Evaluating ECG Results and Screening for ADHD
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Giant Right Atrium Or Ebstein Anomaly? Hacer Kamalı1, Abdullah Erdem1*, Cengiz Erol2 and Atıf Akçevin3
ISSN: 2378-2951 Kamalı et al. Int J Clin Cardiol 2017, 4:104 DOI: 10.23937/2378-2951/1410104 Volume 4 | Issue 4 International Journal of Open Access Clinical Cardiology CASE REPORT Giant Right Atrium or Ebstein Anomaly? Hacer Kamalı1, Abdullah Erdem1*, Cengiz Erol2 and Atıf Akçevin3 1Department of Pediatric Cardiology, Istanbul Medipol University, Turkey 2Department of Radiology, Istanbul Medipol University, Turkey 3Department of Cardiovascular Surgery, Istanbul Medipol University, Turkey *Corresponding author: Abdullah Erdem, Associate Professor of Pediatric Cardiology, Department of Pediatric Cardiology, Istanbul Medipol University, Istanbul, Turkey, Tel: +905-0577-05805, Fax: +90212467064, E-mail: drabdullaherdem@ hotmail.com partment for exercise intolerance. She had been diag- Abstract nosed and followed up as Ebstein anomaly for ten years. Many acquired and congenital pathologies could cause en- Her heart rate was 75/min with normal jugular venous largement of Right Atrium (RA). In rare pathologies we couldn’t find an obvious reason for enlargement of RA. Sometimes it pressure. Findings of lung examination were normal. leads to misdiagnosis if we try to explain an unknown pa- Auscultation of heart revealed a 2/6 systolic murmur on thology with well known pathology. In this article we de- the lower left edge of the sternum. On abdominal ex- scribe a woman who presented with giant enlargement of amination, there was no organomegaly. Arterial Oxygen the RA misdiagnosed as Ebstein anomaly. Cardiac MRI is highly helpful for differential diagnosis of these kinds of atrial Saturation (SpO2) was 98%. Electrocardiography (ECG) pathologies. Although it is rare, giant RA should be consid- revealed absence of atrial fibrillation, the rhythm was ered in differential diagnosis of huge RA without an obvious sinus but there was right axis deviation and right bundle cause. -
179 the Pre-Operative Assessment of Acyanotic Pediatric Patients
179 ORIGINAL ARTICLE Th e Pre-operative Assessment of Acyanotic Pediatric Patients Presented with Heart Murmur and Role of Surgry in congenital heart diseases, A retrospective analysis Dhafer O Alqahtani, Ali A. Alakfash, Omar R .Altamim Abstract Objectives: Th e aim of this study is to evaluate the incidence of congenital heart disease in patients referred solely because of heart murmur in pediatric age group and to assess the rule of medical and surgical management in patient with heart defects. Study design: It is retrospective analysis of all paediatric cases who presented with cardiac murmur. Materials and Methods:A retrospective database and echocardiographic review. All patients referred to King Abdulaziz Cardiac Center (KA CC) Riyadh, Kingdom of Saudi Arabia dur- ing the period from July 2007 till March 2009 for cardiovascular evaluation because of heart murmur detected during routine physical exam. We included any pediatric patient from the neonatal period till 14 years of age who had echocardiography in our center. Any patient with cyanosis, those with diff erence in the blood pressure between the upper limbs and lower limbs of more than 15 mmHg, preterm neonates, any acquired heart disease and syndromic and critically ill patients were excluded from the study. Results: A total of 245 patients met the inclusion criteria. Median age and weight is 7 months (one day – 12 years), 7.85 Kg (1.9 – 54 Kg) respectively. Normal echocardiography was pres- ent in 163 patients (66.5%). Th e most encountered anomaly found was patent ductus arte- riosious (PDA) which was diagnosed in 27 patients (11 %) followed by atrial septal defect (ASD) secundum in 26 patients (10.6%), then the VSD in 22 patients (9%), atrio-ventricular septal defect (AVSD) in 1 patient (0.4%), Coarctation of Aorta in 3 patients (1.2%), Tortuous of arch in 1 patients (0.4%), Pulmonary stenosis in 10 patients (4%), Mitral valve prolapse in 4 patients (1.6%) and the false tendon in 6 patients (2.4 %). -
Clinical Manifestation and Survival of Patients with I Diopathic Bilateral
ORIGINAL ARTICLE Clinical Manifestation and Survival of Patients with Mizuhiro Arima, TatsujiI diopathicKanoh, Shinya BilateralOkazaki, YoshitakaAtrialIwama,DilatationAkira Yamasaki and Sigeru Matsuda Westudied the histories of eight patients who lacked clear evidence of cardiac abnormalities other than marked bilateral atrial dilatation and atrial fibrillation, which have rarely been dis- cussed in the literature. From the time of their first visit to our hospital, the patients' chest radio- graphs and electrocardiograms showed markedly enlarged cardiac silhouettes and atrial fibrilla- tion, respectively. Each patient's echocardiogram showed a marked bilateral atrial dilatation with almost normal wall motion of both ventricles. In one patient, inflammatory change was demonstrated by cardiac catheterization and endomyocardial biopsy from the right ventricle. Seven of our eight cases were elderly women.Over a long period after the diagnosis of cardiome- galy or arrhythmia, diuretics or digitalis offered good results in the treatment of edema and congestion in these patients. In view of the clinical courses included in the present study, we conclude that this disorder has a good prognosis. (Internal Medicine 38: 112-118, 1999) Key words: cardiomegaly, atrial fibrillation, elder women,good prognosis Introduction echocardiography. The severity of mitral and tricuspid regur- gitation was globally assessed by dividing into three equal parts Idiopathic enlargement of the right atrium was discussed by the distance from the valve orifice. The regurgitant jet was de- Bailey in 1955(1). This disorder may be an unusual congenital tected on color Doppler recording in the four-chamber view malformation. A review of the international literature disclosed and classified into one of the three regions (-: none, +: mild, that although several cases have been discussed since Bailey's ++:moderate, +++: severe). -
Young Adults. Look for ST Elevation, Tall QRS Voltage, "Fishhook" Deformity at the J Point, and Prominent T Waves
EKG Abnormalities I. Early repolarization abnormality: A. A normal variant. Early repolarization is most often seen in healthy young adults. Look for ST elevation, tall QRS voltage, "fishhook" deformity at the J point, and prominent T waves. ST segment elevation is maximal in leads with tallest R waves. Note high take off of the ST segment in leads V4-6; the ST elevation in V2-3 is generally seen in most normal ECG's; the ST elevation in V2- 6 is concave upwards, another characteristic of this normal variant. Characteristics’ of early repolarization • notching or slurring of the terminal portion of the QRS wave • symmetric concordant T waves of large amplitude • relative temporal stability • most commonly presents in the precordial leads but often associated with it is less pronounced ST segment elevation in the limb leads To differentiate from anterior MI • the initial part of the ST segment is usually flat or convex upward in AMI • reciprocal ST depression may be present in AMI but not in early repolarization • ST segments in early repolarization are usually <2 mm (but have been reported up to 4 mm) To differentiate from pericarditis • the ST changes are more widespread in pericarditis • the T wave is normal in pericarditis • the ratio of the degree of ST elevation (measured using the PR segment as the baseline) to the height of the T wave is greater than 0.25 in V6 in pericarditis. 1 II. Acute Pericarditis: Stage 1 Pericarditis Changes A. Timing 1. Onset: Day 2-3 2. Duration: Up to 2 weeks B. Findings 1. -
Subclinical Subaortic Stenosis in a Golden Retriever
CASE ROUTES h CARDIOLOGY h PEER REVIEWED Subclinical Subaortic Stenosis in a Golden Retriever Kursten Pierce, DVM, DACVIM (Cardiology) Colorado State University THE CASE THE CASE A 12-month-old intact female golden retriever is pre- Diagnostic investigation of the heart murmur via echo- sented for a wellness examination and to discuss the cardiography is discussed with the owner but declined pros and cons of breeding the patient versus pursuing due to the patient’s lack of clinical signs and the costs ovariohysterectomy. The owner would like her to pro- associated with additional testing. duce one litter of puppies prior to being spayed. What are the next steps? On physical examination, the patient is bright, alert, and responsive. She is extremely energetic with a good THE CHOICE IS YOURS … BCS (4/9) and appropriate musculature. Cardiovascu- CASE ROUTE 1 lar examination reveals pink mucous membranes, no To provide information on breeding and caring for a obvious jugular venous distension, and a normal heart pregnant bitch and neonatal puppies and plan to spay rate and rhythm with normal synchronous femoral the patient after the puppies have been weaned, go to pulses. Auscultation is difficult and brief because the page 28. patient is rambunctious and panting. Despite the pant- ing, she is eupneic with clear bronchovesicular sounds. CASE ROUTE 2 A grade II/VI left basilar systolic heart murmur is aus- To avoid providing additional recommendations cultated. A murmur had not previously been docu- regarding breeding and ovariohysterectomy to the mented at her puppy wellness visits. The owner has not owner until a diagnostic investigation with a cardiolo- observed any coughing, trouble breathing, exercise gist has been pursued, go to page 32. -
EKG Workshop Texas Academy of Physician Assistants 2018 TAPA 43Rd Annual CME Conference
EKG Workshop Texas Academy of Physician Assistants 2018 TAPA 43rd Annual CME Conference David J. Klocko, MPAS, PA-C Associate Professor and Academic Coordinator UT Southwestern Department of PA Studies Disclosures Content in this presentation is licensed under a Creative Commons Attribution 4.0 International license. David J. Klocko is receiving a stipend from TAPA to do this workshop. Objectives Review basic electrophysiology. Recognize common cardiac dysrhythmias. Recognize common conductive disorders. Review axis, bundle branch block, chamber enlargement and hypertrophy. Recognize common ECG changes associated with myocardial ischemia and infarction. Systematic Evaluation of the 12- lead EKG –Our ultimate goal ! Step Check for: LOOK AT: 1. Rhythm Rhythm Strip 2. Axis I, AvF ** 3. BBB V1, V6 4. Ischemia, Injury or I-AvL, V5-V6 Infarct V1-V4 II, III, AvF 5. Chamber II, V1 Enlargement Intervals & Segments PR interval (PRI): start of atrial depolarization to start of ventricular depolarization Normal: 0.12 – 0.20 sec QRS complex: depolarization of ventricles Normal: 0.04 – 0.11 sec When QRS >0.12 sec, think about an interventricular conduction delay Intervals & Segments ST segment: end ventricular depolarization to start of repolarization QT interval (QTI): both ventricular depolarization & repolarization Normal: 0.3 – 0.4 sec; varies with heartrate Faster heartrates….shorter QTIs; slower heartrates ….longer QTIs Heart Rate For regular rhythms, measure the interval between complexes in large boxes 300-150-100-75-60-50-43-37-33 -
Sudan's Guidelines for Diagnosis, Management and Prevention
Acute Rheumatic Fever and Rheumatic Heart Disease: Sudan’s Guidelines for Diagnosis, Management and Prevention 1 2 Sudan’s Federal Ministry of Health Sudan Heart Society-Working Group on Pediatric Cardiology Sudanese Association of Pediatricians Sudanese Children’s Heart Society Writing Committee: Sulafa Khalid M Ali, FRCPCH, FACC, Consultant Pediatric Cardiologist Professor-University of Khartoum Mohamed Saeed Al Khaleefa, FRCP, Consultant Cardiologist Professor-University of Al Zaem Al Azhari Siragedeen Mohamed Khair, MD, Consultant Pediatrician Professor- University of Al Zaem Al Azhari Second Edition Jan/2017 3 Contents Chapter Title Page Preface 5 Chapter 1 Rheumatic Heart Disease : General Considerations 6 Chapter 2 Diagnosis and Management of Streptococcal 11 Pharyngitis Chapter 3 Acute Rheumatic Fever 15 Chapter 4 Rheumatic Heart Disease 25 Chapter 5 Rheumatic Heart Disease in Pregnancy 49 Chapter 6 Acute Rheumatic Fever & Rheumatic Heart Disease 57 Control Appendices Rheumatic Heart Disease Protocols, Manuals, 63 Brochures and Educational Websites 4 Preface to the Second Edition: This is the second edition of Sudan’s Guidelines for acute rheumatic fever (ARF) and rheumatic heart disease (RHD) diagnosis, management and prevention. RHD is a devastating sequel of ARF, initiated by a simple throat infection with group A streptococcus in susceptible population. Eradication of RHD can be achieved by improvement of health care system as has been witnessed in developed countries. In many developing countries like Sudan, RHD is still prevalent causing significant mortality and premature cardiovascular death as well as an undesired burden on the health system. An RHD control program has been established in Sudan in 2012 aiming to increase the awareness of both the public and medical personnel, to introduce primary and consolidate secondary prevention and to strengthen the surveillance system. -
Changes in Cardiac Geometry Due to Hypertrophy
Changes in Cardiac Geometry Due to Hypertrophy By Marta Ellen Pedersen A Master’s Paper Submitted in Partial Fulfillment of The Requirements for the Degree of Master of Science in Clinical Exercise Physiology Dr. Joseph O’ Kroy Date University of Wisconsin-River Falls 2014 Introduction For any given body size, men have larger hearts than women, athletes have larger hearts than nonathletes, and often times, an enlarged heart is a symptom of an underlying disorder that is causing the heart to work harder than normal. This review will emphasize the differences between a pathologically enlarged heart and an athletically enlarged heart. Pathologically induced hypertrophy (myopathy) When heart cells get bigger, (often is the case when heart disease is present) the total heart works less efficiently. Some people suffer from conditions like hypertrophic cardiomyopathy, which includes significant heart muscle enlargement, and can be genetic or caused by high blood pressure. Cardiomyopathy decreases the size of the heart's chambers, reducing blood flow. Hypertrophy, or thickening, of the heart muscle can occur in response to increased stress on the heart. The most common causes of Cardiomyopathy are related to increased blood pressure. The extra work of pumping blood against the increased pressure causes the ventricle to thicken over time, the same way a body muscle increases in mass in response to weightlifting. Cardiomyopathy can occur in both the right and left atrium and the right and left ventricles. Blood travels through the right ventricle to the lungs. If conditions occur that decrease pulmonary circulation, extra stress can be placed on the right ventricle, and can lead to right ventricular myopathy. -
Problems in Family Practice Heart Murmurs in Infants and Children
Problems in Family Practice Heart Murmurs in Infants and Children Thomas A. Riemenschneider, MD Sacramento, California A system is presented for evaluation of heart murmurs in in fants and children. The system places emphasis on identifica tion of functional murmurs, which the physician encounters so frequently in daily practice. A three-part approach is presented which includes: (1) evaluation of cardiovascular status, (2) as sessment of the heart murmur, and (3) decision regarding the need for further evaluation. This approach relieves the physi cian of the necessity to remember the multiple details of the many congenital cardiac lesions, and requires only the knowl edge of a few easily remembered details about functional murmurs. The system enables the physician to confidently distinguish organic and functional murmurs and to decide which children need further evaluation and referral to the pediatric cardiologist. The physician who cares for infants, children, with “normal” murmurs for reassurance to the and adolescents will frequently encounter heart parents.2 Using his/her knowledge of the myriad murmurs during the course of a careful physical details of the many congenital cardiac malforma examination. It has been estimated that a heart tions, the pediatric cardiologist seeks evidence murmur may be heard at some time in almost that the murmur is due to an organic lesion. The every child.1 Murmurs may be classified as “func family physician cannot expect to retain all of tional” (physiologic, normal, benign, or innocent), these details, and therefore often feels in or “organic” (associated with an anatomic car adequately prepared to assess the child with a diovascular abnormality). -
Abnormal ECG Findings in Athletes Normal ECG
SEATTLE CRITERIA Abnormal ECG findings in athletes These ECG findings are unrelated to regular training or expected physiologic adaptation to exercise, may suggest the presence of pathologic cardiovascular disease, and require further diagnostic evaluation. Abnormal ECG finding Definition T wave inversion > 1 mm in depth in two or more leads V2-V6, II and aVF, or I and aVL (excludes III, aVR, and V1) ST segment depression ≥ 0.5 mm in depth in two or more leads Pathologic Q waves > 3 mm in depth or > 40 ms in duration in two or more leads (except III and aVR) Complete left bundle branch block QRS ≥ 120 ms, predominantly negative QRS complex in lead V1 (QS or rS), and upright monophasic R wave in leads I and V6 Intra-ventricular conduction delay Any QRS duration ≥ 140 ms Left axis deviation -30° to -90° Left atrial enlargement Prolonged P wave duration of > 120 ms in leads I or II with negative portion of the P wave ≥ 1 mm in depth and ≥ 40 ms in duration in lead V1 Right ventricular hypertrophy R-V1 + S-V5 > 10.5 mm and right axis deviation > 120° pattern Ventricular pre-excitation PR interval < 120 ms with a delta wave (slurred upstroke in the QRS complex) and wide QRS (> 120 ms) Long QT interval* QTc ≥ 470 ms (male) QTc ≥ 480 ms (female) QTc ≥ 500 ms (marked QT prolongation) Short QT interval* QTc ≤ 320 ms Brugada-like ECG pattern High take-off and downsloping ST segment elevation followed by a negative T wave in ≥ 2 leads in V1-V3 Profound sinus bradycardia < 30 BPM or sinus pauses ≥ 3 sec Mobitz type II 2° AV block Intermittently non-conducted P waves not preceded by PR prolongation and not followed by PR shortening 3° AV block Complete heart block Atrial tachyarrhythmias Supraventricular tachycardia, atrial fibrillation, atrial flutter Premature ventricular contractions ≥ 2 PVCs per 10 second tracing Ventricular arrhythmias Couplets, triplets, and non-sustained ventricular tachycardia *The QT interval corrected for heart rate is ideally measured with heart rates of 60-90 bpm. -
Accuracy of Electrocardiographic Criteria for Atrial Enlargement: Validation with Cardiovascular Magnetic Resonance
Accuracy of Electrocardiographic Criteria for Atrial Enlargement: Validation with Cardiovascular Magnetic Resonance The Harvard community has made this article openly available. Please share how this access benefits you. Your story matters Citation Tsao, Connie W., Mark E. Josephson, Thomas H. Hauser, T. David O'Halloran, Anupam Agarwal, Warren J. Manning, and Susan B. Yeon. 2008. Accuracy of electrocardiographic criteria for atrial enlargement: Validation with cardiovascular magnetic resonance. Journal of Cardiovascular Magnetic Resonance 10(1): 7. Published Version doi://10.1186/1532-429X-10-7 Citable link http://nrs.harvard.edu/urn-3:HUL.InstRepos:10140325 Terms of Use This article was downloaded from Harvard University’s DASH repository, and is made available under the terms and conditions applicable to Other Posted Material, as set forth at http:// nrs.harvard.edu/urn-3:HUL.InstRepos:dash.current.terms-of- use#LAA Journal of Cardiovascular Magnetic Resonance BioMed Central Research Open Access Accuracy of electrocardiographic criteria for atrial enlargement: validation with cardiovascular magnetic resonance Connie W Tsao1, Mark E Josephson1, Thomas H Hauser1, T David O'Halloran1, Anupam Agarwal1,2, Warren J Manning1,3 and Susan B Yeon*1 Address: 1Harvard-Thorndike Laboratory and the Department of Medicine, Cardiovascular Division, Beth Israel Deaconess Medical Center and Harvard Medical School, 330 Brookline Avenue, Boston, Massachusetts, USA, 2Cardiovascular and Metabolic Division, GlaxoSmithKline Pharmaceuticals, 1250 Collegeville -
Surgical Treatment of Idiopathic Enlargement of the Right Atrium
Hindawi Case Reports in Surgery Volume 2018, Article ID 7241309, 5 pages https://doi.org/10.1155/2018/7241309 Case Report Surgical Treatment of Idiopathic Enlargement of the Right Atrium Francesca Chiara Surace, Federica Iezzi , Massimo Colaneri, and Marco Pozzi Department of Paediatric and Congenital Cardiac Surgery and Cardiology, Azienda Ospedaliero Universitaria, Ospedali Riuniti Ancona “Umberto I-G. M. Lancisi-G. Salesi”, Ancona, Italy Correspondence should be addressed to Federica Iezzi; [email protected] Received 25 June 2018; Accepted 26 August 2018; Published 20 September 2018 Academic Editor: Shuli Silberman Copyright © 2018 Francesca Chiara Surace et al. This is an open access article distributed under the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited. Idiopathic enlargement of the right atrium (IERA) is a very rare abnormality. Approximately one-half (48%) of the patients with a congenital enlargement of the right atrium have no symptoms. When they occur, symptoms include shortness of breath (28% of cases), palpitations (17%), arrhythmias (12%), and in rare cases, right heart failure and extreme tiredness. We report one such case of a young man with a disproportionally enlarged right atrium. The basal transthoracic echocardiogram demonstrated a huge right atrium with a thick smoke pattern and mild tricuspid regurgitation in the absence of congenital heart disease. Magnetic resonance imaging confirmed the right atriomegaly, with initial compression of the right ventricle, and excluded congenital heart defects or absence of pericardium. The patient underwent surgical resection of the right atrial wall and the atriotomy was closed, leaving an atrial chamber of normal consistency and size.