BDJ 2012. Letter to Editor. TMJ Dysfunction

Total Page:16

File Type:pdf, Size:1020Kb

BDJ 2012. Letter to Editor. TMJ Dysfunction Send your letters to the Editor, British Dental Journal, 64 Wimpole Street, London, W1G 8YS Letters to the Editor Email [email protected] LETTERS Priority will be given to letters less than 500 words long. Authors must sign the letter, which may be edited for reasons of space. Readers may now comment on letters via the BDJ website (www.bdj.co.uk). A 'Readers' Comments' section appears at the end of the full text of each letter online. TMJ DYSFUNCTION as a side effect of long-term treatment TRAUMA BY IPAD Sir, the general dental practitioner may with antipsychotic drugs and approxi- Sir, an 11-year-old boy attended the be the first point of contact for patients mately 10% of patients exposed to these emergency clinic at Glasgow Dental Hos- with TMJ problems. Presenting com- will experience a dystonic reaction.4 It pital and School’s paediatric department plaints may include pain, limitation in is important that GDPs are aware of this in March 2012 following a second inci- mouth opening, clicking and disloca- condition for successful management. dent of trauma to his upper right central tion with occlusal changes. There may Such dislocation of the TMJ is usually incisor. The first incident, a skateboard- also be associated tooth wear. Often the unilateral, although reports have shown ing accident in May 2011, resulted in a symptoms may be related to functional that the mechanical energy derived from middle third comminuted root fracture, habits such as bruxism. the oromandibular dystonia can occa- which healed with connective tissue However, various medications may also sionally cause bilateral dislocations. union after a period of splinting and result in TMJ dysfunction, due to side Classic signs of TMJ dislocation the tooth remained vital. The second effects such as bruxism and TMJ disloca- include pain, inability to completely traumatic incident resulted in an oblique tion. There have been several reported close the mouth, deviation of the jaw to crown fracture at the level of the cervi- incidents of iatrogenic bruxism involving the side, forward protrusion of the man- cal margin (see accompanying radio- diurnal bruxism1 thought to be associated dible, difficulty swallowing, drooling graph, Fig. 1). The coronal fragment was with dopaminergic blockade, and noctur- from the mouth, difficulty chewing and subsequently removed, the exposed pulp nal bruxism. Nocturnal bruxism has been difficulty articulating. Treatment of the extirpated and an overdenture provided reported with venlafaxine, a serotonin/ dislocation requires manual manipula- to restore aesthetics in the short term. noradrenaline reuptake inhibitor, which tion of the joint, aided with the use of Ideally, the root will be obturated and responded to gabapentin,2 as well as three analgesics and benzodiazepines. Botuli- retained in order to facilitate placement selective serotonin reuptake inhibitors num toxin A (BTA) injection therapy is of a single osseointegrated implant at an (SSRIs), paroxetine, fluoxetine and setra- indicated where conservative treatment appropriate age. line. In both reports the SSRI-associated has failed, and surgery carries an unac- bruxism was treated with buspirone. ceptable risk. BTA is also beneficial in Nocturnal bruxism may also be second- patients who suffer recurrent disloca- ary to the SSRI citalopram.3 tion of the TMJ as a result of impaired Phenothiazines are widely used for muscle coordination secondary to their antipsychotic, antiemetic and seda- mandibular dystonia, early and late tive properties. They are known to block dyskinesias, epilepsy and brain stem the dopamine receptors in the brain, syndromes of various origins. which may account for their extrapy- K. Bhuva, W. Matthews, ramidal side effects. These side effects L. M. Carter, A. Kanatas cause various movement disorders Leeds including oral bucco-lingual dyskinesia 1. Brown E S, Hong S C. Antidepressant-induced (continual side to side or circular chew- bruxism successfully treated with gabapentin. J Am Dent Assoc 1999; 130: 1467–1469. ing movements of the jaw) and oroman- 2. Ellison J M, Stanziani P. SSRI-associated nocturnal dibular dystonia (OMD). OMD is a focal bruxism in four patients. J Clin Psychiatry 1993; 54: 432–434. dystonia manifested by involuntary 3. Wise M. Citalopram-induced bruxism. Br J Psychia- muscle contractions producing repeti- try 2001; 178: 182. 4. Maestre-Ferrín L, Burguera J A, Peñarrocha-Diago tive, patterned mouth, jaw, and tongue M, Peñarrocha-Diago M. Oromandibular dystonia: movements.4 This is often referred to as a dental approach. Med Oral Pathol Oral Cir Bucal primary (idiopathic) or secondary dysto- 2009; 15: e25–27. Fig. 1 Oblique crown fracture at the cervical margin nia. The latter most commonly develops DOI: 10.1038/sj.bdj.2012.728 BRITISH DENTAL JOURNAL VOLUME 213 NO. 4 AUG 25 2012 145 © 2012 Macmillan Publishers Limited. All rights reserved. .
Recommended publications
  • Management of Saliva and Drooling Excessive Saliva and Drooling Affects up to 50% of People with Parkinson’S (PD)
    Management of Saliva and Drooling Excessive saliva and drooling affects up to 50% of people with Parkinson’s (PD). Drooling can be embarrassing and can limit social interactions for the person with PD. Saliva and Drooling Parkinson Information Parkinson It can also be an important symptom of swallowing difficulty, which can increase the risk of choking on saliva. People with Parkinson’s disease do not swallow automatically due to rigidity and impaired mobility of the muscles of the palate, throat and esophagus. Saliva pools in the mouth and can potentially become a hazard since swallowing into the lungs carries the risk of pneumonia. If you have poor posture, saliva collects in the front of the mouth, resulting in drooling. Cause and symptoms Decreased control of saliva is most often caused by changes in the ability to swallow, rather than from producing too much saliva. A common cause of drooling for people with PD is the weakening and/or loss of motor control of the muscles involved in swallowing. You may experience one or more of the following symptoms: • Decreased ability to keep your mouth closed at rest, known as the “open mouth posture” • Difficulty keeping lips closed • Lack of awareness of the saliva in your mouth • Wetness at the sides of your mouth • A wet sounding voice • Drooling with posture changes • Coughing and/or choking Evaluation and treatment Speak with your physician about all symptoms that may not be related to PD. If you are experiencing drooling or choking on your saliva, you may require a swallowing evaluation by a Speech Language Pathologist.
    [Show full text]
  • 16. Questions and Answers
    16. Questions and Answers 1. Which of the following is not associated with esophageal webs? A. Plummer-Vinson syndrome B. Epidermolysis bullosa C. Lupus D. Psoriasis E. Stevens-Johnson syndrome 2. An 11 year old boy complains that occasionally a bite of hotdog “gives mild pressing pain in his chest” and that “it takes a while before he can take another bite.” If it happens again, he discards the hotdog but sometimes he can finish it. The most helpful diagnostic information would come from A. Family history of Schatzki rings B. Eosinophil counts C. UGI D. Time-phased MRI E. Technetium 99 salivagram 3. 12 year old boy previously healthy with one-month history of difficulty swallowing both solid and liquids. He sometimes complains food is getting stuck in his retrosternal area after swallowing. His weight decreased approximately 5% from last year. He denies vomiting, choking, gagging, drooling, pain during swallowing or retrosternal pain. His physical examination is normal. What would be the appropriate next investigation to perform in this patient? A. Upper Endoscopy B. Upper GI contrast study C. Esophageal manometry D. Modified Barium Swallow (MBS) E. Direct laryngoscopy 4. A 12 year old male presents to the ER after a recent episode of emesis. The parents are concerned because undigested food 3 days old was in his vomit. He admits to a sensation of food and liquids “sticking” in his chest for the past 4 months, as he points to the upper middle chest. Parents relate a 10 lb (4.5 Kg) weight loss over the past 3 months.
    [Show full text]
  • Osteoid-Osteoma
    Benign tumors of soft tissues and bones of head at children. Classification, etiology. Diagnostics, differential diagnostics, treatment and rehabilitation of children. Pediatric Surgical Dentistry Lector - Kolisnik I.A. 0504044002 (Viber, Telegram) Plan of lecture and their organizational structure. № The main stages of the Type of lecture. Means of time distribution lecture and activating students. Methodical their content support materials 1. Preparatory stage. look p 1 and 2 5 % definitionrelevance of the topic, learning objectives of the lecture and motivation 2. The main stage. Teaching lectureClinical lecture. 85 % material according to the plan: -90% 1. 1 Frequency of malignant processes of SHLD in children. 2. Phases of carcinogenesis 3. Signs of benign and malignant process. 4. Methods of diagnosis of SHLD tumors. 5. The structure of malignant pathology of the thyroid gland in children. 6. Clinical and morphological features of malignant tumors of the thyroid gland in children. Principles of treatment. 7. Basic principles of rehabilitation of children with oncological pathology. 8. Stages of formation of bone regenerate. 9. Clinical case. 1. The final stage Answers to possible 5 % 2. Lecture summary. questions. 3. General conclusions. Classification of benign neoplasm Type of tissue Type of neoplasm Pavement epithelium Squamous cell papilloma Secretory (glandular) epithelium Adenoma Connective Fibroma Adipose Lipoma Smooth muscle Leiomyoma Osseous Osteoma Cartilaginous Chondroma Lymphoid Lymphoma Transversal striated muscle Rhabdomioma
    [Show full text]
  • Oral Pathology Unmasking Gastrointestinal Disease
    Journal of Dental Health Oral Disorders & Therapy Review Article Open Access Oral pathology unmasking gastrointestinal disease Abstract Volume 5 Issue 5 - 2016 Different ggastrointestinal disorders, such as Gastroesophageal Reflux Disease (GERD), Celiac Disease (CD) and Crohn’s disease, may manifest with alterations of the oral cavity Fumagalli LA, Gatti H, Armano C, Caruggi S, but are often under and misdiagnosed both by physicians and dentists. GERD can cause Salvatore S dental erosions, which are the main oral manifestation of this disease, or other multiple Department of Pediatric, Università dell’Insubria, Italy affections involving both hard and soft tissues such as burning mouth, aphtous oral ulcers, Correspondence: Silvia Salvatore, Pediatric Department of erythema of soft palate and uvula, stomatitis, epithelial atrophy, increased fibroblast number Pediatric, Università dell’Insubria, Via F. Del Ponte 19, 21100 in chorion, xerostomia and drooling. CD may be responsible of recurrent aphthous stomatitis Varese, Italy, Tel 0039 0332 299247, Fax 0039 0332 235904, (RAS), dental enamel defects, delayed eruption of teeth, atrophic glossitis and angular Email chelitis. Crohn’s disease can occur with several oral manifestations like indurated tag-like lesions, clobbestoning, mucogingivitis or, less specifically, with RAS, angular cheilitis, Received: October 30, 2016 | Published: December 12, 2016 reduced salivation, halitosis, dental caries and periodontal involvement, candidiasis, odynophagia, minor salivary gland enlargement, perioral
    [Show full text]
  • Neurology and the Gastrointestinal System
    J Neurol Neurosurg Psychiatry 1998;65:291–300 291 J Neurol Neurosurg Psychiatry: first published as 10.1136/jnnp.65.3.291 on 1 September 1998. Downloaded from NEUROLOGY AND MEDICINE Neurology and the gastrointestinal system G D Perkin, I Murray-Lyon The interrelation of neurology and the gas- that the two techniques are complementary, trointestinal system includes defects of gut acetylcholinesterase staining being particularly innervation, primary disorders of the nervous helpful when the biopsy material does not system (or muscle) which lead to gastrointesti- include submucosa, or in older infants or chil- nal symptoms—for example, dysphagia—and, dren in whom the population of distal submu- finally, certain gut disorders which include cosal ganglion cells may be less dense.6 neurological features in their clinical range. The first of this trio will be discussed only Gastrointestinal disorders due to briefly in this review, the second and third in neurological disease more detail. DYSPHAGIA A neurogenic mechanism for dysphagia, which Defects of innervation may have either sensory or motor components, ACHALASIA or both, can result from a disorder at the oral, Achalasia is characterised by an absence of pharyngeal, or oesophageal phase of swallow- peristalsis in the oesophageal body accompa- ing. In most patients, the neurological disorder nied by a failure of relaxation of the lower is evident, but in others, dysphagia is the oesophageal sphincter.1 Although the condi- presenting feature. Besides the dysphagia, tion can be secondary to other disease other symptoms suggesting a neurogenic copyright. processes—for example, Chagas’ disease—in mechanism include drooling of saliva, nasal Europeans it is usually a primary disorder.
    [Show full text]
  • A 10-Year Retrospective Review of Botulinum Toxin Injections and Surgical Management of Sialorrhea
    Open Access Original Article DOI: 10.7759/cureus.7916 A 10-year Retrospective Review of Botulinum Toxin Injections and Surgical Management of Sialorrhea Rachel E. Weitzman 1 , Kosuke Kawai 2 , Roger Nuss 3 , Amy Hughes 4 1. Otolaryngology, Harvard Medical School, Boston, USA 2. Otolaryngology, Boston Children’s Hospital, Boston, USA 3. Otolaryngology, Boston Children's Hospital, Boston, USA 4. Otolaryngology, Connecticut Children's Medical Center, Hartford, USA Corresponding author: Amy Hughes, [email protected] Abstract Background Sialorrhea is a common comorbidity among children with neurologic disorders. Botulinum toxin injections and surgical procedures are recommended for the management of pathological sialorrhea in patients who fail conservative management or with concerns for salivary aspiration. The following review evaluates outcomes following botulinum toxin injections and surgical interventions for sialorrhea over a 10-year period with a focus on treatment options and outcomes for patients with anterior and posterior drooling. Methods The study included all patients less than 25 years of age who underwent a procedure for drooling (Current Procedural Terminology (CPT) codes 42440, 42450, 42509, 42510, 64611 matched with the International Classification of Diseases (ICD)-9 and ICD-10 codes 527.7 and K11.7) from January 1, 2006 to December 31, 2015. A chart review collected demographics, drooling medication use, and type of drooling (anterior, posterior, both). Outcome variables included pre- and post-procedure number of bibs, parent-reported outcomes, post-intervention drooling medication requirement, post-procedure length of stay, and complications. Results Seventy-one patients were included in our analysis, with 88 total procedures performed. The average age at first intervention was 8.9 years; 43 patients were male and 40 patients had cerebral palsy.
    [Show full text]
  • Oral Complications at the End of Life
    CE HOURS Continuing Education1.5 Oral Complications at the End of Life Although dysphagia and stomatitis can have devastating effects on the quality of a patient’s life, there are many ways to manage them. By Constance Dahlin, MSN, APRN, BC, PCM en Eldredge, a 76-year-old retired truck driver, has end-stage heart disease and dementia. In the last year, his health has worsened considerably. He is bedbound, extremely weak, and occasionally short of breath. He has a poor appetite and is losing weight. After an evaluation including a com- plete blood count and computed tomographic scans of the chest and abdomen revealed colon cancer, he received chemotherapy, which ended Ktwo months ago. Mr. Eldredge is being cared for at home by his 75-year- old wife, Jean, and two of their adult children. He complains of dry mouth and says that foods “don’t taste right.” During meals he often coughs and sputters and says his mouth hurts when he eats and that food gets caught in his throat. His wife reports that he often must be coaxed to eat more. Swallowing difficulties (dysphagia) and oral mucosal inflammation (stomatitis) are common in patients who have progressive terminal ill- ness. Perhaps because these conditions are common within the context of a patient’s deconditioning near the end of life, many providers consider them relatively trivial and they’re therefore underreported, underesti- mated, and frequently overlooked for more prominent symptoms such as pain or shortness of breath.1-4 But swallowing difficulties and oral mucosal inflammation can have tremendous adverse consequences for patients and families and should not be dismissed.
    [Show full text]
  • Salivary Gland Disorders and Tumours
    Salivary gland disorders and tumours Sumamry This lesson is one we all tend to avoid because most of them sound the same! Hopefully this lesson will help you with clarification. Introduction to Salivary gland disorders and tumours: List of Salivary gland disorders and tumours Viral sialadenitis (mumps) Bacterial sialadenitis Sialosis (sialadenosis) Sialolithiasis Mucocele Acute Necotising Sialometaplasia Tumours: Pleomorphic adenoma Warthins Tumour Mucoepidermoid carcinoma Adenoid cystic carcinoma Acinic cell carcinoma Low-grade polymorphic adenoma Sjogren's syndrome Xerostomia Sialorrhea ReviseDental.com Key words: Sialosis non-pathogenic, non-neoplastic increase in salivary gland size Sialodenitis ductal infection Sialolithiasis duct obstruction Sialectasis cystic widening of the duct Sialorrhea excessive salivation/drooling (1) Acute Viral Sialadenitis Aetiology and epidemiology Common in the childhood disease Mumps caused by the RNA virus Parmyxovirus Typically affects the parotid gland Spread by droplet spread or direct contact 2-3 week incubation period precedes the clinical symptoms Can cause extrasalivary manifestations such as Orchitis Oophoritis Pancreatitis Clinical features Painful Typically bilateral enlargement of parotid glands Skin over the glands is not affected which distinguishes from bacterial sialodenitis Malaise, fever and headaches Histopathology Accumulation of neutrophils and fluid in the lumen of the ductal structures Diagnosis Made on clinical presentation Management FluidsReviseDental.com and medication for
    [Show full text]
  • ODONTOGENTIC INFECTIONS Infection Spread Determinants
    ODONTOGENTIC INFECTIONS The Host The Organism The Environment In a state of homeostasis, there is Peter A. Vellis, D.D.S. a balance between the three. PROGRESSION OF ODONTOGENIC Infection Spread Determinants INFECTIONS • Location, location , location 1. Source 2. Bone density 3. Muscle attachment 4. Fascial planes “The Path of Least Resistance” Odontogentic Infections Progression of Odontogenic Infections • Common occurrences • Periapical due primarily to caries • Periodontal and periodontal • Soft tissue involvement disease. – Determined by perforation of the cortical bone in relation to the muscle attachments • Odontogentic infections • Cellulitis‐ acute, painful, diffuse borders can extend to potential • fascial spaces. Abscess‐ chronic, localized pain, fluctuant, well circumscribed. INFECTIONS Severity of the Infection Classic signs and symptoms: • Dolor- Pain Complete Tumor- Swelling History Calor- Warmth – Chief Complaint Rubor- Redness – Onset Loss of function – Duration Trismus – Symptoms Difficulty in breathing, swallowing, chewing Severity of the Infection Physical Examination • Vital Signs • How the patient – Temperature‐ feels‐ Malaise systemic involvement >101 F • Previous treatment – Blood Pressure‐ mild • Self treatment elevation • Past Medical – Pulse‐ >100 History – Increased Respiratory • Review of Systems Rate‐ normal 14‐16 – Lymphadenopathy Fascial Planes/Spaces Fascial Planes/Spaces • Potential spaces for • Primary spaces infectious spread – Canine between loose – Buccal connective tissue – Submandibular – Submental
    [Show full text]
  • Treatments for Ankyloglossia and Ankyloglossia with Concomitant Lip-Tie Comparative Effectiveness Review Number 149
    Comparative Effectiveness Review Number 149 Treatments for Ankyloglossia and Ankyloglossia With Concomitant Lip-Tie Comparative Effectiveness Review Number 149 Treatments for Ankyloglossia and Ankyloglossia With Concomitant Lip-Tie Prepared for: Agency for Healthcare Research and Quality U.S. Department of Health and Human Services 540 Gaither Road Rockville, MD 20850 www.ahrq.gov Contract No. 290-2012-00009-I Prepared by: Vanderbilt Evidence-based Practice Center Nashville, TN Investigators: David O. Francis, M.D., M.S. Sivakumar Chinnadurai, M.D., M.P.H. Anna Morad, M.D. Richard A. Epstein, Ph.D., M.P.H. Sahar Kohanim, M.D. Shanthi Krishnaswami, M.B.B.S., M.P.H. Nila A. Sathe, M.A., M.L.I.S. Melissa L. McPheeters, Ph.D., M.P.H. AHRQ Publication No. 15-EHC011-EF May 2015 This report is based on research conducted by the Vanderbilt Evidence-based Practice Center (EPC) under contract to the Agency for Healthcare Research and Quality (AHRQ), Rockville, MD (Contract No. 290-2012-00009-I). The findings and conclusions in this document are those of the authors, who are responsible for its contents; the findings and conclusions do not necessarily represent the views of AHRQ. Therefore, no statement in this report should be construed as an official position of AHRQ or of the U.S. Department of Health and Human Services. The information in this report is intended to help health care decisionmakers—patients and clinicians, health system leaders, and policymakers, among others—make well-informed decisions and thereby improve the quality of health care services. This report is not intended to be a substitute for the application of clinical judgment.
    [Show full text]
  • CDHO Factsheet Oral Cancer
    Disease/Medical Condition ORAL CANCER Date of Publication: August 7, 2014 (also known as “oral cavity cancer”) Is the initiation of non-invasive dental hygiene procedures* contra-indicated? Possibly (dental hygiene procedures should not be scheduled while the patient/client is experiencing oral ulcerations and pain, has an acute oral infection, has an absolute neutrophil count ≤ 1.0 X 109/L, or has a platelet count ≤ 50 X 109/L) Is medical consult advised? ..................................... Possibly (e.g., if suspicious lesion is detected; if intraoral infection and/or immunosuppression is suspected, particularly if the patient/client is undergoing radiation therapy and/or chemotherapy) Is the initiation of invasive dental hygiene procedures contra-indicated?** Possibly (contra-indicated for persons undergoing radiotherapy and/or chemotherapy for oral cancer); furthermore, dental hygiene procedures should not be scheduled while the patient/client is experiencing oral ulcerations and pain, has an acute oral infection, has an absolute neutrophil count ≤ 1000/mm3, or has a platelet count ≤ 50,000/mm3) Is medical consult advised? ...................................... See above. Is medical clearance required? .................................. Yes, if the patient/client is about to undergo or is undergoing active chemotherapy or radiation therapy for oral cancer. – Yes, if the patient/client is scheduled for major oral surgery for oral cancer. Is antibiotic prophylaxis required? ............................. No, not typically (although cancer or treatment-induced immunosuppression may warrant consideration of antibiotic prophylaxis). Is postponing treatment advised? .............................. Possibly (depends on whether cancer and its treatment may interfere with invasive procedures and whether there is immunosuppression associated with cancer treatment).1 Oral management implications Dental hygienists play an important role in early detection of oral cancer, leading to timely medical/dental referral and potential biopsy, endoscopy, and imaging.
    [Show full text]
  • Ptyalism & Pseudoptyalism
    Diagnostic Tree Internal Medicine Peer Reviewed Julie Allen, BVMS, MS, DACVIM (Small Animal) Ptyalism & Antech Diagnostics Pseudoptyalism Drooling* Ptyalism (excessive saliva** production) Pseudoptyalism (inability to swallow normal saliva production; drooling**) History & examination • Complete physical examination, evaluating salivary gland size and symmetry • Full neurologic examination • Breed: Giant breeds (eg, St. Bernard) or Yorkshire and Maltese terriers (increased PSS incidence) • Age: Young animals likely to ingest toxins/FBs or have congenital issues; neoplasia likely in older animals • History of vaccination (eg, rabies, calicivirus) or trauma (eg, electric cord injury) • Exposure to toxins, medications, topical products • Duration: Acute (eg, FB) vs chronic (eg, neoplasm) • Discoloration of saliva (eg, blood, purulent discharge) suggestive of oral problem • Halitosis may indicate oral, esophageal, or gastric disease • Pawing at face/mouth may indicate orofacial pain, hypocalcemia • Change in eating behavior: Dropping food, chewing on one side of the mouth, pseudoanorexia or hyporexia • Diet (eg, high-protein) can cause drooling because of precipitation of HE in patients with liver dysfunction • Other GI (ie, retching, regurgitation, vomiting, weight loss) or neurologic signs (ie, seizures, gagging, dysphagia) Known toxin Known medication exposure Toxicity Medication reaction • Household cleaners • Medications/topical products given topically or PO • Plants/trees (eg, Kentucky coffee tree, poinsettia) (eg, selamectin, moxidectin–imidacloprid)
    [Show full text]