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Juvenile Spondyloarthropathies: Inflammation in Disguise
PP.qxd:06/15-2 Ped Perspectives 7/25/08 10:49 AM Page 2 APEDIATRIC Volume 17, Number 2 2008 Juvenile Spondyloarthropathieserspective Inflammation in DisguiseP by Evren Akin, M.D. The spondyloarthropathies are a group of inflammatory conditions that involve the spine (sacroiliitis and spondylitis), joints (asymmetric peripheral Case Study arthropathy) and tendons (enthesopathy). The clinical subsets of spondyloarthropathies constitute a wide spectrum, including: • Ankylosing spondylitis What does spondyloarthropathy • Psoriatic arthritis look like in a child? • Reactive arthritis • Inflammatory bowel disease associated with arthritis A 12-year-old boy is actively involved in sports. • Undifferentiated sacroiliitis When his right toe starts to hurt, overuse injury is Depending on the subtype, extra-articular manifestations might involve the eyes, thought to be the cause. The right toe eventually skin, lungs, gastrointestinal tract and heart. The most commonly accepted swells up, and he is referred to a rheumatologist to classification criteria for spondyloarthropathies are from the European evaluate for possible gout. Over the next few Spondyloarthropathy Study Group (ESSG). See Table 1. weeks, his right knee begins hurting as well. At the rheumatologist’s office, arthritis of the right second The juvenile spondyloarthropathies — which are the focus of this article — toe and the right knee is noted. Family history is might be defined as any spondyloarthropathy subtype that is diagnosed before remarkable for back stiffness in the father, which is age 17. It should be noted, however, that adult and juvenile spondyloar- reported as “due to sports participation.” thropathies exist on a continuum. In other words, many children diagnosed with a type of juvenile spondyloarthropathy will eventually fulfill criteria for Antinuclear antibody (ANA) and rheumatoid factor adult spondyloarthropathy. -
The Painful Heel Comparative Study in Rheumatoid Arthritis, Ankylosing Spondylitis, Reiter's Syndrome, and Generalized Osteoarthrosis
Ann Rheum Dis: first published as 10.1136/ard.36.4.343 on 1 August 1977. Downloaded from Annals of the Rheumatic Diseases, 1977, 36, 343-348 The painful heel Comparative study in rheumatoid arthritis, ankylosing spondylitis, Reiter's syndrome, and generalized osteoarthrosis J. C. GERSTER, T. L. VISCHER, A. BENNANI, AND G. H. FALLET From the Department of Medicine, Division of Rheumatology, University Hospital, Geneva, Switzerland SUMMARY This study presents the frequency of severe and mild talalgias in unselected, consecutive patients with rheumatoid arthritis, ankylosing spondylitis, Reiter's syndrome, and generalized osteoarthosis. Achilles tendinitis and plantar fasciitis caused a severe talalgia and they were observed mainly in males with Reiter's syndrome or ankylosing spondylitis. On the other hand, sub-Achilles bursitis more frequently affected women with rheumatoid arthritis and rarely gave rise to severe talalgias. The simple calcaneal spur was associated with generalized osteoarthrosis and its frequency increased with age. This condition was not related to talalgias. Finally, clinical and radiological involvement of the subtalar and midtarsal joints were observed mainly in rheumatoid arthritis and occasionally caused apes valgoplanus. copyright. A 'painful heel' syndrome occurs at times in patients psoriasis, urethritis, conjunctivitis, or enterocolitis. with inflammatory rheumatic disease or osteo- The antigen HLA B27 was present in 29 patients arthrosis, causing significant clinical problems. Very (80%O). few studies have investigated the frequency and characteristics of this syndrome. Therefore we have RS 16 PATIENTS studied unselected groups of patients with rheuma- All of our patients had the complete triad (non- toid arthritis (RA), ankylosing spondylitis (AS), gonococcal urethritis, arthritis, and conjunctivitis). -
The Pathomechanics of Plantar Fasciitis
Sports Med 2006; 36 (7): 585-611 REVIEW ARTICLE 0112-1642/06/0007-0585/$39.95/0 2006 Adis Data Information BV. All rights reserved. The Pathomechanics of Plantar Fasciitis Scott C. Wearing,1 James E. Smeathers,1 Stephen R. Urry,1 Ewald M. Hennig2 and Andrew P. Hills1 1 Institute of Health and Biomedical Innovation, Queensland University of Technology, Kelvin Grove, Queensland, Australia 2 Biomechanik Labor, University Duisburg-Essen, Essen, Germany Contents Abstract ....................................................................................585 1. Anatomy of the Plantar Fascia ............................................................586 1.1 Gross Anatomy of the Plantar Fascia...................................................586 1.2 Histological Anatomy of the Plantar Fascia .............................................589 1.2.1 Neuroanatomy ................................................................590 1.2.2 Microcirculation ...............................................................590 1.2.3 The Enthesis ...................................................................590 2. Mechanical Properties of the Plantar Fascia ................................................592 2.1 Structural Properties ..................................................................593 2.2 Material Properties ...................................................................593 3. Mechanical Function of the Plantar Fascia .................................................594 3.1 Quasistatic Models of Plantar Fascial Function ..........................................594 -
Upper Extremity
Upper Extremity Shoulder Elbow Wrist/Hand Diagnosis Left Right Diagnosis Left Right Diagnosis Left Right Adhesive capsulitis M75.02 M75.01 Anterior dislocation of radial head S53.015 [7] S53.014 [7] Boutonniere deformity of fingers M20.022 M20.021 Anterior dislocation of humerus S43.015 [7] S43.014 [7] Anterior dislocation of ulnohumeral joint S53.115 [7] S53.114 [7] Carpal Tunnel Syndrome, upper limb G56.02 G56.01 Anterior dislocation of SC joint S43.215 [7] S43.214 [7] Anterior subluxation of radial head S53.012 [7] S53.011 [7] DeQuervain tenosynovitis M65.42 M65.41 Anterior subluxation of humerus S43.012 [7] S43.011 [7] Anterior subluxation of ulnohumeral joint S53.112 [7] S53.111 [7] Dislocation of MCP joint IF S63.261 [7] S63.260 [7] Anterior subluxation of SC joint S43.212 [7] S43.211 [7] Contracture of muscle in forearm M62.432 M62.431 Dislocation of MCP joint of LF S63.267 [7] S63.266 [7] Bicipital tendinitis M75.22 M75.21 Contusion of elbow S50.02X [7] S50.01X [7] Dislocation of MCP joint of MF S63.263 [7] S63.262 [7] Bursitis M75.52 M75.51 Elbow, (recurrent) dislocation M24.422 M24.421 Dislocation of MCP joint of RF S63.265 [7] S63.264 [7] Calcific Tendinitis M75.32 M75.31 Lateral epicondylitis M77.12 M77.11 Dupuytrens M72.0 Contracture of muscle in shoulder M62.412 M62.411 Lesion of ulnar nerve, upper limb G56.22 G56.21 Mallet finger M20.012 M20.011 Contracture of muscle in upper arm M62.422 M62.421 Long head of bicep tendon strain S46.112 [7] S46.111 [7] Osteochondritis dissecans of wrist M93.232 M93.231 Primary, unilateral -
Frequency and Criticality of Diagnoses in Family Medicine Practices: from the National Ambulatory Medical Care Survey (NAMCS)
J Am Board Fam Med: first published as 10.3122/jabfm.2018.01.170209 on 12 January 2018. Downloaded from ORIGINAL RESEARCH Frequency and Criticality of Diagnoses in Family Medicine Practices: From the National Ambulatory Medical Care Survey (NAMCS) Michael R. Peabody, PhD, Thomas R. O’Neill, PhD, Keith L. Stelter, MD, MMM, and James C. Puffer, MD Background: Family medicine is a specialty of breadth, providing comprehensive health care for the individual and the family that integrates the broad scope of clinical, social, and behavioral sciences. As such, the scope of practice (SOP) for family medicine is extensive; however, over time many family phy- sicians narrow their SOP. We sought to provide a nationally representative description of the most com- mon and the most critical diagnoses that family physicians see in their practice. Methods: Data were extracted from the 2012 National Ambulatory Medical Care Survey (NAMCS) to select all ICD-9 codes reported by family physicians. A panel of family physicians then reviewed 1893 ICD-9 codes to place each code into an American Board of Family Medicine Family Medicine Certifica- tion Examination test plan specifications (TPS) category and provide a rating for an Index of Harm (IoH). Results: An analysis of all 1893 ICD-9 codes seen by family physicians in the 2012 NAMCS found that 198 ICD-9 codes could not be assigned a TPS category, leaving 1695 ICD-9 codes in the dataset. Top 10 lists of ICD-9 codes by TPS category were created for both frequency and IoH. Conclusions: This study provides a nationally representative description of the most common diag- copyright. -
Conditions Related to Inflammatory Arthritis
Conditions Related to Inflammatory Arthritis There are many conditions related to inflammatory arthritis. Some exhibit symptoms similar to those of inflammatory arthritis, some are autoimmune disorders that result from inflammatory arthritis, and some occur in conjunction with inflammatory arthritis. Related conditions are listed for information purposes only. • Adhesive capsulitis – also known as “frozen shoulder,” the connective tissue surrounding the joint becomes stiff and inflamed causing extreme pain and greatly restricting movement. • Adult onset Still’s disease – a form of arthritis characterized by high spiking fevers and a salmon- colored rash. Still’s disease is more common in children. • Caplan’s syndrome – an inflammation and scarring of the lungs in people with rheumatoid arthritis who have exposure to coal dust, as in a mine. • Celiac disease – an autoimmune disorder of the small intestine that causes malabsorption of nutrients and can eventually cause osteopenia or osteoporosis. • Dermatomyositis – a connective tissue disease characterized by inflammation of the muscles and the skin. The condition is believed to be caused either by viral infection or an autoimmune reaction. • Diabetic finger sclerosis – a complication of diabetes, causing a hardening of the skin and connective tissue in the fingers, thus causing stiffness. • Duchenne muscular dystrophy – one of the most prevalent types of muscular dystrophy, characterized by rapid muscle degeneration. • Dupuytren’s contracture – an abnormal thickening of tissues in the palm and fingers that can cause the fingers to curl. • Eosinophilic fasciitis (Shulman’s syndrome) – a condition in which the muscle tissue underneath the skin becomes swollen and thick. People with eosinophilic fasciitis have a buildup of eosinophils—a type of white blood cell—in the affected tissue. -
A+ Mobile Ultrasound Services LLC Seattle, WA 206-799-3301 [email protected] Musculoskeletal (MSK)
A+ Mobile Ultrasound Services LLC Seattle, WA 206-799-3301 [email protected] Musculoskeletal (MSK) www.APlusUltrasound.com Shoulder Hip • Rotator Cuff Tear/Tedonosis • Bowel Hernia • Biceps Tendon Tear • Sports Hernia • Tendinitis/Tenosynovitis/Subluxation • Snapping Hip Syndrome • Shoulder Impingement • Effusion • AC joint separation • Gluteal or thigh muscle injury • Fluid Collections – Bursitis / Effusion Knee Elbow • MCL / LCL Injury • Tennis Elbow – Lateral Epicondylitis • Iliotibial Band Syndrome • Golfer’s Elbow – Medial Epicondylitis • Jumper’s Knee – Patellar Tendon Injury • Biceps Tendon Insertion • Lateral, Medial or Posterior Meniscus Tear • Ulnar Nerve Entrapment • Runner’s knee • (Cubital Tunnel Syndrome) • Fluid collection – Bursitis / Effusion / Baker’s • Ulnar Collateral Ligament (UCL) Injury Cyst • Triceps Tendon Tear/Tendonosis Ankle/Foot • Fluid Collection – Bursitis / Effusion • Achilles’ Tendon Tear/Tendinitis Wrist • Tibial Tendon Tear/Tendinitis/Tenosynovitis • Medial Nerve Entrapment • Peroneal • Carpal Tunnel Syndrome Tear/Tendinitis/Tenosynovitis/Subluxation • Extensor Tendonosis/Tenosynovitis • Ankle Sprain – Ligament Injury (ATFL) • De Quervain’s Syndrome • High Ankle Sprain – Tibiofibular Ligament Tear • Flexor Tendonosis/Tenosynovitis • Tibial Nerve Entrapment Hand/Finger • Fluid Collection – Bursitis / Effusion • Trigger Finger • Plantar Fasciitis • Avulsion • Morton’s Neuroma • Fracture • Turf Toe MSK Jaw and Neck Ultrasound MSK Extremity – Non Joint • Neck Pain • Muscle Sprain/Tear • Whiplash -
Plantar Fasciitis
Recent Advances in Orthotic Therapy for Plantar Fasciitis An Evidence Based Approach Lawrence Z. Huppin, D.P.M. Assistant Clinical Professor, Western University of Health Sciences, College of Podiatric Medicine Disclosures I disclose the following financial relationships with commercial entities that produce health care related products and services relevant to the content of this lecture: ◦ Employee (Medical Director) of ProLab Orthotics, manufacturer of foot orthoses Our Goals . Review Three Studies that Help Optimize Clinical Outcome of Custom Foot Orthoses Used to Treat Plantar Fasciitis • Cause of Plantar Fascial Tension • Forefoot Wedging Effect on Plantar Fascial Tension • Arch Contour Effect on Plantar Fascial Tension . Develop a “pathology specific” orthosis prescription to most effectively treat plantar fasciitis . Offloading Excessive Fascia Tension is Goal of Orthotic Therapy . What Causes Tension? Tissue Stress Theory McPoil, Hunt. Evaluation and Management of Foot and Ankle Disorders: Present Problems and Future Directions. Journal of Orthopaedic & Sports Physical Therapy, 1995 Effect of STJ Pronation on PF Strain . Cannot by itself cause strain of plantar fascia . Can only increase plantar fascia strain via the MTJ • (inversion of the forefoot on the rearfoot) Scherer. “Heel Pain Syndrome: Pathomechanics and Non-surgical Treatment. JAPMA 1991 Effect of Forefoot Inversion on Plantar Fascial Strain 73 patients with 118 painful heels 91% had foot deformity compensated by supination of long axis of MTJ Out of 118 painful heels 63 had forefoot valgus 33 had everted rearfoot 20 had plantarflexed first ray Scherer 1991 What foot types supinate the MTJ? . 47% forefoot valgus . 24% everted heel . 20% plantarflexed first ray Scherer 1991 What foot types supinate the MTJ? . -
Chronic Foot Pain
Revised 2020 American College of Radiology ACR Appropriateness Criteria® Chronic Foot Pain Variant 1: Chronic foot pain. Unknown etiology. Initial imaging. Procedure Appropriateness Category Relative Radiation Level Radiography foot Usually Appropriate ☢ US foot Usually Not Appropriate O MRI foot without and with IV contrast Usually Not Appropriate O MRI foot without IV contrast Usually Not Appropriate O CT foot with IV contrast Usually Not Appropriate ☢ CT foot without and with IV contrast Usually Not Appropriate ☢ CT foot without IV contrast Usually Not Appropriate ☢ Bone scan foot Usually Not Appropriate ☢☢☢ Variant 2: Persistent posttraumatic foot pain. Radiographs negative or equivocal. Clinical concern includes complex regional pain syndrome type I. Next imaging study. Procedure Appropriateness Category Relative Radiation Level MRI foot without IV contrast Usually Appropriate O 3-phase bone scan foot Usually Appropriate ☢☢☢ MRI foot without and with IV contrast May Be Appropriate O US foot Usually Not Appropriate O CT foot with IV contrast Usually Not Appropriate ☢ CT foot without and with IV contrast Usually Not Appropriate ☢ CT foot without IV contrast Usually Not Appropriate ☢ Variant 3: Chronic metatarsalgia including plantar great toe pain. Radiographs negative or equivocal. Clinical concern includes sesamoiditis, Morton’s neuroma, intermetatarsal bursitis, chronic plantar plate injury, or Freiberg’s infraction. Next imaging study. Procedure Appropriateness Category Relative Radiation Level MRI foot without IV contrast Usually Appropriate O US foot May Be Appropriate O MRI foot without and with IV contrast May Be Appropriate O CT foot without IV contrast May Be Appropriate ☢ Bone scan foot May Be Appropriate ☢☢☢ CT foot with IV contrast Usually Not Appropriate ☢ CT foot without and with IV contrast Usually Not Appropriate ☢ ACR Appropriateness Criteria® 1 Chronic Foot Pain Variant 4: Chronic plantar heel pain. -
Eosinophilic Fasciitis (Shulman Syndrome)
CONTINUING MEDICAL EDUCATION Eosinophilic Fasciitis (Shulman Syndrome) Sueli Carneiro, MD, PhD; Arles Brotas, MD; Fabrício Lamy, MD; Flávia Lisboa, MD; Eduardo Lago, MD; David Azulay, MD; Tulia Cuzzi, MD, PhD; Marcia Ramos-e-Silva, MD, PhD GOAL To understand eosinophilic fasciitis to better manage patients with the condition OBJECTIVES Upon completion of this activity, dermatologists and general practitioners should be able to: 1. Recognize the clinical presentation of eosinophilic fasciitis. 2. Discuss the histologic findings in patients with eosinophilic fasciitis. 3. Differentiate eosinophilic fasciitis from similarly presenting conditions. CME Test on page 215. This article has been peer reviewed and is accredited by the ACCME to provide continuing approved by Michael Fisher, MD, Professor of medical education for physicians. Medicine, Albert Einstein College of Medicine. Albert Einstein College of Medicine designates Review date: March 2005. this educational activity for a maximum of 1 This activity has been planned and implemented category 1 credit toward the AMA Physician’s in accordance with the Essential Areas and Policies Recognition Award. Each physician should of the Accreditation Council for Continuing Medical claim only that credit that he/she actually spent Education through the joint sponsorship of Albert in the activity. Einstein College of Medicine and Quadrant This activity has been planned and produced in HealthCom, Inc. Albert Einstein College of Medicine accordance with ACCME Essentials. Drs. Carneiro, Brotas, Lamy, Lisboa, Lago, Azulay, Cuzzi, and Ramos-e-Silva report no conflict of interest. The authors report no discussion of off-label use. Dr. Fisher reports no conflict of interest. We present a case of eosinophilic fasciitis, or Shulman syndrome apart from all other sclero- Shulman syndrome, in a 35-year-old man and dermiform states. -
Haglund's Syndrome, Retrocalaneal Exostosis
Open Access Review Article DOI: 10.7759/cureus.820 Haglund’s Syndrome: A Commonly Seen Mysterious Condition Raju Vaishya 1 , Amit Kumar Agarwal 1 , Ahmad Tariq Azizi 2 , Vipul Vijay 1 1. Orthopaedics, Indraprastha Apollo Hospitals 2. Orthopaedics, Herat Regional Hospital, Herat, Afghanistan Corresponding author: Amit Kumar Agarwal, [email protected] Abstract Haglund’s deformity was first described by Patrick Haglund in 1927. It is also known as retrocalcaneal exostosis, Mulholland deformity, and ‘pump bump.' It is a very common clinical condition, but still poorly understood. Haglund’s deformity is an abnormality of the bone and soft tissues in the foot. An enlargement of the bony section of the heel (where the Achilles tendon is inserted) triggers this condition. The soft tissue near the back of the heel can become irritated when the large, bony lump rubs against rigid shoes. The aetiology is not well known, but some probable causes like a tight Achilles tendon, a high arch of the foot, and heredity have been suggested as causes. Middle age is the most common age of affection, females are more affected than males, and the occurence is often bilateral. A clinical feature of this condition is pain in the back of the heel, which is more after rest. Clinical evaluation and lateral radiographs of the ankle are mostly enough to make a diagnosis of Haglund’s syndrome. Haglund’s syndrome is often treated conservatively by altering the heel height in shoe wear, orthosis, physiotherapy, and anti-inflammatory drugs. Surgical excision of the bony exostoses of the calcaneum is only required in resistant cases. -
20-0420 ) Issued: April 21, 2021 U.S
United States Department of Labor Employees’ Compensation Appeals Board __________________________________________ ) D.H., Appellant ) ) and ) Docket No. 20-0420 ) Issued: April 21, 2021 U.S. POSTAL SERVICE, BEVERLY POST ) OFFICE, Beverly, MA, Employer ) __________________________________________ ) Appearances: Case Submitted on the Record Katherine A. Harrell, for the appellant1 Office of Solicitor, for the Director DECISION AND ORDER Before: ALEC J. KOROMILAS, Chief Judge PATRICIA H. FITZGERALD, Alternate Judge VALERIE D. EVANS-HARRELL, Alternate Judge JURISDICTION On December 13, 2019 appellant, through his representative, filed a timely appeal from an October 16, 2019 nonmerit decision of the Office of Workers’ Compensation Programs (OWCP). As more than 180 days has elapsed from the last merit decision dated December 18, 2018 to the filing of this appeal, pursuant to the Federal Employees’ Compensation Act2 (FECA) and 20 C.F.R. §§ 501.2(c) and 501.3, the Board lacks jurisdiction over the merits of this case. 1 In all cases in which a representative has been authorized in a matter before the Board, no claim for a fee for legal or other service performed on appeal before the Board is valid unless approved by the Board. 20 C.F.R. § 501.9(e). No contract for a stipulated fee or on a contingent fee basis will be approved by the Board. Id. An attorney or representative’s collection of a fee without the Board’s approval may constitute a misdemeanor, subject to fine or imprisonment for up to one year or both. Id.; see also 18 U.S.C. § 292. Demands for payment of fees to a representative, prior to approval by the Board, may be reported to appropriate authorities for investigation.