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NDUFS4
High-Throughput, Pooled Sequencing Identifies Mutations in NUBPL And
Proteomic and Metabolomic Analyses of Mitochondrial Complex I-Deficient
Low Abundance of the Matrix Arm of Complex I in Mitochondria Predicts Longevity in Mice
Novel NDUFS4 Gene Mutation in an Atypical Late-Onset Mitochondrial Form of Multifocal Dystonia Celine Bris, Tiphaine Rouaud, Valerie Desquiret-Dumas, Et Al
Leigh Syndrome
Impaired Mitochondrial Complex I Function As a Candidate Driver in The
A Novel Mutation in NDUFS4 Causes Leigh Syndrome in an Ashkenazi Jewish Family
Accessory Subunits Are Integral for Assembly and Function of Human Mitochondrial Complex I
Analysis of Human Mutations in the Supernumerary Subunits of Complex I
Mitochondrial Complex I Inhibition Is Not Required for Dopaminergic Neuron Death Induced by Rotenone, MPP؉, Or Paraquat
Mitochondrial DNA (Mtdna) Test Requisition
Leigh Syndrome Associated with Mitochondrial Complex I Deficiency Due to a Novel Mutation in the NDUFS1 Gene
An NAD+ Dependent/Sensitive Transcription System: Toward a Novel Anti-Cancer Therapy
Mitochondrial Complex I Deficiency
Mutant NDUFS3 Subunit of Mitochondrial Complex I Causes Leigh Syndrome
Diagnosis and Management of Mitochondrial Disease: a Consensus Statement from the Mitochondrial Medicine Society
The Genetic Evidence of Burn-Induced Cardiac Mitochondrial Metabolism Dysfunction
Low Abundance of NDUFV2 and NDUFS4 Subunits of the Hydrophilic Complex I Domain and VDAC1 Predicts Mammalian Longevity T
Top View
Comprehensive Diagnosis for Mitochondrial Disorders
Metabolic Consequences of NDUFS4 Gene Deletion in Immortalized Mouse Embryonic fibroblasts☆
Fatal Breathing Dysfunction in a Mouse Model of Leigh Syndrome
Gene Replacement Therapy Provides Benefit in an Adult Mouse Model of Leigh Syndrome
FOXRED1 Silencing in Mice: a Possible Animal Model for Leigh Syndrome
Genequery™ Human Parkinson's Disease Qpcr Array Kit (GQH-PKD)
The NDUFS4 Nuclear Gene of Complex I of Mitochondria and the Camp Cascade
NDUFA10 Mutations Cause Complex I Deficiency in a Patient with Leigh Disease
Investigation Into the Effects and Mechanisms of Rapamycin Treatment
Targeting NAD+ Metabolism As Interventions for Mitochondrial Disease Received: 25 October 2018 Chi Fung Lee1,2,5, Arianne Caudal1,3, Lauren Abell1,4, G