Eruption of Teeth Assistant Professor Aseel Haidar
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Glossary for Narrative Writing
Periodontal Assessment and Treatment Planning Gingival description Color: o pink o erythematous o cyanotic o racial pigmentation o metallic pigmentation o uniformity Contour: o recession o clefts o enlarged papillae o cratered papillae o blunted papillae o highly rolled o bulbous o knife-edged o scalloped o stippled Consistency: o firm o edematous o hyperplastic o fibrotic Band of gingiva: o amount o quality o location o treatability Bleeding tendency: o sulcus base, lining o gingival margins Suppuration Sinus tract formation Pocket depths Pseudopockets Frena Pain Other pathology Dental Description Defective restorations: o overhangs o open contacts o poor contours Fractured cusps 1 ww.links2success.biz [email protected] 914-303-6464 Caries Deposits: o Type . plaque . calculus . stain . matera alba o Location . supragingival . subgingival o Severity . mild . moderate . severe Wear facets Percussion sensitivity Tooth vitality Attrition, erosion, abrasion Occlusal plane level Occlusion findings Furcations Mobility Fremitus Radiographic findings Film dates Crown:root ratio Amount of bone loss o horizontal; vertical o localized; generalized Root length and shape Overhangs Bulbous crowns Fenestrations Dehiscences Tooth resorption Retained root tips Impacted teeth Root proximities Tilted teeth Radiolucencies/opacities Etiologic factors Local: o plaque o calculus o overhangs 2 ww.links2success.biz [email protected] 914-303-6464 o orthodontic apparatus o open margins o open contacts o improper -
Non-Syndromic Occurrence of True Generalized Microdontia with Mandibular Mesiodens - a Rare Case Seema D Bargale* and Shital DP Kiran
Bargale and Kiran Head & Face Medicine 2011, 7:19 http://www.head-face-med.com/content/7/1/19 HEAD & FACE MEDICINE CASEREPORT Open Access Non-syndromic occurrence of true generalized microdontia with mandibular mesiodens - a rare case Seema D Bargale* and Shital DP Kiran Abstract Abnormalities in size of teeth and number of teeth are occasionally recorded in clinical cases. True generalized microdontia is rare case in which all the teeth are smaller than normal. Mesiodens is commonly located in maxilary central incisor region and uncommon in the mandible. In the present case a 12 year-old boy was healthy; normal in appearance and the medical history was noncontributory. The patient was examined and found to have permanent teeth that were smaller than those of the average adult teeth. The true generalized microdontia was accompanied by mandibular mesiodens. This is a unique case report of non-syndromic association of mandibular hyperdontia with true generalized microdontia. Keywords: Generalised microdontia, Hyperdontia, Permanent dentition, Mandibular supernumerary tooth Introduction [Ullrich-Turner syndrome], Chromosome 13[trisomy 13], Microdontia is a rare phenomenon. The term microdontia Rothmund-Thomson syndrome, Hallermann-Streiff, Oro- (microdentism, microdontism) is defined as the condition faciodigital syndrome (type 3), Oculo-mandibulo-facial of having abnormally small teeth [1]. According to Boyle, syndrome, Tricho-Rhino-Phalangeal, type1 Branchio- “in general microdontia, the teeth are small, the crowns oculo-facial syndrome. short, and normal contact areas between the teeth are fre- Supernumerary teeth are defined as any supplementary quently missing” [2] Shafer, Hine, and Levy [3] divided tooth or tooth substance in addition to usual configuration microdontia into three types: (1) Microdontia involving of twenty deciduous and thirty two permanent teeth [7]. -
Ankylosed Primary Molars, Andlaw (1974) Described Surface Defects from Bicuspids Preceded by Non-Ankylosed 11 Molars
PEDIATRICDENTISTRY/Copyright (~) 1980 The AmericanAcademy of Pedodontics/Vol. 2, No, 1 Ankylosedprimary mola.rs: Results and treatment recommendat,onsfrom an eight-year longitudinal study Louise Brearley Messer,B.D.Sc., L.D.S., M.D.Sc. Jay T. Cline, D.D.S., M.A. Abstract continues concomitantly with vertical alveolar bone growth,~,3 and the tooth is immobile to manual rock- A total of 263 ankyloscd primarymolars in 107 ing.4,5 children aged three to 12 years was studied for four years. Forty-six children remainedin the study for eight years. The etiology of the condition remains unknown. Extrinsic causative factors implicated are local me- Observationof affected dentitions showedthat the con- ~ ~ dition waslikely to recur. Threeclinical pa~ternsfor the chanical trauma, disturbed local metabolism, local- condition are described. Typically, maxillary molars be- ized infection, 6 chemical or thermal irritation 7 and came ankylosed earlier and demonstrated more severe tooth reimplantation, s Intrinsic factors cited include a in[raocclusion than mandibularmolars. Mandibularfirst genetic or congenital gap in the periodontal liga- molars usually remainedslightly or moderatelyin#a- ment. Since both erupting and exfoliating teeth show occluded; mandibularsecond molars and maxillary first alternating periods of resorption and deposition of and second molars showedprogressively severe infra- bone and cementum,° aberrant deposition of these tis- occlusion. Followingeither extraction or ex~oliation of the suesI° may produce an area of ankylosing tissue.Z, affected molars, the succedaneousbicuspids did not differ In a summaryof studies reporting the prevalence of in either coronal morphologyor in distribution of enamel ankylosed primary molars, Andlaw (1974) described surface defects from bicuspids preceded by non-ankylosed 11 molars. -
Establishment of a Dental Effects of Hypophosphatasia Registry Thesis
Establishment of a Dental Effects of Hypophosphatasia Registry Thesis Presented in Partial Fulfillment of the Requirements for the Degree Master of Science in the Graduate School of The Ohio State University By Jennifer Laura Winslow, DMD Graduate Program in Dentistry The Ohio State University 2018 Thesis Committee Ann Griffen, DDS, MS, Advisor Sasigarn Bowden, MD Brian Foster, PhD Copyrighted by Jennifer Laura Winslow, D.M.D. 2018 Abstract Purpose: Hypophosphatasia (HPP) is a metabolic disease that affects development of mineralized tissues including the dentition. Early loss of primary teeth is a nearly universal finding, and although problems in the permanent dentition have been reported, findings have not been described in detail. In addition, enzyme replacement therapy is now available, but very little is known about its effects on the dentition. HPP is rare and few dental providers see many cases, so a registry is needed to collect an adequate sample to represent the range of manifestations and the dental effects of enzyme replacement therapy. Devising a way to recruit patients nationally while still meeting the IRB requirements for human subjects research presented multiple challenges. Methods: A way to recruit patients nationally while still meeting the local IRB requirements for human subjects research was devised in collaboration with our Office of Human Research. The solution included pathways for obtaining consent and transferring protected information, and required that the clinician providing the clinical data refer the patient to the study and interact with study personnel only after the patient has given permission. Data forms and a custom database application were developed. Results: The registry is established and has been successfully piloted with 2 participants, and we are now initiating wider recruitment. -
The Cat Mandible (II): Manipulation of the Jaw, with a New Prosthesis Proposal, to Avoid Iatrogenic Complications
animals Review The Cat Mandible (II): Manipulation of the Jaw, with a New Prosthesis Proposal, to Avoid Iatrogenic Complications Matilde Lombardero 1,*,† , Mario López-Lombardero 2,†, Diana Alonso-Peñarando 3,4 and María del Mar Yllera 1 1 Unit of Veterinary Anatomy and Embryology, Department of Anatomy, Animal Production and Clinical Veterinary Sciences, Faculty of Veterinary Sciences, Campus of Lugo—University of Santiago de Compostela, 27002 Lugo, Spain; [email protected] 2 Engineering Polytechnic School of Gijón, University of Oviedo, 33203 Gijón, Spain; [email protected] 3 Department of Animal Pathology, Faculty of Veterinary Sciences, Campus of Lugo—University of Santiago de Compostela, 27002 Lugo, Spain; [email protected] 4 Veterinary Clinic Villaluenga, calle Centro n◦ 2, Villaluenga de la Sagra, 45520 Toledo, Spain * Correspondence: [email protected]; Tel.: +34-982-822-333 † Both authors contributed equally to this manuscript. Simple Summary: The small size of the feline mandible makes its manipulation difficult when fixing dislocations of the temporomandibular joint or mandibular fractures. In both cases, non-invasive techniques should be considered first. When not possible, fracture repair with internal fixation using bone plates would be the best option. Simple jaw fractures should be repaired first, and caudal to rostral. In addition, a ventral approach makes the bone fragments exposure and its manipulation easier. However, the cat mandible has little space to safely place the bone plate screws without damaging the tooth roots and/or the mandibular blood and nervous supply. As a consequence, we propose a conceptual model of a mandibular prosthesis that would provide biomechanical Citation: Lombardero, M.; stabilization, avoiding any unintended (iatrogenic) damage to those structures. -
Feline Tooth Resorption Feline Odontoclastic Resorptive Lesions (FORL)
Feline Tooth Resorption Feline Odontoclastic Resorptive Lesions (FORL) What is tooth resorption? Tooth resorption is a destructive process that eats away at teeth and is quite common in cats. Up to 50% of cats over the age of 8 will have resorptive lesions. Of those 50% with lesions, 50% of them will have more than one. This process can be very painful, and due to the nature of the cat, many will not show obvious signs of pain. These lesions will often require immediate treatment. Feline tooth resorption does progress and will require treatment to avoid pain and loss of function. This process is not necessarily preventable, but studies do show that cats who do not receive oral hygiene care are at an increased risk of development of resorptive lesions. Feline patients diagnosed with Feline Immunodeficiency Virus and Feline Leukemia Virus are also more likely to develop lesions. Despite the health status of your cat, it is important to know tooth resorption is a common and treatable disease. What causes tooth resorption? Tooth resorption is an idiopathic disease of the teeth. This means that the cause is unknown. We do know odontoclasts, the cat’s own cells, begin to destroy the structure of the tooth. Sometimes resorption will be associated with a tooth root abscess, although this is uncommon. What are the clinical signs of this? Cats with tooth resorption will often have inflammation of the gum tissue surrounding the affected tooth. Your veterinarian will comment on the teeth and gums during the oral part of the physical examination. Even though an oral exam is done, many of these lesions are below the gum line and require dental radiography to fully diagnose. -
Dental Anomalies: Foundational Articles and Consensus Recommendations, 2021
Dental Anomalies: Foundational Articles and Consensus Recommendations, 2021 Adekoya-Sofowora CA. Natal and neonatal teeth: a review. Niger Postgrad Med J 2008;15:38-41 Al-Ani AH, Antoun JS, Thomson WM, Merriman TR, Farella M. Hypodontia: An Update on Its Etiology, Classification, and Clinical Management. Biomed Res Int. 2017:9378325. doi.org/10.1155/2017/9378325. Anthonappa RP, King NM, Rabie AB. Aetiology of supernumerary teeth: A literature review. Eur Arch Paediatr Dent. 2013;14:279-88. Dashash, M. Yeung CA, Jamous I, Blinkhorn A. Interventions for the restorative care of amelogenesis imperfecta in children and adolescents. Cochrane Database Syst Rev 2013;6:CD007157. Gallacher A, Ali R, Bhakta S. Dens invaginatus: diagnosis and management strategies. Br Dent J 2016;221:383-7. Gill DS, Barker CS. The multidisciplinary management of hypodontia: a team approach. Br Dent J 2015;218:143-9. Khalaf K, Miskelly J, Voge E, Macfarlane TV. Prevalence of hypodontia and associated factors: a systematic review and meta-analysis. J Orthod. 2014; 41:299-316. Lammi L. Arte S, Somer M, Javinen H, et al. Mutations in AXIN2 cause familial tooth agenesis and predispose to colorectal cancer. Am. J. Hum. Genet. 2004, 74:1043–1050. Marvin ML, Mazzoni S, Herron CM, Edwards S, et al. AXIN2-associated autosomal dominant ectodermal dysplasia and neoplastic syndrome. Am J Med Genet A. 2011,155 898–902. Seow WK. Developmental defects of enamel and dentine: Challenges for basic science research and clinical management. Aust Dent J 2014;59:143-54. Shields ED, Bixler D, El-Kafrawy AM. A proposed classification for heritable human dentine defects with a description of a new entity. -
Feline Alveolar Osteitis Treatment Planning: Implant Protocol with Osseodensification and Early Crown Placement Rocco E
Feline Alveolar Osteitis Treatment Planning: Implant Protocol with Osseodensification and Early Crown Placement Rocco E. Mele DVM1, Gregori M. Kurtzman, DDS, MAGD, DICOI,DIDIA2 1 Eastpoint Pet Clinic, Tucson, A, USA 2 Silver Spring, MD, USA Abstract: Feline dental implants are becoming a predictable and viable treatment option for the replacement of lost canines due to maxillary Alveolar Osteitis (AO) a painful condition, commonly experienced by a growing number of cats. Surgical extraction and debridement remains the treatment of choice for this complex inflammatory process. However, future complications can be a common sequela of maxillary canine loss. This case will demonstrate the successful surgical extraction of a maxillary canine with implant placement following the osseodensification protocol and utilizing the sockets osteitis buttressing bone formation to promote a positive result with final crown restoration 13 weeks following implant placement. Introduction: Alveolar Osteitis (AO) is a chronic inflammatory process more often diagnosed in maxillary canine sockets of the feline patient. Clinical presentation may include oral pain, bleeding, periodontitis, tooth resorption (ORL), and alveolar buccal bone changes.1-5 Clinical Features: A presumptive diagnosis of (AO) is made on the awake patient, documenting clinical features such as; gingivitis with soft tissue swelling, gingival mucosal erythema, buccal bone expansion, and coronal extrusion. (Figure 1) Radiographic Features: Radiographic changes are identified under general anesthesia. These bony changes and pathology may include; deep palatal probing (Figure 2 red), alveolar bone expansion (Figure 2 green), buttressing condensing bone (Figure 2 blue) and a mottled osseous appearance mimicking rough, large trabeculae (Figure 2 yellow) Osseodensification (OD): OD is a novel biomechanical bone preparation technique for dental implant placement to improve bone quality by increasing its density utilizing Densah burs. -
An Insight Into Internal Resorption
Hindawi Publishing Corporation ISRN Dentistry Volume 2014, Article ID 759326, 7 pages http://dx.doi.org/10.1155/2014/759326 Review Article An Insight into Internal Resorption Priya Thomas, Rekha Krishna Pillai, Bindhu Pushparajan Ramakrishnan, and Jayanthi Palani Department of Oral and Maxillofacial Pathology, Annoor Dental College & Hospital, Muvattupuzha, Ernakulam District, Kerala 686673, India Correspondence should be addressed to Priya Thomas; [email protected] Received 20 January 2014; Accepted 27 March 2014; Published 12 May 2014 Academic Editors: S.-C. Choi and G. Mount Copyright © 2014 Priya Thomas et al. This is an open access article distributed under the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited. Internal resorption, a rare phenomenon, has been a quandary from the standpoints of both its diagnosis and treatment. It is usually asymptomatic and discovered by chance on routine radiographic examinations or by a classic clinical sign, “pink spot” in the crown. This paper emphasizes the etiology and pathophysiologic mechanisms involved in internal root resorption. Prognosis is good for smaller lesions; however, for those with extensive resorption associated with perforation the tooth structure is greatly weakened and the prognosis remains poor. 1. Introduction teeth. Unlike the deciduous teeth, the permanent teeth rarely undergo resorption unless stimulated by a pathological Tooth resorption presents itself either as a physiological process. Pathologic resorption occurs following traumatic or a pathological process occurring internally (pulpally injuries, orthodontic tooth movement, or chronic infections derived) or externally (periodontally derived). According to of the pulp or periodontal structures [1]. -
Pharmacology of Local Anesthesia
By Mohammad Hussein Zaki Lecturer Oral & Maxillofacial Surgery Faculty of Dentistry – Minia University . Impacted tooth. Failure of the tooth to fully erupt into the oral cavity within its expected developmental time period and can no longer reasonably be expected to do so. A tooth that can not, or will not, erupt into its normal functioning positions. Impacted tooth. An impacted tooth is one that is erupted, partially erupted or unerupted and will not eventually assume a normal arch relationship with the other teeth and tissues. Unerupted tooth. Includes impacted teeth and teeth that are in the process of erupting. Impacted teeth seen in the following order of frequency: 1. Mandibular third molars. 2. Maxillary third molars. 3. Maxillary canine. 4. Mandibular premolar. 5. Maxillary premolar. 6. Mandibular canine. 7. Maxillary central incisors. 8. Maxillary lateral incisors. Systemic factors: • Syndromes. Cleidocranial dysplasia. • Endocrine deficiencies. Hypothyroidism. • Irradiation. Local factors: • Obstruction for eruption. Irregularity in position of an adjacent tooth. Density of the overlying soft tissue or bone. Prolonged deciduous tooth retention. Supernumerary teeth. • Tooth ankylosis. • Dilaceration of roots. • Malposed tooth germs. Local factors: • Pathological lesions. • Cleft lip and palate. • Arch-length deficiency. Phylogenic theory. Mendelian theory. Clinical. Radiographic. • Periapical. Radiographic. • Periapical. Radiographic. • Occlusal. Radiographic. • Panorama. Radiographic. • Panorama. Radiographic. • CT. Radiographic. • CBCT. Impacted teeth other than third molars. Exposure. • A procedure that allows natural eruption of impacted teeth. • It should be done as soon as it is determined that the tooth is not going to erupt spontaneously. • Spontaneous eruption versus orthodontic assistance. Impacted teeth other than third molars. Exposure. Impacted teeth other than third molars. -
Eruption Abnormalities in Permanent Molars: Differential Diagnosis and Radiographic Exploration
DOI: 10.1051/odfen/2014054 J Dentofacial Anom Orthod 2015;18:403 © The authors Eruption abnormalities in permanent molars: differential diagnosis and radiographic exploration J. Cohen-Lévy1, N. Cohen2 1 Dental surgeon, DFO specialist 2 Dental surgeon ABSTRACT Abnormalities of permanent molar eruption are relatively rare, and particularly difficult to deal with,. Diagnosis is founded mainly on radiographs, the systematic analysis of which is detailed here. Necessary terms such as non-eruption, impaction, embedding, primary failure of eruption and ankylosis are defined and situated in their clinical context, illustrated by typical cases. KEY WORDS Molars, impaction, primary failure of eruption (PFE), dilaceration, ankylosis INTRODUCTION Dental eruption is a complex developmen- at 0.08% for second maxillary molars and tal process during which the dental germ 0.01% for first mandibular molars. More re- moves in a coordinated fashion through cently, considerably higher prevalence rates time and space as it continues the edifica- were reported in retrospective studies based tion of the root; its 3-dimensional pathway on orthodontic consultation records: 2.3% crosses the alveolar bone up to the oral for second molar eruption abnormalities as epithelium to reach its final position in the a whole, comprising 1.5% ectopic eruption, occlusion plane. This local process is regu- 0.2% impaction and 0.6% primary failure of lated by genes expressing in the dental fol- eruption (PFE) (Bondemark and Tsiopa4), and licle, at critical periods following a precise up to 1.36% permanent second molar iim- chronology, bilaterally coordinated with fa- paction according to Cassetta et al.6. cial growth. -
Teliangectaticum Granuloma Associated to a Natal Tooth Granuloma Telangiectásico Asociado a Diente Natal
www.medigraphic.org.mx Revista Odontológica Mexicana Facultad de Odontología Vol. 20, No. 1 January-March 2016 pp 29-32 CASE REPORT Teliangectaticum granuloma associated to a natal tooth Granuloma telangiectásico asociado a diente natal Katherine Vásquez Sanjuán,* Ary López Álvarez,§ Jonathan Harris RicardoII ABSTRACT RESUMEN Oral teliangectaticum granuloma, also known as pyogenic granuloma Granuloma telangiectásico bucal, también conocido como granulo- is a proliferation of exuberant granulation tissue caused by chronic ma piógeno, es una proliferación de tejido de granulación exuberan- inflammation or local irritation. It is a rare lesion in newborn; it te, a una infl amación crónica o una irritación local, poco frecuente appears as an isolated tumor lesion, mostly located in the anterior del recién nacido, que se presenta como una lesión tumoral única, zone of the alveolar crest. This lesion bleeds spontaneously and localizada con mayor frecuencia en la zona anterior de la cresta shows predilection for the female gender. Its etiology is related to alveolar, de sangrado espontáneo, tiene predilección por el género trauma factors, local irritation and hormonal changes. Due to its size femenino, la etiología se relaciona con factores traumáticos, irrita- it can affect the newborn’s feeding. Surgical removal is the choice ción local y cambios hormonales, por el tamaño puede afectar la treatment for this type of lesions. The present study presents the alimentación del neonato, la remoción quirúrgica es el tratamiento case of a newborn with diagnosis of telangiectaticum granuloma de elección. Se presenta caso clínico de recién nacido con diagnós- in the mandibular ridge associated to a natal tooth.