JUGULAR VENOUS PRESSURE Maddury Jyotsna
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Heart Rate Information Sheet Finding Your Pulse Taking Your Pulse
Heart rate In this activity you will measure your heart rate and investigate the effect that other things have on your heart rate. Information sheet You can measure your heart (pulse) rate anywhere on your body where a major artery is close to the surface of your skin. The easiest places are: • on the front of your forearm • just above your wrist on your thumb side • on the side of your neck about half way between your chin and your ear. Finding your pulse Try to locate your pulse in one of these places, using the tips of your index and middle fingers. You should feel a gentle, regular beat. This is your heart rate. Do not use your thumb, as your thumb has a pulse of its own. Taking your pulse When you find your pulse, use a stopwatch or a watch with a second hand to count how many beats there are in a full minute (60 seconds). In most situations, taking your pulse rate over one minute will give a reasonably accurate result, but if you want to take your pulse after exercise, you should do so over a much shorter time interval. After exercise your pulse rate will be changing rapidly. To get a reasonably accurate result, start to measure the rate immediately after the exercise and count the number of beats in 10 seconds. Multiply this number by six to find your heart rate. For an adult, a normal resting heart rate is between 60–100 beats a minute. The fitter you are, the lower your resting heart beat will be. -
Some Observations on the Atrial Sound*
15 Oktober 1960 S.A. TYDSKRIF VIR GENEESKUNDE 887 SOME OBSERVATIONS ON THE ATRIAL SOUND* JOHN R BARLOW, M.R, RCH., M.R.C.P., Department of Medicine, University of The WiTwaTersralld. Johallllesburg The atrial sound, also known as the fourth heart sound Emotional factors probably play a large part in this; the or. presystolic gallop, has been defined' as an audible P-G interval may quickly horten again on the introduction ibration occurring more than 0·07 second after the of an intravenous needle or imilar procedure. beginning of the P wave of the simultaneous electrocardio 3. EffeCT of respiration. Whereas re piration produce gram (ECG) but preceding the onset of the QRS complex. no effect on the P-G interval in patients with hyperten ion It has been shown' that, unrelated to any alteration in the or ischaemia. the atrial sound of cor pulmonale both in P-R interval, the position of the atrial sound varies in creases in intensity and occurs earlier in the cardiac cycle relation to the first heart sound and to the simultaneous during inspiration as compared with expiration (Fig. 4). ECG. 4. EffeCT of various procedures. Jt has already been Jt is the purpose of this paper to demonstrate this mentioned that the intravenous administration of hypo variation in the timing of the atrial sound and to discuss tensive drugs will increase the P-G interval in hypertensive some of the clinical implications resulting from this varia patients. The inhalation of amyl nitrite has a similar effect bility. in hypertension (Fig. 5), though a change in the P-G METHODS AND OBSERVATIO S interval is seldom seen in cases with ischaemic heart disease following inhalation of this drug. -
Chronotropic and Dromotropic Responses to Localized Glutamate Microinjections in the Rat $ Nucleus Ambiguus
brain research 1542 (2014) 93–103 Available online at www.sciencedirect.com www.elsevier.com/locate/brainres Research Report Chronotropic and dromotropic responses to localized glutamate microinjections in the rat $ nucleus ambiguus Karla N. Sampaio1,He´lder Mauad2, K. Michael Spyer, Timothy W. Fordn Division of Biosciences, Faculty of Life Sciences, University College London, Gower Street, London WC1E 6BT, UK article info abstract Article history: The cardioinhibitory effects of cardiac vagal motoneurons (CVMs) are mediated by Accepted 18 October 2013 activation of postganglionic neurons in the epicardial ganglia which have been shown to Available online 24 October 2013 exert functionally selective effects on heart rate and atrioventricular conduction in the rat. Here we investigate whether CVMs producing these responses may occupy different Keywords: rostrocaudal positions within the nucleus ambiguus. Excitation of CVMs was attempted Autonomic nervous system by microinjections of glutamate into the nucleus ambiguus of an arterially perfused Vagus nerve preparation in a grid extending over 2 mm in the rostrocaudal plane using the obex as a Nucleus ambiguus reference point. Microinjections were paired, one made during pacing to measure changes Heart rate in atrioventricular conduction (P-R interval) independent of changes in heart rate and the Glutamate other looking for changes in heart period (P-P interval) un-paced. Although evidence of a differential distribution was found in 7 cases, in the majority (13/20), sites producing maximal effects on both variables coincided. Maximal changes in atrioventricular conduc- tion resulted from more rostral sites in 6 cases and from a more caudal site in only one. Overall, the ratio of the change in atrioventricular conduction to the change in heart rate for a given site was significantly greater 1 mm rostral to the obex than at either end of the test grid. -
Heart to Heart - STEAM Activity
Heart to Heart - STEAM Activity Purpose: The main objective of this exercise is to introduce how the human heart works. This lesson will be divided into four different lessons, including an introduction to heart anatomy, heart beats and pulses, and the circulatory system. We will be using coloring activities, stethoscopes, and handmade pumps to reinforce the concepts seen in this lesson. Vocabulary ● Artery: A blood vessel that carries blood high in oxygen content away from the heart to the farthest reaches of the body. ● Vein: a Blood vessel that carries blood low in oxygen content from the body back to the heart. ● Atrium: One of the two upper cavities of the heart that passes blood to the ventricles. ● Ventricles: One of the two lower chambers of the heart that receives blood from the atria. ● Valves: Tissue-paper thin membranes attached to the heart wall that constantly open and close to regulate blood flow. ● Pulse: A rhythmical, mechanical throbbing of the arteries as blood pumps through them. ● Heart Rate: The number of times per minute that the heart contracts - the number of heart beats per minute (bpm). ● Taquicardia: A high resting heart rate that is usually higher than 100 beats per minute. ● Bradycardia: A low resting heart rate that is usually lower than 60 beats per minute. ● Circulation: The movement of blood through the vessels of the body by the pumping action of the heart. It distributes nutrients and oxygen and removes waste products from all parts of the body. ● Pulmonary Circulation: The portion of the circulatory system that carries deoxygenated blood from the heart to the lungs and oxygenated blood back to the heart. -
Cardiac Symptoms and Physical Signs 11
CHAPTER 1 1 Cardiac Symptoms and Physical Signs 1.1 Common Cardiac Symptoms Angina Typical angina presents as a chest tightness or heaviness brought on by effort and relieved by rest. The sensation starts in the retrosternal region and radi- ates across the chest. Frequently it is associated with a leaden feeling in the arms. Occasionally it may present in more unusual sites, e.g. pain in the jaw or teeth on effort, without pain in the chest. It may be confused with oesopha- geal pain, or may present as epigastric or even hypochondrial pain. The most important feature is its relationship to effort. Unilateral chest pain (sub- mammary) is not usually cardiac pain, which is generally symmetrical in distribution. Angina is typically exacerbated by heavy meals, cold weather (just breath- ing in cold air is enough) and emotional disturbances. Arguments with col- leagues or family and watching exciting television are typical precipitating factors. Stable Angina This is angina induced by effort and relieved by rest. It does not increase in frequency or severity, and is predictable in nature. It is associated with ST- segment depression on ECG. Decubitus Angina This is angina induced by lying down at night or during sleep. It may be caused by an increase in LVEDV (and hence wall stress) on lying fl at, associ- ated with dreaming or getting between cold sheets. Coronary spasm may occur in REM sleep. It may respond to a diuretic, calcium antagonist or nitrate taken in the evening. Swanton’s Cardiology, sixth edition. By R. H. Swanton and S. -
Cardiology-EKG Michael Bradley
Cardiology/EKG Board Review Michael J. Bradley D.O. DME/Program Director Family Medicine Residency Objectives • Review general method for EKG interpretation • Review specific points of “data gathering” and “diagnoses” on EKG • Review treatment considerations • Review clinical cases/EKG’s • Board exam considerations EKG EKG – 12 Leads • Anterior Leads - V1, V2, V3, V4 • Inferior Leads – II, III, aVF • Left Lateral Leads – I, aVL, V5, V6 • Right Leads – aVR, V1 11 Step Method for Reading EKG’s • “Data Gathering” – steps 1-4 – 1. Standardization – make sure paper and paper speed is standardized – 2. Heart Rate – 3. Intervals – PR, QT, QRS width – 4. Axis – normal vs. deviation 11 Step Method for Reading EKG’s • “Diagnoses” – 5. Rhythm – 6. Atrioventricular (AV) Block Disturbances – 7. Bundle Branch Block or Hemiblock of – 8. Preexcitation Conduction – 9. Enlargement and Hypertrophy – 10. Coronary Artery Disease – 11. Utter Confusion • The Only EKG Book You’ll Ever Need Malcolm S. Thaler, MD Heart Rate • Regular Rhythms Heart Rate • Irregular Rhythms Intervals • Measure length of PR interval, QT interval, width of P wave, QRS complex QTc • QTc = QT interval corrected for heart rate – Uses Bazett’s Formula or Fridericia’s Formula • Long QT syndrome – inherited or acquired (>75 meds); torsades de ponites/VF; syncope, seizures, sudden death Axis Rhythm • 4 Questions – 1. Are normal P waves present? – 2. Are QRS complexes narrow or wide (≤ or ≥ 0.12)? – 3. What is relationship between P waves and QRS complexes? – 4. Is rhythm regular or irregular? -
Central Venous Pressure Venous Examination but Underestimates Ultrasound Accurately Reflects the Jugular
Ultrasound Accurately Reflects the Jugular Venous Examination but Underestimates Central Venous Pressure Gur Raj Deol, Nicole Collett, Andrew Ashby and Gregory A. Schmidt Chest 2011;139;95-100; Prepublished online August 26, 2010; DOI 10.1378/chest.10-1301 The online version of this article, along with updated information and services can be found online on the World Wide Web at: http://chestjournal.chestpubs.org/content/139/1/95.full.html Chest is the official journal of the American College of Chest Physicians. It has been published monthly since 1935. Copyright2011by the American College of Chest Physicians, 3300 Dundee Road, Northbrook, IL 60062. All rights reserved. No part of this article or PDF may be reproduced or distributed without the prior written permission of the copyright holder. (http://chestjournal.chestpubs.org/site/misc/reprints.xhtml) ISSN:0012-3692 Downloaded from chestjournal.chestpubs.org at UCSF Library & CKM on January 21, 2011 © 2011 American College of Chest Physicians CHEST Original Research CRITICAL CARE Ultrasound Accurately Refl ects the Jugular Venous Examination but Underestimates Central Venous Pressure Gur Raj Deol , MD ; Nicole Collett , MD ; Andrew Ashby , MD ; and Gregory A. Schmidt , MD , FCCP Background: Bedside ultrasound examination could be used to assess jugular venous pressure (JVP), and thus central venous pressure (CVP), more reliably than clinical examination. Methods: The study was a prospective, blinded evaluation comparing physical examination of external jugular venous pressure (JVPEXT), internal jugular venous pressure (JVPINT), and ultrasound collapse pressure (UCP) with CVP measured using an indwelling catheter. We com- pared the examination of the external and internal JVP with each other and with the UCP and CVP. -
Central Venous Pressure: Uses and Limitations
Central Venous Pressure: Uses and Limitations T. Smith, R. M. Grounds, and A. Rhodes Introduction A key component of the management of the critically ill patient is the optimization of cardiovascular function, including the provision of an adequate circulating volume and the titration of cardiac preload to improve cardiac output. In spite of the appearance of several newer monitoring technologies, central venous pressure (CVP) monitoring remains in common use [1] as an index of circulatory filling and of cardiac preload. In this chapter we will discuss the uses and limitations of this monitor in the critically ill patient. Defining Central Venous Pressure What is the Central Venous Pressure? Central venous pressure is the intravascular pressure in the great thoracic veins, measured relative to atmospheric pressure. It is conventionally measured at the junction of the superior vena cava and the right atrium and provides an estimate of the right atrial pressure. The Central Venous Pressure Waveform The normal CVP exhibits a complex waveform as illustrated in Figure 1. The waveform is described in terms of its components, three ascending ‘waves’ and two descents. The a-wave corresponds to atrial contraction and the x descent to atrial relaxation. The c wave, which punctuates the x descent, is caused by the closure of the tricuspid valve at the start of ventricular systole and the bulging of its leaflets back into the atrium. The v wave is due to continued venous return in the presence of a closed tricuspid valve. The y descent occurs at the end of ventricular systole when the tricuspid valve opens and blood once again flows from the atrium into the ventricle. -
Practical Cardiac Auscultation
LWW/CCNQ LWWJ306-08 March 7, 2007 23:32 Char Count= Crit Care Nurs Q Vol. 30, No. 2, pp. 166–180 Copyright c 2007 Wolters Kluwer Health | Lippincott Williams & Wilkins Practical Cardiac Auscultation Daniel M. Shindler, MD, FACC This article focuses on the practical use of the stethoscope. The art of the cardiac physical exam- ination includes skillful auscultation. The article provides the author’s personal approach to the patient for the purpose of best hearing, recognizing, and interpreting heart sounds and murmurs. It should be used as a brief introduction to the art of auscultation. This article also attempts to illustrate heart sounds and murmurs by using words and letters to phonate the sounds, and by presenting practical clinical examples where auscultation clearly influences cardiac diagnosis and treatment. The clinical sections attempt to go beyond what is available in standard textbooks by providing information and stethoscope techniques that are valuable and useful at the bedside. Key words: auscultation, murmur, stethoscope HIS article focuses on the practical use mastered at the bedside. This article also at- T of the stethoscope. The art of the cardiac tempts to illustrate heart sounds and mur- physical examination includes skillful auscul- murs by using words and letters to phonate tation. Even in an era of advanced easily avail- the sounds, and by presenting practical clin- able technological bedside diagnostic tech- ical examples where auscultation clearly in- niques such as echocardiography, there is still fluences cardiac diagnosis and treatment. We an important role for the hands-on approach begin by discussing proper stethoscope selec- to the patient for the purpose of evaluat- tion and use. -
Toolbox-Talks--Blood-Pressure.Pdf
TOOLBOX Toolbox Talk #1 TALKS Blood Pressure vs. Heart Rate While your blood pressure is the force of your blood moving through your blood vessels, your heart rate is the number of times your heart beats per minute. They are two separate measurements and indicators of health. • For people with high blood pressure (HBP or hypertension), there’s no substitute for measuring blood pressure. • Heart rate and blood pressure do not necessarily increase at the same rate. A rising heart rate does not cause your blood pressure to increase at the same Quarter: rate. Even though your heart is beating more times a minute, healthy blood BLOOD vessels dilate (get larger) to allow more blood to flow through more easily. PRESSURE When you exercise, your heart speeds up so more blood can reach your muscles. It may be possible for your heart rate to double safely, while your blood pressure may respond by only increasing a modest amount. Talk Number: Heart Rate and Exercise 1 In discussions about high blood pressure, you will often see heart rate Blood mentioned in relation to exercise. Your target heart rate is based on age and Pressure can help you monitor the intensity of your exercise. vs. • If you measure your heart rate (take your pulse) before, during and after Heart Rate physical activity, you’ll notice it will increase over the course of the exercise. • The greater the intensity of the exercise, the more your heart rate will increase. • When you stop exercising, your heart rate does not immediately return to your normal (resting) heart rate. -
Bates' Pocket Guide to Physical Examination and History Taking
Lynn S. Bickley, MD, FACP Clinical Professor of Internal Medicine School of Medicine University of New Mexico Albuquerque, New Mexico Peter G. Szilagyi, MD, MPH Professor of Pediatrics Chief, Division of General Pediatrics University of Rochester School of Medicine and Dentistry Rochester, New York Acquisitions Editor: Elizabeth Nieginski/Susan Rhyner Product Manager: Annette Ferran Editorial Assistant: Ashley Fischer Design Coordinator: Joan Wendt Art Director, Illustration: Brett MacNaughton Manufacturing Coordinator: Karin Duffield Indexer: Angie Allen Prepress Vendor: Aptara, Inc. 7th Edition Copyright © 2013 Wolters Kluwer Health | Lippincott Williams & Wilkins. Copyright © 2009 by Wolters Kluwer Health | Lippincott Williams & Wilkins. Copyright © 2007, 2004, 2000 by Lippincott Williams & Wilkins. Copyright © 1995, 1991 by J. B. Lippincott Company. All rights reserved. This book is protected by copyright. No part of this book may be reproduced or transmitted in any form or by any means, including as photocopies or scanned-in or other electronic copies, or utilized by any information storage and retrieval system without written permission from the copyright owner, except for brief quotations embodied in critical articles and reviews. Materials appear- ing in this book prepared by individuals as part of their official duties as U.S. government employees are not covered by the above-mentioned copyright. To request permission, please contact Lippincott Williams & Wilkins at Two Commerce Square, 2001 Market Street, Philadelphia PA 19103, via email at [email protected] or via website at lww.com (products and services). 9 8 7 6 5 4 3 2 1 Printed in China Library of Congress Cataloging-in-Publication Data Bickley, Lynn S. Bates’ pocket guide to physical examination and history taking / Lynn S. -
Cardiovascular Physiology
CARDIOVASCULAR PHYSIOLOGY Ida Sletteng Karlsen • Trym Reiberg Second Edition 15 March 2020 Copyright StudyAid 2020 Authors Trym Reiberg Ida Sletteng Karlsen Illustrators Nora Charlotte Sønstebø Ida Marie Lisle Amalie Misund Ida Sletteng Karlsen Trym Reiberg Booklet Disclaimer All rights reserved. No part oF this book may be reproduced in any Form on by an electronic or mechanical means, without permission From StudyAid. Although the authors have made every efFort to ensure the inFormation in the booklet was correct at date of publishing, the authors do not assume and hereby disclaim any liability to any part For any inFormation that is omitted or possible errors. The material is taken From a variety of academic sources as well as physiology lecturers, but are Further incorporated and summarized in an original manner. It is important to note, the material has not been approved by professors of physiology. All illustrations in the booklet are original. This booklet is made especially For students at the Jagiellonian University in Krakow by tutors in the StudyAid group (students at JU). It is available as a PDF and is available for printing. If you have any questions concerning copyrights oF the booklet please contact [email protected]. About StudyAid StudyAid is a student organization at the Jagiellonian University in Krakow. Throughout the academic year we host seminars in the major theoretical subjects: anatomy, physiology, biochemistry, immunology, pathophysiology, supplementing the lectures provided by the university. We are a group of 25 tutors, who are students at JU, each with their own Field oF specialty. To make our seminars as useful and relevant as possible, we teach in an interactive manner often using drawings and diagrams to help students remember the concepts.