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Paramedic National EMS Education Standard
NORTHWEST COMMUNITY EMERGENCY MEDICAL SERVICES SYSTEM CCCooonnntttiiinnnuuuiiinnnggg EEEddduuucccaaatttiiiooonnn SSSeeepppttteeemmmbbbeeerrr 222000111222 EEyyee && EEaarr DDiissoorrddeerrss && TTrraauummaa Questions/comments are welcome. Please direct to Jen Dyer, RN, EMT-P EMS Educator NWC EMSS Con-Ed Eye and Ear Disorders and Trauma September 2012 – page 1 Paramedic National EMS Education Standard Integrates assessment findings with principles of pathophysiology to formulate a field impression and implement a treatment/disposition plan for patients with eye and ear disorders/trauma. Objectives: Upon completion of the class and review of the independent study materials and post-test question bank, each participant will do the following with a degree of accuracy that meets or exceeds the standards established for their scope of practice: 1. Identify the anatomical structures of the eye and describe the corresponding physiologic function of each. (C) 2. Explain the physiology of normal vision. (C) 3. Identify the anatomic structures of the ear and describe the corresponding physiologic function of each. (C) 4. Explain the physiology of normal hearing. (C) 5. Explain the physiology of equilibrium. (C) 6. Select and discuss maneuvers for assessing eye structures and functions (C) and demonstrate a thorough EMS assessment of ocular structures, visual acuity, pupils and ocular movements. (P) 7. Distinguish abnormal assessment findings/conditions of the eye: blurred vision, diplopia, photophobia, changes in vision, flashing, pupil exam, Adie’s pupil, oculomotor nerve paralysis, Horner’s Syndrome, blindness, deviation/paralytic strabismus, orbit fracture, cataracts, conjunctivitis, color blindness, near sightedness, farsightedness, astigmatism, amblyopia, burns of the eye, corneal abrasions, foreign body, inflammation of the eyelid, glaucoma, hyphema, iritis, orbital cellulitis, macular degeneration and trauma. -
Week of July 27, 2015 – Eye Injuries Protecting Your Eyes from Injury Is
Week of July 27, 2015 – Eye Injuries Protecting your eyes from injury is one of the most basic things you can do to keep your vision healthy throughout your life. And the most basic step a person can take to protect his/her eyes is wearing the proper protective eyewear. According to a national survey by the American Academy of Ophthalmology, only 35 percent of respondents said they always wear protective eyewear when performing home repairs or maintenance; even fewer do so while playing sports. Eye emergencies include: cuts, scratches, getting objects in the eye, burns, chemical exposures, and blunt injuries to the eye or eyelid. Certain eye infections and other medical conditions, such as blood clots or glaucoma, also represent serious conditions. Since the eye is easily damaged, any of these conditions can lead to vision loss. A black eye is a bruise and usually caused by direct trauma to the eye or face. The bruise is caused by bleeding under the skin. The tissue around the eye turns black and blue, gradually, over a few days, it changes to purple, green, and yellow. The abnormal color disappears within 2 weeks. Swelling of the eyelid and tissue around the eye may also occur. Certain types of skull fractures may also result in bruising around the eyes, even without direct injury to the eye. Sometimes, serious damage to the eye itself occurs from the pressure of a swollen eyelid or face and can result is a hyphema; which is blood in the front area of the eye. Trauma is a common cause of the condition and is often due to a direct hit to the eye from a ball. -
The Use of Hydrofera Blue™ on a Chemical Burn By
Case Study: The Use of Hydrofera Blue™ on a Chemical Burn by Cyhalothrin Jeanne Alvarez, FNP, CWS Independent Medical Associates, Bangor, ME History of Present Illness/Injury: This 70 year old white male was spraying a product containing cyhalothrin (Hot Shot Home Insect Control) overhead to kill spiders. Some of the product dripped and came in contact with his skin in five locations on his upper right arm and hand. He states he washed his arm and hand with copious amounts of soap and water right after the contact of the product on his skin. He presented to the office for evaluation four days after the incidence complaining of burning pain, paresthesia and blistering at the sites. A colleague initially saw this patient and contacted poison control who provided information regarding the procedure for decontamination and monitoring. Prolonged exposure can cause symptoms similar to frostbite. Paresthesia related to dermal exposure is reported but there was no available guidance for treatment options for the blistered areas and/or treatment options for the paresthesia given. Washing the contact area with soap and water was indicated by the guidelines. Past Medical History: This patient has a significant history of hypertension. Medications/Allergies: This patient takes Norvasc 10mg daily. He has used Tylenol 1000mg every 4-6 hours as needed for pain without significant improvement in his pain level. He has no known allergies. Treatments: Day 4 (after exposure): The patient presented for evaluation after a dermal chemical exposure complaining of burning pain, blisters and paresthesia. He had washed the area after exposure with soap and water and had applied a triple antibiotic ointment. -
Diagnosing Depressed Skull Fracture in a Young Child
Nursing Practice Keywords: Skull fracture/Children/ Neurology Case study Neurology Skull fractures associated with intracranial injury are a leading cause of traumatic death in childhood. Children with head injuries should be monitored for signs of deterioration Diagnosing depressed skull fracture in a young child respiratory rate 32/min and oxygen satura- In this article... tion 97% in air. He was drowsy but easily Risks associated with head injury and skull fracture roused by his parents. The Glasgow Coma Scale score was 12/15, which could indicate The importance of monitoring and early diagnosis an intracranial injury and raised intracra- nial pressure. A neurological examination revealed Authors Shameem Ahmed is assistant left-sided hemi-paresis indicative of raised professor in neurosurgery; Rupa Thenseen intracranial pressure. A right-sided lateral Frank is sister in charge, neurosurgical soft tissue swelling and haematoma along operation theatre; both at Gauhati Medical with a depression of the underlying bones College, Guwahati, India; Siba Prosad Paul was noted on palpation of his skull. is specialty trainee in neonates at South- He was reviewed by an anaesthetist and mead Hospital, Bristol his condition was considered to be stable. An urgent non-contrast computed tomog- ead injury is the most common raphy scan revealed a large depressed frac- cause of death and disability in ture (>5mm) involving the right fronto- people aged below 40 years parieto-occipital bone (Fig 1). The boy was H(National Institute for Health Fig 1. Large right fronto-parieto-occipital reviewed by the neurosurgical team and an and Care Excellence, 2014). It accounts for bone simple depressed fracture exploration and elevation of the depressed 1.4 million attendances at accident and fragment was carried out on the same day. -
Trauma Clinical Guideline: Major Burn Resuscitation
Washington State Department of Health Office of Community Health Systems Emergency Medical Services and Trauma Section Trauma Clinical Guideline Major Burn Resuscitation The Trauma Medical Directors and Program Managers Workgroup is an open forum for designated trauma services in Washington State to share ideas and concerns about providing trauma care. The workgroup meets regularly to encourage communication among services, and to share best practices and information to improve quality of care. On occasion, at the request of the Emergency Medical Services and Trauma Care Steering Committee, the group discusses the value of specific clinical management guidelines for trauma care. The Washington State Department of Health distributes this guideline on behalf of the Emergency Medical Services and Trauma Care Steering Committee to assist trauma care services with developing their trauma patient care guidelines. Toward this goal, the workgroup has categorized the type of guideline, the sponsoring organization, how it was developed, and whether it has been tested or validated. The intent of this information is to assist physicians in evaluating the content of this guideline and its potential benefits for their practice or any particular patient. The Department of Health does not mandate the use of this guideline. The department recognizes the varying resources of different services, and approaches that work for one trauma service may not be suitable for others. The decision to use this guideline depends on the independent medical judgment of the physician. We recommend trauma services and physicians who choose to use this guideline consult with the department regularly for any updates to its content. The department appreciates receiving any information regarding practitioners’ experience with this guideline. -
Causes and Characteristics of Peri-Orbital Contusions and Their Relationship with Intracranial Injuries in Inward Patients in Two Tertiary Care Hospitals in Sri Lanka
Medico-Legal Journal of Sri Lanka, 2020 December Vol. 8, Issue 2 Original article Causes and Characteristics of Peri-Orbital Contusions and Their Relationship with Intracranial Injuries in Inward Patients in Two Tertiary Care Hospitals in Sri Lanka Warushahennadi J1*, Senavirathne AS2, Godakandage SSP3, Pathirana MD4, Jayarathne UGB5, Ambepitiya SGH2 1Department of Forensic Medicine, Faculty of Medicine, University of Ruhuna, Sri Lanka, 2Office of the Judicial Medical Officer, District General Hospital, Matara, Sri Lanka, 3Family Health Bureau, Sri Lanka, 4National Hospital of Sri Lanka, 5Office of the Judicial Medical Officer, Teaching Hospital, Karapitiya, Sri Lanka Abstract Introduction: The peri-orbital contusion (PC) is a common injury in day to day surgical casualties. It is a common injury observed in patients who are in an unconscious state following head injuries. The aim of the study is to describe characteristics of PC and understand its relationship with associated injuries, especially with facial injuries and intracranial injuries. Methods: This retrospective study reviewed the medico-legal examination forms (MLEF) of 67 inward patients in Teaching Hospital, Karapitiya and District General Hospital, Matara with peri-orbital contusions following trauma during a period of six months from January 2020 to June 2020. Results: A total number of 67 patients were included with 81% being male patients. The commonest soft tissue injuries around the PCs were abrasions (n=39, 71%) and 25 (38%) of the study sample had fractures of the skull. The majority (n=22, 88%) of them had fractures of facial bones followed by vault and basal skull fractures. The majority of PCs (45%) were blue in colour and only 8% were red. -
Head Injury Policy
Date January 2020 Review Date January 2021 Responsibility Senior Sister HEAD INJURY POLICY The following has been developed in accordance with NICE clinical guideline 56 - Head Injury, International Rugby Board Concussion Guidelines and the RFU Guidelines for schools and colleges. Background Injuries to the head can occur in many situations in the school environment i.e. any time that pupil’s head comes into contact with a hard object such as the floor, a desk, or another pupil’s body. The potential is probably greatest during activities where collisions can occur such as in the playground, during sport and PE, and if messing around indoors during breaks. The nature of rugby means that concussion can occur during both training and in matches. Concussion is a disturbance of the normal working of the brain without causing any structural damage. It usually follows a blow directly to the head, or indirectly if the head is shaken when the body is struck. It is important to recognise that it is not necessary to lose consciousness to sustain a concussion following a blow to the head. The risk of injury is dependent upon the velocity and the force of the impact, the part of the head involved in the impact, and any pre-existing medical conditions. Symptoms may not develop for some hours, or even days, after a knock to the head, and in rare cases can develop weeks after a head injury. Whilst an initial concussion is unlikely to cause any permanent damage, a repeat injury to the head soon after a prior, unresolved concussion can have serious consequences. -
My Burn Wound Have So That You Can Be Treated for It
ered ‘natures Band-Aid’ as they keep infec- and get help right away. Signs of infection tion out and keep the wound moist and include: redness/heat/swelling around the warm. In such blisters, the body can usual- wound, increased drainage, drainage that ly re-absorb the fluid inside, and; is green or pus and/or foul smelling, in- Break blisters that are large, that keep creased or new pain, and fever (38*C); you from moving your joints or that are in Stop smoking; a spot that may cause the blister to break Eat a well-balanced diet; on its own, or that are filled with unclear Take your medications as prescribed; and/or bloody fluid. Keep your blood sugars in good control (if you have diabetes); Medications Get to and/or maintain a healthy body Burns can be painful, especially superficial and weight; superficial-partial thickness burns, as they involve Avoid using aloe Vera, vitamin E, butter, your nerve endings. It is important that you tell eggs, or table honey on your burns. Alt- your healthcare providers about any pain you hough these treatments are old ‘home My Burn Wound have so that you can be treated for it. Pain con- remedies’, there is little research to say trol may include simple pain medications, like they work. Medical grade honey may be Ibuprofen (Advil) or acetaminophen (Tylenol), or used if your health care provider feels it is stronger pain medications like morphine. right for you; Protect your burn from further injury, In addition to pain medications, your doctor may and; prescribe you anti-anxiety medications and/or Protect your healed burn from the sun Tips on how to care antibiotics. -
OCULAR TRAUMA Accidents and from High Velocity Missiles at the Workplace
!!!!!!!!!!!!!! !Kr!ieg!er !Eye!Ins!tit!ute!at!Sin!ai!Ho!spi!tal ! !!!!!!!!!!!!!! !!!!!!!!!!!!!! !e!y!!e !!l!i!g!!h!!t!s ! !!!!!!!!!!!!!! Spring 2006 of injury can occur from a shattered windschield in road traffic OCULAR TRAUMA accidents and from high velocity missiles at the workplace. Foreign bodies are most frequently found on the cornea and under the eyelid where they EYE INJURY can be easily removed. We have seen a progressive increase in eye trauma resulting Eye injury occurs frequently in the United States where nearly from automobile accidents in the past seven years. Frontal air two million individuals require treatment in the hospital (60%) bag deployment was associated with a statistically significant, or doctor’s office (40%) every year. Males are four times more two-fold increased risk of eye injury, whereas seat belt use was likely than females to have ocular injuries, and eye injuries occur associated with a two-fold reduced eye injury risk. Seat belt use mostly among persons in their 20s or younger. However, as the is the most effective means of occupant protection against auto - population ages, we are seeing an increasing number of eye mobile accident-related eye injury. injuries in the elderly. Older age, being female, passenger seat position and collision Most injuries occur in the home, are sports-related or work- severity were also associated with eye injury risk. related or are the result of an assault or result from a motor vehicle accident. The most common objects EYE PROTECTION to strike the eye are fists, thrown objects Many cases of ocular injury can be prevented by wearing (e.g., stones, balls), BBs, pellets and sticks. -
6 Chemical Skin Burns
53 6 Chemical Skin Burns Magnus Bruze, Birgitta Gruvberger, Sigfrid Fregert Contents aged to a point where there is no return to viability; in other words, a necrosis develops [7, 43, 45]. One 6.1 Introduction . 53 single skin exposure to certain chemicals can result 6.2 Definition . 53 in a chemical burn. These chemicals react with intra- 6.3 Diagnosis . 56 and intercellular components in the skin. However, 6.4 Clinical Features . 56 the action of toxic (irritant) chemicals varies caus- 6.5 Treatment . 57 ing partly different irritant reactions morphologically. 6.6 Complications . 58 They can damage the horny layer, cell membranes, 6.7 Prevention . 59 6.8 Summary . 59 lysosomes, mast cells, leukocytes, DNA synthesis, References . 60 blood vessels, enzyme systems, and metabolism. The corrosive action of chemicals depends on their chem- ical properties, concentration, pH, alkalinity, acidity, temperature, lipid/water solubility, interaction with 6.1 Introduction other substances, and duration and type (for exam- ple, occlusion) of skin contact. It also depends on the Chemical skin burns are particularly common in in- body region, previous skin damage, and possibly on dustry, but they also occur in non-work-related en- individual resistance capacity. vironments. Occupationally induced chemical burns Many substances cause chemical burns only when are frequently noticed when visiting and examining they are applied under occlusion from, for example, workers at their work sites. Corrosive chemicals used gloves, boots, shoes, clothes, caps, face masks, ad- in hobbies are an increasing cause of skin burns. Dis- hesive plasters, and rings. Skin folds may be formed infectants and cleansers are examples of household and act occlusively in certain body regions, e.g., un- products which can cause chemical burns. -
International Council of Ophthalmology and Based on Their Curriculum 2009
HANDBOOK FOR JUNIOR RESIDENTS AND MEDICAL STUDENTS LEARNING EMERGENCY OPHTHALMOLOGY Compiled by The Task Force on Undergraduate Teaching in Ophthalmology of the International Council of Ophthalmology and based on their curriculum 2009 1 In this booklet we have put together common ophthalmic emergency conditions that we think you need to know and key ophthalmic disorders we think you need to have seen. There are descriptions and colour pictures of these conditions. This pocket sized book summaries the key points in the ophthalmology curriculum complied by the Task Force of the International Council of Ophthalmology and is a format that is very portable! Sue Lightman, Do Nhu Hon and Peter McCluskey On behalf of the International Council of Ophthalmology and Vietnam National Institute of Ophthalmology, Hanoi Medical University 2010 Other Contributing Authors with thanks Anh Dinh Kim , Anh Nguyen Quoc, Chau Hoang Thi Minh, Dong Pham Ngoc, Ha Tran Minh, Hon Do Nhu, Ngoc Do Quang, Quan Bui Dao, Richard Andrews, Thang Nguyen Canh, Thanh Pham Thi Kim, Thuy Nguyen Thi Thu, Thuy Vu Thi Bich, Tung Mai Quoc, Van Pham Thi Khanh, Van Pham Trong, Yen Nguyen Thu, Simon Taylor 2 Have you seen? Tick Do you Tick Note for you: if yes know if yes Remember how it is to look it up caused and treated? Trauma Periorbital haematoma Orbital blowout Lid laceration Subconjunctival Haemorrhage Chemical burns – cornea and conjunctiva Foreign body Corneal abrasion Hyphema Iridodialysis Cataract Lens subluxation /dislocation Intraocular foreign body Scleral rupture 3 Painful Red Eye Chalazion Dacryocystitis Orbital cellulitis Conjunctivitis Scleritis Episcleritis Viral keratitis Bacterial keratitis Shingles Uveitis Acute angle-closure glaucoma Endophthalmitis Sudden Painless Loss of Vision Vitreous haemorrhage Retinal tear/detachment Central retinal artery occlusion Central retinal vein occlusion Others 4 Proptosis VII nerve palsy TRAUMA Ocular trauma is very common, especially in developing countries. -
Chemical Burn Injuries
DERLEME/ REVİEW Kocaeli Med J 2018; 7; 1:54-58 Chemical Burn Injuries Kimyasal Yanıklar Ayten Saraçoğlu1, Mehmet Yılmaz2, Kemal Tolga Saraçoğlu2 1Marmara Üniversitesi Tıp Fakültesi, Anesteziyoloji ve Reanimasyon Anabilim Dalı, İstanbul, Türkiye 2Sağlık Bilimleri Üniversitesi Tıp Fakültesi, Derince SUAM Anesteziyoloji ve Reanimasyon Kliniği, Kocaeli, Türkiye ÖZET ABSTRACT Kimyasal yanıklar sıklıkla koroziv maddelere maruziyet Chemical burns often develop after exposure to corrosive sonrasında gelişmektedirler. Tüm yanık türlerinin %10,7’sini, substances. They include 10.7% of all burn types and 2-6% of yanık merkezine hasta kabullerinin de %2-6’sını the patient admissions to the burn center. Chemical compounds oluşturmaktadır. Kimyasal komponentlere bağlı hasar 6 farklı possess 6 different types of damaging mechanisms; reduction, mekanizmayla ortaya çıkmaktadır. Bunlar redüksyon, oxidation, corrosion, protoplasmic toxins, vesicants and oksidasyon, korozyon, protoplazmik toksinler, yakıcı desiccants. The characteristics of chemical burn injuries kimyasallar ve kurutuculardır. Kimyasal yanık hasarının include skin discoloration and contractures, having rarely karakteristikleri arasında ciltte renk değişiklikleri ve korozyon, regular shape, perforation in the gastrointestinal tract with the nadiren regüler bir yapı, gastrointestinal kanalda perforasyon, risk of severe systemic toxicity and mortality. Compared to the ciddi sistemik toksisite ve mortalite riski yer almaktadır. thermal burns, the wound healing process following chemical Termal yanıklarla karşılaştırıldığında yara iyileşme süreci burn injuries is markedly slower and also frequently related belirgin derecede daha yavaş olup sıklıkla hastanede uzamış with a prolonged stay at the hospital. Moreover, generally the yatış süresiyle ilişkilidir. Ayrıca yanık hasarı genellikle burn injury results following a prolonged exposure to the kimyasal ajana uzamış maruziyet sonrasında oluşmaktadır. chemical agent. White phosphorus burns are good examples Beyaz fosfor yanıkları bunun iyi bir örneğidir.