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Produktinformation Diagnostik & molekulare Diagnostik Laborgeräte & Service Zellkultur & Verbrauchsmaterial Forschungsprodukte & Biochemikalien Weitere Information auf den folgenden Seiten! See the following pages for more information! Lieferung & Zahlungsart Lieferung: frei Haus Bestellung auf Rechnung SZABO-SCANDIC Lieferung: € 10,- HandelsgmbH & Co KG Erstbestellung Vorauskassa Quellenstraße 110, A-1100 Wien T. +43(0)1 489 3961-0 Zuschläge F. +43(0)1 489 3961-7 [email protected] • Mindermengenzuschlag www.szabo-scandic.com • Trockeneiszuschlag • Gefahrgutzuschlag linkedin.com/company/szaboscandic • Expressversand facebook.com/szaboscandic Tel:240-252-7368(USA) Fax:240-252-7376(USA) www.elabscience.com ® E-mail:[email protected] Elabscience Elabscience Biotechnology Inc. RMND5B Polyclonal Antibody Catalog No. E-AB-52625 Reactivity H,M Storage Store at -20℃. Avoid freeze / thaw cycles. Host Rabbit Applications IHC,ELISA Isotype IgG Note: Centrifuge before opening to ensure complete recovery of vial contents. Images Immunogen Information Immunogen Fusion protein of human RMND5B Gene Accession BC009911 Swissprot Q96G75 Synonyms FLJ22318,MGC112682,Protein RMD5 homolog B,Required for meiotic nuclear division 5 homolog B (S. cerevisiae),RP23-79E13.4 Immunohistochemistry of paraffin- Product Information embedded Human esophagus cancer Buffer PBS with 0.05% NaN3 and 40% Glycerol,pH7.4 tissue using RMND5B Polyclonal Purify Antigen affinity purification Antibody at dilution of 1:50(×200) Dilution IHC 1:50-1:300, ELISA 1:5000-1:10000 Background The function of this protein remains unknown. For Research Use Only Focus on your research Thank you for your recent purchase. If you would like to learn more about antibodies,please visit www.elabscience.com. Service for life science Applications:WB-Western Blot IHC-Immunohistochemistry IF-Immunofluorescence IP-Immunoprecipitation FC-Flow cytometry ChIP- Chromatin Immunoprecipitation Reactivity: H-Human R-Rat M-Mouse Mk-Monkey Dg-Dog Ch-Chicken Hm-Hamster Rb-Rabbit Sh- Sheep Pg-Pig Z-Zebrafish X-Xenopus C-Cow. Powered by TCPDF (www.tcpdf.org).

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A Computational Approach for Defining a Signature of Β-Cell Golgi Stress in Diabetes Mellitus
Page 1 of 781 Diabetes A Computational Approach for Defining a Signature of β-Cell Golgi Stress in Diabetes Mellitus Robert N. Bone1,6,7, Olufunmilola Oyebamiji2, Sayali Talware2, Sharmila Selvaraj2, Preethi Krishnan3,6, Farooq Syed1,6,7, Huanmei Wu2, Carmella Evans-Molina 1,3,4,5,6,7,8* Departments of 1Pediatrics, 3Medicine, 4Anatomy, Cell Biology & Physiology, 5Biochemistry & Molecular Biology, the 6Center for Diabetes & Metabolic Diseases, and the 7Herman B. Wells Center for Pediatric Research, Indiana University School of Medicine, Indianapolis, IN 46202; 2Department of BioHealth Informatics, Indiana University-Purdue University Indianapolis, Indianapolis, IN, 46202; 8Roudebush VA Medical Center, Indianapolis, IN 46202. *Corresponding Author(s): Carmella Evans-Molina, MD, PhD ([email protected]) Indiana University School of Medicine, 635 Barnhill Drive, MS 2031A, Indianapolis, IN 46202, Telephone: (317) 274-4145, Fax (317) 274-4107 Running Title: Golgi Stress Response in Diabetes Word Count: 4358 Number of Figures: 6 Keywords: Golgi apparatus stress, Islets, β cell, Type 1 diabetes, Type 2 diabetes 1 Diabetes Publish Ahead of Print, published online August 20, 2020 Diabetes Page 2 of 781 ABSTRACT The Golgi apparatus (GA) is an important site of insulin processing and granule maturation, but whether GA organelle dysfunction and GA stress are present in the diabetic β-cell has not been tested. We utilized an informatics-based approach to develop a transcriptional signature of β-cell GA stress using existing RNA sequencing and microarray datasets generated using human islets from donors with diabetes and islets where type 1(T1D) and type 2 diabetes (T2D) had been modeled ex vivo. To narrow our results to GA-specific genes, we applied a filter set of 1,030 genes accepted as GA associated.
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Integrating Single-Step GWAS and Bipartite Networks Reconstruction Provides Novel Insights Into Yearling Weight and Carcass Traits in Hanwoo Beef Cattle
animals Article Integrating Single-Step GWAS and Bipartite Networks Reconstruction Provides Novel Insights into Yearling Weight and Carcass Traits in Hanwoo Beef Cattle Masoumeh Naserkheil 1 , Abolfazl Bahrami 1 , Deukhwan Lee 2,* and Hossein Mehrban 3 1 Department of Animal Science, University College of Agriculture and Natural Resources, University of Tehran, Karaj 77871-31587, Iran; [email protected] (M.N.); [email protected] (A.B.) 2 Department of Animal Life and Environment Sciences, Hankyong National University, Jungang-ro 327, Anseong-si, Gyeonggi-do 17579, Korea 3 Department of Animal Science, Shahrekord University, Shahrekord 88186-34141, Iran; [email protected] * Correspondence: [email protected]; Tel.: +82-31-670-5091 Received: 25 August 2020; Accepted: 6 October 2020; Published: 9 October 2020 Simple Summary: Hanwoo is an indigenous cattle breed in Korea and popular for meat production owing to its rapid growth and high-quality meat. Its yearling weight and carcass traits (backfat thickness, carcass weight, eye muscle area, and marbling score) are economically important for the selection of young and proven bulls. In recent decades, the advent of high throughput genotyping technologies has made it possible to perform genome-wide association studies (GWAS) for the detection of genomic regions associated with traits of economic interest in diﬀerent species. In this study, we conducted a weighted single-step genome-wide association study which combines all genotypes, phenotypes and pedigree data in one step (ssGBLUP). It allows for the use of all SNPs simultaneously along with all phenotypes from genotyped and ungenotyped animals. Our results revealed 33 relevant genomic regions related to the traits of interest.
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Supplementary Table S4. FGA Co-Expressed Gene List in LUAD
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Meta-Analysis of Gene Expression in Individuals with Autism Spectrum Disorders
Meta-analysis of Gene Expression in Individuals with Autism Spectrum Disorders by Carolyn Lin Wei Ch’ng BSc., University of Michigan Ann Arbor, 2011 A THESIS SUBMITTED IN PARTIAL FULFILLMENT OF THE REQUIREMENTS FOR THE DEGREE OF Master of Science in THE FACULTY OF GRADUATE AND POSTDOCTORAL STUDIES (Bioinformatics) The University of British Columbia (Vancouver) August 2013 c Carolyn Lin Wei Ch’ng, 2013 Abstract Autism spectrum disorders (ASD) are clinically heterogeneous and biologically complex. State of the art genetics research has unveiled a large number of variants linked to ASD. But in general it remains unclear, what biological factors lead to changes in the brains of autistic individuals. We build on the premise that these heterogeneous genetic or genomic aberra- tions will converge towards a common impact downstream, which might be reflected in the transcriptomes of individuals with ASD. Similarly, a considerable number of transcriptome analyses have been performed in attempts to address this question, but their findings lack a clear consensus. As a result, each of these individual studies has not led to any significant advance in understanding the autistic phenotype as a whole. The goal of this research is to comprehensively re-evaluate these expression profiling studies by conducting a systematic meta-analysis. Here, we report a meta-analysis of over 1000 microarrays across twelve independent studies on expression changes in ASD compared to unaffected individuals, in blood and brain. We identified a number of genes that are consistently differentially expressed across studies of the brain, suggestive of effects on mitochondrial function. In blood, consistent changes were more difficult to identify, despite individual studies tending to exhibit larger effects than the brain studies.
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Identification of Genomic Targets of Krüppel-Like Factor 9 in Mouse Hippocampal
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An Integrated Analysis of Cashmere Fineness Lncrnas in Cashmere Goats
G C A T T A C G G C A T genes Article An Integrated Analysis of Cashmere Fineness lncRNAs in Cashmere Goats Yuan Y. Zheng 1 , Sheng D. Sheng 1, Tai Y. Hui 1, Chang Yue 1, Jia M. Sun 1, Dan Guo 2, Su L. Guo 3, Bo J. Li 1, Hui L. Xue 1, Ze Y. Wang 1,* and Wen L. Bai 1,* 1 College of Animal Science & Veterinary Medicine, Shenyang Agricultural University, Shenyang 110866, China; [email protected] (Y.Y.Z.); [email protected] (S.D.S.); [email protected] (T.Y.H.); [email protected] (C.Y.); [email protected] (J.M.S.); [email protected] (B.J.L.); [email protected] (H.L.X.) 2 Animal Genetic Breeding, Academy of Animal Husbandry Science of Liaoning Province, Liaoyang 130062, China; [email protected] 3 Prosperous Community, Changshun Town, Huade 013350, China; [email protected] * Correspondence: [email protected] (Z.Y.W.); [email protected] (W.L.B.); Tel.: +86-15998362195 (Z.Y.W.) Received: 29 January 2019; Accepted: 28 March 2019; Published: 2 April 2019 Abstract: Animal growth and development are regulated by long non-coding RNAs (lncRNAs). However, the functions of lncRNAs in regulating cashmere fineness are poorly understood. To identify the key lncRNAs that are related to cashmere fineness in skin, we have collected skin samples of Liaoning cashmere goats (LCG) and Inner Mongolia cashmere goats (MCG) in the anagen phase, and have performed RNA sequencing (RNA-seq) approach on these samples. The high-throughput sequencing and bioinformatics analyses identified 437 novel lncRNAs, including 93 differentially expressed lncRNAs.
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Of the T Cell by G-CSF and IL-10 Cell Mobilization Requires Direct
Downloaded from http://www.jimmunol.org/ by guest on October 1, 2021 is online at: average * The Journal of Immunology , 26 of which you can access for free at: 2014; 192:3180-3189; Prepublished online 28 from submission to initial decision 4 weeks from acceptance to publication February 2014; doi: 10.4049/jimmunol.1302315 http://www.jimmunol.org/content/192/7/3180 Modification of T Cell Responses by Stem Cell Mobilization Requires Direct Signaling of the T Cell by G-CSF and IL-10 Kelli P. A. MacDonald, Laetitia Le Texier, Ping Zhang, Helen Morris, Rachel D. Kuns, Katie E. Lineburg, Lucie Leveque, Alistair L. Don, Kate A. Markey, Slavica Vuckovic, Frederik O. Bagger, Glen M. Boyle, Bruce R. Blazar and Geoffrey R. Hill J Immunol cites 45 articles Submit online. Every submission reviewed by practicing scientists ? is published twice each month by Submit copyright permission requests at: http://www.aai.org/About/Publications/JI/copyright.html Receive free email-alerts when new articles cite this article. Sign up at: http://jimmunol.org/alerts http://jimmunol.org/subscription http://www.jimmunol.org/content/suppl/2014/02/28/jimmunol.130231 5.DCSupplemental This article http://www.jimmunol.org/content/192/7/3180.full#ref-list-1 Information about subscribing to The JI No Triage! Fast Publication! Rapid Reviews! 30 days* Why • • • Material References Permissions Email Alerts Subscription Supplementary The Journal of Immunology The American Association of Immunologists, Inc., 1451 Rockville Pike, Suite 650, Rockville, MD 20852 Copyright © 2014 by The American Association of Immunologists, Inc. All rights reserved. Print ISSN: 0022-1767 Online ISSN: 1550-6606.
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Supplemental Material
Supplemental Material Methods Laboratory Analyses We measured levels of serum and urine electrolytes and creatinine in samples obtained from all patients using routine methods. Intact parathyroid hormone (iPTH) levels were measured by chemiluminescent immunoassay (CLIA Immulite 2000XP, Siemens, Germany), FGF-23 plasma concentrations were determined by ELISA (Human FGF23 C-terminal Elisa kit, Immutopics International, San Clemente, CA, USA) (reference range 26-110 kRU/mL)(for details see below), 25-OH-D3 was measured by a direct competitive chemiluminescent immunoassay (CLIA, Liaison Analyzer, Diasorin S.p.A.), and 1,25-(OH)2D3 was measured after purification by radioimmunoassay (IDS kit, Immunodiagnostic Systems, Germany). Tubular phosphate reabsorption Renal phosphate handling was assessed according to Stark et al. as TmP/GFR = SPO4 – (UPO4 x 1 1,2 SCr / UCr) . For SPO4 and TmP/GFR, age dependent reference values were used . Mean and SD for TmP/GFR were converted to mmol/L GFR resulting in the following reference ranges (mean ±2SD): Newborn=1.4-3.0mmol/l; 1month to 2years = 0.9-2.1mmol/l, 2-12years = 1.0- 1.8mmol/l, 12-16years = 0.9-1.7mmol/l, >16years = 0.8-1.2mmol/l. Genome wide linkage / LOD score calculation DNA of four affected individuals shown in the pedigrees F1, F2, F3 (Figure S1) was used to map the disease locus. Individuals F1.1, F1.2 and F2.1 were analyzed using Illumina Human660W-Quad BeadChips, individual F3.1 was run on a Human OmniExpress BeadChip. For combined analysis only those SNPs present on both chip types were used where a genotype was obtained for all four individuals (about 239.000 SNPs).
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Autocrine IFN Signaling Inducing Profibrotic Fibroblast Responses by a Synthetic TLR3 Ligand Mitigates
Downloaded from http://www.jimmunol.org/ by guest on September 28, 2021 Inducing is online at: average * The Journal of Immunology published online 16 August 2013 from submission to initial decision 4 weeks from acceptance to publication http://www.jimmunol.org/content/early/2013/08/16/jimmun ol.1300376 A Synthetic TLR3 Ligand Mitigates Profibrotic Fibroblast Responses by Autocrine IFN Signaling Feng Fang, Kohtaro Ooka, Xiaoyong Sun, Ruchi Shah, Swati Bhattacharyya, Jun Wei and John Varga J Immunol Submit online. Every submission reviewed by practicing scientists ? is published twice each month by http://jimmunol.org/subscription Submit copyright permission requests at: http://www.aai.org/About/Publications/JI/copyright.html Receive free email-alerts when new articles cite this article. Sign up at: http://jimmunol.org/alerts http://www.jimmunol.org/content/suppl/2013/08/20/jimmunol.130037 6.DC1 Information about subscribing to The JI No Triage! Fast Publication! Rapid Reviews! 30 days* Why • • • Material Permissions Email Alerts Subscription Supplementary The Journal of Immunology The American Association of Immunologists, Inc., 1451 Rockville Pike, Suite 650, Rockville, MD 20852 Copyright © 2013 by The American Association of Immunologists, Inc. All rights reserved. Print ISSN: 0022-1767 Online ISSN: 1550-6606. This information is current as of September 28, 2021. Published August 16, 2013, doi:10.4049/jimmunol.1300376 The Journal of Immunology A Synthetic TLR3 Ligand Mitigates Proﬁbrotic Fibroblast Responses by Inducing Autocrine IFN Signaling Feng Fang,* Kohtaro Ooka,* Xiaoyong Sun,† Ruchi Shah,* Swati Bhattacharyya,* Jun Wei,* and John Varga* Activation of TLR3 by exogenous microbial ligands or endogenous injury-associated ligands leads to production of type I IFN.
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Mycobacterium Tuberculosis Induced Transcription in Macrophages: the Role of TPL2/ERK Signalling in the Negative Regulation of T
Mycobacterium tuberculosis induced transcription in macrophages: the role of TPL2/ERK signalling in the negative regulation of type I interferon production and implications for control of tuberculosis John Benson Ewbank August 2012 Division of Immunoregulation MRC National Institute for Medical Research The Ridgeway, Mill Hill London NW7 1AA Submitted to University College London for the Degree of Doctor of Philosophy I, John Benson Ewbank, confirm that the work presented in this thesis is my own. Where information has been derived from other sources, I confirm that this has been indicated in the thesis Abstract Abstract Mycobacterium tuberculosis is an important global cause of mortality and morbidity. The major host cell of Mycobacterium tuberculosis is the macrophage, and Mycobacterium tuberculosis is able to subvert the macrophage response in order to survive and replicate. The majority of infected individuals mount an immune response capable of controlling Mycobacterium tuberculosis infection. This requires the cytokines IL-12, TNFα, IL-1 and IFNγ, which promote eradication or control of infection. However, other immune factors, including IL-10 and type I IFN, can inhibit this protective response. In this study we have used microarray analysis to study the temporal response of macrophages to Mycobacterium tuberculosis infection, in an unbiased fashion. In response to Mycobacterium tuberculosis infection, macrophages produced cytokines and chemokines, upregulated genes involved with major histocompatability class I antigen presentation, activated both pro- and anti-apoptotic genes and downregulated many genes involved in cell-division and metabolism. We also observed the early induction of genes regulated by the extracellular-regulated kinase (ERK) MAP kinase pathway, and the upregulation of genes known to be induced by type I IFN, leading us to further investigate the role of these pathways in the macrophage response to Mycobacterium tuberculosis.
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A Biochemical Landscape of A-To-I RNA Editing in the Human Brain Transcriptome
Downloaded from genome.cshlp.org on October 3, 2021 - Published by Cold Spring Harbor Laboratory Press Resource A biochemical landscape of A-to-I RNA editing in the human brain transcriptome Masayuki Sakurai,1,4 Hiroki Ueda,1,4 Takanori Yano,1 Shunpei Okada,1 Hideki Terajima,1 Toutai Mitsuyama,2 Atsushi Toyoda,3 Asao Fujiyama,3 Hitomi Kawabata,1 and Tsutomu Suzuki1,5 1Department of Chemistry and Biotechnology, Graduate School of Engineering, University of Tokyo, Tokyo 113-8656, Japan; 2Computational Biology Research Center (CBRC), National Institute of Advanced Industrial Science and Technology (AIST), Tokyo 135-0064, Japan; 3Comparative Genomics Laboratory, Center for Genetic Resource Information, National Institute of Genetics, Shizuoka 411-8540, Japan Inosine is an abundant RNA modification in the human transcriptome and is essential for many biological processes in modulating gene expression at the post-transcriptional level. Adenosine deaminases acting on RNA (ADARs) catalyze the hydrolytic deamination of adenosines to inosines (A-to-I editing) in double-stranded regions. We previously established a biochemical method called ‘‘inosine chemical erasing’’ (ICE) to directly identify inosines on RNA strands with high reliability. Here, we have applied the ICE method combined with deep sequencing (ICE-seq) to conduct an unbiased genome-wide screening of A-to-I editing sites in the transcriptome of human adult brain. Taken together with the sites identified by the conventional ICE method, we mapped 19,791 novel sites and newly found 1258 edited mRNAs, including 66 novel sites in coding regions, 41 of which cause altered amino acid assignment. ICE-seq detected novel editing sites in various repeat elements as well as in short hairpins.
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Analysis of Transciptome and Splicing Changes In
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