprolonged personal decelerations use may onlydeceleration” are distinct entities indicate cord compression from oligohy- In general parlance, we often use the terms dramnios. Even more troubling, a pro- “fetal bradycardia” and “prolonged decel- longed deceleration may occur for the first eration” loosely. In practice, we must dif- IN THIS ARTICLE time during the evolution of a profound ferentiate these entities because underlying catastrophe, such as amniotic fluid pathophysiologic mechanisms and clinical 3 FHR patterns: embolism or uterine rupture during vagi- management may differ substantially. What would nal birth after cesarean delivery (VBAC). The problem: Since the introduction In some circumstances, a prolonged decel- of electronic fetal monitoring (EFM) in you do? eration may be the terminus of a progres- the 1960s, numerous descriptions of FHR ❙ Complete heart sion of nonreassuring fetal heart rate patterns have been published, each slight- block (FHR) changes, and becomes the immedi- ly different from the others.
    [Show full text]
  • Retained Placenta and Postpartum Haemorrhage
    Digital Comprehensive Summaries of Uppsala Dissertations from the Faculty of Medicine 1077 Retained Placenta and Postpartum Haemorrhage JOHANNA BELACHEW ACTA UNIVERSITATIS UPSALIENSIS ISSN 1651-6206 ISBN 978-91-554-9182-6 UPPSALA urn:nbn:se:uu:diva-246185 2015 Dissertation presented at Uppsala University to be publicly examined in Rosénsalen, Ing 95/96, Akademiska sjukhuset, Uppsala, Thursday, 23 April 2015 at 13:15 for the degree of Doctor of Philosophy (Faculty of Medicine). The examination will be conducted in Swedish. Faculty examiner: Professor Kjell Å Salvesen (Lunds Universitet). Abstract Belachew, J. 2015. Retained Placenta and Postpartum Haemorrhage. Digital Comprehensive Summaries of Uppsala Dissertations from the Faculty of Medicine 1077. 59 pp. Uppsala: Acta Universitatis Upsaliensis. ISBN 978-91-554-9182-6. The aim was to explore the possibility to diagnose retained placental tissue and other placental complications with 3D ultrasound and to investigate the impact of previous caesarean section on placentation in forthcoming pregnancies. 3D ultrasound was used to measure the volumes of the uterine body and cavity in 50 women with uncomplicated deliveries throughout the postpartum period. These volumes were then used as reference, to diagnose retained placental tissue in 25 women with secondary postpartum haemorrhage. All but three of the 25 women had retained placental tissue confirmed at histopathology. The volume of the uterine cavity in women with retained placental tissue was larger than the reference in most cases, but even cavities with no retained placental tissue were enlarged (Studies I and II). Women with their first and second birth, recorded in the Swedish medical birth register, were studied in order to find an association between previous caesarean section and retained placenta.
    [Show full text]
  • Concurrent Intraoperative Uterine Rupture and Placenta Accreta. Do
    Concurrent intraoperative uterine rupture and placenta accreta Romanian Journal of Anaesthesia and Intensive Care 2018 Vol 25 No 1, 83-85 CASE REPORT DOI: http://dx.doi.org/10.21454/rjaic.7518.251.acc Concurrent intraoperative uterine rupture and placenta accreta. Do preoperative chronic hypertension, preterm premature rupture of membranes, chorioamnionitis, and placental abruption provide warning to this rare occurrence? M. Anthony Cometa, Scott M. Wasilko, Adam L. Wendling Department of Anesthesiology, Division of Obstetric Anesthesiology, University of Florida College of Medicine, Gainesville, FL, USA Abstract Uterine and placental pathology can be a major cause of morbidity and mortality in the parturient and infant. When presenting alone, placental abruption, uterine rupture, or placenta accreta can result in significant peripartum hemorrhage, requiring aggressive surgical and anesthetic management; however, the presence of multiple concurrent uterine and placental pathologies can result in significant morbidity and mortality. We present the anesthetic management of a parturient who underwent an urgent cesarean delivery for non- reassuring fetal tracing in the setting of chronic hypertension, preterm premature rupture of membranes, and chorioamnionitis who was subsequently found to have placental abruption, uterine rupture, and placenta accreta. Keywords: preterm premature rupture of membranes (PPROM), chorioamnionitis, placental abruption, uterine rupture, placenta accrete, cesarean-hysterectomy Received: 14 March 2018 / Accepted: 30 March 2018 Rom J Anaesth Intensive Care 2018; 25: 83-85 operative bleeding that requires aggressive fluid and blood product management and resuscitation [5]. At present, the literature does not reference the Introduction presentation and anesthetic management of these aforementioned conditions being concurrently present Various placental and uterine pathologies can result in one parturient.
    [Show full text]
  • OBGYN-Study-Guide-1.Pdf
    OBSTETRICS PREGNANCY Physiology of Pregnancy: • CO input increases 30-50% (max 20-24 weeks) (mostly due to increase in stroke volume) • SVR anD arterial bp Decreases (likely due to increase in progesterone) o decrease in systolic blood pressure of 5 to 10 mm Hg and in diastolic blood pressure of 10 to 15 mm Hg that nadirs at week 24. • Increase tiDal volume 30-40% and total lung capacity decrease by 5% due to diaphragm • IncreaseD reD blooD cell mass • GI: nausea – due to elevations in estrogen, progesterone, hCG (resolve by 14-16 weeks) • Stomach – prolonged gastric emptying times and decreased GE sphincter tone à reflux • Kidneys increase in size anD ureters dilate during pregnancy à increaseD pyelonephritis • GFR increases by 50% in early pregnancy anD is maintaineD, RAAS increases = increase alDosterone, but no increaseD soDium bc GFR is also increaseD • RBC volume increases by 20-30%, plasma volume increases by 50% à decreased crit (dilutional anemia) • Labor can cause WBC to rise over 20 million • Pregnancy = hypercoagulable state (increase in fibrinogen anD factors VII-X); clotting and bleeding times do not change • Pregnancy = hyperestrogenic state • hCG double 48 hours during early pregnancy and reach peak at 10-12 weeks, decline to reach stead stage after week 15 • placenta produces hCG which maintains corpus luteum in early pregnancy • corpus luteum produces progesterone which maintains enDometrium • increaseD prolactin during pregnancy • elevation in T3 and T4, slight Decrease in TSH early on, but overall euthyroiD state • linea nigra, perineum, anD face skin (melasma) changes • increase carpal tunnel (median nerve compression) • increased caloric need 300cal/day during pregnancy and 500 during breastfeeding • shoulD gain 20-30 lb • increaseD caloric requirements: protein, iron, folate, calcium, other vitamins anD minerals Testing: In a patient with irregular menstrual cycles or unknown date of last menstruation, the last Date of intercourse shoulD be useD as the marker for repeating a urine pregnancy test.
    [Show full text]
  • A Rare Case of Placenta Percreta Causing Uterine Rupture and Massive Haemoperitoneum in 2Nd Trimester: an Obvious Near Miss
    International Journal of Reproduction, Contraception, Obstetrics and Gynecology Parmar C et al. Int J Reprod Contracept Obstet Gynecol. 2021 Jun;10(6):2495-2497 www.ijrcog.org pISSN 2320-1770 | eISSN 2320-1789 DOI: https://dx.doi.org/10.18203/2320-1770.ijrcog20212201 Case Report A rare case of placenta percreta causing uterine rupture and massive haemoperitoneum in 2nd trimester: an obvious near miss Chirayu Parmar*, Mittal Parmar, Gayatri Desai Department of Gynecology, Kasturba Hospital, SEWA Rural, Jhagadi, Gujarat, India Received: 30 December 2020 Accepted: 05 May 2021 *Correspondence: Dr. Chirayu Parmar, E-mail: [email protected] Copyright: © the author(s), publisher and licensee Medip Academy. This is an open-access article distributed under the terms of the Creative Commons Attribution Non-Commercial License, which permits unrestricted non-commercial use, distribution, and reproduction in any medium, provided the original work is properly cited. ABSTRACT Placenta accreta spectrum is very rarely encountered with ruptured uterus and is commonly seen in third trimester of pregnancy. Hereby, a case of placenta percreta with uterine rupture in second trimester of pregnancy is presented. 40 year old women with previous 2 LSCS presented in emergency department with ninteen weeks pregnancy and massive haemoperitoneum. Emergency laprotomy revealed uterine rupture alnong with placenta percreta for which obstetric hysterectomy was done. Although, a rare occurrence, obstetricians should consider patients placenta accreta spectrum in
    [Show full text]
  • Reduction of Subacute Uterine Inversion by Haultain's Method: A
    This open-access article is distributed under Creative Commons licence CC-BY-NC 4.0. CASE REPORT Reduction of subacute uterine inversion by Haultain’s method: A case report E Ziki,1 MB ChB, MMed; S Madombi,1 MB ChB; C Chidhakwa,1 FCOG; M G Madziyire,1 MMed; N Zakazaka,2 MMed 1 Department of Obstetrics and Gynaecology, University of Zimbabwe, College of Health Sciences, Harare, Zimbabwe 2 Department of Obstetrics and Gynaecology, Parirenyatwa Central Hospital, Harare, Zimbabwe Corresponding author: E Ziki ([email protected]) Uterine inversion is a rare but potentially life-threatening obstetric emergency of unknown aetiology, which is often associated with inadvertent traction on the umbilical cord before separation of the placenta. Here we report a case of a 26-year-old woman who presented with a day’s history of uterine inversion after an attempt to remove a retained placenta following a second-trimester miscarriage. Reduction was attempted in casualty without success and she was taken to theatre for surgical reduction under anaesthesia. Reduction was eventually achieved using the Haultain method. S Afr J Obstet Gynaecol 2017;23(3):78-79. DOI:10.7196/SAJOG.2017.v23i3.1274 Uterine inversion is defined as the ‘turning inside out of the fundus of subacute uterine inversion was made and resuscitation was into the uterine cavity’ and the incidence is about 1 in 3 737.[1] performed using ringer’s lactate fluid and 4 units of packed red Active management of the third stage of labour resulted in a 4.4- blood cells. She was commenced on intravenous antibiotics.
    [Show full text]
  • Treating Postpartum Hemorrhage and Invasive Placenta Endovascular Techniques to Improve Outcomes in Patients with Abnormal Invasive Placenta
    WOMEN’S HEALTH Treating Postpartum Hemorrhage and Invasive Placenta Endovascular techniques to improve outcomes in patients with abnormal invasive placenta. BY ANDREW C. PICEL, MD; RORY L. COCHRAN, PHD; AND KARI J. NELSON, MD ostpartum hemorrhage is associated with a signifi- ABNORMAL INVASIVE PLACENTA cant risk of maternal morbidity and mortality. The The AIP disease spectrum is defined by the depth of American College of Obstetricians and Gynecologists placental invasion into the uterine wall. Placenta accreta recently defined primary postpartum hemorrhage refers to trophoblastic attachment to the myometrium, Pas blood loss ≥ 1,000 mL or blood loss accompanied by whereas placenta increta occurs when there is penetration signs or symptoms of hypovolemia within 24 hours after into the myometrium. Placenta percreta is the most severe birth.1 Poor uterine tone accounts for 70% to 80% of pri- form, with invasion into the serosa and surrounding viscera.3 mary postpartum hemorrhage.1,2 Other causes of primary Increasing rates of uterine interventions, particularly cesarean hemorrhage include lacerations, retained placental tissue, deliveries, are resulting in a rapidly increasing incidence of invasive placenta, uterine inversion, and coagulation defects. invasive placenta, which is now occurring in approximately Secondary postpartum hemorrhage may be caused by 1 in 533 pregnancies.4 subinvolution of the placental site, retained products of AIP imparts a high risk of obstetric hemorrhage, surgical conception, infection, and coagulation defects.1 Clinically, injury, morbidity, and mortality. The invasive placenta does primary postpartum hemorrhage is the most common and not typically separate from the uterine wall and may invade most frequently encountered form of postpartum hemor- surrounding structures, resulting in a complex operation rhage seen in interventional radiology.
    [Show full text]
  • Simmomtm Birthing Simulation Solutions
    Improving maternal and neonatal care SimMomTM Birthing Simulation Solutions www.laerdal.com/simmom Innovative simulation Improving patient safety Preventing adverse outcomes during birth Simulation has gathered increasing acceptance over the Simulation training is unique in its ability to facilitate effective years as an integral part of healthcare training and as a team training. By bringing together multi-disciplinary fundamental approach to help improve patient safety. healthcare professionals in a simulation to rehearse both common birthing scenarios and emergency critical incidents; In the field of obstetrics, research into suboptimal patient early recognition of birth complications, correct diagnosis outcomes have identified the following key contributory and rapid, coordinated action from the delivery team can factors: confusion in roles and responsibilities; lack of cross be perfected for improved outcomes. monitoring; failure to prioritize and perform clinical tasks in a structured, coordinated manner; poor communication and lack of organizational support. Reference : The Joint Commission, 2004, Draycott et al. 2009 2 Simulation by Laerdal and Limbs & Things SimMomTM A complete solution A progressive partnership SimMomTM is an advanced full-body birthing simulator with By integrating the strengths of the PROMPT birthing accurate anatomy and functionality. SimMom has been simulator from Limbs & Things with the ALS Simulator developed to provide you with a comprehensive simulation from Laerdal, SimMom provides anatomical accuracy and solution to support multi-disciplined staff in obstetric and authentic simulation experiences that together, facilitate midwifery care, enabling refinement of individual skills and valuable learning experiences for a wide range of midwifery team performance. and obstetric skills. With a range of Technical and Educational Services as well as pre-programmed scenarios to ease educator preparation time, SimMom is the optimal simulation experience.
    [Show full text]