208 Dysbarism and Complications of Diving

Total Page:16

File Type:pdf, Size:1020Kb

208 Dysbarism and Complications of Diving References 1 208 Dysbarism and Complications of Diving REFERENCES 1. Strauss MB, Borer RC: Diving medicine: contemporary topics and their controversies. Am J Emerg Med 19: 232, 2001. 2. Becker GD, Parell GJ: Barotrauma of the ears and sinuses after scuba diving. Euro Arch Otorhinolaryngol 258: 159, 2001. 3. Harker CP, Neuman TS, Olson LK, et al: The roentgenographic findings associated with air embolism in sport SCUBA divers. J Emerg Med 11: 443, 1993. 4. Neuman TS: Arterial gas embolism and decompression sickness. News Physiol Sci 17: 77, 2001. 5. Doolette DJ, Mitchell SJ: The physiologic kinetics of nitrogen and the prevention of decompression sickness. Clin Pharmacokinet 40: 1, 2001. 6. Neuman TS, Bove AA: Combined arterial gas embolism and decompression sickness following no-stop dives. Undersea Biomed Res 17: 429, 1990. 7. Millar I: Post diving altitude exposure. SPUMS J 26: 135, 1996. 8. Newton HB: Neurologic complications of scuba diving. Am Fam Physician 63: 2211, 2001. 9. Grover IR, Reed W, Neuman TS: The SANDHOG criteria and its validation for the diagnosis of DCS arising from bounce diving. Undersea Hyper Med 34: 165, 2007. 10. Martin JD, Thom SR: Vascular leukocyte sequestration in decompression sickness and prophylactic hyperbaric oxygen therapy in rats. Aviat Space Environ Med 73: 565, 2002. 11. Cianci P, Slade JB Jr: Delayed treatment of decompression sickness with short, no-air- break tables: review of 140 cases. Aviat Space Environ Med 77: 1003, 2006. 12. Catron PW, Flynn ET: Adjuvant drug therapy for decompression sickness: a review. Undersea Biomed Res 9: 161, 1982. 13. Dutka AJ, Mink R, McDermott J, et al: Effect of lidocaine on somatosensory evoked response and cerebral blood flow after canine cerebral air embolism. Stroke 23: 1515, 1992. 14. Mitchell SJ, Pellett O, Gorman DF: Cerebral protection by lidocaine during cardiac operations. Ann Thorac Surg 67: 1117, 1999. 15. Mitchell SJ: Lidocaine for the treatment of decompression illness: a review of the liter- ature. Undersea Hyper Med 28: 165, 2001. 16. Tetzlaff K, Muth CM, Waldhauser LK: A review of asthma and scuba diving. J Asthma 39: 557, 2002. 17. Hampson NB, Dunford RG: Pulmonary edema of scuba divers. Undersea Hyper Med 24: 29, 1997. 18. Demchenko IT, Boso AE, O’Neill TJ, et al: Nitric oxide and cerebral blood flow responses to hyperbaric oxygen. J Appl Physiol 88: 1381, 2000. ■ USEFUL WEB RESOURCES Divers Alert Network—http://www.diversalertnetwork.org DAN Japan—http://www.danjapan.gr.jp DAN Europe—http://www.daneurope.org DAN Asia-Pacific—http://www.danasiapacific.org DAN Southern Africa—http://www.dansa.org Undersea and Hyperbaric Medical Society—http://www.uhms.org Copyright © 2010 The McGraw-Hill Companies, Inc. All rights reserved..
Recommended publications
  • Heat Stroke Heat Exhaustion
    Environmental Injuries Co lin G. Ka ide, MD , FACEP, FAAEM, UHM Associate Professor of Emergency Medicine Board-Certified Specialist in Hyperbaric Medicine Specialist in Wound Care The Ohio State University Wexner Medical Center The Most Dangerous Drug Combination… Accidental Testosterone Hypothermia and Alcohol! The most likely victims… Photo: Ralf Roletschek 1 Definition of Blizzard Hypothermia of Subnormal T° when the body is unable to generate sufficient heat to sustain normal functions Core Temperature < 95°F 1979 (35°C) Most Important Temperatures Thermoregulation 95°F (35° C) Hyper/Goofy The body uses a Poikilothermic shell to maintain a Homeothermic core 90°F (32°C) Shivering Stops Maintains core T° w/in 1.8°F(1°C) 80°F (26. 5°C) Vfib, Coma Hypothalamus Skin 65°F (18°C) Asystole Constant T° 96.896.8-- 100.4° F 2 Thermoregulation The 2 most important factors Only 3 Causes! Shivering (10x increase) Decreased Heat Production Initiated by low skin temperature Increased Heat Loss Warming the skin can abolish Impaired Thermoregulation shivering! Peripheral vasoconstriction Sequesters heat Predisposing Predisposing Factors Factors Decreased Production Increased Loss –Endocrine problems Radiation Evaporation • Thyroid Conduction* • Adrenal Axis Convection** –Malnutrition *Depends on conducting material **Depends on wind velocity –Neuromuscular disease 3 Predisposing Systemic Responses CNS Factors T°< 90°F (34°C) Impaired Regulation Hyperactivity, excitability, recklessness CNS injury T°< 80°F (27°C) Hypothalamic injuries Loss of voluntary
    [Show full text]
  • Environmental Injuries
    Environmental Injuries Colin G. Kaide, MD, FACEP, FAAEM, UHM Associate Professor of Emergency Medicine Board-Certified Specialist in Hyperbaric Medicine Specialist in Wound Care The Ohio State University Wexner Medical Center 1 The Most Dangerous Drug Combination… Testosterone and Alcohol! The most likely victims… Photo: Ralf Roletschek Accidental Hypothermia 2 Blizzard of 1979 Definition of Hypothermia Subnormal T° when the body is unable to generate sufficient heat to sustain normal functions Core Temperature < 95°F (35°C) 3 Most Important Temperatures 95°F (35° C) Hyper/Goofy 90°F (32°C) Shivering Stops 80°F (26.5°C) Vfib, Coma 65°F(18F (18°C) AtlAsystole Thermoregulation The body uses a Poikilothermic shell to maintain a Homeothermic core Maintains core T° w/in 1.8°F(1°C) Hypothalamus Skin CttTConstant T° 96. 8- 100.4° F 4 Thermoregulation The 2 most important factors Shivering (10x increase) Initiated by low skin temperature Warming the skin can abolish shivering! Peripheral vasoconstriction Sequesters heat Only 3 Causes! Decreased Heat Production Increased Heat Loss Impaired Thermoregulation 5 Predisposing Factors Decreased Production –Endocrine problems • Thyroid • Adrenal Axis –Malnutrition –Neuromuscular disease Predisposing Factors Increased Loss RRditiadiation Evaporation Conduction* Convection** *DDdepends on cond dtitilucting material **Depends on wind velocity 6 Predisposing Factors Impaired Regulation CNS injury Hypothalamic injuries Peripheral Injury Atherosclerosis Neuropathy Interfering Agents Systemic Responses CNS
    [Show full text]
  • Dysbarism - Barotrauma
    DYSBARISM - BAROTRAUMA Introduction Dysbarism is the term given to medical complications of exposure to gases at higher than normal atmospheric pressure. It includes barotrauma, decompression illness and nitrogen narcosis. Barotrauma occurs as a consequence of excessive expansion or contraction of gas within enclosed body cavities. Barotrauma principally affects the: 1. Lungs (most importantly): Lung barotrauma may result in: ● Gas embolism ● Pneumomediastinum ● Pneumothorax. 2. Eyes 3. Middle / Inner ear 4. Sinuses 5. Teeth / mandible 6. GIT (rarely) Any illness that develops during or post div.ing must be considered to be diving- related until proven otherwise. Any patient with neurological symptoms in particular needs urgent referral to a specialist in hyperbaric medicine. See also separate document on Dysbarism - Decompression Illness (in Environmental folder). Terminology The term dysbarism encompasses: ● Decompression illness And ● Barotrauma And ● Nitrogen narcosis Decompression illness (DCI) includes: 1. Decompression sickness (DCS) (or in lay terms, the “bends”): ● Type I DCS: ♥ Involves the joints or skin only ● Type II DCS: ♥ Involves all other pain, neurological injury, vestibular and pulmonary symptoms. 2. Arterial gas embolism (AGE): ● Due to pulmonary barotrauma releasing air into the circulation. Epidemiology Diving is generally a safe undertaking. Serious decompression incidents occur approximately only in 1 in 10,000 dives. However, because of high participation rates, there are about 200 - 300 cases of significant decompression illness requiring treatment in Australia each year. It is estimated that 10 times this number of divers experience less severe illness after diving. Physics Boyle’s Law: The air pressure at sea level is 1 atmosphere absolute (ATA). Alternative units used for 1 ATA include: ● 101.3 kPa (SI units) ● 1.013 Bar ● 10 meters of sea water (MSW) ● 760 mm of mercury (mm Hg) ● 14.7 pounds per square inch (PSI) For every 10 meters a diver descends in seawater, the pressure increases by 1 ATA.
    [Show full text]
  • Delayed Treatment of Decompression Sickness with Short, No-Air-Break Tables: Review of 140 Cases
    REVIEW ARTICLE Delayed Treatment of Decompression Sickness with Short, No-Air-Break Tables: Review of 140 Cases Paul Cianci and John B. Slade,Jr. CIANCI P, SLADE JR JB. Delayed treatment of decompression sick- increasing frequency, often having dived very provoc- ness with short, no-air-break tables: review of 140 cases. Aviat Space ative profiles, many suffering from severe DCS, and Environ Med 2006; 77:1003–8. Introduction: Most cases of decompression sickness (DCS) in the U.S. with long delays to treatment. are treated with hyperbaric oxygen using U.S. Navy Treatment Tables 5 In 1963 and 1964, the Navy Experimental Dive Unit and 6, although detailed analysis shows that those tables were based on received reports of 133 cases of DCS in which the stan- limited data. We reviewed the development of these protocols and offer dard USN tables at the time were used (28). Full relief an alternative treatment table more suitable for monoplace chambers did not result in 24% of initial recompressions. When that has proven effective in the treatment of DCS in patients presenting to our facility. Methods: We reviewed the outcomes for 140 cases of outcomes using USN Tables 3 and 4 were analyzed, a DCS in civilian divers treated with the shorter tables at our facility from 47% incidence of failure of the first treatment was January 1983 through December 2002. Results: Onset of symptoms noted. However, there were no instances of treatment averaged 9.3 h after surfacing. At presentation, 44% of the patients failure when DCS had occurred following rigid USN demonstrated mental aberration.
    [Show full text]
  • Short Update on Dysbaric Osteonecrosis: Concepts and Decompression Management
    Dysbaric Osteonecrosis Review Short Update on Dysbaric Osteonecrosis: Concepts and Decompression Management Niladri Kumar Mahato1 Abstract Dysbaric osteonecrosis (DON) is caused by inadequate decompression after exposure to very high ambient air pressures. Different pathological events have been shown to occur in conjunction to result in DON. New observations from clinical and experimental data in this field have led investigators to reformat the etiology, pathogenesis and modify existing modalities of treatment of DON. This short review revisits concepts related to pathophysiology of DON occurring as a part of generalized decompression sickness and discusses newer therapeutic insights stemming from recent advancements in DON research. Keywords: Decompression; Dysbarism; Embolism; Hyperbaric Juxta-articular; Metaphysis; Rhizomelic J Bangladesh Soc Physiol. 2015, December; 10(2): 76-81 For Authors Affiliation, see end of text. http://www.banglajol.info/index.php/JBSP Introduction ysbarism or musculoskeletal pointed towards several etiologies of DON8-13. decompression sickness (DCS) is Given the typical anatomy of vasculature around Dusually observed in people who undergo the end of long bones, it seems plausible that deep-sea diving or are exposed to environments DON is linked to occurrence of micro-embolisms of high air pressures1-5. Dysbarism or Caisson with gas or lipid bubbles following a rapid Disease (CD) is characterized by an array of decrease in surrounding air pressure at those systemic complications involving soft tissues, the sites3,10,14-17. Other investigators have proposed cardio-pulmonary, nervous, renal and external compression of blood flow in such musculoskeletal systems when individuals or vessels due to similar bubbles arising in structures experimental animals are brought back to normal around an artery or a vein to induce external pressure.
    [Show full text]
  • Cardiovascular Responses to Diving in the Turtle, Pseudemys Scripta Stuart Keith Ware Iowa State University
    Iowa State University Capstones, Theses and Retrospective Theses and Dissertations Dissertations 1980 Cardiovascular responses to diving in the turtle, Pseudemys scripta Stuart Keith Ware Iowa State University Follow this and additional works at: https://lib.dr.iastate.edu/rtd Part of the Animal Sciences Commons, Physiology Commons, and the Veterinary Physiology Commons Recommended Citation Ware, Stuart Keith, "Cardiovascular responses to diving in the turtle, Pseudemys scripta " (1980). Retrospective Theses and Dissertations. 6813. https://lib.dr.iastate.edu/rtd/6813 This Dissertation is brought to you for free and open access by the Iowa State University Capstones, Theses and Dissertations at Iowa State University Digital Repository. It has been accepted for inclusion in Retrospective Theses and Dissertations by an authorized administrator of Iowa State University Digital Repository. For more information, please contact [email protected]. INFORMATION TO USERS This was produced from a copy of a document sent to us for microfilming. While the most advanced technological means to photograph and reproduce this document have been used, the quality is heavily dependent upon the quality of the material submitted. The following explanation of techniques is provided to help you understand markings or notations which may appear on this reproduction. 1. The sign or "target" for pages apparently lacking from the document photographed is "Missing Page(s)". If it was possible to obtain the missing page(s) or section, they are spliced into the film along with adjacent pages. This may have necessitated cutting through an image and duplicating adjacent pages to assure you of complete continuity. 2. When an image on the film is obliterated with a round black mark it is an indication that the film inspector noticed either blurred copy because of movement during exposure, or duplicate copy.
    [Show full text]
  • Power Equation for Predicting the Risk of Central Nervous System Oxygen Toxicity at Rest
    fphys-11-01007 August 17, 2020 Time: 14:44 # 1 ORIGINAL RESEARCH published: 17 August 2020 doi: 10.3389/fphys.2020.01007 Power Equation for Predicting the Risk of Central Nervous System Oxygen Toxicity at Rest Ben Aviner1, Ran Arieli1,2* and Alexandra Yalov3 1 The Israel Naval Medical Institute, Israel Defense Forces Medical Corps, Haifa, Israel, 2 Eliachar Research Laboratory, Western Galilee Medical Center, Nahariya, Israel, 3 HP – Indigo Division, Nes Ziona, Israel Patients undergoing hyperbaric oxygen therapy and divers engaged in underwater activity are at risk of central nervous system oxygen toxicity. An algorithm for predicting CNS oxygen toxicity in active underwater diving has been published previously, but not for humans at rest. Using a procedure similar to that employed for the derivation of our active diving algorithm, we collected data for exposures at rest, in which subjects breathed hyperbaric oxygen while immersed in thermoneutral water at 33◦C(n = 219) Edited by: or in dry conditions (n = 507). The maximal likelihood method was employed to solve for Costantino Balestra, Haute École Bruxelles-Brabant the parameters of the power equation. For immersion, the CNS oxygen toxicity index 2 10:93 (HE2B), Belgium is KI = t × PO2 , where the calculated risk from the Standard Normal distribution 0:5 2 12:99 Reviewed by: is ZI = [ln(KI ) – 8.99)]/0.81. For dry exposures this is KD = t × PO2 , with risk Enrico M. Camporesi, 0:5 ZD = [ln(KD ) – 11.34)]/0.65. We propose a method for interpolating the parameters at USF Health, United States Thijs Wingelaar, metabolic rates between 1 and 4.4 MET.
    [Show full text]
  • Decompression Sickness in Inside Attendants
    SECTION VI 643 CHAPTER 2 DECOMPRESSION SICKNESS IN INSIDE ATTENDANTS PAUL J. SHEFFIELD AND CHRISTY J. PIRONE WHAT IS DECOMPRESSION SICKNESS? Decompression Sickness (DCS) Defined: DCS is an illness that occurs when environmental pressure is reduced sufficiently to cause gases that are dissolved in body tissues to evolve as bubbles. Primarily consisting of nitrogen, the bubbles evolve from solution when the inside attendant surfaces too fast for the body to compensate. Patients do not have the problem because the oxygen they breathe during hyperbaric oxygen treatment elimi- nates the nitrogen from their bodies. Signs and Symptoms of DCS: Bubbles that cause DCS can form in all parts of the body and the anatomic location accounts for the variety of signs and symptoms. (1) DCS can manifest itself from minor to life-threatening symptoms. Minor skin itching or tingling usually passes within 20 to 30 min- utes, and no treatment is necessary, all other forms of DCS are treated in the hyperbaric chamber with immediate compression and hyperbaric oxygen. “Bends pain” seen in about 90% of cases, may appear anywhere in the body, but is more frequent in legs or arms that were exercised during the exposure. Neurologic symptoms involving the brain or spinal cord occur in about 25% of cases, and are manifested by a wide variety of symptoms but mainly by headache, numbness, paralysis of an arm or leg, loss of sensation, vertigo, visu- al distortions or blindness, and extreme fatigue. Chokes is a rare but life-threat- ening respiratory disorder caused by gas emboli in the lungs, and manifested by wheezing, chest pain, or troublesome cough.
    [Show full text]
  • Time to Treatment for Decompression Illness
    Health and Safety Executive Time to treatment for decompression illness Prepared by North Sea Medical Centre for the Health and Safety Executive 2007 RR550 Research Report Health and Safety Executive Time to treatment for decompression illness Dr Werner Stipp North Sea Medical Centre 3 Lowestoft Road Gorleston on Sea Great Yarmouth Norfolk NR 31 6SG Hyperbaric oxygen treatment (HBO) is the standard and definitive treatment for divers with decompression illness (DCI). There is conflicting evidence in the medical literature on whether DCI is more responsive to early rather than late treatment with hyperbaric oxygen (HBO). The main aim of this study is to investigate the influence of time to treatment with HBO in divers with neurological DCI. Here we show that early HBO treatment in divers with neurological DCI is robustly associated with a better outcome. There is a suggestion in this study that divers with DCI are less responsive if HBO treatment is delayed for 350 minutes or more (or approximately six hours) after surfacing from the incident dive. An interesting observation in this study is that if normobaric oxygen is administered before HBO, it tends to protect divers against delay in treatment with HBO. An additional analysis provides medical evidence that time limits for HBO as specified in the current ACOP1 should remain in place. This study recommends that a time to treatment action plan (TTT action plan), which specifies what to do when a diver develops suspicious DCI symptoms, will help to ensure that divers have prompt HBO treatment. Divers with serious DCI should be given the appropriate level of medical care in a hospital setting.
    [Show full text]
  • Wisconsin Standardized Intermediate Curriculum December 2012 Wisconsin Department of Health Services
    Wisconsin Standardized Intermediate Curriculum December 2012 Wisconsin Department of Health Services This page is intentionally blank. 2012 – Wisconsin Intermediate Curriculum Table of Contents 0.0 – INTRODUCTION .......................................................................................................................................................... 13 0.1 – WISCONSIN INTERMEDIATE PROGRAM OUTCOMES ............................................................................................. 13 0.2 – CURRICULUM BACKGROUND AND EMS TRAINING CENTER ADAPTATION ....................................................... 13 0.3 – PROGRAM PREREQUISITES / PRESUMPTION OF PREREQUISITE EDUCATION .................................................. 13 0.4 – WISCONSIN 2012 INTERMEDIATE CURRICULUM COMMITTEE MEMBERS ...................................................... 14 0.5 – COURSE STRUCTURE AND TOPICAL HOUR GUIDELINES ...................................................................................... 14 0.6 – CLINICAL AND FIELD EXPERIENCES, MINIMUM HOURS AND COMPETENCY REQUIREMENTS ....................... 16 1.0 – PREPARATORY ............................................................................................................................................................ 20 1.1 – EMS SYSTEMS ............................................................................................................................................................. 20 1.1.1 - Quality Improvement ..........................................................................................................................................
    [Show full text]
  • Diving and Hyperbaric Medicine Journal 2020;50(1)
    Diving and Hyperbaric Medicine The Journal of the South Pacific Underwater Medicine Society and the European Underwater and Baromedical Society© Volume 50 No. 1 March 2020 Exhaled breath to measure pulmonary O2 toxicity? Predictors of outcome in fisherman-divers with serious DCS HBOT effect on endothelial function in diabetics Blood viscosity after prolonged cold-water immersion Occupational diver satisfaction with health surveillance Efficiency of different first aid oxygen administration devices Validation of a US Navy air diving decompression profile Diving with hypertension and antihypertensive drugs Wearable sensors in breath-hold diving Pulmonary barotrauma radiology Cerebral oxygen toxicity at one atmosphere inspired oxygen? E-ISSN 2209-1491 ABN 29 299 823 713 CONTENTS Diving and Hyperbaric Medicine Volume 50 No.1 March 2020 1 The Editor's offering Letter to the Editor Original articles 75 Effect of an air break on the occurrence of seizures in 2 Assessment of pulmonary oxygen toxicity in special hyperbaric oxygen therapy operations forces divers under operational circumstances may be predicted by the power using exhaled breath analysis equation for hyperoxia at rest Thijs T Wingelaar, Paul Brinkman, Rigo Hoencamp, Pieter-Jan AM van Ran Arieli Ooij, Anke-Hilse Maitland-van der Zee, Markus W Hollmann, Rob A van Hulst 9 Factors influencing the severity of long-term sequelae in Obituary fishermen-divers with neurological decompression sickness Jean-Eric Blatteau, Kate Lambrechts, Jean Ruffez 77 Francis Alfred (Frank) Blackwood 17 Measurement
    [Show full text]
  • Report of the Decompression Illness Adjunctive Therapy Committee of the Undersea and Hyperbaric Medical Society
    REPORT OF THE DECOMPRESSION ILLNESS ADJUNCTIVE THERAPY COMMITTEE OF THE UNDERSEA AND HYPERBARIC MEDICAL SOCIETY Including Proceedings of the Fifty-Third Workshop of the Undersea and Hyperbaric Medical Society Chaired by Richard E. Moon, MD Editor Richard E. Moon, MD Copyright ©2003 Undersea and Hyperbaric Medical Society, Inc. 10531 Metropolitan Avenue Kensington, MD 20895, USA ISBN 0-930406-22-2 The views, opinions, and findings contained herein are those of the author(s) and should not be construed as an official Agency position, policy, or decision unless so designated by other official documentation. Authors Richard D. Vann, Ph.D. Assistant Research Professor of Anesthesiology Duke University Medical Center Director of Research, Divers Alert Network Durham, NC 27710 Edward D. Thalmann, M.D. Assistant Clinical Professor of Anesthesiology Assistant Clinical Professor of Occupational and Environmental Medicine Duke University Medical Center Assistant Medical Director, Divers Alert Network Durham, NC 27710 John M. Hardman, M.D. Professor and Chairman Dept. of Pathology John A. Burns School of Medicine University of Hawaii Honolulu, HI 96822 Ward Reed, M.D., M.P.H. Fellow in Undersea and Hyperbaric Medicine Duke University Medical Center Durham, NC 27710 W. Dalton Dietrich, Ph.D. Departments of Neurological Surgery and Neurology The Miami Project to Cure Paralysis University of Miami School of Medicine Miami, FL 33101 Frank Butler, CAPT, MC, USN Biomedical Research Director Naval Special Warfare Command Pensacola, FL Richard E. Moon, M.D. Professor of Anesthesiology Associate Professor of Medicine Medical Director, Center for Hyperbaric Medicine and Environmental Physiology Duke University Medical Center Medical Director, Divers Alert Network Durham, NC 27710 2 Joseph Dervay, M.D., M.M.S., F.A.C.E.P Flight Surgeon Medical Operations and Crew Systems NASA Johnson Space Center Houston, TX 77058 David S.
    [Show full text]