Recommendations and Guidelines for Preoperative Evaluation
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Clinical Excellence in Cardiology
Clinical Excellence in Cardiology Roy C. Ziegelstein, MD* A recent study identified 7 domains of clinical excellence on the basis of interviews with “clinically excellent” physicians at academic institutions in the United States: (1) commu- nication and interpersonal skills, (2) professionalism and humanism, (3) diagnostic acu- men, (4) skillful negotiation of the health care system, (5) knowledge, (6) taking a scholarly approach to clinical practice, and (7) having passion for clinical medicine. What constitutes clinical excellence in cardiology has not previously been defined. The author discusses clinical excellence in cardiology using the framework of these 7 domains and also considers the additional domain of clinical experience. Specific aspects of the domains of clinical excellence that are of greatest relevance to cardiology are highlighted. In conclusion, this discussion characterizes what constitutes clinical excellence in cardiology and should stimulate additional discussion of the topic and an examination of how the domains of clinical excellence in cardiology are related to specific patient outcomes. © 2011 Elsevier Inc. All rights reserved. (Am J Cardiol 2011;108:607–611) On the basis of interviews with 24 academic physicians and clinical excellence has not been clearly demonstrated. deemed “clinically excellent,” Christmas et al1 identified 7 For example, the compliance of hospitals with performance domains of clinical excellence relevant to all disciplines in measures is not associated with improved heart failure out- medicine: (1) communication and interpersonal skills, (2) comes.10,11 The speed with which an interventional cardi- professionalism and humanism, (3) diagnostic acumen, (4) ologist achieves reperfusion of the culprit vessel, the so- skillful negotiation of the health care system, (5) knowl- called door-to-balloon time, is an important performance edge, (6) taking a scholarly approach to clinical practice, measure in the treatment of patients with acute ST-segment and (7) having passion for clinical medicine. -
Carcinoid) Tumours Gastroenteropancreatic
Downloaded from gut.bmjjournals.com on 8 September 2005 Guidelines for the management of gastroenteropancreatic neuroendocrine (including carcinoid) tumours J K Ramage, A H G Davies, J Ardill, N Bax, M Caplin, A Grossman, R Hawkins, A M McNicol, N Reed, R Sutton, R Thakker, S Aylwin, D Breen, K Britton, K Buchanan, P Corrie, A Gillams, V Lewington, D McCance, K Meeran, A Watkinson and on behalf of UKNETwork for neuroendocrine tumours Gut 2005;54;1-16 doi:10.1136/gut.2004.053314 Updated information and services can be found at: http://gut.bmjjournals.com/cgi/content/full/54/suppl_4/iv1 These include: References This article cites 201 articles, 41 of which can be accessed free at: http://gut.bmjjournals.com/cgi/content/full/54/suppl_4/iv1#BIBL Rapid responses You can respond to this article at: http://gut.bmjjournals.com/cgi/eletter-submit/54/suppl_4/iv1 Email alerting Receive free email alerts when new articles cite this article - sign up in the box at the service top right corner of the article Topic collections Articles on similar topics can be found in the following collections Stomach and duodenum (510 articles) Pancreas and biliary tract (332 articles) Guidelines (374 articles) Cancer: gastroenterological (1043 articles) Liver, including hepatitis (800 articles) Notes To order reprints of this article go to: http://www.bmjjournals.com/cgi/reprintform To subscribe to Gut go to: http://www.bmjjournals.com/subscriptions/ Downloaded from gut.bmjjournals.com on 8 September 2005 iv1 GUIDELINES Guidelines for the management of gastroenteropancreatic neuroendocrine (including carcinoid) tumours J K Ramage*, A H G Davies*, J ArdillÀ, N BaxÀ, M CaplinÀ, A GrossmanÀ, R HawkinsÀ, A M McNicolÀ, N ReedÀ, R Sutton`, R ThakkerÀ, S Aylwin`, D Breen`, K Britton`, K Buchanan`, P Corrie`, A Gillams`, V Lewington`, D McCance`, K Meeran`, A Watkinson`, on behalf of UKNETwork for neuroendocrine tumours .............................................................................................................................. -
Septoplasty, Rhinoplasty, Septorhinoplasty, Turbinoplasty Or
Septoplasty, Rhinoplasty, Septorhinoplasty, 4 Turbinoplasty or Turbinectomy CPAP • If you have obstructive sleep apnea and use CPAP, please speak with your surgeon about how to use it after surgery. Follow-up • Your follow-up visit with the surgeon is about 1 to 2 weeks after Septoplasty, Rhinoplasty, Septorhinoplasty, surgery. You will need to call for an appointment. Turbinoplasty or Turbinectomy • During this visit any nasal packing or stents will be removed. Who can I call if I have questions? For a healthy recovery after surgery, please follow these instructions. • If you have any questions, please contact your surgeon’s office. Septoplasty is a repair of the nasal septum. You may have • For urgent questions after hours, please call the Otolaryngologist some packing up your nose or splints which stay in for – Head & Neck (ENT) surgeon on call at 905-521-5030. 7 to 14 days. They will be removed at your follow up visit. When do I need medical help? Rhinoplasty is a repair of the nasal bones. You will have a small splint or plaster on your nose. • If you have a fever 38.5°C (101.3°F) or higher. • If you have pain not relieved by medication. Septorhinoplasty is a repair of the nasal septum and the nasal bone. You will have a small splint or plaster cast on • If you have a hot or inflamed nose, or pus draining from your nose, your nose. or an odour from your nose. • If you have an increase in bleeding from your nose or on Turbinoplasty surgery reduces the size of the turbinates in your dressing. -
Ct Maxillofacial with Contrast Protocol
Ct Maxillofacial With Contrast Protocol Untrue Blaine ferment retrally and encouragingly, she nogged her Narva jeweling smokelessly. Unsaluted Zak scollop: he bards his self-sacrifice lordly and trichotomously. Eliott companions perdurably. In contrast both ultra-low dose protocols that combined a larger voxel. Although some elements on maxillofacial lesions in protocol change was limited in addition, protocols were reported are extremely thin slices. Contact us see it kills thyroid functions, which are related disorders such as they safe. CT The American College of Radiology with regret than. If the protocol is changed by one then our radiologists to condition more suitable. PRACTICE PARAMETER CT American College of Radiology. Assume that ct? Separate requests for concurrent imaging of the arteries and the veins in separate head are inappropriate. You have had an hour prior to maxillofacial fibrosarcoma using special room, protocol is no additional effects research to help your details. Patient lead a candidate for curative surgery. No citing articles found no other precautions can be stored in your email address ct maxillofacial radiology facilities may affect your contrast ct with maxillofacial lesions. Pillows may est will usually, with maxillofacial ct with persistent dysesthesia as radiation. It personnel also used to narrate at blood vessels and lymph nodes in the abdomen. RESULTS Compared with the reference dose protocol with FBP the. MRI lumbar spine pain and without IV contrast is best appropriate; CT lumbar spine system or without IV contrast can be performed if MRI is contraindicated. Ordered CT exams under ARA protocols For any coding. Studies by maxillofacial with other. -
Handheld Devices and Informatics in Anesthesia Prasanna Vadhanan,MD1, Adinarayanan S., DNB2
COMMENTRY Handheld devices and informatics in anesthesia Prasanna Vadhanan,MD1, Adinarayanan S., DNB2 1Associate Professor, Department of Anaesthesiology, Vinayaka Missions Medical College & Hospital, Keezhakasakudimedu, Kottucherry Post, Karaikal, Puducherry 609609, (India) 2Associate Professor, Department of Anaesthesiology & Critical Care. The Jawaharlal Institute of Postgraduate Medical Education & Research, Dhanvantri Nagar, Gorimedu, Puducherry, 605006, (India) Correspondence: Dr. Prasanna Vadhanan, MD, No. 6, P&T Nagar, Mayiladuthurai 609001 (India); Phone: +919486489690; E-mail: [email protected] Received: 02 April 2016; Reviewed: 2 May 2016; Corrected: 02 June 2016; Accepted: 10 June 2016 ABSTRACT Handheld devices like smartphones, once viewed as a diversion and interference in the operating room have become an integral part of healthcare. In the era of evidence based medicine, the need to remain up to date with current practices is felt more than ever before. Medical informatics helps us by analysing complex data in making clinical decisions and knowing recent advances at the point of patient care. Handheld devices help in delivering such information at the point of care; however, too much reliance upon technology might be hazardous especially in an emergency situation. With the recent approval of robots to administer anesthesia, the question of whether technology can replace anesthesiologists from the operating room looms ahead. The anesthesia and critical care related applications of handheld devices and informatics -
Significance of ST-Segment Depression During Supraventricular Tachycardia
Significance of ST-segment depression during supraventricular tachycardia. Clues offered by its return to normal at the end of the episode Gianaugusto Slavich, Daisy Pavoni, Luigi Badano, Malgorzata Popiel* Cardiology Unit, S. Maria della Misericordia Hospital, Udine, *I Division of Cardiology, Department of Medicine, University of Poznan, Poland Key words: The finding of transient ST-segment depression during episodes of supraventricular tachycardia ST-segment depression; is common but its ischemic significance is usually uncertain. Several authors came to the conclusion Supraventricular that in the absence of positive myocardial scintigraphy these alterations are not associated with a tachyarrhythmias. coronary flow-limiting stenosis. Our report tends to confirm this view but we suggest to observe the evolution of ST-segment changes at the very end of the episodes; these mechanisms have not been ad- equately addressed in previous studies and could provide useful clues to the ischemic or non-ischemic origin of ST-segment abnormalities. (Ital Heart J 2002; 3 (3): 206-210) © 2002 CEPI Srl The finding of transient ST-segment de- Description of cases pression during episodes of supraventricu- Received June 20, 2001; lar tachycardia is rather common but its is- Case 1. A 63-year-old male, with a nega- revision received January 17, 2002; accepted chemic significance is usually uncertain. tive cardiovascular history, no coronary January 19, 2002. Two clinical cases prompted us to ad- risk factors and practicing frequent and in- dress this issue and to review the pertinent tense recreational activity, presented with Address: literature. Our report suggests that the ob- complaints of recurrent sudden-onset pal- Dr. -
Essentials of Bedside Cardiology CONTEMPORARY CARDIOLOGY
Essentials of Bedside Cardiology CONTEMPORARY CARDIOLOGY CHRISTOPHER P. CANNON, MD SERIES EDITOR Aging, Heart Disease and Its Management: Facts and Controversies, edited by Niloo M. Edwards, MD, Mathew S. Maurer, MD, and Rachel B. Wellner, MD, 2003 Peripheral Arterial Disease: Diagnosis and Treatment, edited by Jay D. Coffman, MD, and Robert T. Eberhardt, MD, 2003 Essentials ofBedside Cardiology: With a Complete Course in Heart Sounds and Munnurs on CD, Second Edition, by Jules Constant, MD, 2003 Primary Angioplasty in Acute Myocardial Infarction, edited by James E. Tcheng, MD,2002 Cardiogenic Shock: Diagnosis and Treatment, edited by David Hasdai, MD, Peter B. Berger, MD, Alexander Battler, MD, and David R. Holmes, Jr., MD, 2002 Management of Cardiac Arrhythmias, edited by Leonard I. Ganz, MD, 2002 Diabetes and Cardiovascular Disease, edited by Michael T. Johnstone and Aristidis Veves, MD, DSC, 2001 Blood Pressure Monitoring in Cardiovascular Medicine and Therapeutics, edited by William B. White, MD, 2001 Vascular Disease and Injury: Preclinical Research, edited by Daniell. Simon, MD, and Campbell Rogers, MD 2001 Preventive Cardiology: Strategies for the Prevention and Treatment of Coronary Artery Disease, edited by JoAnne Micale Foody, MD, 2001 Nitric Oxide and the Cardiovascular System, edited by Joseph Loscalzo, MD, phD and Joseph A. Vita, MD, 2000 Annotated Atlas of Electrocardiography: A Guide to Confident Interpretation, by Thomas M. Blake, MD, 1999 Platelet Glycoprotein lIb/IlIa Inhibitors in Cardiovascular Disease, edited by A. Michael Lincoff, MD, and Eric J. Topol, MD, 1999 Minimally Invasive Cardiac Surgery, edited by Mehmet C. Oz, MD and Daniel J. Goldstein, MD, 1999 Management ofAcute Coronary Syndromes, edited by Christopher P. -
Anemia in Heart Failure - from Guidelines to Controversies and Challenges
52 Education Anemia in heart failure - from guidelines to controversies and challenges Oana Sîrbu1,*, Mariana Floria1,*, Petru Dascalita*, Alexandra Stoica1,*, Paula Adascalitei, Victorita Sorodoc1,*, Laurentiu Sorodoc1,* *Grigore T. Popa University of Medicine and Pharmacy; Iasi-Romania 1Sf. Spiridon Emergency Hospital; Iasi-Romania ABSTRACT Anemia associated with heart failure is a frequent condition, which may lead to heart function deterioration by the activation of neuro-hormonal mechanisms. Therefore, a vicious circle is present in the relationship of heart failure and anemia. The consequence is reflected upon the pa- tients’ survival, quality of life, and hospital readmissions. Anemia and iron deficiency should be correctly diagnosed and treated in patients with heart failure. The etiology is multifactorial but certainly not fully understood. There is data suggesting that the following factors can cause ane- mia alone or in combination: iron deficiency, inflammation, erythropoietin levels, prescribed medication, hemodilution, and medullar dysfunc- tion. There is data suggesting the association among iron deficiency, inflammation, erythropoietin levels, prescribed medication, hemodilution, and medullar dysfunction. The main pathophysiologic mechanisms, with the strongest evidence-based medicine data, are iron deficiency and inflammation. In clinical practice, the etiology of anemia needs thorough evaluation for determining the best possible therapeutic course. In this context, we must correctly treat the patients’ diseases; according with the current guidelines we have now only one intravenous iron drug. This paper is focused on data about anemia in heart failure, from prevalence to optimal treatment, controversies, and challenges. (Anatol J Cardiol 2018; 20: 52-9) Keywords: anemia, heart failure, intravenous iron, ferric carboxymaltose, quality of life Introduction g/dL in men) (2). -
Noninvasive Positive Pressure Ventilation in the Home
Technology Assessment Program Noninvasive Positive Pressure Ventilation in the Home Final Technology Assessment Project ID: PULT0717 2/4/2020 Technology Assessment Program Project ID: PULT0717 Noninvasive Positive Pressure Ventilation in the Home (with addendum) Prepared for: Agency for Healthcare Research and Quality U.S. Department of Health and Human Services 5600 Fishers Lane Rockville, MD 20857 www.ahrq.gov Contract No: HHSA290201500013I_HHSA29032004T Prepared by: Mayo Clinic Evidence-based Practice Center Rochester, MN Investigators: Michael Wilson, M.D. Zhen Wang, Ph.D. Claudia C. Dobler, M.D., Ph.D Allison S. Morrow, B.A. Bradley Beuschel, B.S.P.H. Mouaz Alsawas, M.D., M.Sc. Raed Benkhadra, M.D. Mohamed Seisa, M.D. Aniket Mittal, M.D. Manuel Sanchez, M.D. Lubna Daraz, Ph.D Steven Holets, R.R.T. M. Hassan Murad, M.D., M.P.H. Key Messages Purpose of review To evaluate home noninvasive positive pressure ventilation (NIPPV) in adults with chronic respiratory failure in terms of initiation, continuation, effectiveness, adverse events, equipment parameters and required respiratory services. Devices evaluated were home mechanical ventilators (HMV), bi-level positive airway pressure (BPAP) devices, and continuous positive airway pressure (CPAP) devices. Key messages • In patients with COPD, home NIPPV as delivered by a BPAP device (compared to no device) was associated with lower mortality, intubations, hospital admissions, but no change in quality of life (low to moderate SOE). NIPPV as delivered by a HMV device (compared individually with BPAP, CPAP, or no device) was associated with fewer hospital admissions (low SOE). In patients with thoracic restrictive diseases, HMV (compared to no device) was associated with lower mortality (low SOE). -
Infarto Auricular, Infarto De Miocardio Inferior Y Arritmia Auricular, Una Tríada Olvidada
Ronda de Enfermedad Coronaria Infarto auricular, infarto de miocardio inferior y arritmia auricular, una tríada olvidada ID : LMFB, 66 years old, female, born and living in Pacatuba - Ceará, Brazil. Main complaint: “chest pain and shortness of breath” History of current disease: the patient informed about very intense constrictive precordial pain associated to nausea and vomits with delta-T (ΔT) of 4 hours (ΔT is the time of arrival of each patient to the Emergency Department). She informed about dyspnea to great and moderate strain for the last six months, with worsening after the onset of precordial pain. Personal pathological history: she mentioned high blood pressure and smoker for a long time. Stroke in 2009 with no sequelae. She denied having Diabetes mellitus, dyslipidemia or other risk factor. Physical examination: oriented, Glasgow scale 15. CPA: Split, regular heart rhythm, normal sounds, no murmurs. Systemic blood pressure: 169x78 mmHg, Heart Rate: 104 bpm. Pulmonary auscultation: vesicular murmur present, with no adventitious sounds. Respiratory rate: 29 rpm. It is decided to treat her with Primary Percutaneous Coronary Intervention (PPCI) and three stents where implanted. Questions: 1. Which is the “culprit” artery and obstruction location? And why? 2. What is the heart rhythm of the first ECG? 3. What is/are the mechanism(s) of P wave alterations? Sessão coronariana ID : L.M.F.B., 66 anos, natural e residente em Pacatuba-CE. Queixa Principal: “dor no peito e falta de ar” História da doença atual: paciente refere dor precordial de forte intensidade associada a náuseas e vômitos com delta-T de 4 horas. -
Carcinoid Case Presentation and Discussion: the American Perspective
Endocrine-Related Cancer (2003) 10 489–496 NEUROENDOCRINE TUMOURS Carcinoid case presentation and discussion: the American perspective R R P Warner Department of Medicine, Gastrointestinal Division, The Mount Sinai School of Medicine, One Gustave L Levy Place, New York, New York 10029, USA (Requests for offprints should be addressed toRRPWarner; Email: rwarner—[email protected]) Abstract The rationale underlying an aggressive approach in the management of some carcinoid patients is explained and illustrated by the presented case of a middle-aged man with advanced classic typical midgut carcinoid. The patient exhibited somatostatin receptor scintigraphy-positive massive liver metastases, carcinoid syndrome, severe tricuspid and pulmonic cardiac valve disease with congestive heart failure, ascites and malnutrition. He had been treated for several years with supportive medications and biotherapy including octreotide and alpha interferon but his tumor eventually progressed and his overall condition was markedly deteriorated when he first sought more aggressive treatment. This consisted of prompt replacement of both tricuspid and pulmonic valves, followed by hepatic artery chemoembolus (HACE) injection and then surgical tumor debulking including excision of the primary tumor in the small intestine. In addition, radiofrequency ablation was utilized to reduce the volume of metastases in the liver. Prophylactic cholecystectomy was also performed and a biopsy of tumor was submitted for cell culture drug resistance testing. This was followed by systemic chemotherapy utilizing the drug (docetaxel) which the in vitro studies suggested as most likely to be effective. His excellent response to this succession of treatments exemplifies the successful application of aggressive sequential multi-modality therapy. Endocrine-Related Cancer (2003) 10 489–496 Introduction and sometimes aggressive treatment (O¨ berg 1998). -
Systolic Murmurs
Murmurs and the Cardiac Physical Exam Carolyn A. Altman Texas Children’s Hospital Advanced Practice Provider Conference Houston, TX April 6 , 2018 The Cardiac Physical Exam Before applying a stethoscope….. Some pearls on • General appearance • Physical exam beyond the heart 2 Jugular Venous Distention Pallor Cyanosis 3 Work of Breathing Normal infant breathing Quiet Tachypnea Increased Rate, Work of Breathing 4 Beyond the Chest Clubbing Observed in children older than 6 mos with chronic cyanosis Loss of the normal angle of the nail plate with the axis of the finger Abnormal sponginess of the base of the nail bed Increasing convexity of the nail Etiology: ? sludging 5 Chest ❖ Chest wall development and symmetry ❖ Long standing cardiomegaly can lead to hemihypertrophy and flared rib edge: Harrison’s groove or sulcus 6 Ready to Examine the Heart Palpation Auscultation General overview Defects Innocent versus pathologic 7 Cardiac Palpation ❖ Consistent approach: palm of your hand, hypothenar eminence, or finger tips ❖ Precordium, suprasternal notch ❖ PMI? ❖ RV impulse? ❖ Thrills? ❖ Heart Sounds? 8 Cardiac Auscultation Where to listen: ★ 4 main positions ★ Inching ★ Ancillary sites: don’t forget the head in infants 9 Cardiac Auscultation Focus separately on v Heart sounds: • S2 normal splitting and intensity? • Abnormal sounds? Clicks, gallops v Murmurs v Rubs 10 Cardiac Auscultation Etiology of heart sounds: Aortic and pulmonic valves actually close silently Heart sounds reflect vibrations of the cardiac structures after valve closure Sudden