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RIPK3
Necroptosis in Intestinal Inflammation and Cancer
Global Survey of Cell Death Mechanisms Reveals Metabolic Regulation of Ferroptosis
Regulation of Caspase-8 Activity at the Crossroads of Pro-Inflammation
Molecular Insights Into the Mechanism of Necroptosis: the Necrosome As a Potential Therapeutic Target
Caspase-8 Promotes C-Rel–Dependent Inflammatory Cytokine Expression and Resistance Against Toxoplasma Gondii
Conformational Interconversion of MLKL and Disengagement from RIPK3 Precede Cell Death by Necroptosis
Ninth UK Neuromuscular Translational Research Conference 22Nd – 23Rd March 2016
Network-Assisted Analysis of Primary Sjögren's Syndrome GWAS Data In
RIPK3 / RIP3 Antibody (Aa480-530) Rabbit Polyclonal Antibody Catalog # ALS11903
Mouse Ripk3 Conditional Knockout Project (CRISPR/Cas9)
RIPK3 Antibody (N-Term) Purified Rabbit Polyclonal Antibody (Pab) Catalog # Ap7184a
RIPK3 Antibody (C-Term) Affinity Purified Rabbit Polyclonal Antibody (Pab) Catalog # Ap14554b
1 LUBAC Is Essential for Embryogenesis by Preventing Cell
Deletion of Ripk3 Prevents Motor Neuron Death in Vitro but Not in Vivo
Shear Stress Associated with Cardiopulmonary Bypass Induces Expression of Inflammatory Cytokines and Necroptosis in Monocytes
RIP3: a Molecular Switch for Necrosis and Inflammation
A Missense Mutation in the MLKL Brace Region Promotes Lethal Neonatal Inflammation and Hematopoietic Dysfunction
RIPK3 Upregulation Confers Robust Proliferation and Collateral Cystine
Top View
Salmonella Enterica Serovar Typhimurium Infection in Mouse Pregnancy
Mutations That Prevent Caspase Cleavage of RIPK1 Cause Autoinflammatory Disease
The Role of RIPK3 Ubiquitylation and MLKL Signalling During Cell Death and Autophagy
A Missense Mutation in the MLKL Brace Region Promotes Lethal Neonatal Inflammation and Hematopoietic Dysfunction