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Sequestosome 1
Mtor: a Pharmacologic Target for Autophagy Regulation
SQSTM1 Mutations in Familial and Sporadic Amyotrophic Lateral Sclerosis
NF-B in Hematological Malignancies
Hydroxyurea Scavenges Free Radicals and Induces the Expression of Antioxidant Genes in Human Cell Cultures Treated with Hemin
Molecular Insights Into an Ancient Form of Paget's Disease of Bone
The Discovery and Evaluation of Inhibitors of the Keap1-Nrf2 Protein-Protein Interaction
Sequestosome-1 (SQSTM1) Sequence Variants in ALS Cases in the UK: Prevalence and Coexistence of SQSTM1 Mutations in ALS Kindred with PDB
Abstract Fibroblasts Can Be Directly Reprogrammed Into Induced
YOD1/TRAF6 Association Balances P62- Dependent IL-1 Signaling To
Dimerisation of the UBA Domain of P62 Inhibits Ubiquitin Binding and Regulates NF-Κb Signalling
The N-Terminus and Phe52 Residue of LC3 Recruit P62/SQSTM1 Into Autophagosomes
P62 Is Negatively Implicated in the TRAF6-BECN1 Signaling Axis for Autophagy Activation and Cancer Progression by Toll-Like Receptor 4 (TLR4)
ERBB3 and IGF1R Signaling Are Required for Nrf2-Dependent
Assessing Autophagy in Sciatic Nerves of a Rat Model That Develops Inflammatory Autoimmune Peripheral Neuropathies
Molecular Immunology 58 (2014) 27–31
TRAF6 Phosphorylation Prevents Its Autophagic Degradation and Re-Shapes LPS-Triggered Signaling Networks
Cellular Immunology Ubiquitination and Phosphorylation of The
Autocrine IFN Signaling Inducing Profibrotic Fibroblast Responses by a Synthetic TLR3 Ligand Mitigates
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Deficiency of P62/Sequestosome 1 Causes Hyperphagia Due to Leptin Resistance in the Brain
Nuclear Factor
Transcriptome Analysis Defines Myocardium Gene Signatures In
Proteome-Scale Amino-Acid Resolution Footprinting of Protein-Binding Sites
Novel TRAF6-Protein Interactions and the Molecular Mechanisms by Which They Regulate TRAF6 Epd Endent Signaling An-Jey Andrew Su
SQSTM1 Upregulation Constitutes a Survival Mechanism That Occurs During Granulocytic Differentiation of Acute Myeloid Leukemia Cells
The Regulation of NFE2L2 (NRF2) Signalling and Epithelial-To-Mesenchymal Transition in Age-Related Macular Degeneration Pathology
Filamentous Aggregation of Sequestosome-1/P62 in Brain Neurons and Neuroepithelial Cells Upon Tyr-Cre-Mediated Deletion of the Autophagy Gene Atg7
Sequestosome 1 /P62 and TRAF6 Serve As a Bridge to Connect IRS-1 with Akt in Insulin Signaling
Tumorigenesis in Tuberous Sclerosis Complex Is Autophagy and P62/Sequestosome 1 (SQSTM1)-Dependent
Autophagy Roles in Genome Maintenance
Autophagy Is an Important Cellular Process That Serves As a Com- Biochemical Reactions (16)
1 YOD1/TRAF6 Association Balances P62-Dependent IL-1 Signaling to NF-Κb
SQSTM1 Gene Sequestosome 1
N-3 Pufas Induce Inflammatory Tolerance by Formation of KEAP1
NRF2 Activation in Cancer: from DNA to Protein Erica W
NRF2, a Transcription Factor for Stress Response and Beyond
Mtor-Dependent Cell Survival Mechanisms
Molecular Insights Into Mechanisms of Hepatoblastoma Pathogenesis
Precision Autophagy Directed by Receptor Regulators – Emerging Examples Within the TRIM Family Tomonori Kimura*, Michael Mandell and Vojo Deretic*
The Pathways Underlying the Multiple Roles of P62 in Inflammation And
KLF4-SQSTM1/P62-Associated Prosurvival Autophagy Contributes to Carfilzomib Resistance in Multiple Myeloma Models
Natural Variation in the Sequestosome-Related Gene, Sqst-5, Underlies
Full-Text Version of This Article Contains a Data Supplement
Therapeutic Targeting of the NRF2 Signaling Pathway in Cancer
Variable IPSS DIPSS DIPSS-Plus
Rainbow Trout Red Blood Cells Exposed to Viral