Signs and Symptoms of Heart Failure: Are You Asking the Right Questions?

Total Page:16

File Type:pdf, Size:1020Kb

Signs and Symptoms of Heart Failure: Are You Asking the Right Questions? Cardiovascular Critical Care SIGNS AND SYMPTOMS OF HEART FAILURE: ARE YOU ASKING THE RIGHT QUESTIONS? By Nancy Albert, PhD, CCNS, CCRN, NE-BC, Kathleen Trochelman, RN, MSN, Jianbo Li, PhD, and Songhua Lin, MS Background Patients may not verbalize common and atypical signs and symptoms of heart failure and may not understand their association with worsening disease and treatments. Objectives To examine prevalence of signs and symptoms relative to demographics, care setting, and functional class. Methods A convenience sample of 276 patients (164 ambula- tory, 112 hospitalized) with systolic heart failure completed a 1-page checklist of signs and symptoms experienced in the preceding 7 days (ambulatory) or in the 7 days before hospital- ization. Demographic and medical history data were collected. C E 1.0 Hour Results Mean age was 61.6 (SD, 14.8) years, 65% were male, 58% were white, and 45% had ischemic cardiomyopathy. Hospitalized patients reported more sudden weight gain, Notice to CE enrollees: weight loss, severe cough, low/orthostatic blood pressure, A closed-book, multiple-choice examination profound fatigue, decreased exercise, restlessness/confusion, following this article tests your under standing of irregular pulse, and palpitations (all P < .05). Patients in func- the following objectives: tional class IV reported more atypical signs and symptoms of 1. Recognize signs and atypical symptoms heart failure (severe cough, nausea/vomiting, diarrhea or loss that may be associated with worsening of appetite, and restlessness, confusion, or fainting, all P ≤.001). heart failure and functional class. Sudden weight gain increased from 5% in functional class I 2. Define key elements of a patient teaching to 37.5% in functional class IV (P < .001). Dyspnea occurred in plan related to heart failure and response all functional classes (98%-100%) and both settings (92%-100%). to treatment. Profound fatigue was associated with worsening functional 3. Define reliable indicators of functional class class (P < .001) and hospital setting (P = .001); paroxysmal noc- for heart failure patients. turnal dyspnea was associated with functional class IV (P = .02) and hospital setting (P <.001). To read this article and take the CE test online, Conclusion Profound fatigue is more reliable than dyspnea as visit www.ajcconline.org and click “CE Articles an indicator of functional class. Nurses must recognize atypi- in This Issue.” No CE test fee for AACN members. cal signs and symptoms of worsening functional class to deter- mine clinical status and facilitate patient care decisions. ©2009 American Association of Critical-Care Nurses (American Journal of Critical Care. 2010;19:443-453) doi: 10.4037/ajcc2009314 www.ajcconline.org AJCC AMERICAN JOURNAL OF CRITICAL CARE, September 2010, Volume 19, No. 5 443 eart failure is a clinical syndrome characterized by a group of signs and symp- toms. The incidence of heart failure continues to increase, with 660000 new cases diagnosed annually in adults aged 45 and over. For men and women at age 40, the lifetime risk of heart failure developing is currently 1 in 5.1 Thus it is important for health care providers to better understand the signs and symp- Htoms of heart failure so that diagnosis is not missed at first presentation and worsening status is identified early and treated promptly. Additionally, common signs and symptoms of heart failure may be well known to health care providers who treat heart failure routinely, but atypical signs and symptoms associated with worsening status may not be recognized at presentation. Heart failure represents a substantial burden to patients and nurses recognize signs and symptoms of the health care system, with estimated direct and heart failure and understand the management of heart indirect costs in 2008 expected to approach $35 failure, interconnections between bodily changes, sen- billion,1 so it is important for health care providers sations, and behaviors may become more relevant. and patients to quantify common and atypical Patients may not verbalize signs or symptoms to signs and symptoms of heart failure in order to health care providers, either because providers do not optimize diagnostic testing and treat- ask or because patients believe or perceive the signs Recognizing signs ment decisions and facilitate appro- and symptoms to be unrelated to the heart. Alterna- priate monitoring of overall status. tively, patients who have not been educated in symp- and symptoms of Because signs and symptoms of tom monitoring or those who are fearful or uncertain heart failure are important determi- about taking actions when symptomatic may cope by worsening heart nants of worsening status, learning ignoring signs and symptoms, taking action only failure may affect their frequency relative to care set- when signs and symptoms are severe, or behaving in ting and clinical status (New York ways that ultimately exacerbate heart failure. a patient’s decision Heart Association [NYHA] functional Many behavioral theorists include signs and to seek treatment. class) provides insight about determi- symptoms as a precursor to coping or self-care nants of current heart failure–related behaviors. Behavioral models that directly or indi- health status that may affect decisions about the rectly link the signs and symptoms of a disease or need for hospitalization, readiness for discharge, and condition and behavior are the Common Sense frequency of monitoring. Model of Illness,3-5 the Health Promotion Model,6 Further, recognition of signs and symptoms of the Health Belief Model,7 the Self- worsening heart failure may affect a patient’s decision Regulation Model,8 and the Symptom Management to seek treatment, follow self-care recommendations, Model.9 For example, in the Common Sense Model and adhere to medications and other aspects of the of Illness, implicit sensations and symptoms of ill- treatment plan. In qualitative research, patients with ness are processed on both cognitive and emotional heart failure did not recognize common but not heart- levels to form a conscious level of danger and threat specific symptoms such as dyspnea and fatigue as that leads to goals for coping and coping actions. If important markers of worsening condition.2 When acknowledgment of signs and symptoms is a precur- sor to coping by adhering to self-care behaviors, health care professionals need to better understand About the Authors the scope of patients’ signs and symptoms so they Nancy Albert is director of nursing research and innova- can adequately assess patients’ status and provide tion in the Nursing Institute and a clinical nurse special- ist in the Kaufman Center for Heart Failure, Kathleen education and counseling. Trochelman is a nurse researcher in nursing research The primary aim of this study was to determine and innovation at the Nursing Institute, and Jianbo Li is patients’ perception of signs and symptoms of heart a statistician and Songhua Lin is a statistical program- mer, both in Quantitative Health Sciences, at Cleveland failure before an ambulatory visit or hospitalization. Clinic in Cleveland, Ohio. The secondary aims were to use a preprinted check- list of possible signs and symptoms of heart failure Corresponding author: Nancy Albert, PhD, CCNS, CCRN, NE-BC, FAHA, FCCM, Cleveland Clinic, 9500 Euclid Avenue, Mail to examine if symptoms differed relative to demo- code J3-4, Cleveland, OH 44195 (e-mail: [email protected]). graphics, NYHA functional class, and (for patients in NYHA functional class III or IV) care setting. 444 AJCCAMERICAN JOURNAL OF CRITICAL CARE, September 2010, Volume 19, No. 5 www.ajcconline.org Methods used in another study11 before this research. The label Setting and Sample NYHA functional class I is commonly thought of as This descriptive, cross-sectional study was con- asymptomatic heart failure; however, in this study it ducted at the Cleveland Clinic in Cleveland, Ohio, matched the original definition a large tertiary care medical center with both ambu- and reflected that symptoms did The checklist latory care and hospital services, including cardiac not prohibit or limit carrying out transplantation, for patients with heart failure. The ordinary physical activities of daily format prompted institutional review board approved the study pro- living. Patients could have reported patients to report tocol, and work was completed with the ethical symptoms on the checklist and standards set forth in the Helsinki Declaration of reported NYHA functional class I all signs and 1975. Study candidates were 276 adults: 164 sched- status if they believed ordinary uled for an ambulatory visit in the heart failure dis- physical activity did not cause symptoms they ease management program (a clinic led by an undue dyspnea, fatigue, palpita- were experiencing, advanced practice nurse) and 112 patients being tions, or chest pain. treated in the hospital for exacerbation of chronic The checklist format prompted rather than only heart failure. Convenience sampling was used to patients to report all signs and those they thought collect data for 11 months. Overall sample size was symptoms they were experiencing, not predetermined. The enrollment goal was to rather than reporting only those were related to achieve a minimum sample of 40 patients per NYHA that they thought were related to heart failure. functional class so that adequate assessment by heart failure. Wording of items was functional class could be completed. Inclusion crite- simplified to enhance patients’ understanding;
Recommended publications
  • Noninvasive Positive Pressure Ventilation in the Home
    Technology Assessment Program Noninvasive Positive Pressure Ventilation in the Home Final Technology Assessment Project ID: PULT0717 2/4/2020 Technology Assessment Program Project ID: PULT0717 Noninvasive Positive Pressure Ventilation in the Home (with addendum) Prepared for: Agency for Healthcare Research and Quality U.S. Department of Health and Human Services 5600 Fishers Lane Rockville, MD 20857 www.ahrq.gov Contract No: HHSA290201500013I_HHSA29032004T Prepared by: Mayo Clinic Evidence-based Practice Center Rochester, MN Investigators: Michael Wilson, M.D. Zhen Wang, Ph.D. Claudia C. Dobler, M.D., Ph.D Allison S. Morrow, B.A. Bradley Beuschel, B.S.P.H. Mouaz Alsawas, M.D., M.Sc. Raed Benkhadra, M.D. Mohamed Seisa, M.D. Aniket Mittal, M.D. Manuel Sanchez, M.D. Lubna Daraz, Ph.D Steven Holets, R.R.T. M. Hassan Murad, M.D., M.P.H. Key Messages Purpose of review To evaluate home noninvasive positive pressure ventilation (NIPPV) in adults with chronic respiratory failure in terms of initiation, continuation, effectiveness, adverse events, equipment parameters and required respiratory services. Devices evaluated were home mechanical ventilators (HMV), bi-level positive airway pressure (BPAP) devices, and continuous positive airway pressure (CPAP) devices. Key messages • In patients with COPD, home NIPPV as delivered by a BPAP device (compared to no device) was associated with lower mortality, intubations, hospital admissions, but no change in quality of life (low to moderate SOE). NIPPV as delivered by a HMV device (compared individually with BPAP, CPAP, or no device) was associated with fewer hospital admissions (low SOE). In patients with thoracic restrictive diseases, HMV (compared to no device) was associated with lower mortality (low SOE).
    [Show full text]
  • Respiratory Insufficiency in Patients with ALS at Or Near the End of Life
    Amyotrophic lateral sclerosis (ALS) is a devastating motor neuron disease causing progressive paralysis and eventual death, usually from respiratory failure. Treatment for ALS is focused primarily on optimal symptom manage- ment because there is no known cure. Respiratory symptoms that occur are related to the disease process and can be very distressing for patients and their loved ones. Recommendations on the management of respira- tory insufficiency are provided to help guide clinicians caring for patients with ALS. Hospice and Palliative Care Feature The Management of Andrea L. Torres, APN, CNP Respiratory Insufficiency in Patients With ALS at or Near the End of Life 186 Home Healthcare Nurse www.homehealthcarenurseonline.com Copyright © 2012 Lippincott Williams & Wilkins. Unauthorized reproduction of this article is prohibited. Introduction 2007). By the time most patients are definitively Amyotrophic lateral sclerosis (ALS) is a devastat- diagnosed, they are often already in an advanced ing motor neuron disease characterized by pro- stage of the disease (Wood-Allum & Shaw, 2010). gressive muscle weakness eventually leading to Life expectancy is typically 3-5 years from the paralysis and death. The onset typically occurs onset of symptoms (Elman et al., 2007). in late middle age, with men slightly more af- fected than women (Wood-Allum & Shaw, 2010). Palliative Care Approaches for ALS Patients The majority of cases of ALS have no known Due to the progressive nature of ALS, early pal- cause; about 10% of ALS cases are linked to a fa- liative care is an essential component in the milial trait (Ferguson & Elman, 2007). Treatment treatment plan, and should begin as soon as the is primarily focused on optimal symptom man- diagnosis of ALS is confirmed (Elman et al., 2007).
    [Show full text]
  • CT Children's CLASP Guideline
    CT Children’s CLASP Guideline Chest Pain INTRODUCTION . Chest pain is a frequent complaint in children and adolescents, which may lead to school absences and restriction of activities, often causing significant anxiety in the patient and family. The etiology of chest pain in children is not typically due to a serious organic cause without positive history and physical exam findings in the cardiac or respiratory systems. Good history taking skills and a thorough physical exam can point you in the direction of non-cardiac causes including GI, psychogenic, and other rare causes (see Appendix A). A study performed by the New England Congenital Cardiology Association (NECCA) identified 1016 ambulatory patients, ages 7 to 21 years, who were referred to a cardiologist for chest pain. Only two patients (< 0.2%) had chest pain due to an underlying cardiac condition, 1 with pericarditis and 1 with an anomalous coronary artery origin. Therefore, the vast majority of patients presenting to primary care setting with chest pain have a benign etiology and with careful screening, the patients at highest risk can be accurately identified and referred for evaluation by a Pediatric Cardiologist. INITIAL INITIAL EVALUATION: Focused on excluding rare, but serious abnormalities associated with sudden cardiac death EVALUATION or cardiac anomalies by obtaining the targeted clinical history and exam below (red flags): . Concerning Pain Characteristics, See Appendix B AND . Concerning Past Medical History, See Appendix B MANAGEMENT . Alarming Family History, See Appendix B . Physical exam: - Blood pressure abnormalities (obtain with manual cuff, in sitting position, right arm) - Non-innocent murmurs . Obtain ECG, unless confident pain is musculoskeletal in origin: - ECG’s can be obtained at CT Children’s main campus and satellites locations daily (Hartford, Danbury, Glastonbury, Shelton).
    [Show full text]
  • A Case of Extreme Hypercapnia
    119 Emerg Med J: first published as 10.1136/emj.2003.005009 on 20 January 2004. Downloaded from CASE REPORTS A case of extreme hypercapnia: implications for the prehospital and accident and emergency department management of acutely dyspnoeic patients L Urwin, R Murphy, C Robertson, A Pollok ............................................................................................................................... Emerg Med J 2004;21:119–120 64 year old woman was brought by ambulance to the useful non-invasive technique to aid the assessment of accident and emergency department. She had been peripheral oxygen saturation. In situations of poor perfusion, Areferred by her GP because of increasing dyspnoea, movement and abnormal haemoglobin, however, this tech- cyanosis, and lethargy over the previous four days. On arrival nique may not reliably reflect PaO2 values. More importantly, of the ambulance crew at her home she was noted to be and as shown in our case, there is no definite relation tachycardic and tachypnoeic (respiratory rate 36/min) with a between SaO2 values measured by pulse oximetry and PaCO2 GCS of 5 (E 3, M 1, V 1). She was given oxygen at 6 l/min via values although it has been shown that the more oxygenated a Duo mask, and transferred to hospital. The patient arrived at the accident and emergency department 18 minutes later. In transit, there had been a clinical deterioration. The GCS was now 3 and the respiratory rate 4/min. Oxygen saturation, as measured by a pulse oximeter was 99%. The patient was intubated and positive pressure ventilation started. Arterial blood gas measurements taken at the time of intubation were consistent with acute on chronic respiratory failure (fig 1).
    [Show full text]
  • Chapter 17 Dyspnea Sabina Braithwaite and Debra Perina
    Chapter 17 Dyspnea Sabina Braithwaite and Debra Perina ■ PERSPECTIVE Pathophysiology Dyspnea is the term applied to the sensation of breathlessness The actual mechanisms responsible for dyspnea are unknown. and the patient’s reaction to that sensation. It is an uncomfort- Normal breathing is controlled both centrally by the respira- able awareness of breathing difficulties that in the extreme tory control center in the medulla oblongata, as well as periph- manifests as “air hunger.” Dyspnea is often ill defined by erally by chemoreceptors located near the carotid bodies, and patients, who may describe the feeling as shortness of breath, mechanoreceptors in the diaphragm and skeletal muscles.3 chest tightness, or difficulty breathing. Dyspnea results Any imbalance between these sites is perceived as dyspnea. from a variety of conditions, ranging from nonurgent to life- This imbalance generally results from ventilatory demand threatening. Neither the clinical severity nor the patient’s per- being greater than capacity.4 ception correlates well with the seriousness of underlying The perception and sensation of dyspnea are believed to pathology and may be affected by emotions, behavioral and occur by one or more of the following mechanisms: increased cultural influences, and external stimuli.1,2 work of breathing, such as the increased lung resistance or The following terms may be used in the assessment of the decreased compliance that occurs with asthma or chronic dyspneic patient: obstructive pulmonary disease (COPD), or increased respira- tory drive, such as results from severe hypoxemia, acidosis, or Tachypnea: A respiratory rate greater than normal. Normal rates centrally acting stimuli (toxins, central nervous system events).
    [Show full text]
  • Chest Auscultation: Presence/Absence and Equality of Normal/Abnormal and Adventitious Breath Sounds and Heart Sounds A
    Northwest Community EMS System Continuing Education: January 2012 RESPIRATORY ASSESSMENT Independent Study Materials Connie J. Mattera, M.S., R.N., EMT-P COGNITIVE OBJECTIVES Upon completion of the class, independent study materials and post-test question bank, each participant will independently do the following with a degree of accuracy that meets or exceeds the standards established for their scope of practice: 1. Integrate complex knowledge of pulmonary anatomy, physiology, & pathophysiology to sequence the steps of an organized physical exam using four maneuvers of assessment (inspection, palpation, percussion, and auscultation) and appropriate technique for patients of all ages. (National EMS Education Standards) 2. Integrate assessment findings in pts who present w/ respiratory distress to form an accurate field impression. This includes developing a list of differential diagnoses using higher order thinking and critical reasoning. (National EMS Education Standards) 3. Describe the signs and symptoms of compromised ventilations/inadequate gas exchange. 4. Recognize the three immediate life-threatening thoracic injuries that must be detected and resuscitated during the “B” portion of the primary assessment. 5. Explain the difference between pulse oximetry and capnography monitoring and the type of information that can be obtained from each of them. 6. Compare and contrast those patients who need supplemental oxygen and those that would be harmed by hyperoxia, giving an explanation of the risks associated with each. 7. Select the correct oxygen delivery device and liter flow to support ventilations and oxygenation in a patient with ventilatory distress, impaired gas exchange or ineffective breathing patterns including those patients who benefit from CPAP. 8. Explain the components to obtain when assessing a patient history using SAMPLE and OPQRST.
    [Show full text]
  • Community-Acquired Pneumonia in Adults: Diagnostic Reliability of Physical Examination Techniques and Their Teaching in Academia
    James Madison University JMU Scholarly Commons Physician Assistant Capstones The Graduate School Fall 12-14-2018 Community-acquired pneumonia in adults: Diagnostic reliability of physical examination techniques and their teaching in academia Amber Tordoff James Madison University Lauren A. Williams James Madison University Follow this and additional works at: https://commons.lib.jmu.edu/pacapstones Part of the Bacteria Commons, Bacterial Infections and Mycoses Commons, Diagnosis Commons, Investigative Techniques Commons, Medical Pathology Commons, Respiratory Tract Diseases Commons, Virus Diseases Commons, and the Viruses Commons Recommended Citation Tordoff AL, Williams LA. Community-Acquired Pneumonia in Adults: Diagnostic Reliability of Physical Examination Techniques and their Teaching in Academia. JMU Scholarly Commons Physician Assistant Capstones. https://commons.lib.jmu.edu/pacapstones/44/. Published December 12, 2018. This Presentation is brought to you for free and open access by the The Graduate School at JMU Scholarly Commons. It has been accepted for inclusion in Physician Assistant Capstones by an authorized administrator of JMU Scholarly Commons. For more information, please contact [email protected]. Community-Acquired Pneumonia in Adults: Diagnostic Reliability of Physical Examination Techniques and their Teaching in Academia Amber Tordoff, PA-S and Lauren Williams, PA-S, James Madison University, Harrisonburg, Virginia _____________________________________________________________________________________ ABSTRACT Background:
    [Show full text]
  • Chapter 11 Dyspnea, Orthopnea, and Paroxysmal Nocturnal Dyspnea
    2/12/2015 Dyspnea, Orthopnea, and Paroxysmal Nocturnal Dyspnea ­ Clinical Methods ­ NCBI Bookshelf NCBI Bookshelf. A service of the National Library of Medicine, National Institutes of Health. Walker HK, Hall WD, Hurst JW, editors. Clinical Methods: The History, Physical, and Laboratory Examinations. 3rd edition. Boston: Butterworths; 1990. Chapter 11 Dyspnea, Orthopnea, and Paroxysmal Nocturnal Dyspnea Vaskar Mukerji. Definition Dyspnea refers to the sensation of difficult or uncomfortable breathing. It is a subjective experience perceived and reported by an affected patient. Dyspnea on exertion (DOE) may occur normally, but is considered indicative of disease when it occurs at a level of activity that is usually well tolerated. Dyspnea should be differentiated from tachypnea, hyperventilation, and hyperpnea, which refer to respiratory variations regardless of the patients" subjective sensations. Tachypnea is an increase in the respiratory rate above normal; hyperventilation is increased minute ventilation relative to metabolic need, and hyperpnea is a disproportionate rise in minute ventilation relative to an increase in metabolic level. These conditions may not always be associated with dyspnea. Orthopnea is the sensation of breathlessness in the recumbent position, relieved by sitting or standing. Paroxysmal nocturnal dyspnea (PND) is a sensation of shortness of breath that awakens the patient, often after 1 or 2 hours of sleep, and is usually relieved in the upright position. Two uncommon types of breathlessness are trepopnea and platypnea. Trepopnea is dyspnea that occurs in one lateral decubitus position as opposed to the other. Platypnea refers to breathlessness that occurs in the upright position and is relieved with recumbency. Technique A patient with dyspnea may say: "I feel short of breath," "I"m having difficulty breathing," "I can"t catch my breath," "I feel like I"m suffocating." Because it is a subjective phenomenon, the perception of dyspnea and its interpretation vary from patient to patient.
    [Show full text]
  • Many Faces of Chest Pain Ian Mcleod, MS, Med, PA-C, ATC Northern Arizona University ASAPA Spring Conference 2019 Disclosures
    Many Faces of Chest Pain Ian McLeod, MS, MEd, PA-C, ATC Northern Arizona University ASAPA Spring Conference 2019 Disclosures • I have no financial disclosures to report Objectives • Following the presentation attendees will be able to: • Develop a concise differential diagnosis for patients with chest pain including cardiac and non-cardiac causes. • Describe key clinical characteristics and management of the following chest pain etiologies: angina, embolism, gastroesophageal reflux, costochondritis, costochondral dysfunction, anxiety and pneumonia. • Discuss appropriate use of diagnostic studies utilized in the evaluation of patients presenting with chest pain. Chest Pain – Primary Care Setting • ~1.5% of all visits are for chest pain • Musculoskeletal 35-50% • Gastrointestinal 10-20% • Cardiac 10-15% • Pulmonary 5-10% • Psychogenic 1-2% Chest Pain Differentials • Cardiac • Pulmonary • Stable angina • Pneumonia • Acute coronary syndrome • Pulmonary embolism • Pericarditis • Spontaneous pneumothorax • Aortic dissection • Psych • MSK • Panic disorder • Costochondritis • Tietze syndrome • Costovertebral joint dysfunction • GI • Gastroesophageal reflux disease (GERD) • Medication induced esophagitis Setting the stage • Non-traumatic • Acute chest pain • Primary care setting • H&P • ECG • CXR Myocardial Ischemia Risk Factors • Increasing age • Male sex • Chronic renal insufficiency • Diabetes Mellitus • Known atherosclerotic disease → coronary or peripheral • Early family history of coronary artery disease • 1st degree male relative < 55 y/o
    [Show full text]
  • The Mechanism of Cheyne-Stokes Respiration
    THE MECHANISM OF CHEYNE-STOKES RESPIRATION Ramon L. Lange, Hans H. Hecht J Clin Invest. 1962;41(1):42-52. https://doi.org/10.1172/JCI104465. Research Article Find the latest version: https://jci.me/104465/pdf Journal of Clinical Investigation Vol. 41, No. 1, 1962 THE MECHANISM OF CHEYNE-STOKES RESPIRATION * By RAMON L. LANGE t AND HANS H. HECHT (From the Department of Medicine, University of Utah College of Medicine, Salt Lake City, Utah) (Submitted for publication June 1, 1961; accepted September 14, 1961) The unusual breathing pattern characterized by on the part of the patient"); d) the crescendo- cycles of hyperventilation and apnea was first de- decrescendo respiratory pattern; e) return to nor- scribed by Cheyne in 1818 (1) but wider attention mal breathing when circulation is improved. was attracted to the phenomenon by Stokes in his Pryor (3) demonstrated a phasic temporal shift work "The Diseases of the Heart and Aorta" in between ventilatory function and arterial blood gas 1854 (2). This pattern is most commonly seen values in individuals with Cheyne-Stokes (C-S) in patients with heart disease. The form of cardiac respiration and heart disease with arterial oxygen dysfunction associated with this respiratory dis- saturation highest during the period of apnea. order is characterized by left ventricular dilatation, This was not true in patients with intracranial pulmonary congestion, and a low cardiac output. hemorrhage or increased intracranial pressure who Hypertensive disease or ischemic heart disease are also exhibited intermittent respiration. He as- the usual etiological factors. sumed that in C-S breathing an increased lung-to- The underlying mechanism of this disorder has brain circulation time caused oscillatory variations been the subject of debate since Cheyne described in arterial saturation resulting from a delayed it in a man with "heart disease and apoplexy." feedback of the ventilatory effects (output) to the Stokes implicated heart disease more specifically respiratory center (input) (Figure 1).
    [Show full text]
  • Physical Diagnosis the Pulmonary Exam What Should We Know About the Examination of the Chest?
    PHYSICAL DIAGNOSIS THE PULMONARY EXAM WHAT SHOULD WE KNOW ABOUT THE EXAMINATION OF THE CHEST? • LANDMARKS • PERTINENT VOCABULARY • SYMPTOMS • SIGNS • HOW TO PERFORM AN EXAM • HOW TO PRESENT THE INFORMATION • HOW TO FORMULATE A DIFFERENTIAL DIAGNOSIS IMPORTANT TOPOGRAPHY OF THE CHEST TOPOGRAPHY OF THE BACK LOOK AT THE PATIENT • RESPIRATORY DISTRESS • ANXIOUS • CLUTCHING • ACCESSORY MUSCLES •CYANOSIS • GASPING • STRIDOR • CLUBBING TYPES OF BODY HABITUS WHAT IS A BARRELL CHEST? • THORACIC INDEX – RATIO OF THE ANTERIORPOSTERIOR TO LATERAL DIAMETER NORMAL 0.70 – 0.75 IN ADULTS - >0.9 IS CONSIDERED ABNORMAL • NORMALS - ILLUSION •COPD AM J MED 25:13-22,1958 PURSED – LIPS BREATHING • COPD – DECREASES DYSPNEA • DECREASES RR • INCREASES TIDAL VOLUME • DECREASES WORK OF BREATHING CHEST 101:75-78, 1992 WHITE NOISE (NOISY BREATHING) • THIS NOISE CAN BE HEARD AT THE BEDSIDE WITHOUT THE STETHOSCOPE • LACKS A MUSICAL PITCH • AIR TURBULENCE CAUSED BY NARROWED AIRWAYS • CHRONIC BRONCHITIS CHEST 73:399-412, 1978 RESPIRATORY ALTERNANS • NORMALLY BOTH CHEST AND ABDOMEN RISE DURING INSPIRATION • PARADOXICAL RESPIRATION IMPLIES THAT DURING INSPIRATION THE CHEST RISES AND THE ABDOMEN COLLAPSES • IMPENDING MUSCLE FATIGUE DO NOT FORGET THE TRACHEA • TRACHEAL DEVIATION • AUSCULTATE - STRIDOR • TRACHEAL TUG (OLIVERS SIGN) – DOWNWARD DISPLACEMENT OF THE CRICOID CARTILAGE WITH VENTRICULAR CONTRACTION – OBSERVED IN PATIENTS WITH AN AORTIC ARCH ANEURYSM • TRACHEAL TUG (CAMPBELL’S SIGN) – DOWNWARD DISPACEMENT OF THE THYROID CARTILAGE DURING INSPIRATION – SEEN IN PATIENTS
    [Show full text]
  • Examen Préopératoire De L'enfant
    Preanesthetic examination of the child Red flags Francis Veyckemans BAPA-SKA Refresher course Leuven 2020 ? We only find what we look for We only look for what we know The child with • Sleep-disordered breathing • Bronchopulmonary dysplasia • A mediastinal mass Sleep-disordered breathing • Simple snoring : 20% of children microarousals during sleep • Increased upper airway resistance some behavioural problems • Hypopnea partial airway obstruction • Obstructive apnea : 1-3% episodes of complete airway obstruction Obstructive sleep apnea syndrome 3 types of OSAS: 1: hypertrophy of tonsils /adenoids 2: obesity with moderate tonsils /adenoids 3: orofacial pathology - midfacial hypoplasia (achondroplasia …) - macroglossia, small pharynx - hypotonia (T 21, Prader Willi, polyhandicap) - micrognathia Normal Snoring OSAS Signs & symptoms of OSAS * During sleep: - snoring - respiratory arrests - restlessness, nightmares - neck hyperextension * Adenoid facies (open mouth) * Diurnal somnolence * Mood disorders * Increased risk in African American ethnicity Risks of OSAS • Mood disorders • Learning difficulties • Risk of chronic cor pulmonale • More respiratory complications : obstruction, desaturation, laryngospasm • Decreased response to CO2 • Increased sensitivity to opiates ( n of µ opiates in the forebrain) Polysomnography: gold standard Obstructive apnea /hypopnea index per hour of sleep > 1.5 = pathologic 1.5 -5= mild OSAS > 5 = moderate OSAS > 10 = severe OSAS 80% during REM sleep (>< in adults: non-REM sleep) Sleep oximetry : McGill Oximetry
    [Show full text]