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PARL
Mitochondrial Rhomboid PARL Regulates Cytochrome C Release During Apoptosis Via OPA1-Dependent Cristae Remodeling
PARL (Human) Recombinant Protein (P01)
Genetic Correction of HAX1 in Induced Pluripotent Stem Cells from a Patient with Severe Congenital Neutropenia Improves Defective Granulopoiesis
Human CLPB) Is a Potent Mitochondrial Protein Disaggregase That Is Inactivated By
The Incomplete Puzzle of the BCL2 Proteins
Insights Into the Catalytic Properties of the Mitochondrial Rhomboid
PARL Antibody Cat
Characterization of PARL-Mediated Intramembrane Proteolysis
Skd3 (Human Clpb) Is a Potent Mitochondrial Protein Disaggregase That Is Inactivated by 3-Methylglutaconic Aciduria-Linked Mutations Ryan R Cupo1,2, James Shorter1,2*
Loss of Parla Function Results in Inactivity, Olfactory Impairment, and Dopamine Neuron Loss in Zebrafish
Mutations in CLPB Cause Intellectual Disability, Congenital Neutropenia
PARL Deficiency in Mouse Causes Complex III Defects, Coenzyme Q Depletion, and Leigh-Like Syndrome
Mitochondrial Dynamics, ROS, and Cell Signaling: a Blended Overview
The BCL-2 Protein Family: Opposing Activities That Mediate Cell Death
Heterozygous Variants of CLPB Are a Cause of Severe Congenital Neutropenia
PARL Leu262val Is Not Associated with Fasting Insulin Levels in UK Populations
Structural and Mechanistic Basis of Parl Activity and Regulation
Association of PARL Gene Rs3732581, Rs73887537
Top View
Integrated Gene Expression Analysis of Controls Across Datasets Available of the Genes Implicated in Type 2 Diabetes Mellitus Complications
PINK1 Autophosphorylation Upon Membrane Potential Dissipation Is Essential for Parkin Recruitment to Damaged Mitochondria
Snapshot: Mitochondrial Dynamics
Activation of Mitochondrial Protease OMA1 by Bax and Bak Promotes Cytochrome C Release During Apoptosis
Atypical Protein Phosphatases: Emerging Players in Cellular Signaling
Rhomboid Proteins: Conserved Membrane Proteases with Divergent Biological Functions
Identification of Novel Genomic Imbalances in Saudi Patients with Congenital Heart Disease Zuhair N
MOMP, Cell Suicide As a BCL-2 Family Business
Kill One Or Kill the Many: Interplay Between Mitophagy and Apoptosis
Genetic Association of the PARL-ABCC5-HTR3D-HTR3C Locus with Primary Angle-Closure Glaucoma in Chinese
PINK1 Import Regulation; a Fine System to Convey Mitochondrial Stress to the Cytosol Shiori Sekine and Richard J
1 Mitochondrial Protease PARL Mediates the Processing of the Cell
PARL Antibody Catalog # ASC10749
BF0639-PARL Antibody
A Bridge Between Mitochondria and Parkinson's Disease
Mitochondrial Membrane Potential Regulates PINK1 Import and Proteolytic Destabilization by PARL
A Zebrafish Model for HAX1-Associated Congenital Neutropenia Ferrata Storti Foundation
Submission Disease Characterized by Disrupted Biology Study Proposedpdf That Parlexerts Pro-Apoptotic Effects Via 94 27 Energy Production