Breathe Right! Is an Oregon OSHA Standards and Technical Resources Section Publication
Total Page:16
File Type:pdf, Size:1020Kb
Load more
Recommended publications
-
Safety Data Sheets (Sdss) Information and Glossary
Safety Data Sheets (SDSs) Information and Glossary 2408 Wanda Daley Drive Ames, Iowa 50011 (515) 294-5359 | www.ehs.iastate.edu Copyright© Reviewed 2018 Safety Data Sheets (SDSs) Information Safety Data Sheets (SDSs) are informational sheets required by the Occupational Safety and Health Administration (OSHA) for hazardous substances (chemicals). The OSHA Hazard Communication Standard (29 CFR 1910.1200) and the Iowa Hazardous Chemical Risks Right to Know Standard (Iowa Administrative Code Section 875, Chapters 110-140) requires SDSs be “readily accessible” for any hazardous chemical in the workplace. Employees must be made aware of the SDS content and chemical storage location. SDSs can be obtained from manufacturers and distributors at the time of initial shipment. Each university workplace must maintain an SDS for each hazardous chemical in its inventory. SDSs that are available electronically meet the “readily accessible” criteria, as long as computer access is available to all employees whenever work is being conducted. EH&S suggests that each workspace also maintain paper copies of commonly used hazardous chemicals for ease of access. OSHA requires specific information be included on an SDS, in a 16-section format as described in the UN Globally Harmonized System of Classification and Labeling of Chemicals (GHS). The SDS must be in English and must include at least the following information: Section 1: Identification includes product identifier; manufacturer or distributor name, address, phone number; emergency phone number; recommended use; restrictions on use. Section 2: Hazard(s) Identification includes all hazards regarding the chemical; required label elements. Section 3: Composition/Information on Ingredients includes information on chemical ingredients; trade secret claims. -
Job Hazard Analysis
Identifying and Evaluating Hazards in Research Laboratories Guidelines developed by the Hazards Identification and Evaluation Task Force of the American Chemical Society’s Committee on Chemical Safety Copyright 2013 American Chemical Society Table of Contents FOREWORD ................................................................................................................................................... 3 ACKNOWLEDGEMENTS ................................................................................................................................. 5 Task Force Members ..................................................................................................................................... 6 1. SCOPE AND APPLICATION ..................................................................................................................... 7 2. DEFINITIONS .......................................................................................................................................... 7 3. HAZARDS IDENTIFICATION AND EVALUATION ................................................................................... 10 4. ESTABLISHING ROLES AND RESPONSIBILITIES .................................................................................... 14 5. CHOOSING AND USING A TECHNIQUE FROM THIS GUIDE ................................................................. 17 6. CHANGE CONTROL .............................................................................................................................. 19 7. ASSESSING -
G.J. Chemical Company, Inc. Safety Data Sheet
G.J. CHEMICAL COMPANY, INC. SAFETY DATA SHEET . PRODUCT IDENTIFIER 1.1 PRODUCT NAME: ACETONE PRODUCT NUMBER(S):100100, 100101,100110, 100120, 100130, 100140,100150, 100160 & 100180 TRADE NAMES/SYNONYMS: 2-Propanone; Dimethylformaldehyde; Dimethyl Ketone; Beta-Ketopropane; Methyl Ketone; CAS-No: 67-74-1 CHEMICAL FAMILY: Ketone, Aliphatic 1.2 RELEVANT IDENTIFIED USES OF THE SUBSTANCE OR MIXTURE AND USES ADVISED AGAINST IDENTIFIED USES: 1. Manufacture, process and distribution of substances and mixtures * 2. Use in laboratories 3. Uses in coatings 4. Use as binders and release agents 5. Rubber production and processing 6. Polymer manufacturing 7. Polymer processing 8. Use in Cleaning Agents 9. Use in Oil and Gas Field drilling and production operations 10. Blowing agents 11. Mining chemicals USES ADVISED AGAINST: No information available 1.3 DETAILS OF THE SUPPLIER OF THE SAFETY DATA SHEET Company: G.J. CHEMICAL CO., INC. Address: 40 VERONICA AVENUE SOMERSET, NJ 08873 Telephone: 1-973-589-1450 Fax: 1-973-589-3072 1.4 Emergency Telephone Number Emergency Phone: 1-800-424-9300 (CHEMTREC) . HAZARDS IDENTIFICATION 2.1 Classification of the substance or mixture GHS Classification in accordance with 29CFR 1910 (OSHA HCS) Flammable liquids (Category 2), H225 Eye irritation (Category 2A), H319 Specific target organ toxicity - single exposure (Category 3), Central Nervous system, H336 2.2 GHS Label elements, including precautionary statements Pictogram GHS02 GHS07 Signal word: DANGER Hazard statement(s) H225 Highly flammable liquid and vapor. H319 Causes serious eye irritation. H336 May cause drowsiness or dizziness. Precautionary statement(s) Prevention: P210 Keep away from heat/sparks/open flames/hot surfaces. -
CARBON MONOXIDE: the SILENT KILLER Information You Should
CARBON MONOXIDE: THE SILENT KILLER Information You Should Know Carbon monoxide is a silent killer that can lurk within fossil fuel burning household appliances. Many types of equipment and appliances burn different types of fuel to provide heat, cook, generate electricity, power vehicles and various tools, such as chain saws, weed eaters and leaf blowers. When these units operate properly, they use fresh air for combustion and vent or exhaust carbon dioxide. When fresh air is restricted, through improper ventilation, the units create carbon monoxide, which can saturate the air inside the structure. Carbon Monoxide can be lethal when accidentally inhaled in concentrated doses. Such a situation is referred to as carbon monoxide poisoning. This is a serious condition that is a medical emergency that should be taken care of right away. What Is It? Carbon monoxide, often abbreviated as CO, is a gas produced by burning fossil fuel. What makes it such a silent killer is that it is odorless and colorless. It is extremely difficult to detect until the body has inhaled a detrimental amount of the gas, and if inhaled in high concentrations, it can be fatal. Carbon monoxide causes tissue damage by blocking the body’s ability to absorb enough oxygen. In fact, poisoning from this gas is one of the leading causes of unintentional death from poison. Common Sources of CO Kerosene or fuel-based heaters Fireplaces Gasoline powered equipment and generators Charcoal grills Automobile exhaust Portable generators Tobacco smoke Chimneys, furnaces, and boilers Gas water heaters Wood stoves and gas stoves Properly installed and maintained appliances are safe and efficient. -
Breathing Protection 02
PRODUCT CATALOG US BREATHING PROTECTION 02 CONTENTS 4. SELECTING RESPIRATORY PROTECTION 28. CHOOSE A SUITABLE FACEPIECE 5. TWO TYPES OF RESPIRATORY PROTECTION 28. SR 520 - SR 530 6. SELECTING BREATHING PROTECTION ON 30. SR 570 THE BASIS OF LEVEL OF CONTAMINATION 34. SR 580 7. SELECTING BREATHING PROTECTION BASED 36. SR 580/SR 584 ON WORK DURATION AND WORKLOAD 38. SR 580/SR 587/SR 588-1 & SR 588-2 9. HALF MASKS AND FULL FACE MASKS 39. FILTER 10. HALF MASK SR 100 40. FILTER FOR HALF- AND FULL FACE MASK 12. HALF MASK SR 900 42. FILTER FOR FAN UNIT 14. REMOTE FILTER KIT OPTIONS 16. REMOTE FILTER HOLDER SR 905 43. CONTINUOUS FLOW AIR LINE RESPIRATORY PROTECTION EQUIPMENT 16. FULL FACE MASK SR 200 44. SR 200 AIRLINE 46. SR 307 19. READY-TO-GO KITS 19. FULL FACE MASK OPTIONS 47. COMPRESSED AIR FILTER 20. HALF MASK OPTIONS 47. SR 99-1 23. POWER ASSISTED FILTER PROTECTION POWERED AIR PURIFYING RESPIRATORS 48. COMPRESSED AIR HOSES 24. SR 500 48. SR 358 26. READY-TO-GO-KITS 48. SR 359 50. FILTER RECOMMENDATIONS 03 WORLD CLASS RESPIRATORY PROTECTION EQUIPMENT MADE IN SWEDEN SINCE 1926 Sundström Safety protects people from SUNDSTRÖMS FLEXIBLE SYSTEM contaminated air and is not content to MAKES WORK MORE COMFORTABLE, simply meet official requirements. SAFER AND MORE EFFICIENT Our aim is to always design and manu- facture the best and most comfortable The base for Sundström Safety's System is always the respiratory protection equipment actual respiratory protection over nose and mouth. on the market. -
Effects of Anaesthesia Techniques and Drugs on Pulmonary Function
Review Article Effects of anaesthesia techniques and drugs on pulmonary function Address for correspondence: Vijay Saraswat Dr. Vijay Saraswat, Department of Anaesthesiology, Apollo Hospitals, Nashik, Maharashtra, India Apollo Hospitals, Nashik, Maharashtra, India. E‑mail: drvsaraswat@gmail. ABSTRACT com The primary task of the lungs is to maintain oxygenation of the blood and eliminate carbon dioxide through the network of capillaries alongside alveoli. This is maintained by utilising ventilatory reserve capacity and by changes in lung mechanics. Induction of anaesthesia impairs pulmonary functions by the loss of consciousness, depression of reflexes, changes in rib cage and haemodynamics. All drugs used during anaesthesia, including inhalational agents, affect pulmonary functions directly by acting on respiratory system or indirectly through their actions on other systems. Volatile anaesthetic agents have more pronounced effects on pulmonary functions compared to intravenous induction agents, leading to hypercarbia and hypoxia. The posture of the patient also leads to major changes in pulmonary functions. Anticholinergics and neuromuscular blocking agents have little effect. Analgesics and Access this article online sedatives in combination with volatile anaesthetics and induction agents may exacerbate Website: www.ijaweb.org their effects. Since multiple agents are used during anaesthesia, ultimate effect may be DOI: 10.4103/0019‑5049.165850 different from when used in isolation. Literature search was done using MeSH key words ‘anesthesia’, -
142, May 1999 Reuse of Organic Vapor Chemical Cartridges
Technical Data Bulletin #142, May 1999 Reuse of Organic Vapor Chemical Cartridges Introduction One of the most significant changes in the Occupational Safety and Health Administration’s (OSHA’s) new 1910.134 respiratory protection standard is the requirement to establish change schedules for chemical cartridges used for gases and vapors. Change schedules are often based on service life measurements or estimates. To best use the service life information, it is necessary to understand how chemical cartridges work. It is especially important when organic vapor cartridges are used against volatile chemicals during more than one work shift. These chemicals may desorb from the carbon when not in use. Inappropriate reuse of the organic vapor cartridges can result in breakthrough occurring earlier than predicted by the service life estimate. For example, when the organic vapor cartridge has been used for chemicals that migrate through the cartridge during the storage or nonuse period, it should not be reused. The decision to reuse the cartridge may have an impact on worker protection and the respiratory protection program Background Chemical cartridges are used on respirators to help remove and lower worker exposures to harmful gases and vapors in the workplace. There are several types of chemical cartridges: organic vapor, ammonia, formaldehyde, mercury vapor and acid gases, such as hydrogen chloride, chlorine and sulfur dioxide. It is important to understand how the different cartridge types work. All chemical cartridges consist of a container filled with a sorbent. A chemical cartridge sorbent is a granular porous material that interacts with the gas or vapor molecule to remove it from the air. -
Respiratory System
Respiratory System 1 Respiratory System 2 Respiratory System 3 Respiratory System 4 Respiratory System 5 Respiratory System 6 Respiratory System 7 Respiratory System 8 Respiratory System 9 Respiratory System 10 Respiratory System 11 Respiratory System • Pulmonary Ventilation 12 Respiratory System 13 Respiratory System 14 Respiratory System • Measuring of Lung Function œ Compliance œ the ease at which the lungs and thoracic wall can be expanded œ if reduced it is more difficult to inflate the lungs œ causes: • Damaged lung tissue • Fluid within lung tissue • Decrease in pulmonary surfactant • Anything that impedes lung expansion or contraction œ Respiratory Volumes and Capacities will be covered in Lab œ 15 Respiratory System • Exchange of Oxygen and Carbon Dioxide œ Charles‘ Law œ the volume of a gas is directly proportional to the absolute temperature, assuming the pressure remains constant As gases enter the lung they warm and expand, increasing lung volume œ Dalton‘s Law œ each gas of a mixture of gases exerts its own pressure as if all the other gases were not present œ Henry‘s Law œ the quantity of a gas that will dissolve in a liquid is proportional to the partial pressure of the gas and its solubility coefficient, when the temperature remains constant 16 Respiratory System • External and Internal Respiration 17 Respiratory System • Transport of Oxygen and Carbon Dioxide by the Blood œ Oxygen Transport • 1.5% dissolved in plasma • 98.5% carried with Hbinside of RBC‘s as oxyhemoglobin œ Hbœ made up of protein portion called the globinportion -
THE 6 MAJOR BODY SYSTEMS and How They Interact with Each Other to Keep the “Body Machine” Alive and Working Well
THE 6 MAJOR BODY SYSTEMS And how they interact with each other to keep the “body machine” alive and working well. CIRCULATORY SYSTEM / CARDIOVASCULAR SYSTEM PRIMARY PURPOSE: transport blood throughout the body by circulating PRIMARY ORGANS/PARTS: Heart, blood vessels (arteries, veins, capillaries) (1) Transports/carries nutrients and oxygen through the blood to most parts of the body (2) Transports/carries waste in cells and carbon-dioxide (CO2) away from the parts: (a) Cell waste goes to the kidneys for filter and disposal (b) Carbon-dioxide (CO2) goes to the lungs to exhale (breathe out) Kidneys and Lungs have a close relationship with Cardiovascular system Kidneys: filter through blood to take out the waste and get it eventually out of the body Lungs: breathes in oxygen and gives it to the blood for Circulatory system to carry throughout the body; and takes unneeded carbon-dioxide (CO2) from the blood and breathes that out. Circulatory/Cardiovascular System through the blood to most parts of the body provides nutrients and oxygen which is needed for our bodies to have ENERGY! RESPIRATORY SYSTEM PRIMARY PURPOSE: Breathing - taking in Oxygen, pushing out Carbon-Dioxide (CO2) PRIMARY ORGANS: Lungs, trachea (tube going from lungs to nose/mouth) (1) Inhales (breathes in) Oxygen - good for the body - gives it to the Circulatory System to be transported throughout the body through the blood. (2) Exhales (breathes out) Carbon-Dioxide (CO2) - lungs get this gas from the blood (Circ. Sys.) and pushes it out of the body DIGESTIVE SYSTEM PRIMARY PURPOSE: take in food; break down food into nutrients (good) and waste (unneeded) PRIMARY ORGANS: Stomach, large and small intestines, esophagus (tube from stomach to mouth) (1) Digestive System gets nutrients (good) from food and hands it over to the blood and Circulatory System then carries those nutrients where they need to go. -
Oxygen Toxicity in Neonatal Rats: the Effect of Endotoxin Treatment on Survival During and Post-02 Exposure
0031-3998/87/2102-0 I 09$02.00/0 PEDIATRIC RESEARCH Vol. 21. No.2, 1987 Copyright© 1987 International Pediatric Research Foundation, Inc. Printed in U.S.A. Oxygen Toxicity in Neonatal Rats: The Effect of Endotoxin Treatment on Survival during and post-02 Exposure LEE FRANK Department ofMedicine, Pulmonary Research Labora10ry, University of Miami School of Medicine, Miami, Florida 33136 ABSTRACT. Neonatal rats were treated with low doses of monary 0 2 toxicity (I, 2). However, there are unique pulmonary bacterial lipopolysaccharide (endotoxin) to test for a pro complications associated with prolonged hyperoxic exposure in tective effect of endotoxin against 02 toxicity and the the neonatal animal which could importantly influence both severe inhibition of normal lung development which occurs short and long-term survival post-02 exposure. These complica during prolonged exposure to hyperoxia. The rationale for tions relate to the known inhibitory action of hyperoxia on lung the prophylactic use of endotoxin included its marked biosynthetic processes (1, 3, 4), and the morphological conse protective effect against pulmonary 0 2 toxicity in adult quences this inhibitory action has on the rapidly growing and rats and its lung growth-promoting effect in experimental differentiating newborn (animal) lung. Several studies have now pulmonary stress models. Neonatal rats (4-5 days old) demonstrated that early postnatal exposure to hyperoxia is as survived a 14-day exposure to >95% 0 2 equally well sociated with marked inhibition of the normal septation process whether treated with saline (39/51 = 76%) or with endo by which the large saccular airspaces characteristic of the new toxin (41/51 = 80%). -
Respiratory System
Respiratory System Course Rationale Anatomy & To pursue a career in health care, proficiency in anatomy and physiology is Physiology vital. Unit XIII Objectives Respiratory Upon completion of this lesson, the student will be able to: System • Describe biological and chemical processes that maintain homeostasis • Analyze forces and the effects of movement, torque, tension, and Essential elasticity on the human body Question • Define and decipher terms pertaining to the respiratory system How long can • Distinguish between the major organs of the respiratory system the body be • Analyze diseases and disorders of the respiratory system without • Label a diagram of the respiratory system oxygen? Engage TEKS Perform the following in front of the class using a paper towel and a hand 130.206 (c) mirror: 1 (A)(B) • Use the paper towel to clean and dry the mirror. 2(A)(D) • Hold the mirror near, but not touching, your mouth. 3 (A)(B)(E) • Exhale onto the mirror two or three times. 5 (B)(C)(D) • Examine the surface of the mirror. 6 (B) What happens to the mirror? 8 (A)(B)(C) Why does the mirror become fogged? 9 (A)(B) 10 (A)(B)(C) Or Prior Student Of all the substances the body must have to survive, oxygen is by far the most Learning critical. Think about the following: Cardiovascular system – • Without food - live a few weeks Pulmonary • Without water - live a few days Circulation • Without oxygen - live 4 – 6 minutes Estimated time 4 - 6 hours Key Points 1. Introduction – Respiratory System A. General Functions *Teacher note: 1. Brings oxygenated air to the alveoli invite a 2. -
The Future of Mechanical Ventilation: Lessons from the Present and the Past Luciano Gattinoni1*, John J
Gattinoni et al. Critical Care (2017) 21:183 DOI 10.1186/s13054-017-1750-x REVIEW Open Access The future of mechanical ventilation: lessons from the present and the past Luciano Gattinoni1*, John J. Marini2, Francesca Collino1, Giorgia Maiolo1, Francesca Rapetti1, Tommaso Tonetti1, Francesco Vasques1 and Michael Quintel1 Abstract The adverse effects of mechanical ventilation in acute respiratory distress syndrome (ARDS) arise from two main causes: unphysiological increases of transpulmonary pressure and unphysiological increases/decreases of pleural pressure during positive or negative pressure ventilation. The transpulmonary pressure-related side effects primarily account for ventilator-induced lung injury (VILI) while the pleural pressure-related side effects primarily account for hemodynamic alterations. The changes of transpulmonary pressure and pleural pressure resulting from a given applied driving pressure depend on the relative elastances of the lung and chest wall. The term ‘volutrauma’ should refer to excessive strain, while ‘barotrauma’ should refer to excessive stress. Strains exceeding 1.5, corresponding to a stress above ~20 cmH2O in humans, are severely damaging in experimental animals. Apart from high tidal volumes and high transpulmonary pressures, the respiratory rate and inspiratory flow may also play roles in the genesis of VILI. We do not know which fraction of mortality is attributable to VILI with ventilation comparable to that reported in recent clinical practice surveys (tidal volume ~7.5 ml/kg, positive end-expiratory pressure (PEEP) ~8 cmH2O, rate ~20 bpm, associated mortality ~35%). Therefore, a more complete and individually personalized understanding of ARDS lung mechanics and its interaction with the ventilator is needed to improve future care.