Musculoskeletal Signs

Total Page:16

File Type:pdf, Size:1020Kb

Musculoskeletal Signs CHAPTER 1 MUSCULOSKELETAL SIGNS 1 Anterior drawer test Anterior drawer test Mechanism/s The ACL arises from the anterior aspect of the tibial plateau and inserts into the medial aspect of the lateral femoral condyle. It limits anterior movement of the tibia upon the femur. Loss of continuity of the ACL permits 90º inappropriate anterior movement of the tibia and thus knee joint instability. Sign value A literature review of six studies reported variable sensitivity of 27–88%, specificity of 91–99%, FIGURE 1.1 positive LR of 11.5 and negative LR of Anterior drawer test for anterior cruciate 2 ligament injury 0.5. A literature review by Solomon DH et al. of nine studies reported a sensitivity of 9–93% and specificity of 23–100%.1 Description While a positive anterior drawer With the patient lying supine, the knee sign (+LR 11.5)2 has been suggested to at 90° flexion and the foot immobilised be strong evidence of ACL injury, the by the examiner, the proximal third results are not uniform, with another of the tibia is pulled towards the study reporting a +LR 2.0 (sensitivity examiner. In a positive test, there is 83%, specificity 57%, –LR 0.3).3 A anterior (forward) movement of the negative anterior drawer sign cannot tibia without an abrupt stop.1 reliably exclude ACL injury (sensitivity Condition/s 27–88%; –LR 0.5).2 When strong clinical suspicion persists, further associated with diagnostic steps are necessary (e.g. • Anterior cruciate ligament (ACL) interval re-examination, MRI, injury arthroscopy). CLINICAL PEARL 2 Apley’s grind test Apley’s grind test 1 is increased rotation relative to the unaffected side, this is suggestive of a ligamentous lesion. If rotation plus compression is more painful or there is decreased rotation relative to the unaffected side, this is suggestive of meniscal injury.4 Condition/s associated with • Meniscal injury Mechanism/s FIGURE 1.2 Direct mechanical force upon the Apley’s grind test injured meniscus elicits tenderness. Sign value A review by Hegedus EJ et al. reported Description a pooled sensitivity of 60.7% and With the patient lying prone and the specificity of 70.2% with an odds ratio knee at 90° flexion, the lower leg is of 3.4.5 Significant heterogeneity in passively internally and externally the data limits its accuracy. Overall, rotated while axial pressure is applied Apley’s grind test has limited diagnostic to the lower leg. The test is considered utility, limited supporting data and, positive if tenderness is elicited. in the acute setting, the manoeuvre The process can also be combined produces severe pain.6 with or without distraction. If rotation McMurray’s grind test has more plus distraction is more painful or there robust supporting data. 3 Apley’s scratch test Apley’s scratch test Condition/s associated with Common • Rotator cuff muscle injury • Labral tear • Anterior shoulder dislocation • Bicipital tendonitis • Adhesive capsulitis (frozen shoulder) • Acromioclavicular joint injury Mechanism/s The shoulder joint is a complex structure. Its components include the humeral head, glenoid fossa, acromion, clavicle, scapula and surrounding soft FIGURE 1.3 tissue structures. Under normal One of three manoeuvres of Apley’s circumstances the shoulder joint is scratch test capable of a vast range of movement. Based on Woodward T, Best TM. The painful Apley’s scratch test assesses shoulder: part 1, clinical evaluation. Am Fam Phys 2000; 61(10): 3079–3088. glenohumeral abduction, adduction, flexion, extension, internal rotation and external rotation. Tenderness or limited range of movement suggests injury to Description one or more components of the Apley’s scratch test is a general range shoulder joint. of movement assessment of the shoulder joint (i.e. glenohumeral, Sign value acromioclavicular, sternoclavicular and Apley’s scratch test is a useful scapulothoracic joints). The patient is component of the general shoulder instructed to touch the unaffected exam but has limited utility for a shoulder anteriorly and posteriorly specific diagnosis. The position of the (behind their head), and touch the shoulder at which tenderness or limited inferior scapula posteriorly (behind range of movement occurs should be their back). Tenderness and/or limited noted. In the patient with an abnormal range of movement while performing Apley’s scratch test, further diagnostic these movements is considered an manoeuvres should be performed to abnormal test.7 narrow the differential diagnosis. 4 Apparent leg length inequality (functional leg length) Apparent leg length 1 inequality (functional leg length) ABC FIGURE 1.4 Measurement of leg lengths A The apparent leg length is the distance from the umbilicus to the medial malleolus; B pelvic rotation causing an apparent leg length discrepancy; C the true leg length is the distance from the anterior superior iliac spine to the medial malleolus. Based on Firestein GS, Budd RC, Harris ED et al., Kelley’s Textbook of Rheumatology, 8th edn, Philadelphia: WB Saunders, 2008: Fig 42-24. Description Ligamentous laxity A disparity between the relative The ligaments on one side (e.g. in the distance from the umbilicus to the hip joint) may be more flexible or medial malleolus of each leg.8 By longer than their counterparts, making definition it implies asymmetry of the the femur sit lower in the joint capsule. lower extremities in the absence of a Joint contracture bony abnormality. (See ‘True leg A joint contracture impairs full range length inequality’ in this chapter.) of movement. If the knee joint is contracted in a flexed position, the Condition/s length of the affected side will be less associated with than the opposite leg during maximal • Altered foot mechanics attempted extension. • Adaptive shortening of soft tissues Altered foot mechanics • Joint contractures Excessive pronation of the foot eventuates in and/or may be • Ligamentous laxity accompanied by a decreased arch • Axial malalignments height compared to the ‘normal’ foot, resulting in a functionally shorter Mechanism/s limb.8 An apparent or functional leg length inequality may occur at any point from the pelvis to the foot.8 5 Apparent leg length inequality (functional leg length) Sign value significant effect is controversial.8 The test should be interpreted in relation The distance (anywhere from to the patient’s history and full gait 3–22 mm) at which apparent leg length assessment. inequality results in a clinically 6 Apprehension test Apprehension test 1 • Rotator cuff muscle injury • Glenoid labrum injury • Glenoid defect (e.g. Bankart’s fracture) • Humeral head defect (e.g. Hill– Sachs fracture) Less common – atraumatic • Connective tissue disorder: Ehlers–Danlos syndrome, Marfan’s syndrome FIGURE 1.5 • Congenital absence of glenoid Apprehension test The arm is abducted and placed in an Mechanism/s externally rotated position. Note the right Glenohumeral joint instability is caused arm of the examiner is providing anterior traction on the humerus, pulling the by dysfunction of the bony and/or soft posterior part of the humeral head forward. tissue structures that maintain joint The same test can be done from the back, stability: glenoid, humeral head, joint with the patient sitting up and the examiner capsule, capsuloligamentous or pushing forward on the posterior head of glenohumeral ligaments, labrum, and the humerus. rotator cuff muscles. The shoulder joint is susceptible to instability due to its inherent mobility and complex soft Description tissue structures responsible for stability. The apprehension test is an assessment In the apprehension test, the joint is of glenohumeral joint instability. With placed into a position vulnerable to the patient sitting or lying supine, the instability. It is the typical position shoulder is placed into 90° abduction, precipitating traumatic anterior 90° external rotation and 90° elbow shoulder dislocation. For this reason, a flexion. The examiner applies pressure significant number of healthy patients to the posterior aspect of the proximal will experience apprehension during humerus and attempts to move the this manoeuvre. humeral head anteriorly (see Figure 1.5). The test is positive if the Sign value patient experiences apprehension due to T’Jonck L et al. reported a sensitivity impending subluxation or dislocation of 88.0%, specificity of 50%, positive of the glenohumeral joint.9 likelihood ratio of 1.8 and negative likelihood ratio of 0.23.10 Condition/s The apprehension test for associated with glenohumeral joint instability is a moderately useful screening test. Based More common on available data, the test has limited – traumatic utility to rule in the diagnosis. It is not • Recurrent glenohumeral joint used in the setting of acute anterior subluxation or dislocation shoulder dislocation. 7 Apprehension–relocation test (Fowler’s sign) Apprehension–relocation test (Fowler’s sign) • Glenoid labrum injury • Glenoid defect (e.g. Bankart’s fracture) • Humeral head defect (e.g. Hill– Sachs fracture) Less common – atraumatic • Connective tissue disorder: FIGURE 1.6 Ehlers–Danlos syndrome, Marfan’s Apprehension–relocation (Fowler) test syndrome Note that pressure is applied anteriorly to the proximal humerus. • Congenital absence of glenoid Description Mechanism/s The apprehension–relocation test is an The underlying anatomy and causes of assessment of glenohumeral joint glenohumeral joint instability are instability. The relocation manoeuvre outlined under ‘Apprehension test’ and is typically performed following the apply here. In the apprehension– apprehension test (see ‘Apprehension relocation test, symptomatic relief is test’). With the patient sitting or lying due to restoration of the normal supine, the shoulder is placed into 90° anatomical relationship of the humeral abduction, 90° external rotation and head in the glenohumeral joint. 90° elbow flexion. The examiner applies pressure to the anterior aspect Sign value of the proximal humerus and attempts T’Jonck L et al. reported a sensitivity to move the humeral head posteriorly. of 85%, specificity of 87%, positive The test is positive if the patient likelihood ratio of 6.5 and negative 10 experiences relief of apprehension (i.e.
Recommended publications
  • Effusion =S Fluid in Pleural Space (Outside of Lung) Fremitus - Pathophysiology • Fremitus: – Increased W/Consolidation (E.G
    General Part Head and Neck Cardiovascular Abdomen Lung Muscles Lung Exam • Includes Vital Signs & Cardiac Exam • 4 Elements (cardiac & abdominal too) – Observation – Palpation – Percussion – Auscultation Pulmonary Review of Systems • All organ systems have an ROS • Questions to uncover problems in area • Need to know right questions & what the responses might mean! Exposure Is Key – You Cant Examine What You Can’t See! Anatomy Of The Spine Cervical: 7 Vertebrae Thoracic: 12 Vertebrae Lumbar: 5 Vertebrae Sacrum: 5 Fused Vertebrae Note gentle curve ea segment Hammer & Nails icon indicates A Slide Describing Skills You Should Perform In Lab Spine Exam As Relates to the Thorax • W/patient standing, observe: – shape of spine. – Stand behind patient, bend @ waist – w/Scoliosis (curvature) one shoulder appears “higher” Pathologic Changes In Shape Of Spine – Can Affect Lung Function Scoliosis (curved to one side) Thoracic Kyphosis (bent forward) Observation • ? Ambulates w/out breathing difficulty? • Readily audible noises (e.g. wheezing)? • Appearance →? sitting up, leaning forward, inability to speak, pursed lips → significant compromise • ? Use of accessory muscles of neck (sternocleidomastoids, scalenes), inter-costals → significant compromise / Make Note of Chest Shape: Changes Can Give Insight into underlying Pathology Barrel Chested (hyperinflation secondary to emphysema) Examine Nails/Fingers: Sometimes Provides Clues to Pulmonary Disorders Cyanosis Nicotine Staining Clubbing Assorted other hand and arm abnormalities: Shape, color, deformity
    [Show full text]
  • ICD-10 Diagnoses on Router
    L ARTHRITIS R L HAND R L ANKLE R L FRACTURES R OSTEOARTHRITIS: PRIMARY, 2°, POST TRAUMA, POST _____ CONTUSION ACHILLES TEN DYSFUNCTION/TENDINITIS/RUPTURE FLXR TEN CLAVICLE: STERNAL END, SHAFT, ACROMIAL END CRYSTALLINE ARTHRITIS: GOUT: IDIOPATHIC, LEAD, CRUSH INJURY AMPUTATION TRAUMATIC LEVEL SCAPULA: ACROMION, BODY, CORACOID, GLENOID DRUG, RENAL, OTHER DUPUYTREN’S CONTUSION PROXIMAL HUMERUS: SURGICAL NECK 2 PART 3 PART 4 PART CRYSTALLINE ARTHRITIS: PSEUDOGOUT: HYDROXY LACERATION: DESCRIBE STRUCTURE CRUSH INJURY PROXIMAL HUMERUS: GREATER TUBEROSITY, LESSER TUBEROSITY DEP DIS, CHONDROCALCINOSIS LIGAMENT DISORDERS EFFUSION HUMERAL SHAFT INFLAMMATORY: RA: SEROPOSITIVE, SERONEGATIVE, JUVENILE OSTEOARTHRITIS PRIMARY/SECONDARY TYPE _____ LOOSE BODY HUMERUS DISTAL: SUPRACONDYLAR INTERCONDYLAR REACTIVE: SECONDARY TO: INFECTION ELSEWHERE, EXTENSION OR NONE INTESTINAL BYPASS, POST DYSENTERIC, POST IMMUNIZATION PAIN OCD TALUS HUMERUS DISTAL: TRANSCONDYLAR NEUROPATHIC CHARCOT SPRAIN HAND: JOINT? OSTEOARTHRITIS PRIMARY/SECONDARY TYPE _____ HUMERUS DISTAL: EPICONDYLE LATERAL OR MEDIAL AVULSION INFECT: PYOGENIC: STAPH, STREP, PNEUMO, OTHER BACT TENDON RUPTURES: EXTENSOR OR FLEXOR PAIN HUMERUS DISTAL: CONDYLE MEDIAL OR LATERAL INFECTIOUS: NONPYOGENIC: LYME, GONOCOCCAL, TB TENOSYNOVITIS SPRAIN, ANKLE, CALCANEOFIBULAR ELBOW: RADIUS: HEAD NECK OSTEONECROSIS: IDIOPATHIC, DRUG INDUCED, SPRAIN, ANKLE, DELTOID POST TRAUMATIC, OTHER CAUSE SPRAIN, ANKLE, TIB-FIB LIGAMENT (HIGH ANKLE) ELBOW: OLECRANON WITH OR WITHOUT INTRA ARTICULAR EXTENSION SUBLUXATION OF ANKLE,
    [Show full text]
  • Brachial-Plexopathy.Pdf
    Brachial Plexopathy, an overview Learning Objectives: The brachial plexus is the network of nerves that originate from cervical and upper thoracic nerve roots and eventually terminate as the named nerves that innervate the muscles and skin of the arm. Brachial plexopathies are not common in most practices, but a detailed knowledge of this plexus is important for distinguishing between brachial plexopathies, radiculopathies and mononeuropathies. It is impossible to write a paper on brachial plexopathies without addressing cervical radiculopathies and root avulsions as well. In this paper will review brachial plexus anatomy, clinical features of brachial plexopathies, differential diagnosis, specific nerve conduction techniques, appropriate protocols and case studies. The reader will gain insight to this uncommon nerve problem as well as the importance of the nerve conduction studies used to confirm the diagnosis of plexopathies. Anatomy of the Brachial Plexus: To assess the brachial plexus by localizing the lesion at the correct level, as well as the severity of the injury requires knowledge of the anatomy. An injury involves any condition that impairs the function of the brachial plexus. The plexus is derived of five roots, three trunks, two divisions, three cords, and five branches/nerves. Spinal roots join to form the spinal nerve. There are dorsal and ventral roots that emerge and carry motor and sensory fibers. Motor (efferent) carries messages from the brain and spinal cord to the peripheral nerves. This Dorsal Root Sensory (afferent) carries messages from the peripheral to the Ganglion is why spinal cord or both. A small ganglion containing cell bodies of sensory NCS’s sensory fibers lies on each posterior root.
    [Show full text]
  • Cervical Spine Injuries
    Essential Sports Medicine Essential Sports Medicine Joseph E. Herrera Editor Department of Rehabilitation Medicine, Interventional Spine and Sports, Mount Sinai Medical Center New York, NY Grant Cooper Editor New York Presbyterian Hospital New York, NY Editors Joseph E. Herrera Grant Cooper Department of Rehabilitation Medicine New York Presbyterian Hospital Interventional Spine & Sports New York, NY Mount Sinai Medical Center New York, NY Series Editors Grant Cooper Joseph E. Herrera New York Presbyterian Hospital Department of Rehabilitation Medicine New York, NY Interventional Spine and Sports Mount Sinai Medical Center New York, NY ISBN: 978-1-58829-985-7 e-ISBN: 978-1-59745-414-8 DOI: 10.1007/978-1-59745-414-8 Library of Congress Control Number © 2008 Humana Press, a part of Springer Science + Business Media, LLC All rights reserved. This work may not be translated or copied in whole or in part without the written permission of the publisher (Humana Press, 999 Riverview Drive, Suite 208, Totowa, NJ 07512 USA), except for brief excerpts in connection with reviews or scholarly analysis. Use in connection with any form of information storage and retrieval, electronic adaptation, computer software, or by similar or dissimilar methodology now known or hereafter developed is forbidden. The use in this publication of trade names, trademarks, service marks, and similar terms, even if they are not identified as such, is not to be taken as an expression of opinion as to whether or not they are subject to proprietary rights. While the advice and information in this book are believed to be true and accurate at the date of going to press, neither the authors nor the editors nor the publisher can accept any legal responsibility for any errors or omissions that may be made.
    [Show full text]
  • Piriformis Syndrome Is Overdiagnosed 11 Robert A
    American Association of Neuromuscular & Electrodiagnostic Medicine AANEM CROSSFIRE: CONTROVERSIES IN NEUROMUSCULAR AND ELECTRODIAGNOSTIC MEDICINE Loren M. Fishman, MD, B.Phil Robert A.Werner, MD, MS Scott J. Primack, DO Willam S. Pease, MD Ernest W. Johnson, MD Lawrence R. Robinson, MD 2005 AANEM COURSE F AANEM 52ND Annual Scientific Meeting Monterey, California CROSSFIRE: Controversies in Neuromuscular and Electrodiagnostic Medicine Loren M. Fishman, MD, B.Phil Robert A.Werner, MD, MS Scott J. Primack, DO Willam S. Pease, MD Ernest W. Johnson, MD Lawrence R. Robinson, MD 2005 COURSE F AANEM 52nd Annual Scientific Meeting Monterey, California AANEM Copyright © September 2005 American Association of Neuromuscular & Electrodiagnostic Medicine 421 First Avenue SW, Suite 300 East Rochester, MN 55902 PRINTED BY JOHNSON PRINTING COMPANY, INC. ii CROSSFIRE: Controversies in Neuromuscular and Electrodiagnostic Medicine Faculty Loren M. Fishman, MD, B.Phil Scott J. Primack, DO Assistant Clinical Professor Co-director Department of Physical Medicine and Rehabilitation Colorado Rehabilitation and Occupational Medicine Columbia College of Physicians and Surgeons Denver, Colorado New York City, New York Dr. Primack completed his residency at the Rehabilitation Institute of Dr. Fishman is a specialist in low back pain and sciatica, electrodiagnosis, Chicago in 1992. He then spent 6 months with Dr. Larry Mack at the functional assessment, and cognitive rehabilitation. Over the last 20 years, University of Washington. Dr. Mack, in conjunction with the Shoulder he has lectured frequently and contributed over 55 publications. His most and Elbow Service at the University of Washington, performed some of the recent work, Relief is in the Stretch: End Back Pain Through Yoga, and the original research utilizing musculoskeletal ultrasound in order to diagnose earlier book, Back Talk, both written with Carol Ardman, were published shoulder pathology.
    [Show full text]
  • Disorders of the Knee
    DisordersDisorders ofof thethe KneeKnee PainPain Swelling,Swelling, effusioneffusion oror hemarthrosishemarthrosis LimitedLimited jointjoint motionmotion Screw home mechanism – pain, stiffness, fluid, muscular weakness, locking InstabilityInstability – giving way, laxity DeformityDeformity References: 1. Canale ST. Campbell’s operative orthopaedics. 10th edition 2003 Mosby, Inc. 2. Netter FH. The Netter collection of Medical illustrations – musculoskeletal system, Part I & II. 1997 Novartis Pharmaceuticals Corporation. 3. Magee DJ. Orthopedic Physical assessment. 2nd edition 1992 W. B. Saunders Company. 4. Hoppenfeld S. Physical examination of the spine and extremities. 1976 Appleton-century-crofts. AnteriorAnterior CruciateCruciate LigamentLigament Tibial insertion – broad, irregular, diamond-shaped area located directly in front of the intercondylar eminence Femoral attachment Femoral attachment Figure 43-24 In addition to their – semicircular area on the posteromedial synergistic functions, cruciate aspect of the lateral condyle and collateral ligaments exercise 33 mm in length basic antagonistic function 11 mm in diameter during rotation. A, In external Anteromedial bundle — tight in flexion rotation it is collateral ligaments that tighten and inhibit excessive Posterolateral bundle — tight in extension rotation by becoming crossed in 90% type I collagen space. B, In neutral rotation none 10% type III collagen of the four ligaments is under unusual tension. C, In internal Middle geniculate artery rotation collateral ligaments Fat
    [Show full text]
  • Meniscus Injury
    Introduction Role of menisci • Medial meniscus lesions are more common than 01 lateral meniscus because it is attached to the improving articular capsule that make it less mobile thus it cannot congruency and increasing easily to accommodate the abnormal stresses. the stability of the knee • In increasing age – gradual degeneration and change in the material properties of the menisci Meniscus controlling the complex thus splits and tears are more likely that usually associated with osteoarthritic articular damage or rolling and gliding actions of chondrocalcinosis. Injury the joint • In younger people - meniscal tears are usually the result of trauma, with a specific injury identified in distributing load during the history. movement Tear of Meniscus Pathology Pathology • Usually, meniscus more likely to tear along its Vertical tear Horizontal tear length than across its width because the Bucket-handle tear usually ‘degenerative’ or due to repetitive minor trauma meniscus consists mainly of circumferential the separated fragment remains attached front complex with the tear pattern lying in many collagen fibres held by a few radial strands. and back planes The torn portion can sometimes displace towards may be displaced or likely to displace • The meniscus is usually torn by a twisting the centre of the joint and becomes jammed If the loose piece of meniscus can be displaced, it between femur and tibia acts as a mechanical irritant, giving rise to force with the knee bent and taking weight. This causes a block to movement with the patient recurrent synovial effusion and mechanical describing a ‘locked knee’ symptoms • In middle life, tears can occur with relatively posterior or anterior horn tears Some are associated with meniscal cysts little force when fibrotic change has the very back or front of the meniscus is It is also suggested that synovial cells infiltrate into the vascular area between meniscus and restricted mobility of the meniscus.
    [Show full text]
  • Knee Examination (ACL Tear) (Please Tick)
    Year 4 Formative OSCE (September) 2018 Station 3 Year 4 Formative OSCE (September) 2018 Reading for Station 3 Candidate Instructions Clinical Scenario You are an ED intern at the Gold Coast University Hospital. Alex Jones, 20-years-old, was brought into the hospital by ambulance. Alex presents with knee pain following an injury playing soccer a few hours ago. Alex has already been sent for an X-ray. The registrar has asked you to examine Alex. Task In the first six (6) minutes: • Perform an appropriate physical examination of Alex and explain what you are doing to the registrar as you go. In the last two (2) minutes, you will be given Alex’s X-ray and will be prompted to: • Interpret the radiograph • Provide a provisional diagnosis to the registrar • Provide a management plan to the registrar You do not need to take a history. The examiner will assume the role of the registrar. Year 4 Formative OSCE (September) 2018 Station 3 Simulated Patient Information The candidate has the following scenario and task Clinical Scenario You are an ED intern at the Gold Coast University Hospital. Alex Jones, 20-years-old, was brought into the hospital by ambulance. Alex presents with knee pain following an injury playing soccer a few hours ago. Alex has already been sent for an X-ray. The registrar has asked you to examine Alex. Task In the first six (6) minutes: • Perform an appropriate physical examination of Alex and explain what you are doing to the registrar as you go. In the last two (2) minutes, you will be given Alex’s X-ray and will be prompted to: • Interpret the radiograph • Provide a provisional diagnosis to the registrar • Provide a management plan to the registrar You do not need to take a history.
    [Show full text]
  • Comparison of the Thesslay Test and Mcmurray Test: a Systematic
    py & Ph ra ys e i th c Alexanders et al.,Physiother Rehabil 2016, 1:1 a io l s R y e Journal of DOI: 10.4172/2573-0312.1000104 h h a P b f i o l i l t a ISSN:a 2573-0312 t n i r o u n o J Physiotherapy & Physical Rehabilitation Research Article Open Access Comparison of the Thesslay Test and McMurray Test: A Systematic Review of Comparative Diagnostic Studies Jenny Alexanders1*, Anna Anderson2, Sarah Henderson1 and Ulf Clausen3 1Sport, Health and Sciences Department, The University of Hull, Washburn Building, Cottingham Road, Hull, United Kingdom 2Leeds Teaching Hospitals, Beckett Street, Leeds, LS9 7TF, United Kingdom 3Dr Hill and Partners, Beverly Health Practice, Manor Road, Hull, HU17 7BZ, United Kingdom Abstract Background: The Thessaly test is a relatively recently developed meniscal test; therefore research compared to other meniscal tests is somewhat limited. In addition, a systematic review comparing the Thessaly’s test with a long standing test such as the McMurray test has not been previously conducted. Objective: To systematically identify and appraise all empirical studies comparing the diagnostic accuracy of the Thessaly test and McMurray test. Procedure: Eligible studies were identified through a rigorous search of ScienceDirect, CINAHL Plus, Pubmed, PEDro, EMBASE and Cochrane Library from January 2004 until August 2014. Full English reports of studies investigating the accuracy of the Thessaly test and McMurray test. Quality Assessment of Studies of Diagnostic Accuracy (QUADAS) scores were completed on each selected article. Results: The Thessaly test reported to have higher diagnostic accuracy values (61-96%) compared to the McMurray test (56-84%).
    [Show full text]
  • Physical Esxam
    Pearls in the Musculoskeletal Exam Frank Caruso MPS, PA-C, EMT-P Skin, Bones, Hearts & Private Parts 2019 Examination Key Points • Area that needs to be examined, gown your patients - well exposed • Understand normal functional anatomy • Observe normal activity • Palpation • Range of Motion • Strength/neuro-vascular assessment • Special Tests General Exam Musculoskeletal Overview Physical Exam Preview Watch Your Patients Walk!! Inspection • Posture – Erectness – Symmetry – Alignment • Skin and subcutaneous tissues – Swelling – Redness – Masses Inspection • Extremities – Size – Deformities – Enlargement – Alignment – Contour – Symmetry Inspection • Muscles – Bilateral symmetry – Hypertrophy – Atrophy – Fasciculations – Spasms Palpation • Palpate bones, joints, and surrounding muscles for the following: – Heat – Tenderness – Swelling – Fluctuation – Crepitus – Resistance to pressure – Muscle tone Muscles • Size and strength affected by the following: – Genetics – Exercise – Nutrition • Muscles move joints through range of motion (ROM). Muscle Strength • Compare bilateral muscles – Strength – Symmetry – Equality – Resistance End Feel Think About It!! • The sensation the examiner feels in the joint as it reaches the end of the range of motion of each passive movement • Bone to bone: This is hard, unyielding – normal would be elbow extension. • Soft–tissue approximation: yielding compression that stops further movement – elbow and knee flexion. End Feel • Tissue stretch: hard – springy type of movement with a slight give – toward the end of range of motion – most common type of normal end feel : knee extension and metacarpophalangeal joint extension. Abnormal End Feel • Muscle spasm: invoked by movement with a sudden dramatic arrest of movement often accompanied by pain - sudden hard – “vibrant twang” • Capsular: Similar to tissue stretch but it does not occur where one would expect – range of motion usually reduced.
    [Show full text]
  • Evaluation and Management of Sports Injuries in Children
    2019 Frontiers in Pediatrics Sports Medicine Mini-Symposium Presented by MUSC Health Sports Medicine Sports Medicine Panel of Experts Michael J. Barr, PT, DPT, MSR Sports Medicine Manager MUSC Health Sports Medicine Alec DeCastro, MD Assistant Professor CAQ Sports Medicine Director, MUSC/Trident Family Medicine Residency MUSC Health Sports Medicine MUSC Department of Family Medicine Harris S. Slone, MD Associate Professor Orthopaedic Surgery and Sports Medicine MUSC Health Sports Medicine MUSC Department of Orthopaedics Sports Medicine Breakout Group Leaders Aaron Brown, ATC Athletic Trainer MUSC Health Sports Medicine Amelia Brown, MS, ATC Athletic Trainer MUSC Health Sports Medicine Brittney Lang, MS, ATC Athletic Trainer MUSC Health Sports Medicine Bobby Weisenberger, MS, ATC, PES Athletic Trainer MUSC Health Sports Medicine Sports Medicine Schedule Approximate Timeline: 2:00: Introduction – Michael Barr, PT, DPT, MSR – Sports Medicine Manager 2:05: Ankle Case Report – Harris Slone, MD 2:20: Knee Case Report – Harris Slone, MD 2:35: Shoulder Instability Case Report – Michael Barr, PT, DPT, MSR 2:50: Back Case Report – Alec DeCastro, MD 3:05: High BP Case Report – Alec DeCastro, MD 3:20: Hands On Practice of Exam Techniques – All + Athletic Trainers 3:50: Question/Answer Open Forum – All 4:00: End Sports Medicine Disclosers No relevant financial disclosers Sports Medicine Learning Objectives Learning Objectives: 1. Describe mechanisms of injury and clinical presentation for common pediatric sports related injuries of the ankle, knee, back and shoulder. 2. Demonstrate examination techniques to support the diagnosis of common pediatric sports related injuries of the ankle, knee, back and shoulder 3. Determine what imaging studies should be ordered and when to refer to a sports med/orthopaedic surgeon or to physical therapy 4.
    [Show full text]
  • Upper Extremity
    Upper Extremity Shoulder Elbow Wrist/Hand Diagnosis Left Right Diagnosis Left Right Diagnosis Left Right Adhesive capsulitis M75.02 M75.01 Anterior dislocation of radial head S53.015 [7] S53.014 [7] Boutonniere deformity of fingers M20.022 M20.021 Anterior dislocation of humerus S43.015 [7] S43.014 [7] Anterior dislocation of ulnohumeral joint S53.115 [7] S53.114 [7] Carpal Tunnel Syndrome, upper limb G56.02 G56.01 Anterior dislocation of SC joint S43.215 [7] S43.214 [7] Anterior subluxation of radial head S53.012 [7] S53.011 [7] DeQuervain tenosynovitis M65.42 M65.41 Anterior subluxation of humerus S43.012 [7] S43.011 [7] Anterior subluxation of ulnohumeral joint S53.112 [7] S53.111 [7] Dislocation of MCP joint IF S63.261 [7] S63.260 [7] Anterior subluxation of SC joint S43.212 [7] S43.211 [7] Contracture of muscle in forearm M62.432 M62.431 Dislocation of MCP joint of LF S63.267 [7] S63.266 [7] Bicipital tendinitis M75.22 M75.21 Contusion of elbow S50.02X [7] S50.01X [7] Dislocation of MCP joint of MF S63.263 [7] S63.262 [7] Bursitis M75.52 M75.51 Elbow, (recurrent) dislocation M24.422 M24.421 Dislocation of MCP joint of RF S63.265 [7] S63.264 [7] Calcific Tendinitis M75.32 M75.31 Lateral epicondylitis M77.12 M77.11 Dupuytrens M72.0 Contracture of muscle in shoulder M62.412 M62.411 Lesion of ulnar nerve, upper limb G56.22 G56.21 Mallet finger M20.012 M20.011 Contracture of muscle in upper arm M62.422 M62.421 Long head of bicep tendon strain S46.112 [7] S46.111 [7] Osteochondritis dissecans of wrist M93.232 M93.231 Primary, unilateral
    [Show full text]