Head Injury/Concussion
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Early Management of Retained Hemothorax in Blunt Head and Chest Trauma
World J Surg https://doi.org/10.1007/s00268-017-4420-x ORIGINAL SCIENTIFIC REPORT Early Management of Retained Hemothorax in Blunt Head and Chest Trauma 1,2 1,8 1,7 1 Fong-Dee Huang • Wen-Bin Yeh • Sheng-Shih Chen • Yuan-Yuarn Liu • 1 1,3,6 4,5 I-Yin Lu • Yi-Pin Chou • Tzu-Chin Wu Ó The Author(s) 2018. This article is an open access publication Abstract Background Major blunt chest injury usually leads to the development of retained hemothorax and pneumothorax, and needs further intervention. However, since blunt chest injury may be combined with blunt head injury that typically requires patient observation for 3–4 days, other critical surgical interventions may be delayed. The purpose of this study is to analyze the outcomes of head injury patients who received early, versus delayed thoracic surgeries. Materials and methods From May 2005 to February 2012, 61 patients with major blunt injuries to the chest and head were prospectively enrolled. These patients had an intracranial hemorrhage without indications of craniotomy. All the patients received video-assisted thoracoscopic surgery (VATS) due to retained hemothorax or pneumothorax. Patients were divided into two groups according to the time from trauma to operation, this being within 4 days for Group 1 and more than 4 days for Group 2. The clinical outcomes included hospital length of stay (LOS), intensive care unit (ICU) LOS, infection rates, and the time period of ventilator use and chest tube intubation. Result All demographics, including age, gender, and trauma severity between the two groups showed no statistical differences. -
NIH Public Access Author Manuscript J Neuropathol Exp Neurol
NIH Public Access Author Manuscript J Neuropathol Exp Neurol. Author manuscript; available in PMC 2010 September 24. NIH-PA Author ManuscriptPublished NIH-PA Author Manuscript in final edited NIH-PA Author Manuscript form as: J Neuropathol Exp Neurol. 2009 July ; 68(7): 709±735. doi:10.1097/NEN.0b013e3181a9d503. Chronic Traumatic Encephalopathy in Athletes: Progressive Tauopathy following Repetitive Head Injury Ann C. McKee, MD1,2,3,4, Robert C. Cantu, MD3,5,6,7, Christopher J. Nowinski, AB3,5, E. Tessa Hedley-Whyte, MD8, Brandon E. Gavett, PhD1, Andrew E. Budson, MD1,4, Veronica E. Santini, MD1, Hyo-Soon Lee, MD1, Caroline A. Kubilus1,3, and Robert A. Stern, PhD1,3 1 Department of Neurology, Boston University School of Medicine, Boston, Massachusetts 2 Department of Pathology, Boston University School of Medicine, Boston, Massachusetts 3 Center for the Study of Traumatic Encephalopathy, Boston University School of Medicine, Boston, Massachusetts 4 Geriatric Research Education Clinical Center, Bedford Veterans Administration Medical Center, Bedford, Massachusetts 5 Sports Legacy Institute, Waltham, MA 6 Department of Neurosurgery, Boston University School of Medicine, Boston, Massachusetts 7 Department of Neurosurgery, Emerson Hospital, Concord, MA 8 CS Kubik Laboratory for Neuropathology, Department of Pathology, Massachusetts General Hospital, Harvard Medical School, Boston, Massachusetts Abstract Since the 1920s, it has been known that the repetitive brain trauma associated with boxing may produce a progressive neurological deterioration, originally termed “dementia pugilistica” and more recently, chronic traumatic encephalopathy (CTE). We review the 47 cases of neuropathologically verified CTE recorded in the literature and document the detailed findings of CTE in 3 professional athletes: one football player and 2 boxers. -
Post-Concussion Syndrome
Post-Concussion Syndrome BY DAVID COPPEL Over the last decade, sport-related concussions have fatigue, irritability, sleep disturbance and sensitivity to become an important focus within the general sports inju- light and noise may continue over the next few days. Oth- WHAT CAN COACHES DO? ry and sports medicine field. Clinical and research studies SIGNS AND SYMPTOMS • Make sure student-athletes who sustain a concus- er symptoms seen on post-concussion symptom checklists According to the Diagnostic and Statistical Manual regarding this form/context of mild traumatic brain injury sion are immediately removed from play and that include attention and concentration difficulties, slowed of Mental Disorders – 4th edition (DSM-4) – an individu- have increased geometrically as its position as a public they do not feel pressure from the coaching staff to processing, distractibility, memory problems, slowed visu- al with post-concussion disorder experiences objective health concern elevated and the Centers for Disease Con- return to play before fully recovered. Communicating al tracking or vision problems, balance disturbance, and declines in attention, concentration, learning or memo- with team members before the season about con- trol and Prevention (CDC) became involved. ry. The individual also reports three or more subjective anxiety or depressed mood. Typically, depressed mood or cussion safety, and verbally reinforcing the impor- The CDC has compiled guidelines and resources for symptoms, present for at least three months: tance of concussion safety throughout the season health care providers, coaches, parents and athletes re- • Becoming fatigued easily are important ways to encourage student-athletes garding concussions. Great progress has been made in • Disordered sleep to feel comfortable reporting concussion symptoms WHAT CAN ATHLETIC • Headache understanding and managing sport-related concussions, to medical personnel. -
Concussion Guide for Coaches
Concussion Guide for Youth and High School Coaches SIGNS OBSERVED BY COACHING STAFF THE FACTS • Appears dazed or stunned (such as glassy eyes) • All concussions are serious. • Is confused about assignment or position • Most concussions occur without loss of • Forgets an instruction or play consciousness. • Is unsure of score or opponent • Recognition and proper response to concussions • Moves clumsily or poor balance when they first occur can help aid recovery and • Answers questions slowly prevent further injury, or even death. • Loses consciousness (even briefly) • Shows mood, behavior, or personality changes • Can’t recall events prior to hit or fall A bump, blow, or jolt to the head can cause a concussion, • Can’t recall events after hit or fall a type of traumatic brain injury (TBI). Concussions can also occur from a blow to the body that causes the head and brain to move rapidly back and forth. Even a “ding,” SYMPTOMS REPORTED BY ATHLETE “getting your bell rung,” or what seems to be mild bump • Headache or “pressure” in head or blow to the head can be serious. • Nausea or vomiting • Balance problems or dizziness • Double or blurry vision RECOGNIZING A POSSIBLE CONCUSSION • Sensitivity to light or noise To help recognize a concussion, watch for or ask others to • Feeling sluggish, hazy, foggy, or groggy report the following two things among your athletes: • Concentration or memory problems 1. A forceful bump, blow, or jolt to the head or body • Confusion that results in rapid movement of the head. • Feeling more emotional, nervous, or anxious – and – • Does not “feel right” or is “feeling down” 2. -
The Relationships Between Rugby Union, and Health and Well-Being
Review Br J Sports Med: first published as 10.1136/bjsports-2020-102085 on 28 October 2020. Downloaded from The relationships between rugby union, and health and well- being: a scoping review Steffan A Griffin ,1,2 Nirmala Kanthi Panagodage Perera ,3,4 Andrew Murray ,1,5 Catherine Hartley ,6 Samantha G Fawkner,7 Simon P T Kemp ,2,8 Keith A Stokes ,2,9 Paul Kelly 10 1Centre for Sport and Exercise, ABSTRACT Indeed, there are widely published health benefits The University of Edinburgh, Objective To scope the relationships between rugby associated with participating in various individual Edinburgh, UK 7 8 2 and team sports, including improved aerobic Medical Services, Rugby union, and health and well- being. Football Union, London, UK Design Scoping review. fitness, cardiovascular function, metabolic fitness 3Centre for Sport, Exercise and Data sources Published and unpublished reports of and in some cases reduced mortality.9 Sport is also Osteoarthritis Research Versus any age, identified by searching electronic databases, considered an ‘underused yet important’ contrib- Arthritis, Botnar Research platforms and reference lists. utor to physical activity (PA) and health10 in the Centre, University of Oxford, Oxford, UK Methods A three- step search strategy identified World Health Organisation’s (WHO) 2019 Global 4Sports Medicine, Australian relevant published primary, secondary studies and grey Action Plan for Physical Activity. Institute of Sport, Bruce, literature, which were screened using a priori inclusion Despite global participation in rugby union, there Australian Capital Territory, criteria. Data were extracted using a standardised tool, has not been an overarching review of the evidence Australia 5 on the specific relationships between rugby union, Scottish Rugby Union, to form (1) a numerical analysis and (2) a thematic Edinburgh, UK summary. -
Mild Traumatic Brain Injury and Concussion: Information for Adults
Mild Traumatic Brain Injury and Concussion: Information for Adults Discharge Instructions You were seen today for a mild traumatic brain injury (mild TBI) or concussion. Watch for Danger Signs Use this handout to help you watch In rare cases, a dangerous blood clot that for changes in how you are feeling crowds the brain against the skull can develop or acting and to help you feel better. after a TBI. The people checking on you should call 911 or take you to an emergency Be sure to let a family member or department right away if you have: friend know about your injury and the types of symptoms to look out • A headache that gets worse and does not for. They may notice symptoms go away before you do and can help you. • Significant nausea or repeated vomiting • Unusual behavior, increased confusion, Schedule a follow-up appointment restlessness, or agitation with your regular doctor. • Drowsiness or inability to wake up Due to your injury, you may need to take some • Slurred speech, weakness, numbness, or time off from things like work or school. If so, ask decreased coordination your doctor for written instructions about when • Convulsions or seizures (shaking or you can safely return to work, school, sports, twitching) or other activities such as driving a car, riding a • Loss of consciousness (passing out) bike, or operating heavy equipment. More information on mild TBI and concussion, as well as tips to help you feel better, can be found at www.cdc.gov/TraumaticBrainInjury. Learn About Your Injury Mild TBI and concussions are brain injuries. -
Guidelines for BLS/ALS Medical Providers Current As of March 2019
Tactical Emergency Casualty Care (TECC) Guidelines for BLS/ALS Medical Providers Current as of March 2019 DIRECT THREAT CARE (DTC) / HOT ZONE Guidelines: 1. Mitigate any immediate threat and move to a safer position (e.g. initiate fire attack, coordinated ventilation, move to safe haven, evacuate from an impending structural collapse, etc). Recognize that threats are dynamic and may be ongoing, requiring continuous threat assessments. 2. Direct the injured first responder to stay engaged in the operation if able and appropriate. 3. Move patient to a safer position: a. Instruct the alert, capable patient to move to a safer position and apply self-aid. b. If the patient is responsive but is injured to the point that he/she cannot move, a rescue plan should be devised. c. If a patient is unresponsive, weigh the risks and benefits of an immediate rescue attempt in terms of manpower and likelihood of success. Remote medical assessment techniques should be considered to identify patients who are dead or have non-survivable wounds. 4. Stop life threatening external hemorrhage if present and reasonable depending on the immediate threat, severity of the bleeding and the evacuation distance to safety. Consider moving to safety prior to application of the tourniquet if the situation warrants. a. Apply direct pressure to wound, or direct capable patient to apply direct pressure to own wound and/or own effective tourniquet. b. Tourniquet application: i. Apply the tourniquet as high on the limb as possible, including over the clothing if present. ii. Tighten until cessation of bleeding and move to safety. -
Concussion and Epilepsy: What Is the Link?
COVER STORY Concussion and Epilepsy: What is the Link? Individuals who suffer a traumatic brain injury are at increased risk for developing seizures. Here’s an update the current evidence and implications for monitoring. INSIGHTS FROM G. ALEXANDER HISHAW, MD ccording to a 2003 report by the CDC, mild trau- matic injury results in roughly 500,000 emergency Significant risk factors for later department visits without hospitalization and seizures included brain contusion almost 200,000 hospitalizations per year. However, Ait’s believed this number is an underestimate because a with subdural hematoma, skull number of people with mild traumatic brain injury (TBI) fracture, loss of consciousness or are never seen in an ED. Increasingly, TBI is associated with amnesia for more than one day, and various neurolgical sequelae, as listed in Table 1. Among these, is epilepsy. age of 65 years or older. A population study in seizures after TBI by John Annegers, et al. showed that the standardized incidence UNDERSTANDING THE INJURY ratio was 1.5 after mild injury, 2.9 after moderate injury, According to the Department of Defense definition, acute and 17 after severe injury. Significant risk factors for later injury results from primary (mechanical) physical disrup- seizures included brain contusion with subdural hema- toma, skull fracture, loss of consciousness or amnesia for TABLE 1. NEUROLOGICAL CONDITIONS more than one day, and an age of 65 years or older. LINKED TO TBI Additional data are available from the Southern Arizona VA registry, which show: of 184 patients with TBI, three • Epilepsy percent of mild TBI cases experienced seizures, five per- • Dissociation cent of moderate TBI cases experienced seizures, and 32 • Frontal lobe syndrome percent of severe TBI cases experienced seizures. -
CDC Concussion Definition and Pathophysiology
Concussion Definition and Pathophysiology The Evolving Definition of Concussion CDC Physicians Toolkit; Collins, Gioia et al 2006 Regarding Cerebral Concussion…… A concussion (or mild traumatic brain injury) is a complex pathophysiological process affecting the brain, induced by traumatic biomechanical forces secondary to direct or indirect forces to the head. Disturbance of brain function is related to neurometabolic dysfunction, rather than structural brain injury, and is typically associated with normal structural imaging findings (CT Scan, MRI). Concussion may or may not involve a loss of consciousness. Concussion results in a constellation of physical, cognitive, emotional, and sleep‐related symptoms. Recovery is a sequential process and symptoms may last from several minutes to days, weeks, months, or even longer in some cases.” . Following a concussion, there are metabolic chemical changes that take place in the brain. Brain injury can occur even if there is NO loss of consciousness. More than 90% of concussions DO NOT involve loss of consciousness. Memories of events BEFORE and AFTER the concussion are MORE accurate assessments of SEVERITY than loss of consciousness. Pathophysiology of Concussions – Dr. Micky Collins, Director UPMC Sports Medicine Concussion Program Neurometabolic Cascade of Concussion During activities a concussion can occur with any type of external force to the head. These events can cause the brain to accelerate and decelerate with translational, rotational and/or angular forces. Brain injuries can result on the side of the force, cu, or the opposite side of the force, contra cu. So what exactly do these forces do to the brain? Researchers believe it leads to a wave of energy that passes through the brain tissue triggering neuronal dysfunction. -
Neurologic Injuries in Hockey Richard A
University of North Dakota UND Scholarly Commons Librarian Publications Chester Fritz Library 2009 Neurologic Injuries in Hockey Richard A. Wennberg Howard B. Cohen Stephanie Walker University of North Dakota, [email protected] Follow this and additional works at: https://commons.und.edu/cfl-lp Recommended Citation Wennberg, Richard A.; Cohen, Howard B.; and Walker, Stephanie, "Neurologic Injuries in Hockey" (2009). Librarian Publications. 9. https://commons.und.edu/cfl-lp/9 This Article is brought to you for free and open access by the Chester Fritz Library at UND Scholarly Commons. It has been accepted for inclusion in Librarian Publications by an authorized administrator of UND Scholarly Commons. For more information, please contact [email protected]. Neurologic Injuries in Hockey Richard A.Wennberg, MD, MSc, FRCPCa,*, Howard B. Cohen, DDS, MA, PhDb, Stephanie R.Walker, MA, MLSc KEYWORDS: Hockey Head injury Concussion Spinal cord injury What they worry about most is injuries,.[in] this combination of ballet and murder. Al Purdy, Hockey Players, 1965 References to hockey injuries can be found not only in poetry but also in the medical literature, where authors have described the game as a combination of ‘‘finesse and controlled aggression’’.1,2 Hockey players attain skating speeds of more than 30 miles per hour and may shoot the puck at more than 100 miles per hour, all in the setting of a contact sport played on an ice surface enclosed by unyielding boards and glass.1 There is an understandable risk for injuries, -
Early Post-Traumatic Pulmonary-Embolism in Patients Requiring ICU Admission: More Complicated Than We Think!
3854 Editorial Early post-traumatic pulmonary-embolism in patients requiring ICU admission: more complicated than we think! Mabrouk Bahloul1, Mariem Dlela1, Nadia Khlaf Bouaziz2, Olfa Turki1, Hedi Chelly1, Mounir Bouaziz1 1Department of Intensive Care, Habib Bourguiba University Hospital, Sfax, Tunisia; 2Centre Intermédiaire, Rte El MATAR Km 4, Sfax, Tunisia Correspondence to: Professor Mabrouk Bahloul. Department of Intensive Care, Habib Bourguiba University Hospital, 3029 Sfax, Tunisia. Email: [email protected]. Submitted Jun 10, 2018. Accepted for publication Sep 12, 2018. doi: 10.21037/jtd.2018.09.49 View this article at: http://dx.doi.org/10.21037/jtd.2018.09.49 We read with interest the article entitled “Prevalence and traumatic thromboembolism in patients who are requiring main determinants of early post-traumatic thromboembolism an ICU admission. Nevertheless, this study as mentioned in patients requiring ICU admission” (1). In this retrospective by the authors suffers from many limitations. In fact, the study, Kazemi Darabadi et al. (1) had included to their small sample size, and also the retrospective design of database, the records of 240 trauma-patients requiring the study lead to an increased probability of some bias ICU admission, with a confirmed diagnosis of pulmonary when collecting data. In fact, in this study (1), PE was embolism (PE). The patients were categorized as subjects not screened on a daily basis. As a consequence, the late with an early PE (≤3 days) and those with a late PE (>3 days). stage PE may only be a delayed diagnosis. For instance, According to their analysis, 48.5% of the patients suffering a patient who develops with PE on day 2 but without any from PE had developed this complication within 72 h symptom(s); then he is accidentally diagnosed with PE on following a trauma event and/or after ICU admission. -
The Fund at a Glance
CytoSorbentsTM Trauma and Crush Injury TRAUMA AND CRUSH INJURIES ARE CURRENTLY LEADING CAUSES OF DEATH. Trauma and crush injuries occur frequently and from multiple sources. For example, automobile or motorcycle accidents, natural disasters, and acts of criminal violence or terrorism can all yield mild to severe trauma and crush injuries. The breadth of life-threatening conditions from trauma-induced injuries, such as lung and bowel rupture, closed head injury, crush injury and rhabdomyolysis, traumatic fractures, penetrating wound injury, and limb loss, creates a very challenging environment for clinicians to employ effective treatment strategies. Better therapies are needed to reduce the mortality in patients with severe trauma injury, reduce the risk of organ failure, reduce complications (e.g. rhabdomyolysis and infection), and decrease hospital stay length. CYTOKINE STORM AND RHABDOMYOLYSIS IN TRAUMA CAN CAUSE ORGAN FAILURE AND DEATH Trauma is a well-known trigger of the immune response, and can result in the massive, uncontrolled production of cytokines, or “cytokine storm.” Cytokines are small proteins that normally help stimulate and regulate the immune system to fight infection or injury. However, in trauma, cytokine storm can be toxic, leading to a massive systemic inflammatory response syndrome (SIRS) and a cascade of events that can kill cells and damage organs, leading to organ failure and often death. Cytokine storm exacerbates physical trauma in many ways. For example, trauma can cause: Hemorrhagic shock due to blood loss while cytokine storm causes capillary leak and intravascular volume loss, and the induction of nitric oxide that can lead to myocardial depression and peripheral vasodilation, all of which exacerbate shock Severe limb injuries that often require a tourniquet.