Wound Care Hyperbaric Medicine
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Therapeutic Hypothermia: Where Do We Stand?
5/29/2015 Therapeutic Hypothermia: Where Do We Stand? Melina Aguinaga-Meza, MD Assistant Professor of Medicine Gill Heart Institute University of Kentucky Disclosure Information Melina Aguinaga-Meza, MD “Therapeutic Hypothermia: Where Do We Stand?” • FINANCIAL DISCLOSURE: – No relevant financial relationship exists • UNLABELED/UNAPPROVED USES DISCLOSURE: – No relevant relationship exists 1 5/29/2015 The Clinical Problem • Out-of-hospital cardiac arrest (OHCA) is a leading cause of death among adults in the US • Approx. 300,000 OHCA events occur each year in the US • Resuscitation is attempted in 100,000 of these arrests • Less than 40 000 survive to hospital admission MMWR / July 29, 2011 / Vol. 60 / No. 8 2 5/29/2015 Consequences From Cardiac Arrest Myocardial Brain injury dysfunction Post-Cardiac Arrest Syndrome Systemic ischemia Disorder that + reperfusion caused the cardiac responses arrest • The effects of this syndrome are severe and pervasive MMWR / July 29, 2011 / Vol. 60 / No. 8 Survival and Neurological Outcomes after OHCA • Only one third of patients admitted to the hospital survive to hospital discharge • Approx. one out of ten people who experience OHCA survive to hospital discharge • Only 2 out of 3 of them have a good/moderate neurologic recovery MMWR / July 29, 2011 / Vol. 60 / No. 8: CARES 3 5/29/2015 “Chain of Survival” • Actions needed to improve chances of survival from out-of-hospital cardiac arrest Circulation 2010; 122:S676-84 • Try to identify and treat the precipitating causes of the arrest and prevent recurrent -
Hypothermia and Respiratory Heat Loss While Scuba Diving
HYPOTHERMIA AND RESPIRATORY HEAT LOSS WHILE SCUBA DIVING Kateřina Kozáková Faculty of Physical Education and Sport, Charles University in Prague, Department of Biomedical Labo- ratory Abstract One of the factors affecting length of stay under water of a diver is heat comfort. During scuba diving there is an increased risk of hypothermia. Hypothermia is one of the most life threatening factors of a diver and significantly affects his performance. The body heat loss runs by different mechanisms. One of them is the respiratory mechanism, which is often overlooked. Compressed dry air or other media is coming out from the cylinder, which have to be heated and humidified to a suitable value. Thus the organism loses body heat and consequently energy. Based on literature search the article will describe safe dive time in terms of hypo- thermia in connection to respiratory heat loss. Key words: hypothermia, heat loss, respiration, scuba diving, water environment Souhrn Jedním z faktorů ovlivňujících délku pobytu potápěče pod vodou je tepelný komfort. Během výkonu přístro- jového potápění hrozí zvýšené riziko hypotermie. Hypotermie představuje jedno z nejzávažnějších ohrožení života potápěče a zásadně ovlivňuje jeho výkon. Ke ztrátám tělesného tepla dochází různými mechanismy. Jednou cestou tepelných ztrát je ztráta tepla dýcháním, která je často opomíjená. Z potápěčského přístroje vychází suchý stlačený vzduch nebo jiné médium, který je třeba při dýchání ohřát a zvlhčit na potřebnou hodnotu. Tím organismus ztrácí tělesné teplo a potažmo energii. Tento článek, na základě literární rešerše, popíše bezpečnou dobou ponoru z hlediska hypotermie a v souvislosti se ztrátou tepla dýcháním. Klíčová slova: hypotermie, ztráta tepla, dýchání, přístrojové potápění, vodní prostředí Introduction amount of body heat. -
Alterations in the Specific Gravity of the Blood Plasma with Onset of Diuresis in Heart Failure
MECHANISM OF DIURESIS: ALTERATIONS IN THE SPECIFIC GRAVITY OF THE BLOOD PLASMA WITH ONSET OF DIURESIS IN HEART FAILURE Harold J. Stewart J Clin Invest. 1941;20(1):1-6. https://doi.org/10.1172/JCI101189. Research Article Find the latest version: https://jci.me/101189/pdf MECHANISM OF DIURESIS: ALTERATIONS IN THE SPECIFIC GRAVITY OF THE BLOOD PLASMA WITH ONSET OF DIURESIS IN HEART FAILURE By HAROLD J. STEWART (From the Department of Medicine of.the New York Hospital and Cornell University Medical College and the Hospital of the Rockefeller Institute for Medical Research, New York) (Received for publication July 3, 1940) There are divergent views concerning the Moreover, its duration may be brief before res- mechanism by which diuresis is initiated. Many toration is attempted, or it may be long enough of the observations on this subject relate to mer- and of such magnitude that it can be detected. curial drugs. Crawford and McIntosh (1) con- On the other hand, if diuresis is initiated at cluded that novasurol induced primary dilution, the tissue side of the system so that fluid enters followed by concentration of the blood in edema- the blood stream first, dilution of the blood would tous patients. Bryan, Evans, Fulton, and Stead occur. Equilibrium would be disturbed until the (2) thought that salyrgan resulted in concentra- kidneys began to excrete the surplus fluid. If di- tion of the blood, since sustained rise in its spe- lution of the blood was of sufficient duration and cific gravity occurred coincident with diuresis in magnitude, it might be detected. -
Critical Care in the Monoplace Hyperbaric Chamber
Critical Care in the Monoplace Hyperbaric Critical Care - Monoplace Chamber • 30 minutes, so only key points • Highly suggest critical care medicine is involved • Pitfalls Lindell K. Weaver, MD Intermountain Medical Center Murray, Utah, and • Ventilator and IV issues LDS Hospital Salt Lake City, Utah Key points Critical Care in the Monoplace Chamber • Weaver LK. Operational Use and Patient Care in the Monoplace Chamber. In: • Staff must be certified and experienced Resp Care Clinics of N Am-Hyperbaric Medicine, Part I. Moon R, McIntyre N, eds. Philadelphia, W.B. Saunders Company, March, 1999: 51-92 in CCM • Weaver LK. The treatment of critically ill patients with hyperbaric oxygen therapy. In: Brent J, Wallace KL, Burkhart KK, Phillips SD, and Donovan JW, • Proximity to CCM services (ed). Critical care toxicology: diagnosis and management of the critically poisoned patient. Philadelphia: Elsevier Mosby; 2005:181-187. • Must have study patient in chamber • Weaver, LK. Critical care of patients needing hyperbaric oxygen. In: Thom SR and Neuman T, (ed). The physiology and medicine of hyperbaric oxygen therapy. quickly Philadelphia: Saunders/Elsevier, 2008:117-129. • Weaver LK. Management of critically ill patients in the monoplace hyperbaric chamber. In: Whelan HT, Kindwall E., Hyperbaric Medicine Practice, 4th ed.. • CCM equipment North Palm Beach, Florida: Best, Inc. 2017; 65-95. • Without certain modifications, treating • Gossett WA, Rockswold GL, Rockswold SB, Adkinson CD, Bergman TA, Quickel RR. The safe treatment, monitoring and management -
Heat Stroke Heat Exhaustion
Environmental Injuries Co lin G. Ka ide, MD , FACEP, FAAEM, UHM Associate Professor of Emergency Medicine Board-Certified Specialist in Hyperbaric Medicine Specialist in Wound Care The Ohio State University Wexner Medical Center The Most Dangerous Drug Combination… Accidental Testosterone Hypothermia and Alcohol! The most likely victims… Photo: Ralf Roletschek 1 Definition of Blizzard Hypothermia of Subnormal T° when the body is unable to generate sufficient heat to sustain normal functions Core Temperature < 95°F 1979 (35°C) Most Important Temperatures Thermoregulation 95°F (35° C) Hyper/Goofy The body uses a Poikilothermic shell to maintain a Homeothermic core 90°F (32°C) Shivering Stops Maintains core T° w/in 1.8°F(1°C) 80°F (26. 5°C) Vfib, Coma Hypothalamus Skin 65°F (18°C) Asystole Constant T° 96.896.8-- 100.4° F 2 Thermoregulation The 2 most important factors Only 3 Causes! Shivering (10x increase) Decreased Heat Production Initiated by low skin temperature Increased Heat Loss Warming the skin can abolish Impaired Thermoregulation shivering! Peripheral vasoconstriction Sequesters heat Predisposing Predisposing Factors Factors Decreased Production Increased Loss –Endocrine problems Radiation Evaporation • Thyroid Conduction* • Adrenal Axis Convection** –Malnutrition *Depends on conducting material **Depends on wind velocity –Neuromuscular disease 3 Predisposing Systemic Responses CNS Factors T°< 90°F (34°C) Impaired Regulation Hyperactivity, excitability, recklessness CNS injury T°< 80°F (27°C) Hypothalamic injuries Loss of voluntary -
Environmental Injuries
Environmental Injuries Colin G. Kaide, MD, FACEP, FAAEM, UHM Associate Professor of Emergency Medicine Board-Certified Specialist in Hyperbaric Medicine Specialist in Wound Care The Ohio State University Wexner Medical Center 1 The Most Dangerous Drug Combination… Testosterone and Alcohol! The most likely victims… Photo: Ralf Roletschek Accidental Hypothermia 2 Blizzard of 1979 Definition of Hypothermia Subnormal T° when the body is unable to generate sufficient heat to sustain normal functions Core Temperature < 95°F (35°C) 3 Most Important Temperatures 95°F (35° C) Hyper/Goofy 90°F (32°C) Shivering Stops 80°F (26.5°C) Vfib, Coma 65°F(18F (18°C) AtlAsystole Thermoregulation The body uses a Poikilothermic shell to maintain a Homeothermic core Maintains core T° w/in 1.8°F(1°C) Hypothalamus Skin CttTConstant T° 96. 8- 100.4° F 4 Thermoregulation The 2 most important factors Shivering (10x increase) Initiated by low skin temperature Warming the skin can abolish shivering! Peripheral vasoconstriction Sequesters heat Only 3 Causes! Decreased Heat Production Increased Heat Loss Impaired Thermoregulation 5 Predisposing Factors Decreased Production –Endocrine problems • Thyroid • Adrenal Axis –Malnutrition –Neuromuscular disease Predisposing Factors Increased Loss RRditiadiation Evaporation Conduction* Convection** *DDdepends on cond dtitilucting material **Depends on wind velocity 6 Predisposing Factors Impaired Regulation CNS injury Hypothalamic injuries Peripheral Injury Atherosclerosis Neuropathy Interfering Agents Systemic Responses CNS -
Dysbarism - Barotrauma
DYSBARISM - BAROTRAUMA Introduction Dysbarism is the term given to medical complications of exposure to gases at higher than normal atmospheric pressure. It includes barotrauma, decompression illness and nitrogen narcosis. Barotrauma occurs as a consequence of excessive expansion or contraction of gas within enclosed body cavities. Barotrauma principally affects the: 1. Lungs (most importantly): Lung barotrauma may result in: ● Gas embolism ● Pneumomediastinum ● Pneumothorax. 2. Eyes 3. Middle / Inner ear 4. Sinuses 5. Teeth / mandible 6. GIT (rarely) Any illness that develops during or post div.ing must be considered to be diving- related until proven otherwise. Any patient with neurological symptoms in particular needs urgent referral to a specialist in hyperbaric medicine. See also separate document on Dysbarism - Decompression Illness (in Environmental folder). Terminology The term dysbarism encompasses: ● Decompression illness And ● Barotrauma And ● Nitrogen narcosis Decompression illness (DCI) includes: 1. Decompression sickness (DCS) (or in lay terms, the “bends”): ● Type I DCS: ♥ Involves the joints or skin only ● Type II DCS: ♥ Involves all other pain, neurological injury, vestibular and pulmonary symptoms. 2. Arterial gas embolism (AGE): ● Due to pulmonary barotrauma releasing air into the circulation. Epidemiology Diving is generally a safe undertaking. Serious decompression incidents occur approximately only in 1 in 10,000 dives. However, because of high participation rates, there are about 200 - 300 cases of significant decompression illness requiring treatment in Australia each year. It is estimated that 10 times this number of divers experience less severe illness after diving. Physics Boyle’s Law: The air pressure at sea level is 1 atmosphere absolute (ATA). Alternative units used for 1 ATA include: ● 101.3 kPa (SI units) ● 1.013 Bar ● 10 meters of sea water (MSW) ● 760 mm of mercury (mm Hg) ● 14.7 pounds per square inch (PSI) For every 10 meters a diver descends in seawater, the pressure increases by 1 ATA. -
THE PHYSICIAN's GUIDE to DIVING MEDICINE the PHYSICIAN's GUIDE to DIVING MEDICINE Tt",,.,,,,., , ••••••••••• ,
THE PHYSICIAN'S GUIDE TO DIVING MEDICINE THE PHYSICIAN'S GUIDE TO DIVING MEDICINE tt",,.,,,,., , ••••••••••• , ......... ,.", •••••••••••••••••••••••• ,. ••. ' ••••••••••• " .............. .. Edited by Charles W. Shilling Catherine B. Carlston and Rosemary A. Mathias Undersea Medical Society Bethesda, Maryland PLENUM PRESS • NEW YORK AND LONDON Library of Congress Cataloging in Publication Data Main entry under title: The Physician's guide to diving medicine. Includes bibliographies and index. 1. Submarine medicine. 2. Diving, Submarine-Physiological aspects. I. Shilling, Charles W. (Charles Wesley) II. Carlston, Catherine B. III. Mathias, Rosemary A. IV. Undersea Medical Society. [DNLM: 1. Diving. 2. Submarine Medicine. WD 650 P577] RC1005.P49 1984 616.9'8022 84-14817 ISBN-13: 978-1-4612-9663-8 e-ISBN-13: 978-1-4613-2671-7 DOl: 10.1007/978-1-4613-2671-7 This limited facsimile edition has been issued for the purpose of keeping this title available to the scientific community. 10987654 ©1984 Plenum Press, New York A Division of Plenum Publishing Corporation 233 Spring Street, New York, N.Y. 10013 All rights reserved No part of this book may be reproduced, stored in a retrieval system, or transmitted in any form or by any means, electronic, mechanical, photocopying, microfilming, recording, or otherwise, without written permission from the Publisher Contributors The contributors who authored this book are listed alphabetically below. Their names also appear in the text following contributed chapters or sections. N. R. Anthonisen. M.D .. Ph.D. Professor of Medicine University of Manitoba Winnipeg. Manitoba. Canada Arthur J. Bachrach. Ph.D. Director. Environmental Stress Program Naval Medical Research Institute Bethesda. Maryland C. Gresham Bayne. -
Diving and Hyperbaric Medicine
Diving and Hyperbaric Medicine 7KH-RXUQDORIWKH6RXWK3DFL¿F8QGHUZDWHU0HGLFLQH6RFLHW\ ,QFRUSRUDWHGLQ9LFWRULD $% ISSN 1833 - 3516 Volume 37 No. 4 ABN 29 299 823 713 December 2007 Diving expeditions: from Antarctica to the Tropics Diving deaths in New Zealand Epilepsy and diving – time for a change? Mechanical ventilation of patients at pressure Print Post Approved PP 331758/0015 9^k^c\VcY=neZgWVg^XBZY^X^cZKdajbZ(,Cd#)9ZXZbWZg'%%, PURPOSES OF THE SOCIETY IdegdbdiZVcY[VX^a^iViZi]ZhijYnd[VaaVheZXihd[jcYZglViZgVcY]neZgWVg^XbZY^X^cZ Idegdk^YZ^c[dgbVi^dcdcjcYZglViZgVcY]neZgWVg^XbZY^X^cZ IdejWa^h]V_djgcVa IdXdckZcZbZbWZghd[i]ZHdX^ZinVccjVaanViVhX^Zci^ÄXXdc[ZgZcXZ OFFICE HOLDERS EgZh^YZci 9g8]g^h6Xdii (%EVg`6kZcjZ!GdhhancEVg` :çbV^a1XVXdii5deijhcZi#Xdb#Vj3 Hdji]6jhigVa^V*%,' EVhiçEgZh^YZci 9gGdWncLVa`Zg &'7VggVaa^ZgHigZZi!<g^[Äi] :çbV^a1GdWnc#LVa`Zg5YZ[ZcXZ#\dk#Vj3 68I'+%( HZXgZiVgn 9gHVgV]H]Vg`Zn E#D#7DM&%*!CVggVWZZc :çbV^a1hejbhhZXgZiVgn5\bV^a#Xdb3 CZlHdji]LVaZh'&%& IgZVhjgZg 9g<jnL^aa^Vbh E#D#7dm&.%!GZY=^aaHdji] :çbV^a1hejbh5[VhibV^a#cZi3 K^Xidg^V(.(, :Y^idg 6hhdX#Egd[#B^`Z9Vk^h 8$d=neZgWVg^XBZY^X^cZJc^i :çbV^a1hejbh_5XY]W#\dki#co3 8]g^hiX]jgX]=dhe^iVa!Eg^kViZ7V\),&%!8]g^hiX]jgX]!CO :YjXVi^dcD[ÄXZg 9g;^dcVH]Vge ').XC^X]dahdcGdVY!H]ZcidcEVg` :çbV^a1h]Vge^Z[5YdXidgh#dg\#j`3 LZhiZgc6jhigVa^V+%%- EjWa^XD[ÄXZg 9gKVcZhhV=VaaZg E#D#7dm-%'(!8Vggjb9dlch :çbV^a1kVcZhhV#]VaaZg5XYbX#Xdb#Vj3 K^Xidg^V('%& 8]V^gbVc6CO=B< 9g9Vk^YHbVgi 9ZeVgibZcid[9^k^c\VcY=neZgWVg^XBZY^X^cZ :çbV^a1YVk^Y#hbVgi5Y]]h#iVh#\dk#Vj3 GdnVa=dWVgi=dhe^iVa!=dWVgi!IVhbVc^V,%%% LZWbVhiZg -
Training Objectives for a Diving Medical Physician
The Diving Medical Advisory Committee Training Objectives for a Diving Medicine Physician This guidance includes all the training objectives agreed by the Diving Medical Advisory Committee, the European Diving Technology Committee and the European Committee for Hyperbaric Medicine in 2011. Rev 1 - 2013 INTRODUCTION The purpose of this document is to define more closely the training objectives in diving physiology and medicine that need to be met by doctors already fully accredited or board-certified in a clinical speciality to national standards. It is based on topic headings that were originally prepared for a working group of European Diving Technology Committee (EDTC) and the European Committee of Hyperbaric Medicine (ECHM) as a guide for diving medicine some 20 years ago by J.Desola (Spain), T.Nome (Norway) & D.H.Elliott (U.K.). The training now required for medical examiners of working divers and for specialist diving medicine physicians was based on a EDTC/ECHM standard 1999 and subsequently has been enhanced by the Diving Medical Advisory Committee (DMAC), revised and agreed in principle by DMAC, EDTC and ECHM in 2010 and then ratified by EDTC and ECHM in 2011. The requirements now relate to an assessment of competence, the need for some training in occupational medicine, the need for maintenance of those skills by individual ‘refresher training’. Formal recognition of all this includes the need to involve a national authority for medical education. These objectives have been applied internationally to doctors who provide medical support to working divers. (Most recreational instructors and dive guides are, by their employment, working divers and so the guidance includes the relevant aspects of recreational diving. -
A Rare Complication of Carbon Monoxide Poisoning
International Clinical Pathology Journal Case Report Open Access Compartment syndrome of the lower limb: a rare complication of carbon monoxide poisoning Abstract Volume 6 Issue 5 - 2018 Carbon monoxide poisoning (CO) nicknamed “Silent killer” remains the leading cause Ghassen Drissi, Mohamed Khaled, Houssem of poisoning deaths in most countries. In Tunisia, an estimated number of 2000 to 4000 cases of poisoning was noted per a year with an estimated percentage of death up Kraiem, Khaled Zitouna, Mohamed lassed to 90% at the site of intoxication. This intoxication is very exceptionally complicated kanoun by a compartment syndrome that can aggravate the functional and vital prognosis Department of Orthopaedics and Traumatology, La Rabta Hospital of Tunis, Tunisia when it is dominated by other symptoms, particularly neurological ones. We report the case of a patient who presented a compartment syndrome during CO intoxication Correspondence: Ghassen Drissi, Department of that evolved favorably. Orthopaedics and Traumatology, La Rabta Hospital of Tunis, Tunisia, Email Received: October 08, 2018 | Published: November 15, 2018 Introduction an inhalation syndrome causing acute respiratory failure associated with a compartmental syndrome of the left lower limb syndrome Carbon monoxide is a colorless, odourless, non-irritating gas. It is causing acute kidney failure (tubular necrosis) with rhabdomyolysis particularly toxic to mammals but undetectable by them giving it an requiring intubation associated to mechanical ventilation with insidious “Silent killer” character. In humans, it is the cause of much emergency hemodialysis sessions. With regard to the compartmental domestic intoxication, often fatal. Its causes are most often accidental, syndrome, two fasciotomies in emergency were made in our service. -
THE EFFECT of CONTINUOUS NEGATIVE PRESSURE BREATH- ING on WATER and ELECTROLYTE EXCRETION by the HUMAN KIDNEY by HERBERT 0
THE EFFECT OF CONTINUOUS NEGATIVE PRESSURE BREATH- ING ON WATER AND ELECTROLYTE EXCRETION BY THE HUMAN KIDNEY By HERBERT 0. SIEKER,1 OTTO H. GAUER,2 AND JAMES P. HENRY (From the Aero-Medical Laboratory, Wright-Patterson Air Force Base, Ohio) (Submitted for publication September 4, 1953; accepted December 30, 1953) The effects of decreased intrathoracic pressure ment. Negative pressure breathing during both inspiration on arterial blood pressure (1), venous pressure and expiration was applied through a standard U. S. Air (2, 3), cardiac output (4), and pres- Force pressure breathing oxygen mask attached by a pulmonary short tubing to a 40 liter cylinder. This cylinder was sure and volume (5) have been investigated in the ventilated by a suction pump with fresh air at the rate of past. The present study was prompted by the as- 100 to 160 liters per minute and rebreathing was pre- sociation of marked diuresis with continuous nega- vented by means of a two-way valve in the face mask. tive pressure breathing in anesthetized animals (6) The desired negative pressure, which in these experiments and the observation that in unanesthetized man was a mean pressure of 15 to 18 centimeters of water, was obtained by varying the air inlet to the container. continuous positive pressure breathing leads to an Control studies duplicated the procedure exactly except oliguria (7). The purpose of this investigation that the negative pressure breathing was omitted. Re- was to demonstrate that human subjects like an- peat studies were done in all but two subjects. esthetized animals have an increased urine flow in The urine volume for each 15-minute interval was noted response to continuous negative pressure breathing.