Compartment Syndrome, Chronic

Total Page:16

File Type:pdf, Size:1020Kb

Compartment Syndrome, Chronic CLINICAL Compartment Syndrome, Chronic REVIEW Indexing Metadata/Description › Title/condition: Compartment Syndrome, Chronic › Synonyms: Chronic exertional compartment syndrome; exercise-induced compartment syndrome; recurrent compartment syndrome; subacute compartment syndrome; chronic exertional compartment syndrome; compartment syndrome, chronic exertional; compartment syndrome, exercise-induced; compartment syndrome, recurrent; chronic compartment syndrome › Anatomical location/body part affected: Most often affects the lower leg, especially the anterior and lateral compartments,(35) but can affect all compartments, including gluteus, foot, shoulder, hand(35), upper leg, and forearm(1) › Area(s) of specialty: Orthopedic rehabilitation, sports rehabilitation, cardiovascular rehabilitation › Description • Compartment syndrome – “A condition in which the circulation and function of tissues within a closed space are compromised by increased pressure within that space”(2) • There are two types of compartment syndrome, acute and chronic. Acute compartment syndrome is a surgical emergency;(2) please see Clinical Review…Compartment Syndrome, Acute; Item Number: T708485 for details • Chronic compartment syndrome– A reversible rise in tissue pressure to abnormal levels brought on by exertion and relieved by rest that occurs within an enclosed space and has the potential to threaten perfusion and tissue viability(2) • The prevalence of chronic compartment syndrome is not known since it is not uncommon for patients to administer self-directed treatment or activity modification and not seek medical attention(32) • 95% of all chronic compartment syndrome cases affect the leg(32) Authors › ICD-9 codes Ellenore Palmer, BScPT, MSc • 729.7 nontraumatic compartment syndrome Cinahl Information Systems, Glendale, CA Andrea Callanen, MPT • 729.71 nontraumatic compartment syndrome of upper extremity: nontraumatic Cinahl Information Systems, Glendale, CA compartment syndrome of shoulder, arm, forearm, wrist, hand, and fingers • 729.72 nontraumatic compartment syndrome of lower extremity: nontraumatic Reviewers compartment syndrome of hip, buttock, thigh, leg, foot, and toes Andrea Callanen, MPT • 958.90 compartment syndrome, unspecified Cinahl Information Systems, Glendale, CA • ICD codes are provided for the reader’s reference, not for billing purposes Abigail Grover Snook, PT, MS, MEd Cinahl Information Systems, Glendale, CA › ICD-10 codes Rehabilitation Operations Council • M62.2 ischemic infarction of muscle (non-traumatic compartment syndrome) Glendale Adventist Medical Center, • ICD codes are provided for the reader’s reference, not for billing purposes Glendale, CA › G-Codes Editor • Mobility G-code set Sharon Richman, MSPT –G8978, Mobility: walking & moving around functional limitation, current status, at Cinahl Information Systems, Glendale, CA therapy episode outset and at reporting intervals –G8979, Mobility: walking & moving around functional limitation; projected goal status, at therapy episode outset, at reporting intervals, and at discharge or to end January 8, 2016 reporting Published by Cinahl Information Systems, a division of EBSCO Information Services. Copyright©2016, Cinahl Information Systems. All rights reserved. No part of this may be reproduced or utilized in any form or by any means, electronic or mechanical, including photocopying, recording, or by any information storage and retrieval system, without permission in writing from the publisher. Cinahl Information Systems accepts no liability for advice or information given herein or errors/omissions in the text. It is merely intended as a general informational overview of the subject for the healthcare professional. Cinahl Information Systems, 1509 Wilson Terrace, Glendale, CA 91206 –G8980, Mobility: walking & moving around functional limitation, discharge status, at discharge from therapy or to end reporting • Changing & Maintaining Body Position G-code set –G8981, Changing & maintaining body position functional limitation, current status, at therapy episode outset and at reporting intervals –G8982, Changing & maintaining body position functional limitation, projected goal status, at therapy episode outset , at reporting intervals, and at discharge or to end reporting –G8983, Changing & maintaining body position functional limitation, discharge status, at discharge from therapy or to end reporting • Carrying, Moving & Handling Objects G-code set –G8984, Carrying, moving & handling objects functional limitation, current status, at therapy episode outset and at reporting intervals –G8985, Carrying, moving & handling objects functional limitation, projected goal status, at therapy episode outset, at reporting intervals, and at discharge or to end reporting –G8986, Carrying, moving & handling objects functional limitation, discharge status, at discharge from therapy or to end reporting • Self Care G-code set –G8987, Self care functional limitation, current status, at therapy episode outset and at reporting intervals –G8988, Self care functional limitation, projected goal status, at therapy episode outset, at reporting intervals, and at discharge or to end reporting –G8989, Self care functional limitation, discharge status, at discharge from therapy or to end reporting • Other PT/OT Primary G-code set –G8990, Other physical or occupational primary functional limitation, current status, at therapy episode outset and at reporting intervals –G8991, Other physical or occupational primary functional limitation, projected goal status, at therapy episode outset, at reporting intervals, and at discharge or to end reporting –G8992, Other physical or occupational primary functional limitation, discharge status, at discharge from therapy or to end reporting • Other PT/OT Subsequent G-code set –G8993, Other physical or occupational subsequent functional limitation, current status, at therapy episode outset and at reporting intervals –G8994, Other physical or occupational subsequent functional limitation, projected goal status, at therapy episode outset, at reporting intervals, and at discharge or to end reporting –G8995, Other physical or occupational subsequent functional limitation, discharge status, at discharge from therapy or to end reporting › . G-code Modifier Impairment Limitation Restriction CH 0 percent impaired, limited or restricted CI At least 1 percent but less than 20 percent impaired, limited or restricted CJ At least 20 percent but less than 40 percent impaired, limited or restricted CK At least 40 percent but less than 60 percent impaired, limited or restricted CL At least 60 percent but less than 80 percent impaired, limited or restricted CM At least 80 percent but less than 100 percent impaired, limited or restricted CN 100 percent impaired, limited or restricted Source: http://www.cms.gov . › Reimbursement: No specific issues or information regarding reimbursement have been identified › Presentation/signs and symptoms • Presentation – there is a variety of different presentations(39) –Gradual onset of symptoms(3,4) –Patients typically present with a long duration of preexisting symptoms(32) –Symptoms frequently occur during repetitious movements (e.g., jogging, biking) and will cease once activity stops(3,5,25) –Over time, a low-intensity ache may persist even when activity has ceased(3) –Typically occurs bilaterally and at the same point (duration/intensity) during exercise(6) –Most often affects the lower leg compartments(1) - Anterior compartment – most often affected - 95% of cases occur in anterior compartment(40) - Lateral compartment - Superficial posterior compartment - Deep posterior compartment –Also reported in the thigh, forearm, and foot(27) - Thigh contains 3 compartments: anterior, posterior, and medial - Forearm contains 2 compartments: anterior and posterior - Foot contains the medial, lateral, superficial, and deep compartments –Signs/symptoms - Pain during exercise at a predictable point that resolves with cessation of activity(7) - Pain gradually intensifies to be severe enough to prevent the individual from continuing activity such as running or walking(26) - Mild weakness or tenderness of muscle group of impaired compartment(7) - Soft tissue within the affected compartment feels tense/tight(27) - Physical examination is asymptomatic at rest(7) - Burning, cramping, and aching over involved compartment during and immediately following exertion(4,6) - Paresthesia (e.g., numbness, tingling) corresponding to the nerve within the affected compartment during aggravating activity(3,4,27) - Paresthesia occurs over the dorsal aspect of the foot if the anterior compartment is affected(27) - Paresthesia occurs on the plantar aspect of the foot if the deep compartment is affected(27) Causes, Pathogenesis, & Risk Factors › Causes • Result of increased or prolonged exertion (exercise) that leads to elevated pressure in the involved compartment(2,4) • Other potential causative factors(2,4) –Sudden escalation in training –Alteration in type of surface practiced/trained on –New footwear –Faulty biomechanics › Pathogenesis • The pathophysiology is not fully understood. Hypotheses include the following: –Muscle volume normally increases during exercise; however, in chronic compartment syndrome, the osseofascial compartment is unable to accommodate to the increase in size associated with muscle contraction and swelling. The increased volume increases intracompartmental pressure and reduces perfusion of the tissues within the fascial compartment and ischemia
Recommended publications
  • Gluteal Muscle Contracture: Diagnosis and Management Options
    SICOT J 2017, 3,1 Ó The Authors, published by EDP Sciences, 2017 DOI: 10.1051/sicotj/2016036 Available online at: www.sicot-j.org REVIEW ARTICLE OPEN ACCESS Gluteal muscle contracture: diagnosis and management options Saroj Rai1, Chunqing Meng1,*, Xiaohong Wang1, Nabin Chaudhary2, Shengyang Jin1, Shuhua Yang1, and Hong Wang1 1 Department of Orthopedics, Wuhan Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, #1277 Jiefang Avenue, 430022 Wuhan, P.R. China 2 Department of Radiology, Tongji Hospital, Tongji Medical College, Huazhong University of Science and Technology, #1095 Jiefang Avenue, 430030 Wuhan, P.R. China Received 14 April 2016, Accepted 29 October 2016, Published online 6 January 2017 Abstract – Gluteal muscle contracture (GMC), a debilitating disease, exists all over the globe but it is much more prevalent in China. Patients typically present with abduction and external rotation of the hip and are unable to bring both the knees together while squatting. Multiple etiologies have been postulated, the commonest being repeated intramuscular injection into the buttocks. The disease is diagnosed primarily by clinical features but radiological features are necessary for the exclusion of other pathological conditions. Non-operative treatment with physiotherapy can be tried before surgery is considered but it usually fails. Different surgical techniques have been described and claimed to have a better outcome of one over another but controversy still exists. Based on published literatures, the clinical outcome is exceptionally good in all established methods of surgery. However, endoscopic surgery is superior to conventional open surgery in terms of cosmetic outcome with fewer complications. Nevertheless, its use has been limited by lack of adequate knowledge, instrumentations, and some inherent limitations.
    [Show full text]
  • Cancer Cachexia Decreases Specific Force and Accelerates Fatigue in Limb Muscle
    Biochemical and Biophysical Research Communications 435 (2013) 488–492 Contents lists available at SciVerse ScienceDirect Biochemical and Biophysical Research Communications journal homepage: www.elsevier.com/locate/ybbrc Cancer cachexia decreases specific force and accelerates fatigue in limb muscle ⇑ B.M. Roberts a, G.S. Frye b, B. Ahn b, L.F. Ferreira b,1, A.R. Judge a,1, a 1225 Center Drive, HPNP Building Room 1142, Department of Physical Therapy, University of Florida, Gainesville, FL 32610, USA b 1864 Stadium Road, Department of Applied Physiology & Kinesiology, University of Florida, Gainesville, FL 32610, USA article info abstract Article history: Cancer cachexia is a complex metabolic syndrome that is characterized by the loss of skeletal muscle Received 30 April 2013 mass and weakness, which compromises physical function, reduces quality of life, and ultimately can Available online 11 May 2013 lead to mortality. Experimental models of cancer cachexia have recapitulated this skeletal muscle atro- phy and consequent decline in muscle force generating capacity. However, more recently, we provided Keywords: evidence that during severe cancer cachexia muscle weakness in the diaphragm muscle cannot be Muscle weakness entirely accounted for by the muscle atrophy. This indicates that muscle weakness is not just a conse- Muscle atrophy quence of muscle atrophy but that there is also significant contractile dysfunction. The current study C-26 aimed to determine whether contractile dysfunction is also present in limb muscles during severe Contractile dysfunction Colon-26 (C26) carcinoma cachexia by studying the glycolytic extensor digitorum longus (EDL) muscle and the oxidative soleus muscle, which has an activity pattern that more closely resembles the dia- phragm.
    [Show full text]
  • Case Report Forearm Compartment Syndrome Following Thrombolytic Therapy for Massive Pulmonary Embolism: a Case Report and Review of Literature
    Hindawi Publishing Corporation Case Reports in Orthopedics Volume 2011, Article ID 678525, 4 pages doi:10.1155/2011/678525 Case Report Forearm Compartment Syndrome following Thrombolytic Therapy for Massive Pulmonary Embolism: A Case Report and Review of Literature Ravi Badge and Mukesh Hemmady Department of Trauma and Orthopaedics Surgery, Wrightington, Wigan and Leigh NHS Trust, Wigan WN1 2NN, UK Correspondence should be addressed to Ravi Badge, [email protected] Received 2 November 2011; Accepted 6 December 2011 Academic Editor: M. K. Lyons Copyright © 2011 R. Badge and M. Hemmady. This is an open access article distributed under the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited. Use of thrombolytic therapy in pulmonary embolism is restricted in cases of massive embolism. It achieves faster lysis of the thrombus than the conventional heparin therapy thus reducing the morbidity and mortality associated with PE. The compartment syndrome is a well-documented, potentially lethal complication of thrombolytic therapy and known to occur in the limbs involved for vascular lines or venepunctures. The compartment syndrome in a conscious and well-oriented patient is mainly diagnosed on clinical ground with its classical signs and symptoms like disproportionate pain, tense swollen limb and pain on passive stretch. However these findings may not be appropriately assessed in an unconscious patient and therefore the clinicians should have high index of suspicion in a patient with an acutely swollen tense limb. In such scenarios a prompt orthopaedic opinion should be considered. In this report, we present a case of acute compartment syndrome of the right forearm in a 78 years old male patient following repeated attempts to secure an arterial line for initiating the thrombolytic therapy for the management of massive pulmonary embolism.
    [Show full text]
  • Spinal Muscular Atrophy
    FACT SHEET SPINAL MUSCULAR ATROPHY Spinal Muscular Atrophy (SMA) is a Motor Neuron Disease. It is caused by the mutation of the Survival of SYMPTOMS IN INFANTS • Muscle weakness. Motor Neuron (SMN) gene. It occurs due to the loss of • Muscle atrophy (wasting). motor neurons within the spinal cord and brain. It results • Poor muscle tone. in the progressive wasting away of muscles (atrophy) and • Areflexia (delayed reflexes). muscle weakness. SMA can affect people of all ages, races • Weak cry. or genders; however, the majority of cases occur in infancy • Difficulty sucking or swallowing. or childhood. There are four types of SMA. • Feeding difficulties. FORMS OF SMA • Weak cough. • Lack of developmental milestones (inability to lift head TYPE I (ACUTE INFANTILE) or sit up). • Also called Wernig-Hoffman Disease. • Limpness or a tendency to flop. • Most severe form of SMA. • Accumulations of secretions in the lungs or throat. • Usually diagnosed before six months of age. • Those affected cannot sit without support, lungs may SYMPTOMS IN ADULTS not fully develop, swallowing and breathing may be • Muscle weakness. difficult and there is weakness of the intercostal muscles • Muscle atrophy (wasting). (muscles between the ribs). • Weak tongue. • 95 per cent fatal by 18 • Stiffness. • Cramps. TYPE II (CHRONIC INFANTILE) • Fasciculation (twitching). • Usually diagnosed before the age of two, with the • Clumsiness. majority of cases diagnosed by 15 months. • Dyspnea (shortness of breath). • May be able to sit without assistance or even stand with support. DIAGNOSIS • Increased risk for complications from respiratory • A diagnosis can be made by an SMN gene test which infections.
    [Show full text]
  • Skeletal Muscle Damage in COVID-19: a Call for Action
    medicina Review Skeletal Muscle Damage in COVID-19: A Call for Action Amira Mohammed Ali 1,2,* and Hiroshi Kunugi 3,4 1 Department of Psychiatric Nursing and Mental Health, Faculty of Nursing, Alexandria University, Alexandria 21527, Egypt 2 Department of Behavioral Medicine, National Institute of Mental Health, National Center of Neurology and Psychiatry, Tokyo 187-8553, Japan 3 Department of Psychiatry, School of Medicine, Teikyo University, Tokyo 173-8605, Japan; [email protected] 4 Department of Mental Disorder Research, National Institute of Neuroscience, National Center of Neurology and Psychiatry, Tokyo 187-8551, Japan * Correspondence: [email protected]; Tel.: +81-042-346-1986 Abstract: Both laboratory investigations and body composition quantification measures (e.g., com- puted tomography, CT) portray muscle loss in symptomatic Coronavirus disease 2019 (COVID-19) patients. Muscle loss is associated with a poor prognosis of the disease. The exact mechanism of muscle damage in COVID-19 patients, as well as the long-term consequences of muscle injury in disease survivors, are unclear. The current review briefly summarizes the literature for mechanisms, assessment measures, and interventions relevant to skeletal muscle insult in COVID-19 patients. Muscle injury is likely to be attributed to the cytokine storm, disease severity, malnutrition, prolonged physical inactivity during intensive care unit (ICU) stays, mechanical ventilation, and myotoxic drugs (e.g., dexamethasone). It has been assessed by imaging and non-imaging techniques (e.g., CT and electromyography), physical performance tests (e.g., six-minute walk test), anthropometric measures (e.g., calf circumference), and biomarkers of muscle dystrophy (e.g., creatine kinase). Interventions Citation: Ali, A.M.; Kunugi, H.
    [Show full text]
  • Orthopedic Trauma Postoperative Care and Rehab
    Orthopedic Trauma Postoperative Care and Rehab Serge Charles Kaska, MD Name that Beach 100$ Still 100$ 75$ 50$ 1$ Omaha • June 6th 1941 • !st Infantry Division • 2000 KIA LIFE OR LIMB THREAT 1. Compartment Syndrome 2. Fat Emboli Syndrome 3. Pulmonary Embolism 4. Shock Compartment syndrome case A 16 year old male was retrieving a tire from his truck bed on the side of the highway in the pouring rain when a car careens off of the road and sandwiches the patients legs between the bumpers at freeway speed. Acute compartment syndrome Compartment syndrome DEFINED Definition: Elevated tissue pressure within a closed fascial space • Pathogenesis – Too much in-flow: results in edema or hemorrhage – Decreased outflow: results in venous obstruction caused by tight dressing and/or cast. • Reduces tissue perfusion • Results in cell death Compartment syndrome tissue survival • Muscle – 3-4 hours: reversible changes – 6 hours: variable damage – 8 hours: irreversible changes • Nerve – 2 hours: looses nerve conduction – 4 hours: neuropraxia – 8 hours: irreversible changes Physical exam 1. Pain 2. Pain 3. Pain Physical exam • Inspection – Swelling, skin is tight and shiny • Motion – Active motion will be refused or unable. Must see dorsiflexion • Palpation – Severe pain with palpation • Alarming pain with passive stretch Physical exam • Dorsiflexion Physical Exam • Palpation – Severe pain with palpation • Alarming pain with passive stretch Physical exam • Evaluations from nurses, therapists, and orthotech’s are CRITICAL • If you call a doctor and say that
    [Show full text]
  • Mechanical Ventilation Guide
    MAYO CLINIC MECHANICAL VENTILATION GUIDE RESP GOALS INITIAL MONITORING TARGETS FAILURE SETTINGS 6 P’s BASIC HEMODYNAMIC 1 BLOOD PRESSURE SBP > 90mmHg STABILITY PEAK INSPIRATORY 2 < 35cmH O PRESSURE (PIP) 2 BAROTRAUMA PLATEAU PRESSURE (P ) < 30cmH O PREVENTION PLAT 2 SAFETY SAFETY 3 AutoPEEP None VOLUTRAUMA Start Here TIDAL VOLUME (V ) ~ 6-8cc/kg IBW PREVENTION T Loss of AIRWAY Female ETT 7.0-7.5 AIRWAY / ETT / TRACH Patent Airway MAINTENANCE Male ETT 8.0-8.5 AIRWAY AIRWAY FiO2 21 - 100% PULSE OXIMETRY (SpO2) > 90% Hypoxia OXYGENATION 4 PEEP 5 [5-15] pO2 > 60mmHg 5’5” = 350cc [max 600] pCO2 40mmHg TIDAL 6’0” = 450cc [max 750] 5 VOLUME 6’5” = 500cc [max 850] ETCO2 45 Hypercapnia VENTILATION pH 7.4 GAS GAS EXCHANGE BPM (RR) 14 [10-30] GAS EXCHANGE MINUTE VENTILATION (VMIN) > 5L/min SYNCHRONY WORK OF BREATHING Decreased High Work ASSIST CONTROL MODE VOLUME or PRESSURE of Breathing PATIENT-VENTILATOR AC (V) / AC (P) 6 Comfortable Breaths (WOB) SUPPORT SYNCHRONY COMFORT COMFORT 2⁰ ASSESSMENT PATIENT CIRCUIT VENT Mental Status PIP RR, WOB Pulse, HR, Rhythm ETT/Trach Position Tidal Volume (V ) Trachea T Blood Pressure Secretions Minute Ventilation (V ) SpO MIN Skin Temp/Color 2 Connections Synchrony ETCO Cap Refill 2 Air-Trapping 1. Recognize Signs of Shock Work-up and Manage 2. Assess 6Ps If single problem Troubleshoot Cause 3. If Multiple Problems QUICK FIX Troubleshoot Cause(s) PROBLEMS ©2017 Mayo Clinic Foundation for Medical Education and Research CAUSES QUICK FIX MANAGEMENT Bleeding Hemostasis, Transfuse, Treat cause, Temperature control HYPOVOLEMIA Dehydration Fluid Resuscitation (End points = hypoxia, ↑StO2, ↓PVI) 3rd Spacing Treat cause, Beware of hypoxia (3rd spacing in lungs) Pneumothorax Needle D, Chest tube Abdominal Compartment Syndrome FLUID Treat Cause, Paralyze, Surgery (Open Abdomen) OBSTRUCTED BLOOD RETURN Air-Trapping (AutoPEEP) (if not hypoxic) Pop off vent & SEE SEPARATE CHART PEEP Reduce PEEP Cardiac Tamponade Pericardiocentesis, Drain.
    [Show full text]
  • Acute Compartment Syndrome Complicating Deep Venous Thrombosis
    SMGr up Case Report SM Journal of Acute Compartment Syndrome Case Reports Complicating Deep Venous Thrombosis Senthil Dhayalan1*, David Jardine1*, Tony Goh2 and Nicholas Lash3 1Senior registrar and General Physician, Dept of General Medicine, NZ 2Consultant Radiologist, Dept of Radiology, NZ 3Orthopaediac Surgeon, Dept of Orthopaedics, NZ A 40-yr old, heavily-built man initially presented to his general practitioner 2 days before Article Information admission with recent onset of pain and swelling in his left calf. A duplex ultrasound scan Received date: Nov 21, 2018 demonstrated a popliteal and lower leg deep vein thrombosis [DVT], extending 15 cm above the Accepted date: Nov 26, 2018 knee into the femoral vein. He was started appropriately on enoxaparin 130 mg bd. Despite the Published date: Nov 27, 2018 treatment, the pain got worse, particularly when standing, and he was admitted for symptom control. Five weeks earlier he had undergone anterior cruciate ligament repair of the left knee, *Corresponding author [without heparin prophylaxis] and made a satisfactory recovery (Figure 1). Jardine D, General Medicine On examination he was mildly distressed with a low-normal blood pressure [110/70 mmHg] Department, Christchurch hospital, and a persistent low-grade fever [temperature ranged from 37.0-37.8 oC]. The left leg was moderately Private Bag 4710, Christchurch 8140, diffusely swollen, slightly warm and darker in colour [figure]. The pain was localised to the posterior New Zealand, compartments and was exacerbated by dorsi-flexing the ankle and squeezing the gastrocnemius. Email: [email protected] There were no signs of joint effusion, thrombophlebitis, lymphadenopathy or cellulitis.
    [Show full text]
  • Bilateral Atraumatic Compartment Syndrome of the Legs Leading to Rhabdomyolysis and Acute Renal Failure Following Prolonged Kneeling in a Heroin Addict
    PAJT 10.5005/jp-journals-10030-1075 CASE REPORTBilateral Atraumatic Compartment Syndrome of the Legs Leading to Rhabdomyolysis and Acute Renal Failure Bilateral Atraumatic Compartment Syndrome of the Legs Leading to Rhabdomyolysis and Acute Renal Failure Following Prolonged Kneeling in a Heroin Addict. A Case Report and Review of Relevant Literature Saptarshi Biswas, Ramya S Rao, April Duckworth, Ravi Kothuru, Lucio Flores, Sunil Abrol ABSTRACT cerrado, que interfiere con la circulación de los componentes mioneurales del compartimento. Síndrome compartimental Introduction: Compartment syndrome is defined as a symptom bilateral de las piernas es una presentación raro que requiere complex caused by increased pressure of tissue fluid in a closed una intervención quirúrgica urgente. En un reporte reciente osseofascial compartment which interferes with circulation (Khan et al 2012), ha habido reportados solo 8 casos de to the myoneural components of the compartment. Bilateral síndrome compartimental bilateral. compartment syndrome of the legs is a rare presentation Se sabe que el abuso de heroína puede causar el síndrome requiring emergent surgical intervention. In a recent case report compartimental y rabdomiólisis traumática y atraumática. El (Khan et al 2012) there have been only eight reported cases hipotiroidismo también puede presentarse independiente con cited with bilateral compartment syndrome. rabdomiólisis. Heroin abuse is known to cause compartment syndrome, traumatic and atraumatic rhabdomyolysis. Hypothyroidism can Presentación del caso: Presentamos un caso de una mujer also independently present with rhabdomyolysis. de 22 años quien presentó con tumefacción bilateral de las piernas asociado con la perdida de la sensación, después Case presentation: We present a case of a 22 years old female de pasar dos días arrodillado contra una pared después de who presented with bilateral swelling of the legs with associated usar heroína intravenosa.
    [Show full text]
  • Assessment, Management and Decision Making in the Treatment of Polytrauma Patients with Head Injuries
    Compartment Syndrome Andrew H. Schmidt, M.D. Professor, Dept. of Orthopedic Surgery, Univ. of Minnesota Chief, Department of Orthopaedic Surgery Hennepin County Medical Center April 2016 Disclosure Information Andrew H. Schmidt, M.D. Conflicts of Commitment/ Effort Board of Directors: OTA Critical Issues Committee: AOA Editorial Board: J Knee Surgery, J Orthopaedic Trauma Medical Director, Director Clinical Research: Hennepin County Med Ctr. Disclosure of Financial Relationships Royalties: Thieme, Inc.; Smith & Nephew, Inc. Consultant: Medtronic, Inc.; DGIMed; Acumed; St. Jude Medical (spouse) Stock: Conventus Orthopaedics; Twin Star Medical; Twin Star ECS; Epien; International Spine & Orthopedic Institute, Epix Disclosure of Off-Label and/or investigative Uses I will not discuss off label use and/or investigational use in my presentation. Objectives • Review Pathophysiology of Acute Compartment Syndrome • Review Current Diagnosis and Treatment – Risk Factors – Clinical Findings – Discuss role and technique of compartment pressure monitoring. Pathophysiology of Compartment Syndrome Pressure Inflexible Fascia Injured Muscle Vascular Consequences of Elevated Intracompartment Pressure: A-V Gradient Theory Pa (High) Pv (Low) artery arteriole capillary venule vein Local Blood Pa - Pv Flow = R Matsen, 1980 Increased interstitial pressure Pa (High) Tissue ischemia artery arteriole capillary venule vein Lysis of cell walls Release of osmotically active cellular contents into interstitial fluid Increased interstitial pressure More cellular
    [Show full text]
  • Steroid-Induced Myopathy and Its Significance to Respiratory Disease: a Known Disease Rediscovered
    Eur Respir J REVIEW 1992, 5, 997-1003 Steroid-induced myopathy and its significance to respiratory disease: a known disease rediscovered P.N.R. Dekhuijzen, M. Decramer Steroid-induced myopathy and its significance to respiratory disease: a known dis­ Respiratory Muscle Research Unit, ease rediscovered. P.N.R. Dekhuijzen, M. Decramer. Laboratory for Pneumology and Respi­ ABSTRACT: Skeletal muscle myopathy is a well-known side-effect of systemi­ ratory Division, University Hospital, cally administered corticosteroids. In recent years renewed attention is being Katholieke Universiteit Leuven, B-3000 Leuvcn, Belgium. paid to the involvement of the respiratory muscles and its consequent signifi­ cance in pulmonary patients. Two different clinical patterns of steroid-induced Correspondence: M. Decramer, Respira­ muscular changes are known. In acute myopathy and atrophy after short term tory Division, University Hospital, treatment with high doses of steroids, generalized muscle atrophy and Weligerveld I, 3212 Pellenberg, Bel­ rhabdomyolysis occur, including the respiratory muscles. Chronic steroid my­ gium opathy, occurring after prolonged treatment with moderate doses, is character­ ized by the gradual onset of proximal limb muscle weakness and may be Keywords: Atrophy accompanied by reduced respiratory muscle force. corticosteroids Animal studies demonstrated diaphragmatic myopathy and atrophy similar myopathy respiratory muscles to the alterations in peripheral skeletal muscles. Fluorinated steroids induced selective type lib (fast-twitch
    [Show full text]
  • Western Trauma Association Critical Decisions in Trauma: Penetrating Chest Trauma
    WTA 2014 ALGORITHM Western Trauma Association Critical Decisions in Trauma: Penetrating chest trauma Riyad Karmy-Jones, MD, Nicholas Namias, MD, Raul Coimbra, MD, Ernest E. Moore, MD, 09/30/2020 on BhDMf5ePHKav1zEoum1tQfN4a+kJLhEZgbsIHo4XMi0hCywCX1AWnYQp/IlQrHD3Ypodx1mzGi19a2VIGqBjfv9YfiJtaGCC1/kUAcqLCxGtGta0WPrKjA== by http://journals.lww.com/jtrauma from Downloaded Martin Schreiber, MD, Robert McIntyre, Jr., MD, Martin Croce, MD, David H. Livingston, MD, Jason L. Sperry, MD, Ajai K. Malhotra, MD, and Walter L. Biffl, MD, Portland, Oregon Downloaded from http://journals.lww.com/jtrauma his is a recommended algorithm of the Western Trauma Historical Perspective TAssociation for the acute management of penetrating chest The precise incidence of penetrating chest injury, varies injury. Because of the paucity of recent prospective randomized depending on the urban environment and the nature of the trials on the evaluation and management of penetrating chest review. Overall, penetrating chest injuries account for 1% to injury, the current algorithms and recommendations are based 13% of trauma admissions, and acute exploration is required in by on available published cohort, observational and retrospective BhDMf5ePHKav1zEoum1tQfN4a+kJLhEZgbsIHo4XMi0hCywCX1AWnYQp/IlQrHD3Ypodx1mzGi19a2VIGqBjfv9YfiJtaGCC1/kUAcqLCxGtGta0WPrKjA== 5% to 15% of cases; exploration is required in 15% to 30% of studies, and the expert opinion of the Western Trauma Asso- patients who are unstable or in whom active hemorrhage is ciation members. The two algorithms should be reviewed in the suspected. Among patients managed by tube thoracostomy following sequence: Figure 1 for the management and damage- alone, complications including retained hemothorax, empy- control strategies in the unstable patient and Figure 2 for the ema, persistent air leak, and/or occult diaphragmatic injuries management and definitive repair strategies in the stable patient.
    [Show full text]