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Index A Acute actinic prurigo, 26 Abscess, 2 Acute febrile neutrophilic dermatosis, see Sweet’s Acanthamoebiasis, 3 syndrome Acantholytic acanthoma, 4 Acute generalized exanthematous pustulosis (AGEP), 29 Acantholytic actinic keratosis, 25 Acute grade 2 GvHD, 260 Acantholytic squamous cell carcinoma, 578 Acute grade 3 GvHD, 260 Acanthoma fissuratum, 202 Acute irritant contact dermatitis, 308 Acanthosis nigricans, 5 Acute lupus erythematosus, 30 Acanthotic seborrheic keratosis, 565 Acute radiation dermatitis, 536 Accessory digit, see Supernumerary digit Adenoid cystic basal cell carcinoma, 75 Accessory nipple, see Supernumerary nipple Adenoid cystic carcinoma, 31 Accessory tragus, 6 Adenoma Sebaceum, see Angiofibroma Acne agminata, 7 Adult T-cell lymphoma/leukemia, 32, 149, 150 Acne conglobata, 8 Adult xanthogranuloma (AXG), 436, 664 Acne fulminans, 9 African tick bite, 33 Acne inversa, see Hidradenitis suppurativa AGEP, see Acute generalized exanthematous Acne keloidalis nuchae, 10, 553 pustulosis (AGEP) Acne rosacea, see Rosacea Aggressive digital papillary adenocarcinoma, 34 Acne vulgaris Albinism, 35 inflammatory, 11, 12 Albright’s syndrome, 94 noninflammatory, 11 Allergic contact dermatitis, 36 Acquired digital fibrokeratoma, 13 acute, 36 Acral fibrokeratoma, 13 chronic, 36 Acral fibromyxoma, 14 subacute, 36 Acral papular mucinosis, 398 Allergic granulomatosis with polyangiitis, see Churg Acral persistent papular mucinosis, 15, 342 Strauss disease Acral vesicular dermatitis, see Dyshidrotic dermatitis Alopecia areata, 37 Acroangiodermatitis, 16 Alopecia mucinosa, see Follicular mucinosis Acrochordon, 17 Alpha-1 antitrypsin deficiency associated panniculitis, Acrodermatitis chronica atrophicans, 18, 364 see Alpha-1 antitrypsin panniculitis early lesions, 18 Alpha-1 antitrypsin panniculitis, 38 established lesions, 18 Alternariosis, 39 Acrodermatitis enteropathica, 418 Amalgam tattoo, 40 Acrokeratosis verruciformis of Hopf, 19 Ameloblastoma, 41 Acrospiroma, 20, 21 Amputation neuroma, 619 Actinic cheilitis, 22 Amyloidosis, 42 Actinic elastosis, see Solar elastosis lichen, 42 Actinic granuloma, 23 macular, 42 Actinic keratosis, 24, 25, 144 nodular/plaque, 42 Actinic lentigo, see Solar lentigo Androgenic alopecia, 43 Actinic prurigo, 26, 352 Anetoderma, 44 acute, 26 early lesion, 44 chronic, 26 established lesion, 44 Actinic reticuloid, 27, 352 Angiocentric lymphoma, see Extranodal NK/T-cell Actinomycosis, 28 lymphoma, nasal type © Springer International Publishing AG, part of Springer Nature 2018 671 V. Vincek, Dermatopathology A–Z, https://doi.org/10.

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Periodontal Assessment and Treatment Planning Gingival description Color: o pink o erythematous o cyanotic o racial pigmentation o metallic pigmentation o uniformity Contour: o recession o clefts o enlarged papillae o cratered papillae o blunted papillae o highly rolled o bulbous o knife-edged o scalloped o stippled Consistency: o firm o edematous o hyperplastic o fibrotic Band of gingiva: o amount o quality o location o treatability Bleeding tendency: o sulcus base, lining o gingival margins Suppuration Sinus tract formation Pocket depths Pseudopockets Frena Pain Other pathology Dental Description Defective restorations: o overhangs o open contacts o poor contours Fractured cusps 1 ww.links2success.biz [email protected] 914-303-6464 Caries Deposits: o Type . plaque . calculus . stain . matera alba o Location . supragingival . subgingival o Severity . mild . moderate . severe Wear facets Percussion sensitivity Tooth vitality Attrition, erosion, abrasion Occlusal plane level Occlusion findings Furcations Mobility Fremitus Radiographic findings Film dates Crown:root ratio Amount of bone loss o horizontal; vertical o localized; generalized Root length and shape Overhangs Bulbous crowns Fenestrations Dehiscences Tooth resorption Retained root tips Impacted teeth Root proximities Tilted teeth Radiolucencies/opacities Etiologic factors Local: o plaque o calculus o overhangs 2 ww.links2success.biz [email protected] 914-303-6464 o orthodontic apparatus o open margins o open contacts o improper
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Skin Lesions in Diabetic Patients
Rev Saúde Pública 2005;39(4) 1 www.fsp.usp.br/rsp Skin lesions in diabetic patients N T Foss, D P Polon, M H Takada, M C Foss-Freitas and M C Foss Departamento de Clínica Médica. Faculdade de Medicina de Ribeirão Preto. Universidade de São Paulo. Ribeirão Preto, SP, Brasil Keywords Abstract Skin diseases. Dermatomycoses. Diabetes mellitus. Metabolic control. Objective It is yet unknown the relationship between diabetes and determinants or triggering factors of skin lesions in diabetic patients. The purpose of the present study was to investigate the presence of unreported skin lesions in diabetic patients and their relationship with metabolic control of diabetes. Methods A total of 403 diabetic patients, 31% type 1 and 69% type 2, underwent dermatological examination in an outpatient clinic of a university hospital. The endocrine-metabolic evaluation was carried out by an endocrinologist followed by the dermatological evaluation by a dermatologist. The metabolic control of 136 patients was evaluated using glycated hemoglobin. Results High number of dermophytosis (82.6%) followed by different types of skin lesions such as acne and actinic degeneration (66.7%), pyoderma (5%), cutaneous tumors (3%) and necrobiosis lipoidic (1%) were found. Among the most common skin lesions in diabetic patients, confirmed by histopathology, there were seen necrobiosis lipoidic (2 cases, 0.4%), diabetic dermopathy (5 cases, 1.2%) and foot ulcerations (3 cases, 0.7%). Glycated hemoglobin was 7.2% in both type 1 and 2 patients with adequate metabolic control and 11.9% and 12.7% in type 1 and 2 diabetic patients, respectively, with inadequate metabolic controls.
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Malignant Hidradenoma: a Report of Two Cases and Review of the Literature
ANTICANCER RESEARCH 26: 2217-2220 (2006) Malignant Hidradenoma: A Report of Two Cases and Review of the Literature I.E. LIAPAKIS1, D.P. KORKOLIS2, A. KOUTSOUMBI3, A. FIDA3, G. KOKKALIS1 and P.P. VASSILOPOULOS2 1Department of Plastic and Reconstructive Surgery, 2First Department of Surgical Oncology and 3Department of Surgical Pathology, Hellenic Anticancer Institute, "Saint Savvas" Hospital, Athens, Greece Abstract. Introduction: Malignant tumors of the sweat glands difficult (1). Clear cell hidradenoma is an extremely rare are very rare. Clear cell hidradenoma is a lesion with tumor with less than 50 cases reported (2, 3). histopathological features resembling those of eccrine poroma The cases of two patients, suffering from aggressive and eccrine spiradenoma. The biological behavior of the tumor dermal lesions invading the abdominal wall and the axillary is aggressive, with local recurrences reported in more than 50% region, are described here. Surgical resection and of the surgically-treated cases. Materials and Methods: Two histopathological examination ascertained the presence of patients are presented, the first with tumor in the right axillary malignant clear cell hidradenoma. In addition to these region, the second with a recurrent tumor of the abdominal cases, a review of the literature is also presented. wall. The first patient underwent wide excision with clear margins and axillary lymph node dissection and the second Case Reports patient underwent wide excision of the primary lesion and bilateral inguinal node dissection due to palpable nodes. Patient 1. Patient 1 was a 68-year-old Caucasian male who had Results: The patients had uneventful postoperative courses. No undergone excision of a rapidly growing, ulcerous lesion of the additional treatment was administered.
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Topical Treatments for Seborrheic Keratosis: a Systematic Review
SYSTEMATIC REVIEW AND META-ANALYSIS Topical Treatments for Seborrheic Keratosis: A Systematic Review Ma. Celina Cephyr C. Gonzalez, Veronica Marie E. Ramos and Cynthia P. Ciriaco-Tan Department of Dermatology, College of Medicine and Philippine General Hospital, University of the Philippines Manila ABSTRACT Background. Seborrheic keratosis is a benign skin tumor removed through electrodessication, cryotherapy, or surgery. Alternative options may be beneficial to patients with contraindications to standard treatment, or those who prefer a non-invasive approach. Objectives. To determine the effectiveness and safety of topical medications on seborrheic keratosis in the clearance of lesions, compared to placebo or standard therapy. Methods. Studies involving seborrheic keratosis treated with any topical medication, compared to cryotherapy, electrodessication or placebo were obtained from MEDLINE, HERDIN, and Cochrane electronic databases from 1990 to June 2018. Results. The search strategy yielded sixty articles. Nine publications (two randomized controlled trials, two non- randomized controlled trials, three cohort studies, two case reports) covering twelve medications (hydrogen peroxide, tacalcitol, calcipotriol, maxacalcitol, ammonium lactate, tazarotene, imiquimod, trichloroacetic acid, urea, nitric-zinc oxide, potassium dobesilate, 5-fluorouracil) were identified. The analysis showed that hydrogen peroxide 40% presented the highest level of evidence and was significantly more effective in the clearance of lesions compared to placebo. Conclusion. Most of the treatments reviewed resulted in good to excellent lesion clearance, with a few well- tolerated minor adverse events. Topical therapy is a viable option; however, the level of evidence is low. Standard invasive therapy remains to be the more acceptable modality. Key Words: seborrheic keratosis, topical, systematic review INTRODUCTION Description of the condition Seborrheic keratoses (SK) are very common benign tumors of the hair-bearing skin, typically seen in the elderly population.
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Actinic Keratoses Final Report
Actinic Keratoses Final Report Mark Helfand, MD, MPH Annalisa K. Gorman, MD Susan Mahon, MPH Benjamin K.S. Chan, MS Neil Swanson, MD Submitted to the Agency for Healthcare Research and Quality under contract 290-97-0018, task order no. 6 Oregon Health & Science University Evidence-based Practice Center 3181 SW Sam Jackson Park Road Portland, Oregon 97201 May 19, 2001 Actinic Keratoses Structured Abstract Objective: To examine evidence about the natural history and management of actinic keratoses (AKs). Search Strategy: We searched the MEDLINE database from January 1966 to January 2001, the Cochrane Controlled Trials Registry, and a bibliographic database of articles about skin cancer. We identified additional articles from reference lists and experts. Selection Criteria: We selected 45 articles that contained original data relevant to treatment of actinic keratoses, progression of AKs to squamous cell cancer (SCC ), means of identifying a high-risk group, or surveillance of patients with AKs to detect and treat SCCs early in their course. Data Collection and Analysis: We abstracted information from these studies to construct evidence tables. We also developed a simple mathematical model to examine whether estimates of the rate of progression of AK to SCC were consistent among studies. Finally, we analyzed data from the Medicare Statistical System to estimate the frequency of procedures attributable to AK among elderly beneficiaries. Main Results: The yearly rate of progression of an AK in an average-risk person in Australia is between 8 and 24 per 10,000. High-risk individuals with multiple AKs have progression rates as high as 12-30 percent over 3 years.
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Fundamentals of Dermatology Describing Rashes and Lesions
Dermatology for the Non-Dermatologist May 30 – June 3, 2018 - 1 - Fundamentals of Dermatology Describing Rashes and Lesions History remains ESSENTIAL to establish diagnosis – duration, treatments, prior history of skin conditions, drug use, systemic illness, etc., etc. Historical characteristics of lesions and rashes are also key elements of the description. Painful vs. painless? Pruritic? Burning sensation? Key descriptive elements – 1- definition and morphology of the lesion, 2- location and the extent of the disease. DEFINITIONS: Atrophy: Thinning of the epidermis and/or dermis causing a shiny appearance or fine wrinkling and/or depression of the skin (common causes: steroids, sudden weight gain, “stretch marks”) Bulla: Circumscribed superficial collection of fluid below or within the epidermis > 5mm (if <5mm vesicle), may be formed by the coalescence of vesicles (blister) Burrow: A linear, “threadlike” elevation of the skin, typically a few millimeters long. (scabies) Comedo: A plugged sebaceous follicle, such as closed (whitehead) & open comedones (blackhead) in acne Crust: Dried residue of serum, blood or pus (scab) Cyst: A circumscribed, usually slightly compressible, round, walled lesion, below the epidermis, may be filled with fluid or semi-solid material (sebaceous cyst, cystic acne) Dermatitis: nonspecific term for inflammation of the skin (many possible causes); may be a specific condition, e.g. atopic dermatitis Eczema: a generic term for acute or chronic inflammatory conditions of the skin. Typically appears erythematous,
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Advanced Technology with Maximum Versatility for Dermatology & Aesthetic Procedures
ADVANCED TECHNOLOGY WITH MAXIMUM VERSATILITY FOR DERMATOLOGY & AESTHETIC PROCEDURES Fractional and Non-Fractional Applications Skin Resurfacing Skin Rejuvenation Skin Tightening Scar Remodeling Skin Tags & Benign Skin Lesion Skin Imperfections “The Pixel CO2 is brilliant; it is a must - have device for all dermatologists and plastic surgeons.” Dr. Michael Shochat, MD, Dermatologist. 2 | ALMA Pixel CO2 ALMA The carbon dioxide (CO2) laser has been known to provide some of the most dramatic, age-defying results in the treatment of challenging skin imperfections including wrinkles, fine lines, photodamage, uneven skin tone and skin laxity, as well as in scar treatment, skin tags and benign tumors. Using the power of the CO2 laser, the optimal mix of ablative and thermal effects and an array of applicators and treatment modes for highly tailored procedures, Alma’s Pixel CO2 brings unparalleled precision and innovation to the field of dermatology and plastic surgery. Alma Pixel CO2 is a highly flexible system for char-free tissue ablation, vaporization, excision, incision and coagulation of soft tissue. It allows physicians full control of treatment parameters, including level and depth of ablation and thermal control via pulse duration and mode of energy delivery. This versatility maximizes precision and treatment results while minimizing unnecessary tissue damage. The CO2 laser uses a 10,600nm wavelength, which is ideal for collagen matrix renewal and an optimal choice for treating an extensive range of dermatological issues. The CO2 laser has the ability to perform efficient, highly precise fractional and non-fractional laser treatments using the widest assortment of advanced applicators. With powerful performance and hundreds of treatment options, Alma Pixel CO2 opens the door to new possibilities in dermatological and surgical treatments.
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Cryosurgery Using the Cryopen®
Cryosurgery using the CryoPen® FAQ CRYOSURGERY What is cryosurgery? Cryosurgery is a procedure that uses extreme cold to destroy tissue. How can my practice benefit from using cryosurgery in my practice? Cryosurgery in the office offers an excellent modality for eliminating referral time while creating an added source of revenue. How can my patients benefit from having cryosurgery in my practice? Patients will appreciate the efficient use of their time and decreased cost of services by avoiding secondary visits to specialists. By keeping the procedure in house, patients will put a greater value on your practice. How is cryosurgery better than other methods of removing skin lesions? Cryosurgery requires no anesthesia and has less scarring than other techniques of skin lesion removal with minimal post-op care. What is the mechanism of cell destruction in cryosurgery? Cell destruction occurs when a cell is rapidly brought down to a very low temperature. When these two criteria are met (varies with cell type), ice crystals form, destroying the cell organelles and protein matrixes. Water then rushes into the surrounding area causing a blister and a disruption of the local blood supply. Cytologic evidence of cell destruction can be seen as soon as two hours after the procedure. What types of lesions are appropriate to freeze? Almost any unwanted skin lesions are appropriate such as warts, moles, actinic keratosis, seborrheic keratosis, keloids, lentigos, dermatofibromas, and hemangiomas to just name a few. In most practices, over 90% of unwanted lesions encountered are amenable to using cryosurgery. What types of lesions are not appropriate to freeze? All Melanomas and Recurrent Basal Cell Carcinomas are contraindicated for cryosurgery.
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Copyrighted Material
Trim size: Trim Size: 216mm x 279mm Holm bindex.tex V3 - 05/25/2015 11:50 A.M. Page 361 Index figures are in italics; tables/boxes are in bold aging and periodontal disease anticoagulant therapy epidemiology, 212–213 ADP anticoagulant platelet inhibitor, A periodontal inflammation, systemic 257–258 acetaminophen (paracetamol), 92, 95, 99, 270, diseases and aging, 218 cyclooxygenase inhibitors, 257 275 risk indicators/population, 213–215 warfarin, 257 acidulated phosphate fluoride (AFP), 195 susceptibility, 215–218 antihistamines, 256 actinic cheilitis (solar cheilitis), 72 alcohol aphasia, 345 Actinic elastosis of lower lip, 235 dementia, 112 Appletree dental truck, 330 Actinomyces gerencseriae, 186 mouth cancer, 158 apraxia, 345 Actinomyces Israelii, 186 oral cancer, 136, 236 Arthritis and oral hygiene/denture insertion,72 Active coronal/root caries in mandibular sugars, 138 ‘artificial salivas’, 251 anterior teeth,74 tooth wear, 139 Assessment tools for oral examination,76 activities of daily living (ADL), 64, 70, 121–122, alendronate see bisphosphonate asthma 315 allergy dental management, 97–98 acupuncture (salivary stimulation), 251 dental management, 82–83 overview, 97 adaptation overview, 82 Atrophic mandibular posterior ridge,75 aging, 56 aloe vera, 251 attrition (occlusal wear), 73–74 response, 13 Alveolar bone loss prevention, 160 Attrition (occlusal wear), 74 theories, 7 alveolar ridge, 75, 142 atypical presentation of disease, 62 Addison’s disease, 72, 94 Alzheimer’s disease (AD) auditory function ADP antagonist platelet inhibitor,
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Keratitis-Ichthyosis-Deafness Syndrome in Association With
Genes and skin Eur J Dermatol 2005; 15 (5): 347-52 Laura MAINTZ1 Regina C. BETZ2 Keratitis-ichthyosis-deafness syndrome Jean-Pierre ALLAM1 in association with follicular occlusion triad Jörg WENZEL1 Axel JAKSCHE3 Nicolaus FRIEDRICHS4 Thomas BIEBER1 Keratitis-Ichthyosis-Deafness syndrome is a rare congenital disorder of Natalija NOVAK1 the ectoderm caused by mutations in the connexin-26 gene (GJB2) on 1 chromosome 13q11-q12, giving rise to keratitis, erythrokeratoderma Department of Dermatology, University of and neurosensory deafness. We report the case of a 31-year-old black Bonn, Sigmund-Freud-Str. 25, 53105 Bonn, Germany male diagnosed as having KID syndrome. Sequencing analysis showed 2 Institute of Human Genetics, University of a heterozygous missense mutation D50N (148G > A) in the GJB2 gene. Bonn, Wilhelmstr. 31, 53115 Bonn, In addition to the classical features of vascularizing keratitis, erythro- Germany 3 Department of Ophthalmology, University keratoderma and congenital deafness, our patient presented a follicular of Bonn, Sigmund-Freud-Str. 25, 53105 occlusion triad with hidradenitis suppurativa (HS, alias acne inversa), Bonn, Germany acne conglobata and dissecting cellulitis of the scalp, leading to cicatri- 4 Institute of Pathology, University of Bonn, Sigmund-Freud-Str. 25, 53105 Bonn, cial alopecia and disfiguring, inflammatory vegetations of his scalp. Germany Conservative therapy such as a keratolytic, rehydrating and antiseptic external therapy, antibiotic, antimycotic and retinoids were only of Reprints: N. Novak moderate benefit, so we finally chose the curative possibility of surgery Fax: (+49) 228 287 4883 <[email protected]> therapy of the axillar papillomas and of the scalp. The inflammatory papillomatous regions of the axillae and of the scalp were radically debrided.
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Expert-Level Diagnosis of Nonpigmented Skin Cancer by Combined Convolutional Neural Networks
Supplementary Online Content Tschandl P, Rosendahl C, Akay BN, et al. Expert-level diagnosis of nonpigmented skin cancer by combined convolutional neural networks. JAMA Dermatol. Published online November 28, 2018. doi:10.1001/jamadermatol.2018.4378 eFigure. Sensitivities (Blue) and Specificities (Orange) at Different Threshold Cutoffs (Green) of the Combined Classifier Evaluated on the Validation Set eAppendix. Neural Network Training eTable 1. Complete List of Diagnoses and Their Frequencies Within the Test-Set eTable 2. Education of Users According to Their Experience Group eTable 3. Percent of Correct Prediction of the Malignancy Status for Specific Diagnoses of a CNN Using Either Close-up or Dermatoscopic Images This supplementary material has been provided by the authors to give readers additional information about their work. © 2018 American Medical Association. All rights reserved. Downloaded From: https://jamanetwork.com/ on 09/25/2021 eFigure. Sensitivities (Blue) and Specificities (Orange) at Different Threshold Cutoffs (Green) of the Combined Classifier Evaluated on the Validation Set A threshold cut at 0.2 (black) is found for a minimum of 51.3% specificity. © 2018 American Medical Association. All rights reserved. Downloaded From: https://jamanetwork.com/ on 09/25/2021 eAppendix. Neural Network Training We compared multiple architecture and training hyperparameter combinations in a grid-search fashion, and used only the single best performing network for dermoscopic and close-up images, based on validation accuracy, for further analyses. We trained four different CNN architectures (InceptionResNetV2, InceptionV3, Xception, ResNet50) and used model definitions and ImageNet pretrained weights as available in the Tensorflow (version 1.3.0)/ Keras (version 2.0.8) frameworks.
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371 a Acne Excoriee , 21, 22 Acneiform Disorders , 340 Acne
Index A African American community Acne excoriee , 21, 22 cocoa butter , 302 Acneiform disorders , 340 diagnosis codes, dermatologist Acne keloidalis nuchae (AKN) , 340 visit , 301 description , 130 hair myths , 303 diagnosis , 131, 132 patient care , 304 differential diagnosis , 133 skin myths , 301–303 epidemiology , 130–131 African descent, cultural considerations histopathology , 133 description , 300 laser hair removal , 244 health services utilization , 300–301 pathogenesis , 131 misconceptions , 301 prevalence , 130–131 AGA. See Androgenetic alopecia (AGA) treatment Aging effects, ethnic skin , 248–249 ﬁ rst line therapy , 134–135 AKN. See Acne keloidalis nuchae (AKN) minimally invasive therapy , 135 Alaluf, S. , 6 surgical , 135 Alexis, A.F. , 23 Acne vulgaris (AV) Alopecia areata , 99–100 aggravating factors , 23 Alopecia syphilitica , 101–102 clinical features , 21–23 Alpha hydroxy acids , 286–288 epidemiology , 23–24 Alster, T. , 197 management Anagen ef ﬂ uvium (AE) , 99 oral therapy , 27 Androgenetic alopecia (AGA) , 97–98, procedural therapy , 27–28 355–356 topical therapy , 24–26 Antimalarials pathogenic factors , 23 lupus erythematosus , 55 PIH , 22 sarcoidosis , 71 vs. rosacea , 29 Aramaki, J. , 10 sequelae , 28 Aromatherapy, traditional Asian practice Acupuncture alopecia areata , 309 traditional Asian practice, cutaneous contact dermatitis , 309, 310 conditions description , 307 adverse effects , 311 phototoxic reaction , 309 description , 309 Ashy dermatosis. See Erythema evaluation process , 310 dyschromicum perstans
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