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Tyler Jacks
Tyler Jacks, Phd President, Break Through Cancer; Founding Director, Koch Institute for Integrative Cancer Research, Massachusetts Institute of Technology
Francisco J. Sánchez-Rivera
The Role of HIF-1Α in Sarcoma Metastasis and Response to Radiation Therapy
Susceptibility to Astrocytoma in Mice Mutant for Nf1 and Trp53 Is Linked to Chromosome 11 and Subject to Epigenetic Effects
The David H. Koch Institute for Integrative Cancer Research at MIT
Conference Program and Schedule
Development and Characterization of Immunogenic Genetically Engineered Mouse Models of Pancreatic Cancer
Ras Activation in Astrocytomas and Neurofibromas
3Rd Joint Meeting of BSA and NCAB
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Statement of Dr. Tyler Jacks, Phd Director, Koch Institute For
Conditional Mouse Lung Cancer Models Using Adenoviral Or Lentiviral Delivery of Cre Recombinase
Celebrating 40 Years of Rita Allen Foundation Scholars 1 PEOPLE Rita Allen Foundation Scholars: 1976–2016
Department of Health and Human Services Public
Wen Xue, Ph.D
Mutant P53 Drives Metastasis and Overcomes Growth Arrest/Senescence in Pancreatic Cancer
Ovarian Cancer Side Population Defines Cells with Stem Cell-Like Characteristics and Mullerian Inhibiting Substance Responsiveness
Targeting Cancer: an Interview with Tyler Jacks
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The Retinoblastoma Protein Tumor Suppressor Is Important for Appropriate Osteoblast Differentiation and Bone Development
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Research Highlights
CRISPR-Mediated Interrogation of Small Cell Lung Cancer
Astrocyte-Specific Expression of CDK4 Is Not Su Cient for Tumor Formation
P53-Independent Apoptosis Induced by Paclitaxel Through an Indirect Mechanism
A Mouse Model of MYCN-Driven Retinoblastoma Reveals MYCN-Independent Tumor Reemergence
Exploring Cell Apoptosis and Senescence to Understand and Treat Cancer: an Interview with Scott Lowe
An Organoid Platform to Study Alveolar Stem Cells in Lung Generation and Cancer
Cyclooxygenase-1 Is Overexpressed in Multiple Genetically Engineered Mouse Models of Epithelial Ovarian Cancer
Tyler Jacks Director, Koch Institute for Integrative Cancer Research David H
Mitochondrial Apoptotic Priming Is a Key Determinant of Cell Fate Upon P53 Restoration