James-Michael-CV.Pdf

Total Page:16

File Type:pdf, Size:1020Kb

James-Michael-CV.Pdf DUKE UNIVERSITY MEDICAL CENTER CURRICULUM VITAE Permanent Record and the Appointments and Promotions Committee Date Prepared: 04/28/2017 Name: Michael Lucas James, MD Primary academic appointment: Associate Professor Primary academic department: Anesthesiology Secondary appointment: Neurology Present academic rank and title: Associate Professor without Tenure, Track 1 Date and rank of first Duke Faculty appointment: 07/01/2006, Assistant Professor Medical Licensure: North Carolina License #200001390 Date of License: 09/26/2000 Specialty certifications and dates American Board of Psychiatry & Neurology 05/12/2004 Recertification 2014 Subspecialty Vascular Neurology 04/16/2009 American Board of Anesthesiology 05/25/2007 United Council of Neurological Subspecialties Neurocritical Care 12/31/2008 Date of birth: 09/26/1973 Place: Baton Rouge, LA USA Citizen of: United States of America Education: Institution Date (Year) Degree High School LSU Laboratory School 1991 College Vanderbilt University 1995 BS Professional LSU School of Medicine in New Orleans 1999 MD Scholarly societies: Psi Chi Professional training and academic career: Training: Position/Title Dates Duke University Medical Center Medical Intern 07/1999 – 06/2000 Neurology Resident 07/2000 – 06/2002 Neurology Chief Resident 07/2002 – 06/2003 Anesthesiology Resident 07/2003 – 06/2006 Neurocritical Care Fellow 07/2005 – 06/2006 Professional appointments: Assistant Professor Departments of Anesthesiology & Neurology Duke University 07/2006 – 10/2014 Associate Director Multidisciplinary Neuroprotection Laboratories, Department of Anesthesiology, Duke University 07/2010 – 06/2015 Associate Director Brain Injury Translational Research Center, Department of Neurology, Duke University 07/2009 – present Associate Professor Departments of Anesthesiology & Neurology Duke University 10/2014 – present Program Director Neuroanesthesiology Fellowship, Department of Anesthesiology Duke University 01/2015 – present Associate Director Clinical Endeavors in Anesthesia Research, Department of Anesthesiology Duke University 07/2015 – present Liaison Clinical Research, Duke-NUS Affairs Duke University 07/2016 – present Associate Professor Department of Clinical Science Duke-NUS Graduate Medical School 10/2016 – present Publications: Refereed journals: 1. James ML, Panni MK. Extremely prolonged unilateral block (20 hours) with spinal ropivacaine used for cervical cerclage placement. Anesth Analg. 2005 Mar;100(3):897-8. PMID: 15728090 2. James ML, Husain AM. Brainstem auditory evoked potential monitoring: when is change in wave V significant? Neurology. 2005 Nov 22;65(10):1551-5. PMID: 16301480 3. Bennett SS, Graffagnino C, Borel CO, James ML. Use of high frequency oscillatory ventilation (HFOV) in neurocritical care patients. Neurocrit Care. 2007;7(3):221-6. PMID: 17805492 4. James ML, Warner DS, Laskowitz DT. Preclinical models of intracerebral hemorrhage: a translational perspective. Neurocrit Care. 2008;9(1):139-52. PMID: 18058257 5. James ML, Sullivan PM, Lascola CD, Vitek MP, Laskowitz DT. Pharmacogenomic effects of apolipoprotein e on intracerebral hemorrhage. Stroke. 2009 Feb;40(2):632-9. PMID: 19109539 6. James ML, Blessing R, Bennett E, Laskowitz DT. Apolipoprotein E modifies neurological outcome by affecting cerebral edema but not hematoma size after intracerebral hemorrhage in humans. J Stroke Cerebrovasc Dis. 2009 Mar-Apr;18(2):144-9. PMID: 19251191 7. James ML, Blessing R, Phillips-Bute BG, Bennett E, Laskowitz DT. S100B and brain natriuretic peptide predict functional neurological outcome after intracerebral haemorrhage. Biomarkers. 2009 Sep;14(6):388-94. PMID: 19505208 8. James ML, Wang H, Venkatraman T, Song P, Lascola CD, Laskowitz DT. Brain natriuretic peptide improves long-term functional recovery after acute CNS injury in mice. J Neurotrauma. 2010 Jan;27(1):217-28. PMID: 19803787 9. Kim GS, Amato A, James ML, Britz GW, Zomorodi A, Graffagnino C, Zomorodi M, Olson DM. Continuous and intermittent CSF diversion after subarachnoid hemorrhage: a pilot study. Neurocrit Care. 2011 Feb;14(1):68-72. PMID: 20596794 10. James ML, Keifer JC. Posterior optic nerve ischemic neuropathy in the setting of phenoxybenzamine therapy after uneventful spinal fusion. J Neurosurg Anesthesiol. 2011 Apr;23(2):169-70. PMID: 21270647 11. James ML, Olson DM. Brainstem dysfunction as a predictor of death in the nonneurologically injured. Crit Care Med. 2011 Aug;39(8):2012-3. PMID: 21768813 12. Amato A, Britz GW, James ML, Graffagnino C, Zomorodi AR, Zomorodi ME, Olson DM. An observational pilot study of CSF diversion in subarachnoid haemorrhage. Nurs Crit Care. 2011 Sep- Oct;16(5):252-60. PMID: 21824230 13. Laskowitz DT, Lei B, Dawson HN, Wang H, Bellows ST, Christensen DJ, Vitek MP, James ML. The apoE-mimetic Peptide, COG1410, Improves Functional Recovery in a Murine Model of Intracerebral Hemorrhage. Neurocrit Care. 2011 Oct 12. PMID: 21989844 14. Indraswari F, Wang H, Lei B, James ML, Kernagis D, Warner DS, Dawson HN, Laskowitz DT. Statins improve outcome in murine models of intracranial hemorrhage and traumatic brain injury: A translational approach. J Neurotrauma. 2012 Jan 10. PMID: 22233347 15. James ML, Wang H, Cantillana V, Lei B, Kernagis DN, Dawson HN, Klaman LD, Laskowitz DT. TT-301 inhibits microglial activation and improves outcome after central nervous system injury in adult mice. Anesthesiology. 2012 Jun;116(6):1299-311. PMID: 22487803 16. Macy GE, James ML. Guilty by association?*. Crit Care Med. 2012 Jul;40(7):2230-1. PMID: 22710208. 17. Sun W, Peacock A, Becker J, Phillips-bute BG, Laskowitz DT, James ML. Correlation of leukocytosis with neurological deterioration following supratentorial intracerebral hemorrhage. J Clin Neurosci. 2012 Aug;19(8):1096-100. Epub 2012 Jun 16. PMID: 22704946 18. Sheng SP, Lei B, James ML, Lascola CD, Venkatraman TN, Jung JY, Maze M, Franks NP, Pearlstein RD, Sheng H, Warner DS. Xenon Neuroprotection in Experimental Stroke: Interactions with Hypothermia and Intracerebral Hemorrhage. Anesthesiology. 2012 Nov 7. PMID: 23143806 19. James ML, Olson DM, Graffagnino C. A pilot study of cerebral and haemodynamic physiological changes during sedation with dexmedetomidine or propofol in patients with acute brain injury. Anaesth Intensive Care. 2012 Nov;40(6):949-57. Anesthesia & Intensive Care. 2012 Nov; 40(6). PMID: 23194203 20. Wang H, Anderson LG, Lascola CD, James ML, Venkatraman TN, Bennett ER, Acheson SK, Vitek MP, Laskowitz DT. ApolipoproteinE mimetic peptides improve outcome after focal ischemia. Exp Neurol. 2013 Mar;241:67-74. Epub 2012 Dec 6. PMID: 23219883 21. Olson DM, Grissom JL, Williamson RA, Bennett SN, Bellows ST, James ML. Interrater Reliability of the Bedside Shivering Assessment Scale. Am J Crit Care. 2013 Jan;22(1):70-4. PMID: 23283091 22. James ML, Grau-Sepulveda MV, Olson DM, Smith EE, Hernandez AF, Peterson ED, Schwamm LH, Bhatt DL, Fonarow GC. Insurance Status and Outcome after Intracerebral Hemorrhage: Findings from Get with The Guidelines-Stroke. J Stroke Cerebrovasc Dis. 2013 Mar 26. PMID: 23537567 23. James ML, Andersen ND, Swaminathan M, Phillips-Bute B, Hanna JM, Smigla GR, Barfield ME, Bhattacharya SD, Williams JB, Gaca JG, Husain AM, Hughes GC. Predictors of electrocerebral inactivity with deep hypothermia. J Thorac Cardiovasc Surg. 2013 Apr 11. PMID: 23582829 24. Ouyang M, McDonagh DL, Phillips-Bute B, James ML, Friedman AH, Gan TJ. Comparison of Postoperative Nausea between Benign and Malignant Brain Tumor Patients undergoing Awake Craniotomy: A Retrospective Analysis. Curr Med Res Opin. 2013 Jun 4. [Epub ahead of print] PMID: 23731201 25. Ouyang M, McDonagh DL, Phillips-Bute B, James ML, Friedman AH, Gan TJ. Does Midline Shift Predict Postoperative Nausea in Brain Tumor Patients Undergoing Awake Craniotomy? A Retrospective Analysis. Curr Med Res Opin. 2013 Jun 4. [Epub ahead of print] PMID: 23731200 26. James ML, Dority J, Gray MC, Bellows ST, McDonagh DL, Brambrink AM. Survey of Anesthesiologists Practicing in American Neurointensive Care Units as Neurointensivists. J Neurosurg Anesth. 2013 Jul 24. PMID: 23887679 27. Khan S, Adogwa O, Gan TJ, Null UT, Verla T, Gokhale S, White WD, Britz GW, Zomorodi AR, James ML, McDonagh DL. Efffect of 6% hydroxyethylStarch 130/0.4 in 0.9% sodium chloride (Voluven®) on complications after subarachnoid hemorrhage: a retrospective analysis. SpringerPlus. 2013 Jul 15;2(1):314. PMID: 23888282 28. Lei B, Dawson HN, Roulhac-Wilson B, Wang H, Laskowitz DT, James ML. Tumor necrosis factor alpha antagonism improves neurobehavioral recovery in murine intracerebral hemorrhage. J Neuroinflammation. 2013 Aug 20;10(1):103. PMID: 23962089 29. Sun W, Pan W, Kranz PG, Hailey CE, Williamson RA, Sun W, Laskowitz DT, James ML. Predictors of late neurological deterioration after spontaneous intracerebral hemorrhage. Neurocrit Care. 2013 Aug 24. PMID: 2397979 30. Woo D, Rosand J, Kidwell C, McCauley JL, Osborne J, Brown MW, West SE, Rademacher EW, Waddy S, Roberts JN, Koch S, Gonzales NR, Sung G, Kittner SJ, Birnbaum L, Frankel M, Testai FD, Hall CE, Elkind MSV, Flaherty M, Coull B, Chong JY, Warwick T, Malkoff M, James ML, Ali LK, Worrall BB, Jones F, Watson T, Leonard A, Martinez R, Sacco RI, Langefeld CD. The Ethnic Variations of Intracerebral Hemorrhage (ERICH) Study protocol. Stroke. 2013 Sep 10. PMIID: 24021679 31. Olson DM, Zomorodi MG, James ML, Cox CE, Moretti EW, Riemen KE, Graffagnino C. Exploring the impact of augmenting sedation assessment with physiologic monitors. Aust Crit Care. 2013 Oct 5.
Recommended publications
  • Electrophysiologic Monitoring in Neurointensive Care
    Ovid: Electrophysiologic monitoring in neurointensive care. Main Search Page Ask A LibrarianDisplay Knowledge BaseHelpLogoff Full Text Save Article TextEmail Article TextPrint Preview Electrophysiologic monitoring in neurointensive care Procaccio, Francesco MD*†; Polo, Alberto MD*; Lanteri, Paola MD†; Sala, ISSN: Author(s): Francesco MD† 1070- 5295 Issue: Volume 7(2), April 2001, pp 74-80 Accession: Publication Type: [Neuroscience] 00075198- Publisher: © 2001 Lippincott Williams & Wilkins, Inc. 200104000- University and City Hospital Neuroanesthesia and Intensive Care, Department 00004 of Neurological Sciences and Vision, Divisions of *Neurology and Full †Neurosurgery, Verona, Italy. Institution(s): Text Correspondence to Francesco Procaccio, MD, Neuroanesthesia and Intensive (PDF) Care, University and City Hospital, Pz Stefani, 1, 37124 Verona, Italy; e-mail: 69 K [email protected] Email Jumpstart Table of Contents: Find ≪ Neurologic complications in intensive care. Citing ≫ Pediatric neurologic emergencies. Articles ≪ Abstract Table Links of Cumulative evidence of potential benefits of Contents Abstract electroencephalography (EEG) and evoked potentials in About Complete Reference the management of patients with acute cerebral this ExternalResolverBasic damage has been confirmed. Continuous EEG Journal Outline monitoring is the best method for detecting ≫ nonconvulsive seizures and is strongly recommended for the treatment of status epilepticus. Continuously displayed, ● Abstract validated quantitative EEG may facilitate early detection
    [Show full text]
  • Critical Care Neurology and Neuro Critical Care
    CRITICAL CARE NEUROLOGY AND NEURO CRITICAL CARE 1 of 86 ROADMAP CRITICAL CARE NEUROLOGY • Analgesia, sedation and neuromuscular blockade o Basic principles, goals, general guidelines and assessment o Table of established drugs o Sedation for endotracheal intubation in critical care o Specialist analgesia in critical care • Sleep • Neurological dysfunction in critical care o Acute brain dysfunction o Delirium o Autonomic dysfunction o Critical illness neuromyopathy o Encephalopathy o Disease specific / syndromal encephalopathies ( ° Hypertensive encephalopathies ° Toxic and metabolic encephalopathies o Common infectious and inflammatory diseases of the nervous system ° Meningitis ° Encephalitis - gereralised and limbic • Transverse myelitis ° Acute inflammatory demyelinating polyneuropathy (AIDP) ° Myaesthenic crises ° Therapeutic plasma exchange and IVIg o Epilespy and seizures ° EEG ° Pathophysiology ° Epidemiology and epileptogenesis ° Epilepsy in ICU • Post injury epilepsy • Status epilepticus ° Anti-epileptic drugs (AEDs) • The controversy of prophylactic AEDs • Proconvulsant drugs o Persistent disorders of consciousness o Brain stem death: diagnosis, pathophysiology and management of the potential organ donor. CORE TOPICS IN NEURO CRITICAL CARE • Secondary brain injury o Pathophysiology – oedema, vascular autoregulation, sodium, glucose, temperature (metabolic supply / demand imbalance), oxygen, carbon dioxide o Prevention and neuroprotection – discussed for each topic above plus pharmacological therapies (epo, progesterone, magnesium
    [Show full text]
  • Molecular Mechanisms of Neuroimmune Crosstalk in the Pathogenesis of Stroke
    International Journal of Molecular Sciences Review Molecular Mechanisms of Neuroimmune Crosstalk in the Pathogenesis of Stroke Yun Hwa Choi 1, Collin Laaker 2, Martin Hsu 2, Peter Cismaru 3, Matyas Sandor 4 and Zsuzsanna Fabry 2,4,* 1 School of Pharmacy, University of Wisconsin-Madison, Madison, WI 53705, USA; [email protected] 2 Neuroscience Training Program, University of Wisconsin-Madison, Madison, WI 53705, USA; [email protected] (C.L.); [email protected] (M.H.) 3 Chemistry, University of Wisconsin-Madison, Madison, WI 53705, USA; [email protected] 4 Department of Pathology and Laboratory Medicine, University of Wisconsin-Madison, Madison, WI 53705, USA; [email protected] * Correspondence: [email protected] Abstract: Stroke disrupts the homeostatic balance within the brain and is associated with a significant accumulation of necrotic cellular debris, fluid, and peripheral immune cells in the central nervous system (CNS). Additionally, cells, antigens, and other factors exit the brain into the periphery via damaged blood–brain barrier cells, glymphatic transport mechanisms, and lymphatic vessels, which dramatically influence the systemic immune response and lead to complex neuroimmune communi- cation. As a result, the immunological response after stroke is a highly dynamic event that involves communication between multiple organ systems and cell types, with significant consequences on not only the initial stroke tissue injury but long-term recovery in the CNS. In this review, we discuss the complex immunological and physiological interactions that occur after stroke with a focus on how the peripheral immune system and CNS communicate to regulate post-stroke brain homeostasis. First, Citation: Choi, Y.H.; Laaker, C.; Hsu, we discuss the post-stroke immune cascade across different contexts as well as homeostatic regulation M.; Cismaru, P.; Sandor, M.; Fabry, Z.
    [Show full text]
  • The Range of Neurological Complications in Chikungunya Fever
    Neurocrit Care DOI 10.1007/s12028-017-0413-8 REVIEW ARTICLE The Range of Neurological Complications in Chikungunya Fever 1 2 3 4 5,6 T. Cerny • M. Schwarz • U. Schwarz • J. Lemant • P. Ge´rardin • E. Keller1 Ó Springer Science+Business Media New York 2017 Abstract assigning categories A–C, category A representing the Background Chikungunya fever is a globally spreading highest level of quality. Only A and B cases were con- mosquito-borne disease that shows an unexpected neu- sidered for further analysis. After general analysis, cases rovirulence. Even though the neurological complications were clustered according to geospatial criteria for subgroup have been a major cause of intensive care unit admission analysis. and death, to date, there is no systematic analysis of their Results Thirty-six of 1196 studies were included, yielding spectrum available. 130 cases. Nine were ranked as category A (diagnosis of Objective To review evidence of neurological manifesta- Neuro-Chikungunya probable), 55 as B (plausible), and 51 tions in Chikungunya fever and map their epidemiology, as C (disputable). In 15 cases, alternative diagnoses were clinical spectrum, pathomechanisms, diagnostics, therapies more likely. Patient age distribution was bimodal with a and outcomes. mean of 49 years and a second peak in infants. Fifty per- Methods Case report and systematic review of the litera- cent of the cases occurred in patients <45 years with no ture followed established guidelines. All cases found were reported comorbidity. Frequent diagnoses were encephali- assessed using a 5-step clinical diagnostic algorithm tis, optic neuropathy, neuroretinitis, and Guillain–Barre´ syndrome. Neurologic conditions showing characteristics of a direct viral pathomechanism showed a peak in infants Electronic supplementary material The online version of this and a second one in elder patients, and complications and article (doi:10.1007/s12028-017-0413-8) contains supplementary neurologic sequelae were more freque material, which is available to authorized users.
    [Show full text]
  • Patient-Tailored Connectomics Visualization for the Assessment of White Matter Atrophy in Traumatic Brain Injury
    Patient-Tailored Connectomics Visualization for the Assessment of White Matter Atrophy in Traumatic Brain Injury The Harvard community has made this article openly available. Please share how this access benefits you. Your story matters Citation Irimia, Andrei, Micah C. Chambers, Carinna M. Torgerson, Maria Filippou, David A. Hovda, Jeffry R. Alger, Guido Gerig, et al. 2012. Patient-tailored connectomics visualization for the assessment of white matter atrophy in traumatic brain injury. Frontiers in Neurology 3:10. Published Version doi://10.3389/fneur.2012.00010 Citable link http://nrs.harvard.edu/urn-3:HUL.InstRepos:8462352 Terms of Use This article was downloaded from Harvard University’s DASH repository, and is made available under the terms and conditions applicable to Other Posted Material, as set forth at http:// nrs.harvard.edu/urn-3:HUL.InstRepos:dash.current.terms-of- use#LAA METHODS ARTICLE published: 06 February 2012 doi: 10.3389/fneur.2012.00010 Patient-tailored connectomics visualization for the assessment of white matter atrophy in traumatic brain injury Andrei Irimia1, Micah C. Chambers 1, Carinna M.Torgerson1, Maria Filippou 2, David A. Hovda2, Jeffry R. Alger 3, Guido Gerig 4, Arthur W.Toga1, Paul M. Vespa2, Ron Kikinis 5 and John D. Van Horn1* 1 Laboratory of Neuro Imaging, Department of Neurology, University of California Los Angeles, Los Angeles, CA, USA 2 Brain Injury Research Center, Departments of Neurology and Neurosurgery, University of California Los Angeles, Los Angeles, CA, USA 3 Department of Radiology, David
    [Show full text]
  • Neurosurgeons and Neurocritical Care
    American Association of Neurological Surgeons and Congress of Neurological Surgeons POSITION STATEMENT on Neurosurgeons and Neurocritical Care Summary Statement Accreditation Council for Graduate Medical Education (ACGME)-approved neurosurgical residency training includes critical care management of patients with neurological disorders. Neurosurgeons are fully trained in neurointensive care by reason of training program requirements, and upon completion of training are competent to independently manage and direct treatment of patients with neurological disorders requiring critical care. Additional training in critical care is optional, but not necessary for neurosurgeons to manage neurocritical care patients following residency training. Certification in neurological surgery is through the American Board of Neurological Surgery (ABNS), and includes certification for critical care of patients with neurological conditions. No other certification is required for ABNS diplomats for privileges in neurological surgery or neurocritical care management. Additional certification by organizations unrecognized by the American Board of Medical Specialties (ABMS) is unnecessary for ensuring neurosurgeon training, competency, or credentialing in intensive or critical care. Patient Access to Neurosurgeon Care in Critical Care Settings The American Association of Neurological Surgeons (AANS) (http://www.aans.org) and the Congress of Neurological Surgeons (CNS) (http://www.cns.org) are professional scientific and educational associations with over 7,400 members worldwide. All active members of the AANS are board certified by the American Board of Neurological Surgery (ABNS), the Royal College of Physicians and Surgeons of Canada, or the Mexican Council of Neurological Surgery, AC. The AANS and the CNS are dedicated to advancing the specialty of neurological surgery in order to provide the highest quality of neurosurgical care to the public.
    [Show full text]
  • ACGME Program Requirements for Graduate Medical Education In
    ACGME Program Requirements for Graduate Medical Education in Neuroendovascular Intervention (Proposed name change for Endovascular Surgical Neuroradiology) Summary and Impact of Major Requirement Revisions Requirement #: N/A – Subspecialty Name Requirement Revision (significant change only): Endovascular Surgical Neuroradiology Neuroendovascular Intervention 1. Describe the Review Committee’s rationale for this revision: The Review Committee, in collaboration with the Review Committees for Neurology and Neurological Surgery, determined that while this area of study goes by many different names in practice, the current name of endovascular surgical neuroradiology is not inclusive as it does not encompass the intervention portion of the subspecialty. 2. How will the proposed requirement or revision improve resident/fellow education, patient safety, and/or patient care quality? N/A 3. How will the proposed requirement or revision impact continuity of patient care? N/A 4. Will the proposed requirement or revision necessitate additional institutional resources (e.g., facilities, organization of other services, addition of faculty members, financial support; volume and variety of patients), if so, how? N/A 5. How will the proposed revision impact other accredited programs? N/A Requirement #: Int.C. Requirement Revision (significant change only): The program shall offer one year of graduate medical education in endovascular surgical neuroradiology. (Core)* The educational program in neuroendovascular intervention must be at least 24 months in length. (Core) 1. Describe the Review Committee’s rationale for this revision: This major revision reflects efforts to align the requirements with the Committee on Advanced Subspecialty Training (CAST) requirements for neuroendovascular surgery. The CAST requirements emphasize a need for greater proficiency in the outpatient evaluation and care of pre- and post-procedure endovascular patients.
    [Show full text]
  • Fnb- Neuro Anesthesia and Critical Care
    Guidelines for Competency Based Training Programme in FNB- NEURO ANESTHESIA AND CRITICAL CARE NATIONAL BOARD OF EXAMINATIONS Medical Enclave, Ansari Nagar, New Delhi-110029, INDIA Email: [email protected] Phone: 011 45593000 1 CONTENTS I. OBJECTIVES OF THE PROGRAMME a) Programme goal b) Programme objective II. ELIGIBILITY CRITERIA FOR ADMISSION III. TEACHING AND TRAINING ACTIVITIES IV. SYLLABUS V. COMPETENCIES VI. LOG BOOK VII. NBE LEAVE GUIDELINES VIII. EXAMINATION – a) FORMATIVE ASSESSMENT b) FELLOWSHIP EXIT THEORY & PRACTICAL IX. RECOMMENDED TEXT BOOKS AND JOURNALS 2 PROGRAMME GOAL The course has been designed to train candidates by the anesthesiologists in the principles and practice of Neuroanaesthesia and Neurocritical care. The training program should enable the candidates to function independently as faculty / consultant in the anaesthetic / intensive care management of the patients with neurological disorders coming for neurosurgical / radiological intervention. PROGRAMME OBJECTIVES At the end of the course, the candidate should be able to: Understand physiological and pathological basis of central nervous system disorders. Understand the theoretical basis of organ dysfunction and critical illness Develop the knowledge and skills to diagnose critical illnesses and their complications Critically evaluate published literature Learn to practice evidence-based medicine in managing neurological patients Develop skills of communication with family members of critically ill patients Apply the highest ethical standards in the
    [Show full text]
  • The Neurointensive Care Nursery and Evolving Roles for Nursing
    The Neurointensive Care Nursery and Evolving Roles for Nursing Susan Peloquin, RN, MS, PNP Annette Carley, DNP, NNP, PNP Sonia L. Bonifacio, MD Hannah C. Glass, MDCM, MAS Disclosure ABSTRACT The authors have no relevant financial interest or affiliations Neonatal neurocritical care is an emerging subspecialty that combines the expertise of critical care with any commercial interests medicine and neurology with that of nursing and other providers in an interprofessional team related to the subjects discussed approach to care.1,2 Neurocritical care of the neonate has roots in adult and pediatric practice. It within this article. has been demonstrated that adults with acute neurologic conditions who are treated in a specialized No commercial support or neurocritical care unit have reduced morbidity and mortality, as well as decreased length of stay, lower sponsorship was provided for this educational activity. costs, and reduced need for neurosurgical procedures. In pediatrics, neurocritical care has focused on various primary and secondary neurologic conditions complicating critical care that also contribute to mortality, morbidity, and duration of hospitalization. However, the concept of neurocritical care as a subspecialty in pediatric practice is still evolving, and evidence demonstrating improved outcomes is lacking.3–5 In the neonatal intensive care nursery, neurocritical care is also evolving as a subspecialty concept to address both supportive and preventive care and optimize neurologic outcomes for an at-risk neonatal patient population. To enhance effectiveness of this care approach, nurses must be prepared to appropriately recognize acute changes in neurologic status, implement protocols that specifically address neurologic conditions, and carefully monitor neurologic status to help prevent secondary injury.
    [Show full text]
  • Textbook of Neurointensive Care
    Netherlands Journal of Critical Care Accepted October 2014 BOOK REVIEW Textbook of neurointensive care A.J. Layon, A. Gabrielli, W.A. Friedman (Eds.) This textbook aims to cover all aspects of neurointensive care. bit redundant in this volume, in my opinion, as is an extensive Although not explicitly stated by the authors in the foreword, chapter on spinal cord surgeries. Neuroradiology, ethical issues it seems to be aimed primarily at educating neurointensive and brain death are handled quite extensively and a chapter on care, which is an acknowledged subspeciality in intensive care applied pharmacotherapy concludes this volume. in the United States. It is edited by acknowledged critical care Apart from some critiques as stated above, this seems to be physicians and a neurosurgeon (two of them also edited the an essential book to be available for libraries of ICUs where well-known Civetta, Taylor and Kirbey’s Critical Care). Authors neuroinjured patients are managed. Keywords for this volume include seasoned neurointensive care researchers and clinicians. are: comprehensive, well-illustrated and well organised. The book is organised in introductory sections covering neurocritical care organisation, quality management and neurocritical care as emerging subspeciality in the US. The following sections involve ‘neuroanatomy and pathophysiology’, ‘(non)invasive neuromonitoring’, ‘general intensive care issues specifically applied to the neuroinjured patient’, ‘(evidence- based) management of specific neurointensive care diagnoses (e.g. subarachnoid haemorrhage, traumatic brain injury)’ and ‘situations of special interest’. The neuroanatomy and pathology chapters cover the basics very well and are richly illustrated. The neuromonitoring chapters provide a good overview although a more detailed discussion with regard to the current status and usefulness of different monitors for clinical practice would have been valuable.
    [Show full text]
  • Clinical Outcome of Children Admitted with Acute Encephalitis Syndrome in a Tertiary Care Hospital, Dhaka, Bangladesh Dr
    Scholars Journal of Applied Medical Sciences Abbreviated Key Title: Sch J App Med Sci ISSN 2347-954X (Print) | ISSN 2320-6691 (Online) Paediatric Neuroscience Journal homepage: https://saspublishers.com Clinical Outcome of Children Admitted with Acute Encephalitis Syndrome in a Tertiary Care Hospital, Dhaka, Bangladesh Dr. Mustafa Mahbub1*, Dr. Suraj C Majumder2, Dr. AZM Mosiul Azam3 1Professor, Paediatric neuroscience department, Bangladesh institute of child health (BICH), Dhaka Shishu (children) Hospital, Dhaka, Bangladesh 2Registrar, Paediatric neuroscience department, Dhaka Shishu (Children) Hospital, Dhaka, Bangladesh 3Associate Professor, Paediatric neuroscience department, Bangladesh institute of child health (BICH), Dhaka Shishu (Children) Hospital, Dhaka, Bangladesh DOI: 10.36347/sjams.2021.v09i05.004 | Received: 26.03.2021 | Accepted: 03.05.2021 | Published: 08.05.2021 *Corresponding author: Mustafa Mahbub Abstract Original Research Article Background: Acute encephalitides are the most common cause of acute neurological disease in children. This is a potentially devastating neurological syndrome with different etiologies including direct central nervous system infection with different agents most commonly viral and those mediated by immune system. The management has changed dramatically in recent years for several reasons including improved diagnostic and brain imaging techniques, better antiviral and more advanced neurointensive care and rehabilitation. Objective: To observe the presentation, diagnostic approach and management of the children admitted in neuroscience department of Dhaka Shishu Hospital. Method: A retrospective analysis of suspected encephalitis was done from records of discharged patients of the Neurosciences Department at Dhaka Shishu Hospital from January 2011-February 2013. SPSS is used for statistical analysis. Result: This study examined 31 patients (17 boys; 56.5 %, 14 Girls;45.16) with a mean onset age of 5.32 years (range 0.5-12 years).Out of 31 cases of AES, 8 patients (25.81%) were JE and 24 patients (77.42%) were non- JE.
    [Show full text]
  • INTENSIVE CARE in NEUROLOGY and NEUROSURGERY Pathophysiological Basis for the Management of Acute Cerebral Injury
    Daniel Agustín Godoy INTENSIVE CARE IN NEUROLOGY AND NEUROSURGERY Pathophysiological Basis for the Management of Acute Cerebral Injury VOLUME 1 Head Editor Daniel Agustín Godoy, MD, FCCM Neurointensive Care Unit- Sanatorio Pasteur Intensive Care Unit Hospital Interzonal de Agudos ‘’San Juan Bautista’’ Catamarca. Argentina Associate Editor Gustavo Rene Piñero, MD, FCCM Intensive Care Unit Hospital Municipal ‘’Leonidas Lucero’’ Assistant Professor Critical and Emergency MedicineHealth Sciences Department - South University Bahia Blanca, Buenos Aires. Argentina © SEEd srl. All rights reserved Piazza Carlo Emanuele II, 19 – 10123 Torino, Italy Tel. 011.566.02.58 – Fax 011.518.68.92 www.edizioniseed.it [email protected] First edition February 2013 VOLUME 1 ISBN 978-88-9741-939-6 Printed on acid-free paper by Tipografia Graphot, Torino (Italy) Although the information about medication given in this book has been carefully checked, the author and publisher accept no liability for the accuracy of this information. In every individual case the user must check such information by consulting the relevant literature. This work is subject to copyright. All rights are reserved, whether the whole or part of the material is concerned, specifically the rights of translation, reprinting, reuse of illustrations, recitation, broadcast- ing, reproduction on microfilm or in any other way, and storage in data banks. Duplication of this publi- cation or parts thereof is permitted only under the provisions of the Italian Copyright Law in its current version, and permission for use must always be obtained from SEEd Medical Publishers Srl. Violations are liable to prosecution under the Italian Copyright Law. To my parents Mirtha and Justino for giving me the life, education and the opportunity to study and acquire this wonderful profession.
    [Show full text]